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Journal articles on the topic 'Hormones gastrointestinales'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2024

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1

REHFELD, JENS F. "The New Biology of Gastrointestinal Hormones." Physiological Reviews 78, no. 4 (1998): 1087–108. http://dx.doi.org/10.1152/physrev.1998.78.4.1087.

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Rehfeld, Jens F. The New Biology of Gastrointestinal Hormones. Physiol. Rev. 78: 1087–1108, 1998. — The classic concept of gastrointestinal endocrinology is that of a few peptides released to the circulation from endocrine cells, which are interspersed among other mucosal cells in the upper gastrointestinal tract. Today more than 30 peptide hormone genes are known to be expressed throughout the digestive tract, which makes the gut the largest endocrine organ in the body. Moreover, development in cell and molecular biology now makes it feasible to describe a new biology for gastrointestinal hor

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2

Lucas, Alan, Stephen R. Bloom, and Albert Aynsley Green. "Gastrointestinal peptides and the adaptation to extrauterine nutrition." Canadian Journal of Physiology and Pharmacology 63, no. 5 (1985): 527–37. http://dx.doi.org/10.1139/y85-092.

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The adaptation to extrauterine nutrition involves complex physiological changes at birth which may be regulated by genetic endowment; enteral nutrients, secretions, and bacteria; and endogenous hormones and exogenous hormones in breast milk. The hypothesis is explored that enteral feeding after birth may trigger key adaptations in the gut and in metabolism partly through the mediation of gastrointestinal hormone secretion. Gut peptides are found in the early human fetal gut and by the second trimester some are found in high concentrations in the fetal circulation and amniotic fluid. Major plas

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Mai, Yang, Francesca K. H. Gavins, Liu Dou, et al. "A Non-Nutritive Feeding Intervention Alters the Expression of Efflux Transporters in the Gastrointestinal Tract." Pharmaceutics 13, no. 11 (2021): 1789. http://dx.doi.org/10.3390/pharmaceutics13111789.

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Intestinal interactions with nutrients, xenobiotics and endogenous hormones can influence the expression of clinically relevant membrane transporters. These changes in the gastrointestinal (GI) physiology can in turn affect the absorption of numerous drug substrates. Several studies have examined the effect of food on intestinal transporters in male and female humans and animal models. However, to our knowledge no studies have investigated the influence of a non-nutritive fibre meal on intestinal efflux transporters and key sex and GI hormones. Here, we show that a fibre meal increased the acu

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Idrovo, Juan-Pablo, Jill A. Shults, Brenda J. Curtis, Michael M. Chen, and Elizabeth J. Kovacs. "Alcohol Intoxication and the Postburn Gastrointestinal Hormonal Response." Journal of Burn Care & Research 40, no. 6 (2019): 785–91. http://dx.doi.org/10.1093/jbcr/irz083.

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Abstract Gastrointestinal hormones are essential in postburn metabolism. Since near 50% of burn victims test positive for blood alcohol levels at hospital admission and have inferior outcomes compared to nonintoxicated burn patients; we hypothesized that the gastrointestinal hormone secretion is compromised in intoxicated burn victims. To test our theory, we quantified gastrointestinal hormones serum levels in a combine ethanol intoxication and burn injury mouse model. Thus, mice received a daily dose of ethanol for 3 days, rested 4 days, and were given ethanol 3 additional days. Mice underwen

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Somogyi, V., A. Gyorffy, T. J. Scalise, et al. "Endocrine factors in the hypothalamic regulation of food intake in females: a review of the physiological roles and interactions of ghrelin, leptin, thyroid hormones, oestrogen and insulin." Nutrition Research Reviews 24, no. 1 (2011): 132–54. http://dx.doi.org/10.1017/s0954422411000035.

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Controlling energy homeostasis involves modulating the desire to eat and regulating energy expenditure. The controlling machinery includes a complex interplay of hormones secreted at various peripheral endocrine endpoints, such as the gastrointestinal tract, the adipose tissue, thyroid gland and thyroid hormone-exporting organs, the ovary and the pancreas, and, last but not least, the brain itself. The peripheral hormones that are the focus of the present review (ghrelin, leptin, thyroid hormones, oestrogen and insulin) play integrated regulatory roles in and provide feedback information on th

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6

Liddle, R. A. "Regulation of cholecystokinin secretion by intraluminal releasing factors." American Journal of Physiology-Gastrointestinal and Liver Physiology 269, no. 3 (1995): G319—G327. http://dx.doi.org/10.1152/ajpgi.1995.269.3.g319.

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Ingested nutrients stimulate secretion of gastrointestinal hormones that are necessary for the coordinated processes of digestion and absorption of food. One of the most important hormonal regulators of the digestive process is cholecystokinin (CCK). This hormone is concentrated in the proximal small intestine and is secreted into the blood on the ingestion of proteins and fats. The physiological actions of CCK include stimulation of pancreatic secretion and gallbladder contraction, regulation of gastric emptying, and induction of satiety. Therefore, in a highly coordinated manner CCK regulate

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Sharman‐Koendjbiharie, Meenakoemarie, Wim P. M. Hopman, Marjolein Piena‐Spoel, Marcel J. I. J. Albers, Jan B. M. J. Jansen, and Dick Tibboel. "Gut Hormones in Preterm Infants With Necrotizing Enterocolitis During Starvation and Reintroduction of Enteral Nutrition." Journal of Pediatric Gastroenterology and Nutrition 35, no. 5 (2002): 674–79. http://dx.doi.org/10.1002/j.1536-4801.2002.tb07928.x.

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ABSTRACTObjectivesGastrointestinal hormones control gut functions in response to enteral nutrition. Diseases involving the gastrointestinal tract, such as necrotizing enterocolitis, may affect gut hormone secretion and therefore influence gut functions. Because bowel rest is an important part of the treatment, infants with this disease are especially at risk for an altered gut hormone secretion and thus for compromised gut functions.MethodsIn the current study, the gastrointestinal hormone profiles of eight preterm infants with an ileostomy after necrotizing enterocolitis (Bell stages 2 and 3)

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Romero Suárez, Tatiana Lissett, Juan José Reyes Vélez, Henry Wellington Calderón Cornejo, Anllelyne Elizabeth Gorozabel Alman, and María Nikolle Intriago Freire. "Rol neuroendocrino del sistema gastrointestinal." Anatomía Digital 6, no. 3.1 (2023): 59–73. http://dx.doi.org/10.33262/anatomiadigital.v6i3.1.2656.

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Introducción: el sistema gastrointestinal, más lejos de su clásico rol en la alimentación e inmunidad, cumple también funciones endocrinas mediante la secreción de hormonas que participan en la homeostasis. Objetivo: exponer las generalidades del rol neuroendocrino del sistema gastrointestinal y su impacto en la homeostasis. Métodos: revisión narrativa de la literatura que incluyó 46 artículos seleccionados en base a su disponibilidad abierta y ser publicados en inglés y español. Conclusiones: el estudio de las hormonas gastrointestinales permite el desarrollo de nuevas tecnologías diagnóstica

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9

Lu, Van B., Fiona M. Gribble, and Frank Reimann. "Nutrient-Induced Cellular Mechanisms of Gut Hormone Secretion." Nutrients 13, no. 3 (2021): 883. http://dx.doi.org/10.3390/nu13030883.

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The gastrointestinal tract can assess the nutrient composition of ingested food. The nutrient-sensing mechanisms in specialised epithelial cells lining the gastrointestinal tract, the enteroendocrine cells, trigger the release of gut hormones that provide important local and central feedback signals to regulate nutrient utilisation and feeding behaviour. The evidence for nutrient-stimulated secretion of two of the most studied gut hormones, glucagon-like peptide 1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP), along with the known cellular mechanisms in enteroendocrine cells r

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10

Harty, Richard F. "Gastrointestinal hormones." Current Opinion in Gastroenterology 7, no. 6 (1991): 906–12. http://dx.doi.org/10.1097/00001574-199112000-00012.

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11

Chaudhri, Owais, Caroline Small, and Steve Bloom. "Gastrointestinal hormones regulating appetite." Philosophical Transactions of the Royal Society B: Biological Sciences 361, no. 1471 (2006): 1187–209. http://dx.doi.org/10.1098/rstb.2006.1856.

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The role of gastrointestinal hormones in the regulation of appetite is reviewed. The gastrointestinal tract is the largest endocrine organ in the body. Gut hormones function to optimize the process of digestion and absorption of nutrients by the gut. In this capacity, their local effects on gastrointestinal motility and secretion have been well characterized. By altering the rate at which nutrients are delivered to compartments of the alimentary canal, the control of food intake arguably constitutes another point at which intervention may promote efficient digestion and nutrient uptake. In rec

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Xie, Cong, Karen L. Jones, Christopher K. Rayner, and Tongzhi Wu. "Enteroendocrine Hormone Secretion and Metabolic Control: Importance of the Region of the Gut Stimulation." Pharmaceutics 12, no. 9 (2020): 790. http://dx.doi.org/10.3390/pharmaceutics12090790.

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It is now widely appreciated that gastrointestinal function is central to the regulation of metabolic homeostasis. Following meal ingestion, the delivery of nutrients from the stomach into the small intestine (i.e., gastric emptying) is tightly controlled to optimise their subsequent digestion and absorption. The complex interaction of intraluminal nutrients (and other bioactive compounds, such as bile acids) with the small and large intestine induces the release of an array of gastrointestinal hormones from specialised enteroendocrine cells (EECs) distributed in various regions of the gut, wh

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13

Prodhan, Utpal K., Shikha Pundir, Vic S. C. Chiang, et al. "Comparable Postprandial Amino Acid and Gastrointestinal Hormone Responses to Beef Steak Cooked Using Different Methods: A Randomised Crossover Trial." Nutrients 12, no. 2 (2020): 380. http://dx.doi.org/10.3390/nu12020380.

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Cooking changes the texture and tenderness of red meat, which may influence its digestibility, circulatory amino acids (AA) and gastrointestinal (GI) hormonal responses in consumers. In a randomised crossover intervention, healthy males (n = 12) consumed a beef steak sandwich, in which the beef was cooked by either a pan-fried (PF) or sous-vide (SV) method. Plasma AA were measured by ultrahigh performance liquid chromatography (UPLC), while plasma GI hormones were measured using a flow cytometric multiplex array. Following meat ingestion, the circulatory concentrations of some of the essential

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14

Dauncey, M. J., and A. Morovat. "Investigation of mechanisms mediating the increase in plasma concentrations of thyroid hormones after a meal in young growing pigs." Journal of Endocrinology 139, no. 1 (1993): 131–41. http://dx.doi.org/10.1677/joe.0.1390131.

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ABSTRACT These studies investigated a number of possible mechanisms which could mediate the increase in plasma concentrations of thyroid hormones after a meal in young growing pigs. It has been established that in animals fed one meal a day, an immediate rise in plasma 3,5,3′-tri-iodothyronine (T3) and a slightly delayed increase in thyroxine (T4) levels are followed by a more sustained peak in both hormones several hours later. The increase in thyroid hormones involves both total and free T3 and T4, and there is no change in plasma albumin, the high-capacity thyroid hormone-binding protein in

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15

Gu, Xiaojing, Wei Zhou, and Juan Han. "Factors Affecting the Readmission of Patients with Pancreatic Cancer after Surgery." Applied Bionics and Biomechanics 2022 (April 29, 2022): 1–6. http://dx.doi.org/10.1155/2022/6106914.

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Objective. Pancreatic cancer is one of the deadliest solid malignancies. Its surgical resection is technically very challenging and has a high risk of complications even after discharge. This study analyzed the risk factors associated with unplanned readmission after pancreatic cancer surgery. Methods. Pancreatic cancer patients who were readmitted within 30 days after surgery were classified as the observation group, while those not readmitted within 30 days postsurgery were classified as the control group. The serum levels of gastrointestinal hormones, stress hormones, and peripheral immune

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16

Borer, Katarina T., Po-Ju Lin, and Elizabeth Wuorinen. "Timing of Meals and Exercise Affects Hormonal Control of Glucoregulation, Insulin Resistance, Substrate Metabolism, and Gastrointestinal Hormones, but Has Little Effect on Appetite in Postmenopausal Women." Nutrients 13, no. 12 (2021): 4342. http://dx.doi.org/10.3390/nu13124342.

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The current prevalence of obesity in the US is strongly associated with excessive food intake and insufficient physical activity. This study examined whether changing the timing of exercise before or after two daily meals could alter human appetite for food. Fifty-four healthy postmenopausal women were matched by body weight and assigned to two groups: (1) two bouts of 2-h moderate-intensity exercise ending one hour before each weight-maintenance meal (XM, n = 23), (2) two-hour moderate-intensity exercise starting 1 h after each weight-maintenance meal (MX, n = 23), and one sedentary control (

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17

Sippel, Rebecca S., Jennifer E. Carpenter, Muthusamy Kunnimalaiyaan, Sara Lagerholm, and Herbert Chen. "Raf-1 activation suppresses neuroendocrine marker and hormone levels in human gastrointestinal carcinoid cells." American Journal of Physiology-Gastrointestinal and Liver Physiology 285, no. 2 (2003): G245—G254. http://dx.doi.org/10.1152/ajpgi.00420.2002.

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Gastrointestinal carcinoid cells secrete multiple neuroendocrine markers and hormones including 5-HT and chromogranin A. The intracellular signaling pathways that regulate production of bioactive molecules are not completely understood. Our aim was to determine whether activation of the raf-1/MEK/MAPK signal transduction pathway in carcinoid cells could modulate production of neuroendocrine markers and hormones. Human pancreatic carcinoid cells (BON) were stably transduced with an estrogen-inducible raf-1 construct creating BON-raf cells. Activation of raf-1 in BON-raf cells led to a marked in

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Wang, Xiaomei, Huifeng Jing, and Yuyue Guo. "Effects of different intensity aerobic exercise on intestinal flora and gastrointestinal hormones in type 2 diabetic rats." Journal of Computational Methods in Sciences and Engineering 24, no. 3 (2024): 1355–62. http://dx.doi.org/10.3233/jcm-247158.

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To examine the impact and mechanisms of the aerobics exercise on gut flora and gastrointestinal hormones in type 2 diabetes rats. Methods Adult male SD rats aged 8 weeks were divided into 5 groups at random (n= 10): a peaceful comparison group (N), a diabetic comparison group (D), a diabetic low-intensity exercise group (LD), a diabetic middle-intensity exercise group (MD), and a diabetes highintensity exercise group. (HD). The rat groups LD, MD, and HD performed aerobic exercise five times per week for a total of 6 weeks and compared general condition, blood glucose, blood lipids, insulin, le

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Goldspink, Deborah A., Frank Reimann, and Fiona M. Gribble. "Models and Tools for Studying Enteroendocrine Cells." Endocrinology 159, no. 12 (2018): 3874–84. http://dx.doi.org/10.1210/en.2018-00672.

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Abstract Gut hormones produced by gastrointestinal enteroendocrine cells modulate key physiological processes including glucose homeostasis and food intake, making them potential therapeutic candidates to treat obesity and diabetes. Understanding the function of enteroendocrine cells and the molecular mechanisms driving hormone production is a key step toward mobilizing endogenous hormone reserves in the gut as a therapeutic strategy. In this review, we will discuss the variety of ex vivo and in vitro model systems driving this research and their contributions to our current understanding of n

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Dietrich, Jean-Bernard. "AR4-2J cells: A model to study polypeptide hormone receptors." Bioscience Reports 16, no. 4 (1996): 273–88. http://dx.doi.org/10.1007/bf01855012.

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The AR4-2J cell line is derived from a transplantable tumour of the exocrine rat pancreas. Acinar in origin, this cell line contains significant amounts of amylase and can be grown in continuous culture. Many in vitro studies have been done using these cells; these studies were often complemented with in vivo experiments on animals. Particularly, many polypeptide hormones interacting with specific receptors located on the cell membrane have been analysed. The accurate knowledge of the hormone-receptor interactions has allowed to design interesting analogs of these hormones. In several cases, t

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Premen, A. J., P. R. Kvietys, and D. N. Granger. "Postprandial regulation of intestinal blood flow: role of gastrointestinal hormones." American Journal of Physiology-Gastrointestinal and Liver Physiology 249, no. 2 (1985): G250—G255. http://dx.doi.org/10.1152/ajpgi.1985.249.2.g250.

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Systemic arterial pressure, jejunal perfusion pressure, and jejunal blood flow were measured in eight autoperfused canine jejunum preparations (5 dogs) before and during local intra-arterial infusion of physiological doses of secretin (18.5 pM), neurotensin (233 pM), and cholecystokinin octapeptide (CCK-8, 30 pM). Intra-arterial infusion of secretin, neurotensin, or CCK-8 alone did not affect either systemic or jejunal arterial pressures. Likewise, jejunal blood flow was not significantly altered by secretin (3 +/- 3%), neurotensin (-5 +/- 4%), or CCK-8 (-5 +/- 5%). Even when all three hormone

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Vukic, Vera, Dragana Jovanovic, and Vesna Skodric-Trifunovic. "Lung carcinoma with paraneoplastic hyponatremia and hypercalcemia." Medical review 63, no. 7-8 (2010): 512–15. http://dx.doi.org/10.2298/mpns1008512v.

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Introduction. Among the numerous paraneoplastic syndromes of the lung carcinoma, metabolic manifestations are of the particular clinical relevance. They are the consequence of the prominent neurosecretory activity of certain peptides and hormones, mostly within amine precursor uptake and decarboxylation (APUD) secretion. Small cell lung carcinoma is the most common cause of paraneoplastic syndromes. Ectopically produced proteins by the tumor are structurally similar but biologically less active than the ?true? hormones, which may be clinically manifested by absent, less frequent or milder symp

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Kim, Ji-Eun, Yun-Ju Choi, Su-Jin Lee, et al. "Laxative Effects of Phlorotannins Derived from Ecklonia cava on Loperamide-Induced Constipation in SD Rats." Molecules 26, no. 23 (2021): 7209. http://dx.doi.org/10.3390/molecules26237209.

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This study investigated the laxative effects of phlorotannins (Pt) derived from Ecklonia cava (E. cave) on chronic constipation by evaluating alterations in stool parameters, gastrointestinal motility, histopathological structure, mucin secretion, gastrointestinal hormones, muscarinic cholinergic regulation, and fecal microbiota in SD rats with loperamide (Lop)-induced constipation subjected to Pt treatment. Stool-related parameters (including stool number, weight, and water contents), gastrointestinal motility, and length of intestine were significantly enhanced in the Lop+Pt-treated group as

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Saito, H., Y. Nakakita, S. Segawa, and Y. Tsuchiya. "Oral administration of heat-killed Lactobacillus brevis SBC8803 elevates the ratio of acyl/des-acyl ghrelin in blood and increases short-term food intake." Beneficial Microbes 10, no. 6 (2019): 671–77. http://dx.doi.org/10.3920/bm2018.0091.

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It is known that gastrointestinal microbiota, probiotics and heat-killed microbes can regulate intestinal immunity; however, their effect on the secretion of gastrointestinal hormones is unclear. The secretion of gastrointestinal hormones can be mediated by the elevation of intracellular Ca2+ concentration, suggesting that these hormones may act through common mechanisms. We have previously shown that heat-killed Lactobacillus brevis SBC8803 (hk-SBC8803) induced the secretion of serotonin and elevated intracellular Ca2+ concentration in serotonin-producing RIN-14B cells, suggesting that hk-SBC

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Guo, Bao-Lian, and Fang-Ping Lou. "The Clinical Observation on PONV with the Intervention of Thumbtack Needles Combined with Chinese Medicine Ironing Therapy." Evidence-Based Complementary and Alternative Medicine 2022 (July 30, 2022): 1–6. http://dx.doi.org/10.1155/2022/2498995.

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Objective. This study aimed to analyze the clinical efficacy of the intervention of thumbtack needles (applicable to subcutaneous embedding) combined with Chinese medicine ironing therapy on postoperation nausea and vomiting (PONV). Methods. 106 patients who scheduled elective surgery were enrolled and randomized into control group and experimental group, with 53 cases in each group. The control group received modern medication, while the experimental group was given thumbtack needles combined with Chinese medicine ironing therapy based on the control group. The PONV score, incidence rate, gas

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Leinskold, T., TE Adrian, U. Arnelo, J. Larsson, and J. Permert. "Gastrointestinal growth factors and pancreatic islet hormones during postoperative IGF-I supplementation in man." Journal of Endocrinology 167, no. 2 (2000): 331–38. http://dx.doi.org/10.1677/joe.0.1670331.

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Insulin-like growth factor-I (IGF-I) has been demonstrated to exert a nitrogen sparing effect, both experimentally and in patients after abdominal surgery. IGF-I is a major mediator for the anabolic effects of growth hormone (GH). Whether elevated circulating IGF-I levels are the sole mediator of the anabolic effects following GH has not been clarified. IGF-I influences glucose metabolism, both through its own specific receptor and by activating the insulin receptor, and has also been proposed to influence pancreatic islet secretion directly. In the present study, the postoperative effects of

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Townsend, Courtney M., Pomila Singh, and James C. Thompson. "Gastrointestinal hormones and gastrointestinal and pancreatic carcinomas." Gastroenterology 91, no. 4 (1986): 1002–6. http://dx.doi.org/10.1016/0016-5085(86)90707-9.

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Moss, S., and J. Calam. "Pathophysiology of gastrointestinal hormones." Current Opinion in Gastroenterology 6, no. 6 (1990): 877–81. http://dx.doi.org/10.1097/00001574-199012000-00005.

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29

Straus, Eugene. "Radioimmunoassay of Gastrointestinal Hormones." Journal of Chemical Education 76, no. 6 (1999): 788. http://dx.doi.org/10.1021/ed076p788.

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Eggstein, S., and E. H. Farthmann. "Hormones and Gastrointestinal Cancer." Digestive Surgery 13, no. 3 (1996): 159–60. http://dx.doi.org/10.1159/000172424.

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Liu, Yifei. "Clinical Effect of Omeprazole Combined with Amoxicillin in the Treatment of Gastric Ulcer." Proceedings of Anticancer Research 7, no. 5 (2023): 1–6. http://dx.doi.org/10.26689/par.v7i5.5300.

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Objective: To investigate the therapeutic effect of omeprazole + amoxicillin in patients with gastric ulcers. Methods: 62 cases of patients with gastric ulcers who were treated from January 2022 to December 2022 were recruited and randomly divided into groups. Omeprazole + amoxicillin treatment was included in the study group, and amoxicillin treatment was included in the control group. The score of gastric ulcer symptoms, time of symptom resolution, gastrointestinal hormone index, and adverse reactions were compared. Results: The gastric ulcer symptom scores in the study group were lower than

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Farhadipour, Mona, and Inge Depoortere. "The Function of Gastrointestinal Hormones in Obesity—Implications for the Regulation of Energy Intake." Nutrients 13, no. 6 (2021): 1839. http://dx.doi.org/10.3390/nu13061839.

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The global burden of obesity and the challenges of prevention prompted researchers to investigate the mechanisms that control food intake. Food ingestion triggers several physiological responses in the digestive system, including the release of gastrointestinal hormones from enteroendocrine cells that are involved in appetite signalling. Disturbed regulation of gut hormone release may affect energy homeostasis and contribute to obesity. In this review, we summarize the changes that occur in the gut hormone balance during the pre- and postprandial state in obesity and the alterations in the diu

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Jordinson, Mark, Robert A. Goodlad, Audrey Brynes, et al. "Gastrointestinal responses to a panel of lectins in rats maintained on total parenteral nutrition." American Journal of Physiology-Gastrointestinal and Liver Physiology 276, no. 5 (1999): G1235—G1242. http://dx.doi.org/10.1152/ajpgi.1999.276.5.g1235.

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Total parenteral nutrition (TPN) causes atrophy of gastrointestinal epithelia, so we asked whether lectins that stimulate epithelial proliferation can reverse this effect of TPN. Two lectins stimulate pancreatic proliferation by releasing CCK, so we asked whether lectins that stimulate gastrointestinal proliferation also release hormones that might mediate their effects. Six rats per group received continuous infusion of TPN and a once daily bolus dose of purified lectin (25 mg ⋅ rat−1 ⋅ day−1) or vehicle alone (control group) for 4 days via an intragastric cannula. Proliferation rates were es

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Trofimova, I. V., L. B. Drygina, and V. N. Ellinidi. "FEATURES OF HORMONE-RECEPTOR INTERACTION BETWEEN STEROID HORMONES AND VITAMIN D IN OSTEOPENIC SYNDROME." Medicо-Biological and Socio-Psychological Problems of Safety in Emergency Situations, no. 2 (September 26, 2018): 101–8. http://dx.doi.org/10.25016/2541-7487-2018-0-2-101-108.

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Relevance. The interrelation of the state of the upper gastrointestinal tract with the change of bone mineral density (BMD) is proved. It is known that hormonal regulation plays a key role against the background of complicated gastroduodenal pathology. Its effect on target organs hormones are mediated by specific receptors, but the level of expression of steroid hormone receptors and vitamin D in acid-dependent diseases of the stomach and in concomitant metabolic disorders in bone tissue remains not sufficiently investigated.Intention.The aim is to study the content of steroid hormone receptor

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Abd-rabou, Ahmed, Hanaa Ahmed, and Emad Eskander. "CLEMASTINE, THE H1 HISTAMINE RECEPTOR ANTAGONIST, ALTERS THE HUMAN SEX AND THYROID HORMONAL PROFILES." Asian Journal of Pharmaceutical and Clinical Research 10, no. 8 (2017): 327. http://dx.doi.org/10.22159/ajpcr.2017.v10i8.19456.

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Objective: Clemastine is widely used as an antihistaminic drug. However, clemastine effectively acts as an antagonist of H1 histamine receptor, it has significant burden adverse effects causing common nervous system, psychiatric, and gastrointestinal ailments, as well as rare cardiac and immune system disorders. The objective of this study is to investigate whether there is a remarkable impact of clemastine administration on the human hormonal pituitary-thyroid-adrenal axis.Methods: To achieve that, hormonal profile was tested in the sera of males and females treated and untreated individuals

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Hellström, Per M., Allan Geliebter, Erik Näslund, et al. "Peripheral and central signals in the control of eating in normal, obese and binge-eating human subjects." British Journal of Nutrition 92, S1 (2004): S47—S57. http://dx.doi.org/10.1079/bjn20041142.

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The worldwide increase in the incidence of obesity is a consequence of a positive energy balance, with energy intake exceeding expenditure. The signalling systems that underlie appetite control are complex, and the present review highlights our current understanding of key components of these systems. The pattern of eating in obesity ranges from over-eating associated with binge-eating disorder to the absence of binge-eating. The present review also examines evidence of defects in signalling that differentiate these sub-types. The signalling network underlying hunger, satiety and metabolic sta

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Henz, Leo J., and Frank E. Johnson. "Ultrastructure of E. coli: An in vitro study of cells cultured in the presence of four gastrointestinal (GI) hormones." Proceedings, annual meeting, Electron Microscopy Society of America 48, no. 3 (1990): 628–29. http://dx.doi.org/10.1017/s0424820100160698.

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Hormones are found in exocrine secretions entering the gut. They alter the morphology of many eukaryotic cells; whether they affect the morphology of enteric flora is unknown. In this study, we examined the ultrastructure of E. coli, a common bacterium in the mammalian gut, for morphological changes resulting from exposure to GI hormones.E. coli (#11775 from American Type Culture Collection) were grown in protease-free trypticase soy broth (TSB) at 37°C for 18 hr to a concentration of 2 x 107 cells/ml. Pure synthetic hormones were used: sulfated C-terminal cholecystokinin octapeptide (CCK), pe

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Puzio, Iwona, Siemowit Muszyński, Piotr Dobrowolski, et al. "Alterations in Small Intestine and Liver Morphology, Immunolocalization of Leptin, Ghrelin and Nesfatin-1 as Well as Immunoexpression of Tight Junction Proteins in Intestinal Mucosa after Gastrectomy in Rat Model." Journal of Clinical Medicine 10, no. 2 (2021): 272. http://dx.doi.org/10.3390/jcm10020272.

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The stomach is responsible for the processing of nutrients as well as for the secretion of various hormones which are involved in many activities throughout the gastrointestinal tract. Experimental adult male Wistar rats (n = 6) underwent a modified gastrectomy, while control rats (n = 6) were sham-operated. After six weeks, changes in small intestine (including histomorphometrical parameters of the enteric nervous plexuses) and liver morphology, immunolocalization of leptin, ghrelin and nesfatin-1 as well as proteins forming adherens and tight junctions (E-cadherin, zonula occludens-1, occlud

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Gribble, Fiona M. "The gut endocrine system as a coordinator of postprandial nutrient homoeostasis." Proceedings of the Nutrition Society 71, no. 4 (2012): 456–62. http://dx.doi.org/10.1017/s0029665112000705.

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Hormones from the gastrointestinal (GI) tract are released following food ingestion and trigger a range of physiological responses including the coordination of appetite and glucose homoeostasis. The aim of this review is to discuss the pathways by which food ingestion triggers secretion of cholecystokinin (CCK), glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) and the altered patterns of gut hormone release observed following gastric bypass surgery. Our understanding of how ingested nutrients trigger secretion of these gut hormones has increased dramatica

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Farias, Gisele, Bárbara Dal Molin Netto, Solange Cravo Bettini, Ana Raimunda Dâmaso, and Alexandre Coutinho Teixeira de Freitas. "Neuroendocrine regulation of energy balance: Implications on the development and surgical treatment of obesity." Nutrition and Health 23, no. 3 (2017): 131–46. http://dx.doi.org/10.1177/0260106017719369.

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Introduction: Obesity, a serious public health problem, occurs mainly when food consumption exceeds energy expenditure. Therefore, energy balance depends on the regulation of the hunger–satiety mechanism, which involves interconnection of the central nervous system and peripheral signals from the adipose tissue, pancreas and gastrointestinal tract, generating responses in short-term food intake and long-term energy balance. Increased body fat alters the gut- and adipose-tissue-derived hormone signaling, which promotes modifications in appetite-regulating hormones, decreasing satiety and increa

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Stern, H. Patrick, Suzanne E. Stroh, Stephen C. Fiedorek, et al. "Increased Plasma Levels of Pancreatic Polypeptide and Decreased Plasma Levels of Motilin in Encopretic Children." Pediatrics 96, no. 1 (1995): 111–17. http://dx.doi.org/10.1542/peds.96.1.111.

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Objective. Abnormalities of hormones affecting gastrointestinal motility have been found in "functional" disorders of the gastrointestinal system in adults. One such disorder of childhood, encopresis, is frequently associated with constipation, the treatment of which often eliminates the soiling. We hypothesized that hormones affecting gastrointestinal motility were different between encopretic patients and matched controls. Methods. Ten encopretic patients were matched by age, race, and sex with controls who had no history of encopresis or constipation. After an overnight fast, each child con

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Lecomte, Marie-José, Chloé Bertolus, Nélina Ramanantsoa, et al. "Acetylcholine Modulates the Hormones of the Growth Hormone/Insulinlike Growth Factor-1 Axis During Development in Mice." Endocrinology 159, no. 4 (2018): 1844–59. http://dx.doi.org/10.1210/en.2017-03175.

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Abstract Pituitary growth hormone (GH) and insulinlike growth factor (IGF)-1 are anabolic hormones whose physiological roles are particularly important during development. The activity of the GH/IGF-1 axis is controlled by complex neuroendocrine systems including two hypothalamic neuropeptides, GH-releasing hormone (GHRH) and somatostatin (SRIF), and a gastrointestinal hormone, ghrelin. The neurotransmitter acetylcholine (ACh) is involved in tuning GH secretion, and its GH-stimulatory action has mainly been shown in adults but is not clearly documented during development. ACh, together with th

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Uvnäs-Moberg, Kerstin. "Gastrointestinal Hormones and Pathophysiology of Functional Gastrointestinal Disorders." Scandinavian Journal of Gastroenterology 22, sup128 (1987): 138–46. http://dx.doi.org/10.3109/00365528709090982.

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Kiss, Rita, Georgina Pesti-Asbóth, Mária Magdolna Szarvas, et al. "Diosgenin and Its Fenugreek Based Biological Matrix Affect Insulin Resistance and Anabolic Hormones in a Rat Based Insulin Resistance Model." BioMed Research International 2019 (April 4, 2019): 1–13. http://dx.doi.org/10.1155/2019/7213913.

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Fenugreek is known since ancient times as a traditional herbal medicine of its multiple beneficial effects. Fenugreek’s most studied and employed effect is its hypoglycemic property, but it can also be useful for the treatment of certain thyroid disorders or for the treatment of anorexia. The regulation of glucose homeostasis is a complex mechanism, dependent on the interaction of different types of hormones and neurotransmitters or other compounds. For the study of how diosgenin and fenugreek seeds modify insulin sensitivity, we used a rat insulin resistance model induced by high-fat diet. Di

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Stote, Kim, Adele Corkum, Marva Sweeney, Nicole Shakerley, Terri Kean, and Katherine Gottschall-Pass. "Postprandial Effects of Blueberry (Vaccinium angustifolium) Consumption on Glucose Metabolism, Gastrointestinal Hormone Response, and Perceived Appetite in Healthy Adults: A Randomized, Placebo-Controlled Crossover Trial." Nutrients 11, no. 1 (2019): 202. http://dx.doi.org/10.3390/nu11010202.

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The consumption of blueberries, as well as the phenolic compounds they contain, may alter metabolic processes related to type 2 diabetes. The study investigated the effects of adding 140 g of blueberries to a higher-carbohydrate breakfast meal on postprandial glucose metabolism, gastrointestinal hormone response, and perceived appetite. As part of a randomized crossover design study, 17 healthy adults consumed a standardized higher-carbohydrate breakfast along with 2 treatments: (1) 140 g (1 cup) of whole blueberries and (2) a placebo gel (matched for calories, sugars, and fiber of the whole b

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Kim, Sung Eun, Seulki Kim, Shin-Hee Kim, et al. "Causality between Sex Hormones and Bone Mineral Density in Childhood: Age- and Tanner-Stage-Matched Sex Hormone Level May Be an Early Indicator of Pediatric Bone Fragility." Biomedicines 12, no. 6 (2024): 1173. http://dx.doi.org/10.3390/biomedicines12061173.

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This study aimed to investigate the impact of hypogonadism on bone mineral density (BMD) in children and adolescents with chronic diseases to determine the relationship between sex hormones and BMD. This retrospective study included 672 children and adolescents with chronic diseases such as hemato-oncologic, rheumatoid, gastrointestinal, and endocrinologic diseases. The relationship between the sex- and Tanner-stage-matched Z-scores for sex hormones and the sex- and age-matched lumbar spine BMD (LSBMD) Z-scores was evaluated. Adjustments were made for confounders such as underlying diseases, a

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Beck, B. "Gastric inhibitory polypeptide: a gut hormone with anabolic functions." Journal of Molecular Endocrinology 2, no. 3 (1989): 169–74. http://dx.doi.org/10.1677/jme.0.0020169.

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Summary The gastrointestinal hormone, gastric inhibitory polypeptide (GIP), has been isolated and characterized because of its enterogastrone-type effects. It is also named glucose-dependent insulinotropic polypeptide and is actually considered to be the main incretin factor of the entero-insular axis. Besides these well-described effects on gastric secretion and pancreatic β cells, it also has direct metabolic effects on other tissues and organs, such as adipose tissue, liver, muscle, gastrointestinal tract and brain. In adipose tissue it is involved in the activation and regulation of lipopr

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Walton, Kristen L. W. "Teaching the role of secretin in the regulation of gastric acid secretion using a classic paper by Johnson and Grossman." Advances in Physiology Education 33, no. 3 (2009): 165–68. http://dx.doi.org/10.1152/advan.00023.2009.

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The regulation of gastric acid secretion has been the subject of investigation for over a century. Inhibition of gastrin-induced acid secretion by the intestine-derived hormone secretin provides a classic physiological example of negative feedback in the gastrointestinal tract. A classic paper by Leonard R. Johnson and Morton I. Grossman clearly shows the ability of secretin to negatively regulate gastric acid secretion, providing students with an example of this feedback loop. In addition, this article demonstrates the step forward in gastrointestinal endocrinology that occurred when pure pre

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Peeters, Theo L. "Gastrointestinal hormones and gut motility." Current Opinion in Endocrinology & Diabetes and Obesity 22, no. 1 (2015): 9–13. http://dx.doi.org/10.1097/med.0000000000000123.

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Aylwin, Simon. "Gastrointestinal surgery and gut hormones." Current Opinion in Endocrinology & Diabetes 12, no. 1 (2005): 89–98. http://dx.doi.org/10.1097/01.med.0000152979.05456.b6.

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