Relevant bibliographies by topics / Histamine

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Academic literature on the topic 'Histamine'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 June 2025

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Journal articles on the topic "Histamine"

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Chen, M., and L. A. Brown. "Histamine stimulation of surfactant secretion from rat type II pneumocytes." American Journal of Physiology-Lung Cellular and Molecular Physiology 258, no. 4 (1990): L195—L200. http://dx.doi.org/10.1152/ajplung.1990.258.4.l195.

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The present study examined the effect of histamine on the secretion of phosphatidylcholine, the predominant component of pulmonary surfactant from adult rat alveolar type II pneumocytes in primary culture. Histamine stimulated surfactant secretion in a time- and dose-dependent manner. At a concentration of 10 microM, histamine stimulated surfactant release by 5.2-fold over the basal secretory rate. The concentration producing half the maximal response for histamine-induced secretion was 70 nM. Histamine-induced secretion was blocked by both the selective histamine1 receptor antagonist pyrilamine and the selective histamine2 receptor antagonist cimetidine, but not by the beta-adrenergic antagonist alprenolol. Histamine activated adenylate cyclase and intracellular adenosine 3',5'-cyclic monophosphate (cAMP) production. These effects on adenylate cyclase and cAMP induced by histamine were inhibited by the H2 antagonist cimetidine. This would suggest that surfactant secretion was stimulated by histamine through H2 receptors and by cAMP. When histamine and isoproterenol were added concomitantly, the effects on phosphatidylcholine secretion were additive; however, there was no additive effect on adenylate cyclase activation. This coupled with H2 antagonist pyrilamine inhibition of histamine-induced secretion would suggest that histamine stimulated surfactant secretion through another mechanism in addition to a cAMP-dependent mechanism.
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Kaunsui, Marledi, Charles Masinambou, Ella Dertina Saragih, et al. "Efek Penambahan Ekstrak Daun Tagalolo (Ficus Septica Burm. F) terhadap Kadar Histamin dan Total Bakteri Ikan Cakalang (Katsuwonus pelamis L)." Media Teknologi Hasil Perikanan 11, no. 1 (2023): 51–58. https://doi.org/10.35800/mthp.11.1.2023.54138.

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Histamine is one of the important parameters in determining the quality of fish quality, especially in fishery products that will be exported. This study aims to determine the effect of adding tagalolo leaf water extract on histamine levels and total plate counts in fresh skipjack (Katsuwonus pelamis L). The results showed that tagalolo leaves have the potential to slow down the rate of histamine and bacterial development in fish Keyword: skipjack, histamine, tagalolo leaves, total plate count Histamin merupakan salah satu parameter penting dalam penentuan kualitas mutu ikan terutama pada produk perikanan yang akan diekspor. Penelitian ini bertujuan untuk mengetahui efek penambahan ekstrak air daun tagalolo terhadap kadar histamin dan angka lempeng total pada ikan cakalang (Katsuwonus pelamis L) segar. Hasil penelitian menunjukkan bahwa daun tagalolo memiliki potensi untuk dapat memperlambat laju perkembangan histamin dan bakteri pada ikan. Kata kunci: ikan, histamine, daun tagalolo, angka lempeng total
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Bilovol, A. M., S. G. Tkachenko, and A. A. Berehova. "COMPARATIVE CHARACTERISTICS OF INDICATORS OF NEUROENDOCRINE REGULATION IN PATIENTS WITH LICHENOID DERMATOSES." Dermatology and Venerology, no. 1-2 (2022): 14–16. http://dx.doi.org/10.33743/2308-1066-2022-1-2-14-16.

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The aim is to study the indicators of neuroendocrine regulation in patients with chronic lichenoid dermatoses. Materials and methods. Studies of neuroendocrine system indicators were performed in two groups of patients: 1–60 patients with psoriasis, 2–56 patients with lichen planus, the control group consisted of 15 healthy individuals, whose indicators were considered normal. Determination of serotonin, histamine, histaminase, corticotropin, adrenaline in the serum of patients was performed by enzyme-linked immunosorbent assay. Results. There was a reliable increase in mean serum serotonin levels relative to control in both patients with psoriasis (228%) and patients with lichen planus (182%), histamine (85% and 76%, respectively); reliable decrease in serum histaminase level by 35% in patients with psoriasis and by 33% in patients with lichen planus relative to control and significant increase in histamine / histaminase index by 167% and 142%, respectively; reliable increase in serum corticotropin levels relative to control by 179% in patients with psoriasis and by 31% in patients with lichen planus. The level of adrenaline also significantly exceeded the control values by 43% in patients with psoriasis and by 37% in patients with lichen planus. Analysis of the average value of the adrenaline / serotonin ratio showed a decrease in its relative control by 57% in patients with psoriasis and by 52% in patients with lichen planus. Conclusions. Unidirectional changes in biochemical, enzymatic and hormonal constants of ergo- and trophotropic systems in both psoriasis and lichen planus have been reported, with the predominance of histamine biosynthesis over its inactivation and the prevalence of serotonergic system over adrenergic one, which was accompanied by significant increase in serum level of serotonin, histamin, corticotropin and adrenalin relative to control and a decrease in histaminase and the average adrenaline / serotonin ratio.
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Wodi, Stevy Imelda, and Eko Cahyono. "KAJIAN TOTAL BAKTERI DAN KADAR HISTAMIN TUNA PASCA TANGKAP DI PERAIRAN SANGIHE." Jurnal Ilmiah Tindalung 7, no. 1 (2021): 28–32. http://dx.doi.org/10.54484/jit.v7i1.385.

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Tuna yang berkualitas sangat ditentukan oleh penanganan pasca tangkap, yang kita akui aspek-aspek pasca tangkap ini belum merata dikuasai oleh masyarakat nelayan di Kepulauan Sangihe, sehingga mengakibatkan permasalahan keamanan pangan terutama kadar histamin yang melampaui batas. Histamin terbentuk akibat adanya kesalahan selama proses penanganan dan pengolahan. Tingginya temperatur adalah penyebab utama terbentuknya histamin. Histamin dapat menyebabkan keracunan pada orang yang mengkonsumsinya. Penelitian ini bertujuan untuk menentukan total bakteri dan kadar histamin tuna pasca tangkap di perairan Sangihe. Parameter uji dalam penelitian ini meliputi pengambilan dan preparasi sampel, Uji total bakteri, dan kadar histamin pada tuna segar. Analisis histamin menggunakan metode Enzyme Linked Immunosorbent Assay (ELISA). Hasil penelitian menunjukan bahwa TPC pada ketiga lokasi, yaitu Kauhis 9,7 x 104 cfu/g Santiago 2,8 x 104 cfu/g masih memenuhi standar SNI untuk tuna segar yaitu 5,0 x 105 cfu/g, sedangkan pada pasar Towo 8,2 x 105 cfu/g telah melewati batas standar. Kadar histamin dari ketiga lokasi masih dikategorikan sangat aman < 5 ppm dari standar yaitu 100 ppm.
 
 Quality tuna is very much determined by post-capture handling, which we admit that these post-catch aspects have not been evenly controlled by the fishing community in the Sangihe Islands, resulting in food safety problems, especially histamine levels that exceed the limit. Histamine is formed due to errors during the handling and processing process. The high temperature is the main cause of the formation of histamine. Histamine can cause poisoning in people who take it. This study aims to determine the total bacteria and histamine levels of post-caught tuna in Sangihe waters. The test parameters in this study include sample collection and preparation, total bacterial test, and histamine levels in fresh tuna. Histamine analysis used the Enzyme-Linked Immunosorbent Assay (ELISA) method. The results showed that the TPC in the three locations, namely Kauhis 9.7 x 104 CFU / g Santiago 2.8 x 104 CFU / g still met the SNI standard for fresh tuna, namely 5.0 x 105 CFU / g, while in the Towo 8 market, 2 x 105 CFU / g has exceeded the standard limit. The histamine levels from the three locations were still categorized as very safe < 5 ppm from the standard, namely 100 ppm.
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Yoshida, Hiroyuki, Mika Aoki, Aya Komiya, et al. "HYBID (alias KIAA1199/CEMIP) and hyaluronan synthase coordinately regulate hyaluronan metabolism in histamine-stimulated skin fibroblasts." Journal of Biological Chemistry 295, no. 8 (2020): 2483–94. http://dx.doi.org/10.1074/jbc.ra119.010457.

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The immune-regulatory compound histamine is involved in the metabolism of the essential skin component hyaluronan (HA). We previously reported that histamine up-regulates the expression of HYBID (hyaluronan-binding protein involved in hyaluronan depolymerization, also called CEMIP or KIAA1199), which plays a key role in HA degradation. However, no information is available about histamine's effects on HA synthase (HAS) expression, the molecular sizes of HA species produced, and histamine receptors and their signaling pathways in skin fibroblasts. Moreover, histamine's effects on photoaged skin remain elusive. Here, we show that histamine increases HA degradation by up-regulating HYBID and down-regulating HAS2 in human skin fibroblasts in a dose- and time-dependent manner and thereby decreases the total amounts and sizes of newly produced HA. Histamine H1 blocker abrogated the histamine effects on HYBID up-regulation, HAS2 suppression, and HA degradation. Histamine H1 agonist exhibited effects on HA levels, composition, and breakdown similar to those of histamine. Of note, blockade of protein kinase Cδ or PI3K–Akt signaling abolished histamine-mediated HYBID stimulation and HAS2 suppression, respectively. Immunohistochemical experiments revealed a significant ∼2-fold increase in tryptase-positive mast cells in photoaged skin, where HYBID and HAS2 expression levels were increased and decreased, respectively, compared with photoprotected skin. These results indicate that histamine controls HA metabolism by up-regulating HYBID and down-regulating HAS2 via distinct signaling pathways downstream of histamine receptor H1. They further suggest that histamine may contribute to photoaged skin damage by skewing HA metabolism toward degradation.
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Kljun, Jakob, Saša Petriček, Dušan Žigon, Rosana Hudej, Damijan Miklavčič, and Iztok Turel. "Synthesis and Characterization of Novel Ruthenium(III) Complexes with Histamine." Bioinorganic Chemistry and Applications 2010 (2010): 1–6. http://dx.doi.org/10.1155/2010/183097.

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Novel ruthenium(III) complexes with histamine[RuCl4(dmso-S)(histamineH)]⋅O(1a) and[RuCl4(dmso-S)(histamineH)](1b) have been prepared and characterized by X-ray structure analysis. Their crystal structures are similar and show a protonated amino group on the side chain of the ligand which is not very common for a simple heterocyclic derivative such as histamine. Biological assays to test the cytotoxicity of the compound1bcombined with electroporation were performed to determine its potential for future medical applications in cancer treatment.
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Putra, Debriga, Henny A. Dien, Roike I. Montolalu, et al. "Efek Suhu dan Waktu Simpan terhadap Kualitas Bagian Tengah Yellowfin Tuna Segar (Thunnus albacares)." Media Teknologi Hasil Perikanan 8, no. 3 (2020): 100. http://dx.doi.org/10.35800/mthp.8.3.2020.29537.

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Decline quality of Tuna as a result of abuse temperature and long shelf life time. Histamine, microbial and sensory odours become one of the global problems that can cause health problems. Large number of bacteria dominated in viscera area rather than other fish parts. This study was conducted to determine the effect of storage temperature on histamine, microbial and sensory odours on middle of Yellowfin Tuna. This study used descriptive explorative method, sampel were periodically taken for analyses at intervals 48 hours (0ºC), 8 hours (10ºC) and 4 hours (25ºC). Yellowfin Tuna was rejected earlier by the sensory odours than TPC and histamine. During storage at 0ºC for 720 hours histamine levels still acceptable for consumption however TPC already exceeded the limit after 672 hours and rejected by sensory odours for 624 hours storage. During storage at temperature 10ºC did not reached the limit for 120 hours while TPC value already reached 2x106 cfu/g for 88 hours storage and rejected by sensory odours for 72 hours storage. During storage at temperature 25ºC histamine reached 67.3 ppm for 32 hours, ALT reached 5.5x105 cfu/g for 24 hours and rejected by sensory odour for 20 hours of storage. Histamine formation correlates with the growth of microbial counts and decrease of sensory odours value. Suhu yang tinggi dalam waktu simpan lama berpengaruh terhadap penurunan kualitas ikan tuna. Histamin, mikroba dan bau busuk menjadi salah satu permasalahan global yang dapat menyebabkan masalah kesehatan. Bagian tengah ikan menjadi sumber bakteri paling tinggi dibandingkan bagian lainnya. Penelitian ini bertujuan untuk mengkaji perkembangan histamin, ALT dan sensori bau pada bagian tengah Tuna sirip kuning dalam penyimpanan suhu yang berbeda. Penelitian ini menggunakan metode deskriptif eksploratif, sample di analisis secara berkala pada suhu 0ºC (48 jam), 10ºC (8 jam) dan 25ºC (4 jam). Tuna sirip kuning mengalami pembusukan lebih awal berdasarkan parameter sensori bau, kemudian disusul ALT dan Histamin. Kandungan histamine pada suhu 0ºC masih layak untuk dikonsumsi setelah 720 jam, namun nilai ALT melebihi batas aman setelah 672 jam penyimpanan dan nilai sensori bau setelah 624 jam penyimpanan. Penyimpanan suhu 10ºC tidak menyebabkan peningkatan histamin melebihi batas limit setelah penyimpanan selama 120 jam, sedangkan nilai ALT mencapai 2x106 cfu/g setelah 88 jam dan nilai sensori bau menyatakan ikan busuk setelah penyimpanan selama 72 jam. Penyimpanan suhu 25ºC histamin mencapai 67,3 ppm setelah penyimpanan 32 jam dan nilai ALT 5,5x105 cfu/g pada penyimpanan ke 24 jam serta ikan dinyatakan busuk berdasarkan sensori bau setelah 20 jam penyimpanan. Peningkatan histamin berkorelasi dengan pertumbuhan jumlah mikroba dan penurunan nilai sensori bau.
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Gasiorek, Friederike, Ervice Pouokam, Martin Diener, Sabine Schlecht, and Mathias S. Wickleder. "Effects of multivalent histamine supported on gold nanoparticles: activation of histamine receptors by derivatized histamine at subnanomolar concentrations." Organic & Biomolecular Chemistry 13, no. 39 (2015): 9984–92. http://dx.doi.org/10.1039/c5ob01354b.

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Usuki, Seigo, Noriko Tamura, Tomohiro Tamura, et al. "Konjac Ceramide (kCer)-Mediated Signal Transduction of the Sema3A Pathway Promotes HaCaT Keratinocyte Differentiation." Biology 11, no. 1 (2022): 121. http://dx.doi.org/10.3390/biology11010121.

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Histamines suppress epidermal keratinocyte differentiation. Previously, we reported that konjac ceramide (kCer) suppresses histamine-stimulated cell migration of HaCaT keratinocytes. kCer specifically binds to Nrp1 and does not interact with histamine receptors. The signaling mechanism of kCer in HaCaT cells is also controlled by an intracellular signaling cascade activated by the Sema3A-Nrp1 pathway. In the present study, we demonstrated that kCer treatment induced HaCaT keratinocyte differentiation after migration of immature cells. kCer-induced HaCaT cell differentiation was accompanied by some features of keratinocyte differentiation markers. kCer induced activating phosphorylation of p38MAPK and c-Fos, which increased the protein levels of involucrin that was the latter differentiation marker. In addition, we demonstrated that the effects of both kCer and histamines are regulated by an intracellular mechanism of Rac1 activation/RhoA inhibition downstream of the Sema3A/Nrp1 receptor and histamine/GPCR pathways. In summary, the effects of kCer on cell migration and cell differentiation are regulated by cascade crosstalk between downstream Nrp1 and histamine-GPCR pathways in HaCaT cells.
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Barman, S. A., and A. E. Taylor. "Histamine's effect on pulmonary vascular resistance and compliance at elevated tone." American Journal of Physiology-Heart and Circulatory Physiology 257, no. 2 (1989): H618—H625. http://dx.doi.org/10.1152/ajpheart.1989.257.2.h618.

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Histamine's effect on the longitudinal resistance and compliance distribution in the canine pulmonary circulation was determined under control and elevated vascular tone using the thromboxane analogue U46619. The arterial-, venous-, and double-occlusion techniques were used in isolated blood-perfused dog lungs at both constant flow and constant pressure. Large and small blood vessel resistances and compliances were studied in lungs given the following treatments: 1) histamine; 2) histamine in lungs pretreated with the H1-receptor antagonist diphenhydramine, and 3) histamine in lungs pretreated with the H2-receptor antagonist cimetidine. The results of this study indicate that histamine constricts small and large veins through H1-receptor mediation at both normal and elevated vascular tone. When vascular tone was elevated, H2-receptor vasodilatation was also apparent in all blood vessel segments. Histamine decreased total, middle compartment, and large vessel vascular compliances by an H1-receptor effect. When vascular tone was elevated, histamine's H1-receptor-mediated vasoconstrictor effect on compliance vessels was less due to the presence of an H2-receptor-mediated vasodilatory system.
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Dissertations / Theses on the topic "Histamine"

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Cumming, Paul Kenneth. "Pharmacology of cerebral histamine." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/30687.

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Four aspects of the function of histaminergic systems were studied in the rat brain: toxicology, catabolism, release in vivo, and high affinity binding of histamine. Preparations of histamine-N-methyltransferase (HNMT) derived from kidney and brain were employed in the radioenzymatic quantification of histamine in biological samples. Tritiated S-adenosyl-L-methionine ([³H]-SAM) served as the co-substrate. A toxicological study was conducted to determine the sensitivity of the HA innervation to prenatal treatment with methylazoxymethanol (MAM), an inhibitor of mitosis. In adult rats, the MAM treatment was without effect on cerebral histamine content, although forebrain HNMT activity was 50% reduced. In another study, C-57 mice were treated with the selective dopamine neurotoxin l-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP). Substantial dopamine depletions were not associated with alterations in the cerebral histamine content. In a study of the structural requirements for HNMT inhibition, 9-amino-1,2,3,4-tetrahydroacridine (THA), was found to be one of the most potent inhibitors yet described. The β-carboline alkaloids, of which harmaline is the prototype, were also found to be moderately potent HNMT inhibitors. Because of the lack of high-affinity re-uptake and the absence of alternate catabolic pathways, blockade of HNMT can potentially alter central histaminergic tone. Peripheral administration of THA was able to produce dose-dependent increases in cerebral histamine content, as was the more potent HNMT inhibitor, metoprine. The issue of structural requirements for HNMT inhibition are discussed in the light of these results. The in vivo release of histamine was studied by the cerebral microdialysis technique. After chronic implantation of horizontal probes, TTX-insensitive and partially calcium-sensitive efflux of histamine was detected in the dorsal striatum and the bed nucleus of the stria terminalis. In striatum, histamine efflux was elevated 50% after peripheral histidine loading (500 mg/kg, i.p.). After synthesis blockade with α-fluoromethylhistidine (100 mg/kg,i.p.), extracellular histamine levels in striatum disappeared in a bi-exponential manner. The half-lives of this disappearance, 32 minutes and 7 hours, indicate the presence of at least two histamine pools. Striatal histamine efflux was elevated by yohimbine treatment (10 mg/kg, i.p.), suggesting the presence of a tonic α₂-adrenergic inhibition of histamine release in vivo. In addition to the classical H₁ and H₂ receptors, histamine is able to bind to a pharmacologically distinct site, H₃, recently characterized as an autoreceptor regulating the synthesis and release of histamine. The binding properties of the H₃ ligand [³H]-N[symbol omitted]-methylhistamine ([³H]-N-MeHA) were studied in forebrain cryostat sections by autoradiography. Determination of Bmax (25 fmole/section) and displacement studies indicated that [³H]-N-MeHA bound to the same site as [³H]-histamine: the high affinity histamine binding site. Binding was greatest in the basal ganglia and had a complex distribution within the cerebral cortex. Quinolinic acid lesion studies indicated that the majority of the binding in the basal ganglia was on striato-nigral projection neurons. Cortical binding was also sensitive to local excitotoxic lesions. Therefore, the majority of H₃ binding is located on postsynaptic structures intrinsic to these brain regions, rather than on presynaptic autoreceptors on terminals of histamine neurons.<br>Medicine, Faculty of<br>Graduate
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Halpern, Georges M. "Hyperréactivité cutanée et histamine." Université de Paris-Sud. Faculté de pharmacie (Châtenay-Malabry, Hauts-de-Seine), 1992. http://www.theses.fr/1992PA114832.

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Takemoto, Emy. "Desenvolvimento de metodologia por cromatografia líquida de ultra eficiência para determinação de histamina em pescados in natura e em conservas." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/74/74132/tde-19092016-134317/.

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No Brasil, o consumo de pescado in natura cresce a cada ano e sua ingestão tem sido associada a problemas de saúde, principalmente, surtos de intoxicação alimentar causado pela histamina, podendo representar risco à saúde do consumidor. A histamina pode provocar erupções na pele, náuseas, dor de cabeça, palpitações, vômitos, dores abdominais, distúrbios respiratórios e taquicardia. O Brasil exporta pescado para os principais mercados consumidores e tem enfrentado barreiras comerciais pela exigência de análises de histamina, com a finalidade de assegurar a qualidade do pescado exportado. Assim sendo, foi desenvolvido e validado um método por cromatografia líquida de ultra eficiência (CLUE) para a determinação dos teores de histamina em peixes. O método desenvolvido mostrou ter boa linearidade, seletividade, exatidão e precisão, ser robusto e com os limites de detecção e quantificação determinados de 0,03 &micro;g mL-1 e 0,10 &micro;g mL-1, respectivamente. A metodologia foi aplicada a amostras de pescados (atum e sardinha) in natura e em conservas. Das 12 amostras analisadas de atum in natura somente uma apresentou teor de histamina de 1,07 mg.kg-1, 05 amostras de sardinha in natura apresentaram teores de 26,81, 0,35, 37,25, 9,97 e 0,94 mg kg-1, respectivamente. Nas amostras de atum em conserva, 02 apresentaram teores de 1,30 e 0,13 mg kg-1. Enquanto que, 04 amostras de sardinha em conserva continham teores de histamina de 2,49, 68,96 e 11,66 mg kg-1, e uma das amostras de sardinha em conserva estava com o teor muito acima, cerca de 17 vezes do limite máximo estabelecido pelo MAPA, de 100 mg kg-1 para conservas de sardinha. Essa quantidade de histamina encontrada pode sugerir a ocorrência de uma intoxicação, representando risco à saúde humana. Além disso, foi calculada a incerteza de medição, pois garante uma maior confiabilidade dos resultados analíticos para tomadas de decisões importantes em Vigilância Sanitária e Saúde Pública.<br>In Brazil, the consumption of fish in nature grows every year and its intake has been linked to health problems, especially food poisoning outbreaks caused by histamine, which may pose a risk to consumer health. Histamine can cause skin rashes, nausea, headache, palpitations, vomiting, abdominal pain, respiratory disorders and tachycardia. Brazil exports fish to the main consumer markets and has faced trade barriers by requiring histamine analysis, in order to ensure the quality of exported fish. Thus, it was a method developed and validated liquid chromatography ultra efficiency (CLUE) for determining histamine levels in fish. The method was proven to have good linearity, selectivity, accuracy and precision and be robust with the limits of detection and quantification of certain 0,03 ug mL-1 and 0.10 ug mL-1, respectively. The methodology was applied to fish samples (tuna and sardines) in fresh and canned. Of the 12 samples analyzed tuna in fresh only one showed histamine content of 1.07 mg.kg-1, 05 sardine in fresh samples showed levels of 26.81, 0.35, 37.25, 9.97 and 0.94 mg kg-1, respectively. In the samples of canned tuna, 02 showed levels of 1.30 and 0.13 mg kg-1. While 04 canned sardines containing histamine concentrations of 2.49, 68.96 and 11.66 mg kg-1, and one sample canned sardine content was much higher, about 17 times higher than the maximum limit established by MAPA, 100 mg kg-1 for canned sardine. This amount of histamine found may suggest the occurrence of intoxication, representing a risk to human health. In addition, the measurement uncertainty was calculated as it ensures a higher reliability of the analytical results for taking important decisions on Health Surveillance and Public Health.
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Evans, Debra. "Mechanisms controlling gastric histamine biosynthesis." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.262323.

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Brine, J. M. "Histamine and its brain receptors." Thesis, University of Southampton, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.356768.

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Geng, Tian. "Structural studies on histamine receptors." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/56628.

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The work presented in this thesis concerns a sub-family of the biologically important family of G-protein coupled receptors (GPCRs) known as histamine receptors. They are recognized as important drug targets for many disorders in mankind, such as allergic rhinitis and Alzheimer’s disease. This thesis has its roots in the first structure of histamine receptor H1, determined in complex with a first generation antihistamine (doxepin) in 2011. Here, extensive biological, computational and structural investigations have been carried out for H1 in complex with two second-generation antihistamines (cetirizine and fexofenadine). Crystal structures of H1-cetirizine (3.1 Å) and H1-fexofenadine (3.4 Å) show the binding modes of second-generation antihistamines correspond well with the previous docking studies. The so-called “anion binding site” on H1 is responsible for the high specificity of the ligands. Some regions on the receptor were difficult to define in the crystal structures. These regions were characterized by radioligand binding assays, together with molecular dynamic simulations (carried out by our collaborators from Oxford University) suggested that, second generation ligands have a significantly large impact on receptor dynamics. While H1 mediates many immune-related disorders, the histamine receptor H3 is a drug target to many mental disorders. In addition to the H1 work presented here, it is also reported the initial work done on the histamine receptor H3. With extensive screenings on various H3 constructs (from human and turkey organisms) and expression systems, it was observed that the best construct was from turkey. An optimal expression protocol in insect cells was obtained. This has established a secure starting point for future structural studies on H3.
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Stutts, William A. (William Anderson). "The Role of Neuronal Histamine in Memory Processing." Thesis, University of North Texas, 1995. https://digital.library.unt.edu/ark:/67531/metadc278349/.

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Neuronal histamine(HA) may play a role in memory processing. This hypothesis is based upon evidence that the action of histamine at central H1 and H2 histamine receptor sites has been shown to modulate memory of rats and mice in adversely-motivated tasks. The purpose of this study was to test this hypothesis more thoroughly in mice using two distinct approaches to neuronal HA manipulation. One approach involved the use of new pharmacological agents which act at the histamine H3 receptor. It has been demonstrated that the selective H3 antagonist thioperamide increases HA release in the brain of mice whereas the H3 agonist imetit decreases HA release via modulation of presynaptic H3 autoreceptors. It was expected that an increase in neuronal HA via the autoreceptor mechanism would result in facilitation of memory processing whereas a decrease in HA release would disrupt memory processing. The second approach involved the manipulation of cerebral HA levels via the specific enzyme inhibiting compounds alpha-flouromethylhistidine (alpha-FMH), a potent neuronal HA depleter and metoprine, a histamine-methyl transferase inhibitor which results in accumulation of neuronal HA. Again, effects of increased HA due to metoprine and decreased HA levels due to alpha-FMH were expected to facilitate and disrupt memory processing respectively. One trial inhibitory (passive) avoidance training was employed in each experiment in order to evaluate the effect of each drug on memory. Each compound was tested for effects on memory consolidation and memory retrieval as well as for the presence of state dependent effects. The pattern of effects obtained with thioperamide suggested facilitation of acquisition or memory storage (consolidation) processes, with no effect on the retrieval phase of memory processing. In accordance with those findings, significant disruption of memory occurred when imetit was present during the consolidation phase of memory processing, but not when presented prior to the retrieval phase. These findings suggest that H3 receptor sites play a significant role in the modulation of memory processes via some mechanism which exclusively affects the acquisition or memory consolidation process, while the retrieval of previously laid down memory traces is unaffected.
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Morel, Anne. "Immunoanalyse des neuromédiateurs : histamine et indolamines." Aix-Marseille 2, 1990. http://www.theses.fr/1990AIX22040.

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L'immunoanalyse de l'histamine et des indolamines (serotonine et 5hiaa) est rendue difficile par la faible taille (m inferieure a 200) de ces neuromediateurs. Nous avons donc cherche, par modification chimique de ces haptenes a atteindre la masse minimale de 400, necessaire pour l'obtention d'anticorps de bonne affinite. Le meme motif antigenique (haptene modifie) doit etre reproduit pour tous les protagonistes de l'immunoessai. L'amelioration de l'immunoessai du 5hiaa illustre l'importance de l'homologie de structure a reproduire pour l'immunogene, le traceur et l'haptene a doser. La modification chimique doit etre reproduite dans des milieux biologiques complexes, ce qui en limitera le choix. L'utilisation des maillons succinyl-glycyl pour les immunoessais de l'histamine et de la serotonine, permet un gain d'affinite d'un facteur superieur a 100000 et une grande specificite. L'amplification de l'immuno-reconnaissance apportee par chaque maillon et les caracteristiques de la liaison antigene-anticorps ont ete etudiees plus particulierement pour l'histamine. Le dosage radioimmunologique de l'histamine ainsi construit est adapte a la determination de l'histamine dans des milieux biologiques varies, offrant un outil nouveau en allergologie tant en recherche fondamentale qu'en routine clinique
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Rigal, Dominique. "Action de l'histamine sur la physiologie des lymphocytes : synthèse bibliographique et apport personnel." Lyon 1, 1987. http://www.theses.fr/1987LYO1H073.

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Dewachter, Pascale. "Inhibiteurs calciques, activateurs potassiques et bronchospasme expérimental à l'histamine." Nancy 1, 1995. http://docnum.univ-lorraine.fr/public/SCD_T_1995_0459_DEWACHTER.pdf.

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Books on the topic "Histamine"

1

Uvnäs, Börje, ed. Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9.

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-M, Arrang J., and Uvnäs Börje 1913-, eds. Histamine and histamine antagonists. Springer-Verlag, 1991.

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Jarisch, Reinhart, ed. Histamine Intolerance. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-55447-6.

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Blandina, Patrizio, and Maria Beatrice Passani, eds. Histamine Receptors. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40308-3.

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Hattori, Yuichi, and Roland Seifert, eds. Histamine and Histamine Receptors in Health and Disease. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58194-1.

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Thurmond, Robin L., ed. Histamine in Inflammation. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-8056-4.

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Jean-Charles, Schwartz, and Haas Helmut L. 1942-, eds. The Histamine receptor. Wiley-Liss, 1992.

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Falus, András. Histamine and inflammation. R.G. Landes, 1994.

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Khardori, Nancy, Rahat Ali Khan, and Trivendra Tripathi, eds. Biomedical Aspects of Histamine. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-90-481-9349-3.

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Harvima, Rauno. Histamine radio enzyme assay. Dept. of Dermatology, University of Kuopio, 1989.

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Book chapters on the topic "Histamine"

1

Lorenz, W. "Suggestions to Those Who Have Become Histaminologists at a Time of Overflooding and Biased Information." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_1.

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Lorenz, W., E. Neugebauer, B. Uvnäs, et al. "Munich Consensus Development Conference on Histamine Determination." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_10.

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Barrett, K. E., and F. L. Pearce. "Heterogeneity of Mast Cells." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_11.

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Gomperts, B. D. "Control of the Exocytotic Mechanism in Rat Mast Cells." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_12.

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Watanabe, T., Y. Taguchi, K. Maeyama, and H. Wada. "Formation of Histamine: Histidine Decarboxylase." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_13.

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Maslinski, C., and W. A. Fogel. "Catabolism of Histamine." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_14.

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Schwartz, J. C., J. M. Arrang, M. L. Bouthenet, M. Garbarg, H. Pollard, and M. Ruat. "Histamine Receptors in Brain." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_15.

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Yamatodani, A., N. Inagaki, P. Panula, N. Itowi, T. Watanabe, and H. Wada. "Structure and Functions of the Histaminergic Neurone System." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_16.

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Foreman, J. C. "Histamine H2 Receptors and Lung Function." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_17.

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Öbrink, K. J. "Histamine and the Parietal Cell." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_18.

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Conference papers on the topic "Histamine"

1

Shkodra, Bajramshahe, Mattia Petrelli, Antonio Altana, et al. "Organic Memristive Devices with Capacitive-Coupled Effect: A Novel Approach for Histamine Sensing." In 2024 IEEE BioSensors Conference (BioSensors). IEEE, 2024. http://dx.doi.org/10.1109/biosensors61405.2024.10712669.

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Horie, Masafumi, Tadashi Kohyama, Yasuhiro Yamauchi, et al. "Histamine Stimulates Human Lung Fibroblast Mediated Gel Contraction." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4299.

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Ritter, Arthur, Christine M. Genua, Janardan Yadav, Swamy Laxminarayan, and David Kristol. "Active analog approach to Histamine HI receptor antagonists." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5760929.

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Ritter, Gerula, Yadav, Laxminarayan, and Kristol. "Active Analog Approach To Histamine H1 Receptor Antagonists." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.589633.

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Sbeih, N., R. Rignault, J. Callebert, et al. "AB0014 HISTAMINE AND BASOPHIL GRANULOCYTES IN AA AMYLOIDOSIS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.5030.

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Wei, Xiaona, and Yuzong Chen. "Computational model of VEGF, thrombin and histamine signalling network." In 2010 IEEE International Conference on Bioinformatics and Biomedicine Workshops (BIBMW). IEEE, 2010. http://dx.doi.org/10.1109/bibmw.2010.5703939.

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Newick, Kheng, Shaun O'Brien, Veena Kapoor, et al. "Abstract B78: Histamine dihydrochloride in the treatment of mesothelioma." In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-b78.

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Gajowniczek-Ałasa, Dorota, Dominik Szwajgier, and Ewa Baranowska-Wójcik. "Biogenic amines in foods excluded from low histamine diets." In 1st International PhD Student’s Conference at the University of Life Sciences in Lublin, Poland: ENVIRONMENT – PLANT – ANIMAL – PRODUCT. Publishing House of The University of Life Sciences in Lublin, 2022. http://dx.doi.org/10.24326/icdsupl1.t005.

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NAKAMURA, TADAHO, TAKEO YOSHIKAWA, NAOYA NOGUCHI та ін. "THE ROLE OF HISTAMINE H3 RECEPTOR IN PANCREATIC β-CELLS". У Proceedings of the Tohoku University Global Centre of Excellence Programme. IMPERIAL COLLEGE PRESS, 2012. http://dx.doi.org/10.1142/9781848169067_0034.

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Naila, A., S. Flint, G. C. Fletcher, P. J. Bremer, G. Meerdink, and R. H. Morton. "Degradation of histamine in tuna soup by diamine oxidase (DAO)." In FOOD AND ENVIRONMENT 2011. WIT Press, 2011. http://dx.doi.org/10.2495/fenv110111.

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Reports on the topic "Histamine"

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Draganova-Filipova, Milena, Elisaveta Apostolova, and Plamen Zagorchev. Effects of Rosmarinus officinalis Oil on Histamine-induced Acute Inflammation. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.02.14.

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Draganova-Filipova, Milena, Elisaveta Apostolova, and Plamen Zagorchev. Effects of Rosmarinus officinalis Oil on Histamine-induced Acute Inflammation. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/grabs2018.2.14.

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Fowler, Joanna S., and Michael Furey. Radiotracer Synthesis and PET Imaging Evaluation for Brain Histamine Receptors. Office of Scientific and Technical Information (OSTI), 2014. http://dx.doi.org/10.2172/1149990.

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Zagorchev, Plamen, Elisaveta Apostolova, Vesela Kokova, Lyudmil Peychev, Zhivko Peychev, and Milena Draganova-Filipova. Activation of Kv7.2 to Kv7.5 Channels by Retigabine Modulates the Effects of Histamine and 2‑(2‑Pyridyl) Ethylamine on Smooth Muscles. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.10.18.

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