Relevant bibliographies by topics / Histamine H2 receptor antagonist

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  2. Dissertations / Theses
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Academic literature on the topic 'Histamine H2 receptor antagonist'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Histamine H2 receptor antagonist"

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Chen, M., and L. A. Brown. "Histamine stimulation of surfactant secretion from rat type II pneumocytes." American Journal of Physiology-Lung Cellular and Molecular Physiology 258, no. 4 (1990): L195—L200. http://dx.doi.org/10.1152/ajplung.1990.258.4.l195.

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The present study examined the effect of histamine on the secretion of phosphatidylcholine, the predominant component of pulmonary surfactant from adult rat alveolar type II pneumocytes in primary culture. Histamine stimulated surfactant secretion in a time- and dose-dependent manner. At a concentration of 10 microM, histamine stimulated surfactant release by 5.2-fold over the basal secretory rate. The concentration producing half the maximal response for histamine-induced secretion was 70 nM. Histamine-induced secretion was blocked by both the selective histamine1 receptor antagonist pyrilami
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Neely, C. F., I. Matot, D. Haile, J. Nguyen, and V. Batra. "Tone-dependent responses of histamine in feline pulmonary vascular bed." American Journal of Physiology-Heart and Circulatory Physiology 268, no. 2 (1995): H653—H661. http://dx.doi.org/10.1152/ajpheart.1995.268.2.h653.

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Under conditions of controlled pulmonary blood flow and constant left atrial pressure, histamine produced tone-dependent responses in the pulmonary vascular (PV) bed of intact-chest, spontaneously breathing cats. At low, baseline PV tone, histamine produced dose-dependent increases in mean lobar arterial pressure that were antagonized by the selective histamine H1-receptor antagonist, diphenhydramine. The cyclooxygenase inhibitor, meclofenamate, and the thromboxane A2 (TxA2) receptor antagonist, SQ-29548, had no effect on these vasoconstrictor responses of histamine. After an increase in PV to
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Baloch, Naseer Khan, Nusrat Jafri, and Ahmed Danyal. "H2 RECEPTOR ACTIVITY." Professional Medical Journal 21, no. 05 (2018): 933–35. http://dx.doi.org/10.29309/tpmj/2014.21.05.2499.

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… Histamine can stimulate the heart by directly interacting with cardiac histaminereceptors. In the present study we have investigated the H2 receptor activity in isolated rabbitheart. Cimetidine, a specific H2 receptor antagonist was used. The isolated heart was mountedin langendroff apparatus. The heart was perfused at a constant pressure with oxygenatedRinger‘s Locke solution. H2 receptor antagonist produces negative inotropic effect in thepresence of histamine. This indicates that H2 receptors are present in rabbit heart, and plays arole in mediation of positive inotropic effect produced t
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Champion, Hunter C., Trinity J. Bivalacqua, David G. Lambert, Rasheed A. Abassi, and Philip J. Kadowitz. "Analysis of vasoconstrictor responses to histamine in the hindlimb vascular bed of the rabbit." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 277, no. 4 (1999): R1179—R1187. http://dx.doi.org/10.1152/ajpregu.1999.277.4.r1179.

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Hemodynamic responses to histamine were investigated in the anesthetized rabbit. Intravenous injections of histamine induced dose-dependent decreases in systemic arterial pressure that were blocked by the H1-receptor antagonist pyrilamine but not the H2 antagonist cimetidine. Injections of histamine and the H1 agonist 6-[2-(4-imidazolyl)ethylamine]- N-(4-trifuormethylphenyl)-heptanecardoxamide dimaleate (HTMT) into the hindlimb perfusion circuit increased hindlimb perfusion pressure, whereas the H2agonist dimaprit decreased perfusion pressure and the H3-receptor agonist R-(−)-α-methylhistamine
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Barman, S. A., and A. E. Taylor. "Histamine's effect on pulmonary vascular resistance and compliance at elevated tone." American Journal of Physiology-Heart and Circulatory Physiology 257, no. 2 (1989): H618—H625. http://dx.doi.org/10.1152/ajpheart.1989.257.2.h618.

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Histamine's effect on the longitudinal resistance and compliance distribution in the canine pulmonary circulation was determined under control and elevated vascular tone using the thromboxane analogue U46619. The arterial-, venous-, and double-occlusion techniques were used in isolated blood-perfused dog lungs at both constant flow and constant pressure. Large and small blood vessel resistances and compliances were studied in lungs given the following treatments: 1) histamine; 2) histamine in lungs pretreated with the H1-receptor antagonist diphenhydramine, and 3) histamine in lungs pretreated
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Ahmed, T., and M. King. "Suppression of pulmonary and systemic vascular histamine H2-receptors in allergic sheep." Journal of Applied Physiology 60, no. 3 (1986): 791–97. http://dx.doi.org/10.1152/jappl.1986.60.3.791.

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We have previously demonstrated a depression of airway H2-receptor function in sheep allergic to Ascaris suum antigen. To investigate whether this is a generalized defect, we studied the H1- and H2- histamine receptor functions in the pulmonary and systemic circulations of allergic and nonallergic sheep. Pulmonary arterial pressure, and cardiac output were measured for calculation of pulmonary vascular resistance (PVR) and systemic vascular resistance (SVR) before and immediately after a rapid intrapulmonary infusion of histamine (10 micrograms/kg), with and without pretreatment with H1- (chlo
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Mahjour, Marjan, and Arash Khoushabi. "An Herbal H2 Blocker in Melasma Treatment." Current Drug Discovery Technologies 17, no. 3 (2020): 272–77. http://dx.doi.org/10.2174/1570163816666190121145653.

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Background: Melasma is a skin pigmentation disorder that remains resistant to available therapies. The exact cause of melasma is unknown. Histamine is an inflammatory factor. Its involvement in pigmentation is obscure. The aim of this study is to introduce an herbal antihistamine H2 receptor which is effective in these disorders. Methods: This is a review study by searching the electronic databases and also Persian Medicine books, from 2000 to 2018 by the keywords such as H2 antagonist, H2 blocker and melasma. Results: According to the researched studies, histamine can induce melanogenesis and
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Knigge, Ulrich, Steen Matzen, and Jørgen Warberg. "Effects of H2-receptor antagonists on prolactin secretion: Specificity and mediation of the response." Acta Endocrinologica 115, no. 4 (1987): 461–68. http://dx.doi.org/10.1530/acta.0.1150461.

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Abstract. The effects on prolactin secretion of histamine H2-receptor antagonists infused intracerebroventricularly were studied in urethane anaesthetized male rats. A dose of 1.6 μmol cimetidine stimulated basal prolactin secretion and did not affect the histamine-induced release, whereas 0.4 μmol cimetidine inhibited basal and histamine-stimulated prolactin secretion. 0.1 μmol cimetidine had no effect. The more potent H2-receptor antagonist ranitidine at doses of 0.1, 0.4, 1.6 μmol had no effect on basal prolactin secretion, whereas 0.4 and 1.6 μmol inhibited the histamine-stimulated secreti
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Taylor, D. J., J. R. Yoffe, D. M. Brown, and D. E. Woolley. "Histamine H2 receptors on chondrocytes derived from human, canine and bovine articular cartilage." Biochemical Journal 225, no. 2 (1985): 315–19. http://dx.doi.org/10.1042/bj2250315.

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Histamine (1-100 microM) induced a concentration-dependent increase in intracellular cyclic AMP in monolayer cultures of human, canine and foetal-bovine articular chondrocytes. The dose-response curve for histamine in each culture was progressively displaced to the right with increasing concentrations of cimetidine, an H2-receptor antagonist. The histamine-induced cyclic AMP elevation in human articular chondrocytes was also significantly decreased by ranitidine, another H2 antagonist, but not by the H1 antagonists mepyramine and chlorpheniramine. These findings indicate that histamine activat
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Whyment, Andrew D., Andrew M. Blanks, Kevin Lee, Leo P. Renaud, and David Spanswick. "Histamine Excites Neonatal Rat Sympathetic Preganglionic Neurons In Vitro Via Activation of H1 Receptors." Journal of Neurophysiology 95, no. 4 (2006): 2492–500. http://dx.doi.org/10.1152/jn.01135.2004.

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The role of histamine in regulating excitability of sympathetic preganglionic neurons (SPNs) and the expression of histamine receptor mRNA in SPNs was investigated using whole-cell patch-clamp electrophysiological recording techniques combined with single-cell reverse transcriptase polymerase chain reaction (RT-PCR) in transverse neonatal rat spinal cord slices. Bath application of histamine (100 μM) or the H1 receptor agonist histamine trifluoromethyl toluidide dimaleate (HTMT; 10 μM) induced membrane depolarization associated with a decrease in membrane conductance in the majority (70%) of S
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Dissertations / Theses on the topic "Histamine H2 receptor antagonist"

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Dulabh, R. "Histamine and H2-receptor antagonist interactions in the respiratory tract of the guinea pig." Thesis, University of Hertfordshire, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304132.

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Pereira, Francis Solange Vieira Tourinho. "Estudo multicentrico da prescrição de antagonistas de receptores H2 da histamina e de inibidores da bomba de protons." [s.n.], 2000. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309531.

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Orientador: Gun Birgitta Bergsten Mendes<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-07-27T04:08:24Z (GMT). No. of bitstreams: 1 Pereira_FrancisSolangeVieiraTourinho_M.pdf: 1288504 bytes, checksum: d14d47aaa174cc1d3d16d1fed1792303 (MD5) Previous issue date: 2000<br>Resumo: Os antagonistas de receptores H2 da histarnina (AH2) e os inibidores da bomba de prótons (IBP) são efetivos no tratamento de condições clinicas que se acompanham de hipersecreção ácida gástrica, sendo amplamente utilizados na prevenção de
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Birnkammer, Tobias [Verfasser], and Armin [Akademischer Betreuer] Buschauer. "Highly potent and selective acylguanidine-type histamine H2 receptor agonists: synthesis and structure-activity relationships of mono- and bivalent ligands / Tobias Birnkammer. Betreuer: Armin Buschauer." Regensburg : Universitätsbibliothek Regensburg, 2011. http://d-nb.info/1022872559/34.

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Plank, Nicole [Verfasser], and A. [Akademischer Betreuer] Buschauer. "Dimeric histamine H2 receptor agonists as molecular tools and genetically engineered HEK293T cells as an assay platform to unravel signaling pathways of hH1R and hH2R / Nicole Plank. Betreuer: A. Buschauer." Regensburg : Universitätsbibliothek Regensburg, 2016. http://d-nb.info/1100276599/34.

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López, Muñoz Laura. "Homology modeling and structural analysis of the antipsychotic drugs receptorome." Doctoral thesis, Universitat Pompeu Fabra, 2010. http://hdl.handle.net/10803/7228.

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Classically it was assumed that the compounds with therapeutic effect exert their action interacting with a single receptor. Nowadays it is widely recognized that the pharmacological effect of most drugs is more complex and involves a set of receptors, some associated to their positive effects and some others to the side effects and toxicity. Antipsychotic drugs are an example of effective compounds characterized by a complex pharmacological profile binding to several receptors (mainly G protein-coupled-receptors, GPCR). In this work we will present a detailed study of known antipsychotic drug
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Ho, Yu-Hsiang, and 何宇祥. "Study on the analysis of H2 histamine receptor antagonists by capillary electrophoresis." Thesis, 2000. http://ndltd.ncl.edu.tw/handle/07802329296156111829.

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碩士<br>高雄醫學大學<br>藥學研究所<br>88<br>A simple capillary electrophoretic method was established for the determination of cimetidine, ranitidine, famotidine and nizatidine in tablets. The background buffer was 100 mM sodium dihydrogen phosphate adjusted to pH 3.50. Analysis was run at the voltage of 20 kV and a detection wavelength of 214 nm. The method is sensitive with a detection limit of 10 mM (cimetidine), 20 mM (ranitidine), 20 mM (nizatidine) and 10 mM (famotidine) (S/N=3, hydrodynamic injection 1sec.). The calibration curves were linear over a concentration range of 100-300 mM. Mean recoveries
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Erdmann, Daniela [Verfasser]. "Histamine H2- and H3-receptor antagonists : synthesis and characterization of radiolabelled and fluorescent pharmacological tools / vorgelegt von Daniela Erdmann." 2010. http://d-nb.info/1009839616/34.

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Kraus, Anja [Verfasser]. "Highly potent, selective acylguanidine type histamine H2-receptor agonists : synthesis and structure-activity relationships / vorgelegt von Anja Kraus." 2008. http://d-nb.info/987515187/34.

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Chen, Xueyu. "Evaluation of the effect of subject age, hepatic function and the co-administration of the H2-receptor antagonist, cimetidine on the pharmacokinetics and pharmacodynamics of the H1-receptor antagonist, hydroxyzine, and its active metabolite cetirizine in humans and rabbits." 1990. http://hdl.handle.net/1993/17010.

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Ghorai, Prasanta [Verfasser]. "Arpromidine-related acylguanidines : synthesis and structure-activity relationships of a new class of guanidine-type histamine H2 receptor agonists with reduced basicity / vorgelegt von Prasanta Ghorai." 2006. http://d-nb.info/981466257/34.

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Books on the topic "Histamine H2 receptor antagonist"

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E, Pounder R., ed. Histamine H2-receptor antagonists. Science Press, 1990.

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E, Pounder R., ed. Histamine H2-receptor antagonists. Science Press, 1990.

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E, Pounder R., ed. Histamine H2-receptor antagonists. Science Press, 1990.

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1939-, Rocklin Ross E., ed. Histamine and H2 antagonists in inflammation and immunodeficiency. Dekker, 1990.

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Heald, Karen Anne. Histamine H2-receptors: Characterisation and preliminary evaluation of their antagonism in the treatment of pepticulceration. University of Birmingham, 1988.

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Cardiovascular Effects of Histamine and H2-Receptor Antagonists. Zuckschwerdt, W., 1994.

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Roy Pounder MA MD FRCP. Histamine H2-receptor Antagonists: The Landmark Papers (The Landmark Papers). Science Press Ltd, 1990.

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Settipane, Guy A. H1 and H2 Histamine Receptors. New England & Regional Allergy, 1988.

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Tryba, M. H2-Antagonisten in der Prämedikation: Präventive Maßnahmen Zur Vermeidung Von Aspirationspneumonien und Anaphylaktoiden Reaktionen. Springer London, Limited, 2013.

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H2-Antagonisten in der Prämedikation: Präventive Maßnahmen zur Vermeidung von Aspirationspneumonien und anaphylaktoiden Reaktionen. Springer, 1985.

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Book chapters on the topic "Histamine H2 receptor antagonist"

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Sugano, Kentaro. "Histamine H2-receptor antagonists." In Pocket Guide to Gastrointestinal Drugs. John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118481530.ch3.

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Brimblecombe, Roger W., and C. Robin Ganellin. "Cimetidine and Other Histamine H2-Receptor Antagonists." In Drug Discovery and Development. Humana Press, 1987. http://dx.doi.org/10.1007/978-1-4612-4828-6_13.

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Ganellin, C. Robin. "Development of Anti-Ulcer H2-Receptor Histamine Antagonists." In Analogue-based Drug Discovery. Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527608001.ch4.

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Pounder, R. "1976 – 1894: How should we use histamine H2-receptor antagonists?" In Peptic Ulcer Disease: Basic and Clinical Aspects. Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5034-4_9.

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Foreman, J. C. "Histamine H2 Receptors and Lung Function." In Histamine and Histamine Antagonists. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75840-9_17.

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Lorenz, W., H. Sitter, B. Stinner, et al. "Controlled Clinical Trials and Cross-Sectional Studies with Plasma Histamine Measurements and Histamine Receptor Antagonists: Solving the Problem of Preoperative H1- + H2- Prophylaxis by Asking New Questions?" In New Perspectives in Histamine Research. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-7309-3_14.

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Amdrup, E., P. Funch-Jensen, and A. Tøttrup. "Management of recurrent duodenal ulcer after treatment with histamine H2-receptor antagonists and highly selective vagotomy." In Peptic Ulcer Disease: Basic and Clinical Aspects. Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5034-4_28.

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Fritsch, W. P. "Benefits and Risks of Long-term Medical Therapy with Histamine H2-receptor Antagonists in Ulcer Disease — A Physician’s View." In Prostaglandins and Leukotrienes in Gastrointestinal Diseases. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73316-1_58.

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Schumpelick, V., G. Arlt, and G. Winkeltau. "Benefits and Risks of Long-term Medical Therapy with Histamine H2-Receptor Antagonists in Ulcer Disease: A Surgeon’s View." In Prostaglandins and Leukotrienes in Gastrointestinal Diseases. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73316-1_59.

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Douer, D., I. Ben-Bassat, A. Kneller, et al. "Synergism of H2 Histamine Receptor Antagonists with Alpha-Interferon to Inhibit the Growth of Leukemic and Normal Hematopoietic Progenitors." In Acute Leukemias II. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74643-7_14.

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Conference papers on the topic "Histamine H2 receptor antagonist"

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Alfon, Jose, Sonia Sanchez-Gomez, Carolina Salcedo, et al. "Efficacy Of UR-63325, A New Histamine H4 Receptor Antagonist, In House Dust Mite-Induced Mouse Asthma Models." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1302.

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Li, Shen, Sarani Ghoshal, Gunisha Arora, et al. "Abstract 4004: The H2 receptor antagonist nizatidine inhibits carcinogenesis in two rodent models of hepatocellular carcinoma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4004.

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Patscheke, H., K. Stegmeier, W. Hornberger, Ch Staiger, and G. Neugebauer. "INHIBITION OF PLATELET ACTIVATION BY THE NOVEL THROMBOXANE RECEPTOR ANTAGONIST BM 13.505." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643469.

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The effects of BM 13.505 (4-[2-(4-Chlorobenzenesulfonylami-no)ethyl]-benzene acetic acid = BM) on human washed platelets and platelet-rich plasma (PRP) were studied in vitro and after oral application in 10 male volunteers ex vivo/in vitro. BM inhibited the shape change, aggregation and (1H)serotonin release when the platelets were activated by agents that stimulate via the thromboxane Az/prostaglandin H2 (TXA2/PGH2) receptor. Such agonists were collagen, methyl mercury chloride (methyl-Hg), arachidonic acid and the PGH2 analogue U 46,619. BM was 9 times more potent an inhibitor than sulotroba
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Zehender, H., E. C. Witte, K. Stegemeier, and A. Patscheke. "IRREVERSIBLE BLOCKADE OF THE THROMBOXANE A2/PROSTAGLANDIN H2 RECEPTOR OF HUMAN PLATELETS BY AZIDO-BSP." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643755.

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Azido-BSP (4-[2-(4-azido-benzenesulphonylamino)-ethyl]phen-oxyacetic acid) is a photolabile derivative of the competitive thromboxane A2 /prostaglandin H2 (TXA2/PGH2) receptor antagonist sulotroban (=BM 13.177). If protected from short wave light, azido-BSP reversibly inhibited the platelet shape change induced by the PGH2 analogue U 46619 but notthe shape change induced by ADP or PAF. Schild analysis revealed an apparent KD=0.2 μM with washed platelets. The irreversible inhibition requiredirradiation of the platelet suspensionwith UVlight (254 nm) for 5 minutes in the presenceof azido-BSP. Af
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