Academic literature on the topic 'Hominis'

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Journal articles on the topic "Hominis"

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Urciuoli, Alessandro, Clément Zanolli, Sergio Almécija, Amélie Beaudet, Jean Dumoncel, Naoki Morimoto, Masato Nakatsukasa, Salvador Moyà-Solà, David R. Begun, and David M. Alba. "Reassessment of the phylogenetic relationships of the late Miocene apes Hispanopithecus and Rudapithecus based on vestibular morphology." Proceedings of the National Academy of Sciences 118, no. 5 (January 25, 2021): e2015215118. http://dx.doi.org/10.1073/pnas.2015215118.

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Late Miocene great apes are key to reconstructing the ancestral morphotype from which earliest hominins evolved. Despite consensus that the late Miocene dryopith great apes Hispanopithecus laietanus (Spain) and Rudapithecus hungaricus (Hungary) are closely related (Hominidae), ongoing debate on their phylogenetic relationships with extant apes (stem hominids, hominines, or pongines) complicates our understanding of great ape and human evolution. To clarify this question, we rely on the morphology of the inner ear semicircular canals, which has been shown to be phylogenetically informative. Based on microcomputed tomography scans, we describe the vestibular morphology of Hispanopithecus and Rudapithecus, and compare them with extant hominoids using landmark-free deformation-based three-dimensional geometric morphometric analyses. We also provide critical evidence about the evolutionary patterns of the vestibular apparatus in living and fossil hominoids under different phylogenetic assumptions for dryopiths. Our results are consistent with the distinction of Rudapithecus and Hispanopithecus at the genus rank, and further support their allocation to the Hominidae based on their derived semicircular canal volumetric proportions. Compared with extant hominids, the vestibular morphology of Hispanopithecus and Rudapithecus most closely resembles that of African apes, and differs from the derived condition of orangutans. However, the vestibular morphologies reconstructed for the last common ancestors of dryopiths, crown hominines, and crown hominids are very similar, indicating that hominines are plesiomorphic in this regard. Therefore, our results do not conclusively favor a hominine or stem hominid status for the investigated dryopiths.
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Lee, Jaehoon. "From Dignitas Hominis to Perfectio Hominis: From Renaissance to Descartes." Modern Philosophy 19 (April 30, 2022): 5–34. http://dx.doi.org/10.52677/mph.2022.04.19.5.

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Abdul-Wahab, Osama M., Abdulaziz A. Abu Dahish, and Mohamed E. Elawad. "Mycoplasma Hominis." Bahrain Medical Bulletin 39, no. 2 (June 2017): 128–30. http://dx.doi.org/10.12816/0047540.

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Miller, R. A., and B. H. Minshew. "BLASTOCYSTIS HOMINIS." Pediatric Infectious Disease Journal 8, no. 8 (August 1989): 545. http://dx.doi.org/10.1097/00006454-198908000-00036.

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Rosenblatt, J. E. "Blastocystis hominis." Journal of Clinical Microbiology 28, no. 10 (1990): 2379–80. http://dx.doi.org/10.1128/jcm.28.10.2379-2380.1990.

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Murube, Juan. "Demodex hominis." Ocular Surface 13, no. 3 (July 2015): 181–86. http://dx.doi.org/10.1016/j.jtos.2015.04.002.

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Lee, King-chung. "Gastrospirillum hominis." Advances in Anatomic Pathology 2, no. 6 (November 1995): 409–12. http://dx.doi.org/10.1097/00125480-199511000-00047.

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Arisandi, Yessi, Chairil Anwar, Salni Salni, Dadang Hikmah Purnama, Novrikasari Novrikasari, and Ahmad Ghiffari. "Mite Sarcoptes scabiei Varieties Hominis in South Sumatra: Specific Identification and Comparative Study." Open Access Macedonian Journal of Medical Sciences 8, A (December 15, 2020): 938–42. http://dx.doi.org/10.3889/oamjms.2020.5562.

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BACKGROUND: Sarcoptes scabiei mites have more than 15 genetically diverse varieties from various hosts. Identification of S. scabiei mite varieties hominis as an intervention in its prevention is still rarely done. AIM: This study aimed to observe the genetic relationship of the mite S. scabiei varieties hominis compare to the parasite S. scabiei varieties hominis in other regions. METHODS: This research used polymerase chain reaction (PCR) and sequencing methods with 16S gene-specific primers. From 32 S. scabiei samples, 22 samples were identified as varieties hominis that was marked by the appearance of the band at 132 bp. RESULTS: S. scabiei mites hominis varieties from South Sumatra (Yessi Scabies A2 and Yessi Scabies B3) have similarities with deoxyribonucleic acid (DNA) strands with S. scabiei hominis varieties from China (KJ781377 and KJ781376). In contrast, Yessi Scabies A1 has similarities with DNA strands with mite S. scabiei varieties hominis from Australia (AY493402). Still, all the DNA strands, this research is different from S. scabiei mites DNA strands hominis from Panama and Pakistan. CONCLUSION: The PCR method is advantageous and specific in identifying mites S. scabiei hominis varieties, the cause of scabies in humans.
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Che, Ya-Min, Shu-He Mao, Wen-Ling Jiao, and Zhi-Yi Fu. "Susceptibilities of Mycoplasma hominis to Herbs." American Journal of Chinese Medicine 33, no. 02 (January 2005): 191–96. http://dx.doi.org/10.1142/s0192415x05002862.

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To determine the susceptibilities of Mycoplasma homonis (M. hominis) to Chinese medicinal herbs in vitro, 30 clinical strains of M. hominis were isolated and identified from the clinical specimen. The susceptibilities of M. hominis to 19 herbs were determined by serial dilution methods in vitro. The results showed that M. hominis was susceptible to Radix Isatidis, Radix Angelicae Dahuricae, Cortex Phellodendri, Radix et Rhizoma Rhei, Fructus Kochiae and Herba Houttuyniae. These findings laid a foundation in treating M. hominis infection with Chinese herbs.
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Stenzel, D. J., and P. F. Boreham. "Blastocystis hominis revisited." Clinical Microbiology Reviews 9, no. 4 (October 1996): 563–84. http://dx.doi.org/10.1128/cmr.9.4.563.

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Blastocystis hominis is a unicellular organism found commonly in the intestinal tract of humans and many other animals. Very little is known of the basic biology of the organism, and controversy surrounds its taxonomy and pathogenicity. There morphological forms (vacuolar, granular, and ameboid) have been recognized, but recent studies have revealed several additional forms (cyst, avacuolar, and multivacuolar). The biochemistry of the organism has not been studied to any extent, and organelles and structures of unknown function and composition are present in the cells. Several life cycles have been proposed but not experimentally validated. The form used for transmission has not been defined. Infections with the organism are worldwide and appear in both immunocompetent and immunodeficient individuals. Symptoms generally attributed to B. hominis infection are nonspecific, and the need for treatment is debated. If treatment appears warranted, metronidazole is suggested as the drug of choice, although failures of this drug in eradicating the organism have been reported. Infection is diagnosed by light microscopic examination of stained smears or wet mounts of fecal material. Most laboratories identify B. hominis by observing the vacuolar form, although morphological studies indicate that other forms, such as the cyst form and multivacuolar form, also should be sought for diagnosis.
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Dissertations / Theses on the topic "Hominis"

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Aguiló, Regla Josep. "Hominis Presumptions and Evidential Inferences." Pontificia Universidad Católica del Perú, 2017. http://repositorio.pucp.edu.pe/index/handle/123456789/116725.

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The author challenges the terminology «legal presumptions» and «judicial presumptions», and rather refers to presumptions established by rules of presumption and to hominis presumptions. He argues that the best way to differentiate between them is by showing the contrast between «it shall be presumed» (syntagm proper to practical reasoning) and «it is presumable» (syntagm proper to theoretical reasoning). The text clarifies the relationship between the so-called hominis presumptions and the factual inferences or evidential inferences, in general. He answers the question of what the «it is presumed» syntagm (proper to the hominis presumptions) brings with respect to the «it is probable» syntagm (proper of all evidentiary inferences).
El autor cuestiona la terminología «presunciones legales» y «presunciones judiciales» y, más bien, se refiere a las presunciones establecidas por normas de presunción y a las presunciones hominis. Defiende que la mejor manera de diferenciar unas de otras es mostrando la distancia que media entre «debe presumirse» (sintagma propio del razonamiento práctico) y «es presumible» (sintagma propio del razonamiento teórico). El texto aclara las relaciones entre las llamadas presunciones hominis y las inferencias fácticas o inferencias probatorias, en general, respondiendo a la pregunta sobre qué aporta el sintagma «es presumible» (propio de las presunciones hominis) frente al sintagma «es probable» (propio de todas las inferencias probatorias).
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Hopfe, Miriam. "Charakterisierung der Oligopeptidpermease von Mycoplasma hominis." [S.l. : s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963936980.

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Domínguez, Márquez Mª Victoria. "Heterogeneidad genética de blastocystis hominis: implicaciones patogénicas." Doctoral thesis, Universitat de València, 2003. http://hdl.handle.net/10803/10099.

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En los últimos años diversos trabajos ponen de manifiesto la existencia de variaciones intraespecíficas entre aislados de Blastocystis hominis. La descripción de diferentes perfiles proteicos, cariotipos y zimodemas constituyen pruebas de la posible existencia de poblaciones morfológicamente idénticas, que quizás estén dotadas de un potencial patogénico diferente. El objeto de este estudio es el de establecer asociaciones entre variantes genéticas y enzimáticas y distinta significación clínica, para lo cual se realizaron los siguientes procedimientos:(1) Obtener aislados clínicos de pacientes sintomáticos y asintomáticos, cultivarlos y axenizarlos.(2) Comprobar el grado de variación genética críptica en aislados de B. hominis, mediante el análisis de polimorfismo de fragmentos de restricción a partir de dos amplicones diferentes.(3) Evaluar el poder discriminatorio de secuencias repetitivas de ADN como marcadores de diversidad.(4) Demostrar la existencia de poblaciones isoenzimáticas. Se analizaron un total de 260 muestras fecales, de entre las remitidas al Servicio de Microbiología del Hospital Clínico Universitario de Valencia; que fueron sometidas al siguiente protocolo: exámen parasitológico para la identificación de B. hominis y de otros posibles parásitos; y un coprocultivo con el objeto de determinar la existencia de enteropatógenos bacterianos. Se observó B. hominis en 56 de estas muestras, lo que supuso una prevalencia del 21,5%; y se procedió a su cultivo en medio BDM para su posterior axenización, todo ello de acuerdo con el protocolo descrito por Lanuza et al. (1997). Se consiguió la adaptación al cultivo de 38 aislados, de los que se consiguió axenizar 18. Se incluyeron en el estudio 13 cultivos puros procedentes de colección, por lo que finalmente se analizaron un total de 51 aislados: 31 axénicos y 20 monoxénicos. Una vez obtenidos los aislados se llevaron a cabo los ensayos de "riboprinting" (Böhm-Gloning,1997; Clark, 1997), consistentes en amplificar la secuencia que codifica el ARN de la subunidad menor ribosomal y analizar la longitud de los fragmentos obtenidos tras restricción enzimática de dicha región. Extraído el ADN de acuerdo con el procedimiento descrito por Clark (1997) consistente en una extracción fenólica; la amplificación se realizó en dos reacciones distintas: la primera con los cebadores RD3 y RD5 (Clark, 1997) que amplifican la totalidad del gen con lo que se obtiene un amplicón de 2.540 pb; y la segunda con F1 y R1 (Böhm-Gloning et al., 1997) que amplifican sólo una parte del gen, por lo que se obtiene un producto de 1.100 pb. Los productos de amplificación fueron digeridos separadamente con las endonucleasas Rsa I, Hinf I y Alu I. Para el análisis se transformaron las distancias de migración en tamaños relativos al compararlos con marcadores de peso molecular, utilizando el programa informático 1DManager. El criterio establecido para diferenciar entre patrones de RFLP fue considerar que eran distintos los que diferían en uno o en más fragmentos. Analizados los perfiles obtenidos para cada aislado, se obtuvieron los índices de homología entre cepas aplicando el coeficiente de Dice. Los resultados obtenidos tras el análisis conjunto de todos los patrones de bandas fue la obtención de 5 ribodemas (R1 a R5), en los que se valoró si existía relación estadística entre la pertenencia a un ribodema y padecer diarrea aguda o crónica, por lo que se analizaron los datos aplicando el test CHI-CUADRADO y se definieron el riesgo relativo (RR) y el coeficiente de correlación de Pearson (p).Los resultados demostraron una asociación estadísticamente significativa entre diarrea aguda e inclusión en el ribodema R2. Lo cual nos confirmó que la especie B. hominis es genéticamente heterogénea, y que existían diferentes poblaciones con diferencias en la capacidad patogénica. En el estudio también se valoró la eficacia de secuencias repetitivas de ADN como marcadores de diversidad, para lo cual se utilizaron los cebadores TR7 y TR8 (Init et al., 1999), demostrando que se podían establecer diferencias intraespecíficas a partir de un fragmento mayoritario, es decir el que más veces se repetía, considerando que eran cepas pertenecientes al mismo patrón las que coincidían en dicho fragmento. Los resultados demostraron que esta técnica es únicamente válida en la detección de variación intraespecífica en cultivos puros, ya que los cebadores no discriminan suficientemente cuando en la muestra existe ADN bacteriano además del de B. hominis. No se encontraron asociaciones estadísticas entre la pertenencia a alguno de los patrones definidos y la presentación de un proceso diarreico determinado. Finalmente el análisis de las actividades enzimáticas: Glucosa-6-fosfato deshidrogenasa, malato deshidrogenasa, enzima málico, hexoquinasa, fosfoglucomutasa, glucosa fosfato isomerasa, glutámico-oxalacético transaminasa y fosfogluconato deshidrogenasa; consistió en la extracción de las proteínas de los cultivos puros, su separación en geles de poliacrilamida, y el consiguiente revelado utilizando sustancias tamponadas, con los sutratos, cofactores y coenzimas necesarios para cada enzima, de acuerdo con el protocolo de Selander et al. (1985).Todos los aislados presentaron todas las actividades enzimáticas y compartieron idéntico patrón en todos los enzimas a excepción de la malato deshidrogenasa; cuyas diferencias en la movilidad electroforética de una de sus bandas, permitió definir tres poblaciones isoenzimáticas. No se encontraron asociaciones estadísticas entre los zimodemas definidos y la presentación de un determinado proceso diarreico. Los resultados permitieron extraer las siguientes conclusiones:1ª.- Todos los métodos moleculares empleados demostraron, aunque con desigual capacidad de discriminación, que B. hominis es un organismo con marcada heterogeneidad genética.2ª.- El análisis de la secuencia del ARN ribosomal 16S, mediante técnicas de RFLP, ha detectado variantes asociadas a determinados estados mórbidos. El protocolo que utiliza los cebadores RD3 y RD5 es el que ha mostrado un mayor poder de resolución.3ª.- Consideramos que para poder comparar datos de RFLP de forma efectiva es necesaria la utilización de protocolos normalizados, con cepas de referencia, un panel común de enzimas de restricción y una nomenclatura consensuada.4ª.- El estudio de secuencias repetitivas utilizando TR7 y TR8 resulta eficaz en la detección de variación intraespecífica únicamente en aislados axénicos, lo cual limita su utilización como marcador de diversidad.5ª.- Las diferencias encontradas en la movilidad del enzima malato deshidrogenasa demostraron la existencia de tres poblaciones isoenzimáticas, son que ninguna de ellas se asociara con un proceso determinado.6ª.- La homología encontrada entre nuestros aislados y los genotipos de origen humano y animal descritos en otras áreas geográficas sustentan la idea del origen zoonótico de la infección y demuestran la ausencia de variaciones genéticas regionales. Por lo que este trabajo concluye que B. hominis es una especie genéticamente heterogénea, y la asociación estadística observada confirma además la existencia de poblaciones con diferente patogenicidad.
Blastocystis hominis is the most prevalent specie of intestinal protozoa found in men of uncertain role in human disease. This study was undertaken to examine the degree of criptic genetic variation within B. hominis detectable using riboprinting (restriction fragment length polymorphism analysis of polymerase chain reaction amplified small subunit rDNA); the analysis of multiple target sequences by a single set of polymerase chain reaction primers; and the existence of demes with different pathogenic potential after to analyse the enzyme activity. The small subunit ribosomal RNA genes of 51 isolates were amplified using standard polymerase chain reaction conditions and the primers RD5 y RD3 (Clark, CG; 1992). The amplification products were digested with 3 restriction enzymes (Hinf I, Rsa I, Alu I). We used a similar method by the primers F1 y R1 (Böhm-Gloning et al., 1997) and the same restriction endonuclease enzymes. Extensive sequence variation was discovered in B. hominis ribosomal RNA genes and the isolates grouped of at least five differents patterns or ribodemes. The ribodeme R2 was the most frequently isolated variant, is composed of isolates obtained from patients with acute diarrhea. The statistical association found between R2 and acute diarrhea in the absence of other enteropathogens suggest the pathogenic role of this ribodema. The results based on the amplification products obtaines by a single set of polymerase chain reaction primers TR7 y TR8 (Init et al., 1999) showed intrastrain variations among isolates, but this set of PCR primers only can be used in the detection of variable DNA repeat patterns in axenic cultures because the reaction is not selectivity for lower eukayotes, it is present in prokaryotes too . The enzyme polymorphism of this organism has been demostrated by enzyme electrophoresis of 31 axenic B. hominis isolates by electrophoresis on polyacrylamide gel under nondenaturating conditions (PAGE). Eight enzyme systems were studied: GOT (EC 2.6.1.1), GPI (EC 5.3.1.9), EM (EC 1.1.1.40), G6PDH (EC 1.1.1.49), 6PGDH (EC 1.1.1.44), MDH (EC 1.1.1.37), HK (EC 2.7.1.1), PGM (EC 2.7.5.1. ). The isolates showed an identical isoenzyme profile by the seven activities, in the case of MDH the presence of one band with different electrophoretic migrations detected the existence the two zimodemes. The description of different zymodemes confirms the heterogeneity of this organism. This study illustrates the need to reexamine the role of B. hominis in disease taking the genetic diversity of the parasite into account.
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Al-Samarai, Abdul-Ghani M. Ali Hasani. "Chemotherapy of parasitic infection by Herpesvirus hominis." Thesis, University of Sheffield, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318149.

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Meygret, Alexandra. "Caractérisation d'éléments conjugatifs intégratifs (ICE) chez Mycoplasma hominis." Thesis, Bordeaux, 2019. http://www.theses.fr/2019BORD0177/document.

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Les mycoplasmes sont des bactéries à petit génome dérivées d’ancêtres à Gram positif par une succession de pertes de matériel génétique. Il a longtemps été considéré que la réduction génétique était la seule force régissant l’évolution de ces bactéries, cependant, des transferts horizontaux de grandes régions chromosomiques au sein et entre les espèces de mycoplasmes ont été récemment mis en évidence. Des éléments conjugatifs et intégratifs (ICE) découverts chez certaines espèces de mycoplasme pourraient être à l’origine de ces transferts. Ces ICEs codent les systèmes nécessaires pour leur excision, leur transfert conjugatif et leur intégration dans la cellule receveuse.Mycoplasma hominis est un mycoplasme commensal des voies génitales qui peut être responsable d’infections gynécologiques, d’infections néonatales et d’infections extragénitales. L’analyse préliminaire de génomes de M. hominis avait montré la présence de régions codantes caractéristiques des ICEs. Les objectifs de cette thèse étaient de rechercher et caractériser les ICEs chez 12 isolats cliniques de M. hominis entièrement séquencés et de déterminer la prévalence de ces ICEs au sein de l’espèce M. hominis. Pour cela, une étude rétrospective sur une période de 6 ans a été menée sur des isolats cliniques obtenus au CHU de Bordeaux. Les concentrations minimales inhibitrices des tétracyclines et des fluoroquinolones ainsi que les mécanismes de résistance ont été déterminés, permettant de disposer d’une collection d’isolats cliniques caractérisés pour l’étude des ICEs.Des ICEs de près de 30 kpb ont été trouvés en une ou plusieurs copies dans sept des 12 souches de M. hominis séquencées. Seulement cinq de ces ICEs semblaient fonctionnels puisqu’une forme circulaire a pu être détectée. Tous les ICEs de M. hominis présentaient une structure similaire avec un module spécifique de M. hominis d’environ 4-kpb, codant des protéines ayant des caractéristiques structurelles similaires à des effecteurs TAL (transcription activator-like), impliqués dans la reconnaissance de nucléotides et dans la transduction de signaux chez les bactéries symbiotiques. La caractérisation des mécanismes de résistance aux antibiotiques des isolats cliniques de M. hominis collectés au CHU de Bordeaux nous a permis de disposer d’une collection de 183 isolats isolés entre 2010 et 2015, parmi lesquels 14,8% étaient porteur du gène tet(M) responsable de la résistance aux tétracyclines, 2,7% étaient résistant à la lévofloxacine et 1,6% étaient résistants à la moxifloxacine par mutation des gènes de la topoisomérase IV et de l’ADN gyrase. Le screening de 120 de ces isolats cliniques a révélé une prévalence élevée des ICEs dans l’espèce M. hominis, mesurée à 45%. Il n’y avait pas de prédominance des ICEs dans les isolats portant le gène tet(M), suggérant que les ICEs n’étaient pas responsables de la dissémination de la résistance à la tétracycline.Des expériences complémentaires de conjugaison seront nécessaires pour confirmer la fonctionnalité des ICEs retrouvés dans l’espèce M. hominis. Cependant, la forte prévalence et le caractère très conservé des ICEs chez M. hominis suggèrent que ces ICEs pourraient conférer un avantage sélectif pour la physiologie ou la physiopathologie de la bactérie. Ce travail ouvre ainsi la voie à de futures études qui permettront une meilleure compréhension des transferts horizontaux de gènes et des facteurs de virulence chez M. hominis
Mycoplasmas are small-genome bacteria derived from Gram-positive ancestors by a succession of genetic material losses. It has long been considered that genetic reduction was the only force governing the evolution of these bacteria, however, horizontal transfers of large chromosomal regions within and between mycoplasma species have recently been reported. Conjugative and integrative elements (ICE) found in some species of mycoplasma may be responsible for these transfers. These ICEs encode the systems necessary for excision, conjugative transfer and integration into a recipient cell.Mycoplasma hominis is a commensal genital mycoplasma that can be responsible for gynecological infections, neonatal infections and extragenital infections. Preliminary analysis of M. hominis genomes had showed the presence of coding regions characteristic of ICEs. The objectives of this thesis were to search for and characterize ICEs in one reference strain and 11 fully sequenced M. hominis clinical isolates and to determine the prevalence of these ICEs in the M. hominis species. To do so, a retrospective study over a period of 6 years was conducted on clinical isolates collected at the Bordeaux University Hospital. The minimum inhibitory concentrations of tetracyclines and fluoroquinolones as well as resistance mechanisms were determined, providing a collection of clinical isolates characterized for the study of ICEs.ICEs of 27-30 kpb were found in one or two copies in seven of the 12 M. hominis sequenced strains. Only five of these ICEs seemed functional since circular forms of extrachromosomal ICE were detected. All M. hominis ICEs exhibited a similar structure consisting of a 4.0-5.1 kb module composed of five to six juxtaposed CDSs, encoding proteins that share common structural features with transcription activator-like (TAL) effectors, involved in polynucleotide recognition and signal transduction in symbiotic bacteria. The characterization of antibiotic resistance mechanisms in M. hominis clinical isolates collected at Bordeaux University Hospital enabled us to obtain a collection of 183 isolates isolated between 2010 and 2015, of which 14.8% harbored the tet(M) gene responsible for tetracycline resistance, 2.7% were resistant to levofloxacin and 1.6% were resistant to moxifloxacin by mutation in topoisomerase IV and DNA gyrase genes. Screening of 120 of these clinical isolates revealed a high prevalence of ICEs in M. hominis, measured to be 45%. The proportion of ICEs was not higher in isolates carrying the tet (M) gene, suggesting that ICEs were not responsible for the spread of tetracycline resistance.Additional mating experiments will be necessary to confirm the functionality of the ICEs found in the M. hominis species. However, the conserved and specific structure of M. hominis ICEs and the high prevalence in clinical strains suggest that these ICEs may confer a selective advantage for the physiology or pathogenicity of the bacteria. This work opens the way for future studies that will provide a better understanding of horizontal gene transfers and virulence factors in M. hominis
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ROUSSET, MARJOLAINE. "Contribution a l'etude du blastocystis hominis : interet et signification dans un examen de selles." Angers, 1988. http://www.theses.fr/1988ANGE1023.

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Camilo, Cacília da Cunha. "Susceptibilidade de Mycoplasma Hominis e Ureaplasma Sp. a antimicrobianos." Universidade Federal do Amazonas, 2012. http://tede.ufam.edu.br/handle/tede/5317.

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Mycoplasmas are microorganisms lacking cell walls with reduced ability Biosintética, which makes them extremely fastidious growth. The strength of these microorganisms to antimicrobials used in his therapy as tetracyclines, macrolides and quinolones have been reported with increased frequency. The determination of antimicrobial susceptibility is particularly difficult because not shown in broth turbidity which complicates the standardization of the inoculum and are extremely susceptible pH conditions. The method most commonly used for this purpose is based on metabolic inhibition using specific substrates. This study evaluated the susceptibility of isolates of Mycoplasma hominis and Ureaplasma sp. stored in the period from 2011 to 2012. The methodology used for the isolation comprised culture techniques, for storage and freezing of the strains we used selective broth enriched PPLO and BHI, Thawing of isolates was succeeded subculture in broth enriched selective and differential agar A7 for phenotypic characterization. Quantification, identification and antimicrobial susceptibility testing was performed using the the triage system Mycofast Evolution ® Screening (Elitech Group), whose identification is based on bacterial susceptibility by Identibiotiqué system. The susceptibility of the isolates was determined against antimicrobial agents Doxiciclina 8 μg/ml, Roxitromicina 4 μg/ml and Ofloxacina 4 μg/ml, widely used in the empirical treatment of patients with urogenital tract infections. Our study showed high level of resistance of the isolates to doxicycline and ofloxacin. This is the first study involving isolation and susceptibility profile of these agents undertaken in the Amazonia.
Micoplasmas são microrganismos desprovidos de parede celular, com reduzida capacidade Biossintética, o que os torna extremamente fastidiosos ao crescimento. A resistência destes microrganismos aos agentes antimicrobianos utilizados na sua terapia como as tetraciclinas, macrolídeos e quinolonas tem sido relatadas com frequência cada vez maior. A determinação da susceptibilidade a antimicrobianos é particularmente difícil porque não mostram turbidez em caldo o que dificulta a padronização do inóculo e são extremamente susceptíveis as condições de pH. A metodologia utilizada para este fim baseia-se na inibição metabólica utilizando substratos específicos. O presente trabalho avaliou a susceptibilidade de isolados de Mycoplasma hominis e Ureaplasma sp. arrmazenados no período de 2011 a 2012. A metodologia utilizada englobou técnicas de cultura; para a estocagem e congelamento das cepas empregou-se caldo seletivo enriquecido PPLO e BHI, O descongelamento dos isolados foi sucedido de subcultivos em caldos enriquecidos seletivos diferenciais e Agar A7 para caracterização fenotípica. A quantificação, a identificação e o teste de susceptibilidade a antimicrobianos foi realizada através do sistema de triagem Mycofast® Screening Evolution (Elitech Group), cuja identificação se baseia na susceptibilidade bacteriana pelo sistema Identibiotiqué. A susceptibilidade dos isolados foi determinada frente aos antimicrobianos doxiciclina 8 μg/ml, roxitromicina 4 μg/ml e ofloxacina 4 μg/ml, amplamente utilizados no tratamento empírico de pacientes com infecções do trato urogenital. Nosso estudo detectou alto nível de resistência dos isolados armazenados para doxiciclina e ofloxacina. Este é o primeiro estudo envolvendo isolamento e o perfil de susceptibilidade destes agentes realizado na região norte.
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Vanee, Niti. "The Genome Scale Metabolic Model of Cryptosporidium hominis: iNV209." VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1909.

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The apicomplexan Cryptosporidium is a protozoan parasite of humans and other mammals. Cryptosporidium species cause acute gastro-enteritis and diarrheal disease in healthy humans and animals, and cause life-threatening infection in immuno-compromised individuals such as people with AIDS. It has a one-host life cycle and invades intestinal epithelial cells causing diarrhea, or more rarely the pulmonary epithelium. Cryptosporidium carries out all the asexual reproductive stages like several other apicomplexans. Current annotation of this organism predicts it to contain 3884 genes of which only 1581 genes have predicted functions. By using a combination of bioinformatics analysis, biochemical evidence, and high-throughput data, a genome-scale metabolic model of Cryptosporidium hominis is being constructed. The current model is comprised of approximately 213 gene-associated enzymes involved in major metabolic pathways including carbohydrate, nucleotide, amino acid, and energy metabolism. The approach of constructing a genome-scale model provides a link between the genotype and the phenotypic behavior of the organism, making it possible to study and predict behavior based upon genome content. This modeling approach provides an overview for evaluating missing components in a metabolic network and provides an analytical framework for interpreting data as more research becomes available. The goal of constructing this model is to systematically study and analyze various functional behaviors of C. hominis with respect to its stages in life cycle and pathogenicity.
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Glaap, Oliver. "Untersuchungen zu Giannozzo Nanetti "De dignitate et excellentia hominis" /." Stuttgart ; Leipzig : B. G. Teubner, 1994. http://catalogue.bnf.fr/ark:/12148/cb37625239k.

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Rideau, Fabien. "Clonage et modification du génome de Mycoplasma hominis dans la levure Saccharomyces cerevisiae." Thesis, Bordeaux, 2018. http://www.theses.fr/2018BORD0227/document.

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Mycoplasma hominis est un pathogène humain opportuniste responsable d’infections génitales et néo-natales. Modifier génétiquement cette bactérie est nécessaire afin de comprendre les mécanismes de virulence et d’infection de ce pathogène. Il n’existe à ce jour aucun outil moléculaire efficace permettant de manipuler le génome de M. hominis, limitant les recherches sur sa pathogénicité et son métabolisme particulier reposant sur l’arginine. De nouvelles technologies rassemblées sous le terme de Biologie de Synthèse (BS) ont récemment émergé, offrant des perspectives inédites pour l’étude des mycoplasmes en permettant de modifier leurs génomes à grande échelle et de produire des souches mutantes. Ces travaux menés au J. Craig Venter Institute (JCVI, USA) ont montré que le génome de mycoplasmes apparentés pouvait être cloné et manipulé dans la levure avant d’être transplanté dans une cellule receveuse. La levure sert d’hôte d’accueil temporaire pour modifier le génome de la bactérie. Cette approche novatrice ouvre de nombreuses perspectives dans le cadre du développement de la génomique fonctionnelle chez les mycoplasmes pour lesquels les outils génétiques efficaces sont peu nombreux. Le but de cette thèse a été d’adapter pour la première fois certains outils de BS à M. hominis dans le but de créer des mutants déficients pour une fonction donnée. Pour cela, le génome de la souche type de M. hominis PG21 (665 kb) a été cloné dans la levure Saccharomyces cerevisiae par « Transformation-Associated Recombination cloning » (TAR-cloning). Deux clones (B3-2 et B3-4) de levure possédant le génome complet de M. hominis ont été validés par analyse en PCR simplex, PCR multiplex et électrophorèse en champs pulsé (PFGE). Ces clones levures ont ensuite été propagés en milieu sélectif durant 180 générations (30 passages), afin d’évaluer la stabilité du génome bactérien dans son hôte. Cette expérience a montré que (i) si la taille du génome de M. hominis ne variait pas au cours des premiers passages, elle diminuait progressivement à partir du dixième passage (≈60 générations), et que (ii) les zones du génome enrichies en séquence répétées étaient préférentiellement perdues. En tenant compte de ces résultats, le génome de M. hominis a été modifié chez le clone B3-4 par la technique « Clustered Regularly Interspaced Short Palindromic Repeats/Cas9 » (CRISPR/Cas9) lors de passages précoces. Des clones de S. cerevisiae possédant un génome de M. hominis PG21 complet délété du gène vaa, codant une protéine d’adhésion majeure, ont été ainsi produits. La dernière étape de cette approche consistait à transplanter le génome modifié dans une cellule receveuse de M. hominis ou de Mycoplasma arthritidis, espèce phylogénétiquement la plus proche de M. hominis. Aucun protocole de transformation de M. hominis n’étant disponible au début de nos travaux, cette étape constituait un verrou majeur dans la mise en place des outils de BS chez cette espèce. Ce verrou a été en partie levé puisqu’une méthode de transformation de M. hominis basée sur du polyéthylène glycol (PEG) et mettant en jeu le plasposon pMT85 (plasmide contenant un transposon conférant la résistance à la tétracycline) a été mise au point au laboratoire. Cette technique de transformation, développée pour la souche de référence M. hominis M132 (745 kb) reste encore peu efficace ; elle est néanmoins reproductible et a permis d’obtenir des mutants d’intérêt de M. hominis. Le transformant n°28-2 a, ainsi, été muté dans le gène Mhom132_2390, codant le précurseur de la protéine P75, une adhésine putative de M. hominis. Le séquençage des génomes complets d’autres transformants a révélé l’insertion de multiples copies du transposon et la présence d’évènements de duplication et d’inversion de larges fragments d’ADN dans au moins deux génomes de M. hominis
Mycoplasma hominis is an opportunistic human pathogen responsible for genital and neonatal infections. Genetically modifying this bacterium is necessary to understand the virulence and infection mechanisms of this pathogen. There is currently no effective molecular tool to engineer the genome of this bacterium, limiting research on its pathogenicity and its peculiar metabolism based on arginine.New technologies have recently emerged in the field of Synthetic Biology (BS), offering new perspectives for the study of mycoplasmas by allowing large scale genome modifications and the production of mutant strains. Work at the J. Craig Venter Institute (JCVI, USA) has shown that the genome of related mycoplasmas can be cloned and manipulated in yeast before being transplanted into a recipient cell. The yeast serves as a temporary host to modify the genome of the bacterium. This innovative approach opens many perspectives in the development of functional genomics in mycoplasmas for which there are few effective genetic tools. The goal of this thesis was to adapt a number of BS tools to M. hominis for the first time, in order to create mutants deficient for a given function. To achieve this goal, the genome of the M. hominis type strain PG21 (665 kb) was cloned into the yeast Saccharomyces cerevisiae by Transformation-Associated Recombination cloning (TAR-cloning). Two yeast clones (B3-2 and B3-4) possessing the complete genome of M. hominis were validated by simplex PCR, multiplex PCR and Pulsed Field Gel Electrophoresis (PFGE) analyses. These yeast clones were then propagated in a selective medium for 180 generations (30 passages) to evaluate the stability of the bacterial genome in its host. This experiment showed that (i) the size of the genome of M. hominis did not change during the first passages, it decreased progressively from the tenth passage (≈60 generations), and (ii) the enriched genome areas in repeated sequence were preferentially lost. Thus, the genome of M. hominis was modified in the B3-4 clone at early passages using the Clustered Regularly Interspaced Short Palindromic Repeats/Cas9 (CRISPR/Cas9) technology. Yeast clones with a complete M. hominis PG21 genome with a deleted vaa gene, encoding a major adhesion protein, were produced using this approach. The final step of this approach was to transplant the modified genome into a recipient cell of M. hominis or Mycoplasma arthritidis, the species phylogenetically closest to M. hominis. As no M. hominis transformation protocol was available at the beginning of our work, this step constituted a major obstacle in the implementation of BS tools in this species. This barrier has been partially lifted since a method of transformation of M. hominis based on polyethylene glycol (PEG) and involving the plasposon pMT85 (plasmid carrying a transposon conferring resistance to tetracycline) has been developed in the laboratory. This transformation technique, developed for the reference strain M. hominis M132 (745 kb) still remains not very efficient; it is nevertheless reproducible and allowed to obtain M. hominis mutants of interest. The Mhom132_2390 gene, encoding the precursor of the P75 protein, a putative adhesin of M. hominis, was effectively mutated in transformant No. 28-2. Complete genome sequencing of other transformants revealed the insertion of multiple copies of the transposon and the presence of duplication and inversion of large DNA fragments within at least two M. hominis genomes.In conclusion, this data has opened the way for the development and transposition of existing genetic modification approaches to M. hominis, previously considered as a genetically intractable bacterium
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Books on the topic "Hominis"

1

Madách, Imre. Tragoedia hominis. Szeged: Madách Irodalmi Társaság, 2006.

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Micó, Jesús. Natura hominis: Taxonomías. Salamanca: Universidad de Salamanca, 1995.

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Mirandola, Giovanni Pico della. De hominis dignitate. Pisa: Scuola normale superiore, 1985.

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Moreno, Morani, ed. De natura hominis. Leipzig: B.G. Teubner, 1987.

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Mirandola, Giovanni Pico della. Oratio de hominis dignitate. Pordenone: Edizioni studio tesi, 1994.

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Cruzata, Ana Luisa Rodríguez. Opus Dei: Opus hominis. La Habana: Editorial de Ciencias Sociales, 1998.

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Nemesius. Nemesii Emeseni De natura hominis. Leipzig: Teubner, 1987.

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Nemesius Emesenus. Nemesii Emeseni De natura hominis. Edited by Moreno Morani. Berlin, Boston: DE GRUYTER, 1987. http://dx.doi.org/10.1515/9783110333633.

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Mirandola, Giovanni Pico della. De hominis dignitate =: La dignità dell'uomo. 2nd ed. [Milano]: S.Berlusconi, 1995.

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Saprykin, D. L. Regnum Hominis: Imperskiĭ proekt Frensisa Bėkona. Moskva: Izd-vo "Indrik", 2001.

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Book chapters on the topic "Hominis"

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d’Ors, Angel. "Hominis Asinus/Asinus Hominis." In Sophisms in Medieval Logic and Grammar, 382–97. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1767-8_22.

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Stöcker, W. "Mykoplasma hominis." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_2205-1.

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Stöcker, W. "Mykoplasma hominis." In Springer Reference Medizin, 1706. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2205.

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Ringelmann, R., and Beate Heym. "Blastocystis hominis." In Parasiten des Menschen, 92–93. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_18.

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Ringelmann, R., and Beate Heym. "Enteromonas hominis." In Parasiten des Menschen, 144–45. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_43.

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Ringelmann, R., and Beate Heym. "Gastrodiscoides hominis." In Parasiten des Menschen, 150–51. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_46.

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Ringelmann, R., and Beate Heym. "Sarcocystis hominis." In Parasiten des Menschen, 216–17. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_78.

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Ringelmann, R., and Beate Heym. "Trichomonas hominis." In Parasiten des Menschen, 249. Heidelberg: Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_92.

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Mehlhorn, Heinz. "Pentatrichomonas hominis." In Encyclopedia of Parasitology, 2126. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_3461.

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Mehlhorn, Heinz. "Blastocystis hominis." In Encyclopedia of Parasitology, 340–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_416.

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Conference papers on the topic "Hominis"

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Oliveira, Marina Mara Sousa de, Hyan Staytskowy Magalhães Martins, Rafael Pereira de Vasconcelos, Renata Mirian Nunes Eleutério, and José Eleutério Júnior. "Microbiota do colo uterino por Gram em pacientes com infeção por papilomavírus humano e outras infecções sexualmente transmissíveis." In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p035.

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Introdução: A microbiota vaginal é um complexo sistema com diversidade de microrganismos. A disbiose parece aumentar o risco de infecções, principalmente as sexualmente transmissíveis, entre as quais por papilomavírus humano, agente associado a lesões cervicais. Objetivo: Avaliar os diferentes tipos de microbiota cervical e as suas características no esfregaço de material residual de citologia em meio líquido, associando com o papilomavírus humano e com Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma genitalium, Mycoplasma hominis, Ureaplasma urealyticum, Trichomonas vaginalis. Métodos: O estudo analisou 179 casos que tinham material residual de citologia em meio líquido. Alíquota do material foi colocado em lâmina adequada, fixado a seco e corado por método de Gram para leitura em microscópio óptico. Outra alíquota foi utilizada para estudo em reação em cadeia da polimerase - transcriptase reversa e multiplex para pesquisas dos microorganismos associados a infecções sexualmente transmissíveis. O teste exato de Fisher com intervalo de confiança foi utilizado para significância estatística. O projeto foi aprovado em comitê de ética sob número 24071519.9.0000.5049 (UniChristus). Resultados: Os casos foram divididos conforme o escore de Nugent aplicado a esfregaços corados pelo método de Gram. Em microbiota cervical normal (escores de 0 a 3), 100 casos (55,86%); em microbiota intermediária (escore de 4 a 6), 51 casos (28,5%); em sugestivo de disbiose (escore de 7 a 10), 28 casos (15,64%). Nos casos de disbiose, foram observados: Chlamydia trachomatis (1[3,57%]), Mycoplasma hominis (7[25%]), Ureaplasma urealyticum (1[3,57%]), papilomavírus humano 16/45 (1[3,57%]), papilomavírus humano de alto risco (AR) (3[10,71%]) e AR e 16/45 (1[3,57%]). No grupo normal, foi a seguinte distribuição: Ureaplasma urealyticum (1[1%]), papilomavírus humano 16 (2[2%]), papilomavírus humano 18/45 (3[3%]), AR (13[13%]). No grupo intermediário, a distribuição foi: Ureaplasma urealyticum (2[3,9%]), papilomavírus humano AR (5[9,8%]) e papilomavírus humano AR, 16 (1[3,9%]). A única diferença significativa foi de Mycoplasma hominis na disbiose (p<0,0001). Conclusão: O estudo não evidenciou uma associação maior no grupo de disbiose com a maioria das infecções sexualmente transmissíveis, no entanto, com Mycoplasma hominis, foi significativo.
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Qiao, Na-na, Qiang Gao, Qin Zhao, De-pei Wang, Wen-yi Zhao, and Chang-yan Yu. "The Cloning and Analysis of a Partial Lipase Gene Sequence of Staphylococcus hominis GIMT1.079: Partial Lipase Gene Sequencing of S. hominis." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE 2010). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5518078.

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Cláudio, Samuel Rangel, NILCE MARIA PEREIRA DOS SANTOS MENEZES, BÁRBARA DOS ANJOS ROSÁRIO, and MARCELO RICARDO ROSA. "OCORRÊNCIA DO SARCOPTES SCABIEI VAR. HOMINIS (ESCABIOSE) NA BAIXADA SANTISTA." In II Congresso Brasileiro de Saúde Pública On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrasp/11580.

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Feriyawati, L., D. R. Anggraini, and T. A. Nasution. "Characteristic Description of Multiparous Women with Mycoplasma hominis and Ureaplasma urealyticum Infection at Outpatient Clinic in Medan." In International Conference of Science, Technology, Engineering, Environmental and Ramification Researches. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0010093108370840.

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Pecyna, Paulina, Marzena Gajęcka, Zuzanna Babalska, Klaudia Szymendera, Marcin Gabryel, Dorota Mańkowska-Wierzbicka, Dorota Nowak-Malczewska, et al. "QUALITATIVE IDENTIFICATION OF <em>ROSEBURIA HOMINIS </em>IN FAECES SAMPLES OBTAINED FROM PATIENTS WITH IRRITABLE BOWEL SYNDROME AND HEALTHY INDIVIDUALS." In 1st International Electronic Conference on Microbiology. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/ecm2020-07096.

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Telleria, Dr Rubén Eduardo Villalobos, Ender Rafael Cumare, and Sergio Fernando Diaz. "RELAÇÃO ENTRE A QUALIDADE DA ÁGUA E AS PARASITOSES INTESTINAIS, NA POPULAÇÃO DE PALMASOLA, DO MUNICÍPIO DE PALMASOLA, ESTADO FALCÓN, VENEZUELA, JANEIRO-MAIO 2008." In II Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbrapah/23.

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Introdução: A parasitose intestinal é uma doença multifatorial, onde desempenham um papel importante as condições ambientais e os costumes higiênicos da população. Objetivo: Para o que nesta investigação, estudou-se a qualidade da água tendo como objetivo relacionar está com o aparecimento de parasitoses. Material e métodos: Foram determinadas as análises físico-químicas, bacteriológicas e parasitológica da água, bem como o estudo coproparasitológico dos moradores da comunidade de Palmasola, especificamente aos responsáveis pela manipulação dos alimentos das diferentes famílias que integravam a população em estudo (70 amostras de solo), e tirou uma (1) mostra da água para cada análise. Resultados: Nas análises realizadas no poço do Km 28 do Município Palmasola, não houve crescimento bacteriano, interpretado como 2 para colônias coliformes totais, 2 para colônias termorresistentes (fecais) e <100 para colônias aeróbias heterotróficas, enquanto no tanque metálico da População de Palmasola e a rede de distribuição correspondente ao refeitório da EBE "Susana MM Dorante" de Palmasola relatou crescimento de colônias bacterianas, tanto de coliformes totais quanto de coliformes resistentes ao calor (fecais), sendo > 16 e > 100 colônias respectivamente. Quanto à análise físico-química da água, não houve nenhuma alteração em seus diferentes parâmetros; e apesar disso, houve crescimento bacteriano e parasitário (um (01) Helminto) na rede de distribuição. A incidência de Parasitoses Intestinais foi alta, de 52,86% correspondendo a 37 casos de parasitóides, sendo: Giardia lamblia (37,84%), Blastocystis hominis (27,03%), Entamoeba coli (16,22%), Endolimax nana (8,11%), Áscaris lumbricoides (5,11%) e Trichuris trichiura (5,11%), bem como verificou-se que os (86,49%) dos casos parasitóides consomem água sem ferver. Conclusão: Conclui-se que nesta comunidade existe uma alta relação entre o consumo de água não tratada e o aparecimento de parasitoses intestinais (86,49%), além de fatores de risco, como o são: a má disposição do lixo, sistema inadequado deexcreções, falta de educação sanitária, entre outros; para que foram sugeridas as medidas necessárias para a solução deste problema.
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Vieira, Daniella Serafin Couto, Bráulio Leal Fernandes, Érica Elaine Traebert Simezo, Amanda Amaro Pereira, and Mara Scheffer. "STANDARDIZATION OF THE LABORATORY RESEARCH PROTOCOL AND ANATOMOPATHOLOGICAL DIAGNOSIS OF BREAST GRANULOMATOUS INJURIES." In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1020.

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Introduction: Granulomatous breast lesions are multifactorial conditions, with clinical, mammographic, and ultrasound findings like those observed in cases of breast carcinoma. Histological evaluation can present key characteristics to define the lesion pattern. Although this entity is rarely reported in the literature, it is associated with inflammatory conditions such as Ductal Ectasia and foreign body reaction. However, it can also be associated with agents such as bacteria, fungi, and parasites. Furthermore, idiopathic causes and exclusion diagnosis, such as sarcoidosis, can be included in the etiology of the process. Objectives: To establish a correlation between the anatomopathological diagnosis and the laboratory investigation by culture for breast granulomatous lesions diagnosis, with validation of the sample’s analysis protocol. Methods: Samples were selected from 17 women treated at the Mastology Service of a Public Hospital in Brazil, with a history and physical examination that raised suspicion of breast granulomatous lesion and they had previous clinical indication of breast core biopsy. The collection of samples was guided by ultrasonography (USG). In turn, they were stored in a blood culture flask (BD BACTECTM) to perform culture tests by automation (VITEK2), bacterioscopy and fungi exams, and acid resistant bacillus (ARB) tests at the Laboratory of Clinical Analyses. Simultaneously, core biopsy samples, fixed in 10% buffered formaldehyde, were sent to a Laboratory of Pathological Anatomy, for a morphological evaluation and research of ARB, fungi, and other bacteria, using the Ziehl-Nielsen, Grocott, PAS and GRAM histochemical methods. Results: Of the 17 samples, 11 had a chronic inflammatory response pattern with a non-lobulocentric granulomatous reaction component and one of them had a lymphocytic mastitis pattern. The five patients with morphological pattern of lobulocentric granulomatous mastitis presented positive culture, four for Corynebacterium kroppenstedtil, and one for Staphylococcus hominis. Sensitivity, Specificity, Positive Predictive Value and Negative Predictive Value in this sample was 100%. Conclusion: The clinical and radiological aspects can bring difficulties that obscure the diagnostic and etiological interpretation of granulomatous lesions. Thus, the morphological details observed in the anatomical pathological examination and the use of the laboratory investigation protocol with standardization of histochemical reactions associated with the use of tools for microbiological diagnosis show increased sensitivity and specificity for the detection of specific etiologic agents in granulomatous mastitis.
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Campos, Maíra Dias De Oliveira, Ednayra Carvalho Da Silva Barbosa, Edsamara Da Silva Yoshida, Gabriella Eller Gonçalves, and Louise Helena De Freitas Ribeiro. "ESCABIOSE NA INFÂNCIA: A IMPORTÂNCIA DA EDUCAÇÃO ESCOLAR." In II Congresso Brasileiro de Parasitologia Humana On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbrapah/16.

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Introdução: A escabiose, popularmente conhecida como sarna, é uma doença contagiosa causada pelo ácaro Sarcoptes scabiei variedade hominis, artrópode parasita, o qual é transmitida pelo contato direto com uma pessoa infectada. A literatura relata que crianças e pessoas com sistema imunológico enfraquecido são mais suscetíveis à infecção. O número médio de fêmeas do ácaro Sarcoptes scabie em um único ciclo de infestação em adultos é de 12, enquanto em crianças são de 20, resultando em uma maior infestação em crianças do que em adultos, por isso, cuidados voltados à prevenção do surto de sarna nos centros de educação infantil é uma necessidade genuína e indubitável. Objetivos: Explanar a importância da educação infantil em relação à escabiose e sua natureza infecciosa. Material e métodos: As bases de dados Scielo, BVS e PubMed foram utilizadas com o intuito de realizar uma análise crítica fazendo uso dos descritores “escabiose”, “educação infantil” e "saúde pública", assim como as pesquisas tiveram como base idiomas nas línguas português, inglês e espanhol. Utilizou-se materiais publicados entre 2018 a 2021, foram selecionados, também, pesquisas que descreviam a patologia em populações infantis em âmbito escolar. Resultados As crianças são especialmente mais propensas às infecções visto que seus sistemas imunológicos ainda estão em desenvolvimento e por colocarem constantemente as mãos e outros objetos na boca. Isso ocorre porque o indivíduo infantil, na fase oral, é mais propenso a inspecionar o ambiente com as mãos e os lábios. Como resultado, essa parcela de indivíduos compartilha suas secreções entre si, propagando-se assim os parasitas locais. Por isso, intervenções preventivas devem incluir cuidados e educação dos educadores e dos familiares. Conclusão: Tanto no pensamento quanto na prática, o cuidado objetivo e técnico devem ser equilibrados com o cuidado subjetivo e simbólico. Dessa forma, profissionais qualificados e compassivos são necessários a fim de que possam ajudar as crianças a terem experiências diárias que incentivem e desenvolvam o autocuidado. Os comportamentos de higiene pessoal e aspectos culturais da limpeza devem ser considerados. Como resultado, o contato regular entre a escola e a família é necessário para que possam resultar em um ambiente saudável.
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Andrews, Brian. "Hominid." In SIGGRAPH Asia 2012 Computer Animation Festival. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2407603.2407623.

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Kato, I. "Homini-Robotism." In Fifth International Conference on Advanced Robotics 'Robots in Unstructured Environments. IEEE, 1991. http://dx.doi.org/10.1109/icar.1991.240485.

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Reports on the topic "Hominis"

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Karpeeva, E. A. Frequency of Occurrence of Pathogenicity Genes in Case of Coculture of Escherichia Coli With Protozoans Blastocystis Hominis. Prof. Dr Kuznetsov Alexandre Semenovich, March 2015. http://dx.doi.org/10.14526/25_2015_25.

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Perkins, John. Bat Homing. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.2202.

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Huston, G. Architectural Approaches to Multi-homing for IPv6. RFC Editor, September 2005. http://dx.doi.org/10.17487/rfc4177.

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Nguyen, Gam D. Reliability Analysis for FDDI Dual Homing Networks. Fort Belvoir, VA: Defense Technical Information Center, May 1993. http://dx.doi.org/10.21236/ada266133.

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Millan, Jose L. Sequence Motifs Specifying Homing and Metastasis to Bone. Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada407485.

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Speyer, Jason L., and David G. Hull. Advanced Guidance Algorithms for Homing Missiles with Bearing-only Measurements. Fort Belvoir, VA: Defense Technical Information Center, May 1986. http://dx.doi.org/10.21236/ada170833.

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Toro, José Miguel de. Exsilium hominum ignorantia est. Honorius Augustodunensis and Knowledge in the twelfth century. Edicions de la Universitat de Lleida, 2019. http://dx.doi.org/10.21001/itma.2019.13.06.

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Cheng, W., L. Wang, H. Li, J. Dong, and A. D'Alessandro. Dual-Homing Coordination for MPLS Transport Profile (MPLS-TP) Pseudowires Protection. RFC Editor, June 2017. http://dx.doi.org/10.17487/rfc8185.

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Nagami, K., S. Uda, N. Ogashiwa, H. Esaki, R. Wakikawa, and H. Ohnishi. Multi-homing for small scale fixed network Using Mobile IP and NEMO. RFC Editor, June 2007. http://dx.doi.org/10.17487/rfc4908.

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Cheng, W., L. Wang, H. Li, S. Davari, and J. Dong. Dual-Homing Protection for MPLS and the MPLS Transport Profile (MPLS-TP) Pseudowires. RFC Editor, June 2017. http://dx.doi.org/10.17487/rfc8184.

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