Relevant bibliographies by topics / Host defense

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Host defense'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Host defense"

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Hashimoto, Yoshiyuki. "Host-defense." TRENDS IN THE SCIENCES 1, no. 4 (1996): 58–59. http://dx.doi.org/10.5363/tits.1.4_58.

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Kelley, Kenneth J. "Host-defense peptides: Using host defenses to fight infectious diseases." Nature Biotechnology 14, no. 5 (1996): 587–90. http://dx.doi.org/10.1038/nbt0596-587.

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Erwin, Robert L. "Host-defense peptides." Nature Biotechnology 14, no. 7 (1996): 804. http://dx.doi.org/10.1038/nbt0796-804a.

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Frank, Michael M. "Annihilating host defense." Nature Medicine 7, no. 12 (2001): 1285–86. http://dx.doi.org/10.1038/nm1201-1285.

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Clayton, D. H., S. E. Bush, B. M. Goates, and K. P. Johnson. "Host defense reinforces host-parasite cospeciation." Proceedings of the National Academy of Sciences 100, no. 26 (2003): 15694–99. http://dx.doi.org/10.1073/pnas.2533751100.

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Lehrer, Robert I. "Neutrophils and Host Defense." Annals of Internal Medicine 109, no. 2 (1988): 127. http://dx.doi.org/10.7326/0003-4819-109-2-127.

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Di Cara, Francesca. "Peroxisomes in host defense." PLOS Pathogens 16, no. 7 (2020): e1008636. http://dx.doi.org/10.1371/journal.ppat.1008636.

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Ward, Pauline P., Sonia Uribe-Luna, and Orla M. Conneely. "Lactoferrin and host defense." Biochemistry and Cell Biology 80, no. 1 (2002): 95–102. http://dx.doi.org/10.1139/o01-214.

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Lactoferrin is a multifunctional member of the transferrin family of nonheme iron-binding glycoproteins. Lactoferrin is found at the mucosal surface where it functions as a prominent component of the first line of host defense against infection and inflammation. The protein is also an abundant component of the specific granules of neutrophils and can be released into the serum upon neutrophil degranulation. While the iron-binding properties were originally believed to be solely responsible for the host defense properties ascribed to lactoferrin, it is now known that other mechanisms contribute
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Harada, Ruth N., and John E. Repine. "Pulmonary Host Defense Mechanisms." Chest 87, no. 2 (1985): 247–52. http://dx.doi.org/10.1378/chest.87.2.247.

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Nicolas, Pierre, and Yvonne Rosenstein. "Multifunctional host defense peptides." FEBS Journal 276, no. 22 (2009): 6464. http://dx.doi.org/10.1111/j.1742-4658.2009.07356.x.

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Dissertations / Theses on the topic "Host defense"

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Holloway, Laurin Nicole. "Host Defense Mechanisms Against Mycobacterium Tuberculosis." Wright State University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=wright1245878489.

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Li, Yaqiong. "Gamma AApeptides as Host Defense Peptide Mimics." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6301.

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There has been increasing concern regarding the emergence of multi-drug resistant pathogens. The resistance develops when pathogens, especially bacteria, are frequently exposed to conventional antibiotics, as they are heavily used in both human and livestock. This is due to the high target specificity of conventional antibiotics, which places pathogens in high selective pressures and eventually results in drug resistant by mutations. To address this issue, global actions and cooperation are needed. At the same time, new technologies and strategies need to be developed. Host defense peptides (H
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Collins, Cathleen A. "Ubiquitin in host defense against pathogenic mycobacteria." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3359543.

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Sankala, Marko. "Role of macrophage receptor MARCO in host defense /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-686-3.

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Vyboh, Kishanda. "Mapping HIV-1's evasion of host defense." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=110455.

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The HIV-1 RNA genome encodes the main structural polyprotein Gag, Gag/Pol (structural proteins and enzymes), auxiliary (Vpr, Vpu, Vif) and regulatory proteins (Tat, Rev, Nef). The expression of the full complement of viral gene products enhances both the replication and the pathogenic potential of the virus. In our previous work we showed that HIV-1 expression prevented eIF2alpha-dependent stress granule (SG) assembly in cells. SGs are translationally silent sites of RNA triage and can be readily visualized in cells due to clustering of cytoplasmic RNA and the recruitment of multiple cytoplasm
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Prasad, Joni M. "Hemostatic Factors in Bacterial Virulence and Host Defense." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1329495133.

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Fischer, Anthony John. "Augmenting antiviral host defense in the respiratory epithelium." Diss., University of Iowa, 2009. https://ir.uiowa.edu/etd/500.

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The airway epithelium has many roles in innate immunity including detection of pathogens and transmitting danger signals to other cell types. However, its role as a primary defender against infection is not well recognized. We have investigated methods of augmenting antiviral immunity by application of agents that stimulate viral killing, either in the extracellular space or within the cytoplasm. A recently described property of airway epithelial cells is direct oxidative killing of bacteria through the coordination of Duox and lactoperoxidase enzymes. We have exploited this property by supple
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Nakashige, Toshiki G. "Metal sequestration of the host-defense protein human calprotectin." Thesis, Massachusetts Institute of Technology, 2017. http://hdl.handle.net/1721.1/109678.

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Thesis: Ph. D., Massachusetts Institute of Technology, Department of Chemistry, 2017.<br>Vita. Cataloged from PDF version of thesis.<br>Includes bibliographical references.<br>Transition metals are essential nutrients for all organisms. Microbial pathogens must acquire first-row transition metals such as manganese (Mn), iron (Fe), and zinc (Zn) to colonize the mammalian host and to cause disease. As part of the first line of defense against pathogenic infection, the host innate immune response deploys a number of metal-chelating host-defense factors that restrict the access of nutrient metals
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Brophy, Megan Brunjes. "Bioinorganic Chemistry of the Human Host-Defense Protein Calprotectin." Thesis, Massachusetts Institute of Technology, 2015. http://hdl.handle.net/1721.1/98823.

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Thesis: Ph. D. in Biological Chemistry, Massachusetts Institute of Technology, Department of Chemistry, 2015.<br>Vita. Cataloged from PDF version of thesis.<br>Includes bibliographical references.<br>The human innate immune system responds to bacterial and fungal pathogens by releasing the metal-chelating protein calprotectin (CP) at sites of infection and in the upper layers of the epidermis. CP is a Mn(II)- and Zn(ll)-binding protein. The work described in this thesis elucidates the metal-binding properties of CP, and correlates these properties with in vitro growth inhibition of bacteria an
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Cremer, Thomas John IV. "Mechanisms of Host-Defense Against Intracellular Bacterial Pathogens Through The PI3K/Akt Host Signaling Pathway." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1289571696.

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Books on the topic "Host defense"

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1961-, Devine Deirdre, and Hancock R. E. W, eds. Mammalian host defense peptides. Cambridge University Press, 2004.

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W, Ades Edwin, and Lopez Carlos 1942-, eds. Natural killer cells and host defense. Karger, 1989.

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Pourreau, Catherine N. Carp erythrodermatitis: Host defense - pathogen interaction. Landbouwuniversiteit te Wageningen, 1990.

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Hsieh, Shie-Liang, ed. Lectin in Host Defense Against Microbial Infections. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1580-4.

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Faist, Eugen, Jonathan L. Meakins, and Friedrich W. Schildberg, eds. Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8.

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Oeveren, Willem van. Alterations in host defense mechanisms during cardiopulmonary bypass. [s.n.], 1988.

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J, Hoffmann, Janeway Charles A, and Natori Shunji, eds. Phylogenetic perspectives in immunity: The insect host defense. R.G. Landes Co, 1994.

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1941-, Hoffmann J., ed. Phylogenetic perspectives in immunity: The insect host defense. R.G. Landes Co., 1994.

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Epand, Richard M., ed. Host Defense Peptides and Their Potential as Therapeutic Agents. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32949-9.

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Welsh, C. Jane, Mary W. Meagher, and Esther M. Sternberg, eds. Neural and Neuroendocrine Mechanisms in Host Defense and Autoimmunity. Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-48334-4.

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Book chapters on the topic "Host defense"

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Awasthi, L. P., Siddhartha Das, Richard F. Lee, and Sudeepta Pattanayak. "Host Defense." In Plant Pathology. CRC Press, 2024. http://dx.doi.org/10.1201/9781003326915-23.

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Toquin, Valerie, Catherine Sirven, Lutz Assmann, and Haruko Sawada. "Host Defense Inducers." In Modern Crop Protection Compounds. Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527644179.ch26.

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Stanier, Roger Y., John L. Ingraham, Mark L. Wheelis, and Page R. Painter. "Nonspecific Host Defense." In General Microbiology. Macmillan Education UK, 1986. http://dx.doi.org/10.1007/978-1-349-08754-9_29.

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Stanier, Roger Y., John L. Ingraham, Mark L. Wheelis, and Page R. Painter. "Nonspecific Host Defense." In General Microbiology. Macmillan Education UK, 1986. http://dx.doi.org/10.1007/978-1-349-15028-1_29.

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Toquin, Valerie, Christoph A. Braun, Catherine Sirven, Lutz Assmann, and Haruko Sawada. "Host Defense Inducers." In Modern Crop Protection Compounds. Wiley-VCH Verlag GmbH & Co. KGaA, 2019. http://dx.doi.org/10.1002/9783527699261.ch26.

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Cronstein, Bruce N., and Rochelle Hirschhorn. "Adenosine and Host Defense." In Adenosine and Adenosine Receptors. Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-4504-9_12.

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Piyadasa, Hadeesha, Ka-Yee Grace Choi, and Neeloffer Mookherjee. "Antibacterial Host Defense Peptides." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_100.

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Lohner, Karl, and Regina Leber. "Antifungal Host Defense Peptides." In Host Defense Peptides and Their Potential as Therapeutic Agents. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32949-9_2.

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Majcherczyk, Paul Anthony, and Philippe Moreillon. "Inflammation and Host Defense." In The Pneumococcus. ASM Press, 2014. http://dx.doi.org/10.1128/9781555816537.ch12.

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Holliman, John H. "Principles of Host Defense." In Oklahoma Notes. Springer New York, 1995. http://dx.doi.org/10.1007/978-1-4612-0813-6_3.

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Conference papers on the topic "Host defense"

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Kantrow, SP, Z. Shen, P. Zhang, and S. Nelson. "Neutrophil-Mediated Lung Permeability and Host Defense Proteins." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3712.

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Chae, June, Vijayalakshmi Nandakumar, Deanne Hebrink, Theodore Kottom, and Andrew Limper. "The role of CARD9 in Pneumocystis host defense." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa5462.

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Sanders-Reed, John N., Theodore L. Kreifels, Laura A. Mueller, and Kenneth S. Patrick. "Detection and tracking with a hemispherical optical sensor tracker (HOST)." In Defense and Security, edited by Michael K. Masten and Larry A. Stockum. SPIE, 2004. http://dx.doi.org/10.1117/12.562765.

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Rudner, X., S. Ruan, and JE Shellito. "Role of Interleukin-23 in Host Defense AgainstPneumocystis cariniiInfection." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5109.

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Cai, Shanshan, Sanjay Batra, and Samithamby Jeyaseelan. "NLRC4 Inflammasome Regulates Pulmonary Host Defense Against Klebsiella Pneumoniae." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6106.

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Zhang, Tianwei, and Ruby B. Lee. "Host-Based Dos Attacks and Defense in the Cloud." In the Hardware and Architectural Support for Security and Privacy. ACM Press, 2017. http://dx.doi.org/10.1145/3092627.3092630.

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Boggs, Nathaniel, Brenda So, and Ang Cui. "Remote attestation of host-based defense via optical channel." In Cyber Sensing 2020, edited by Peter Chin and Igor V. Ternovskiy. SPIE, 2020. http://dx.doi.org/10.1117/12.2557551.

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Murphy, John P., Vincent H. Berk, and Ian Gregorio-de Souza. "Effectively identifying user profiles in network and host metrics." In SPIE Defense, Security, and Sensing, edited by Edward M. Carapezza. SPIE, 2010. http://dx.doi.org/10.1117/12.852458.

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Havlicsek, Howard S., and Larry J. Zana. "Improving real-time communication between host and motion system in a HWIL simulation." In Defense and Security, edited by Robert Lee Murrer, Jr. SPIE, 2004. http://dx.doi.org/10.1117/12.542637.

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Barnes, Jacob O., Ronald W. Stites, Gary Cook, et al. "Doping transition metal ions into laser host crystals by hot isostatic pressing (HIP) (Conference Presentation)." In Laser Technology for Defense and Security XIII, edited by Stephen G. Post and Mark Dubinskii. SPIE, 2017. http://dx.doi.org/10.1117/12.2266541.

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Reports on the topic "Host defense"

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Morin, Shai, Gregory Walker, Linda Walling, and Asaph Aharoni. Identifying Arabidopsis thaliana Defense Genes to Phloem-feeding Insects. United States Department of Agriculture, 2013. http://dx.doi.org/10.32747/2013.7699836.bard.

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The whitefly (Bemisia tabaci) is a serious agricultural pest that afflicts a wide variety of ornamental and vegetable crop species. To enable survival on a great diversity of host plants, whiteflies must have the ability to avoid or detoxify numerous different plant defensive chemicals. Such toxins include a group of insect-deterrent molecules called glucosinolates (GSs), which also provide the pungent taste of Brassica vegetables such as radish and cabbage. In our BARD grant, we used the whitefly B. tabaci and Arabidopsis (a Brassica plant model) defense mutants and transgenic lines, to gain
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Gafni, Yedidya, Moshe Lapidot, and Vitaly Citovsky. Dual role of the TYLCV protein V2 in suppressing the host plant defense. United States Department of Agriculture, 2013. http://dx.doi.org/10.32747/2013.7597935.bard.

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TYLCV-Is is a major tomato pathogen, causing extensive crop losses in Israel and the U.S. We have identified a TYLCV-Is protein, V2, which acts as a suppressor of RNA silencing. Intriguingly, the counter-defense function of V2 may not be limited to silencing suppression. Our recent data suggest that V2 interacts with the tomato CYP1 protease. CYP1 belongs to the family of papain-like cysteine proteases which participate in programmed cell death (PCD) involved in plant defense against pathogens. Based on these data we proposed a model for dual action of V2 in suppressing the host antiviral defe
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Freeman, Stanley, and Russell J. Rodriguez. The Interaction Between Nonpathogenic Mutants of Colletotrichum and Fusarium, and the Plant Host Defense System. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7573069.bard.

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The intent of this proposal was to study the interaction between nonpathogenic mutants of Colletotrichum magna and Fusarium oxysporum, and the cucurbit host defense system. We had shown previously that a nonpathogenic endophytic mutant path- 1 of C. magna, caused no visible disease symptoms but protected watermelon seedlings from disease caused by the wildtype isolate and F. o. niveum. Objectives were: 1) Determine the microscopic, biochemical and molecular genetic interaction between "protected" (path- 1 colonized) cucurbit hosts and wildtype isolates of C. magna; 2) Isolate non-pathogenic mu
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Chejanovsky, Nor, and Suzanne M. Thiem. Isolation of Baculoviruses with Expanded Spectrum of Action against Lepidopteran Pests. United States Department of Agriculture, 2002. http://dx.doi.org/10.32747/2002.7586457.bard.

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Our long-term goal is to learn to control (expand and restrict) the host range of baculoviruses. In this project our aim was to expand the host range of the prototype baculovirus Autographa cali/arnica nuclear polyhedrosis virus (AcMNPV) towards American and Israeli pests. To achieve this objective we studied AcMNPV infection in the non-permissive hosts L. dispar and s. littoralis (Ld652Y and SL2 cells, respectively) as a model system and the major barriers to viral replication. We isolated recombinant baculoviruses with expanded infectivity towards L. dispar and S. littoralis and tested their
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Greenfield, Edward. Inhibition of Orthopaedic Implant Infections by Immunomodulatory Effects of Host Defense Peptides. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada575574.

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Greenfield, Edward. Inhibition of Orthopaedic Implant Infections by Immunomodulatory Effects of Host Defense Peptides. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada594031.

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Greenfield, Edward. Inhibition of Orthopaedic Implant Infections by Immunomodulatory Effects of Host Defense Peptides. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada555797.

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Dickman, Martin B., and Oded Yarden. Characterization of the chorismate mutase effector (SsCm1) from Sclerotinia sclerotiorum. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7600027.bard.

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Sclerotinia sclerotiorum is a filamentous fungus (mold) that causes plant disease. It has an extremely wide range of hosts (&gt;400 species) and causes considerable damage (annual multimillion dollar losses) in economically important crops. It has proven difficult to control (culturally or chemically) and host resistance to this fungus has generally been inadequate. It is believed that this fungus occurs in almost every country. Virulence of this aggressive pathogen is bolstered by a wide array of plant cell wall degrading enzymes and various compounds (secondary metabolites) produced by the f
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Prusky, Dov, Lisa Vaillancourt, and Robert Fluhr. Host Ammonification by Postharvest Pathogens and its Contribution to Fungal Colonization and Symptom Development. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7592640.bard.

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Postharvest decay of fruits and vegetables caused by pathogenic and saprophytic fungi significantly impairs the quality and quantity of fresh produce brought to market. Consequently, there is considerable interest in identifying factors that determine the susceptibility of these commodities to pathogen infection. Insidious postharvest decays remain quiescent during fruit growth and harvest, but activate during the postharvest period. A key response to the physiological changes occurring during fruit ripening is the initiation of ammonium secretion by the pathogen. Ammonium ions at the infectio
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Sordillo, Lorraine, Don Wojchowski, Gary Perdew, Arthur Saran, and Gabriel Leitner. Identification of Staphylococcus aureaus Virulence Factors Associated with Bovine Mastitis. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574340.bard.

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Staphylococcus aureus is a major cause of mastitis in dairy cattle. The organism is able to adhere to and penetrate mammary epithelium, forming deep seated abscesses that result in chronic infections. This study was based on the observation that certain genotypes of S. aureus are isolated more frequently from field cases of bovine mastitis than others and the most prevalent genotypes of S. aureus have an increased ability to resist neutrophil phagocytosis and killing compared to the rare variants. It was hypothesized that these predominating genotypes differentially express virulence factors t
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