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1

Roychoudhury, Arun K. Human polymorphic genes: World distribution. Oxford University Press, 1988.

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2

Masatoshi, Nei, ed. Humanpolymorphic genes. Oxford University Press, 1988.

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3

Kiaris, Hippokratis. Genes, polymorphisms, and the making of societies: How genetic behavioral traits influence human cultures. Universal-Publishers, 2012.

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4

Bieber, Heidi. Traces of antiquity in genes: Comparative study of serum protein polymorphisms between populations of Africa, Europe and Amerindians of Central America. Shaker Verlag, 1997.

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5

Divan, Aysha, and Janice A. Royds. 2. DNA. Oxford University Press, 2016. http://dx.doi.org/10.1093/actrade/9780198723882.003.0002.

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Another significant milestone was the publication in 2003 of the complete sequence of the human genome—the entire DNA contained within the forty-six chromosomes located in the nucleus of each human somatic (body) cell. Once this was published, further worldwide projects were launched to work out what the functions of these genes and other regions of the genome actually were. ‘DNA’ outlines the components of the human genome and their organization; DNA replication; mutations and correction mechanisms; polymorphisms; and new DNA technologies, including gene cloning, the polymerase chain reaction
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6

Han, Shihui. Gene-culture interaction on human behavior and the brain. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198743194.003.0007.

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Chapter 7 reviews empirical findings that allow consideration of biological and environmental influences on human behavior from an evolutionary perspective (e.g., gene-culture coevolution) and from a perspective of individual development (e.g., gene-culture interaction). It also reviews imaging genetic studies that link genes with brain functional organization. It introduces a cultural neuroscience paradigm for investigating genetic influences on the coupling of brain activity and culture by presenting two studies that examined how serotonin transporter functional polymorphism and oxytocin rec
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7

Hope, James, and Mark P. Dagleish. Prion-protein-related diseases of animals and man. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0041.

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Scrapie, bovine spongiform encephalopathy (BSE), Creutzfeldt–Jakob disease (CJD), and related diseases of mink (transmissible mink encephalopathy), mule deer and elk (chronic wasting disease) are the founder members of a group of diseases called the transmissible degenerative (or spongiform) encephalopathies (TSE). These diseases can be transmitted by prions from affected to healthy animals by inoculation or by feeding diseased tissues. Prions are cellular proteins that can transfer metabolic and pathological phenotypes vertically from parent to progeny or horizontally between cells and animal
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8

Osman, Marwa. In Silico Analysis and Modeling of Deleterious Single Nucleotide Polymorphism (Snps) in Human Gata4 Gene. GRIN Verlag GmbH, 2016.

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9

Kiaris, Hippokratis. Genes, Polymorphisms, and the Making of Societies: A Genetic Perspective of the Divergence Between East and West. Universal Publishers, 2021.

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10

Westberg, Lars, and Hasse Walum. Oxytocin and Vasopressin Gene Variation and the Neural Basis of Social Behaviors. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.011.

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Experimental studies in rodents and humans show that the neuropeptides oxytocin and vasopressin are important regulators of behaviors related to social interactions. Evidence for positive effects of oxytocin treatment on symptoms of psychiatric disorders characterized by impaired social functioning has emerged. Numerous studies report associations between various social behaviors, the risk of autism, and polymorphisms inOXTRandAVPR1A. This chapter provides an overview of these genetic association studies. Although many of the published findings are inconclusive and need replication in independ
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11

Hamilton, Matthew Lloyd. COMT genotypes in pain responses. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0080.

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The landmark study discussed in this chapter is ‘COMT val158met genotype affects μ‎-opioid neurotransmitter responses to a pain stressor’, published by Zubieta et al. in 2003. Catechol-O-methyl-transferase (COMT) is a key modulator of dopaminergic and noradrenergic neurotransmission. This study focused on a single nucleotide polymorphism of the COMT gene encoding the substitution of valine (val) by methionine (met) at Codon 158 (val158met), resulting in a three- to fourfold reduction in its activity. Individuals with the val/val genotype have the highest activity of COMT, val/met genotypes hav
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12

Troisi, Alfonso. Infidelity. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199393404.003.0005.

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This chapter reviews recent data on the evolution of sexual strategies in humans and shows how, in the natural environment, the adaptive functions of sexual infidelity were substantially different in males and females. The meaning of technical terms used by evolutionary biologists to describe different behavioral strategies related to maximization of reproductive success are explained, including the Coolidge effect, good-gene sexual strategy, and serial monogamy. Biological analysis of motivations for sexual infidelity integrate evolutionary hypotheses with recent data from molecular genetic s
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13

Whitworth, Caroline, and Stewart Fleming. Malignant hypertension. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0216.

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Malignant hypertension (MH) is recognized clinically by elevated blood pressure together with retinal haemorrhages or exudates with or without papilloedema (grades III or IV hypertensive retinopathy); and may constitute a hypertensive emergency or crisis when complicated by evidence of end-organ damage including microangiopathic haemolysis, encephalopathy, left ventricular failure, and renal failure. Though reversible, it remains a significant cause of end-stage renal failure, and of cardiovascular and cerebrovascular morbidity and mortality in developing countries.MH can complicate pre-existi
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14

Rabinow, Paul. French DNA: Trouble in Purgatory. University Of Chicago Press, 2002.

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15

Rabinow, Paul. French DNA: Trouble in Purgatory. University Of Chicago Press, 1999.

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