Relevant bibliographies by topics / Homeostasis. eng

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Homeostasis. eng'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 February 2022

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Journal articles on the topic "Homeostasis. eng"

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Yambulatov, A. M., O. S. Ustinova, and K. P. Luzhetskiy. "Violation of homeostasis of the main types of exchange and immune resistance status in children with subclinical hypovitaminosis in conditions of exposure to chemical environmental factors." Health Risk Analysis, no. 1 (January 2016): 77–86. http://dx.doi.org/10.21668/health.risk/2016.1.09.eng.

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Franco, Claudio A., and Holger Gerhardt. "Blood flow boosts BMP signaling to keep vessels in shape." Journal of Cell Biology 214, no. 7 (2016): 793–95. http://dx.doi.org/10.1083/jcb.201609038.

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Bone morphogenic proteins (BMPs) and blood flow regulate vascular remodeling and homeostasis. In this issue, Baeyens et al. (2016. J. Cell Biol. http://dx.doi.org/10.1083/jcb.201603106) show that blood flow sensitizes endothelial cells to BMP9 signaling by triggering Alk1/ENG complexing to suppress cell proliferation and to recruit mural cells, thereby establishing endothelial quiescence.
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Rossi, Elisa, Alexandre Kauskot, François Saller, et al. "Endoglin Is an Endothelial Housekeeper against Inflammation: Insight in ECFC-Related Permeability through LIMK/Cofilin Pathway." International Journal of Molecular Sciences 22, no. 16 (2021): 8837. http://dx.doi.org/10.3390/ijms22168837.

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Endoglin (Eng) is an endothelial cell (EC) transmembrane glycoprotein involved in adhesion and angiogenesis. Eng mutations result in vessel abnormalities as observed in hereditary hemorrhagic telangiectasia of type 1. The role of Eng was investigated in endothelial functions and permeability under inflammatory conditions, focusing on the actin dynamic signaling pathway. Endothelial Colony-Forming Cells (ECFC) from human cord blood and mouse lung/aortic EC (MLEC, MAEC) from Eng+/+ and Eng+/− mice were used. ECFC silenced for Eng with Eng-siRNA and ctr-siRNA were used to test tubulogenesis and p
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Vlassara, Helen, Jaime Uribarri, Weijing Cai, and Gary Striker. "Advanced Glycation End Product Homeostasis." Annals of the New York Academy of Sciences 1126, no. 1 (2008): 46–52. http://dx.doi.org/10.1196/annals.1433.055.

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Gastelum, Cassandra, Lynnea Perez, Jennifer Hernandez, et al. "Adaptive Changes in the Central Control of Energy Homeostasis Occur in Response to Variations in Energy Status." International Journal of Molecular Sciences 22, no. 5 (2021): 2728. http://dx.doi.org/10.3390/ijms22052728.

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Energy homeostasis is regulated in coordinate fashion by the brain-gut axis, the homeostatic energy balance circuitry in the hypothalamus and the hedonic energy balance circuitry comprising the mesolimbcortical A10 dopamine pathway. Collectively, these systems convey and integrate information regarding nutrient status and the rewarding properties of ingested food, and formulate it into a behavioral response that attempts to balance fluctuations in consumption and food-seeking behavior. In this review we start with a functional overview of the homeostatic and hedonic energy balance circuitries;
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Broocks, G., H. Kniep, A. Kemmling, et al. "Effect of intravenous alteplase on ischaemic lesion water homeostasis." European Journal of Neurology 27, no. 2 (2019): 376–83. http://dx.doi.org/10.1111/ene.14088.

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Larkin, Jennie E., Tohei Yokogawa, H. Craig Heller, Paul Franken, and Norman F. Ruby. "Homeostatic regulation of sleep in arrhythmic Siberian hamsters." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 287, no. 1 (2004): R104—R111. http://dx.doi.org/10.1152/ajpregu.00676.2003.

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Sleep is regulated by independent yet interacting circadian and homeostatic processes. The present study used a novel approach to study sleep homeostasis in the absence of circadian influences by exposing Siberian hamsters to a simple phase delay of the photocycle to make them arrhythmic. Because these hamsters lacked any circadian organization, their sleep homeostasis could be studied in the absence of circadian interactions. Control animals retained circadian rhythmicity after the phase shift and re-entrained to the phase-shifted photocycle. These animals displayed robust daily sleep-wake rh
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Yoon, Jong Hyeon, and Ki-Sun Kwon. "Receptor-Mediated Muscle Homeostasis as a Target for Sarcopenia Therapeutics." Endocrinology and Metabolism 36, no. 3 (2021): 478–90. http://dx.doi.org/10.3803/enm.2021.1081.

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Brazhenko, O. N., N. A. Brazhenko, A. I. Loshakova, N. V. Tsygan, and S. G. Zheleznyak. "New diagnostic criteria for assessing homeostasis in patients with respiratory tuberculosis." Bulletin of the Russian Military Medical Academy 20, no. 2 (2018): 10–15. http://dx.doi.org/10.17816/brmma12204.

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The search for available informative criteria for diagnosing the state and dynamics of the homeostatic equilibrium of the organism, which began at the end of the XIXth century, continues to the present. Scientific research establishes the priorities for the study of homeostasis in the clinic and its control over the supra-segmental regulatory centers of the autonomic nervous system. The proposed functional, instrumental, immunological, biochemical and hormonal methods for evaluating it in phthisiology for various reasons, including economic ones, cannot be used fully. The modern clinic needs a
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Bozdemir, Kazım. "Thiol-disulphide homeostasis in chronic sinusitis without polyposis." Turkish Journal of Ear Nose and Throat 28, no. 2 (2018): 66–70. http://dx.doi.org/10.5606/tr-ent.2018.04453.

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Dissertations / Theses on the topic "Homeostasis. eng"

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Blanch, Graziela Torres. "Ajustes cardiovasculares e do equilibrio hidroeletrolítico induzidos por soluções hipertônicas em ratos com lesão do núcleo do trato solitário comissural /." Araraquara : [s.n.], 2010. http://hdl.handle.net/11449/106675.

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Orientador: Débora Simões de Almeida Colombari<br>Banca: Colin Sumners<br>Banca: Cássia Marta de Toledo Bermagaschi<br>Banca: Vagner Roberto Antunes<br>Banca: Lucila Leico Kagohara Elias<br>Resumo: O sistema nervoso central (SNC) tem um papel fundamental na regulação de mecanismos que controlam a osmolaridade dos líquidos corporais. O núcleo do trato solitário (NTS) é o sítio primário das aferências cardiovasculares e de osmorreceptores periféricos e se projeta à áreas prosencefálicas envolvidas com a regulação cardiovascular e do equilíbrio hidroeletrolítico. Demonstramos anteriormente que an
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Freiria-Oliveira, André Henrique. "Participação do grupamento catecolaminérgico A2 do núcleo do trato solitário comissural nos ajustes cardiovasculares e do equilíbrio hidroeletrolitíco induzidos por alterações da osmolaridade ou volume plasmático /." Araraquara : [s.n.], 2010. http://hdl.handle.net/11449/100952.

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Orientador: Débora Simões de Almeida Colombari<br>Banca: Colin Summers<br>Banca: José Antunes-Rodrigues<br>Banca: Marco Antonio Peliky Fontes<br>Banca: Juliana Irani Fratucci De Gobbi<br>Resumo: É de extrema importância para o funcionamento do organismo a manutenção da osmolaridade e volume dos líquidos corporais. O sistema nervoso central (SNC) tem um papel fundamental para esta manuntenção. O núcleo do trato solitário (NTS) é o sítio primário das aferências cardiovasculares e de osmorreceptores periféricos e se projeta à areas prosencefálicas envolvidas com a regulação cardiovascular e do eq
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Damineli, Daniel Santa Cruz. "Contribuições da conectância de rede e complexidade da dinâmica do sistema de trocas gasosas para a estabilidade na utilização de luz por espécies florestais /." Rio Claro : [s.n.], 2008. http://hdl.handle.net/11449/87864.

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Orientador: Gustavo Maia Souza<br>Banca: Carlos Henrique Britto de Assis Prado<br>Banca: Gustavo Habermann<br>Resumo: A estabilidade é fundamental para todos os sistemas biológicos, possibilitando que lidem com a variabilidade ambiental. As propriedades que conferem estabilidade a sistemas biológicos ainda são desconhecidas, mas evidências apontam para a complexidade da dinâmica de certas variáveis fisiológicas e para a força de interação entre elementos de suas redes organizacionais subjacentes. Esta relação foi investigada no sistema de trocas gasosas de espécies florestais tropicais, perten
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Faria, Filho Daniel Emygdio de. "Efeito de dietas com baixo teor protéico, formuladas usando o conceito de proteína ideal, para frangos de corte criados em temperaturas fria, termoneutra e quente /." Jaboticabal : [s.n.], 2003. http://hdl.handle.net/11449/99614.

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Orientador: Renato Luis Furlan<br>Resumo: Foram conduzidos dois experimentos com o objetivo de avaliar a utilização de dietas com baixo teor protéico, formuladas usando o conceito de proteína ideal, para frangos de corte de 7 a 21 dias (experimento 1) e de 21 a 42 dias (experimento 2) criados em diferentes temperaturas. Foram utilizados 900 e 720 frangos machos para os experimentos 01 e 02 respectivamente, da linhagem Cobb-500, distribuídos em um delineamento inteiramente ao acaso em esquema fatorial 3 x 3, com os fatores: níveis de proteína bruta (uma dieta controle e duas com redução protéic
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Gomes, Patricia Rodrigues Lourenço. "Efeito da exposição à fumaça de cigarro sobre a expressão de GLUT4 em ratas prenhes e lactantes e sua prole /." Presidente Prudente : [s.n.], 2010. http://hdl.handle.net/11449/87331.

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Orientador: Patrícia Monteiro Seraphim<br>Banca: Ismael Forte Freitas Júnior<br>Banca: Cecília Edna Mareze da Costa<br>Resumo: A gravidez é um período de ajustes metabólicos e, quando associado ao tabagismo provoca alterações que trazem malefícios tanto à saúde materna quanto à saúde fetal. Assim, o estudo investigou o efeito da exposição à fumaça de cigarro sobre a expressão do transportador de glicose GLUT4 e parâmetros séricos e morfométricos de ratas prenhes e sua prole. Foram utilizadas ratas Wistar divididas em: CG- controle sacrificadas após a gestação, com prole adotada pelo grupo CL;
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Cardoso, Mauro José Lahm. "Marcadores do metabolismo ósseo e homeostase do cálcio no hipertireoidismo felino /." Botucatu : [s.n.], 2006. http://hdl.handle.net/11449/101308.

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Orientador: Lucy Marie Ribeiro Muniz<br>Resumo: Os efeitos do hipertireoidismo experimental (150 g/kg/dia/42 dias) na homeostase do cálcio e nos marcadores do metabolismo ósseo foram estudados em 14 gatos sem raça definida, com idade entre um e três anos. Houve uma clara tendência de aumento das concentrações séricas de PTH intacto a partir do momento inicial com diferença significativa entre este e os demais momentos. O cálcio ionizado demonstrou uma diminuição significativa aos 14 dias em relação ao momento inicial e aos 42 dias em relação aos 14 dias. Os hormônios tireoidianos apresentaram
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Cambri, Lucieli Teresa. "Impacto da desnutrição intrauterina na homeostase glicêmica e na capacidade aeróbia de ratos /." Rio Claro : [s.n.], 2011. http://hdl.handle.net/11449/100424.

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Orientador: Maria Alice Rostom de Mello<br>Banca: Fernando Roberto de Oliveira<br>Banca: Maria Cristina Cintra Gomes Marcondes<br>Banca: Eliete Luciano<br>Banca: Sebastião Gobbi<br>Resumo: Este estudo teve por objetivo analisar as alterações que a desnutrição fetal provoca na homeostase glicêmica e na capacidade aeróbia, e quais alterações persistem na idade adulta, mesmo após a realimentação, em modelo experimental utilizando ratos. Adicionalmente, visou verificar a susceptibilidade de animais precocemente desnutridos aos efeitos metabólicos da sobrecarga de frutose. Ratas Wistar adultas (90
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Martinez, Padua Pedro Pablo. "Participação da excreção renal de cálcio, fósforo, sódio e potássio na homeostase em cães sadios e doentes renais crônicos /." Jaboticabal : [s.n.], 2009. http://hdl.handle.net/11449/89203.

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Orientadora: Marileda Bonafim Carvalho<br>Banca: Flávio Ruas de Moraes<br>Banca: Sandra Regina Ribeiro da Silva<br>Resumo: Os rins desempenham papel fundamental no balanço de água e eletrólitos. Na doença renal crônica (DRC) a manutenção da homeostase de água e sódio é o primeiro problema a ser contornado pelo organismo e com o agravamento das lesões renais surgem outros problemas graves relacionados à homeostase de cálcio e fósforo. O presente estudo tem por escopo avaliar a excreção renal de cálcio, fósforo, sódio e potássio, e o perfil sérico destes eletrólitos em cães normais e em cães com
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Yamada, André Katayama. "Impacto da desnutrição fetal/neonatal e do exercício durante a recuperação nutricional sobre lipídios teciduais e homeostase glicêmica de ratos /." Rio Claro : [s.n.], 2010. http://hdl.handle.net/11449/87441.

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Orientador: Masria Alice Rostom de Mello<br>Banca: Eliete Luciano<br>Banca: Fulvia de Barros Manchado Gobatto<br>Resumo: O presente estudo foi delineado a investigar os efeitos do treinamento físico de natação com característica aeróbia concomitante com a recuperação nutricional sobre marcadores bioquímicos de desnutrição, o teor de lipídios em tecidos insulinosensíveis e a homeostase glicêmica de ratos submetidos à desnutrição protéica fetal/neonatal. Foram avaliados quatro grupos de animais: Normoprotéico (NP): crias de ratas alimentadas com dieta com 17% de caseína durante a gestação e a la
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Souza, Ana Paula de. "Função renal e homeostase de água, sódio e potássio em cães da raça Golden Retriever normais, portadores e afetados pela distrofia muscular /." Jaboticabal : [s.n.], 2007. http://hdl.handle.net/11449/101233.

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Orientadora: Mirela Bonafim Carvalho<br>Banca: Carlos Roberto Daleck<br>Banca: Wilter Ricardo Russiano Vicente<br>Banca: Maria Angélica Miglino<br>Banca: Carlos Eduardo Ambrósio<br>Resumo: A Distrofia Muscular de Duchenne (DMD) é uma doença genética recessiva e hereditária, ligada ao cromossomo X, caracterizada pela ausência ou insuficiência da proteína distrofina no sarcolema das fibras musculares. É uma doença muscular progressiva, que afeta um em cada 3.500 meninos. Atualmente, os cães da raça Golden Retriever, portadores da distrofia muscular do Golden Retriever (GRMD), que exibem mudanças
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Books on the topic "Homeostasis. eng"

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Murer, Heini, Jürg Biber, and Carsten A. Wagner. Phosphate homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0025.

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Inorganic phosphate ions (H2PO4−/ HPO42−) (abbreviated as Pi) are involved in formation of bone and generation of high-energy bonds (e.g. ATP), metabolic pathways, and regulation of cellular functions. In addition, Pi is a component of biological membranes and nucleic acids. Only about 1% of total body Pi content is present in extracellular fluids, at a plasma concentration in adults within the range 0.8–1.4 mMol/L (at pH 7.4 mostly as HPO42−), with diurnal variations of approximately 0.2 mM. A small amount of plasma Pi is bound to proteins or forms complexes with calcium. Under normal, balanc
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Nutt, David J., and Liam J. Nestor. Key elements of addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0003.

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Addiction is characterized by the compulsion to seek and take a substance, the loss of control in limiting substance intake, and the emergence of a negative emotional state (e.g. dysphoria, anxiety) when substance intake is prevented. Importantly, there are elements of addiction that emerge during the addiction trajectory (e.g. liking, wanting, habit, craving) that are a reflection of key changes in the homeostasis of brain networks that control different behaviours. These homeostatic changes ultimately lead to 1) a decreased sensitivity for natural rewards, 2) an enhanced sensitivity for cond
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Harrison, Mark. Cardiovascular system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0049.

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This chapter describes the pathophysiology of the cardiovascular system as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of the control of blood pressure and heart rate, cardiac output, blood flow, cardiac cycle, ECG, pharmacological manipulation of the heart, shock, oxygen delivery and consumption, body fluid homeostasis, crystalloid solutions, colloidal solutions, and exudates and transudates. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.
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Parlato, Marianna, and Jean-Marc Cavaillon. Innate immunity and the inflammatory cascade. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0299.

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Inflammation results from a complex interaction between a large number of mediators able to induce each other and to favour the generation of other inflammatory molecules (e.g. free radicals, lipid mediators, and proteases). The perpetuation of inflammation by these cascades of mediators is favoured by their ability to induce coagulation, leukocyte recruitment, and cell and tissue alteration (apoptosis, necrosis, and barrier disruption). Other cascades of mediators occur to generate anti-inflammatory mediators favouring the healing process. A neuroendocrine loop and neuromediators from central
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Rosenbluth, Glenn, and Christopher P. Landrigan. Sleep, work hours, and medical performance. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0022.

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Physicians are among the few professionals who are expected to work extended duty shifts of 24 hours or more, often with little opportunity for rest. The physiological factors regulating sleepiness, including circadian rhythms, sleep homeostasis, and sleep inertia, are pushed to their limits when meeting the demands of training programmes and patient care. Sleep-deprived physicians experience reduced clinical performance and vigilance, putting patients at risk. Tired physicians are more likely to make both cognitive errors (e.g. diagnostic reasoning) and technical errors (e.g. surgical complic
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Tuschl, Karin, Peter T. Clayton, and Philippa B. Mills. Disorders of Manganese Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0045.

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Manganese is an essential trace metal for numerous metalloenzymes. Manganese homeostasis requires tight regulation in vivo and disruption of this balance can lead to manganese overload and subsequent accumulation of manganese in brain, liver, and blood. Mutations in SLC30A10, a cell surface-localized manganese efflux transporter, cause an autosomal recessive hypermanganesemia syndrome with two distinct phenotypes: childhood onset dystonia and adult onset Parkinsonism, associated with chronic liver disease, polycythemia and features of iron depletion. MRI brain appearances are characteristic of
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Wein, Simon, and Limor Amit. Adjustment disorders and anxiety. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0174.

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Adjustment disorders and anxiety are two of the more common responses to stressors in palliative care. At one end of the spectrum, adjustment and anxiety are normal defences. However, when coping mechanisms fail these responses can become pathological. Judging when a response is pathological is based on two principles: the severity of symptoms and the extent of disruption of normal functioning or homeostatic adaptation. The intimate two-way relationship between physical and psychological symptoms in palliative care means that physical symptoms have to be well controlled and that psychological
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Plutynski, Anya. Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199967452.003.0002.

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Is cancer one or many? If many, how many diseases is cancer, exactly? I argue that this question makes a false assumption; there is no single “natural” classificatory scheme for cancer. Rather, there are many ways to classify cancers, which serve different predictive and explanatory goals. I consider two philosophers’ views concerning whether cancer is a natural kind, that of Khalidi, who argues that cancer is the closest any scientific kind comes to a homeostatic property cluster kind, and that of Lange, whose conclusion is the opposite of Khalidi’s; he argues that cancer is at best a “kludge
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Sidhu, Kulraj S., Mfonobong Essiet, and Maxime Cannesson. Cardiac and vascular physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0001.

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This chapter discusses key components of cardiovascular physiology applicable to clinical practice in the field of anaesthesiology. From theory development to ground-breaking innovations, the history of cardiac and vascular anatomy, as well as physiology, is presented. Utilizing knowledge of structure and function, parameters created have allowed adequate patient clinical assessment and guided interventions. A review of concepts reveals the impact of multiple physiological variables on a patient’s haemodynamic state and the need for more accurate and efficient measurements. In particular, it i
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Book chapters on the topic "Homeostasis. eng"

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Pula, G., and M. Krause. "Role of Ena/VASP Proteins in Homeostasis and Disease." In Handbook of Experimental Pharmacology. Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-72843-6_3.

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Ohlemiller, Kevin K. "A Question of Balance: Free Radicals and Cochlear Homeostasis." In Free Radicals in ENT Pathology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13473-4_3.

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Ruzek, Martin. "Modeling of EEG Signal with Homeostatic Neural Network." In Nostradamus 2013: Prediction, Modeling and Analysis of Complex Systems. Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-00542-3_18.

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Cao, Hoang-Long, Albert De Beir, Pablo Gómez Esteban, et al. "An End-User Interface to Generate Homeostatic Behavior for NAO Robot in Robot-Assisted Social Therapies." In Advances in Computational Intelligence. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59147-6_52.

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"Homeostasis and Body Fluid Regulation: An End Note." In Neurobiology of Body Fluid Homeostasis. CRC Press, 2013. http://dx.doi.org/10.1201/b15544-19.

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Neal, Mark, and Jon Timmis. "Once More Unto the Breach." In Recent Developments in Biologically Inspired Computing. IGI Global, 2005. http://dx.doi.org/10.4018/978-1-59140-312-8.ch014.

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The field of biologically inspired computing has generated many novel, interesting and useful computational systems. None of these systems alone is capable of approaching the level of behaviour for which the artificial intelligence and robotics communities strive. We suggest that it is now time to move on to integrating a number of these approaches in a biologically justifiable way. To this end we present a conceptual framework that integrates artificial neural networks, artificial immune systems and a novel artificial endocrine system. The natural counterparts of these three components are usually assumed to be the principal actors in maintaining homeostasis within biological systems. This chapter proposes a system that promises to capitalise on the self-organising properties of these artificial systems to yield artificially homeostatic systems. The components develop in a common environment and interact in ways that draw heavily on their biological counterparts for inspiration. A case study is presented, in which aspects of the nervous and endocrine systems are exploited to create a simple robot controller. Mechanisms for the moderation of system growth using an artificial immune system are also presented.
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Firth, John D. "Disorders of potassium homeostasis." In Oxford Textbook of Medicine, edited by John D. Firth. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0474.

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Hypokalaemia is defined as a serum potassium concentration lower than 3.5 mmol/litre and is the most common electrolyte abnormality seen in clinical practice, found in about 20% of hospital inpatients. Clinical features and investigation—mild hypokalaemia is asymptomatic, but nonspecific symptoms develop with more severe disturbance, and serious neuromuscular problems sometimes arise at serum potassium concentrations lower than 2.5 mmol/litre. Emergency management is rarely required, but intravenous infusion of potassium should be given in the rare circumstances of life-threatening cardiac arrhythmia or muscular paralysis. There are a very large number of possible causes of hypokalaemia, the most common causes being diuretics (particularly thiazides), vomiting, and diarrhoea. The most common genetic cause is Gitelman’s syndrome. Management is with potassium and magnesium supplements. Other causes of tubular wasting of potassium include Bartter’s syndrome and mineralocorticoid excess. There are several rare conditions in which hypokalaemia is associated with episodes of extreme weakness/paralysis, including thyrotoxic periodic paralysis and familial hypokalaemic periodic paralysis. Hyperkalaemia, defined as a serum potassium concentration of greater than 5.0 mmol/litre, is asymptomatic, and severe hyperkalaemia (&gt;7 mmol/litre) is the most serious of all electrolyte disorders because it can cause cardiac arrest. The electrocardiogram (ECG) is the best guide to the significance of hyperkalaemia in any particular individual. Patients with ECG manifestations more severe than tenting of the T waves should be given intravenous calcium gluconate (10 ml of 10%) followed by intravenous insulin and glucose, or nebulized salbutamol. There are many causes of hyperkalaemia, but by far the most common are renal failure and/or drugs. Other causes include exhaustive exercise, acidosis, drugs, and hyperkalaemic periodic paralysis (very rare).
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Munis, James R. "Man, Machine, and Homeostasis." In Just Enough Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199797790.003.0018.

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Physiologist Claude Bernard lived in a time when very little was known about the mechanisms underlying physiologic findings, and he had ample access to clues garnered from observing machines. Let's consider homeostasis (a concept championed by Bernard), an example for which an engineered machine shed light on a fundamental principle of physiology. Homeostasis is simply the tendency of the body to maintain important physiologic variables (eg, heart rate, blood pressure, PACO<sub>2</sub>) at constant, preset values. An example is a simplified mechanical governor that could be used to regulate the rotational speed of a steam engine shaft. ‘Autoregulate’ might be a more apt word because the governor performs without external help or guidance, provided it is designed and built properly. It doesn't take much imagination to see an analogy between the mechanical governor and the autonomic nervous system. Both maintain specific variables at a constant set point through a process of feedback loops.
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Mifune, Mizuo, and Yoshihiko Kanno. "Hypertension as Three Systematic Dysregulations of Na+ Homeostasis in Terrestrial Mammal, and Salt in Gut Might Cause Brain Inflammation." In Psychology and Patho-physiological Outcomes of Eating [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.98904.

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Although Na+ homeostasis in vivo is essential for mammals, it is known that excessive salt (NaCl) intake has played a major role in the development of hypertension. In vivo, there is a hormonal system, the renin-angiotensin-aldosterone system (RAAS), that specializes in regulating Na+ retention, especially the amount of Na+ in plasma. Na+ homeostasis in vivo has been achieved mainly by the RAAS, through regulation of vascular tonus (blood pressure) and Na+ handling in the kidney (Na+ diuresis). Recent studies have revealed a third mechanism of Na+ homeostasis in vivo: regulation of interstitial Na+ levels in tissues, such as subcutaneous tissues, by tissue macrophage immunity. In the pathogenesis of salt-sensitive hypertension, Recent research have been revealed that three molecular axes (Ang II - Rho/NOX-eNOS system, Aldosterone-rac1 -ENaC system, and tissue Na+ − TonEBP in macrophage -VEGF-c) are significantly involved in maintaining Na+ homeostasis in salt induced hypertension. Furthermore, the mechanism by which salt causes hypertension via the immune system (intestinal, local mucosal, and tissue immunity) has also been reported. In this article, we would like to propose that three molecular dysfunctions are involved in the development of salt-sensitive hypertension through three immunological mechanisms in the maintenance of Na+ homeostasis. Next, I would like to explain the importance of gut-RAAS and abnormality of intestinal microflora (dysbiosis) in salt-sensitive hypertension. It has been known that the metabolites (e.g., short-chain fatty acid neural amino) produced by microflora are deeply involved in central (CNS) and sympathetic nervous system (SNS) activity. In addition, we would like to explain of the importance of brain-RAAS and cerebral inflammation in salt-sensitive hypertension. Moreover, recent research have revealed that the detection-mechanism in the brain for Na+ concentration([Na+]) in vivo and in the tongue for [Na+] in diet. These finding suggests that excessive salt intake may cause brain dysfunction, most delicate organ, before the onset of salt sensitive hypertension, and may also destroy brain structure after the onset of salt sensitive hypertension. Thus, we would like to insist that excessive salt intake might not only induce hypertension, but also be toxic especially for brain. Finally, we would like to explain that The DASH diet (Dietary Approaches to Stop Hypertension) is one of the universal diets for adult human, not only by reducing salt, but also by reducing metabolic stress and improving of dysbiosis.
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Hurt, Ryan T. "Obesity and Nutritional Disorders." In Mayo Clinic Internal Medicine Board Review. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190464868.003.0016.

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The prevalence of overweight and obesity has been increasing in the United States and the westernized world. The cause of the recent obesity epidemic involves a complex interplay between genetic and environmental factors. Specific, rare genetic disruptions of the hypothalamic regulation of energy homeostasis pathways can cause obesity (eg, Prader-Willi syndrome). Most cases of obesity result from a group of gene variants exposed to environmental factors. The 2 major environmental factors that contribute to overweight and obesity are excess caloric intake and low physical activity.
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Conference papers on the topic "Homeostasis. eng"

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Humphrey, J. D. "Constrained Mixture Models of Arterial Homeostasis and Adaptation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204922.

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Phenomenological models of the mechanical behavior of the arterial wall continue to play important roles in vascular mechanics. Indeed, such models revealed the importance of residual stresses in homogenizing the transmural distribution of stress in normalcy [1], which in turn led to one of the most important hypotheses in vascular mechanobiology — the existence of a mechanical homeostasis [2]. Nevertheless, classical models are not able to exploit the growing information on the different mechanical properties and rates and extents of turnover of different structurally significant constituents
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McGah, Patrick M., James J. Riley, Alberto Aliseda, Daniel F. Leotta, and Kirk W. Beach. "Incomplete Restoration of Homeostatic Shear Stress Within Arteriovenous Fistulae." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80458.

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Arteriovenous fistulae are created surgically to provide an adequate access for dialysis in patients with End-Stage Renal Disease (ESRD). Producing an autogenous shunt linking an artery and a vein in the peripheral circulation bypasses the high resistance capillary bed in order to provide the necessary flow rates at sites easily accessible for dialysis. In successful fistulae, venous flow rates can easily exceed 1000 mL/min. It has long been recognized that the hemodynamics constitute the primary external influence on the remodeling process [1]; The high flow rate, together with the exposure o
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Li, Dalong, and Anne M. Robertson. "A Structural Multi-Mechanism Damage Model for Cerebral Arterial Tissue and its Finite Element Implementation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193112.

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Early stage cerebral aneurysms are characterized by the disruption of the internal elastic lamina (IEL). The cause of this breakdown is still not understood, but it has been conjectured to be caused by fatigue failure or alternatively by a breakdown in homeostatic mechanisms in the wall arising from some aspect of the local hemodynamics, e.g. [1].
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Kemeny, Steven F., and Alisa Morss Clyne. "High Glucose Alters Endothelial Cell Response to Shear Stress." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206531.

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Endothelial cells line the walls of all blood vessels, where they maintain homeostasis through control of vascular tone, permeability, inflammation, and the growth and regression of blood vessels. Endothelial cells are mechanosensitive to fluid shear stress, elongating and aligning in the flow direction [1–2]. This shape change is driven by rearrangement of the actin cytoskeleton and focal adhesions [2]. Hyperglycemia, a hallmark of diabetes, affects endothelial cell function. High glucose has been shown to increase protein kinase C, formation of glucose-derived advanced glycation end-products
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Moreno, Michael, Saurabh Biswas, Lewis D. Harrison, et al. "Assessment of Minimally Invasive Device That Provides Simultaneous Adjustable Cardiac Support and Active Synchronous Assist in an Acute Heart Failure Model." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53089.

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Congestive heart failure (CHF) is a debilitating disease that is generally initiated by some index cardiac event and ultimately characterized by left ventricular (LV) remodeling which dramatically alters the mechanical environment about the heart. It is well established that mechanical stimuli (e.g., stress or strain) are important epigenetic factors in cardiovascular development, adaptation, and disease.1–3 Interestingly, abnormal cardiac kinematics is often considered a symptom of heart failure when in actuality it is likely a primary contributing factor to the relentless progression of the
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Graham, Drew A., Danil V. Dobrynin, Alexander Fridman, Gary Friedman, and Alisa Morss Clyne. "A Pin-to-Hole Spark Discharge Plasma Generates Nitric Oxide and Can Be Safely Applied to an Endothelial Cell Monolayer." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206764.

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Endothelial cells line all blood vessels and regulate many homeostatic functions (e.g. platelet aggregation, vascular tone, vascular cell proliferation, leukocyte adhesion) by production of the signaling molecule nitric oxide (NO). NO bioavailability and thus endothelial cell function are compromised in many chronic disease states, including diabetes mellitus and its associated micro- and macrovascular complications (e.g. impaired wound healing and atherosclerosis, respectively) [1]. In the specific case of diabetic wound healing, application of exogenous NO to the diseased tissue may help res
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Li, Lingmin, Shantanu Patil, Nick Steklov, et al. "Computational Simulation of Articular Cartilage Wear in the Patellofemoral Joint During In Vitro Testing." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19636.

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The mechanism by which altered knee joint motions and loads (e.g., following anterior cruciate ligament (ACL) injury) contribute to the development of knee osteoarthritis (OA) is not well understood. One mechanobiological hypothesis is that articular cartilage degradation is initiated when altered knee kinematics increase loading on certain regions of the articular surfaces and decrease loading on other regions [1,2]. If homeostatic loading conditions vary from region to region, then load changes induced by altered kinematics could initiate cartilage degradation in a site-specific manner. This
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Li, Lingmin, Shantanu Patil, Nick Steklov, et al. "Finite Element Modelling of In Vitro Articular Cartilage Wear in the Patellofemoral Joint." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80043.

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The mechanism by which altered knee joint motions and loads (e.g., following anterior cruciate ligament (ACL) injury) contribute to the development of knee osteoarthritis (OA) is not well understood. One mechanobiological hypothesis is that articular cartilage degradation is initiated when altered knee kinematics increase loading on certain regions of the articular surfaces and decrease loading on other regions [1]. If homeostatic loading conditions vary from region to region, then load changes induced by altered kinematics could initiate cartilage degradation in a site-specific manner. This h
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McGah, Patrick M., Alberto Aliseda, Daniel F. Leotta, and Kirk W. Beach. "Effects of Wall Distensibility on the Numerical Simulation of Arteriovenous Fistulae." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14183.

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Arteriovenous fistulae are created surgically to provide an adequate access for dialysis in patients with End-Stage Renal Disease (ESRD). Producing an autogenous shunt linking an artery and a vein in the peripheral circulation bypasses the high resistance capillary bed in order to provide the necessary flow rates at sites easily accessible for dialysis. It has long been recognized that hemodynamics constitute the primary external influence on the remodeling process of anastomosed vascular tissue [1, 2]. The high flow rate, together with the exposure of the venous tissue to the high arterial pr
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Baker, Brendon M., Britta Trappmann, Iris L. Kim, Jason A. Burdick, and Christopher S. Chen. "Engineered Fibrillar Microenvironments With Controllable Architecture and Mechanics for Studying Cellular Stiffness Sensing." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14804.

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The mechanical properties of the extracellular matrix (ECM) have emerged as fundamental players in numerous basic cellular functions such as spreading, migration, proliferation and differentiation, thus impacting many biological processes including embryonic development, adult tissue homeostasis, and disease pathogenesis such as fibrosis and cancer [1,2,3]. Synthetic matrices have been crucial to studying the effect of mechanics on cell behavior, as they allow for precise control of mechanical properties over a wide stiffness range, unachievable in vivo or in many naturally derived material sy
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Reports on the topic "Homeostasis. eng"

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R. Nebreda, Ángel. Señalización de estrés en la homeostasis tumoral: implicaciones terapéuticas. Sociedad Española de Bioquímica y Biología Molecular, 2017. http://dx.doi.org/10.18567/sebbmdiv_anc.2017.10.1.

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