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Journal articles on the topic 'Host Immune Respose'

Author: Grafiati

Published: 6 September 2023

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1

Hamadou, Takieddine, Imene Hamadou, Ahmed Menad, Somia Bouameur, and Souad Ameddah. "COVID-19 : histoire, pathogenèse et réponse immunitaire de l'hôte." Batna Journal of Medical Sciences (BJMS) 8, no. 1 (2021): 52–58. http://dx.doi.org/10.48087/bjmsra.2021.8110.

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By the end of 2019, pneumonia of unknown etiology occurred in Wuhan, China. Local hospitals started receiving patients presenting symptoms like dry cough, fatigue, and breathing difficulties, most of these patients were linked to the Huanan seafood market, Wuhan, China. The pandemic was afterward confirmed to be associated with a novel coronavirus. The virus spread quickly from Wuhan to other provinces of China, then from china to the rest of the world causing thereby one of the most brutal pandemics in the world’s history. SARS-CoV2 has a long incubation period ranging from 3 to 7 days and ca

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2

Haig, David M., Jackie Thomson, Colin McInnes, et al. "Orf virus immuno-modulation and the host immune response." Veterinary Immunology and Immunopathology 87, no. 3-4 (2002): 395–99. http://dx.doi.org/10.1016/s0165-2427(02)00087-9.

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3

Roilides, Emmanuel, Emmanuel Roilides, Maria Simitsopoulou, Aspasia Katragkou, and Thomas J. Walsh. "Host immune response againstScedosporiumspecies." Medical Mycology 47, no. 4 (2009): 433–40. http://dx.doi.org/10.1080/13693780902738006.

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4

Trasia, Reqgi First. "Host Immune Response to Malaria." International Islamic Medical Journal 2, no. 2 (2021): 67–71. http://dx.doi.org/10.33086/iimj.v2i2.1681.

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Malaria is still a health threat, especially for children and pregnant women in endemic areas. The World Health Organization (WHO) reports 228 million cases of malaria occur worldwide and an estimated 405,000 deaths from malaria globally in 2018. A series of malaria control efforts according to WHO recommendations have been carried out widely. However, these programs face obstacles. Therefore, the existence of an effective malaria vaccine is absolutely necessary in a series of malaria control strategies. Development of a malaria vaccine requires a basic concept regarding the host's immune resp

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5

Cheng, E. "Imaging the Host Immune Response." Science Translational Medicine 2, no. 40 (2010): 40ec113. http://dx.doi.org/10.1126/scitranslmed.3001469.

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6

Li, Chaozheng, Shaoping Weng, and Jianguo He. "WSSV–host interaction: Host response and immune evasion." Fish & Shellfish Immunology 84 (January 2019): 558–71. http://dx.doi.org/10.1016/j.fsi.2018.10.043.

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7

Bhopale, Mahendra. "Experimental Hookworm Infection in Laboratory animals: Parasite behavior, Immune response and Chemotherapeutic Studies." Biotechnology and Bioprocessing 2, no. 5 (2021): 01–03. http://dx.doi.org/10.31579/2766-2314/040.

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Hookworm disease is known to be caused allergic manifestation and severe anemic pathogenicity in man and canine hosts. Attempts have been made to establish laboratory models of Necator americaus, Ancylostoma duodenale, and Ancylostoma ceylanicum, together with canine parasite, Ancylostoma caninum. The studies include pathophysiological aspects of the host-parasite relationship, and develop to establish patent infection. Immunological approach to selecting antigen for diagnosis and protective immunity purpose using larval and adult worm antigens and their secretions became the focus with the su

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8

Jamieson, Amanda Mercedes, Meredith Crane, Yun Xu, and Kayla Lee. "Immune triage: prioritization of host immune responses." Journal of Immunology 196, no. 1_Supplement (2016): 197.20. http://dx.doi.org/10.4049/jimmunol.196.supp.197.20.

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Abstract The immune response is important in many functions, including host defense against pathogens, wound healing, development, response to cancer, and maintenance of homeostatic physiological responses. We are interested in the concept of immune triage, in that a given host must be able to deal effectively with multiple insults, and at times prioritize immune responses. It is important for the overall health status of the host that the immune system responds effectively to protect essential organs. We have developed several mouse models, focusing on the lung immune response, that allow us

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9

Elaskandrany, Miar, Rohin Patel, Mintoo Patel, George Miller, Deepak Saxena, and Anjana Saxena. "Fungi, host immune response, and tumorigenesis." American Journal of Physiology-Gastrointestinal and Liver Physiology 321, no. 2 (2021): G213—G222. http://dx.doi.org/10.1152/ajpgi.00025.2021.

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Advances in -omics analyses have tremendously enhanced our understanding of the role of the microbiome in human health and disease. Most research is focused on the bacteriome, but scientists have now realized the significance of the virome and microbial dysbiosis as well, particularly in noninfectious diseases such as cancer. In this review, we summarize the role of mycobiome in tumorigenesis, with a dismal prognosis, and attention to pancreatic ductal adenocarcinoma (PDAC). We also discuss bacterial and mycobial interactions to the host’s immune response that is prevalently responsible for re

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10

Visser, Douwe H., Regan S. Solomons, Katharina Ronacher, et al. "Host Immune Response to Tuberculous Meningitis." Clinical Infectious Diseases 60, no. 2 (2014): 177–87. http://dx.doi.org/10.1093/cid/ciu781.

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11

Solomon, Katie. "The host immune response toClostridium difficileinfection." Therapeutic Advances in Infectious Disease 1, no. 1 (2013): 19–35. http://dx.doi.org/10.1177/2049936112472173.

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12

Boyartchuk, V., and W. Dietrich. "Genetic dissection of host immune response." Genes & Immunity 3, no. 3 (2002): 119–22. http://dx.doi.org/10.1038/sj.gene.6363843.

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13

SCHLUGER, NEIL W, and WILLIAM N ROM. "The Host Immune Response to Tuberculosis." American Journal of Respiratory and Critical Care Medicine 157, no. 3 (1998): 679–91. http://dx.doi.org/10.1164/ajrccm.157.3.9708002.

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14

Moonah, Shannon N., Nona M. Jiang, and William A. Petri. "Host Immune Response to Intestinal Amebiasis." PLoS Pathogens 9, no. 8 (2013): e1003489. http://dx.doi.org/10.1371/journal.ppat.1003489.

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15

Inman, R. D. "Immunogenetic aspects of host immune response." Canadian Journal of Microbiology 34, no. 3 (1988): 319–22. http://dx.doi.org/10.1139/m88-058.

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The central role of histocompatibility leukocyte antigens (HLA) class II molecules in antigen presentation has received great attention in recent years, yet class I molecules have been defined as primarily functioning as a restriction element for cytotoxic T cell killing of virus-infected cells. Extensive clinical evidence, however, indicates that the HLA class I genes are strongly associated with nonseptic complications of enteric and genitourinary bacterial infections. Ninety percent of patients with Reiter's syndrome and reactive arthritis are positive for HLA-B27, yet the mechanism of dise

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16

Sana, Madiha, Muhammad Rashid, Imran Rashid, Haroon Akbar, Jorge E. Gomez-Marin, and Isabelle Dimier-Poisson. "Immune response against toxoplasmosis—some recent updates RH: Toxoplasma gondii immune response." International Journal of Immunopathology and Pharmacology 36 (January 2022): 039463202210784. http://dx.doi.org/10.1177/03946320221078436.

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Aims Cytokines, soluble mediators of immunity, are key factors of the innate and adaptive immune system. They are secreted from and interact with various types of immune cells to manipulate host body’s immune cell physiology for a counter-attack on the foreign body. A study was designed to explore the mechanism of Toxoplasma gondii ( T. gondii) resistance from host immune response. Methods and results The published data on aspect of host (murine and human) immune response against T. gondii was taken from Google scholar and PubMed. Most relevant literature was included in this study. The basic

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17

Korobeinikov, Andrei. "Immune response and within-host viral evolution: Immune response can accelerate evolution." Journal of Theoretical Biology 456 (November 2018): 74–83. http://dx.doi.org/10.1016/j.jtbi.2018.08.003.

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18

Surbatovic, Maja, Milic Veljovic, Jasna Jevdjic, Nada Popovic, Dragan Djordjevic, and Sonja Radakovic. "Immunoinflammatory Response in Critically Ill Patients: Severe Sepsis and/or Trauma." Mediators of Inflammation 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/362793.

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Immunoinflammatory response in critically ill patients is very complex. This review explores some of the new elements of immunoinflammatory response in severe sepsis, tumor necrosis factor-alpha in severe acute pancreatitis as a clinical example of immune response in sepsis, immune response in severe trauma with or without secondary sepsis, and genetic aspects of host immuno-inflammatory response to various insults in critically ill patients.

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19

Brunham, R. C., F. A. Plummer, and R. S. Stephens. "Bacterial antigenic variation, host immune response, and pathogen-host coevolution." Infection and Immunity 61, no. 6 (1993): 2273–76. http://dx.doi.org/10.1128/iai.61.6.2273-2276.1993.

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20

Musundi, Beryl. "An immuno-epidemiological model linking between-host and within-host dynamics of cholera." Mathematical Biosciences and Engineering 20, no. 9 (2023): 16012–30. http://dx.doi.org/10.3934/mbe.2023714.

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<abstract><p>Cholera, a severe gastrointestinal infection caused by the bacterium <italic>Vibrio cholerae</italic>, remains a major threat to public health, with a yearly estimated global burden of 2.9 million cases. Although most existing models for the disease focus on its population dynamics, the disease evolves from within-host processes to the population, making it imperative to link the multiple scales of the disease to gain better perspectives on its spread and control. In this study, we propose an immuno-epidemiological model that links the between-host and with

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21

Buldain, Idoia, Leire Martin-Souto, Aitziber Antoran, et al. "The Host Immune Response to Scedosporium/Lomentospora." Journal of Fungi 7, no. 2 (2021): 75. http://dx.doi.org/10.3390/jof7020075.

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Infections caused by the opportunistic pathogens Scedosporium/Lomentospora are on the rise. This causes problems in the clinic due to the difficulty in diagnosing and treating them. This review collates information published on immune response against these fungi, since an understanding of the mechanisms involved is of great interest in developing more effective strategies against them. Scedosporium/Lomentospora cell wall components, including peptidorhamnomannans (PRMs), α-glucans and glucosylceramides, are important immune response activators following their recognition by TLR2, TLR4 and Dec

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22

Helisa, Yasmin Nur, and Horizon Winangkoso. "Adopting Natural Host Immune Response Against Zoonosis." Journal of Education, Management and Development Studies 2, no. 1 (2022): 52–66. http://dx.doi.org/10.52631/jemds.v2i1.67.

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Zoonosis originated from the transmission of pathogens between species. Rapid mutation causes the pathogens to develop resistance to treatments. Thus, there is an urgent need for medications that could maintain efficacy when encountering new strains. This study aims to discern the possibility of overcoming threats from EIDs by recreating immune responses of natural hosts and reinforcing them in the human system. The methodology used is literature study, as the resarcher utilized data presented by similar studies. References will be taken from clinical trials and studies on related topics from

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23

Lambert, Paul H., Giuseppe del Giudice, and Georges E. Grau. "Host immune response and immunopathology in malaria." Memórias do Instituto Oswaldo Cruz 81, suppl 2 (1986): 185–90. http://dx.doi.org/10.1590/s0074-02761986000600030.

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24

Sabia, C., M. L. Montesano, and C. Napoli. "Transplantation and host immune response toToxoplasma gondii." Transplant Infectious Disease 15, no. 3 (2013): E124—E125. http://dx.doi.org/10.1111/tid.12076.

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25

Dubin, Patricia J., and Jay K. Kolls. "Pseudomonas aeruginosaand the host pulmonary immune response." Expert Review of Respiratory Medicine 1, no. 1 (2007): 121–37. http://dx.doi.org/10.1586/17476348.1.1.121.

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26

Stapleton, J. T. "Host Immune Response To Hepatitis A Virus." Journal of Infectious Diseases 171, Supplement 1 (1995): S9—S14. http://dx.doi.org/10.1093/infdis/171.supplement_1.s9.

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27

Navarro, C. "Predation risk, host immune response, and parasitism." Behavioral Ecology 15, no. 4 (2004): 629–35. http://dx.doi.org/10.1093/beheco/arh054.

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28

Christiaansen, Allison, Steven M. Varga, and Juliet V. Spencer. "Viral manipulation of the host immune response." Current Opinion in Immunology 36 (October 2015): 54–60. http://dx.doi.org/10.1016/j.coi.2015.06.012.

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29

Bhatt, Kamlesh, and Padmini Salgame. "Host Innate Immune Response to Mycobacterium tuberculosis." Journal of Clinical Immunology 27, no. 4 (2007): 347–62. http://dx.doi.org/10.1007/s10875-007-9084-0.

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30

Petry, Franz, Vera Jakobi, and Tesfaye S. Tessema. "Host immune response to Cryptosporidium parvum infection." Experimental Parasitology 126, no. 3 (2010): 304–9. http://dx.doi.org/10.1016/j.exppara.2010.05.022.

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31

Apt, A., and T. K. Kondratieva. "Tuberculosis: Pathogenesis, immune response, and host genetics." Molecular Biology 42, no. 5 (2008): 784–93. http://dx.doi.org/10.1134/s0026893308050154.

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32

Kelly, Ciarán P., and Lorraine Kyne. "The host immune response to Clostridium difficile." Journal of Medical Microbiology 60, no. 8 (2011): 1070–79. http://dx.doi.org/10.1099/jmm.0.030015-0.

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33

Rello, Jordi, and RichardR Watkins. "Monitoring the Host Immune Response in Sepsis." Journal of Translational Critical Care Medicine 4, no. 1 (2022): 18. http://dx.doi.org/10.4103/jtccm-d-22-00013.

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34

Sharma, Tarina, Anwar Alam, Aquib Ehtram, et al. "The Mycobacterium tuberculosis PE_PGRS Protein Family Acts as an Immunological Decoy to Subvert Host Immune Response." International Journal of Molecular Sciences 23, no. 1 (2022): 525. http://dx.doi.org/10.3390/ijms23010525.

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Mycobacterium tuberculosis (M.tb) is a successful pathogen that can reside within the alveolar macrophages of the host and can survive in a latent stage. The pathogen has evolved and developed multiple strategies to resist the host immune responses. M.tb escapes from host macrophage through evasion or subversion of immune effector functions. M.tb genome codes for PE/PPE/PE_PGRS proteins, which are intrinsically disordered, redundant and antigenic in nature. These proteins perform multiple functions that intensify the virulence competence of M.tb majorly by modulating immune responses, thereby

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35

Fremder, Ella, Eyal Jacob, Irena Khononov, et al. "The host response to immune checkpoint inhibitors: From mechanisms to therapeutic implications." Journal of Clinical Oncology 37, no. 15_suppl (2019): e14584-e14584. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e14584.

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e14584 Background: One of the major challenges in clinical immuno-oncology today is predicting which patients will respond to treatment. Although the use of immune checkpoint inhibitors (ICIs) has significantly improved therapeutic outcomes in a subset of patients with advanced malignancies, the majority of patients do not respond to treatment and some even hyper-progress. This response pattern raises questions regarding the possible mechanisms of resistance to ICIs. In the last decade we and others have shown that the host, in response to almost any type of anti-cancer drug, generates pro-tum

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36

Notari, Luigi, Aiping Zhao, Jennifer A. Stiltz, et al. "T1770 Enteric Nematodes Regulates Host Proteolytic Pathway on Immune Cells: Influence on Host Immune Response." Gastroenterology 138, no. 5 (2010): S—575. http://dx.doi.org/10.1016/s0016-5085(10)62648-0.

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37

Näpflin, Kathrin, and Paul Schmid-Hempel. "Immune response and gut microbial community structure in bumblebees after microbiota transplants." Proceedings of the Royal Society B: Biological Sciences 283, no. 1831 (2016): 20160312. http://dx.doi.org/10.1098/rspb.2016.0312.

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Microbial communities are a key component of host health. As the microbiota is initially ‘foreign’ to a host, the host's immune system should respond to its acquisition. Such variation in the response should relate not only to host genetic background, but also to differences in the beneficial properties of the microbiota. However, little is known about such interactions. Here, we investigate the gut microbiota of the bumblebee, Bombus terrestris , which has a protective function against the bee's natural trypanosome gut parasite, Crithidia bombi . We transplanted ‘resistant’ and ‘susceptible’

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38

Riley, Patrick. "Overcoming the problem of the lack of an immune response to cancer: A possible haptogenic approach to cancer immunotherapy." Cancer Research and Cellular Therapeutics 5, no. 1 (2021): 01–04. http://dx.doi.org/10.31579/2640-1053/072.

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Cancer cells possess a number of unusual features, most of which are explicable in the light of the theory of epigenetic carcinogenesis. This includes the remarkable failure of malignant cells to evoke an immunological response from the host which is ascribed to their deviant behaviour resulting from anomalous expression of normal gene products. Given this background a possible approach to eliciting a specific anti-cancer immune response is proposed which involves selective haptenation of an identifiable target protein.

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39

Horak, Richard D., Sean P. Leonard, and Nancy A. Moran. "Symbionts shape host innate immunity in honeybees." Proceedings of the Royal Society B: Biological Sciences 287, no. 1933 (2020): 20201184. http://dx.doi.org/10.1098/rspb.2020.1184.

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The gut microbiome plays a critical role in the health of many animals. Honeybees are no exception, as they host a core microbiome that affects their nutrition and immune function. However, the relationship between the honeybee immune system and its gut symbionts is poorly understood. Here, we explore how the beneficial symbiont Snodgrassella alvi affects honeybee immune gene expression. We show that both live and heat-killed S. alvi protect honeybees from the opportunistic pathogen Serratia marcescens and lead to the expression of host antimicrobial peptides . Honeybee immune genes respond di

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40

Abès, Riad, Sébastien Héritier, Charles-Antoine Dutertre, and Jean-Luc Teillaud. "Immune control of tumors: host immune response and antibody-based immunotherapy." Biomedicine & Pharmacotherapy 62, no. 8 (2008): 516. http://dx.doi.org/10.1016/j.biopha.2008.07.066.

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41

Moller, Anders Pape, and Johannes Erritzoe. "Parasite Virulence and Host Immune Defense: Host Immune Response is Related to Nest Reuse in Birds." Evolution 50, no. 5 (1996): 2066. http://dx.doi.org/10.2307/2410763.

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42

Møller, Anders Pape, and Johannes Erritzøe. "PARASITE VIRULENCE AND HOST IMMUNE DEFENSE: HOST IMMUNE RESPONSE IS RELATED TO NEST REUSE IN BIRDS." Evolution 50, no. 5 (1996): 2066–72. http://dx.doi.org/10.1111/j.1558-5646.1996.tb03592.x.

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43

Steain, Megan, Barry Slobedman, and Allison Abendroth. "The host immune response to varicella zoster virus." Future Virology 7, no. 12 (2012): 1205–20. http://dx.doi.org/10.2217/fvl.12.116.

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44

Hayashi, K. "Host immune response to herpes simplex virus infections." Uirusu 39, no. 1 (1989): 1–19. http://dx.doi.org/10.2222/jsv.39.1.

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45

Raveendran, Ranjith. "“Adapting to terminologies involved in host immune response”." IOSR Journal of Dental and Medical Sciences 11, no. 2 (2013): 01–05. http://dx.doi.org/10.9790/0853-1120105.

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46

Sausen, Daniel, Kirstin Reed, Maimoona Bhutta, Elisa Gallo, and Ronen Borenstein. "Evasion of the Host Immune Response by Betaherpesviruses." International Journal of Molecular Sciences 22, no. 14 (2021): 7503. http://dx.doi.org/10.3390/ijms22147503.

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The human immune system boasts a diverse array of strategies for recognizing and eradicating invading pathogens. Human betaherpesviruses, a highly prevalent subfamily of viruses, include human cytomegalovirus (HCMV), human herpesvirus (HHV) 6A, HHV-6B, and HHV-7. These viruses have evolved numerous mechanisms for evading the host response. In this review, we will highlight the complex interplay between betaherpesviruses and the human immune response, focusing on protein function. We will explore methods by which the immune system first responds to betaherpesvirus infection as well as mechanism

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47

Khadela, Avinash, Vivek P. Chavda, Humzah Postwala, Yesha Shah, Priya Mistry, and Vasso Apostolopoulos. "Epigenetics in Tuberculosis: Immunomodulation of Host Immune Response." Vaccines 10, no. 10 (2022): 1740. http://dx.doi.org/10.3390/vaccines10101740.

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Tuberculosis is a stern, difficult to treat chronic infection caused by acid-fast bacilli that tend to take a long time to be eradicated from the host’s environment. It requires the action of both innate and adaptive immune systems by the host. There are various pattern recognition receptors present on immune cells, which recognize foreign pathogens or its product and trigger the immune response. The epigenetic modification plays a crucial role in triggering the susceptibility of the host towards the pathogen and activating the host’s immune system against the invading pathogen. It alters the

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48

Wang, Rui, Yuta Hozumi, Yong-Hui Zheng, Changchuan Yin, and Guo-Wei Wei. "Host Immune Response Driving SARS-CoV-2 Evolution." Viruses 12, no. 10 (2020): 1095. http://dx.doi.org/10.3390/v12101095.

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The transmission and evolution of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) are of paramount importance in controlling and combating the coronavirus disease 2019 (COVID-19) pandemic. Currently, over 15,000 SARS-CoV-2 single mutations have been recorded, which have a great impact on the development of diagnostics, vaccines, antibody therapies, and drugs. However, little is known about SARS-CoV-2’s evolutionary characteristics and general trend. In this work, we present a comprehensive genotyping analysis of existing SARS-CoV-2 mutations. We reveal that host immune response vi

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49

Uno, Naoko, and Ted M. Ross. "Dengue virus and the host innate immune response." Emerging Microbes & Infections 7, no. 1 (2018): 1–11. http://dx.doi.org/10.1038/s41426-018-0168-0.

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50

Baillie, J. K. "Targeting the host immune response to fight infection." Science 344, no. 6186 (2014): 807–8. http://dx.doi.org/10.1126/science.1255074.

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