Relevant bibliographies by topics / House Dust Mites (HDM)

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Academic literature on the topic 'House Dust Mites (HDM)'

Author: Grafiati
Published: 21 February 2023

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Journal articles on the topic "House Dust Mites (HDM)"

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Smith, Claire, Thorsten Stanley, Julian Crane, and Robert Siebers. "Do Other Components of Bedding Dust Affect Sensitisation to House Dust Mites?" ISRN Allergy 2011 (January 11, 2011): 1–4. http://dx.doi.org/10.5402/2011/426941.

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Bedding dust is a mixture of many components, of which the house dust mite (HDM) allergen, Der p 1, is the most allergenic. There has been little work to investigate the effect of other bedding dust components on HDM sensitisation. The objective of the study was to determine the effect of endotoxin in bedding dust on the allergic response in HDM-sensitised individuals. Twenty-nine house dust mite-sensitised adults were skin prick and allergen patch tested against a sterile solution of their own bedding dust and against a solution containing the same concentration of Der p 1 as the bedding solution for comparison. There was no significant difference in wheal size between the diluted house dust mite solution and the bedding dust in spite of their high levels of endotoxin. Symptomatic subjects had larger, but not statistically significant, responses to commercial house dust mite solution than asymptomatic subjects. Allergen patch test responses were negative in 22/29 of subjects using either bedding dust solutions or comparable diluted house dust mite solutions. An individual's own bedding dust does not appear to contain factors that enhance skin prick test or atopy patch test responses to house dust mites.
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Anasis, Annisa Mulia, Anna Rozaliyani, and Heri Wibowo. "Density of Dermatophagoides spp. and Its Relationship with House-dust Mite Specific Serum IgE in Persistent Asthma." Molecular and Cellular Biomedical Sciences 4, no. 2 (July 1, 2020): 61. http://dx.doi.org/10.21705/mcbs.v4i2.92.

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Background: Asthma is a chronic inflammation of the bronchial tree that emerges as a response to exogenous factors, such as allergens, irritants, and infections. Some asthmatic patients had been reported having symptoms of asthma due to house-dust mites (HDM) allergen exposure. It is associated with immune responses which were increased in the form of specific Immunoglobulin E (IgE) production against HDM allergens. This case-control study aimed to determine the HDM profiles in persistent asthmatic patients, including density of mites, as well as its relationship with specific IgE anti-HDM serum levels.Materials and Methods: A total of 13 patients with persistent asthma and 12 control patients had their specific anti-HDM IgE levels examined using Immulite 2000 xpi. The house dust samples were taken and analyzed with the Fain method.Results: The results have shown that 69% of patients in the persistent asthma group and 25% of normal patients were positive for IgE anti-HDM. Dermatophagoides pteronyssinus is a predominant species with a total of 120 mites (83.9%) of 143 mites. Correlation analysis indicated a positive relationship between IgE anti-HDM levels within the serums of patients and the density of mites in the dust obtained from bedroom spaces (Spearmen Rho, R=0.35, p=0.04).Conclusion: Positive IgE anti-HDM patients in the persistent asthma group were higher (69%) than those in the clinically normal group (25%). The density of mites were dominated by D. Pteronyssinus. The bedroom-dust mites density revealed a positive correlation with serum IgE anti-HDM levels in persistent asthma patients.Keywords: asthma, density, Dermatophagoides spp.,IgE
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Dhaliwal, Amandeep Kaur, Devinder Singh, Ramanpreet Kaur Randhawa, and Atinderpal Singh. "Sensitivity in allergic asthmatic subjects towards house dust mite allergens." Systematic and Applied Acarology 26, no. 1 (January 6, 2021): 75–84. http://dx.doi.org/10.11158/saa.26.1.5.

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Asthma is a common problem that affects about 20 million peoples in India and can be often under-diagnosed or misdiagnosed. It can be allergic or non-allergic though the former type is more common and prevalent. Allergic asthma can be triggered by many allergens and house dust mites (HDM) are one of the common indoor allergens. The present study emphasizes the significance of house dust mites in allergic asthmatic subjects which is based on 115 asthmatic subjects in Punjab, India. For the quantification and the estimation of total serum Immunoglobulin E and HDM specific IgE, a mixture of 14 allergens and a mixture of two mite allergens viz. Dermatophagoides pteronyssinus and D. farinae were used respectively. Total and specific IgE levels were detected on ImmunoCAP Phadia 100. A statistically significant correlation between total and HDM specific IgE levels of 115 asthmatic subjects was found as compared to control group of 30 non-allergic individuals. The specific IgE levels of 54.78% subjects against the allergen of two mite species were found to be positive. Dust samples were taken from various localities of the houses to identify the diversity of house dust mites which were responsible for allergic asthma. Five common house dust mite species viz. D. pteronyssinus Trouessart, D. farinae Hughes, D. microceras Griffiths and Cunnington, D. aureliani Fain and Euroglyphus maynei Cooreman were identified from the dust. The present study observed that total IgE levels were higher with higher specific IgE levels against the mixture of two mite allergens viz. D. pteronyssinus and D. farinae in the blood serum. D. pteronyssinus was the most abundant and prevalent mite species followed by D. farinae. Therefore, present study concluded that HDM specific IgE levels against the mite allergen of D. pteronyssinus and D. farinae in the serum of allergic asthmatic subjects were found to be higher because of the higher prevalence of these two mites (D. pteronyssinus i.e. 69.80% and D. farinae i.e. 20.72%) in the house of allergic asthmatic subjects as compared to other identified mites.
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Yolazenia, Yolazenia, Harianto Harianto, and Iqbal Teguh Riady. "Gambaran Kepadatan Tungau Debu Rumah Sebagai Pencetus Rinitis Alergi pada Anak Panti Asuhan Pekanbaru." Jurnal Ilmu Kedokteran 13, no. 1 (March 1, 2019): 23. http://dx.doi.org/10.26891/jik.v13i1.2019.23-27.

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Allergic rhinitis should be considered as a serious condition because it can affect the quality of life of patients. In manycases, the most common allergen is house dust mites (HDM). The purpose of this study was to describe the density ofhouse dust mites as triggers of allergic rhinitis in Pekanbaru orphanage children. This is a descriptive study with across sectional approach. Data obtained from interviews using the modified International Study of Asthma and Allergiesof Childhood (ISAAC) questionnaire to know the allergic rhinitis status and examination of house dust samples toidentify HDM in the parasitology laboratory. Sampling was carried out in 4 orphanages Pekanbaru. Allergic rhinitissymptoms were experienced by 81.2% of children in the orphanages and HDM was found with the mean density 9.11/g of dust. HDM was found in all four orphanages and was found to have a high incidence of allergic rhinitis.
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Jacquet, Alain. "Innate Immune Responses in House Dust Mite Allergy." ISRN Allergy 2013 (February 28, 2013): 1–18. http://dx.doi.org/10.1155/2013/735031.

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Sensitizations to house dust mites (HDM) trigger strong exacerbated allergen-induced inflammation of the skin and airways mucosa from atopic subjects resulting in atopic dermatitis as well as allergic rhinitis and asthma. Initially, the Th2-biased HDM allergic response was considered to be mediated only by allergen B- and T-cell epitopes to promote allergen-specific IgE production as well as IL-4, IL-5, and IL-13 to recruit inflammatory cells. But this general molecular model of HDM allergenicity must be revisited as a growing literature suggests that stimulations of innate immune activation pathways by HDM allergens offer new answers to the following question: what makes an HDM allergen an allergen? Indeed, HDM is a carrier not only for allergenic proteins but also microbial adjuvant compounds, both of which are able to stimulate innate signaling pathways leading to allergy. This paper will describe the multiple ways used by HDM allergens together with microbial compounds to control the initiation of the allergic response through engagement of innate immunity.
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Yu, Jing-Miao, Qing-Hua Luo, Jin-Lu Sun, Cun-Lian Shi, Jia Yin, Yu-Ling Zhou, Rui Tang, Hui Zhang, Zhang Yu, and Meng Chen. "Diversity of House Dust Mite Species in Xishuangbanna Dai, a Tropical Rainforest Region in Southwest China." BioMed Research International 2015 (2015): 1–6. http://dx.doi.org/10.1155/2015/421716.

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Purpose. To survey the species diversity of home dust mites (HDM) in Xishuangbanna, a tropical rainforest region in Southwest China.Methods. From August 2010 to January 2011, mite-allergic patients and healthy controls were invited to participate. Dust samples from the patients’ homes were collected, and mites in the samples were isolated. Permanent slides were prepared for morphologically based species determination.Results. In total, 6316 mite specimens of morphologically identifiable species were found in 233 dust samples taken from 41 homes. The result shows that the mite family of Pyroglyphidae occupied the highest percentage of the total amount of mites collected, followed by Cheyletidae family. The most common adult Pyroglyphidae mites wereDermatophagoides(D.)farinae,D. pteronyssinus, andD. siboney. The most common mites found from other families wereBlomia tropicalis,Tyrophagus putrescentiae, andAleuroglyphus ovatus. Four main allergenic dust mite speciesD. farinae,D. pteronyssinus,D. siboney, andBlomia tropicaliswere found to be coinhabiting in 6/41 homes.Conclusion. The HDM population in homes in Xishuangbanna, a tropical rainforest region in Southwest China, has its own characteristics. It has rich dust mite species and the dust mite densities do not show significant variation across seasons.
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Trusova, Olga Valerevna, Andrei Viacheslavovich Kamaev, Natalia Leonidovna Liashenko, and Irina Vadimovna Makarova. "Clinical and anamnestic characteristics and results of allergological testing in children with bronchial asthma and allergic rhinitis in case of allergy to house dust mites." Allergology and Immunology in Pediatrics, no. 3 (September 30, 2021): 40–48. http://dx.doi.org/10.53529/2500-1175-2021-3-40-48.

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House dust mites (HDM) are a common cause of allergies, however, it is not always easy to suspect a mite allergy from a patient's history. Objective: to determine clinical characteristics of patients, tactics for demonstrating the role of HDM sensitization in pediatric patients with allergic rhinitis (AR) and bronchial asthma (BA) with domestic sensitization. Materials and methods. The study included 420 patients, 256 boys, 164 girls, aged 5 to 17,9 years (mean 8,44 ± 3,06 years), with AR (100%) and AR in combination with BA (41,9%). The prick test on the forearm skin with water-soluble extracts was carried out and evaluated according to the standard method. The test was considered positive with a maximum wheal diameter of ≥3 mm 15 minutes after applying the prick. Results. Among patients with HDM sensitization (n=268), sensitization to both mites was detected in 218 cases (81,3%), and in 139 cases (51,9%) polysensitization to HDM allergens, house dust and epidermal allergens (cat, dog) was found. Patients noted persistent symptoms of nasal congestion, and rhinorrhea; the provoking factor was not determined by history in 46% of cases. In 69,8% of cases (97 children), it was impossible to isolate the leading allergen according to the history, since patients had contact with cats, dogs, dust, and polysensitization according to the prick tests. Nasal provocation test (NPT) with HDM allergens was performed in 92 children. Positive NPT was obtained in 83 cases (90,2%), negative in 9 cases (9,8%). Conclusions. With HDM sensitization, often (46%) the provoking factor is not determined by history. Monotonous symptoms are characteristic with the absence of obvious relationships with allergens, the location and activities of the child. In terms of frequency, the leading variant is sensitization to both mites D.pteronyssinus, D.farinae (81,3%), and polysensitization to HDM, house dust and cat and dog allergens (51,9%). NPT allows in 90% of cases to show the causal role of HDM allergens in polysensitized patients with insufficiently convincing anamnesis data.
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Eguiluz-Gracia, Ibon, Francisca Palomares, Maria Salas, Almudena Testera-Montes, Adriana Ariza, Ignacio Davila, Joan Bartra, Cristobalina Mayorga, Maria Jose Torres, and Carmen Rondon. "Precision Medicine in House Dust Mite-Driven Allergic Asthma." Journal of Clinical Medicine 9, no. 12 (November 26, 2020): 3827. http://dx.doi.org/10.3390/jcm9123827.

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House dust mites (HDMs) are the allergenic sources most frequently involved in airway allergy. Nevertheless, not every sensitized patient develops respiratory symptoms upon exposure to HDM, and there is a clinical need to differentiate allergic asthmatics (AAs) from atopic non-allergic asthmatics with HDM sensitization. This differentiation sometimes requires in vivo provocations like the bronchial allergen challenge (BAC). Interestingly, recent data demonstrate that non-atopic patients with asthma can also develop positive BAC results. This novel phenotype has been termed local allergic asthma (LAA). The interest in identifying the allergic triggers of asthma resides in the possibility of administering allergen immunotherapy (AIT). AIT is a disease-modifying intervention, the clinical benefit of which persists after therapy discontinuation. Recently, new modalities of sublingual tablets of HDM immunotherapy registered as pharmaceutical products (HDM-SLIT tablets) have become commercially available. HDM-SLIT tablets have demonstrated a robust effect over critical asthma parameters (dose of inhaled corticosteroids, exacerbations, and safety), thus being recommended by international guidelines for patients with HDM-driven AA. In this review, we will summarize the current knowledge on the phenotype and endotype of HDM-driven AA, and LAA, address the difficulties for BAC implementation in the clinic, and discuss the effects of AIT in AA and LAA.
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Reza Anindita, Salma Lailatul Amwia, Maulin Inggraini, and Dede Dwi Nathalia. "Density of House Dust Mites (HDM) Dermathopagoides sp. In Jatimulya Village South Tambun District Bekasi City." International Islamic Medical Journal 3, no. 2 (June 30, 2022): 73–79. http://dx.doi.org/10.33086/iimj.v3i2.3015.

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Background: House Dust Mites (HDM) are arthropods that trigger allergies such as asthma and rhinitis. The percentage of these animals as the cause of asthma according to WHO data (2013) is around 50% -80%. Objective: The purpose of this study was to obtain new information about the density of HDM in the city of Bekasi as well as to complement the existing data so that it can be used as a reference in formulating an allergy prevention program caused by HDM. Methods:This study was designedwith a quasi-experimental study research method using a comparative pre-test post-test non-equivalent control group design.The sample in this study was divided into two groups, namely one control group and one treatment group from the sample selected by purposive sampling. The population studied were students of the Hidayatullah Islamic Boarding School inSurabaya. Result: This type of research is quantitative descriptive with a cross-sectional research design. The sample in this study were 9 houses of residents of the village of Jati Bulak, RT 001/ RW 003, Jatimulya Village, Tambun Selatan District. The location points for dust sampling for each house are mattresses, carpets and floors. The working procedure of this research includes the pre-analytic stage in the form of preparation of tools and materials, the analytical stage in the form of HDM examination with the sedimentation method, the post-analytic stage in the form of confirmation of HDM identification. Conclusion:From research result that has been done, it can be concluded that the HDM figures in 9 houses of Jati Bulak villagers RT 001/ RW 003 Jatimulya Village, Tambun Selatan District are in the low category.
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Gür Çetinkaya, Pınar, Elif Soyak Aytekin, Ayşegül Akarsu, Ümit Murat Şahiner, Bülent Enis Şekerel, and Özge Soyer. "Parental Perspectives on the Implementation of House Dust Mite Avoidance Measures for Children with House Dust Mite Sensitization." Asthma Allergy Immunology 19, no. 3 (December 22, 2021): 125–35. http://dx.doi.org/10.21911/aai.581.

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ABSTRACTObjective: The avoidance of house dust mite (HDM) is crucial in the management of HDM allergies. We aimed to demonstrate the implementation and perspective of the parents whose children had HDM allergy/sensitization to HDM avoidance measures. Materials and Methods: Parents of the patients with HDM sensitization were interviewed via telephone questionnaires.Results: One hundred and three patients with asthma (73.8%), allergic rhinitis (AR) (77.7%) and/or atopic dermatitis (AD) (29.1%) aged four to 18 years were included in the study. Seventy-one patients had multiple allergic diseases (68.9%). Of the parents, 39.8% fully adhered to HDM avoidance measures, and their education status was as follows: 41.5% illiterate/elementary/middle school, 31.7% high school, and 26.8% associate’s degree/university. In addition, 32.2% of the mothers who were partially adherent (n=62) were illiterate or had graduated from elementary/middle school, 33.9% had graduated from high school, and 33.9% had an associate’s degree or had graduated from university. Forty-one (39.8%) mothers were working, and most of them (61%) had graduated from university or had an associate’s degree. Nearly half of the mothers who were partially adherent to HDM measures were working (n=32). In the multivariate analysis, the risk factors for partial adherence to measures was to be a working mother [OR:4.072, 95%CI: 1.350-12.882, p=0.013] and to have the belief that the measures were useless [OR:4.886, 95%CI: 1.499-15.923, p=0.008]. However, no relationship was shown between adherence to the measures and the severity of AR or AD, asthma control status and having multiple allergic diseases.Conclusion: Full adherence to HDM avoidance measures was considerably dependent on the mothers’ working status and belief in the ineffectiveness of the measures whereas there was no relationship to the severity of allergic diseases. This study also revealed how the education status of the mothers affected the adherence to allergen avoidance measures in real life.Keywords: Children, Dermatophagoides pteronyssinus,Dermatophagoides farinae, house dust mites, avoidance measures, parents, pediatrics, questionnaire
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Dissertations / Theses on the topic "House Dust Mites (HDM)"

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Madouri, Fahima. "Asthme allergique induit par un allergène d’acarien, House Dust Mite (HDM) : rôles de la caspase-1 et de la protéine kinase C thêta (PKC-θ)." Thesis, Orléans, 2014. http://www.theses.fr/2014ORLE2055/document.

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Des études menées au laboratoire avaient démontré un rôle critique de l’inflammasome NLRP3 dans l’asthme allergique en réponse à l’ovalbumine en absence d’adjuvant. Mes travaux de thèse ont porté sur le rôle de NLRP3 et de la caspase-1 dans un modèle murin d’inflammation pulmonaire induite par l’allergène d’acarien HDM. Nous avons montré un rôle régulateur de la caspase-1 dépendant de l’inflammasome NLRP3 et la molécule adaptatrice ASC mais pas de l’inflammasome NLRC4. Cette régulation de la réponse allergique se caractérise par une augmentation de l’infiltration des éosinophiles, de l’hyperréactivité bronchique et de la production des cytokines de type Th2 telles que l’IL-4, l’IL-5, l’IL-13 et l’IL-33 dans les poumons. Nous avons montré que les mécanismes responsables de cette régulation sont associés à l’IL-33 produite par les macrophages et que la neutralisation de l’IL-33 par administration locale de la protéine de fusion au récepteur ST2 (muST2-Fc) atténue les caractéristiques de l’asthme allergique. Ces résultats suggèrent que l’activation de la caspase-1 réduit la production d’IL-33 in vivo et régule ainsi la réponse l’inflammation pulmonaire induite par HDM et la réponse Th2. D’autre part, nous nous sommes intéressés au rôle de la Protéine Kinase C thêta (PKC-θ) dans ce même modèle d’inflammation pulmonaire. Nous avons démontré que PKC-θ joue non seulement un rôle protecteur dans l’asthme allergique mais également un rôle critique pour la prolifération et l’activation des cellules lymphoïdes innées (ILC2). D’autre part, l’inhibition de PKC-θ in vivo par administration orale de son inhibiteur spécifique C20 (BIX02656) atténue l’inflammation pulmonaire et la production d’IL-5 et d’IL-13. Nous suggérons que PKC-θ est impliquée dans la différenciation des Th2 et des ILC2 via un mécanisme dépendant des facteurs de transcription IRF4 et NFAT-1. Au total, mes travaux de thèse mettent en exergue deux molécules IL-33 et PKC-θ qui pourraient constituer des cibles thérapeutiques potentielles
Studies from our laboratory have shown a critical role of NLRP3 inflammasome in response to ovalbumin allergen. In the present study we investigate the role of NLRP3 and caspase-1 in a mouse model of pulmonary inflammation induced by HDM. We have shown a regulatory role of caspase-1 dependant of the NLRP3 inflammasome and the adaptator molecule ASC but not NLRC4. The regulation of the allergic response is characterized by an increase of eosinophilia, bronchial hyperreactivity and Th2 cytokines production (IL-4, IL-5, IL-13 and IL-33) in lungs. We have shown that mechanisms responsible of this regulation are associated with IL-33 production by macrophages and that neutralization of IL-33 by local administration of a fusion protein of the ST2 receptor (muST2-Fc) reduce characteristics of asthma. These results suggest that caspase-1 activation reduce IL-33 production in vivo regulating lung inflammation and Th2 response induced by HDM. Moreover, we investigate the role of the Protein Kinase C theta (PKC-θ) in allergic airway inflammation. We have demonstrated that PKC-θ plays a protective role in allergic asthma but is critical for the activation and proliferation of innate lymphoid cells (ILC2). In addition, in vivo inhibition by oral administration of PKC-θ specific inhibitor C20 (BIX02656) reduces pulmonary inflammation with IL-5 and IL-13 production. We suggest that PKC-θ is implicated in Th2 and ILC2 differenciation by a mechanism dependant on transcription factors IRF4 and NFAT-1. Finally, my thesis projects describe IL-33 and PKC-θ as potential therapeutic targets for allergic lung inflammation
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Ullah, MD Ashik. "Novel mechanisms of airway inflammation in mouse models of allergen and virus-induced asthma." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/12830.

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Asthma is a heterogeneous disorder encompassing distinct clinical phenotypes thought to be mediated by distinct mechanisms. The Receptor for Advanced Glycation End products (RAGE) is a pattern-recognition receptor capable of ‘sensing’ exogenous and endogenous molecules; and there is evidence that ligand-RAGE axis is activated in asthma. We investigated the role of RAGE and its ligand high-mobility group box-1 (HMGB1) in the inception and progression of allergen and virus-induced asthma using mouse models. We demonstrate that RAGE is a critical mediator of allergic airway sensitization induced by house dust mite (HDM) and cockroach (CR) allergens. Our studies suggest that RAGE is engaged secondary to the release of HMGB1 by airway epithelial cells; and that HMGB1-RAGE signalling drives type 2 immunity and airway inflammation. RAGE is also required for anti-viral immunity to pneumonia virus of mice (PVM) (equivalent to human respiratory syncytial virus). PVM infection in RAGE-deficient mice led to increased HMGB1 expression in the airways; this contributed to the development of an asthma-like pathology characterised by airway smooth muscle remodelling, airways hyperresponsiveness and the absence of granulocytic inflammation, representing a pauci-granulocytic phenotype of human asthma. In separate studies we investigated whether an anti-IL-6 receptor (IL-6R) antibody protects against allergic airway inflammation. Intriguingly, anti-IL-6R protected against CR-induced airway inflammation but exacerbated the airway inflammatory response to HDM. This differential response was related to differential activation of IL-6 signalling mechanisms in response to CR and HDM. The studies herein expose novel molecular mechanisms that may explain the development of specific phenotypes of human asthma in response to viral or allergic triggers. Selective targeting of these pathways in appropriate patient sub-groups may lead to better outcomes in asthma management.
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Glass, Emmett V. "Environmental control of house dust mites : strategies for limiting exposure to house dust mites and their allergens /." The Ohio State University, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486461246818192.

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Hill, Michael Richard. "Studies on house dust mites and atopic disease." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306515.

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Folisi, Caterina. "Oxidative stress and anti-oxidant response in allergen, virus, and corticosteroids withdrawal-induced asthma exacerbation." Doctoral thesis, Università di Catania, 2015. http://hdl.handle.net/10761/3871.

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Si stima che oltre 300 milioni persone di tutte le età e razze, soffrono di asma.1 L'onere di questa malattia per i governi, le famiglie, e i pazienti è in aumento a livello globale. L'asma è una condizione eterogenea e complessa causata da una combinazione di fattori genetici e ambientali. L'asma è caratterizzata da tosse ricorrente, respiro sibilante, senso di costrizione toracica, e responsività ai broncodilatatori. Iper-reattività bronchiale, infiammazione cronica, alterazioni strutturali delle vie aeree, e iper-secrezione ghiandolare sono altre caratteristiche dell asma. Lo stress ossidativo svolge un ruolo centrale nella patogenesi dell asma.2 Esso si verifica quando la produzione di specie ossidanti supera la capacità dei sistemi biologici di detossificazione o di riparo. La presenza di un alterato status ossidante è ben stabilita in pazienti asmatici. Tuttavia, non è stata ancora eseguita alcuna analisi sistematica del danno ossidativo e sulle risposte cellulari anti-ossidanti negli asmatici. Questa tesi è stata incentrata sulla valutazione dello stress ossidativo e risposta anti-ossidante nell'asma e durante la sua riacutizzazione. La presente tesi ha avuto lo scopo di valutare le conseguenze ossidative che le riacutizzazioni hanno sulle proteine cellulari e di individuare i principali meccanismi anti-ossidanti coinvolti nella risposta cito-protettiva. La tesi ha studiato la relazione tra stress ossidativo, stato anti-ossidante, e sintomi da riacutizzazione asmatica. Una valutazione bio-chimica completa dello stato ossidativo e delle risposte anti-ossidanti è necessaria per identificare la natura e la portata di eventuali deficit anti-ossidanti associati con l'asma e la sua riacutizzazione. Una piena comprensione dello stato ossido-riduttivo nelle riacutizzazioni dell'asma potrebbe sostenere lo sviluppo di nuovi interventi terapeutici sicuri ed efficaci. La popolazione che ha partecipato negli studi inclusi in questa tesi è composta da 4 gruppi di asmatici. Il primo gruppo di nove asmatici è stato esposto al Rhinovirus-16. Il secondo gruppo comprendeva venti asmatici atopici esposti all acaro della polvere. Il terzo trentasette individui sani impiegati in laboratori ed esposti ad allergeni da roditori per un periodo di due anni; alcuni di loro sono diventati allergici. Il quarto gruppo comprendeva ventitré asmatici in trattamento con corticosteroidi la cui sospensione è stata eseguita allo scopo di studiare la riacutizzazione asmatica indotta dalla sospensione di questi farmaci. Sono stati condotti diversi esperimenti in vivo, ex vivo e in vitro per chiarire la relazione tra stato ossidativo e la riacutizzazione asmatica. L ossidazione delle proteine cellulari è stata valutata come stabile indice biologico di stress ossidativo e il livello di espressione è stato misurato per diverse proteine anti-ossidanti nel plasma e nell espettorato indotto. È stata anche determinata la produzione di mediatori pro-infiammatori. I pazienti durante la riacutizzazione dell'asma, come previsto, mostravano livelli più alti di stress ossidativo. L entità del danno ossidativo risultava associata alla severità della sintomatologia. È stato interessante riscontrare che i pazienti durante una riacutizzazione asmatica erano anche più suscettibili a danni ossidativi delle proteine cellulari; questo è stato associato ad una riduzione della capacità anti-ossidante cellulare, ridotta traslocazione nucleare del principale fattore di trascrizione anti-ossidante, e maggiore produzione di mediatori pro-infiammatori. Inoltre, i livelli basali di stress ossidativo sono stati in grado di predire quali pazienti erano più inclini a sviluppare sintomi di riacutizzazione. Questi risultati suggeriscono che il rafforzamento dei meccanismi anti-ossidanti locali e sistemici in asmatici può attenuare l'infiammazione delle vie aeree e il suo aggravamento durante le fasi di riacutizzazione.
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Lawson, Alan. "The domestic hygro-thermal environment, house dust mites and asthma." Thesis, University of Strathclyde, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401343.

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Lassiter, Mark Timothy. "A Survey of House Dust Mites in the Williamsburg Area." W&M ScholarWorks, 1985. https://scholarworks.wm.edu/etd/1539625296.

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Hay, David B. "Ecology of the house dust mite Dermatophagoides pteronyssinus (Trouessart)." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302975.

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Rockwood, Jananie. "House Dust Mite Induced Gene Expression and Cytokine Secretion by Human Dermal Fibroblasts." Wright State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=wright1347976529.

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Shaw, Stephen Charles. "The immune response in canine atopy : hypersensitivity to house dust mites (Dermatophagoides spp.)." Thesis, Open University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340709.

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Books on the topic "House Dust Mites (HDM)"

1

Dust mites. Collingwood, VIC: CSIRO Pub., 2009.

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Solarz, Krzysztof. House dust mites and storage mites (Acari, Oribatida, Astigmatina): Identification keys. Kraków: Institute of Systematics and Evolution of Animals, Polish Academy of Sciences, 2012.

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Charles, Humfrey, Shuker Linda, and Harrison Paul 1953-, eds. IEH assessment on indoor air quality in the home: Nitrogen dioxide, formaldehyde, volatile organic compounds, house dust mites, fung, and bacteria. Leicester: Institue for Environment and Health, 1996.

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Housing and asthma. New York, NY: Spon Press, 2005.

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Mendelson, Cheryl. Home Comforts: The Art & Science of Keeping House. New York, USA: Scribner, 2005.

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Mendelson, Cheryl. Home Comforts: The Art and Science of Keeping House. 4th ed. New York, USA: Scribner, 1999.

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House Dust Mites. Elliot Right Way Books, 1995.

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Colloff, Matthew J. Dust Mites. CSIRO Publishing, 2009. http://dx.doi.org/10.1071/9780643100497.

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Abstract:
Dust mites are present in almost every home – in our beds, clothing and carpets. Conservatively, at least 100 million people are affected by house dust mite allergy worldwide, manifesting itself as asthma, rhinitis or atopic dermatitis. Despite the growing recognition of this major public health problem, there is still no simple, effective, generally applicable strategy for dust mite control. Dust Mites incorporates for the first time in a single volume the topics of systematics and identification, physiology, ecology, allergen biochemistry and molecular biology, epidemiology, mite control and allergen avoidance. It explains key biological and ecological concepts for non-specialist readers, discusses ecological research methods and includes identification keys to dust mite species and life-cycle stage. It also explores how characteristics of population growth, water balance and physiology of dust mites have contributed to their importance as allergenic organisms. Many chapters contain new data, or new analyses of existing data, including global distribution maps of the most important species. Importantly, the book emphasises that studies of the biology and ecology of house dust mites should be regarded within the context of allergic disease rather than as ends in themselves, and that approaches to mite control in clinical management are subject to the same series of ecological rules as any other major problem in pest management. This comprehensive reference is essential reading for anyone involved or interested in house dust mite research and management.
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Boer, Rob De. House Dust Mites: Natural History, Control and Research Techniques. Elsevier Science & Technology Books, 2023.

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Boer, Rob De. House Dust Mites: Natural History, Control and Research Techniques. Elsevier Science & Technology, 2023.

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Book chapters on the topic "House Dust Mites (HDM)"

1

Colloff, Matthew J. "Methods in house dust mite ecology and biology." In Dust Mites, 255–71. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2224-0_6.

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Wahn, U., S. Lau-Schadendorf, B. Ehnert, and A. Weber. "House Dust Mites in German Homes." In New Trends in Allergy III, 447–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46717-2_59.

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Saha, Goutam Kumar. "House Dust Mites—A Precarious Indoor Allergen." In Dust Allergy: Cause & Concern, 33–46. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-1825-1_6.

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Hart, B. J., and A. E. Douglas. "The relationship between house-dust mites and fungi." In The Acari, 319–24. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3102-5_24.

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Mansi, A., L. Frusteri, M. Maroli, A. Salerno, A. M. Marcelloni, G. Muzi, M. P. Accattoli, and G. Abbritti. "House-dust mite infestation in temporary prefabricated homes in Central Italy." In Acarid Phylogeny and Evolution: Adaptation in Mites and Ticks, 425–30. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-017-0611-7_43.

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Lau-Schadendorf, S., A. F. Rusche, B. Ehnert, and U. Wahn. "House Dust Mites and Protective Measures — A Study on Solidified Benzyl Benzoate." In New Trends in Allergy III, 454–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46717-2_60.

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Dubakiene, R., and A. Dautartiene. "House Dust Mites in Stuffed Toys as a Cause of Allergy in Lithuania." In New Trends in Allergy V, 9–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-55994-5_2.

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De Boer, R. "An indirect effect of cleaning on house-dust mites (Dermatophagoides spp.) in carpets." In The Acari, 517–18. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3102-5_57.

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HASHIMOTO, Tomoyuki. "Current Status of House Dust Mites in Japan and Prospects for Control Agents." In ACS Symposium Series, 107–20. Washington DC: American Chemical Society, 2009. http://dx.doi.org/10.1021/bk-2009-1014.ch008.

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Shen, H. D., K. Y. Chua, K. H. Hsieh, and W. R. Thomas. "Molecular Cloning and Immunological Characterization of The Group 7 Allergens of House Dust Mites." In Advances in Experimental Medicine and Biology, 241–42. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-5855-2_33.

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Conference papers on the topic "House Dust Mites (HDM)"

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Tang, Winnie Wan-yee, Yan Shang, and Wayne Mitzner. "Investigation Of Epigenetic Changes In House Dust Mite (HDM)-Induced Asthma." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6376.

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Sato, Makiko, Haruka Aoki-Saito, Hayato Fukuda, Hiroyuki Ikeda, Yasuhiko Koga, Masakiyo Yatomi, Hiroaki Tsurumaki, et al. "Resolvin E3 attenuates House Dust Mite (HDM) -induced asthmatic inflammation in a murine model." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa1111.

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Nieto-Fontarigo, Juan-José, Samuel Cerps, Sangeetha Ramu, Hamid Akbarshahi, Asger Sverrild, Celeste Porsbjerg, and Uller Lena. "Local lung house dust mite (HDM) challenges induce cell necrosis in mouse experimental asthma." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.321.

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Conejero Hall, Laura, Sofía Chayeb Khouili, Sarai Martínez Cano, Helena Izquierdo Fernández, Paola Brandi, and David Sancho Madrid. "LSC Abstract – Batf3-dependent dendritic cells control exacerbated allergic airways inflammation to house dust mite (HDM)." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pp220.

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Lund, Joni, Sabine Bartel, Juryung Chung, Zane Orinska, Skadi Kull, Uta Jappe, John Baines, and Susanne Krauss-Etschmann. "House dust mite (HDM)-induced “asthma” phenotypes differ in 4 mouse strains - link to the microbiome?" In Abstracts from the 17th ERS Lung Science Conference: ‘Mechanisms of Acute Exacerbation of Respiratory Disease’. European Respiratory Society, 2019. http://dx.doi.org/10.1183/23120541.lungscienceconference-2019.pp106.

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Gerthoffer, W. T., S. C. Ramelli, and T. Zhou. "Circular RNA Expression in Mild/Moderate Versus Severe House Dust Mite (HDM) Mouse Models of Asthma." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2855.

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Mateus, E. F., C. Ujaldon, R. Osuna, V. Plaza, and D. Ramos. "Kinetics of Lung Eosinophil and Neutrophil Distribution in a House Dust Mite (HDM)-Induced Murine Asthma Model." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7633.

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Campos Dos Santos, P., S. Dortas Junior, J. Elabras Filho, F. Da Cruz, S. Valle, and P. Ferraiolo. "Association between Staphylococcal Enterotoxin (SE)-specific IgE and specific IgE for house dust mites (HDM) in Brazilian patients with Chronic Rhinosinusitis with Nasal Polyps (CRSwNP)." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.1345.

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Krammer, Susanne, Nina Li, Zuqin Yang, Julia Koelle, Carol Immanuel Geppert, Ralf J. Rieker, Gerard Graham, and Susetta Finotto. "Late Breaking Abstract - The Role of the chemokine receptor CCR3 in House Dust Mite (HDM) induced experimental asthma." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.306.

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Lund, Joni, Sabine Bartel, Juryung Chung, Zane Orinska, Skadi Kull, Uta Jappe, John Baines, and Susanne Krauss-Etschmann. "LSC - 2019 - House dust mite (HDM)-induced “asthma” phenotypes differ in 4 mouse strains - link to the microbiome?" In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4371.

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