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Academic literature on the topic 'Human herpesviruses'

Author: Grafiati

Published: 3 June 2025

Last updated: 22 June 2025

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Journal articles on the topic "Human herpesviruses"

Kutle, Ivana, Anne Dittrich, and Dagmar Wirth. "Mouse Models for Human Herpesviruses." Pathogens 12, no. 7 (2023): 953. http://dx.doi.org/10.3390/pathogens12070953.

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More than one hundred herpesviruses have been isolated from different species so far, with nine infecting humans. Infections with herpesviruses are characterized by life-long latency and represent a significant challenge for human health. To investigate the consequences of infections and identify novel treatment options, in vivo models are of particular relevance. The mouse has emerged as an economical small animal model to investigate herpesvirus infections. However, except for herpes simplex viruses (HSV-1, HSV-2), human herpesviruses cannot infect mice. Three natural herpesviruses have been identified in mice: mouse-derived cytomegalovirus (MCMV), mouse herpesvirus 68 (MHV-68), and mouse roseolovirus (MRV). These orthologues are broadly used to investigate herpesvirus infections within the natural host. In the last few decades, immunocompromised mouse models have been developed, allowing the functional engraftment of various human cells and tissues. These xenograft mice represent valuable model systems to investigate human-restricted viruses, making them particularly relevant for herpesvirus research. In this review, we describe the various mouse models used to study human herpesviruses, thereby highlighting their potential and limitations. Emphasis is laid on xenograft mouse models, covering the development and refinement of immune-compromised mice and their application in herpesvirus research.

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Bouchemal, Amina, Joan Martí Carreras, Lydia Khireddine, et al. "Investigation on colorectal cancer and human herpesvirus infection among Algerian patients." Journal of Infection in Developing Countries 17, no. 05 (2023): 656–64. http://dx.doi.org/10.3855/jidc.17640.

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Introduction: Herpesviruses are a widespread family of double-stranded DNA viruses that establish life-long persistent infection in their hosts. Cumulative evidence tends to argue for the association of human herpesviruses, such as Kaposi's sarcoma herpesvirus (KHSV), Epstein-Barr virus (EBV), and human cytomegalovirus (HCMV) with various human disorders and diseases. The present study aims to investigate the presence of herpesviruses in colorectal cancer (CRC). Methodology: We investigated the presence of herpesviruses in 69 formalin-fixed paraffin embedded tissue (FFPE) biopsies, using a pan-herpesvirus nested polymerase chain reaction (PCR) with degenerate primers and HCMV specific primers to identify the presence of herpesviruses in CRC tissue. Results: None of the samples we examined were positive for herpesviruses. Conclusions: Our results suggest that there is no (or very low) prevalence of lifelong herpesvirus infection in Algerian CRC patients. Larger cohorts may provide more insight into the prevalence of herpesviruses in Algerian CRC biopsies.

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Hassan, Sherif T. S., Miroslava Šudomová, Alena Mazurakova, and Peter Kubatka. "Insights into Antiviral Properties and Molecular Mechanisms of Non-Flavonoid Polyphenols against Human Herpesviruses." International Journal of Molecular Sciences 23, no. 22 (2022): 13891. http://dx.doi.org/10.3390/ijms232213891.

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Herpesviruses are one of the most contagious DNA viruses that threaten human health, causing severe diseases, including, but not limited to, certain types of cancer and neurological complications. The overuse and misuse of anti-herpesvirus drugs are key factors leading to drug resistance. Therefore, targeting human herpesviruses with natural products is an attractive form of therapy, as it might improve treatment efficacy in therapy-resistant herpesviruses. Plant polyphenols are major players in the health arena as they possess diverse bioactivities. Hence, in this article, we comprehensively summarize the recent advances that have been attained in employing plant non-flavonoid polyphenols, such as phenolic acids, tannins and their derivatives, stilbenes and their derivatives, lignans, neolignans, xanthones, anthraquinones and their derivatives, curcuminoids, coumarins, furanocoumarins, and other polyphenols (phloroglucinol) as promising anti-herpesvirus drugs against various types of herpesvirus such as alpha-herpesviruses (herpes simplex virus type 1 and 2 and varicella-zoster virus), beta-herpesviruses (human cytomegalovirus), and gamma-herpesviruses (Epstein–Barr virus and Kaposi sarcoma-associated herpesvirus). The molecular mechanisms of non-flavonoid polyphenols against the reviewed herpesviruses are also documented.

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Chen, Siyu, Yue Deng, and Dongli Pan. "MicroRNA Regulation of Human Herpesvirus Latency." Viruses 14, no. 6 (2022): 1215. http://dx.doi.org/10.3390/v14061215.

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Herpesviruses are ubiquitous human pathogens. After productive (lytic) infection, all human herpesviruses are able to establish life-long latent infection and reactivate from it. Latent infection entails suppression of viral replication, maintenance of the viral genome in infected cells, and the ability to reactivate. Most human herpesviruses encode microRNAs (miRNAs) that regulate these processes during latency. Meanwhile, cellular miRNAs are hijacked by herpesviruses to participate in these processes. The viral or cellular miRNAs either directly target viral transcripts or indirectly affect viral infection through host pathways. These findings shed light on the molecular determinants that control the lytic-latent switch and may lead to novel therapeutics targeting latent infection. We discuss the multiple mechanisms by which miRNAs regulate herpesvirus latency, focusing on the patterns in these mechanisms.

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Nazaryan, R. S., Yu V. Fomenko, N. A. Scheblykina, T. A. Kolesova, N. V. Golik, and E. V. Sukhostavets. "Herpesviruses. Part 1." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 5, no. 6 (2020): 299–307. http://dx.doi.org/10.26693/jmbs05.06.299.

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One of the urgent problems of modern medicine is the high incidence of herpesvirus infections. The high prevalence of herpesviruses in the human population of the world allow us to consider herpes a common systemic disease of the whole organism. Doctors of any specialty are faced with the clinical manifestations of herpes infection in patients, and they themselves are a risk group of chronic herpes infections formation due to constant patient contacts and frequent professional psycho-emotional overload. Herpes infections are a group of infectious diseases caused by human herpesviruses. Now it is known 8 species of herpesvirus causing various human diseases that occur in the acute (during the initial contact with the infection) or chronic form. The herpesvirus family has a number of common properties that distinguish them from other human pathogenic viruses. There are three subfamilies in the Herpesviridae family. Alpha herpesviruses (Аlphaherpesvirinae) include the two serotypes of the herpes simplex virus (HSV-1 and HSV-2), and the varicella-zoster virus (herpes zoster). Beta herpesviruses (Betaherpesvirinae) include cytomegalovirus, human herpes viruses of types 6 and 7 (HHV-5, HHV-6, HHV-7). Gamma herpes viruses (Gammaherpesvirinae) include the Epstein-Barr virus, human herpesvirus type 8 (HHV-4, HHV-8). Clinical manifestations of herpes infection depend more on the immunity state of the infected organism than on the pathogenic properties of the pathogen itself, and develop only in conditions of immunodeficiency caused by various unfavorable factors. Herpesviruses are able to damage organs, weaken the body's immunity, creating conditions for the attachment of the other infections (fungal, bacterial), which in turn can cause organ damage. The herpesvirus ability to infect all organs and tissues of the body determines a significant clinical polymorphism of diseases, as well as the necessity to study various biological liquids. Herpesviruses can be transmitted from person to person by aerosol, contact, sexual and parenteral transmission, as well as from mother to fetus or newborn, they also can act as mutagens. The pathogenesis of herpesvirus infections is rather complex and not completely understood. For a proper understanding of the disease pathogenesis it is necessary to know the main stages of reproduction of human herpesviruses. Modern laboratory techniques are used for diagnosis of herpes infection and allow obtaining more complete information for an accurate diagnosis: virological method, electron microscopy method, the method of biological sample, the polymerase chain reaction methods for the detection of viral antigens, serological, immunological, cytological and histological methods. Conclusion. The high prevalence of diseases caused by herpes viruses, the complex, not fully understood pathogenesis and high contagiousness of diseases, as well as a variety of clinical manifestations of herpesvirus infections with the possibility of the formation of mixed forms with a blurred clinical picture dictate the need for a detailed study of herpes viruses and knowledge of methods for diagnosing herpes infections

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Bharucha, Tehmina, Catherine F. Houlihan, and Judith Breuer. "Herpesvirus Infections of the Central Nervous System." Seminars in Neurology 39, no. 03 (2019): 369–82. http://dx.doi.org/10.1055/s-0039-1687837.

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AbstractThere are over 200 herpesvirus species, of which 10 affect humans. Each of these 10 herpesviruses has a unique clinical syndrome, but common to all is their ability to cause infection and pathology in the central nervous system. In this article, we discuss the epidemiology, clinical presentation, diagnostic modalities, treatment, sequelae, and availability of vaccination of each of the following herpesviruses: herpes simplex virus 1 and 2, varicella zoster virus, human cytomegalovirus, human herpesvirus 6A, 6B, and 7, Epstein–Barr virus, human herpesvirus 8, and simian herpesvirus B.

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Pontejo, Sergio M., Philip M. Murphy, and James E. Pease. "Chemokine Subversion by Human Herpesviruses." Journal of Innate Immunity 10, no. 5-6 (2018): 465–78. http://dx.doi.org/10.1159/000492161.

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Viruses use diverse molecular mechanisms to exploit and evade the immune response. Herpesviruses, in particular, encode functional chemokine and chemokine receptor homologs pirated from the host, as well as secreted chemokine-binding proteins with unique structures. Multiple functions have been described for herpesvirus chemokine components, including attraction of target cells, blockade of leukocyte migration, and modulation of gene expression and cell entry by the virus. Here we review current concepts about how human herpesvirus chemokines, chemokine receptors, and chemokine-binding proteins may be used to shape a proviral state in the host.

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Rickinson, Alan. "Concluding overview: looking back, looking forward." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 356, no. 1408 (2001): 595–604. http://dx.doi.org/10.1098/rstb.2000.0785.

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The γ–herpesviruses are a group of related agents which share the same broad strategy for infection of and persistence within the lymphoid tissues of their hosts. Yet in evolutionary terms these agents are sufficiently diverse to display multiple different molecular mechanisms whereby that strategy can be achieved. Attempts are made to relate the different in vitro growth transforming capacities of the γ 1 –herpesviruses, the T–lymphotropic γ 2 –herpesviruses and the B–lymphotropic γ 2 –herpesviruses to what is known about the biology of these virus infections in their natural or in experimental hosts. The review then summarizes the evidence linking γ–herpesviruses with oncogenesis and proposes that the diverse spectrum of Epstein–Barr virus and human herpesvirus 8–associated human tumours falls into three pathogenetically distinct categories. Many questions remain unanswered in the areas of γ–herpesvirus biology and disease pathogenesis: resolving these questions will require a broadening of our experimental approaches and a willingness to relinquish ‘single–model’ panaceas.

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Yamanishi, Koichi. "New human herpesviruses; human herpesvirus 6 and 7." Clinical Biochemistry 28, no. 3 (1995): 348. http://dx.doi.org/10.1016/0009-9120(95)91425-3.

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Kamel, Mohamed S., Rachel A. Munds, and Mohit S. Verma. "The Quest for Immunity: Exploring Human Herpesviruses as Vaccine Vectors." International Journal of Molecular Sciences 24, no. 22 (2023): 16112. http://dx.doi.org/10.3390/ijms242216112.

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Herpesviruses are large DNA viruses that have long been used as powerful gene therapy tools. In recent years, the ability of herpesviruses to stimulate both innate and adaptive immune responses has led to their transition to various applications as vaccine vectors. This vaccinology branch is growing at an unprecedented and accelerated rate. To date, human herpesvirus-based vectors have been used in vaccines to combat a variety of infectious agents, including the Ebola virus, foot and mouth disease virus, and human immunodeficiency viruses. Additionally, these vectors are being tested as potential vaccines for cancer-associated antigens. Thanks to advances in recombinant DNA technology, immunology, and genomics, numerous steps in vaccine development have been greatly improved. A better understanding of herpesvirus biology and the interactions between these viruses and the host cells will undoubtedly foster the use of herpesvirus-based vaccine vectors in clinical settings. To overcome the existing drawbacks of these vectors, ongoing research is needed to further advance our knowledge of herpesvirus biology and to develop safer and more effective vaccine vectors. Advanced molecular virology and cell biology techniques must be used to better understand the mechanisms by which herpesviruses manipulate host cells and how viral gene expression is regulated during infection. In this review, we cover the underlying molecular structure of herpesviruses and the strategies used to engineer their genomes to optimize capacity and efficacy as vaccine vectors. Also, we assess the available data on the successful application of herpesvirus-based vaccines for combating diseases such as viral infections and the potential drawbacks and alternative approaches to surmount them.

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Dissertations / Theses on the topic "Human herpesviruses"

Taylor-Wiedeman, Jean. "Analysis of human cytomegalovirus in the healthy human carrier." Thesis, Open University, 1992. http://oro.open.ac.uk/57400/.

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Much circumstantial evidence has pointed to peripheral blood leukocytes as one site of persistence of Human Cytomegalovirus (HCMV) in healthy carriers. However, the exact population of peripheral blood cells that carry HCMV and to what extent they express HCMV gene products in not known. I have examined the sites of HCMV persistence in the peripheral blood of healthy carriers. Analysis of pure cell populations by the use of the polymerase chain reaction (PCR), sensitive to between 1 and 10 copies of the HCMV genome, showed that the predominant site of persistence was the monocyte. In addition, analysis of healthy seronegative subjects revealed that a significant number (30%) also harbored HCMV. Finally, study of granulocytes demonstrated no evidence of persistent HCMV. Expression of HCMV during persistence was also analyzed, by using reverse transcription PCR (RT-PCR) with a sensitivity of between 1 and 100 infected fibroblasts. RNA from monocytes showed no evidence of polyadenylated immediate early (IE) or late transcripts. In contrast, in vitro differentiated monocyte-derived macrophages (MDM) did show evidence of HCMV gene expression with the class of HCMV genes expressed dependent on the method of differentiation. MDM treated with hydrocortisone (HC) and phorbol 12-myristate 13-acetate, expressed only lEI, but not IE2, glycoprotein B (gB) or phosphoprotein 28 (pp28) transcripts. Whereas, MDM treated with granulocyte-macrophage colony stimulating factor and HC expressed lEI, IE2 and gB, but not pp28 transcripts. In both cases, cocultivation experiments did not show plaques. Therefore, in the healthy carrier, persistence of HCMV in monocytes is independent of HCMV lytic gene expression, but in vitro differentiation of monocytes to MDM induced endogenous HCMV transcription consistent with the known permissivity of in vivo differentiated macrophages to HCMV infection.

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Chee, Mark. "Analysis of the human cytomegalovirus genome." Thesis, University of Cambridge, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357727.

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Scholes, Andrea Gwendoline Mary. "Effect of human immunodeficiency virus infection on oral shedding of human herpesviruses." Thesis, University of Liverpool, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295830.

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Vargas, Cuero Ana Laura. "Study of CD8'+T lymphocyte responses against human herpesviruses." Thesis, Open University, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342897.

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Baker, Kevin. "Investigations into the ocular involvement of human herpes and papilloma virus infections." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266252.

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Enbom, Malin. "Pathogenesis of the recently identified human herpesviruses 6 and 8 /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4405-9/.

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Strand, Kurt B. "Identification of two distinct lineages of macaque gamma-2 herpesviruses /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/9309.

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NICOLI, Francesco. "Interactions between herpesviruses and the human immune system in old age." Doctoral thesis, Università degli studi di Ferrara, 2022. http://hdl.handle.net/11392/2481326.

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Il processo di invecchiamento è accompagnato da un declino delle funzioni immunitarie che può svolgere un ruolo importante nella bassa efficacia delle vaccinazioni nelle persone anziane. Tuttavia, non è chiaro se esista una cooperazione fra meccanismi intrinseci legati all'età e fattori esterni (come le infezioni con virus persistenti) nella progressione dell'immunosenescenza. Lo scopo di questo lavoro è di studiare l'interazione tra il sistema immunitario dell'ospite e 3 principali herpesvirus (HSV-1, CMV ed EBV). Per raggiungere questo scopo, mi sono concentrato prima sull'effetto delle infezioni persistenti sull'immunosenescenza e, successivamente, sull'effetto dell'immunosenescenza sulle risposte immunitarie alle infezioni. Sono state quindi affrontate 3 domande principali: 1) Le infezioni virali persistenti possono influenzare il compartimento delle cellule T e la risposta immunitaria negli anziani? 2) In che modo l'età influenza le risposte memoria agli herpesvirus? 3) In che modo l'età influenza le risposte alle infezioni primarie (prendendo, a modello, SARS-CoV-2)? Ho inizialmente valutato parametri immunologici relativi ai linfociti T in base allo stato sierologico per i comuni herpesvirus latenti in due coorti indipendenti: 1. individui sani di età compresa tra 19 e 95 anni e 2. individui di età superiore a 70 anni arruolati in uno studio clinico vaccinale. I risultati mostrano, rispettivamente, un effetto prevalente dell'età e dell'infezione da CMV sulle cellule T naive CD8+ e CD4+. Infatti, la sieropositività al CMV è stata associata a una ridotta risposta CD4 e anticorpale in seguito ad una vaccinazione primaria contro encefalite da zecca. La concomitante presenza di diverse infezioni persistenti è inoltre responsabile dell'accumulo (“inflazione”) di cellule T di memoria altamente differenziate nei soggetti più anziani. Quindi, ho misurato le risposte cellulari specifiche per HSV-1, CMV ed EBV in adulti e anziani della Coorte 1, analizzando la capacità secretoria delle diverse sottopopolazioni memoria. In particolare, si è osservato un aumento correlato all'età di cellule T CD8+ di memoria altamente differenziate specifiche per HSV-1, suggerendo che questo virus partecipi all'inflazione del compartimento memoria CD8. Inoltre, si è notato una concomitante diminuzione della capacità secretoria in seguito a stimolazione generale del TCR nelle cellule T CD8+ di memoria poco differenziate. Infine, per confermare se le risposte primarie delle cellule T CD8+ siano difettose in età avanzata ed investigarne le potenziali conseguenze, è stato sfruttato un approccio in vitro per indurre il priming di cellule T CD8+ naive specifiche per SARS-CoV-2 di donatori non esposti al virus, appartenenti a diversi gruppi di età. Rispetto agli adulti più giovani, gli individui anziani mostrano una scarsa capacità di innesco delle cellule T specifiche per SARS-CoV-2 in termini sia di intensità che di qualità della risposta. Inoltre, i soggetti più anziani riconoscono un numero inferiore di epitopi. Nel loro insieme, questi dati suggeriscono fortemente che l'invecchiamento immunitario è associato a risposte primarie alterate. Poiché le cellule T CD8+ virus-specifiche hanno dimostrato di essere protettive nei confronti delle manifestazioni critiche di COVID-19, un'immunità cellulare subottimale può contribuire alla suscettibilità dei soggetti anziani a forme gravi di COVID-19. Inoltre, l'infezione concomitante con CMV influenza negativamente, negli individui anziani, la risposta mediata da CD4 e quindi l'immunità sia cellulare che umorale. Nel complesso, le risposte delle cellule T memoria sono meno influenzate dall'età. Tuttavia, gli herpesvirus causano un’inflazione delle cellule di memoria differenziate che potrebbero avere un impatto negativo sulla memoria immunologica.<br>Advanced age is accompanied by a decline of immune functions, which may play a role in low efficacy of vaccinations in elderly people. However, it is yet unclear at which level age-intrinsic mechanisms and external factors (namely the co-infections with persistent viruses) cooperate, contributing to immunosenescence. The aim of this work was to unravel the age-dependent interplay between the host immune system and 3 main herpesviruses: HSV-1, CMV ad EBV. To reach this scope, I focused on the effect of persistent infections on immunosenescence and of immunosenescence on the immune responses to infections. Three main questions were addressed: 1) May persistent viral infections influence the T-cell compartment and immune responsiveness in older adults? 2) How age affects the memory responses to herpesviruses? 3) How age affects the naïve responses to primary infections (SARS-CoV-2)? To this aim, I first assessed immunological parameters, related to CD8+ and CD4+ T-cell responsiveness, according to the serological status for common latent herpesviruses in two independent cohorts: 1. healthy individuals aged 19y to 95y and 2. individuals above 70y old enrolled in a primo-vaccination clinical trial (n= 137). Results show a prevalent effect of age and CMV infection on CD8+ and CD4+ naive T cells respectively. CMV seropositivity was associated with blunted CD4+ T-cell and antibody responses to primary vaccination against tick-borne encephalitis virus. The concomitant presence of different persistent infections is also responsible of the accumulation (inflation) of late-differentiated memory T cells in older subjects. Then, I measured HSV-, CMV- and EBV-specific cellular responses in middle-aged and elderly adults from Cohort 1 dissecting the secretory capacity of distinct memory subsets. Notably, an age-related increase of late-differentiated, HSV-specific memory CD8+ T cells is observed, suggesting that HSV-1 participate to the memory inflation of the CD8 compartment. In addition, a concomitant decrease of secretory capacity upon general TCR stimulation is noticed in early/intermediate differentiated memory CD8+ T cells. Finally, to confirm whether primary CD8+ T-cell responses are defective in advanced age and assess potential consequences, an in vitro approach to prime SARS-CoV-2-specific naive CD8+ T cells from healthy, unexposed donors of different age groups was exploited. Being an emerging infection, SARS-CoV-2 is the perfect model to study primary responses in unexposed subjects. Compared to younger adults, older individuals display a poor SARS-CoV-2-specific T-cell priming capacity in terms of both magnitude and quality of the response. In addition, older subjects recognize a lower number of epitopes. Collectively, these data implicate that immune ageing is associated with altered primary responses. This implies that elderly subjects present with a low SARS-CoV-2-specific cellular immunity. As virus-specific CD8+ T-cells have shown to be protective toward critical COVID-19 manifestations, an age-related suboptimal cellular immunity may contribute to the age-pattern of the disease. Moreover, the concomitant infection with CMV negatively affects, in elderly individuals, CD4-mediated response and thus both cellular and humoral immunity. This may further explain the reduced responses to emerging infections and de-novo vaccination with advanced age. Overall, recall T-cell responses are less affected by age. Nonetheless, herpesviruses cause an “inflation” of late-differentiated memory cells that could negatively impact on immunological memory towards other antigen specificities (e.g. previously administered vaccines) and is a sign of frequent viral reactivation in life. Altogether, these data provide insights on the changes in adaptive immunity over time and the associated decline in vaccine efficacy with ageing. This knowledge is important for the management of infectious diseases in elderly populations.

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Lepper, Mark William. "Immune modulation by human cytomegalovirus following reactivation in immunocompromised patients." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324251.

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Fok, Yuen-kei Vivien. "Expression of human herpesvirus 6 (HHV-6) genes in virus-infected cells /." View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31385515.

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Books on the topic "Human herpesviruses"

Kawaguchi, Yasushi, Yasuko Mori, and Hiroshi Kimura, eds. Human Herpesviruses. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7230-7.

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Arvin, Ann, Gabriella Campadelli-Fiume, Edward Mocarski, et al., eds. Human Herpesviruses. Cambridge University Press, 2007. http://dx.doi.org/10.1017/cbo9780511545313.

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1929-, Roizman Bernard, Whitley Richard J, and Lopez Carlos 1942-, eds. The Human herpesviruses. Raven Press, 1993.

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M, Arvin Ann, ed. Human herpesviruses: Biology, therapy, and immunoprophylaxis. Cambridge University Press, 2007.

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Becker, Yechiel, and Gholamreza Darai, eds. Pathogenicity of Human Herpesviruses due to Specific Pathogenicity Genes. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-85004-2.

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Yechiel, Becker, and Darai Gholamreza, eds. Pathogenicity of human herpesviruses due to specific pathogenicity genes. Springer-Verlag, 1994.

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Fard, Amir Hossein Mohagheghi. Positivity for and avidity of human herpesviruses IgG antibody determined by ELISA. University of Manchester, 1996.

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Humans, IARC Working Group on the Evaluation of Carcinogenic Risks to. Epstein-Barr virus and Kaposi's sarcoma herpesvirus/human herpesvirus 8. IARC, 1997.

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V, Ablashi D., Krueger G. R. F, and Salahuddin S. Zaki, eds. Human herpesvirus-6: Epidemiology, molecular biology, and clinical pathology. Elsevier, 1992.

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Lopez, Carlos, Ryoichi Mori, Bernard Roizman, and Richard J. Whitley, eds. Immunobiology and Prophylaxis of Human Herpesvirus Infections. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5853-4.

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Book chapters on the topic "Human herpesviruses"

Caserta, Mary T., and Caroline Breese Hall. "Human Herpesviruses: Human Herpesvirus 6." In Viral Infections of Humans. Springer US, 2014. http://dx.doi.org/10.1007/978-1-4899-7448-8_37.

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Tesini, Brenda L., and Mary T. Caserta. "Human Herpesviruses: Human Herpesvirus 6." In Viral Infections of Humans. Springer US, 2023. http://dx.doi.org/10.1007/978-1-4939-9544-8_37-1.

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Labò, Nazzarena, Gianluca Vago, and Giuseppe Gerna. "Human Herpesviruses." In Textbook-Atlas of Intestinal Infections in AIDS. Springer Milan, 2003. http://dx.doi.org/10.1007/978-88-470-2091-7_23.

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Pereira, Lenore. "Glycoproteins Specified by Human Cytomegalovirus." In The Herpesviruses. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2383-9_9.

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Pass, Robert F. "Human Herpesviruses: Cytomegalovirus." In Viral Infections of Humans. Springer US, 2014. http://dx.doi.org/10.1007/978-1-4899-7448-8_35.

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Pass, Robert F. "Human Herpesviruses: Cytomegalovirus." In Viral Infections of Humans. Springer US, 2023. http://dx.doi.org/10.1007/978-1-4939-9544-8_35-2.

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Pass, Robert F. "Human Herpesviruses: Cytomegalovirus." In Viral Infections of Humans. Springer US, 2022. http://dx.doi.org/10.1007/978-1-4939-9544-8_35-1.

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Whitley, Richard J. "A Perspective on the Therapy of Human Herpesvirus Infections." In The Herpesviruses. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4615-8021-8_17.

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Rawls, William E. "Herpes Simplex Viruses and Their Role in Human Cancer." In The Herpesviruses. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2383-9_5.

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Tevethia, Mary J. "Transforming Potential of Herpes Simplex Viruses and Human Cytomegalovirus." In The Herpesviruses. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2383-9_6.

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Conference papers on the topic "Human herpesviruses"

Raposo, Jéssica, Rafaela Pinto, Amanda Lopes, et al. "Investigation of active human herpesviruses 6 and 7 infection before and after renal transplantation." In IV International Symposium on Immunobiologicals & VII Seminário Anual Científico e Tecnológico. Instituto de Tecnologia em Imunobiológicos, 2019. http://dx.doi.org/10.35259/isi.sact.2019_32830.

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Konishcheva, Anna, Valentina Gervazieva та Schodova Svetlana. "Chronic persistence of human γ-herpesviruses and regulatory cells phenotypes in severe bronchial asthma". У ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa568.

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Rattu, Mohammad A., and Wayne Tamaska. "A Case of HHV-6 Viral Meningitis." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.87.

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Meningitis is the inflammation of the meninges and associated with abnormal cell count in the cerebrospinal fluid (CSF). The lack of bacterial growth in cultures, most commonly referred to as aseptic meningitis, is frequently caused by viruses [2]. Viruses have become more common as the prevalence of bacterial meningitis has decreased secondary to vaccination use, with viral meningitis being the most common form among countries. Viral meningitis will present with fever, headache, photophobia, neck stiffness and nausea and vomiting. Younger children however may not show any signs of meningeal irritation. Viral meningitis is usually self-limiting and with good prognosis; however, appropriate evaluation is critical. Enteroviruses such as Coxsackie or Echovirus are the most common cause of viral meningitis across all age groups and parechoviruses are common among children. Herpesviruses leading to meningitis include herpes simplex virus (HSV) 1 and 2, varicella-zoster virus (VZV), cytomegalovirus, Epstein-Barr virus, and human herpesvirus 6. Additional causes include adenovirus, lymphocytic choriomeningitis virus (LCMV), influenza, parainfluenza, and mumps. Arboviruses that can cause viral meningitis include West Nile virus (WNV), Zika, chikungunya, dengue, LaCross, Saint Louis encephalitis, Powassan, and eastern equine encephalitis virus.

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Campos, Leticia, Rafaela Sousa, Gisela Costa, et al. "Detection of human herpesviruses in the oral microbiome of patients with Crohn's disease and ulcerative colitis." In International Symposium on Immunobiological. Instituto de Tecnologia de Imunobiológicos, 2025. https://doi.org/10.35259/isi.2025_70095.

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Touloumes, N., J. Bradley, and M. Saad. "Human Herpesvirus 6 Encephalitis in an Immunocompetent Adult." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a6091.

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Su, Cheng-Chuan, Shih-Ming Tsao, Chun-Liang Lai, Ming-Nan Lin, and Jen-Pi Tsai. "Human herpesvirus type 8 in patients with chronic obstructive pulmonary disease." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3655.

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Reddy, K., R. Bascom, and S. B. Goldfarb. "An Unusual Case of Young Adult Interstitial Pneumonia with Human Herpesvirus 6." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1464.

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Lareau, Caleb A. "1297 Latent human herpesvirus 6 is reactivated in chimeric antigen receptor T cells." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.1297.

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Okada, Y., N. Sasa, S. Kojima, et al. "POS0312 ENDOGENEOUS HUMAN HERPESVIRUS 6B INTEGRATED IN HUMAN GENOME SEQUENCE ASSOCIATES WITH AFFECTION AND SEVERITY OF SYSTEMIC LUPUS ERYTHEMATOSUS." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.1619.

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Chang, Joanne T., Fatma M. Shebl, Ruth M. Pfeiffer, et al. "Abstract 5487: Investigating human herpesvirus 8 infection among adults in Uganda: A factor analysis approach." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5487.

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Reports on the topic "Human herpesviruses"

ภัทรโกศล, ภาวพันธ์, ชิษณุ พันธุ์เจริญ та วรรณา พรรณรักษา. ความชุกของการตรวจพบแอนติบอดีต่อไวรัส human herpesvirus-6 ชนิด IgG ในน้ำเหลืองเด็ก : รายงานผลการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 1999. https://doi.org/10.58837/chula.res.1999.23.

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การศึกษาความชุกของการติดเชื้อ HHV-6 ทำได้โดยการตรวจหาแอนติบอดีจำเพาะต่อเชื้อ HHV-6 ชนิด IgG ด้วยวิธีอีไลซ่า จากตัวอย่างซีรั่มเด็กไทยที่มีสุขภาพแข็งแรงอายุระหว่าง 0-12 ปี (อายุเฉลี่ย 3.35+-3.33 ปี) จำนวน 210 ราย พบมีความชุกของการติดเชื้อ HHV-6 ร้อยละ 88.10 (185/210) ตัวอย่างทั้งหมดเก็บอย่างสุ่มจากผู้ที่มาตรวจสุขภาพที่โรงพยาบาลโดยแบ่งตามช่วงอายุเป็น 7 กลุ่มๆ ละ 30 ตัวอย่าง คือ กลุ่มที่ 1; 0-6 เดือน กลุ่มที่ 2; 6-12 เดือน กลุ่มที่ 3; 12-18 เดือน กลุ่มที่ 4; 18-24 เดือน กลุ่มที่ 5; 2-5 ปี กลุ่มที่ 6; 5-8 ปี และกลุ่มที่ 7; 8-12 พบความชุกของการติดเชื้อ HHV-6 ร้อยละ 63.33, 70, 96.67, 93.33, 100, 100 และ 93.33 ตามลำดับ ค่าเฉลี่ยปริมาณแอนติบอดีจำเพาะต่อ HHV-6 ชนิด IgG ที่อ่านผลบวกในแต่ละกลุ่มจำนวนทั้งสิ้น 185 ตัวอย่าง พบว่าค่าเฉลี่ยปริมาณแอนติบอดีจะเพิ่มสูงจากช่วงอายุ 0-6 เดือน 17.47+-6.32 unit เป็น 27.57+-8.42 unit ในช่วงอายุ 6-12 เดือน สูงสุดช่วงอายุ 18-24 เดือน 33.08+-8.64 unit และจะลดลง โดยค่าเฉลี่ยปริมาณแอนติบอดีในแต่ละกลุ่มมีความแตกต่างกันอย่างมีนัยสำคัญทางสถิติยกเว้นกลุ่มที่ 1 และกลุ่มที่ 7 (p-value = 0.31) ปัจจัยที่เกี่ยวข้องกับการติดเชื้อไวรัส HHV-6 ที่สำคัญคืออายุ (p-value = 0.002) ส่วนปัจจัยอื่นๆ เช่น เพศ เศรษฐานะของครอบครัว จำนวนเด็กในครอบครัว และสถานที่เลี้ยงดูไม่พบมีความสัมพันธ์กับการติดเชื้อ

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Mygatt, Justin G. Pathogenetic Influences of Human Herpesvirus 8 (HHV-8) in Prostate Cancer Progression. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ad1013151.

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