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Journal articles on the topic 'Hofmann, Mark'

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Published: 4 June 2021

Last updated: 27 July 2025

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1

Hardy, B. Carmon, and Richard E. Turley. "Victims: The LDS Church and the Mark Hofmann Case." Journal of American History 80, no. 4 (1994): 1546. http://dx.doi.org/10.2307/2080746.

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2

Shupe, Anson, and Richard E. Turley. "Victims: The LDS Church and the Mark Hofmann Case." Journal for the Scientific Study of Religion 32, no. 2 (1993): 208. http://dx.doi.org/10.2307/1386823.

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3

Evans, Max J. "Victims: The LDS Church and the Mark Hofmann Case." Utah Historical Quarterly 62, no. 2 (1994): 196–97. http://dx.doi.org/10.2307/45062168.

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4

Roberts, Allen D. ""The Truth Is the Most Important Thing": The New Mormon History According to Mark Hofmann." Dialogue: A Journal of Mormon Thought 20, no. 4 (1987): 87–96. http://dx.doi.org/10.2307/45228111.

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5

Schenk, Robyn L., Kaja Kostyrko, Johannes Popow, et al. "Abstract 6506: SOS1 inhibition in combination with HER2 inhibition in models of HER2-driven cancer." Cancer Research 84, no. 6_Supplement (2024): 6506. http://dx.doi.org/10.1158/1538-7445.am2024-6506.

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Abstract Cancers driven by amplifications or mutations of the gene ERBB2 (coding for the receptor tyrosine kinase (RTK) HER2) have been the focus of HER2-targeting antibodies as well as small molecule inhibitors. Despite initial efficacy, resistance to these HER2-targeted therapies inevitably develops. One strategy to tackle this resistance is to identify rational combinations to address common pathways/factors that contribute to resistance. One such common mechanism of resistance is reactivation of the Mitogen-activated protein kinase (MAPK) pathway. SOS1 is a guanine nucleotide exchange fact

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6

Thatikonda, Venu, Kaja Kostyrko, Sabine Jurado, et al. "Abstract 1926: MRAS the forgotten member of the GTPase superfamily plays a role in KRASG12Ci resistance." Cancer Research 84, no. 6_Supplement (2024): 1926. http://dx.doi.org/10.1158/1538-7445.am2024-1926.

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Abstract KRASG12C inhibitors have achieved impressive results in the clinic. However, intrinsic or acquired resistance to therapy invariably develops leading to disease progression in most treated patients. Emerging pre-clinical and clinical evidence points to multiple mechanisms being responsible for resistance to KRASG12C inhibitors, including receptor tyrosine kinase (RTK) activation, KRASG12C amplification, activation of alternative signaling pathways including PI3K, mTOR, YAP/TAZ, acquisition of KRAS mutations that would preclude compound binding (e.g. R68S, Y96C/D, H95D/Q/R), conversion

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Mark, C. Kruit, A. van Buchem Mark, A. M. Hofman Paul, et al. "Migraine as a Risk Factor for Subclinical Brain Lesions." JAMA 291, no. 4 (2004): 427–34. https://doi.org/10.5281/zenodo.1038403.

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Mark C. Kruit, Mark A. van Buchem, Paul A. M. Hofman, Jacobus T. N. Bakkers, Gisela M. Terwindt, Michel D. Ferrari, Lenore J. Launer (2004): Migraine as a Risk Factor for Subclinical Brain Lesions. JAMA 291 (4): 427-434, DOI: 10.5281/zenodo.1038403

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8

Hofmann, Marco H., Sabine Jurado, Daniel Gerlach, et al. "Abstract 3255: Probing and overcoming KRASG12C inhibitor resistance by combination with a pan-KRAS SOS1 inhibitor." Cancer Research 82, no. 12_Supplement (2022): 3255. http://dx.doi.org/10.1158/1538-7445.am2022-3255.

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Abstract The recent accelerated approval of the KRASG12C mutant-selective inhibitor sotorasib (AMG 510) for the treatment of 2nd line KRASG12C mutation-positive NSCLC patients marks the first approved targeted therapy for tumors with any KRAS mutation. While KRASG12C inhibitors deliver clinical benefit, most patients who achieved an objective response ultimately progressed. Recent insights into clinical KRASG12C inhibitor resistance identified reactivation of the RAS/MAPK pathway as a common putative driver mechanism of resistance. Multiple ongoing trials seek to augment responses to KRASG12C

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Rhee, Jongeun, Vicky C. Chang, Weiyin Zhou, et al. "Abstract 7368: Serum concentrations of per- and polyfluoroalkyl substances and mosaic loss of chromosome Y among men in the Prostate, Lung, Colorectal and Ovarian (PLCO) Cancer Screening Trial." Cancer Research 85, no. 8_Supplement_1 (2025): 7368. https://doi.org/10.1158/1538-7445.am2025-7368.

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Abstract Per- and polyfluoroalkyl substances (PFAS) are persistent organic pollutants detectable in the serum of most U.S. adults. Some PFAS are established or possible human carcinogens. Mechanistic studies in human cells and animals suggest PFAS may have genotoxic effects through indirect DNA damage caused by induction of oxidative stress or other mechanisms; however, evidence from human populations is limited. We investigated the relationship between serum PFAS concentrations and mosaic loss of chromosome Y (mLOY) as a biomarker of genotoxicity and genomic instability among men. We analyzed

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Rhee, Jongeun, Jani Koponen, Joshua N. Sampson, et al. "Abstract 840: Serum concentrations of per- and polyfluorinated substances and risk of non-Hodgkin lymphoma." Cancer Research 84, no. 6_Supplement (2024): 840. http://dx.doi.org/10.1158/1538-7445.am2024-840.

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Abstract Per- and polyfluoroalkyl substances (PFAS) are persistent organic pollutants, some immunotoxic, that are detectable in the serum of most U.S. adults. Some studies of highly-exposed individuals have suggested positive associations between PFAS and non-Hodgkin lymphoma (NHL); however, it is unknown whether associations exist at lower exposure levels that are more common in the general population. To address this knowledge gap, we conducted a nested case-control study investigating serum PFAS concentrations and NHL within the Prostate, Lung, Colorectal and Ovarian Cancer Screening Trial.

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Norgard, Robert J., Pratha Budhani, Sarah O'Brian, et al. "Abstract B23: Combination of SOS1::KRAS inhibitor with a MEK inhibitor reconfigures the immune tumor microenvironment of KRASG12D pancreatic ductal adenocarcinomas and sensitizes to immunotherapy." Cancer Immunology Research 10, no. 12_Supplement (2022): B23. http://dx.doi.org/10.1158/2326-6074.tumimm22-b23.

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Abstract KRAS is most frequently mutated in pancreatic adenocarcinomas (PDAC) with more than 90% of cases having a KRAS mutation. This leads to constitutive activation of the downstream signaling pathways, driving tumor cell proliferation whilst co-opting the tumor microenvironment (TME) to promote tumorigenesis and immune escape. The mutant specific KRASG12C inhibitor (AMG510) has been shown to suppress tumor growth and reconfigure the immune TME. However, covalent binding KRASG12C inhibitors have no efficacy on other KRAS alleles, such as G12D and V, the majority of KRAS mutations in PDAC (3

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Tarashevska, Yu Ye, and Ye G. Shyyan. "VARIETY AND PRINCIPLES OF COOPERATION OF DOUBLE CROWNS OF TELESCOPIC CONNECTIN." Ukrainian Dental Almanac, no. 2 (June 19, 2019): 49–54. http://dx.doi.org/10.31718/2409-0255.2.2019.10.

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For today, problem of fixing of partial removable prostheticappliances, and more precisely it remains not fully decidedthe term of endurance and worn down of friction propertiesof telescopic connections. Therefore, the search for improvement of fixative properties of telescopic connection sremains actual. Material and methods: Analytical study of scientific sources on the problems of replacing defects in dentition with removable dentures with fixation on telescopic crowns. The aim of this work consists in that, to educe, to estimateand compare, on thebasis of the dynamic supervisio

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Waizenegger, Irene C., Hengyu Lu, Claus Thamer, et al. "Abstract 2667: Trial in progress: Phase 1 study of BI 1823911, an irreversible KRASG12C inhibitor targeting KRAS in its GDP-loaded state, as monotherapy and in combination with the pan-KRAS SOS1 inhibitor BI 1701963 in solid tumors expressing KRASG12C mutation." Cancer Research 82, no. 12_Supplement (2022): 2667. http://dx.doi.org/10.1158/1538-7445.am2022-2667.

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Abstract Inhibition of KRASG12C mediated signaling and the therapeutic impact in non-small cell lung cancer (NSCLC) whose tumors carry this mutation was demonstrated clinically by sotorasib and adagrasib leading to approval of sotorasib in KRASG12C mutant NSCLC. These encouraging data are currently changing the treatment paradigm for patients with KRASG12C-mutated tumors. However only a fraction of patients is initially responding to these compounds and patients who achieved an objective response ultimately progressed on-treatment. It became clear from these studies that KRASG12C inhibitors re

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Liu, Qinghang, and Polly A. Hofmann. "Modulation of protein phosphatase 2a by adenosine A1 receptors in cardiomyocytes: role for p38 MAPK." American Journal of Physiology-Heart and Circulatory Physiology 285, no. 1 (2003): H97—H103. http://dx.doi.org/10.1152/ajpheart.00956.2002.

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Adenosine A1 receptor activation causes protein phosphatase 2a (PP2a) activation in ventricular myocytes. This attenuates β-adrenergic functional effects in the heart (Liu Q and Hofmann PA. Am J Physiol Heart Circ Physiol 283: H1314–H1321, 2002). The purpose of the present study was to identify the signaling pathway involved in the translocation/activation of PP2a by adenosine A1 receptors in ventricular myocytes. We found that N6-cyclopentyladenosine (CPA; an adenosine A1 receptor agonist)-induced PP2a translocation was blocked by p38 MAPK inhibition but not by JNK inhibition. CPA increased p

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Sigaud, Romain, Lisa Rösch, Charlotte Gatzweiler, et al. "Abstract 5221: The first-in-class ERK inhibitor ulixertinib (BVD-523) shows activity in MAPK-driven pediatric low-grade glioma models as single agent and in combination with MEK inhibitors or senolytics." Cancer Research 82, no. 12_Supplement (2022): 5221. http://dx.doi.org/10.1158/1538-7445.am2022-5221.

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Abstract Ulixertinib (BVD-523) is a well-tolerated, orally delivered, catalytic ERK1/2 inhibitor, which has shown promising responses in adult patients with mitogen-activated protein kinase (MAPK)-driven solid tumors. Pediatric low grade gliomas (pLGGs) are the most common pediatric brain tumors, with the most frequent driving alterations (KIAA:BRAF fusion, BRAF V600E mutation) occurring in the MAPK pathway. To investigate the anti-tumoral activity of ulixertinib in pLGG, cell lines recapitulating both main MAPK alterations were used: DKFZ-BT66 (pilocytic astrocytoma; KIAA:BRAF fusion) and BT4

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16

Colombo Timelli, Maria. "Quand la peinture était dans les livres. Mélanges en l’honneur de François Avril, sous la direction de Mara Hofmann et Caroline Zöhl." Studi Francesi, no. 157 (LIII | I) (May 1, 2009): 149–50. http://dx.doi.org/10.4000/studifrancesi.8182.

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17

Kostyrko, Kaja, Melanie Hinkel, Marco H. Hofmann, and Francesca Trapani. "Abstract 1083: MEKi and SOS1i as a combination treatment option for KRAS mutant endometrial cancer." Cancer Research 83, no. 7_Supplement (2023): 1083. http://dx.doi.org/10.1158/1538-7445.am2023-1083.

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Abstract Endometrial cancer (EC) is the most common cancer of the female genital tract, resulting in over 75,000 deaths per year worldwide. Despite recent advances in medical oncology research, chemo- and radiotherapy are still the standard of care treatments for recurrent or metastatic EC and prognosis for these patients remains poor, with median survival rates of less than one year. Approximately 15-30% of the more common type I EC harbors oncogenic KRAS mutations, which have been linked to disease progression. Preclinical studies have shown that MEK inhibitors exhibit strong anti-tumor acti

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18

Koshias, Andreas, Emma Gray, Graeme Currie, and Jennifer Cleland. "28 Do not attempt resuscitation: university of aberdeen student perspectives." BMJ Supportive & Palliative Care 7, no. 3 (2017): A357.2—A358. http://dx.doi.org/10.1136/bmjspcare-2017-001407.28.

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IntroductionDo Not Attempt Resuscitation Orders(DNAR) and their contemporary counterparts are cornerstones of End of Life care and as such, of importance within medical education. Previous research indicates the need for a better understanding of patient and physicians perceptions of DNAR topics.Aims and methodsThe objective of the study was to explore medical students(MS) and non-medical students perspectives on DNAR discussions(DNARD), and explore any differences. This was a cross-sectional questionnaire study. MS and Education students(ES) were asked how they felt regarding DNARD taking pla

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19

Sale, Matthew J., Neal Mukherjee, Martín Koch, et al. "Abstract 2080: SOS1 inhibition is an effective therapeutic strategy in SOS1-mutant cancer." Cancer Research 84, no. 6_Supplement (2024): 2080. http://dx.doi.org/10.1158/1538-7445.am2024-2080.

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Abstract SOS1 is a principal guanine nucleotide exchange factor for canonical RAS GTPases and activates RAS signaling. Mutations in SOS1 are found in several prominent cancer types, including 2-4% of NSCLC, CRC, and melanoma. The recent development of novel SOS1 inhibitors (SOS1i), including BI-3406, that block interaction between SOS1 and RAS, presents a potential therapy for SOS1-driven cancers. Here we show that the majority of SOS1 mutations observed in patients enhance RAS guanine nucleotide exchange in cells and activate RAS-MAPK signaling. We treated 11 SOS1 mutant cancer cell lines wit

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20

Gidley, Mick. "Topographical Portraits: Seven Views of Richard Avedon'sIn the American West." Prospects 30 (October 2005): 685–713. http://dx.doi.org/10.1017/s0361233300002234.

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This essay addresses some of the ambiguities of the western portrait project conducted by Richard Avedon (1923–2004) between 1978 and 1984, a project that resulted in both a major exhibition, initially mounted at the Anion Carter Museum of Western Art in Fort Worth, Texas, and the portfolio-sized bookIn the American West(1985). The book version, insofar as such a thing is possible, echoed between covers, in sequencing and scale, the design experience of the exhibition, with its large prints of the images, even though, according the person who supervised the photographic printing, Avedon's regu

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21

Weiss, Andreas, Hans Voshol, Diana Graus Porta, et al. "Abstract 4026: JDQ443, a covalent inhibitor of KRASG12C with a novel binding mode, shows broad antitumor activity in KRASG12C preclinical models as a single agent and in combination with inhibitors of SHP2, MEK or CDK4/6." Cancer Research 82, no. 12_Supplement (2022): 4026. http://dx.doi.org/10.1158/1538-7445.am2022-4026.

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Abstract Background: Oncogenic mutations occurring in the KRAS component of the RAS/MAPK pathway slow nucleotide cycling between its active (GTP-bound) and inactive (GDP-bound) states, shifting it towards the active state and increasing oncogenic signaling. Among these mutations, the glycine-to-cysteine mutation of amino acid 12 (KRASG12C), found in ~13% of non-small cell lung cancer (NSCLC) and ~4% of colorectal cancer (CRC), can be specifically targeted and irreversibly locked in the inactive state by covalent modification of Cys12. We have previously reported the discovery and preclinical p

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22

Schmelzle, Tobias, Emilie Chapeau, Daniel Bauer, et al. "Abstract LB319: IAG933, a selective and orally efficacious YAP1/WWTR1(TAZ)-panTEAD protein-protein interaction inhibitor with pre-clinical activity in monotherapy and combinations." Cancer Research 83, no. 8_Supplement (2023): LB319. http://dx.doi.org/10.1158/1538-7445.am2023-lb319.

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Abstract The YAP-TEAD protein-protein interaction (PPI) is a critical event known to mediate YAP oncogenic functions downstream of the Hippo pathway. Current advanced pharmacological agents which aim at inhibiting YAP-TEAD oncogenic function do so by engaging into the lipid pocket of TEAD. Thereby the consequences of a direct pharmacological disruption of the interface of YAP and TEADs remain unexplored. Here we report the identification of IAG933, the first molecule able to potently and directly disrupt the YAP/TAZ-TEADs PPI with suitable properties to enter in clinical trial. The path to dru

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Sale, Matthew J., Neal Mukherjee, Regina Ruzicka, et al. "Abstract 1715: SOS1 inhibition is an effective therapeutic strategy in SOS1-mutant cancer." Cancer Research 83, no. 7_Supplement (2023): 1715. http://dx.doi.org/10.1158/1538-7445.am2023-1715.

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Abstract SOS1 is a principal guanine nucleotide exchange factor for canonical RAS GTPases and activates RAS signaling. Mutations in SOS1 are found in 3-5% of non-small cell lung cancers (NSCLC), colorectal cancers (CRC) and melanomas. The recent development of novel SOS1 inhibitors, including BI-3406 and BI-1701963, that block interaction between SOS1 and RAS, presents a potential therapy for SOS1-driven cancers. Here we show that the majority of SOS1 mutations observed in patients enhance RAS guanine nucleotide exchange in cells and activate RAS-MAPK signaling. Of the 11 available SOS1-mutant

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Salari, Chiara, Jean-Marc Hofman, Magali Mélandri, and David-Sean Thomas. "Hofman, Jean-Marc, Mélandri Magali et Thomas David-Sean. 2020 (dir.). Paris 1910-1937. Promenades dans les collections Albert-Kahn. Cat. exp. (Paris, Cité de l’architecture et du patrimoine, 16 septembre 2020-5 juillet 2021)." Histoires-monde de la photographie, no. 3 (July 7, 2022): 197–98. http://dx.doi.org/10.54390/photographica.679.

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KITLV, Redactie. "Book reviews." New West Indian Guide / Nieuwe West-Indische Gids 85, no. 1-2 (2011): 99–163. http://dx.doi.org/10.1163/13822373-90002439.

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Globalization and the Po st-Creole Imagination: Notes on Fleeing the Plantation,by Michaeline A. Crichlow with Patricia Northover (reviewed by Raquel Romberg)Afro-Caribbean Religions: An Introduction to their Historical, Cultural, and Sacred Traditions, by Nathaniel Samuel Murrell (reviewed by James Houk) Africas of the Americas: Beyond the Search for Origins in the Study of Afro-Atlantic Religions, edited by Stephan Palmié (reviewed by Aisha Khan) Òrìṣà Devotion as World Religion: The Globalization of Yorùbá Religious Culture, edited by Jacob K. Olupona & Terry Rey (reviewed by Brian Braz

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Yvard, Catherine. "Quand la peinture était dans les livres. Mélanges en l’honneur de François Avril, éd. par Mara Hofmann et Caroline Zöhl, à l’occasion de la remise du titre de docteur honoris causa de la Freie Universität Berlin. Préface d’Eberhard König. Paris, Brepols-Bibliothèque nationale de France, 2007, 503 p., ill." Bulletin du bibliophile N° 351, no. 1 (2010): 169–72. http://dx.doi.org/10.3917/bubib.351.0181.

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Amin, Roshdi Soliman, Mahmoud Ahmed Rabab, Abdalla Ahmed, and Soliman Mahmoud. "Study of combination of ascorbic acid and N-acetylcysteine in prevention of contrast induced nephropathy in patients with moderate renal insufficiency." Biolife 5, no. 4 (2022): 527–34. https://doi.org/10.5281/zenodo.7393076.

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<strong>ABSTRACT</strong> Introduction and aims: Despite fluid administration is the most effective strategy in contrast induced nephropathy (CIN) prevention, still the estimated prevalence of CIN up to 50% in high-risk patients. There is still a great debate as regarding the administration of antioxidants in reduction of CIN incidence. This study aims to determine whether addition of ascorbic acid to IV acetylcysteine could reduce CIN prevalence in patients with moderate chronic kidney disease. Methods: We conducted prospective, randomized, double blind, placebo-controlled trial on 106 patien

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Hafida, Jaghror, Serieyssol Karen, Beauger Aude, Loukili Khadija, and Fadli Mohamed. "Diatom Diversity and Ecology of the Sebou Watershed (Morocco)." Biolife 5, no. 4 (2022): 564–77. https://doi.org/10.5281/zenodo.7393291.

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<strong>ABSTRACT</strong> This study investigates biodiversity and structure of spring diatoms associations from Sebou Basin, one of Morocco's largest superficial hydrosystems. The physico-chemical results reflect both anthropogenic and natural pollution, mainly from mineral sources. 226 taxa were identified, 92% are pennates. The result of Shannon and equitability indices showed that diatom populations were generally diversified, which depends on degree of specific richness at each station. Thus, the higher number of taxa in site Og (120 species) reflects an ecologically more stable and m

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"Victims: the LDS Church and Mark Hofmann case." Choice Reviews Online 30, no. 06 (1993): 30–3462. http://dx.doi.org/10.5860/choice.30-3462.

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Pollock, Zach, and Camille Partin. "Geobiology of the Paleoproterozoic Belcher Group, Nunavut, Canada." USURJ: University of Saskatchewan Undergraduate Research Journal 6, no. 1 (2020). http://dx.doi.org/10.32396/usurj.v6i1.507.

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The ~2.0-1.8 Ga (billion years old) Belcher Group on the Belcher Islands in Nunavut provide a unique opportunity for studying Paleoproterozoic geobiology. The Belcher Group includes a sequence of low metamorphic grade peritidal carbonate rocks that preserve putative microbiota, as first described by Hofmann and Jackson (1969). Microbial mats, including stromatolites, are abundant in the peritidal carbonate succession. Additionally, morphologies possibly related to blue-green algae were first described in granular iron formation rocks of the Belcher Group by Moore (1918). The Belcher Group micr

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Stevens, Ian. "The Epistemological Consequences of Artificial Intelligence, Precision Medicine, and Implantable Brain-Computer Interfaces." Voices in Bioethics 10 (June 30, 2024). http://dx.doi.org/10.52214/vib.v10i.12654.

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ABSTRACT I argue that this examination and appreciation for the shift to abductive reasoning should be extended to the intersection of neuroscience and novel brain-computer interfaces too. This paper highlights the implications of applying abductive reasoning to personalized implantable neurotechnologies. Then, it explores whether abductive reasoning is sufficient to justify insurance coverage for devices absent widespread clinical trials, which are better applied to one-size-fits-all treatments. INTRODUCTION In contrast to the classic model of randomized-control trials, often with a large num

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Huot, Jean-Louis. "Jean-Marc Hofman & Emmanuel Pénicaut (dir.), Le Crac des Chevaliers. Chroniques d’un rêve de pierre." Syria, June 4, 2019. http://dx.doi.org/10.4000/syria.8065.

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Nordkvelle, Yngve. "Editorial Vol 2 - issue 2." Seminar.net 2, no. 2 (2006). http://dx.doi.org/10.7577/seminar.2514.

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This journal has a complex subtitle: Media, technology and lifelong learning. The subtitle will to many of our readers be perceived as a synonymous to “ICT in education”. However, ICT in education is strongly influenced by informatics and psychology. Even if schools are main receivers of educational technology, not many inventions in the field stem from the educational field itself. There are many tendencies reminding us of the continual conflict between technology and education. The task of this journal has the aim to discuss media and technology on educational grounds. One might think that i

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"August Wilhelm von Hofmann Medal: M. Quack and S. Shaik / Van't Hoff Prize: G. Meijer and F. Merkt / Janssen Pharmaceutica Prize: I. Marek." Angewandte Chemie International Edition 51, no. 33 (2012): 8150. http://dx.doi.org/10.1002/anie.201205476.

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"August-Wilhelm-von-Hofmann- Denkmünze: M. Quack und S. Shaik / van't Hoff Preis: Preis für G. Meijer und F. Merkt / Janssen-Pharmaceutica-Preis: Preis für I. Marek." Angewandte Chemie 124, no. 33 (2012): 8272. http://dx.doi.org/10.1002/ange.201205476.

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Binh, Luu Thi, Le Thi Thanh Huong, Nguyen Trung Thanh, and Nguyen Phu Hung. "All Trans Retinoic Acid Regulates Expression of Genes Invoved in Cell Senescence Pathway in Gastric Cancer Cell Line MKN45." VNU Journal of Science: Natural Sciences and Technology 35, no. 4 (2019). http://dx.doi.org/10.25073/2588-1140/vnunst.4907.

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Cellular senescence is a state in wich cells can not longer divide. The dysregulation of the cell senescence signaling pathway could lead to uncontrolled cell proliferation and formation of malignant cells. Intervening in cell senessence signaling pathway to bring cancer cells back to cell senessence state is a potential way in cancertreatment. Recent studies show that tumor cells can undergo celluar senessencestate by using special chemotherapy. In this study, we indicatedthat All trans retinoic acid (ATRA) is able to influence the expression of genes involved in cell senessence signaling pat

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Stauff, Markus. "Non-Fiction Transmedia: Seriality and Forensics in Media Sport." M/C Journal 21, no. 1 (2018). http://dx.doi.org/10.5204/mcj.1372.

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At last year’s Tour de France—the three-week cycling race—the winner of one stage was disqualified for allegedly obstructing a competitor. In newspapers and on social media, cycling fans immediately started a heated debate about the decision and about the actual course of events. They uploaded photographs and videos, which they had often edited and augmented with graphics to support their interpretation of the situation or to direct attention to some neglected detail (Simpson; "Tour de France").Due to their competitive character and their audience’s partisanship, modern media sports continuous

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sprotocols. "Registered report: Widespread potential for growth-factor-driven resistance to anticancer kinase inhibitors2." January 9, 2015. https://doi.org/10.5281/zenodo.13851.

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Authors: Edward Greenfield, Erin Griner, The Reproducibility Project: Cancer Biology† ### Abstract The [Reproducibility Project: Cancer Biology](https://osf.io/e81xl/wiki/home/) seeks to address growing concerns about reproducibility in scientific research by conducting replications of 50 papers in the field of cancer biology published between 2010 and 2012. This Registered Report describes the proposed replication plan of key experiments from “Widespread potential for growth-factor-driven resistance to anticancer kinase inhibitors” by Wilson and colleagues, published in Nature in 2012 (Wilson

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