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Relevant bibliographies by topics / HTLV-1 calcium mitochondrial membrane potential phosphorylation

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Academic literature on the topic 'HTLV-1 calcium mitochondrial membrane potential phosphorylation'

Author: Grafiati

Published: 9 March 2023

Last updated: 10 March 2023

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Journal articles on the topic "HTLV-1 calcium mitochondrial membrane potential phosphorylation"

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González-Moreno, Luis, Inés Juaristi, Jorgina Satrústegui, and Araceli del Arco. "Adenine nucleotides transport across the inner mitochondrial membrane in cancer cells: role of ANTs and SCaMCs." IBJ Plus 1, s5 (2022): 20. http://dx.doi.org/10.24217/2531-0151.22v1s5.00020.

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Introduction: The regulation of adenine nucleotide levels in the mitochondria and the cytosol is essential for cell metabolism. To sustain mitochondrial respiration, ATP generated by ATP synthase must be exported to the cytosol, whereas mitochondria must be refilled with ADP and Pi. Adenine Nucleotide Translocases (ANTs), which catalyze the electrogenic ATP4-/ADP3- exchange, have been proposed to work together with ATP synthase and the mitochondrial phosphate carrier to support mitochondrial oxidative phosphorylation by forming the so-called ATP synthasome. Nevertheless, disruption of the main

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Magnus, Gerhard, та Joel Keizer. "Model of β-cell mitochondrial calcium handling and electrical activity. II. Mitochondrial variables". American Journal of Physiology-Cell Physiology 274, № 4 (1998): C1174—C1184. http://dx.doi.org/10.1152/ajpcell.1998.274.4.c1174.

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In the preceding article [ Am. J. Physiol. 274 ( Cell Physiol. 43): C1158–C1173, 1998], we describe the development of a kinetic model for the interaction of mitochondrial Ca2+ handling and electrical activity in the pancreatic β-cell. Here we describe further results of those simulations, focusing on mitochondrial variables, the rate of respiration, and fluxes of metabolic intermediates as a function of d-glucose concentration. Our simulations predict relatively smooth increases of O2consumption, adenine nucleotide transport, oxidative phosphorylation, and ATP production by the tricarboxylic

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Dedkova, Elena N., Xiang Ji, Stephen L. Lipsius, and Lothar A. Blatter. "Mitochondrial calcium uptake stimulates nitric oxide production in mitochondria of bovine vascular endothelial cells." American Journal of Physiology-Cell Physiology 286, no. 2 (2004): C406—C415. http://dx.doi.org/10.1152/ajpcell.00155.2003.

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Although nitric oxide (NO) is a known modulator of cell respiration in vascular endothelium, the presence of a mitochondria-specific nitric oxide synthase (mtNOS) in these cells is still a controversial issue. We have used laser scanning confocal microscopy in combination with the NO-sensitive fluorescent dye DAF-2 to monitor changes in NO production by mitochondria of calf vascular endothelial (CPAE) cells. Cells were loaded with the membrane-permeant NO-sensitive dye 4,5-diaminofluorescein (DAF-2) diacetate and subsequently permeabilized with digitonin to remove cytosolic DAF-2 to allow meas

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Berzabá-Evoli, Edna, Cecilia Zazueta, Jarumi Hishel Cruz Hernández, et al. "Leptin Modifies the Rat Heart Performance Associated with Mitochondrial Dysfunction Independently of Its Prohypertrophic Effects." International Journal of Endocrinology 2018 (August 1, 2018): 1–10. http://dx.doi.org/10.1155/2018/6081415.

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Background. Functional receptors for leptin were described on the surface of cardiomyocytes, and there was a prohypertrophic effect with high concentrations of the cytokine. Therefore, leptin could be a link between obesity and the prevalence of cardiovascular diseases. On the other hand, a deleterious effect of leptin on mitochondrial performance was described, which was also associated with the evolution of cardiac hypertrophy to heart failure. The goal of our study was to analyze the effect of the exposure of rat hearts to a high concentration of leptin on cardiac and mitochondrial function

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Chaanine, Antoine H., Erik Kohlbrenner, Scott I. Gamb, et al. "FOXO3a regulates BNIP3 and modulates mitochondrial calcium, dynamics, and function in cardiac stress." American Journal of Physiology-Heart and Circulatory Physiology 311, no. 6 (2016): H1540—H1559. http://dx.doi.org/10.1152/ajpheart.00549.2016.

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The forkhead box O3a (FOXO3a) transcription factor has been shown to regulate glucose metabolism, muscle atrophy, and cell death in postmitotic cells. Its role in regulation of mitochondrial and myocardial function is not well studied. Based on previous work, we hypothesized that FOXO3a, through BCL2/adenovirus E1B 19-kDa protein-interacting protein 3 (BNIP3), modulates mitochondrial morphology and function in heart failure (HF). We modulated the FOXO3a-BNIP3 pathway in normal and phenylephrine (PE)-stressed adult cardiomyocytes (ACM) in vitro and developed a cardiotropic adeno-associated viru

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Azarashvili, Tamara, Yulia Baburina, Dmitry Grachev, et al. "Calcium-induced permeability transition in rat brain mitochondria is promoted by carbenoxolone through targeting connexin43." American Journal of Physiology-Cell Physiology 300, no. 3 (2011): C707—C720. http://dx.doi.org/10.1152/ajpcell.00061.2010.

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Carbenoxolone (Cbx), a substance from medicinal licorice, is used for antiinflammatory treatments. We investigated the mechanism of action of Cbx on Ca2+-induced permeability transition pore (PTP) opening in synaptic and nonsynaptic rat brain mitochondria (RBM), as well as in rat liver mitochondria (RLM), in an attempt to identify the molecular target of Cbx in mitochondria. Exposure to threshold Ca2+ load induced PTP opening, as seen by sudden Ca2+ efflux from the mitochondrial matrix and membrane potential collapse. In synaptic RBM, Cbx (1 μM) facilitated the Ca2+-induced, cyclosporine A-sen

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Alekseenko, S. N., A. S. Tatevosyan, A. V. Bunyakin, V. G. Zolotukhina, and R. G. Kazarov. "PHYSIOLOGICAL FUNCTIONAL STATES OF MITOCHONDRIA IN THE THERMODYNAMIC AND ELECTROCHEMICAL CYCLE." Kuban Scientific Medical Bulletin 25, no. 5 (2018): 123–35. http://dx.doi.org/10.25207/1608-6228-2018-25-5-123-135.

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Aim.This study was performed to identify the possible physiological and pathogenetic processes taking place in the mitochondrial matrix which create the conditions for lithogenesis of insoluble calcium phosphate salts (calcium carbonate...). Whereas, they can later be deposited in various tissues, taking into account the fact that the formation of calcium phosphate (calcium carbonate ...) in the human body occurs under normal physiological conditions (bone tissue, otolith...). It raises the urgency of the question of understanding the physiological and pathogenetic mechanisms of lithogenesis.M

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Vilventhraraja, Emma, Tetyana Klymenko, Jennifer Edelmann, John Gribben, and Andrejs Ivanov. "Reduction of Mitochondrial Membrane Potential Leads to Enhancement of Type-II CD20-Antibody Cytotoxicity in Diffuse Large B-Cell Lymphoma." Blood 128, no. 22 (2016): 1763. http://dx.doi.org/10.1182/blood.v128.22.1763.1763.

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Abstract Diffuse Large B Cell Lymphoma (DLBCL) is the most prevalent non-Hodgkin lymphoma (NHL) in adults. Since the addition of the Type I anti-CD20 antibody Rituximab to chemotherapy, the overall survival of NHL patients has improved dramatically compared to the pre-Rituximab era. DLBCL however, has the worst survival rates out of all NHLs with an average 5-year survival of 55%. Unfortunately 40% of all DLBCL patients relapse within 2 years, and those that relapse or have refractory disease tend not to respond well to antibody-based salvage therapies. Since the discovery and utilisation of R

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Rosenberg, Evgenia, Valeria Voevoda, Hila Magen, et al. "Venetoclax Reverses Metabolic Reprogramming Induced By S1P Modulator FTY720, Suppresses Oxidative Phosphorylation and Synergistically Targets Multiple Myeloma." Blood 138, Supplement 1 (2021): 1195. http://dx.doi.org/10.1182/blood-2021-149372.

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Abstract Patients with multiple myeloma (MM) invariably relapse with chemotherapy-resistant disease, underscoring the need for new therapeutic modalities that bypass these resistance mechanisms. FTY720, also known as fingolimod, is an S1P modulator approved by the FDA to treat the relapsing form of multiple sclerosis. Previously we reported that FTY720 exhibits potent anti-myeloma effect in vitro and in vivo in disseminated xenograft model of MM (Beider et al., Clin Cancer Res 2017). Cytotoxic activity of FTY720 was associated with down-regulation of anti-apoptotic protein MCL-1 while not affe

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Bahmed, Karim, Chih-Ru Lin, Hannah Simborio, et al. "The role of DJ-1 in human primary alveolar type II cell injury induced by e-cigarette aerosol." American Journal of Physiology-Lung Cellular and Molecular Physiology 317, no. 4 (2019): L475—L485. http://dx.doi.org/10.1152/ajplung.00567.2018.

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The alveolus participates in gas exchange, which can be impaired by environmental factors and toxins. There is an increase in using electronic cigarettes (e-cigarettes); however, their effect on human primary alveolar epithelial cells is unknown. Human lungs were obtained from nonsmoker organ donors to isolate alveolar type II (ATII) cells. ATII cells produce and secrete pulmonary surfactant and restore the epithelium after damage, and mitochondrial function is important for their metabolism. Our data indicate that human ATII cell exposure to e-cigarette aerosol increased IL-8 levels and induc

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Dissertations / Theses on the topic "HTLV-1 calcium mitochondrial membrane potential phosphorylation"

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Biasiotto, Roberta. "Functional characterization of the p13 protein of HTLV-1." Doctoral thesis, Università degli studi di Padova, 2010. http://hdl.handle.net/11577/3422371.

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HTLV-1 is the causative agent of adult T-cell leukemia-lymphoma (ATLL), an aggressive neoplasm of mature CD4+ T cells refractory to current therapies, and of HAM/TSP, a neurodegenerative disease. HTLV-1 is a complex retrovirus whose genome encodes several regulatory and accessory proteins (Rex, Tax, HBZ, p12, p30, p13) in addition to the structural proteins (gag, pol, env) common to all retroviruses. Previous studies demonstrated that HTLV-1 p13, an 87-amino acid protein, is targeted to the inner membrane of mitochondria and induces mitochondrial fragmentation. In vitro studies carried out u

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