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Relevant bibliographies by topics / HIV (Viruses) RNA splicing. Viruses

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Academic literature on the topic 'HIV (Viruses) RNA splicing. Viruses'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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Journal articles on the topic "HIV (Viruses) RNA splicing. Viruses"

1

Younis, Shady, Wael Kamel, Tina Falkeborn, et al. "Multiple nuclear-replicating viruses require the stress-induced protein ZC3H11A for efficient growth." Proceedings of the National Academy of Sciences 115, no. 16 (2018): E3808—E3816. http://dx.doi.org/10.1073/pnas.1722333115.

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The zinc finger CCCH-type containing 11A (ZC3H11A) gene encodes a well-conserved zinc finger protein that may function in mRNA export as it has been shown to associate with the transcription export (TREX) complex in proteomic screens. Here, we report that ZC3H11A is a stress-induced nuclear protein with RNA-binding capacity that localizes to nuclear splicing speckles. During an adenovirus infection, the ZC3H11A protein and splicing factor SRSF2 relocalize to nuclear regions where viral DNA replication and transcription take place. Knockout (KO) of ZC3H11A in HeLa cells demonstrated that severa

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Kim, Geon-Woo, and Aleem Siddiqui. "The role of N6-methyladenosine modification in the life cycle and disease pathogenesis of hepatitis B and C viruses." Experimental & Molecular Medicine 53, no. 3 (2021): 339–45. http://dx.doi.org/10.1038/s12276-021-00581-3.

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AbstractN6-methyladenosine (m6A) is the most prevalent modification of mammalian cellular RNAs. m6A methylation is linked to epigenetic regulation of several aspects of gene expression, including RNA stability, splicing, nuclear export, RNA folding, and translational activity. m6A modification is reversibly catalyzed by methyltransferases (m6A writers) and demethylases (m6A erasers), and the dynamics of m6A-modified RNA are regulated by m6A-binding proteins (m6A readers). Recently, several studies have shown that m6A methylation sites have been identified in hepatitis B virus (HBV) transcripts

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Mahdi, Mohamed, Tamás Richárd Linkner, Zsófia Ilona Szojka, and József Tőzsér. "Elucidating the Role of HIV-2 Viral Protein X." Proceedings 50, no. 1 (2020): 24. http://dx.doi.org/10.3390/proceedings2020050024.

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Human immunodeficiency viruses type 1 and 2 (HIV-1 and HIV-2) are the causative agents of the acquired immunodeficiency syndrome (AIDS). While both viruses share a similar structural and genomic organization, a difference in replication dynamics and the clinical course of infection is evident between the two. Patients dually infected were shown to have lower viral loads and generally a slower rate of progression to AIDS than those who are mono-infected. While the roles of the unique accessory proteins have been studied in detail for HIV-1, those of HIV-2, including viral protein X (Vpx), remai

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Paz, Sean, Michael L. Lu, Hiroshi Takata, Lydie Trautmann, and Massimo Caputi. "SRSF1 RNA Recognition Motifs Are Strong Inhibitors of HIV-1 Replication." Journal of Virology 89, no. 12 (2015): 6275–86. http://dx.doi.org/10.1128/jvi.00693-15.

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ABSTRACTReplication of the integrated HIV-1 genome is tightly regulated by a series of cellular factors. In previous work we showed that transactivation of the HIV-1 promoter is regulated by the cellular splicing factor SRSF1. Here we report that SRSF1 can downregulate the replication of B, C, and D subtype viruses by >200-fold in a cell culture system. We show that viral transcription and splicing are inhibited by SRSF1 expression. Furthermore, SRSF1 deletion mutants containing the protein RNA-binding domains but not the arginine serine-rich activator domain can downregulate viral replicat

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Huang, J., and T. J. Liang. "A novel hepatitis B virus (HBV) genetic element with Rev response element-like properties that is essential for expression of HBV gene products." Molecular and Cellular Biology 13, no. 12 (1993): 7476–86. http://dx.doi.org/10.1128/mcb.13.12.7476.

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Many viruses possess complex mechanisms involving multiple gene products and cis-regulatory elements in order to achieve a fine control of their gene expression at both transcriptional and posttranscriptional levels. Hepatitis B virus (HBV) and retroviruses share many structural and functional similarities. In this study, by genetic and biochemical analyses, we have demonstrated the existence of a novel genetic element within the HBV genome which is essential for high-level expression of viral gene products. This element is located 3' to the envelope coding region. We have shown that this gene

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Huang, J., and T. J. Liang. "A novel hepatitis B virus (HBV) genetic element with Rev response element-like properties that is essential for expression of HBV gene products." Molecular and Cellular Biology 13, no. 12 (1993): 7476–86. http://dx.doi.org/10.1128/mcb.13.12.7476-7486.1993.

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Many viruses possess complex mechanisms involving multiple gene products and cis-regulatory elements in order to achieve a fine control of their gene expression at both transcriptional and posttranscriptional levels. Hepatitis B virus (HBV) and retroviruses share many structural and functional similarities. In this study, by genetic and biochemical analyses, we have demonstrated the existence of a novel genetic element within the HBV genome which is essential for high-level expression of viral gene products. This element is located 3' to the envelope coding region. We have shown that this gene

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Karakama, Yuko, Naoya Sakamoto, Yasuhiro Itsui, et al. "Inhibition of Hepatitis C Virus Replication by a Specific Inhibitor of Serine-Arginine-Rich Protein Kinase." Antimicrobial Agents and Chemotherapy 54, no. 8 (2010): 3179–86. http://dx.doi.org/10.1128/aac.00113-10.

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ABSTRACT Splicing of messenger RNAs is regulated by site-specific binding of members of the serine-arginine-rich (SR) protein family, and SR protein kinases (SRPK) 1 and 2 regulate overall activity of the SR proteins by phosphorylation of their RS domains. We have reported that specifically designed SRPK inhibitors suppressed effectively several DNA and RNA viruses in vitro and in vivo. Here, we show that an SRPK inhibitor, SRPIN340, suppressed in a dose-dependent fashion expression of a hepatitis C virus (HCV) subgenomic replicon and replication of the HCV-JFH1 clone in vitro. The inhibitory

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Tsurutani, Naomi, Jiro Yasuda, Naoki Yamamoto, Byung-Il Choi, Motohiko Kadoki, and Yoichiro Iwakura. "Nuclear Import of the Preintegration Complex Is Blocked upon Infection by Human Immunodeficiency Virus Type 1 in Mouse Cells." Journal of Virology 81, no. 2 (2006): 677–88. http://dx.doi.org/10.1128/jvi.00870-06.

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ABSTRACT Mouse cells do not support human immunodeficiency virus type 1 (HIV-1) replication because of host range barriers at steps including virus entry, transcription, RNA splicing, polyprotein processing, assembly, and release. The exact mechanisms for the suppression, however, are not completely understood. To elucidate further the barriers against HIV-1 replication in mouse cells, we analyzed the replication of the virus in lymphocytes from human CD4/CXCR4 transgenic mice. Although primary splenocytes and thymocytes allowed the entry and reverse transcription of HIV-1, the integration eff

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Boyle, Sarah M., Vivian Ruvolo, Ashish K. Gupta, and Sankar Swaminathan. "Association with the Cellular Export Receptor CRM 1 Mediates Function and Intracellular Localization of Epstein-Barr Virus SM Protein, a Regulator of Gene Expression." Journal of Virology 73, no. 8 (1999): 6872–81. http://dx.doi.org/10.1128/jvi.73.8.6872-6881.1999.

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ABSTRACT Splicing and posttranscriptional processing of eukaryotic gene transcripts are linked to their nuclear export and cytoplasmic expression. Unspliced pre-mRNAs and intronless transcripts are thus inherently poorly expressed. Nevertheless, human and animal viruses encode essential genes as single open reading frames or in the intervening sequences of other genes. Many retroviruses have evolved mechanisms to facilitate nuclear export of their unspliced mRNAs. For example, the human immunodeficiency virus RNA-binding protein Rev associates with the soluble cellular export receptor CRM 1 (e

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Shunaeva, Anastasia, Daria Potashnikova, Alexey Pichugin, et al. "Improvement of HIV-1 and Human T Cell Lymphotropic Virus Type 1 Replication-Dependent Vectors via Optimization of Reporter Gene Reconstitution and Modification with Intronic Short Hairpin RNA." Journal of Virology 89, no. 20 (2015): 10591–601. http://dx.doi.org/10.1128/jvi.01940-15.

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ABSTRACTCell-to-cell transmission is an efficient mechanism to disseminate human immunodeficiency virus type 1 (HIV-1) and human T cell lymphotropic virus type 1 (HTLV-1). However, it has been challenging to quantify the level of cell-to-cell transmission because the virus-producing cells cannot be easily distinguished from infected target cells. We have previously described replication-dependent vectors that can quantify infection events in cocultured cells. These vectors contain an antisense-oriented promoter and reporter gene interrupted by a sense-oriented intron from the human gamma-globi

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Dissertations / Theses on the topic "HIV (Viruses) RNA splicing. Viruses"

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Exline, Colin Michael Stoltzfus C. Martin. "The positive regulation of HIV-1 Vif mRNA splicing is required for efficient virus replication." [Iowa City, Iowa] : University of Iowa, 2009. http://ir.uiowa.edu/etd/356.

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Kok, Tuckweng. "Early events in the replication cycle of human immunodeficiency virus /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phk79.pdf.

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Thesis (Ph. D.)--University of Adelaide, Dept. of Microbiology & Immunology, 1998.<br>Copy of author's previously published article on back end-paper. Includes bibliographical references (leaves 105-158).

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Williams, Claire Amy. "The role of RNA helicases in HIV-1 replication." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610047.

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Zeng, Yingying. "Modeling and structural studies of single-stranded RNA viruses." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/47630.

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My research focuses on structures of the genomes of single-stranded RNA viruses. The first project is concerned with the sequence and secondary structure of HIV-1 RNA. Based on the secondary structure that Watts et al. determined, I performed a series of analysis and the results suggested that the abundance of Adenosines at the wobble position of the codons leads to an unusual structure with numerous unpaired nucleotides. The findings indicated how the virus balances evolutionary pressures on the genomic RNA secondary structure against pressures on the sequence of the viral proteins. The secon

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Lee, Natasha Chun Yi. "RNA interference to study host factors required for human immunodeficiency virus-1 replication." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610703.

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Lindberg, Anette. "Viruses as a Model System for Studies of Eukaryotic mRNA Processing." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3729.

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Sharrocks, Katherine Elizabeth. "Host cell factors facilitating HIV-1 integration." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670169.

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Xhilaga, Miranda 1965. "Proteolytic processing of HIV-1 Gag and GagProPol precursor proteins, genomic RNA rearrangement and virion cor formation are interrelated." Monash University, Dept. of Medicine, 2001. http://arrow.monash.edu.au/hdl/1959.1/9224.

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Campling, Susan J. "The relationships among neuropsychological function, HIV-1 RNA levels, and CD4 counts." View full text, 2002.

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Mishra, Subrata H. "Structure and Energetics of RNA - Protein Interactions for HIV RREIIB Targeting Zinc Finger Proteins." unrestricted, 2008. http://etd.gsu.edu/theses/available/etd-06302008-144759/.

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Thesis (Ph. D.)--Georgia State University, 2008.<br>Title from file title page. Markus W. Germann, committee chair; Kathryn B. Grant , W. David Wilson, committee members. Electronic text (147 p. : ill. (some col.)) : digital, PDF file. Description based on contents viewed Oct. 6, 2008. Includes bibliographical references.

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Books on the topic "HIV (Viruses) RNA splicing. Viruses"

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Yechiel, Becker, ed. Viral messenger RNA: Transcription, processing, splicing, and molecular structure. Nijhoff, 1985.

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2

Matthews, Philippa C. Infections caused by RNA viruses. Edited by Philippa C. Matthews. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737773.003.0009.

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This chapter consists of short notes, diagrams, maps, and tables to summarize RNA viruses that are significant causes of disease in the tropics and subtropics. This includes measles, polio, hepatitis A, C, and E viruses, rabies, arboviruses, and viral haemorrhagic fevers. The chapter also includes sections on important retroviruses, HIV, and human T-lymphotropic virus. For ease of reference, each topic is broken down into sections, including classification, epidemiology, microbiology, pathophysiology, clinical syndromes, diagnosis, treatment, and prevention.

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Karl, Nicholson, ICN Pharmaceuticals inc, and Royal Society of Medicine (Great Britain), eds. HIV and other highly pathogenic viruses. Royal Society of Medicine Services, 1989.

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4

Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur, and Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into

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Grant, Warren, and Martin Scott-Brown. Principles of oncogenesis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0322.

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It is obvious that the process of developing cancer—oncogenesis—is a multistep process. We know that smoking, obesity, and a family history are strong independent predictors of developing malignancy; yet, in clinics, we often see that some heavy smokers live into their nineties and that some people with close relatives affected by cancer spend many years worrying about a disease that, in the end, they never contract. For many centuries scientists have struggled to understand the process that make cancer cells different from normal cells. There were those in ancient times who believed that tumo

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M, Hardy Leslie, and Institute of Medicine (U.S.). Committee on Prenatal and Newborn Screening for HIV Infection., eds. HIV screening of pregnant women and newborns. National Academy Press, 1991.

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HIV Screening of Pregnant Women And Newborns. Natl Academy Pr, 1990.

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Book chapters on the topic "HIV (Viruses) RNA splicing. Viruses"

1

Erickson, J., and D. Kempf. "Structure-based design of symmetric inhibitors of HIV-1 protease." In Positive-Strand RNA Viruses. Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9326-6_3.

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Emini, E. A., V. W. Byrnes, J. H. Condra, W. A. Schleif, and V. V. Sardana. "The genetic and functional basis of HIV-1 resistance to nonnucleoside reverse transcriptase inhibitors." In Positive-Strand RNA Viruses. Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9326-6_2.

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Allison, Robert D., and Shyam Kottilil. "Host Immune Responses in HIV Infection." In RNA Viruses. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812833808_0003.

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Montano, Monty, and Paola Sebastiani. "Efforts to Characterize Host Response to HIV-1 Infection." In RNA Viruses. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812833808_0001.

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Klase, Zachary A., Kuan-Teh Jeang, and Fatah Kashanchi. "HIV-1 and RNA Interference — Examining a Complex System." In RNA Viruses. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812833808_0004.

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Gee, Katrina, Sasmita Mishra, Wei Ma, Marko Kryworuchko, and Ashok Kumar. "Signaling Pathways Activated by HIV and Their Impact on Immune Responses." In RNA Viruses. WORLD SCIENTIFIC, 2009. http://dx.doi.org/10.1142/9789812833808_0002.

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Martin Stoltzfus, C. "Chapter 1 Regulation of HIV-1 Alternative RNA Splicing and Its Role in Virus Replication." In Advances in Virus Research. Elsevier, 2009. http://dx.doi.org/10.1016/s0065-3527(09)74001-1.

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Crawford, Dorothy H. "3. Kill or be killed." In Viruses: A Very Short Introduction. Oxford University Press, 2018. http://dx.doi.org/10.1093/actrade/9780198811718.003.0004.

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‘Kill or be killed’ shows how viruses survive—they must reproduce before the host either dies or its immune system recognizes and eliminates them. The transmission routes of viruses such as flu, measles, common cold, herpes simplex virus, HIV, Epstein–Barr, and hepatitis B are discussed. How do we fight viruses? All living organisms have defences against invading viruses. Vertebrates, and possibly some invertebrates, are immune to re-infection by the same virus. Another protective mechanism, used by plants, but also by insects and other animal species, is gene silencing by RNA interference. The human immune response is explained, discussing the role of lymphocytes and immunopathology, where the immune response may actually harm the body.

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Vlasova-St. Louis, Irina, Andrew Gorzalski, and Mark Pandori. "Diagnostic Applications for RNA-Seq Technology and Transcriptome Analyses in Human Diseases Caused by RNA Viruses." In Applications of RNA-Seq in Biology and Medicine [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99156.

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Human diseases caused by single-stranded, positive-sense RNA viruses, are among the deadliest of the 21st Century. In particular, there are two notable standouts: human immunodeficiency virus (HIV) and severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Detection of these disease-causing viral transcripts, by next-generation RNA sequencing (RNA-Seq), represents the most immediate opportunity for advances in diagnostic, therapeutic, and preventive applicability in infectious diseases (e.g., AIDS and COVID-19). Moreover, RNA-Seq technologies add significant value to public health studies by first, providing real-time surveillance of known viral strains, and second, by the augmentation of epidemiological databases, construction of annotations and classifications of novel sequence variants. This chapter intends to recapitulate the current knowledge of HIV and SARS-CoV-2 transcriptome architecture, pathogenicity, and some features of the host immune response. Additionally, it provides an overview of recent advances in diagnostic sequencing methodologies and discusses the future challenges and prospects on the utilization of RNA-Seq technologies.

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Bartlett, John G., Robert R. Redfield, and Paul A. Pham. "Laboratory Tests." In Bartlett's Medical Management of HIV Infection. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190924775.003.0002.

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This chapter covers the following topics: HIV viruses including viral variants (group O and group N); immune responses to HIV and detection markers; HIV serologic tests; initial tests to detect HIV antibody; confirmatory tests to detect antibody, antigen, or RNA; false-negative and false-positive results; testing strategies and algorithms; alternative testing strategies for resource-limited countries; home tests; tests that use oral fluids; molecular tests to detect and monitor HIV infection; kinetics of viral nucleic acid production (including qualitative and quantitative RNA tests); HIV DNA assessment; uses of viral load tests; rapid molecular tests; resistance testing (including purpose and scope); resistance test methods; tests for sexually transmitted infections (STIs); screening tests for other infectious agents; and recommended reading.

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