Relevant bibliographies by topics / HTLV-1 [Virus T lymphotrope humain de type 1]

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'HTLV-1 [Virus T lymphotrope humain de type 1]'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "HTLV-1 [Virus T lymphotrope humain de type 1]"

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Iwamoto, Masayoshi, Naohiko Nakamura, Hideki Harada, et al. "A Case of HTLV-1 Associated Primary Gastric Lymphoma." Japanese Journal of Gastroenterological Surgery 45, no. 12 (2012): 1170–79. http://dx.doi.org/10.5833/jjgs.45.1170.

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Larkin, Julie, John T. Sinnott, Joshua Weiss, and Douglas A. Holt. "Human T-Cell Lymphotropic Virus-Type I." Infection Control & Hospital Epidemiology 11, no. 6 (1990): 314–18. http://dx.doi.org/10.1086/646177.

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Human T-cell lymphotropic virus type-1 (HTLV-I) is a recently recognized retrovirus identified as the cause of adult T-cell leukemia-lymphoma (ATLL) and HTLV-I-associated myelopathy (TSPI HAM). HTLV-I, a member of theRetroviridaefamily of viruses, was first described in 1980 after the isolation of the virus from a patient with a T-cell lymphoma. These pathogenic retroviruses are typically divided into theOncovirinaeandLentivirinae. The oncovirus group, including HTLV-I, HTLV-II and bovine leukemia virus (BLV), is generally associated with tumors. The lentiviruses are associated with immune def
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Tobinai, Kensei. "3) Human T-lymphotropic Virus Type-I and Adult T-cell Leukemia-lymphoma." Nihon Naika Gakkai Zasshi 104, Suppl (2015): 108b—109a. http://dx.doi.org/10.2169/naika.104.108b.

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Tobinai, Kensei. "3) Human T-lymphotropic Virus Type-I and Adult T-cell Leukemia-lymphoma." Nihon Naika Gakkai Zasshi 104, no. 9 (2015): 1872–77. http://dx.doi.org/10.2169/naika.104.1872.

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Pomier, Carole, Samira Rabaaoui, Jean-François Pouliquen, et al. "Antiretroviral therapy promotes an inflammatory-like pattern of human T-cell lymphotropic virus type 1 (HTLV-1) replication in human immunodeficiency virus type 1/HTLV-1 co-infected individuals." Journal of General Virology 94, no. 4 (2013): 753–57. http://dx.doi.org/10.1099/vir.0.048348-0.

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Upon antiretroviral therapy (ART) human immunodeficiency virus (HIV)/human T-cell lymphotropic virus type 1 (HTLV-1) co-infected individuals frequently develop neurological disorders through hitherto unknown mechanisms. Here, we show that effective anti-HIV ART increases HTLV-1 proviral load through a polyclonal integration pattern of HTLV-1 in both CD4+ and CD8+ T-cell subsets that is reminiscent of that typically associated with HTLV-1-related inflammatory conditions. These data indicate that preventing ART-triggered clonal expansion of HTLV-1-infected cells in co-infected individuals deserv
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Edhom, Karolina af, Christer Lidman, Tobias Granberg, Graham P. Taylor, and Martin Paucar. "Expanding the etiologic spectrum of spastic ataxia syndrome: chronic infection with human T lymphotropic virus type 1." Journal of NeuroVirology 27, no. 2 (2021): 345–47. http://dx.doi.org/10.1007/s13365-020-00932-2.

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Abstract Human T-lymphotropic virus type-1 (HTLV-1) is a neglected infection most often associated with an indolent process. However, a subset of HTLV-1 seropositive patients face the risk to develop life-threatening T-cell lymphoma/leukemia, or the highly disabling and incurable HTLV1-associated myelopathy/tropical spastic paraparesis (HAM/TSP). Over the years, other complications to HTLV-1 have been proposed and debated intensely. One of these, although rare, associations include cerebellar ataxia occurring most often in Japanese patients with manifest HAM/TSP. Here we present a HTLV-1 serop
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SUDA, Takeshi, Keiji SUZUKI, Kazushige ITO, Kazunobu SUZUKI, Hiromi SERIZAWA, and Yasuhisa KOYANAGI. "A CASE OF PRIMARY GASTRIC T-CELL LYMPHOMA (PGTCL) WITHOUT ANTI-HUMAN T-LYMPHOTROPIC VIRUS TYPE-1 (HTLV-1) ANTIBODY." Nihon Rinsho Geka Gakkai Zasshi (Journal of Japan Surgical Association) 62, no. 2 (2001): 381–86. http://dx.doi.org/10.3919/jjsa.62.2_381.

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Tarsis, Sara L., Ming-Tsung Yu, Elizabeth S. Parks, Deborah Persaud, José L. Muñoz, and Wade P. Parks. "Human T-Lymphocyte Transformation with Human T-Cell Lymphotropic Virus Type 2." Journal of Virology 72, no. 1 (1998): 841–46. http://dx.doi.org/10.1128/jvi.72.1.841-846.1998.

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ABSTRACT Human T-cell lymphotrophic virus type 2 (HTLV-2), a common infection of intravenous drug users and subpopulations of Native Americans, is uncommon in the general population. In contrast with the closely related HTLV-1, which is associated with both leukemia and neurologic disorders, HTLV-2 lacks a strong etiologic association with disease. HTLV-2 does shares many properties with HTLV-1, including in vitro lymphocyte transformation capability. To better assess the ability of HTLV-2 to transform lymphocytes, a limiting dilution assay was used to generate clonal, transformed lymphocyte l
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Ghez, David, Yves Lepelletier, Sophie Lambert, et al. "Neuropilin-1 Is Involved in Human T-Cell Lymphotropic Virus Type 1 Entry." Journal of Virology 80, no. 14 (2006): 6844–54. http://dx.doi.org/10.1128/jvi.02719-05.

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ABSTRACT Human T-cell lymphotropic virus type 1 (HTLV-1) is transmitted through a viral synapse and enters target cells via interaction with the glucose transporter GLUT1. Here, we show that Neuropilin-1 (NRP1), the receptor for semaphorin-3A and VEGF-A165 and a member of the immune synapse, is also a physical and functional partner of HTLV-1 envelope (Env) proteins. HTLV-1 Env and NRP1 complexes are formed in cotransfected cells, and endogenous NRP1 contributes to the binding of HTLV-1 Env to target cells. NRP1 overexpression increases HTLV-1 Env-dependent syncytium formation. Moreover, overe
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Mahieux, Renaud, Colombe Chappey, Marie-Claude Georges-Courbot, et al. "Simian T-Cell Lymphotropic Virus Type 1 from Mandrillus sphinx as a Simian Counterpart of Human T-Cell Lymphotropic Virus Type 1 Subtype D." Journal of Virology 72, no. 12 (1998): 10316–22. http://dx.doi.org/10.1128/jvi.72.12.10316-10322.1998.

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ABSTRACT A recent serological and molecular survey of a semifree-ranging colony of mandrills (Mandrillus sphinx) living in Gabon, central Africa, indicated that 6 of 102 animals, all males, were infected with simian T-cell lymphotropic virus type 1 (STLV-1). These animals naturally live in the same forest area as do human inhabitants (mostly Pygmies) who are infected by the recently described human T-cell lymphotropic virus type 1 (HTLV-1) subtype D. We therefore investigated whether these mandrills were infected with an STLV-1 related to HTLV-1 subtype D. Nucleotide and/or amino acid sequence
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Dissertations / Theses on the topic "HTLV-1 [Virus T lymphotrope humain de type 1]"

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Chevalier, Sébastien. "Etude comparative des protéines TAX des virus humains de la leucémie/lymphome à cellules T de type 1, 2 et 3 (HTLV-1, HTLV-2, HTLV-3) et du virus simien STLV-3." Paris 7, 2007. http://www.theses.fr/2007PA077121.

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Les virus htlv-1 et htlv-2 sont des retrovirus complexes du genre deltaretrovirus. Dans le groupe des ptlv pour "primate t lymphotropic virus", on trouve a la fois des virus humains et des virus simiens : htlv-1 et stlv-1, htlv-2 et stlv-2, stlv-3, et plus recemment htlv-3 et htlv-4. Nos etudes ont permis de mettre en evidence plusieurs differences phenotypiques entre les virus htlv-1 et htlv-2. Nous avons d'abord montre que les proteines trans-activatrices tax-1 et tax-2 ne possedaient pas la meme localisation intracellulaire. De plus, la localisation des proteines tax-1 et tax-2 est correlee
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Ghez, David. "La neuropiline-1 est une molécule d'entrée du rétrovirus HTLV-1." Paris 7, 2008. http://www.theses.fr/2008PA077038.

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Pendant plus de 20 suivant la découverte du rétro virus humain HTLV-1, l'identité de son récepteur cellulaire est demeurée inconnue. Différentes molécules ont été proposées mais leur rôle en tant que récepteur d'HTLV-1 rapidement infirmé. Récemment, il a été montré que le transporteur de glucose Glutl présentait des propriétés compatibles avec celles du récepteur d'HTLV-1 et que les héparanes sulfates protéoglycanes facilitaient l'infection virale. Parce qu'elle possédait également de nombreuses caractéristiques compatibles avec celles attendues du récepteur et qu'elle permettait de mieux expl
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Perugi, Fabien. "HDLG, un médiateur cellulaire de l'assemblage des rétrovirus VIH-1 et HTLV-1." Paris 7, 2008. http://www.theses.fr/2008PA077020.

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La compréhension des différentes étapes conduisant à la production des particules virales est, à l'heure actuelle, un axe majeur dans la recherche en rétrovirologie. Dans le souci de trouver une protéine cellulaire capable de faire le pont entre les précurseurs Gag et Env chez HTLV-1, notre équipe a identifié hDIg, lors d'un crible double hybride, comme un des partenaires cellulaires de la région cytoplasmique des glycoprotéines d'enveloppe du HTLV-1. Une étude approfondie de l'interaction de hDIg avec les protéines structurales d'enveloppe (Env) a montré que cette interaction est nécessaire d
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Pais, Correia Ana Monica. "Biofilm-like extracellular viral assemblies mediate HTLV-1 (human T cell leukemia virus type-1) cell-to-cell transmission at virological synapses." Paris 7, 2009. http://www.theses.fr/2009PA077233.

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Près de 20 millions d'individus sont infectés par HTLV-1 (human T cell leukemia virus type-1). La plupart des patients n'ont aucun symptôme, seulement 5-10% d'entre eux développeront des maladies associées à l'infection virale. La particularité d'HTLV-1 est de se transmettre par des contacts cellulaires désignés « synapses virologiques », qui partagent certaines caractéristiques avec les synapses immunologiques. Ce projet a permis de mieux comprendre le processus de transmission cellule à cellule et introduit un nouveau concept de transmission virale par des « biofilms viraux ». Contrairement
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Gessain, Antoine. "Virus HTLV-1 et paraparesie spastique tropicale. Un rétrovirus leucemogene associe a une maladie neurologique : épidémiologie, caractérisation des isolats viraux associes et aspects moléculaires." Paris 7, 1992. http://www.theses.fr/1992PA077309.

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Le virus htlv-i retrovirus isole chez l'homme en 1980 est endemique au japon, dans la region caraibe, en amerique du sud et en afrique intertropicale. Dans ces regions 1 a 30% de la population generale a des anticorps seriques anti-htlv-i. Ce virus est l'agent etiologique de la leucemie t de l'adulte (atl) et d'une neuromyelopathie chronique denommee paraparesie spastique tropicale/myelopathie associee a l'htlv-i (tsp/ham). Ce travail realise entre 1983 et 1991 comporte quatre etapes successives. 1) dans un travail initial d'epidemiologie clinique et de terrain, j'ai participe a la decouverte
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Mauclere, Philippe. "Séroépidémiologie, diversité génétique et transmissions interespèces des infections rétrovirales HTLV/STLV et VIH/SIV au Cameroun." Paris 5, 2002. http://www.theses.fr/2002PA05N098.

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L' Afrique centrale est aujourd'hui considérée comme le berceau de la diversité génétique rétroviale des primates. En 1991 au Cameroun, la connaissance de l'endémie par l'oncovirus HTLV-1 reposait sur des données de séroprévalence très prélíminaires, et aucun cas d'infection par HTLV-2 n'y avait été identifié. L'épidémie de VIH-1 débutait sa progression, mais la circulation des variants majeurs n'avait pas été rapportée, et aucune souche de SIV n'avait été recherchéé chez les primates vivant dans le pays. Le but de ce travail, réalisé à partir d'études menées au Cameroun entre 1991 et 1997, pu
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Desdouits, Marion. "Myopathies virales : étude des interactions entre les cellules musculaires et les virus HTLV-1 et Influenza A." Paris 7, 2011. http://www.theses.fr/2011PA077181.

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Ce travail de thèse porte sur l'étude des myopathies associées à l'infection par les virus HTLV-1 et Influenza A chez l'Homme. Les myopathies inflammatoires associées à HTLV-1 (HAIM) présentent des points communs avec les myopathies inflammatoires idiopathiques (IIM) et avec la paraparésie spastique tropicale ou myélopathie associée à HTLV-1 (TSP/HAM). Une étude basée sur l'analyse de prélèvements musculaires et sanguins de 13 patients atteints d'HAIM nous a permis de montrer que ces patients présentent une charge provirale faible, similaire à celle de personnes infectées asymptomatiques, alor
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Meertens, Laurent. "La diversité génétique des STLV-1 et des STLV-3 et l' étude des mécanismes de répression de l' activité transcriptionnelle de la protéine suppresseur de tumeur p53 par la protéine Tax d' HTLV-2 de sous-type B." Paris 7, 2003. http://www.theses.fr/2003PA077171.

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Delebecque, Frédéric. "Mise au point d'un modèle murin d'infection par HTLV-1 à l'aide de virus chimériques contenant l'enveloppe de Moloney-MuLV." Paris 6, 2003. http://www.theses.fr/2003PA066087.

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Afonso, Philippe. "Paraparésie Spastique Tropicale/Myélopathie Associée à HTLV-1 : mécanismes de rupture de la barrière hémato-encéphalique et interventions thérapeutiques." Paris 7, 2008. http://www.theses.fr/2008PA077059.

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Le retrovirus humain t-lymphotrope de type 1 (htlv-1) est l'agent etiologique d'une maladie neurodegenerative appelee paraparesie spastique tropicale / myelopathie associee a htlv-1 (tsp/ham). La tsp/ham est associee a la presence d'infiltrats lymphocytaires dans le systeme nerveux central (snc) et de proteines seriques dans le parenchyme nerveux. Le snc est normalement isole du reste de l'organisme par la barriere hemato-encephalique (bhe). La bhe est constituee de 3 types cellulaires : les astrocytes, les pericytes et les cellules endotheliales. Ces dernieres forment un capillaire continu et
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Books on the topic "HTLV-1 [Virus T lymphotrope humain de type 1]"

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Mckhann, Guy Mead. Isolation and characterization of human T-cell lymphotropic virus type-1 from patients with tropical spastic paraparesis. s.n.], 1990.

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Scientific Group on Human T-Lymphotropic Virus Tupe-1 (HTLV-1) Infections and Associated Diseases (1992 Kagoshima-shi, Japan). Report: Scientific Group on Human T-Lymphotropic Virus Type-1 (HTLV-1) Infections and Associated Diseases, Kagoshima, Japan, 13-14 October 1992. The Office, 1993.

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Moriuchi, Hiroyuki. Human T-cell Lymphotropic Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0010.

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Human T-cell lymphotropic virus type 1 (HTLV-1), a human retrovirus that infects an estimated 10–20 million people worldwide, has endemic foci in Japan, West and Central Africa, the Caribbean, Central and South America, and Melanesia. Also, it is the etiological agent of a lymphoproliferative malignancy, adult T-cell leukemia/lymphoma (ATLL), as well as chronic inflammatory diseases such as HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP). HTLV-1 can be transmitted vertically, sexually, or by blood-borne transmission. ATLL occurs in approximately 5% of carriers who are infec
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Per, Höllsberg, and Hafler David A, eds. Human T-cell lymphotropic virus type I. J. Wiley and Sons, 1996.

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Araujo, Abelardo Q.-C. Neurological Manifestations of the Human T-lymphotropic Virus Type 1. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0161.

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The human T cell lymphotropic virus type 1 (HTLV-1) is a retrovirus that infects about 20 million individuals worldwide. Its typical neurological presentation is of a chronic, slowly progressive myelopathy named “HTLV-1-associated myelopathy/tropical spastic paraparesis” (HAM/TSP). HAM/TSP emerges as the tip of the iceberg among numerous other neurological clinical syndromes caused by this virus, such as inflammatory myopathies, polyneuropathies, ALS-like syndromes, dysautonomia, etc. HAM/TSP designates a spastic paraparesis with neurogenic bladder, and minor sensory signs. Pathologically, HAM
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Roman, Eve, Alexandra Smith, and Lorelei Mucci. Leukemias. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0028.

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Leukemias are a diverse group of acute and chronic haematological malignancies, that account for 2% to 3% of cancers globally. Recent advances in molecular biology and therapy have transformed the landscape for several leukemia subtypes changing some, but by no means all, from rapidly fatal diseases to treatable conditions with a good prognosis. In general, however, this progress has not been matched by new aetiological insights. Albeit accounting for a relatively small proportion, genetic predisposition syndromes such as neurofibromatosis, Li-Fraumeni and Trisomy 21, have the biggest impact i
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Chen, Irvin S. Y. Transacting Functions Of Human Retroviruses (Current Topics in Microbiology & Immunology). Edited by Irvin S. Y. Chen. Springer, 1995.

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Y, Chen Irvin S., ed. Transacting functions of human retroviruses. Springer-Verlag, 1995.

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Book chapters on the topic "HTLV-1 [Virus T lymphotrope humain de type 1]"

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Bittencourt, Achiléa Lisboa. "Human T-Cell Lymphotropic Virus Type-1 (HTLV-1) Infection in Dermatology." In Dermatology in Public Health Environments. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-33919-1_42.

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Jacobson, Steven, and Raya Massoud. "Human T-Cell Lymphotropic Virus Type 1 Infection." In Viral Infections of the Human Nervous System. Springer Basel, 2012. http://dx.doi.org/10.1007/978-3-0348-0425-7_8.

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Caruso, Breanna, Raya Massoud, and Steven Jacobson. "Human T-Cell Lymphotropic Virus Types 1 and 2." In Manual of Molecular and Clinical Laboratory Immunology. ASM Press, 2016. http://dx.doi.org/10.1128/9781555818722.ch70.

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Araujo, Abelardo, Noreen Sheehy, Hidehiro Takahashi, and William W. Hall. "Concomitant Infections with Human Immunodeficiency Virus Type 1 and Human T-Lymphotropic Virus Types 1 and 2." In Polymicrobial Diseases. ASM Press, 2014. http://dx.doi.org/10.1128/9781555817947.ch5.

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Gallo, Dana. "Laboratory Tests for Human T-Lymphotropic Virus Type I." In Medical Virology 8. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4899-0891-9_2.

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Araujo, Abelardo Q. C., Marco A. Lima, and Marcus Tulius Silva. "Neurological complications of human T-cell lymphotropic virus type-1 infection." In International Neurology. John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118777329.ch88.

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Schönbach, Christian. "Human T-Lymphotropic Virus Type-I-associated Myelopathytropical Spastic Paraparesis." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_753.

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Sarngadharan, M. G., Fulvia diMarzo Veronese, and Robert C. Gallo. "Proteins Encoded by the Human T-Lymphotropic Virus Type III/ Lymphadenopathy Associated Virus (HTLV-III/LAV) Genes." In New Experimental Modalities in the Control of Neoplasia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5242-6_27.

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Yoshimitsu, Makoto, Yohann White, and Naomichi Arima. "Prevention of Human T-Cell Lymphotropic Virus Type 1 Infection and Adult T-Cell Leukemia/Lymphoma." In Viruses and Human Cancer. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-38965-8_12.

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Alais, Sandrine, Hélène Dutartre, and Renaud Mahieux. "Quantitative Analysis of Human T-Lymphotropic Virus Type 1 (HTLV-1) Infection Using Co-Culture with Jurkat LTR-Luciferase or Jurkat LTR-GFP Reporter Cells." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6872-5_4.

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Conference papers on the topic "HTLV-1 [Virus T lymphotrope humain de type 1]"

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Kuriyama, Sachiko, Shinichi Sasaki, Yukiko Nanba, et al. "An Autopsy Case Of MPO-ANCA Positive Microscopic Polyangitis In A Human T-lymphotropic Virus Type 1 Carrier With Interstitial Pneumonia." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4501.

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Forlani, Greta, Rawan Abdallah, Roberto S. Accolla, and Giovanna Tosi. "Abstract B048: The MHC class II transactivator CIITA inhibits the persistent activation of NF-kB by Human T cell Lymphotropic Virus type-1 Tax-1 oncoprotein." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b048.

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Makoto, Ishihara, Natsumi Araya, Tomoo Sato, et al. "Abstract 4803: Quantitative proteome profiling of CD4+CD25+CCR4+ T-cells to identify potential therapeutic targets for adult T-cell leukemia (ATL) and Human T-lymphotropic virus type-1 associated myelopathy (HAM)." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4803.

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