Relevant bibliographies by topics / Host-Pathogen interface

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Reports

Academic literature on the topic 'Host-Pathogen interface'

Author: Grafiati
Published: 25 May 2024
Last updated: 31 July 2025

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Journal articles on the topic "Host-Pathogen interface"

1

Wilson, Van G. "Sumoylation at the Host-Pathogen Interface." Biomolecules 2, no. 2 (2012): 203–27. http://dx.doi.org/10.3390/biom2020203.

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Kuehne, Sarah A. "Communication at the host-pathogen interface." Journal of Oral Microbiology 9, sup1 (2017): 1325269. http://dx.doi.org/10.1080/20002297.2017.1325269.

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Liles, W. Conrad. "The dynamic pathogen–host response interface." Drug Discovery Today: Disease Mechanisms 4, no. 4 (2007): 205–6. http://dx.doi.org/10.1016/j.ddmec.2008.02.005.

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Kaye, Paul, and Phillip Scott. "Leishmaniasis: complexity at the host–pathogen interface." Nature Reviews Microbiology 9, no. 8 (2011): 604–15. http://dx.doi.org/10.1038/nrmicro2608.

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Lonergan, Zachery R., and Eric P. Skaar. "Nutrient Zinc at the Host–Pathogen Interface." Trends in Biochemical Sciences 44, no. 12 (2019): 1041–56. http://dx.doi.org/10.1016/j.tibs.2019.06.010.

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Nosanchuk, Joshua D., and Attila Gacser. "Histoplasma capsulatum at the host–pathogen interface." Microbes and Infection 10, no. 9 (2008): 973–77. http://dx.doi.org/10.1016/j.micinf.2008.07.011.

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Stebbins, C. Erec. "Structural microbiology at the pathogen-host interface." Cellular Microbiology 7, no. 9 (2005): 1227–36. http://dx.doi.org/10.1111/j.1462-5822.2005.00564.x.

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Coombes, Brian K. "Regulatory evolution at the host–pathogen interface." Canadian Journal of Microbiology 59, no. 6 (2013): 365–67. http://dx.doi.org/10.1139/cjm-2013-0300.

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Horizontal gene transfer plays a major role in microbial evolution by innovating the bacterial genome with new genetic blueprints to adapt to previously unexploited niches. However, to benefit from these genetic acquisitions, the bacterium must integrate the expression of these new genes into existing regulatory nodes and deploy them at the right time. There is much to gain from uncovering the genetic diversity in noncoding DNA that is selective during host infection because of the beneficial effect it has on bacterial gene expression. By identifying genes that have undergone regulatory evolut
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Colonna, Marco, Bali Pulendran, and Akiko Iwasaki. "Dendritic cells at the host-pathogen interface." Nature Immunology 7, no. 2 (2006): 117–20. http://dx.doi.org/10.1038/ni0206-117.

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Kelsall, Brian L., Christine A. Biron, Opendra Sharma, and Paul M. Kaye. "Dendritic cells at the host-pathogen interface." Nature Immunology 3, no. 8 (2002): 699–702. http://dx.doi.org/10.1038/ni0802-699.

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Dissertations / Theses on the topic "Host-Pathogen interface"

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Thomas, Graham. "The host-pathogen interface : characterising putative secreted proteins of the honeybee pathogen Nosema ceranae (Microsporidia )." Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/21445.

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Microsporidia are obligate intracellular eukaryotic parasites related to fungi, possessing greatly reduced genomic and cellular components. The microsporidian Nosema ceranae threatens honeybee (Apis mellifera) populations. Nosemosis has a complex epidemiology affected by host, pathogen and environmental factors. Although a draft of the N. ceranae genome has been published, the molecular basis underpinning pathogenicity is not known. The lack of established culturing techniques and a tractable genetic system necessitates use of model systems for both host and parasite such as Saccharomyces cere
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Capewell, Samantha Jessica. "Structural and functional studies of protein targets at the host-pathogen interface." Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/9636.

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Ferric ABC Transporters. Pathogenic bacteria have evolved specialised iron acquisition systems that allow them to effectively colonise a host. One of these systems is the ferric binding protein (Fbp) complex that is a member of the ATP-Binding Cassette (ABC) superfamily of small molecule transporters. The Fbp complex is made up of three-components (FbpABC) that transports ferric iron from the periplasm to the cytoplasm of many Gram negative bacteria. FbpA binds iron in the periplasm and transports it to the FbpB transporter complex that permeates the cytoplasmic membrane. Here the iron is acti
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Caron, Alexandre. "Describing and understanding host-pathogen community interaction at the wildlife/domestic interface." Thesis, University of Pretoria, 2011. http://hdl.handle.net/2263/24464.

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In this thesis, I investigated the relationship between host and pathogen in multi-host and multi-pathogen systems at the interface between wildlife and domestic species. The term “epidemiological interaction” was central to my thesis, and was defined as “any ecological interaction between two host populations resulting in the transmission of one or more pathogen”. Epidemiological interactions are related to the processes of transmission between hosts and I investigated how these epidemiological interactions between different host populations could be investigated in a given ecosystem. I devel
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Walch, Philipp Darius Konstantin [Verfasser], and Athanasios [Akademischer Betreuer] Typas. "Dissecting the host-pathogen interface during Salmonella infection / Philipp Darius Konstantin Walch ; Betreuer: Athanasios Typas." Heidelberg : Universitätsbibliothek Heidelberg, 2021. http://d-nb.info/1234987864/34.

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Milhano, Natacha Alexandra Korni da Fonseca. "Insight into the underlying immune interaction of Rickettsia infection in the vector-pathogen-host interface." Doctoral thesis, Universidade de Évora, 2014. http://hdl.handle.net/10174/17802.

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Ixodid ticks are second only to mosquitos in their notorious role as vectors of pathogens to both animals and humans. Rickettsioses are among the most important tick-borne diseases in Europe, Mediterranean spotted fever in particular. To date, many studies have been performed in order to uncover the underlying mechanisms of this disease, in terms of the interactions among its constituents, i.e., the pathogen, Rickettsia conorii, its vector, Rhipicephalus sanguineus tick, and a vertebrate host. However, important gaps remain in this knowledge, among them studies of the relationship of its vecto
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Fulcher, Jennifer Ann. "Novel galectin-1 functions at the host-pathogen interface interactions with Nipah virus envelope glycoproteins and multifunctional roles in dendritic cell activation and development /." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1680017811&sid=14&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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Montefusco, Pereira Carlos Victor [Verfasser]. "3D air-liquid interface culture of Cystic Fibrosis bronchial epithelia, macrophages and P. aeruginosa to assess host-pathogen interaction and drug efficacy / Carlos Victor Montefusco-Pereira." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1216503478/34.

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Montefusco-Pereira, Carlos Victor [Verfasser]. "3D air-liquid interface culture of Cystic Fibrosis bronchial epithelia, macrophages and P. aeruginosa to assess host-pathogen interaction and drug efficacy / Carlos Victor Montefusco-Pereira." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1216503478/34.

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Edward, Grahame. "Development and use of an in vitro air-liquid interface model of the bovine respiratory tract and multifaceted proteomic approaches to investigate host-pathogen interactions of Mannheimia haemolytica." Thesis, University of Glasgow, 2016. http://theses.gla.ac.uk/7777/.

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In the present study, an air-liquid interface culture system was optimised for the use of bovine airway epithelial cells. This culture system has been adopted for other species, where a pseudostratified epithelium comprising multiple cell types and resembling the in vivo environment is produced. Several aspects of the epithelial culture process were optimised, including (i) the cell harvesting process and the expansion of cells in submerged culture, (ii) the substrate material used for air-liquid interface culture, the effects of collagen-coating of the substrate, and the substrate porosity, (
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Pascault, Alice. "Investigating Candida albicans epithelial infection using a high-throughput microscopy-based assay." Electronic Thesis or Diss., Sorbonne université, 2023. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2023SORUS277.pdf.

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Les infections fongiques représentent un problème émergeant de santé publique dans les pays développés. C. albicans est une levure dimorphique, commensale des muqueuses orales, génitales et intestinales d’une majorité de la population saine. Elle peut cependant conduire à des infections locales, comme les candidoses orales et vaginales voire à des infections systémiques chez les patients immunodéprimés. Si les interactions entre C. albicans et le système immunitaire de l’hôte sont désormais de mieux en mieux connues, l’invasion des épithélia qui est pourtant à l’origine des processus d’infecti
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Books on the topic "Host-Pathogen interface"

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Vogt, Peter K., and Michael J. Mahan, eds. Bacterial Infection: Close Encounters at the Host Pathogen Interface. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80451-9.

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Vogt, Peter K., and Michael J. Mahan. Bacterial Infection: Close Encounters at the Host Pathogen Interface. Springer London, Limited, 2011.

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Vogt, Peter K., and Michael J. Mahan. Bacterial Infection: Close Encounters at the Host Pathogen Interface. Springer London, Limited, 2012.

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Vogt, Peter K., and Michael J. Mahan. Bacterial Infection: Close Encounters at the Host Pathogen Interface. Island Press, 1997.

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Simões, Isaura, Daniel E. Voth, and Luís Jaime Mota, eds. Obligate Intracellular Bacteria: Evasion and Adaptative Tactics Shaping the Host-Pathogen Interface. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-753-3.

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Bacterial Infection: CLOSE ENCOUNTERS AT THE HOST PATHOGEN INTERFACE (Current Topics in Microbiology & Immunology). SPRINGER-VERLAG, 1998.

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Book chapters on the topic "Host-Pathogen interface"

1

Chang, Yung-Chi, and Victor Nizet. "Siglecs at the Host–Pathogen Interface." In Advances in Experimental Medicine and Biology. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1580-4_8.

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Burchmore, Richard. "Proteomics at the host: pathogen interface." In Farm animal proteomics 2013. Wageningen Academic Publishers, 2013. http://dx.doi.org/10.3920/978-90-8686-776-9_11.

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Passalacqua, Karla D., Marie-Eve Charbonneau, and Mary X. D. O'riordan. "Bacterial Metabolism Shapes the Host-Pathogen Interface." In Virulence Mechanisms of Bacterial Pathogens. ASM Press, 2016. http://dx.doi.org/10.1128/9781555819286.ch2.

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Guven-Maiorov, Emine, Chung-Jung Tsai, Buyong Ma, and Ruth Nussinov. "Interface-Based Structural Prediction of Novel Host-Pathogen Interactions." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8736-8_18.

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Kovalyova, Yekaterina, and Stavroula K. Hatzios. "Activity-Based Protein Profiling at the Host–Pathogen Interface." In Current Topics in Microbiology and Immunology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/82_2018_129.

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Comer, Jason E., Ellen A. Lorange, and B. Joseph Hinnebusch. "Examining the Vector–Host–Pathogen Interface With Quantitative Molecular Tools." In Bacterial Pathogenesis. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-032-8_10.

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Naseem, Muhammad, Shabana Shams, and Thomas Roitsch. "Modulating the Levels of Plant Hormone Cytokinins at the Host-Pathogen Interface." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6831-2_11.

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Crozier, Ian. "19 Understanding and Reporting the Natural History of an Infectious Disease." In Principles and Practice of Emergency Research Response. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-48408-7_28.

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AbstractAccurate characterization of the natural history of a disease is often inadequate in outbreaks of novel or re-emerging infectious diseases. Through the lens of the host–pathogen–care interface, the stages, outcomes, and determinants of the natural history are considered, with emphasis on how the natural history might suggest interventions to improve acute and convalescent outcomes. Understanding the natural history directly informs not only clinical care but also preclinical development and discovery of medical countermeasures and sets the stage for design of high-quality clinical tria
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Burchmore, Richard. "Proteomics at the host: pathogen interface." In Farm animal proteomics 2013. Brill | Wageningen Academic, 2013. http://dx.doi.org/10.3920/9789086867769_013.

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Anand, Kushi, and Varadharajan Sundaramurthy. "Mycobacterial lipids in the host–pathogen interface." In Biology of Mycobacterial Lipids. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-323-91948-7.00005-1.

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Reports on the topic "Host-Pathogen interface"

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Horwitz, Benjamin A., and Barbara Gillian Turgeon. Fungal Iron Acquisition, Oxidative Stress and Virulence in the Cochliobolus-maize Interaction. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7709885.bard.

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Our project focused on genes for high affinity iron acquisition in Cochliobolus heterostrophus, a necrotrophic pathogen of maize, and their intertwined relationship to oxidative stress status and virulence of the fungus on the host. An intriguing question was why mutants lacking the nonribosomal peptide synthetase (NRPS) gene (NPS6) responsible for synthesis of the extracellular siderophore, coprogen, are sensitive to oxidative stress. Our overall objective was to understand the mechanistic connection between iron stress and oxidative stress as related to virulence of a plant pathogen to its h
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Dickman, Martin B., and Oded Yarden. Characterization of the chorismate mutase effector (SsCm1) from Sclerotinia sclerotiorum. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7600027.bard.

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Sclerotinia sclerotiorum is a filamentous fungus (mold) that causes plant disease. It has an extremely wide range of hosts (>400 species) and causes considerable damage (annual multimillion dollar losses) in economically important crops. It has proven difficult to control (culturally or chemically) and host resistance to this fungus has generally been inadequate. It is believed that this fungus occurs in almost every country. Virulence of this aggressive pathogen is bolstered by a wide array of plant cell wall degrading enzymes and various compounds (secondary metabolites) produced by the f
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Coplin, David L., Shulamit Manulis, and Isaac Barash. roles Hrp-dependent effector proteins and hrp gene regulation as determinants of virulence and host-specificity in Erwinia stewartii and E. herbicola pvs. gypsophilae and betae. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7587216.bard.

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Gram-negative plant pathogenic bacteria employ specialized type-III secretion systems (TTSS) to deliver an arsenal of pathogenicity proteins directly into host cells. These secretion systems are encoded by hrp genes (for hypersensitive response and pathogenicity) and the effector proteins by so-called dsp or avr genes. The functions of effectors are to enable bacterial multiplication by damaging host cells and/or by blocking host defenses. We characterized essential hrp gene clusters in the Stewart's Wilt of maize pathogen, Pantoea stewartii subsp. stewartii (Pnss; formerly Erwinia stewartii)
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Coplin, David, Isaac Barash, and Shulamit Manulis. Role of Proteins Secreted by the Hrp-Pathways of Erwinia stewartii and E. herbicola pv. gypsophilae in Eliciting Water-Soaking Symptoms and Initiating Galls. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7580675.bard.

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Many bacterial pathogens of plants can inject pathogenicity proteins into host cells using a specialized type III secretion system encoded by hrpgenes. This system deliver effector proteins, into plant cells that function in both susceptible and resistant interactions. We have found that the virulence of Erwinia stewartii(Es; syn. Pantoea stewartii) and Erwinia herbicola pv. gypsophilae (Ehg, syn. Pantoea agglomerans), which cause Stewart's wilt of corn and galls on Gypsophila, respectively, depends on hrpgenes. The major objectives of this project were: To increase expression of hrpgenes in o
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Dickman, Martin B., and Oded Yarden. Genetic and chemical intervention in ROS signaling pathways affecting development and pathogenicity of Sclerotinia sclerotiorum. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7699866.bard.

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Abstract: The long-term goals of our research are to understand the regulation of sclerotial development and pathogenicity in S. sclerotior11111. The focus in this project was on the elucidation of the signaling events and environmental cues involved in the regulation of these processes, utilizing and continuously developing tools our research groups have established and/or adapted for analysis of S. sclerotiorum, Our stated objectives: To take advantage of the recent conceptual (ROS/PPs signaling) and technical (amenability of S. sclerotiorumto manipulations coupled with chemical genomics and
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