Academic literature on the topic 'Horses, abnormalities'

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Journal articles on the topic "Horses, abnormalities"

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O'Neill, HV Masey, J. Keen, and L. Dumbell. "Dental abnormalities in horses." Advances in Animal Biosciences 1, no. 1 (April 2010): 223. http://dx.doi.org/10.1017/s2040470010003663.

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Dyson, Sue, and Danica Pollard. "Application of a Ridden Horse Pain Ethogram and Its Relationship with Gait in a Convenience Sample of 60 Riding Horses." Animals 10, no. 6 (June 17, 2020): 1044. http://dx.doi.org/10.3390/ani10061044.

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A Ridden Horse Pain Ethogram (RHpE) comprising 24 behaviours has been developed to facilitate the identification of musculoskeletal pain. The aim was to further test the RHpE by its application to a convenience sample (n = 60) of sports horses and riding school horses in regular work and assumed by their owners to be working comfortably. All horses performed a purpose-designed dressage-type test of 8.5 min duration in walk, trot and canter, with their normal rider. The RHpE was applied retrospectively to video recordings acquired in a standardised fashion. Seventy-three percent of horses were lame (≤ grade 2/8) on one or more limbs; 47% had gait abnormalities in canter. Ridden Horse Pain Ethogram scores ranged from 3 to 16/24 (median 9); rider skill score ranged from 2.5 to 8/10 (median 4.75). The effect of horse age, breed, sex, work-discipline, epaxial muscle hypertonicity or pain, an ill-fitting saddle, rider skill score, the presence of lameness or gait abnormalities in canter on the RHpE score was assessed using Poisson regression. Two variables were retained in the final multivariable analysis, rider skill score as a continuous variable (p < 0.001), and lameness (p = 0.008). A RHpE score ≥8 was a good indicator of the presence of musculoskeletal pain.
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Smith, Sarah, and Celia M. Marr. "Spontaneous pneumomediastinum in three adult horses." Veterinary Record Case Reports 7, no. 2 (April 2019): e000732. http://dx.doi.org/10.1136/vetreccr-2018-000732.

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The report describes the diagnosis and management of spontaneous pneumomediastinum in three horses which were presented with acute-onset increased respiratory effort. Pneumomediastinum was evident radiographically without evidence of internal or external trauma. Two horses had no obvious inciting cause and one horse had chronic interstitial pneumonia. All horses developed echocardiographic changes consistent with right heart dysfunction. Radiographic evidence of pneumomediastinum resolved in 14–21 days along with all other clinical abnormalities. Despite the severity of the initial clinical signs, all cases reported made a rapid and complete recovery, suggesting that, as in human beings, spontaneous pneumomediastinum in horses is a benign condition with a good prognosis.
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Bugno, Monika, Ewa Słota, Aldona Pieńkowska-schelling, and Claude Schelling. "Identification of chromosome abnormalities in the horse using a panel of chromosome-specific painting probes generated by microdissection." Acta Veterinaria Hungarica 57, no. 3 (September 1, 2009): 369–81. http://dx.doi.org/10.1556/avet.57.2009.3.3.

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Fluorescent in situ hybridisation (FISH) using a panel of molecular probes for all chromosome pairs obtained by chromosome microdissection of the domestic horse ( Equus caballus ) was used to diagnose karyotype abnormalities in 35 horses (32 mares, 2 stallions and 1 intersex), which were selected for the study due to infertility (23 horses), reduced fertility (10 horses) and developmental anomalies (2 horses). The use of the FISH technique with probes for each horse chromosome pair enabled the diagnosis of many different chromosome aberrations in this population. Among the horses analysed, 21 animals had normal karyotype — 64,XX (19 mares) and 64,XY (2 stallions). Fourteen animals, constituting 40% of the population studied, showed the following chromosome abnormalities: 63,X (1 mare); 63,X/64,XX (6 mares); 63,X/64,XX/65,XXX (3 mares); 63,X/65,XXX (1 mare); 64,XX/65,XX+Xp (1 mare); 63,X/64,XX/65,XX+Xq (1 mare), and 63,X/64,XX/65,XX+delY (1 intersex). When only the mares studied because of complete infertility were taken into consideration, this proportion exceeded 56%. Due to the increased frequency of the above-mentioned aberrations in the mosaic form of two or more lines, it was necessary to analyse a large number (100–300) of metaphase spreads. The use of specific molecular probes obtained by chromosome microdissection made these diagnoses much easier.
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Lear∗, T. L. "Assessing Chromosome Abnormalities in Horses with Infertility or Congenital Abnormalities." Journal of Equine Veterinary Science 29, no. 5 (May 2009): 316–17. http://dx.doi.org/10.1016/j.jevs.2009.04.023.

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Bugno-Poniewierska, Monika, and Terje Raudsepp. "Horse Clinical Cytogenetics: Recurrent Themes and Novel Findings." Animals 11, no. 3 (March 16, 2021): 831. http://dx.doi.org/10.3390/ani11030831.

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Clinical cytogenetic studies in horses have been ongoing for over half a century and clearly demonstrate that chromosomal disorders are among the most common non-infectious causes of decreased fertility, infertility, and congenital defects. Large-scale cytogenetic surveys show that almost 30% of horses with reproductive or developmental problems have chromosome aberrations, whereas abnormal karyotypes are found in only 2–5% of the general population. Among the many chromosome abnormalities reported in the horse, most are unique or rare. However, all surveys agree that there are two recurrent conditions: X-monosomy and SRY-negative XY male-to-female sex reversal, making up approximately 35% and 11% of all chromosome abnormalities, respectively. The two are signature conditions for the horse and rare or absent in other domestic species. The progress in equine genomics and the development of molecular tools, have qualitatively improved clinical cytogenetics today, allowing for refined characterization of aberrations and understanding the underlying molecular mechanisms. While cutting-edge genomics tools promise further improvements in chromosome analysis, they will not entirely replace traditional cytogenetics, which still is the most straightforward, cost-effective, and fastest approach for the initial evaluation of potential breeding animals and horses with reproductive or developmental disorders.
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Ramsey, Ewart, Render, Cook, and Latimer. "Congenital ocular abnormalities of Rocky Mountain Horses." Veterinary Ophthalmology 2, no. 1 (March 1999): 47–59. http://dx.doi.org/10.1046/j.1463-5224.1999.00050.x.

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Timothy, A. O. Olusa. "Do dental abnormalities predispose horses to colic?" Journal of Veterinary Medicine and Animal Health 6, no. 7 (July 31, 2014): 192–97. http://dx.doi.org/10.5897/jvmah2014.0290.

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de Mira, M. C., C. Santos, M. A. Lopes, and D. J. Marlin. "Challenges encountered by Federation Equestre Internationale (FEI) veterinarians in gait evaluation during FEI endurance competitions: an international survey." Comparative Exercise Physiology 15, no. 5 (December 10, 2019): 371–78. http://dx.doi.org/10.3920/cep180058.

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Equine endurance competitions are long races over 1-2 days and horses can be eliminated in international competitions by FEI veterinarians for lameness at any time. Elimination rates due to lameness are high, affect the outcome of the races and commonly elicit objections to the decision from endurance competitors. The aims of this study were: (1) to assess the opinion of FEI official endurance veterinarians (OEVs) about the challenges in assessing lameness; (2) the occurrence of confrontations with riders when horses are eliminated from races due to lameness; (3) to assess OEVs’ thoughts about the adoption of user-friendly technology for objective gait evaluation to help detect, quantify and document lameness. All FEI OEVs were asked to complete a questionnaire. There were 157 responses, being most of the respondents from Europe (56.1%) or the Middle East (16.6%). For the majority of respondents, detection of lameness was considered challenging, even for experienced and well-trained veterinarians (57.3%). OEVs also considered it was often hard to classify horses as lame or sound (65.8%). Handlers not trotting the horse appropriately during gait evaluations was considered the most common problem compromising the evaluation (94.3%). Most OEVs (98.2%) responded that they had been confronted at least once by a rider or associate about the decision to eliminate a horse due to lameness. Most OEVs (71.3%) would be interested in having the support of user-friendly technology for objective gait evaluation (33.3% for all evaluations; 38.0% only when horses have more subtle gait abnormalities). The findings of this survey suggest that technology to objectively detect and quantify gait abnormalities during endurance competitions would be beneficial to support decisions made by OEVs when evaluating and or eliminating horses for lameness.
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Plummer, Caryn E., and David T. Ramsey. "A Survey of ocular abnormalities in Miniature Horses." Veterinary Ophthalmology 14, no. 4 (April 18, 2011): 239–43. http://dx.doi.org/10.1111/j.1463-5224.2010.00868.x.

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Dissertations / Theses on the topic "Horses, abnormalities"

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Liupaitė, Laura. "ARKLIŲ DANTŲ PAKITIMŲ KLINIKINIS ĮVERTINIMAS IR GYDYMAS." Master's thesis, Lithuanian Academic Libraries Network (LABT), 2014. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2014~D_20140305_134026-87428.

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Arklių dantų pakitimai gali būti siejami su daugeliu kitų ligų tokių kaip diegliai, išliesėjimas ir pašaro metimas iš burnos ir panašiai, todėl svarbu laiku aptikti dantų pakitimus ir juos gydyti. 2011 - 2013 metais LSMU veterinarijos akademijos stambių gyvulių klinikoje buvo gydyti 1010 arklių, iš jų 2011 metais buvo 10,57 proc. susijusių su dantų ligomis, 2012 metais - 15,97 proc., 2013 metais 15,95 proc. Dažniausi dantų pakitimai arkliams per 2011 - 2013 metus buvo aštrūs emalio kraštai. Arklio dantys dėvisi visą jo gyvenimą kramtant pašarą, o kasmet tai sudaro apie 2 - 3 mm. Dažniausiai atlikta dantų procedūra per 2011 - 2013 metus buvo dantų lyginimas. 2011 - 2013 metų statistika rodo, kad kasmet arklių su dantų problemomis atvedama daugiau, o tai rodo didesnį gyvūnų augintojų susidomėjimą savo augintiniais.
Clinical evaluation and treatment of equine dental abnormalities performed in Large animal clinic at the department of Non-communicable diseases LUHS Veterinary Academy in 2011 - 2013. In total there were registered 1010 horse patients during the 2011 – 2013 years, including 149 patients associated with dental abnormalities. Horses with dental problems were 10.57% in 2011, 15.97% in 2012, 15.95% in 2013 from all horse patient at LUHS Veterinary Academy Large animal clinic . The most common dental lesions in 2011 - 2013 were the sharp enamel edges. These patients were – 24 in 2011, 54 in 2012 and 71 in 2013 patients. According to the literature, the horse's teeth continue to grow throughout its life. The growing horse teeth worn during mastication process, which is about 2-3 mm annually. Another factor influencing the formation of sharp enamel edges - the specific structure of the head of a horse that is difference in the lower and upper jaw size. The upper jaw is wider than the lower, for that reason, the upper jaw teeth sharp enamel edges are formed from the side of the cheek, while mandibular teeth sharp enamel edges formed by the tongue side. Most common dental procedures performed in 2011 - 2013 has been a dental floating. This procedure is used in many dental abnormalities such as tooth overgrowth, sharp enamel edges resulting from, tooth hooks and so on. This procedure is also used as a prophylactic measure. ... [to full text]
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Hanie, Elizabeth Anne. "Comparison of healing of full-thickness cartilage vs. full- thickness cartilage and subchondral bone defects in the equine third carpal bone." Thesis, This resource online, 1991. http://scholar.lib.vt.edu/theses/available/etd-08182009-040427/.

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Eberth, John E. "Chondrodysplasia-Like Dwarfism in the Miniature Horse." UKnowledge, 2013. http://uknowledge.uky.edu/gluck_etds/11.

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Dwarfism is considered one of the most recognized congenital defects of animals and humans and can be hereditary or sporadic in cause and expression. There are two general morphologic categories within this vastly diverse disease. These categories are disproportionate and proportionate dwarfism and within each of these there are numerous phenotypes which have been extensively described in humans, and to a lesser extent in dogs, cattle, mice, chickens, and other domestic species. Ponies and Miniature horses largely differ from full size horses only by their stature. Ponies are often defined as those whose height is not greater than 14.2 hands; however the maximum height for Miniature horses is constitutionally defined as 8.2 hands. Dwarfism is not considered a desirable genetic trait for Miniature horses. A majority of these conformationally inferior horses showed consistent physical abnormalities typical of disproportionate dwarfisms as seen in other mammal species. A whole genome scan with the Illumina Equine SNP50 chip clearly implicated a region on ECA1 as being associated with dwarfism of horses. The region implicated on the horse chromosome 1 (Equus Caballus; ECA1) contained a candidate gene for dwarfism, aggrecan (ACAN). Mutations were found in Exons 2, 6, 11 and 15 with each mutation associated with a distinct type of dwarfism. These mutations are independently transmitted throughout the population. Absence of normal homozygotes for these mutations and absence of normal horses which were heterozygous for these mutations indicated that these alleles caused dwarfism in those genotypes. These genotypes did not explain all observed dwarves in this population.
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Devine, Dustin Vance. "Gait abnormalities caused by selective anesthesia of the suprascapular nerve in horses." 2006. http://digital.library.okstate.edu/etd/umi-okstate-1781.pdf.

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Mirazo, Michelena Javier E. "Use of overground endoscopy for detection of upper airway abnormalities in Thoroughbred racehorses in South Africa." Diss., 2013. http://hdl.handle.net/2263/36797.

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Upper airway endoscopy at rest has been the diagnostic method of choice for diagnosing equine upper respiratory tract (URT) disease since its development in the 1970´s. The development of high-speed treadmill endoscopy (HSTE) improved the sensitivity of URT endoscopy by allowing the examiner to observe the horse’s nasopharynx and larynx during exercise. However, the level of exertion achieved during HSTE may not always represent that achieved during normal exercise as surface, rider, tack, and environmental variables are altered. Recently, the development of dynamic overground endoscopy (DOE) has addressed some of those shortcomings. A retrospective study was undertaken to describe the upper airway abnormalities detected during DOE in horses presenting with poor performance and/or abnormal respiratory noise in South Africa. Patient records of Thoroughbred racehorses undergoing DOE from November 2011 to August 2012 by the Onderstepoort Veterinary Academic Hospital were reviewed. Data collected included signalment, primary complaint, distance exercised, maximum speed attained, and dynamic airway abnormalities detected. XIII A second study was carried out to evaluate agreement within and among examiners of three grading systems for laryngeal function at exercise. The grading systems assessed were an existing system for grading axial deviation of aryepiglottic folds (ADAF), a modified system for grading recurrent laryngeal neuropathy (RLN) at exercise, and a proposed system for grading vocal cord collapse (VCC). For investigation of intra-observer variability, recordings were watched by two of the investigators at the same time, on two different occasions, in real time, slow motion, and at frame-by-frame speed. To evaluate inter-observer variability, recordings were watched by four investigators on one occasion, as described for investigation of intra-observer variability, and scoring sheets completed. Kappa agreement was calculated for both intra- and inter-observer sessions. Fifty-two horses that underwent DOE for investigation of poor performance and/or abnormal respiratory noise were identified. The main abnormalities detected included dorsal displacement of the soft palate (DDSP) (13/52 horses, 25%); recurrent laryngeal neuropathy (RLN) (17/52 horses, 33%); axial deviation of the aryepiglottic folds (ADAF) (21/52 horses, 40%) and vocal cord collapse (VCC) (18/52 horses, 35%). A total of 40 horses presented one or more abnormalities of the URT (77%). Fifteen horses (29%) had a single abnormality, and 25 horses (48%) had multiple abnormalities. Results at frame-by-frame speed from the intra-observer evaluations for all the conditions showed substantial agreement for RLN by both observers (K = 74 - 80). Intra-observer evaluations for VCC were moderate to substantial (K 53 – 63). ADAF was the most difficult URT abnormality to assess for both observers, and agreement within observers was only fair to moderate (K = 36 - 52). Inter-observer evaluations for RLN showed substantial to moderate agreement (K = 62). Inter-observer evaluations for VCC showed moderate agreement (K = 47 – 54), and inter-observer evaluations for ADAF showed only slight to fair agreement and were the lowest for all the conditions (K = 14 – 22). This study showed that DOE is a useful technique for providing valuable information about disease of the URT. Finding multiple abnormalities in 48% of horses examined using DOE suggests that DOE may be indicated even for those horses with an obvious abnormality found during resting endoscopy. The intra-observer evaluations showed that RLN had higher agreement values than those for ADAF and VCC at all speeds, and that ADAF had lower agreement values than those for VCC and RLN at all speeds. Inter-observer agreement was less than intra-observer agreement, presumably because more observers were involved in the inter-observer assessment.
Dissertation (MSc)--University of Pretoria, 2013.
gm2014
Companion Animal Clinical Studies
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Books on the topic "Horses, abnormalities"

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Pamela C. Wagner von Matthiessen. Equine pediatric orthopedics. [Santa Barbara, Calif.]: Veterinary Practice Publ. Co., 1991.

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Pamela C. Wagner von Matthiessen. Equine pediatric orthopedics. [Santa Barbara, Calif.]: Veterinary Practice Publ. Co., 1991.

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Redden, R. F. Radiography of the equine foot: Techniques for enhancing the quality of your films. Versailles, Ky: Nanric Inc., 2002.

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F, Pycock Jonathan, and McKinnon A. O, eds. Current therapy in equine reproduction. St. Louis, Mo: Saunders Elsevier, 2007.

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Yolen, Jane. Centaur rising. New York: Henry Holt & Company, 2014.

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Before Elvis there was nothing. Minneapolis: Coffee House Press, 2005.

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Cervical vertebral malformation of horses. [S.l: American Association of Equine Practitioners, 1986.

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Ansuini, Patti. Spirit of the warhorse: Kitty Silverwings : an appaloosa story. 2013.

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1963-, Douglas Janet, Moyer William, Redden R. F, and University of Guelph. Equine Research Centre., eds. No foot--no horse. Guelph, Ont: Equine Research Centre, University of Guelph, 1994.

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Samper, Juan C., Jonathan Pycock, and Angus O. McKinnon. Current Therapy in Equine Reproduction (Current Veterinary Therapy). Saunders, 2006.

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Book chapters on the topic "Horses, abnormalities"

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Toribio, Ramiro E. "Electrolyte Abnormalities and Neurologic Dysfunction in Horses." In Equine Neurology, 368–85. Hoboken, NJ: John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118993712.ch29.

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McKenzie, Erica C. "Biochemical abnormalities of athletic horses." In Equine Sports Medicine and Surgery, 931–38. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-7020-4771-8.00043-0.

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RICKETTS, S. "Hematologic and biochemical abnormalities in athletic horses." In Equine Sports Medicine and Surgery, 949–66. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-7020-2671-3.50047-7.

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RUSH, B. "Neurologic causes of gait abnormalities in athletic horses." In Equine Sports Medicine and Surgery, 515–31. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-7020-2671-3.50028-3.

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JOSECUNILLERAS, E. "Abnormalities of body fluids and electrolytes in athletic horses." In Equine Sports Medicine and Surgery, 898–918. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-7020-2671-3.50044-1.

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Jose-Cunilleras, Eduard. "Abnormalities of body fluids and electrolytes in athletic horses." In Equine Sports Medicine and Surgery, 881–900. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-7020-4771-8.00040-5.

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"Congenital abnormalities." In Respiratory Diseases of the Horse, 201–12. CRC Press, 2013. http://dx.doi.org/10.1201/b15203-10.

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Evarista Arellano-García, María, Olivia Torres-Bugarín, Maritza Roxana García-García, Daniel García-Flores, Yanis Toledano-Magaña, Cinthya Sofia Sanabria-Mora, Sandra Castro-Gamboa, and Juan Carlos García-Ramos. "Genomic Instability and Cyto-Genotoxic Damage in Animal Species." In Veterinary Medicine and Science. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99685.

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Genomic instability is a condition that may be associated with carcinogenesis and/or physiological disorders when genetic lesions are not repaired. Besides, wild, captive, and domesticated vertebrates are exposed to xenobiotics, leading to health disorders due to cytogenotoxicity. This chapter provides an overview of tests to assess cytogenotoxicity based on micronuclei (MNi) formation. Bone marrow micronuclei test (BmMNt), peripheral blood erythrocyte micronuclei test (PBMNt), and lymphocyte cytokinesis blocking micronuclei assay (CBMN) are discussed. The most illustrative studies of these techniques applied in different vertebrates of veterinary interest are described. The values of spontaneous basal micronuclei in captive, experimental, and farm animals (rodents, hamsters, pigs, goats, cattle, horses, fish) are summarized. In addition, a flow cytometry technique is presented to reduce the time taken to record MNi and other cellular abnormalities. Flow cytometry is helpful to analyze some indicators of genomic instability, such as cell death processes and stages (necrosis, apoptosis) and to efficiently evaluate some biomarkers of genotoxicity like MNi in BmMNt, PBMNt, and CBMN. The intention is to provide veterinary professionals with techniques to assess and interpret cytogenotoxicity biomarkers to anticipate therapeutic management in animals at risk of carcinogenesis or other degenerative diseases.
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Carr, Elizabeth A., and Omar Maher. "Neurologic causes of gait abnormalities in the athletic horse." In Equine Sports Medicine and Surgery, 503–26. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-7020-4771-8.00024-7.

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Gillberg, Christopher. "Double Syndromes:Autism Associated with Genetic, Medical and Metabolic Disorders." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0008.

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A number of conditions—genetic, metabolic, and medical syndromes—have a high rate (more than expected by chance; i.e., more than a few percent) or a very high rate (more than 20%) of several autism symptoms associated with them. In the presence of these syndromes, the diagnosis of autistic disorder or autism spectrum disorder (ASD) is very often appropriate (Gillberg and Coleman 2000). These conditions are listed in Table 2.1. Some conditions, like Down syndrome, have a much lower rate of autistic symptoms than other of the listed syndromes, but, nonetheless, present a much higher rate than expected in the general population. A number of these syndromes are also discussed elsewhere in this volume, where the full range of cognitive and behavioral complications are addressed. Angelman syndrome is characterized by jerky movements, unprovoked laughter, and varying degrees of mental retardation, mostly severe or profound (Horsler and Oliver 2006). The rate of Angelman syndrome in the general population has been estimated at about 1 in 12,000 children, with a 1:1 male-to-female ratio (Steffenburg, Gillberg, Steffenburg, & Kyllerman 1996). Angelman syndrome (formerly known as ‘happy puppet syndrome’) is usually caused by a deletion of chromosome 15q11.2–12, which is similar, but not identical, to that found in children with Prader-Willi syndrome and is maternally, rather than paternally, inherited. The deletion includes a gene for the β-3 subunit of the γ-aminobutyric acid (GABA) receptor (Saitoh et al. 1994). Sixty to 75% of patients have deletions or rearrangements in the long arm of chromosome 15, and the deletion is always on the maternal chromosome. A small proportion of cases have paternal disomy for chromosome 15. At least 20% of affected persons have normal chromosomes and no evidence of disomy. In some of these cases, recurrence in relatives may be observed. Such cases may be due to a dominant mutation of the UBE3 gene at 15q11–13 resulting in an Angelman phenotype only when transmitted by females. Differences in clinical presentation are related to the nature of the genetic defect (Lossie et al. 2001).
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