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Books on the topic 'Histological diagnostics'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

1952-, Wick Mark R., ed. Diagnostic histochemistry. Cambridge University Press, 2008.

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2

Tadrous, Paul Joseph. Diagnostic criteria handbook in histopathology: A surgical pathology vade mecum. John Wiley & Sons, 2007.

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3

C, Cook H., ed. Manual of histological techniques and their diagnostic applications. Churchill Livingstone, 1994.

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4

Yasutake, William T. Collection and preparation of fish specimens for histological examination. U.S. Dept. of the Interior, Fish and Wildlife Service, 1987.

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5

Jasani, Bharat. Immunocytochemistry in diagnostic histopathology. Churchill Livingstone, 1993.

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6

J, O'Leary Timothy. Advanced diagnostic methods in pathology: Principles, practice, and protocols. Saunders, 2003.

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7

Baak, J. P. A. Manual of quantitative pathology in cancer diagnosis and prognosis. Springer-Verlag, 1991.

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8

H, Bach P., and Baker John R. J, eds. Histochemical and immunohistochemical techniques: Applications to pharmacology and toxicology. Chapman and Hall, 1991.

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9

F, Greenleaf James, ed. Tissue characterization with ultrasound. CRC Press, 1986.

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10

Tral’, Tat’yana, Gulrukhsor Tolibova, Igor Kogan, and Anna Olina. Embryo losses. Atlas. Publishing Center RIOR, 2023. http://dx.doi.org/10.29039/978-5-907218-78-9.

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Histologic examination of abortive material is the basic approach to identify the etiology of miscarriage. Morphological diagnostics in case of embryo loss makes it possible to draw up the plan to fully prepare the woman for future pregnancy, whether spontaneous or after fertility treatment, increasing the chance of a favorable outcome. This educational book contains the data from various studies of the endometrium and abortive material undertaken at the Ott Research Institute of Obstetrics, Gynecology and Reproductology. Histology illustrations are supplemented with images of immunohistochemi

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11

Diest, P. J. van. Quantitative cyto- and histoprognosis in breast cancer. Elsevier, 1992.

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12

Automobile Quarterly: Ferraris, Rolls-Royce, Austin-Healey. Book Sales, 1987.

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13

Diagnostic Histopathology of Tumors. Churchill Livingstone, 2000.

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14

(Editor), J. M. Thijssen, and D. Nicholas (Editor), eds. Ultrasonic Tissue Characterization. Springer, 2007.

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15

Butler, Reni S. Architectural Distortion (Radial Scar). Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0030.

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Radial scars are benign lesions of the breast characterized pathologically by a fibroelastic core containing entrapped ducts and lobules that radiate outwards in a stellate pattern. This chapter, highlighting radial scar as a cause of architectural distortion, reviews its imaging features and differential diagnosis on mammography, digital breast tomosynthesis, ultrasound, and MRI; its diagnostic workup using multiple modalities; and its histological confirmation with image-guided core needle biopsy. The particular challenge of radial scar presenting as architectural distortion seen only with t

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16

Tjärnlund, Anna, and Ingrid E. Lundberg. Diagnostic and classification criteria. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0002.

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Diagnosis of idiopathic inflammatory myopathies (IIM) is based on clinical features such as subacute progress of symmetrical weakness of proximal muscle and muscle fatigue, in combination with laboratory confirmation of myopathy, including elevated muscle enzyme levels in serum and histological demonstration of skeletal muscle inflammation, as well as fibre regeneration and degeneration in muscle biopsies. Several classification criteria for IIM have historically been proposed. New classification criteria for IIM have been developed, and are based on real patient data from adult and juvenile I

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17

J, O'Leary Timothy. Advanced Diagnostic Methods in Pathology: Principles, Practice and Protocols (Advanced Diagnostic Methods in Pathology). Saunders, 2002.

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18

Wortsman, Ximena, and Gregor B. E. Jemec. Dermatologic Ultrasound with Clinical and Histologic Correlations. Springer, 2013.

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19

Wortsman, Ximena. Dermatologic Ultrasound with Clinical and Histologic Correlations. 2013.

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20

Jemec, Gregor, and Ximena Wortsman. Dermatologic Ultrasound with Clinical and Histologic Correlations. Springer, 2013.

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21

Jemec, Gregor, and Ximena Wortsman. Dermatologic Ultrasound with Clinical and Histologic Correlations. Springer, 2016.

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22

Histochemical and Immunohistochemical Techniques: Applications to pharmacology and toxicology. Springer, 1991.

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23

Diagnostic cytology and its histopathologic bases. 4th ed. Lippincott, 1992.

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24

Radiologic and histologic pathology of nontumorous diseases of bones and joints. Northbrook Pub. Co., 1990.

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25

Roberts, Ian S. D., Philip Mason, and Agnes B. Fogo. The renal biopsy. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0018.

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This chapter describes the indications for renal biopsy, the procedure, processing of the specimen and the diagnostic method applied by the nephropathologist, and illustrates the spectrum of pathologies seen. The terminology used in renal pathology is explained and illustrated. Diagnostic algorithms are presented. More detailed descriptions of the various pathologies, and in particular the rarer entities, are provided in specific chapters. The focus will be on native renal diseases. The renal biopsy is an invasive procedure associated with a risk of serious complications. The decision to perfo

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26

Newell, Mary S. Round and Punctate Calcifications. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0037.

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Round calcifications are aptly defined as calcifications that are round (or sometimes oval) in shape and smooth in contour. Punctate calcifications are a subset of round calcifications and, by definition, are <0.5 mm in size. Both these types of calcifications are considered typically benign when regional or diffuse in distribution. However, there are certain circumstances where round or punctate calcifications should be evaluated with diagnostic views and possibly recommended for biopsy. Distribution should be carefully evaluated, as should clinical circumstances. This chapter describes th

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27

Bhagavatula, Sharath K., Daniel A. T. Souza, Milana Flusberg, Stuart G. Silverman, and Paul B. Shyn. CT- and PET/CT-Guided Interventional Radiology Procedures. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190495756.003.0011.

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Computed tomography (CT) has become a valuable guidance modality for a large number of diagnostic and therapeutic percutaneous interventions. Diagnostic and therapeutic interventions guided by CT are among the most common interventional radiology procedures performed in clinical practice. Common diagnostic CT-guided interventions include tissue biopsies, fluid aspirations, and catheter drainages. Needle biopsies can be used to obtain tissue for cytologic or histologic assessment from almost any anatomical region of the body. Positron emission tomography (PET)/CT–guided diagnostic and therapeut

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28

Lennon, Rachel, and Neil Turner. The molecular basis of glomerular basement membrane disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0320_update_001.

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The glomerular basement membrane (GBM) is a condensed network of extracellular matrix molecules which provides a scaffold and niche to support the function of the overlying glomerular cells. Within the glomerulus, the GBM separates the fenestrated endothelial cells, which line capillary walls from the epithelial cells or podocytes, which cover the outer aspect of the capillaries. In common with basement membranes throughout the body, the GBM contains core components including collagen IV, laminins, nidogens, and heparan sulphate proteoglycans. However, specific isoforms of these proteins are r

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29

Bunch, Chris. Diagnosis and investigation in haematology. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0278.

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This chapter addresses the interpretation of the full blood count, blood film, bone marrow examination, and related tests in the diagnosis of haematological disorders. Examination of a stained blood film, which should always be requested if a blood count abnormality cannot readily be explained by the clinical context, may give clues to the cause of the abnormality or prove diagnostic. Examination of the bone marrow is essential to the proper evaluation and diagnosis of many haematological disorders. The simplest form of marrow examination involves needle aspiration of marrow cells from the pos

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30

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Nephrotic syndrome. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0161.

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Nephrotic syndrome is a clinical syndrome of heavy proteinuria (greater than 3.5 g per 24 hours), oedema, and hypoalbuminaemia, which is associated with hyperlipidaemia and a procoagulant state. Causes of nephrotic syndrome are traditionally classified by their histopathological descriptions. In most cases, the histological picture can have a primary (idiopathic) or secondary cause. Minimal change, membranous nephropathy, and focal segmental glomerulosclerosis account for over 60% of cases. Diabetic nephropathy and renal amyloidosis are common secondary causes of nephrotic syndrome. Nephrotic-

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31

Faquin, William C., Justin A. Bishop, James S. Lewis, Susan Müller, Lester D. R. Thompson, and John M. Wright. Tumors of the Upper Aerodigestive Tract, Ear, and Jaw. American Registry of PathologyArlington, Virginia, 2025. https://doi.org/10.55418/9781933477619.

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The head and neck region is a marvel of anatomic complexity and it encompasses a broad array of subsites, each with its own architectural nuances. This histologic richness begets a commensurate diversity of disease. Advances in our understanding of the genetic and epigenetic alterations that underlie many of these tumors have not only refined their classification, but also reshaped our approaches to diagnosis, prognosis, and therapy. This Fascicle reflects these scientific and diagnostic evolutions. Departing from prior editions this volume is organized by anatomic subsite. Each chapter delves

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32

Hart, Nicholas, and Tarek Sharshar. Diagnosis, assessment, and management of ICU-acquired weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0248.

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Intensive care unit-acquired weakness (ICU-AW) is the term applied to generalized skeletal muscle weakness developed as a result of critical illness. This condition adversely affects up to three-quarters of patients admitted to the intensive care unit and it is associated with risk factors such as illness severity and duration of mechanical ventilation. Using detailed electrophysiological tests and histological muscle sampling, ICU-AW can be classified as a neuropathy, myopathy, or a neuromyopathy. However, this detailed approach is generally only required when there is diagnostic uncertainty

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33

Kitahara, Cari M., Arthur B. Schneider, and Alina V. Brenner. Thyroid Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0044.

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Thyroid cancer, once considered relatively uncommon in the general population, is now the eighth most commonly diagnosed cancer among women worldwide, and the third most common cancer among women under 45 years of age. The incidence is substantially higher in women than men (3:1 ratio); this differential is highest between ages 15 and 39 and declines with age. Nearly all thyroid cancers derive from the follicular epithelium, and the most common histological type is papillary thyroid cancer (PTC). Incidence of thyroid cancer has been increasing in many countries since the early 1980s. This tren

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34

Probasco, John C. Paraneoplastic Neurological Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0090.

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Paraneoplastic neurological disorders (PNDs) are estimated to affect approximately 0.01% of all patients with cancer. The majority of PNDs are thought to be the byproduct of immune-mediated processes directed against tumor-related antigens, processes which are sometimes effective against a systemic cancer. The inciting cancer is often asymptomatic or occult, with patients presenting to the neurologist with a variety of neurological symptoms and signs depending on the area(s) of the central, peripheral, and autonomic nervous system involved. The diagnosis of a PND is reserved for patients with

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35

Cutter, David, and Martin Scott-Brown. Diagnosis and staging of cancer. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0324.

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The accurate diagnosis of the precise type and stage of a malignancy is a vital part of cancer management. Treatment options and decisions vary significantly between various stages of the same malignancy (e.g. treatment with radical vs palliative intent) and also between specific histological subtypes of a cancer arising from the same organ (e.g. small-cell lung cancer vs non-small-cell lung cancer). It is therefore of critical importance that as much accurate information about each individual case is obtained. This is achieved with a variety of diagnostic procedures which allow the multidisci

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36

Lai, Kar Neng, and Sydney C. W. Tang. Immunoglobulin A nephropathy diagnosis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0067_update_001.

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The defining histological hallmark of immunoglobulin A (IgA) nephropathy is the presence of IgA in the mesangium as the sole or dominant immunoreactant. Light microscopy appearances vary very widely. The most common appearance is mesangial cell proliferation and an increase in mesangial matrix. However, this is not diagnostic in the absence of immunohistology. Focal segmental proliferative or necrotizing glomerulonephritis may be seen in ‘vasculitic’ disease with or without the skin changes of Henoch–Schönlein purpura. Extracapillary proliferation and crescent formation may occur. Occasionally

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37

Noutsias, Michel, and Bernhard Maisch. Myocarditis and pericarditis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0058.

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Transition of acute myocarditis to dilated cardiomyopathy occurs in approximately 20% of patients within a follow-up period of 33 months. Recent research has revealed the adverse prognostic impact of several clinical parameters for this scenario. Acute myocarditis and its sequelae dilated cardiomyopathy and inflammatory cardiomyopathy are often caused by viral infections. Histological evaluation of endomyocardial biopsies is critical for the diagnosis of the cardiomyopathy entity and for the clinical management of around 20% of the patients. Additionally, contemporary diagnostic procedures of

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38

Noutsias, Michel, and Bernhard Maisch. Myocarditis and pericarditis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0058_update_001.

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Transition of acute myocarditis to dilated cardiomyopathy occurs in approximately 20% of patients within a follow-up period of 33 months. Recent research has revealed the adverse prognostic impact of several clinical parameters for this scenario. Acute myocarditis and its sequelae dilated cardiomyopathy and inflammatory cardiomyopathy are often caused by viral infections. Histological evaluation of endomyocardial biopsies is critical for the diagnosis of the cardiomyopathy entity and for the clinical management of around 20% of the patients. Additionally, contemporary diagnostic procedures of

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39

Andrade, M. J. Tumours and masses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0022.

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Transthoracic and transoesophageal echocardiography is the first-line diagnostic tool for imaging space-occupying lesions of the heart. Cardiac masses can be classified as tumours, thrombi, vegetations, iatrogenic material, or normal variants. Occasionally, extracardiac masses may compress the heart and create a mass effect. Cardiac masses may be suspected from the clinical presentation. This is the case in patients with an embolic event presumed of cardiac origin or in patients with infective endocarditis. Otherwise, a cardiac mass can be identified during the routine investigation of common,

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40

Andrade, Maria João, Jadranka Separovic Hanzevacki, and Ricardo Ronderos. Cardiac tumours. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0052.

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Transthoracic and transoesophageal echocardiography represent the first-line diagnostic tools for imaging space-occupying lesions of the heart. Cardiac masses can be classified as tumours, thrombi, vegetations, iatrogenic material, or normal variants. Occasionally, extracardiac masses may compress the heart and create a mass effect. Cardiac masses may be suspected from the clinical presentation. This is the case in patients with an embolic event presumed to be of cardiac origin or in patients with infective endocarditis. Otherwise, a cardiac mass can be identified during the routine investigat

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41

Keshav, Satish, and Alexandra Kent. Immunology and genetics in gastrointestinal and hepatic medicine. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0196.

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The gut has a pivotal role in immune homeostasis. It is constantly exposed to a wide array of antigens in food, and resident and consumed microorganisms. It is estimated that the number of bacterial cells in the gastrointestinal tract is tenfold greater than the number of cells in the human body. The gut needs to recognize harmful bacteria, and consequently contains the largest number of immune cells in the body. However, it must remain tolerant to commensal bacteria. Bacteria express antigens that stimulate an immunological response via the gut-associated lymphoid tissue (GALT). The GALT incl

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42

Hall, Andrew, and Shamima Rahman. Mitochondrial diseases and the kidney. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0340.

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Mitochondrial disease can affect any organ in the body including the kidney. As increasing numbers of patients with mitochondrial disease are either surviving beyond childhood or being diagnosed in adulthood, it is important for all nephrologists to have some understanding of the common renal complications that can occur in these individuals. Mitochondrial proteins are encoded by either mitochondrial or nuclear DNA (mtDNA and nDNA, respectively); therefore, disease causing mutations may be inherited maternally (mtDNA) or autosomally (nDNA), or can arise spontaneously. The commonest renal pheno

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43

Beyond second opinions: Making choices about fertility treatment. University of California Press, 1998.

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