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Journal articles on the topic 'HipBA'

Author: Grafiati

Published: 4 June 2021

Last updated: 27 April 2022

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1

Correia, Frederick F., Anthony D'Onofrio, Tomas Rejtar, et al. "Kinase Activity of Overexpressed HipA Is Required for Growth Arrest and Multidrug Tolerance in Escherichia coli." Journal of Bacteriology 188, no. 24 (2006): 8360–67. http://dx.doi.org/10.1128/jb.01237-06.

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ABSTRACT Overexpression of the HipA protein of the HipBA toxin/antitoxin module leads to multidrug tolerance in Escherichia coli. HipA is a “toxin” that causes reversible dormancy, whereas HipB is an antitoxin that binds HipA and acts as a transcriptional repressor of the hipBA operon. Comparative sequence analysis shows that HipA is a member of the phosphatidylinositol 3/4-kinase superfamily. The kinase activity of HipA was examined. HipA was autophosphorylated in the presence of ATP in vitro, and the purified protein appeared to carry a single phosphate group on serine 150. Thus, HipA is a s

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2

Korch, Shaleen B., and Thomas M. Hill. "Ectopic Overexpression of Wild-Type and Mutant hipA Genes in Escherichia coli: Effects on Macromolecular Synthesis and Persister Formation." Journal of Bacteriology 188, no. 11 (2006): 3826–36. http://dx.doi.org/10.1128/jb.01740-05.

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ABSTRACT Persistence is an epigenetic trait that allows a small fraction of bacteria, approximately one in a million, to survive prolonged exposure to antibiotics. In Escherichia coli an increased frequency of persisters, called “high persistence,” is conferred by mutations in the hipA gene, which encodes the toxin entity of the toxin-antitoxin module hipBA. The high-persistence allele hipA7 was originally identified because of its ability to confer high persistence, but little is known about the physiological role of the wild-type hipA gene. We report here that the expression of wild-type hip

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3

Lin, Chun-Yi, Sanya Hamini, Peter Robert Tupa, and Hisako Masuda. "Cellular Memory of HipA-Induced Growth Arrest: The Length of Cell Growth Arrest Becomes Shorter for Each Successive Induction." Microorganisms 9, no. 12 (2021): 2594. http://dx.doi.org/10.3390/microorganisms9122594.

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Toxin–antitoxin (TA) systems are genetic modules found commonly in bacterial genomes. HipA is a toxin protein encoded from the hipBA TA system in the genome of Escherichia coli. Ectopic expression of hipA induces cell growth arrest. Unlike the cell growth arrest caused by other TA toxins, cells resume growth from the HipA-induced cell growth arrest phase after a defined period of time. In this article, we describe the change in the length of growth arrest while cells undergo repeated cycles of hipA induction, growth arrest and regrowth phases. In the multiple conditions tested, we observed tha

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4

Germain, Elsa, Mohammad Roghanian, Kenn Gerdes, and Etienne Maisonneuve. "Stochastic induction of persister cells by HipA through (p)ppGpp-mediated activation of mRNA endonucleases." Proceedings of the National Academy of Sciences 112, no. 16 (2015): 5171–76. http://dx.doi.org/10.1073/pnas.1423536112.

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The model organismEscherichia colicodes for at least 11 type II toxin–antitoxin (TA) modules, all implicated in bacterial persistence (multidrug tolerance). Ten of these encode messenger RNA endonucleases (mRNases) inhibiting translation by catalytic degradation of mRNA, and the 11th module,hipBA, encodes HipA (high persister protein A) kinase, which inhibits glutamyl tRNA synthetase (GltX). In turn, inhibition of GltX inhibits translation and induces the stringent response and persistence. Previously, we presented strong support for a model proposing (p)ppGpp (guanosine tetra and penta-phosph

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5

Keren, Iris, Devang Shah, Amy Spoering, Niilo Kaldalu, and Kim Lewis. "Specialized Persister Cells and the Mechanism of Multidrug Tolerance in Escherichia coli." Journal of Bacteriology 186, no. 24 (2004): 8172–80. http://dx.doi.org/10.1128/jb.186.24.8172-8180.2004.

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ABSTRACT Bacterial populations produce persisters, cells that neither grow nor die in the presence of bactericidal agents, and thus exhibit multidrug tolerance (MDT). The mechanisms of MDT and the nature of persisters have remained elusive. Our previous research has shown that persisters are largely responsible for the recalcitrance of biofilm infections. A general method for isolating persisters was developed, based on lysis of regular cells by ampicillin. A gene expression profile of persisters contained toxin-antitoxin (TA) modules and other genes that can block important cellular functions

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6

Schumacher, Maria A., Pooja Balani, Jungki Min, et al. "HipBA–promoter structures reveal the basis of heritable multidrug tolerance." Nature 524, no. 7563 (2015): 59–64. http://dx.doi.org/10.1038/nature14662.

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7

Evdokimov, Artem, Igor Voznesensky, Kimberly Fennell, Marie Anderson, James F. Smith, and Douglas A. Fisher. "New kinase regulation mechanism found in HipBA: a bacterial persistence switch." Acta Crystallographica Section D Biological Crystallography 65, no. 8 (2009): 875–79. http://dx.doi.org/10.1107/s0907444909018800.

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8

Hansen, Sonja, Marin Vulić, Jungki Min, et al. "Regulation of the Escherichia coli HipBA Toxin-Antitoxin System by Proteolysis." PLoS ONE 7, no. 6 (2012): e39185. http://dx.doi.org/10.1371/journal.pone.0039185.

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9

Klimkaitė, Laurita, Julija Armalytė, Jūratė Skerniškytė, and Edita Sužiedėlienė. "The Toxin-Antitoxin Systems of the Opportunistic Pathogen Stenotrophomonas maltophilia of Environmental and Clinical Origin." Toxins 12, no. 10 (2020): 635. http://dx.doi.org/10.3390/toxins12100635.

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Stenotrophomonas maltophilia is a ubiquitous environmental bacterium that has recently emerged as a multidrug-resistant opportunistic pathogen causing bloodstream, respiratory, and urinary tract infections. The connection between the commensal environmental S. maltophilia and the opportunistic pathogen strains is still under investigation. Bacterial toxin–antitoxin (TA) systems have been previously associated with pathogenic traits, such as biofilm formation and resistance to antibiotics, which are important in clinical settings. The same species of the bacterium can possess various sets of TA

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10

KAWANO, Hiroaki, Yasutaka HIROKAWA, and Hideo MORI. "Long-Term Survival ofEscherichia coliLacking the HipBA Toxin-Antitoxin System during Prolonged Cultivation." Bioscience, Biotechnology, and Biochemistry 73, no. 1 (2009): 117–23. http://dx.doi.org/10.1271/bbb.80531.

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11

Yadav, Mohit, and Jitendra Singh Rathore. "Functional and transcriptional analysis of chromosomal encoded hipBA type II toxin-antitoxin (TA) module from Xenorhabdus nematophila." Microbial Pathogenesis 162 (January 2022): 105309. http://dx.doi.org/10.1016/j.micpath.2021.105309.

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12

Spoering, Amy L., Marin Vulić, and Kim Lewis. "GlpD and PlsB Participate in Persister Cell Formation in Escherichia coli." Journal of Bacteriology 188, no. 14 (2006): 5136–44. http://dx.doi.org/10.1128/jb.00369-06.

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ABSTRACT Bacterial populations produce dormant persister cells that are resistant to killing by all antibiotics currently in use, a phenomenon known as multidrug tolerance (MDT). Persisters are phenotypic variants of the wild type and are largely responsible for MDT of biofilms and stationary populations. We recently showed that a hipBA toxin/antitoxin locus is part of the MDT mechanism in Escherichia coli. In an effort to find additional MDT genes, an E. coli expression library was selected for increased survival to ampicillin. A clone with increased persister production was isolated and was

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13

Kelly, Curly. "HIPAA Compliance: Lessons from the Repeal of Hawaii's Patient Privacy Law." Journal of Law, Medicine & Ethics 30, no. 2 (2002): 309–12. http://dx.doi.org/10.1111/j.1748-720x.2002.tb00399.x.

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In 1996, Congress passed the Health Insurance Portability and Accountability Act (HIPAA), which required the enactment of new regulations to protect confidential patient health information. In December 2000, the U.S. Department of Health and Human Services (DHHS) published the agency's final rule on patient privacy and the proper use of privileged health information. The HIPAA privacy regulations cover all health-care providers that handle medical records or other identifiable patient health information. Most health-care organizations have until April 14,2003, to comply with HIPPA.

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14

Semanjski, Maja, Elsa Germain, Katrin Bratl, Andreas Kiessling, Kenn Gerdes, and Boris Macek. "The kinases HipA and HipA7 phosphorylate different substrate pools inEscherichia colito promote multidrug tolerance." Science Signaling 11, no. 547 (2018): eaat5750. http://dx.doi.org/10.1126/scisignal.aat5750.

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The bacterial serine-threonine protein kinase HipA promotes multidrug tolerance by phosphorylating the glutamate-tRNA ligase (GltX), leading to a halt in translation, inhibition of growth, and induction of a physiologically dormant state (persistence). The HipA variant HipA7 substantially increases persistence despite being less efficient at inhibiting cell growth. We postulated that this phenotypic difference was caused by differences in the substrates targeted by both kinases. We overproduced HipA and HipA7 inEscherichia coliand identified their endogenous substrates by SILAC-based quantitat

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15

Harrison, Joe J., William D. Wade, Sarah Akierman, et al. "The Chromosomal Toxin Gene yafQ Is a Determinant of Multidrug Tolerance for Escherichia coli Growing in a Biofilm." Antimicrobial Agents and Chemotherapy 53, no. 6 (2009): 2253–58. http://dx.doi.org/10.1128/aac.00043-09.

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ABSTRACT Escherichia coli is refractory to elevated doses of antibiotics when it is growing in a biofilm, and this is potentially due to high numbers of multidrug-tolerant persister cells in the surface-adherent population. Previously, the chromosomal toxin-antitoxin loci hipBA and relBE have been linked to the frequency at which persister cells occur in E. coli populations. In the present study, we focused on the dinJ-yafQ-encoded toxin-antitoxin system and hypothesized that deletion of the toxin gene yafQ might influence cell survival in antibiotic-exposed biofilms. By using confocal laser s

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16

Li, Chaoqun, Yaru Wang, Yan Wang, and Guangju Chen. "Interaction investigations of HipA binding to HipB dimer and HipB dimer + DNA complex: a molecular dynamics simulation study." Journal of Molecular Recognition 26, no. 11 (2013): 556–67. http://dx.doi.org/10.1002/jmr.2300.

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17

Schumacher, M. A., K. M. Piro, W. Xu, S. Hansen, K. Lewis, and R. G. Brennan. "Molecular Mechanisms of HipA-Mediated Multidrug Tolerance and Its Neutralization by HipB." Science 323, no. 5912 (2009): 396–401. http://dx.doi.org/10.1126/science.1163806.

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18

HIPPA, HEIKKI. "New species and new records of Manota Williston (Diptera, Mycetophilidae) from Thailand, with a key to the Oriental and Palaearctic species." Zootaxa 2763, no. 1 (2011): 39. http://dx.doi.org/10.11646/zootaxa.2763.1.3.

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The following new species are described: Manota aquila, M. falcata, M. flammula and M. subcollina. The following are reported as new species for the Thailand fauna: M. calcarata Hippa, M. clausa Hippa, M. curvata Hippa, M. duplex Hippa, M. fera Hippa, M. ferrata Hippa, M. horrida Hippa, M. perangulata Hippa & Ševčík, M. pollex Hippa, and M. transversa Hippa. New records within Thailand are given of the following species: M. aconcinna Hippa, M. acutangula Hippa, M. ancylochaeta Hippa, M. biunculata Hippa, M. dentata Hippa & Papp, M. epigrata Hippa, M. globigera Hippa, M. heptacantha Hip

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19

HIPPA, HEIKKI. "New species and new records of Manota Williston (Diptera, Mycetophilidae) from Thailand." Zootaxa 2017, no. 1 (2009): 1–33. http://dx.doi.org/10.11646/zootaxa.2017.1.1.

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The following 10 new species are described: Manota avita, M. chelapex, M. chi, M. epigrata, M. obtecta, M. prisca, M. seducta, M. subferrata, M. tetrachaeta, and M. vesicaria. New records of the following species are given: Manota aconcinna Hippa, M. acutangula Hippa, M. ancylochaeta Hippa, M. clavulosa Hippa, M. collina Hippa, M. cristata Hippa, M. globigera Hippa, M. inflata Hippa, M. oblonga Hippa, M. oligochaeta Hippa, M. ovata Hippa, M. pectinata Hippa, M. pellii Hippa, M. perlobata Hippa, M. perpusilla Hippa, M. planilobata Hippa, M. procera Hippa, M. roslii Hippa, and M. simplex Hippa.

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20

HIPPA, HEIKKI, and LÁSZLÓ PAPP. "The genus Manota Williston (Diptera: Mycetophilidae) in Thailand, with the description of seven new species." Zootaxa 1528, no. 1 (2007): 41–60. http://dx.doi.org/10.11646/zootaxa.1528.1.2.

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Seven new species of Manota Williston are described from Thailand: M. bifida, M. dentata, M. forceps, M. inusitata, M. mirifica, M. occulta, and M. secreta. Nine Manota species, which were recently described from West Malaysia, are here recorded as new for the fauna of Thailand: M. heptacantha Hippa, M. oligochaeta Hippa, M. ovata Hippa, M. pectinata Hippa, M. perpusilla Hippa, M. plusiochaeta Hippa, M. procera Hippa, M. roslii Hippa, and M. ulu Hippa. M. biunculata Hippa, which was recently described from New Guinea, is also recorded from Thailand. The number of the Oriental species of Manota

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21

HIPPA, HEIKKI, OLAVI KURINA, and ILARI E. SÄÄKSJÄRVI. "The genus Manota Williston (Diptera: Mycetophilidae) in Peruvian Amazonia, with description of sixteen new species and notes on local species richness." Zootaxa 4236, no. 1 (2017): 1. http://dx.doi.org/10.11646/zootaxa.4236.1.1.

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A comprehensive study of material of the worldwide fungus gnat genus Manota Williston, sampled from the Allpahuayo-Mishana National Reserve in Peruvian Amazonia, was conducted. The following 16 species are described as new: M. aligera sp. n., M. aristoseta sp. n., M. calva sp. n., M. ciliata sp. n., M. exigua sp. n., M. digitata sp. n., M. flabellata sp. n., M. iquitosensis sp. n., M. limulata sp. n., M. micella sp. n., M. minutula sp. n., M. nuda sp. n., M. parvula sp. n., M. pauloides sp. n., M. pustulosa sp. n. and M. serrulata sp. n. In addition, the following 16 species are recorded: M. a

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22

Hiatt, Robert A. "HIPAA." Epidemiology 14, no. 6 (2003): 637–739. http://dx.doi.org/10.1097/01.ede.0000092151.80136.ce.

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23

Helmholdt, Robert D. "Hipaa." American Journal of Orthodontics and Dentofacial Orthopedics 124, no. 3 (2003): A17. http://dx.doi.org/10.1016/s0889-5406(03)00632-2.

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24

Berson, Susan W. "HIPAA." Oncology Issues 18, no. 1 (2003): 20. http://dx.doi.org/10.1080/10463356.2003.11883103.

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25

Powell, Suzanne K. "HIPAA." Lippincott's Case Management 8, no. 1 (2003): 1–2. http://dx.doi.org/10.1097/00129234-200301000-00001.

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26

Muller, Lynn S. "HIPAA." Lippincott's Case Management 9, no. 1 (2004): 27–31. http://dx.doi.org/10.1097/00129234-200401000-00006.

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27

Gips, Alexa H. "HIPAA." Annals of Emergency Medicine 71, no. 6 (2018): 787. http://dx.doi.org/10.1016/j.annemergmed.2017.11.029.

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28

Mohrig, Werner, and Frank Menzel. "Revision der neotropischen Trauermücken – Teil I. Die Gattungen Cratyna WINNERTZ, Euricrium ENDERLEIN, Metangela RÜBSAAMEN, Pseudosciara SCHINER und Sciara MEIGEN (Diptera: Sciaridae)." Beiträge zur Entomologie = Contributions to Entomology 64, no. 1 (2014): 135–90. http://dx.doi.org/10.21248/contrib.entomol.64.1.135-190.

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Die vorliegende Revision umfasst 40 Arten aus den Gattungen Cratyna Winnertz, 1867 [4 Arten], Euricrium Enderlein, 1911 [8 Arten], Metangela Rübsaamen, 1894 [4 Arten], Pseudosciara Schiner, 1866 [21 Arten] und Sciara Meigen, 1803 [3 Arten]. Die Arten werden redeskribiert und abgebildet. 63 Spezies wurden neu kombiniert und 4 Namen als neue Synonyma auf dem Artniveau erkannt: Plastosciara barretoi Lane, 1960 als neues Synonym zu Cratyna boracensis (Lane, 1960); Plastosciara downsi Lane, 1960 als neues Synonym zu Cratyna cucarisi (Lane, 1960), Euricrium fulgescens Enderlein, 1911 als neues Synon

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29

HIPPA, HEIKKI, and JAN ŠEVČÍK. "Notes on Oriental and Australasian Manotinae (Diptera, Mycetophilidae), with the description of thirteen new species." Zootaxa 2333, no. 1 (2010): 1. http://dx.doi.org/10.11646/zootaxa.2333.1.1.

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The following new species are described: Manota acehensis (Sumatra), M. anceps (Sumatra), M. bruneiensis (Borneo), M. capillata (Sumatra), M. dolichothrix (Borneo), M. hexacantha (Borneo), M. hyboloma (Borneo), M. perangulata (Borneo), M. radula (Borneo), M. sinepollex (Sumatra), M. stricta (Sumatra), M. subforceps (Sumatra) and Eumanota vilkamaai (New Guinea). New records of the following species are given: Manota bifida Hippa & Papp (Borneo), M. clausa Hippa (Borneo), M. curvata Hippa (Sumatra), M. ferrata Hippa (Borneo), M. forceps Hippa & Papp (Thailand), M. horrida Hippa (Borneo),

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30

HIPPA, HEIKKI, GEIR SØLI, and OLAVI KURINA. "New data on the genus Manota Williston (Diptera: Mycetophilidae) from Africa, with an updated key to the species." Zootaxa 4652, no. 3 (2019): 401–41. http://dx.doi.org/10.11646/zootaxa.4652.3.1.

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A total of 346 male specimens of Manota collected from 13 countries in Africa are studied. They belong to 40 different species including 12 new to science. The new species are: M. burundiensis (Burundi), M. cornuta (Ghana), M. fuscinula (Ghana), M. geniculata (Gabon), M. kirkspriggsi (Madagascar), M. kjaerandseni (Ghana, Côte d’Ivoire), M. leptochaeta (Madagascar), M. limai (São Tomé and Principe), M. oronnai (Nigeria), M. platychaeta (Madagascar), M. polylobata (Nigeria) and M. triseta (Ghana, Guinea). New records of the following 28 species are presented: M. aculifera Hippa & Kurina, 201

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31

KURINA, OLAVI, and HEIKKI HIPPA. "Additions to the knowledge of Manota Williston (Diptera: Mycetophilidae) from the Neotropical region, with description of four new species." Zootaxa 4938, no. 1 (2021): 85–100. http://dx.doi.org/10.11646/zootaxa.4938.1.4.

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The Neotropical species of the genus Manota Williston are studied, based on material of 146 specimens from French Guiana, Ecuador, Nicaragua, Dominica and the Dominican Republic. Four new species are described, viz. M. corniculata sp. n. (French Guiana), M. pseudocavata sp. n. (French Guiana), M. truuverki sp. n. (French Guiana) and M. vladi sp. n. (Dominican Republic). Manota defecta Williston, 1896, the type species of the genus, is listed from Dominica, representing the first record since its description more than a century ago from a Southern Caribbean Island, St. Vincent. New records of 1

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32

Jaschhof, Mathias, and Heikki Hippa. "Pseudoperomyia gen. n. from Malaysia and the phylogeny of the Micromyidi (Diptera: Cecidomyiidae, Lestremiinae)." Beiträge zur Entomologie = Contributions to Entomology 49, no. 1 (1999): 147–71. http://dx.doi.org/10.21248/contrib.entomol.49.1.147-171.

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Die Gallmücken-Gattung Pseudoperomyia gen. n. wird für 8 Lestremiinen-Arten aus Malaysia begründet. Die neubeschriebenen Arten sind: P. acutistyla sp. n., P. bidentata sp. n., P. humilis sp. n., P. parvolobata sp. n., P. intermedia sp. n., P. longicornis sp. n., P. macrostyla sp. n. und P. platistyla sp. n. Innerhalb der Tribus Micromyini ist Anodontoceras die Schwestergruppe von Pseudoperomyia. Das Resultat einer Computeranalyse unterstützt weitgehend eine jüngst vorgeschlagene Hypothese zur Phylogenie der Micromyidi.Nomenklatorische HandlungenPseudoperomyia Jaschhof & Hippa, 1999 (Cecido

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33

KURINA, OLAVI, HEIKKI HIPPA, and DALTON DE SOUZA AMORIM. "Notes on Manota Williston (Diptera: Mycetophilidae) from Australia and Papua New Guinea, with description of two new species." Zootaxa 4555, no. 3 (2019): 385. http://dx.doi.org/10.11646/zootaxa.4555.3.7.

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Two new species, Manota williamsi sp. n. and Manota kerri sp. n., are described from Australia and Papua New Guinea, respectively. The former represents the second Manota species recorded from continental Australia. Characterised by setose anepisternum and non-setose laterotergite, M. williamsi is similar to M. gemella Hippa, 2007, but the presence of the mid tibial organ would group it together with five species from New Zealand. Manota kerri resembles M. alulata Kurina & Hippa, 2015 in having a bilobed gonostylus and sternite 9 entirely fused with the gonocoxa, but differs by other detai

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34

Lovitch, Karen S., and Stephen R. Bentfield. "HIPAA Compliance." Oncology Issues 20, no. 4 (2005): 16. http://dx.doi.org/10.1080/10463356.2005.11884246.

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Grimes, Stephen L. "HIPAA Update." Journal of Clinical Engineering 27, no. 4 (2002): 250. http://dx.doi.org/10.1097/00004669-200202740-00007.

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Grimes, Stephen L. "HIPAA Update." Journal of Clinical Engineering 28, no. 2 (2003): 82–83. http://dx.doi.org/10.1097/00004669-200304000-00009.

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Grimes, Stephen L. "HIPAA Update." Journal of Clinical Engineering 29, no. 1 (2004): 26. http://dx.doi.org/10.1097/00004669-200401000-00035.

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38

Gallagher, Lisa A. "REVISITING HIPAA." Nursing Management (Springhouse) 41, no. 4 (2010): 34–39. http://dx.doi.org/10.1097/01.numa.0000370876.71090.03.

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&NA;. "REVISITING HIPAA." Nursing Management (Springhouse) 41, no. 4 (2010): 39–40. http://dx.doi.org/10.1097/01.numa.0000370877.48219.c8.

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40

Muller, Lynn S. "HIPAA Compliance." Lippincott's Case Management 8, no. 1 (2003): 30–35. http://dx.doi.org/10.1097/00129234-200301000-00006.

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41

Madsen, E. "HIPAA Possumus." Journal of the American Medical Informatics Association 10, no. 3 (2003): 294. http://dx.doi.org/10.1197/jamia.m1355.

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42

Prince, Lori H., Amanda Carroll-Barefield, and Sherry P. Smith. "HIPAA Compliance." Health Care Manager 23, no. 1 (2004): 31–39. http://dx.doi.org/10.1097/00126450-200401000-00007.

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Kiel, Joan M. "HIPAA: SOP." Health Care Manager 29, no. 1 (2010): 80–82. http://dx.doi.org/10.1097/hcm.0b013e3181cd8bb1.

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Sylvan, Sheila E. "HIPAA 5010." Cardiac Cath Lab Director 1, no. 5-6 (2011): 144. http://dx.doi.org/10.1177/2150133512439292.

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Schneider, Carl. "HIPAA-cracy." Hastings Center Report 36, no. 1 (2006): 10–11. http://dx.doi.org/10.1353/hcr.2006.0016.

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&NA;. "Understanding HIPAA." Hearing Journal &NA; (October 2002): 6. http://dx.doi.org/10.1097/01.hj.0000292888.20199.3e.

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Salladay, Susan A. "HIPAA havoc." Nursing 38, no. 2 (2008): 8. http://dx.doi.org/10.1097/01.nurse.0000309703.16617.57.

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&NA;. "Understanding HIPAA." Nursing 38, no. 9 (2008): 64. http://dx.doi.org/10.1097/01.nurse.0000334659.43444.36.

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