Academic literature on the topic 'Human rhinovirus'

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Journal articles on the topic "Human rhinovirus"

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Lang, Judith, Matthias Soddemann, Michael J. Edwards, Gregory C. Wilson, Karl S. Lang, and Erich Gulbins. "Sphingosine Prevents Rhinoviral Infections." International Journal of Molecular Sciences 25, no. 5 (2024): 2486. http://dx.doi.org/10.3390/ijms25052486.

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Rhinoviral infections cause approximately 50% of upper respiratory tract infections and novel treatment options are urgently required. We tested the effects of 10 μM to 20 μM sphingosine on the infection of cultured and freshly isolated human cells with minor and major group rhinovirus in vitro. We also performed in vivo studies on mice that were treated with an intranasal application of 10 μL of either a 10 μM or a 100 μM sphingosine prior and after infection with rhinovirus strains 1 and 2 and determined the infection of nasal epithelial cells in the presence or absence of sphingosine. Final
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Ėmužytė, Regina, Regina Firantienė, Rasa Petraitytė, and Kęstutis Sasnauskas. "Human rhinoviruses, allergy, and asthma: a clinical approach." Medicina 45, no. 11 (2009): 839. http://dx.doi.org/10.3390/medicina45110109.

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The prevalence of allergic diseases is increasing in Lithuania as in the world. The prevalence of allergic sensitization is often higher than 50% of the population. The “hygiene hypothesis” proposed that reduced immune-stimulation by infections may have resulted in the more widespread clinical expression of atopic disease. However, it alone does not provide an adequate explanation for the observed increase of allergic diseases. Human rhinovirus infections are the major infections with a worldwide distribution. Viral infections of the respiratory tract are the most common triggers of acute asth
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Dee, Kieran, Daniel M. Goldfarb, Joanne Haney, et al. "Human Rhinovirus Infection Blocks Severe Acute Respiratory Syndrome Coronavirus 2 Replication Within the Respiratory Epithelium: Implications for COVID-19 Epidemiology." Journal of Infectious Diseases 224, no. 1 (2021): 31–38. http://dx.doi.org/10.1093/infdis/jiab147.

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Abstract Virus-virus interactions influence the epidemiology of respiratory infections. However, the impact of viruses causing upper respiratory infections on severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) replication and transmission is currently unknown. Human rhinoviruses cause the common cold and are the most prevalent respiratory viruses of humans. Interactions between rhinoviruses and cocirculating respiratory viruses have been shown to shape virus epidemiology at the individual host and population level. Here, we examined the replication kinetics of SARS-CoV-2 in the human
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Stone, Cosby A., and E. Kathryn Miller. "Understanding the Association of Human Rhinovirus with Asthma." Clinical and Vaccine Immunology 23, no. 1 (2015): 6–10. http://dx.doi.org/10.1128/cvi.00414-15.

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ABSTRACTHuman rhinoviruses are ubiquitous seasonal pathogens. They have known associations with first onset of wheezing illnesses in children and with asthma exacerbations in patients of all ages. It is not yet certain whether human rhinoviruses play a direct role in the pathogenesis of asthma by activating deleterious inflammatory responses or if they only serve as a catalyst to accelerate the disease in genetically predisposed individuals. There have been previously demonstrated reductions in the development of the asthmatic phenotype with passive immunization against respiratory syncytial v
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Lau, Susanna, Cyril Yip, Patrick Woo, and Kwok-Yung Yuen. "Human rhinovirus C: a newly discovered human rhinovirus species." Emerging Health Threats Journal 3, no. 1 (2010): 7106. http://dx.doi.org/10.3402/ehtj.v3i0.7106.

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Mitra, Manu. "Rhinovirus Physiognomies." SunText Review of Virology 2, no. 2 (2021): 1–3. https://doi.org/10.51737/2766-5003.2021.023.

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Rhinovirus is one of the contagious viral agents in humans and is the principal cause of common cold. Rhinovirus belongs to the genus Enterovrirus in the family Picornaviridae. Rhinovirus are alienated as three standard classes (A, B and C) that includes 160 recognized types of human rhinovirus that differ according to their surface proteins (serotypes). They are lytic in nature and are one of the among the smallest viruses with diameter of about 30 nanometers. Comparatively with other viruses like smallpox and vaccinia are around ten times larger i.e. around 300 nanometers, while flu viruses
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Yamaya, Mutsuo, Hidekazu Nishimura, Yukimasa Hatachi, et al. "Levofloxacin Inhibits Rhinovirus Infection in Primary Cultures of Human Tracheal Epithelial Cells." Antimicrobial Agents and Chemotherapy 56, no. 8 (2012): 4052–61. http://dx.doi.org/10.1128/aac.00259-12.

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ABSTRACTRespiratory virus infections, including infections with rhinoviruses (RVs), are related to exacerbations of chronic obstructive pulmonary disease (COPD). A new quinolone antibiotic, levofloxacin (LVFX), has been used to treat bacterial infections that cause COPD exacerbations as well as bacterial infections that are secondary to viral infection in COPD patients. However, the inhibitory effects of LVFX on RV infection and RV infection-induced airway inflammation have not been studied. We examined the effects of LVFX on type 14 rhinovirus (RV14) (a major human RV) infection of human trac
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Ksenafontov, Andrey D., Maria M. Pisareva, Veronica A. Eder, Tamila D. Musaeva, and Irina V. Kiseleva. "Research of the genetic diversity of human rhinoviruses on the territory of Saint Petersburg 2021–2022." Medical academic journal 2, no. 2 (2022): 89–96. http://dx.doi.org/10.17816/maj108734.

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BACKGROUND: Respiratory viruses circulate everywhere. Rhinoviruses are the most common cause of human upper respiratory tract infections. Therefore, it is necessary to study circulation of their species and types.
 AIM: To study circulation of different species and types of rhinoviruses in Saint Petersburg.
 MATERIALS AND METHODS: Detection of rhinoviruses was carried out by real-time PCR using commercial kits AmpliSens ORVI-screen-FL (Central Research Institute of Epidemiology of Rospotrebnadzor, Moscow); the study of the genetic diversity of rhinoviruses was carried out by Sanger s
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Staunton, D. E., A. Gaur, P. Y. Chan, and T. A. Springer. "Internalization of a major group human rhinovirus does not require cytoplasmic or transmembrane domains of ICAM-1." Journal of Immunology 148, no. 10 (1992): 3271–74. http://dx.doi.org/10.4049/jimmunol.148.10.3271.

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Abstract Intercellular adhesion molecule-1 (CD54), a cell adhesion molecule and the receptor for the major group of rhinoviruses, is a class 1 membrane protein with five Ig-like domains in its extracellular region, a transmembrane domain, and a short cytoplasmic domain. The amino-terminal domains (D1 and D2) are sufficient for virus binding and the first is most important (1). We have investigated whether other extracellular domains, transmembrane or cytoplasmic domains are required for virus entry as determined by postinfection virion protein biosynthesis. We demonstrate that cytoplasmic, tra
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Sittek, Lisa-Marie, Thomas Michael Schmidts, and Peggy Schlupp. "Ingredients Acting as a Physical Barrier for the Prevention and Treatment of the Rhinovirus Infection." Applied Sciences 10, no. 18 (2020): 6511. http://dx.doi.org/10.3390/app10186511.

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Although the common cold, usually caused by human rhinoviruses, is responsible for enormous damage to the economy and health every year, there are hardly any treatment options or prophylaxis for rhinovirus infections. In this work, the potential of a hydrogel complex, based on polymers and an aqueous extract of Icelandic moss in isla® medic lozenges (Engelhard Arzneimittel, Niederdorfelden, Germany), and two other hydrogels (based on solely xanthan gum or sodium hyaluronate) are investigated for the first time in order to prevent and treat rhinovirus infections. By means of rheological investi
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Dissertations / Theses on the topic "Human rhinovirus"

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George, S. N. "Human rhinovirus at naturally occurring COPD exacerbation." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1462147/.

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Chronic obstructive pulmonary disease (COPD) is an inflammatory condition of the lung caused by an abnormal response to particles and noxious gases, primarily cigarette smoke. Patients suffer daily symptoms and can have episodes of worsening symptoms termed acute exacerbations. Exacerbations are associated with impaired quality of life, faster lung function decline, higher mortality and increased risk of hospitalisation. The aetiology of COPD exacerbations is controversial; however respiratory viral and bacterial infections are an important feature of exacerbations. This study utilised real-ti
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Braesch-Andersen, Ken. "Temperature dependence in human Rhinovirus infection of human MRC-5." Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-392331.

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Temperature has been known to be an important factor for in vitro studies where human cell cultures are infected with HRV (human Rhinovirus). The mechanisms behind the temperature effect on the struggle between virulence and cellular defense, are still largely unknown and may be a crucial part in finding a treatment to the common cold. In this study we focused on a few cellular key elements in this struggle and observed behavior changes in regards to the pre-infection growth temperature and the temperature during the viral infection. Past studies have focused mainly on the temperature post ino
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Borman, Andrew Mark. "Internal initiation of translation of human rhinovirus RNA." Thesis, University of Cambridge, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.281908.

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Watson, David G. "Isolation and characterization of human rhinovirus antigenic variants /." The Ohio State University, 1985. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487260135355837.

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Leckie, Gregor Wheelwright. "Molecular studies on a human common cold virus, human rhinovirus nine." Thesis, University of Leicester, 1987. http://hdl.handle.net/2381/35391.

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The molecular cloning of the common cold virus, rhinovirus 9, was achieved using the hybrid (RNA:CDNA) cloning protocol first described by Wood and Lee (1976). Two experiments were required to clone the complete genome; the first was primed by poly dT17 while the second was primed by an oligonucleotide which annealed to a sequence in the P2 region of the genome. Plasmids containing cDNA representative of the rhinovirus 9 genome were identified by in situ hybridisation. Such plasmids were characterised by the restriction endonuclease mapping of their cDNA inserts. This analysis indicated that p
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McIntyre, Chloe Leanne. "Epidemiology, classification and evolution of human rhinoviruses." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/8197.

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Human rhinoviruses (HRV) are extremely common human respiratory pathogens, most commonly associated with mild upper respiratory tract infections. The three known species of HRV (HRV-A, -B and –C) are members of the family Picornaviridae and genus Enterovirus. In contrast to the enterovirus (EV-A-D) species that commonly infect the gut, HRV are generally thought to be acid labile with replication restricted to the respiratory tract. Investigations of the clinical correlations of HRV infections detected on diagnostic screening of respiratory specimens demonstrated no specific association between
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Brown, E. C. "Cellular proteins involved in translation of human rhinovirus RNA." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596963.

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Translation of picornavirus RNA takes place by internal initiation, determined by the presence of an internal ribosome entry site (IRES) in the 5'-untranslated region of the genomic RNA. Efficient translation from the human rhinovirus-2 (HRV-2) IRES is dependent on host cell <I>trans</I>-acting factors. These include unr, p38 and polypyrimidine tract binding protein (PTB). This thesis details the investigation into how these factors act to promote translation from the HRV-2 IRES. Unr, an RNA-binding protein with five cold-shock domains (CSDs), binds to the HRV-2 IRES and this interaction was s
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Wimalasundera, Sunethra Surangani. "The characterisation of a T cell response to human rhinovirus." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267147.

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Byrne, Emma Jane. "The activity of human rhinovirus 14 3C protease in artificial polyproteins." Thesis, University of St Andrews, 1999. http://hdl.handle.net/10023/14310.

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HRV14 3C acts as a protease and has a role in RNA replication in vivo, interacting with a cloverleaf structure in picornaviral genomic RNA. Picornaviral 3C proteases are able to cleave both N- and C-terminally producing 3CDpro, or, 3Cpro and 3DPol, respectively. In order to investigate the mechanisms whereby these alternative processing pathways are adopted an artificial polyprotein system was constructed, composed of viral sequences from the P3 region of the viral polyprotein flanked by reporter genes. Two antibiotic resistance genes (KanR, TetR) were cloned to act as reporter genes flanking
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Thomas, Lynette Hazel. "The effect of respiratory viral infections on human circulatory leukocytes." Thesis, University of Southampton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.242504.

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Books on the topic "Human rhinovirus"

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Schreiber, Michael Thomas. Determinants of Human Rhinovirus Cellular Tropism in Monocyte-Lineage Cells. [publisher not identified], 2016.

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Bartlett, Nathan, Peter Wark, and Darryl Knight. Rhinovirus Infections: Rethinking the Impact on Human Health and Disease. Elsevier Science & Technology Books, 2019.

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Bartlett, Nathan, Peter Wark, and Darryl Knight. Rhinovirus Infections: Rethinking the Impact on Human Health and Disease. Elsevier Science & Technology, 2019.

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Fenaux, Martijn. Alkynyl Nucleoside Analogs As Inhibitors of Human Rhinovirus: United States Patent 9988416. Independently Published, 2020.

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Newton, Pippa. Upper respiratory tract infections, including influenza. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0128.

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Infections of the nasal cavity, sinuses, pharynx, epiglottis, and larynx are termed upper respiratory tracts infections. These include acute coryza, pertussis, sinusitis, pharyngitis, tonsillitis, epiglottitis, laryngitis, laryngotracheobronchitis, and influenza. Rhinoviruses and coronaviruses account for the majority of acute coryzal illnesses. Acute sinusitis (&lt;4 weeks duration) is also usually viral in origin. About 70% of pharyngitis and tonsillitis cases are viral in etiology. Haemophilus influenzae (Type B) is responsible for most cases of epiglottitis. Acute laryngitis and laryngotra
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Book chapters on the topic "Human rhinovirus"

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Blaas, Dieter. "Human Rhinovirus Minor Group Receptors." In Molecular Biology of Picornavirus. ASM Press, 2014. http://dx.doi.org/10.1128/9781555817916.ch9.

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Singh, Anne Marie, and William W. Busse. "Human Rhinovirus Models in Asthma." In Contributions to Microbiology. KARGER, 2007. http://dx.doi.org/10.1159/000107051.

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Jensen, Lora M., Erin J. Walker, David A. Jans, and Reena Ghildyal. "Proteases of Human Rhinovirus: Role in Infection." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1571-2_10.

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Tomusange, Khamis, Danushka Wijesundara, Eric James Gowans, and Branka Grubor-Bauk. "Human Rhinovirus-A1 as an Expression Vector." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6869-5_11.

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Kastner, Markus, Christian Rankl, Andreas Ebner, et al. "Nanoimaging, Molecular Interaction, and Nanotemplating of Human Rhinovirus." In Scanning Probe Microscopy in Nanoscience and Nanotechnology 2. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-10497-8_21.

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Lee, Wai-Ming, Wensheng Wang, Yury A. Bochkov, and Iris Lee. "Reverse Genetics System for Studying Human Rhinovirus Infections." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1571-2_12.

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Lee, Wai-Ming, Yin Chen, Wensheng Wang, and Anne Mosser. "Infectivity Assays of Human Rhinovirus-A and -B Serotypes." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1571-2_7.

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Dutko, Frank J., Mark A. McKinlay, and Michael G. Rossmann. "Antiviral Compounds Bind to a Specific Site Within Human Rhinovirus." In Concepts in Viral Pathogenesis III. Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4613-8890-6_39.

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Wasik, Bethany R., Brian R. Wasik, Ellen F. Foxman, Akiko Iwasaki, and Paul E. Turner. "Experimental Evolution of Human Rhinovirus Strains Adapting to Mouse Cells." In Evolution in Action: Past, Present and Future. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39831-6_12.

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Smith, Thomas J., John Badger, Marcia Kremer, et al. "Crystallographic and Pharmacological Studies of Antiviral Agents Against Human Rhinovirus." In Crystallographic and Modeling Methods in Molecular Design. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3374-9_2.

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Conference papers on the topic "Human rhinovirus"

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Chughtai, N., J. Madsen, and H. Clark. "Collectin Interaction With Human Rhinovirus." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4194.

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Iwasaki, Jua, Wendy-Anne Smith, Wayne R. Thomas, and Belinda J. Hales. "Sero-Epidemiology Of Human Rhinovirus C." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3293.

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Numata-Nakamura, M., H. Lee, H. W. Chu, M. A. Seibold, and D. R. Voelker. "Pulmonary Surfactant Lipids Antagonize Human Rhinovirus Infections." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5757.

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Yang, A., L. Daly, Y. Yamamoto, J. Baker, and K. Ito. "S113 Cellular senescence ameliorates human rhinovirus clearance." In British Thoracic Society Winter Meeting 2023, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 22 to 24 November 2023, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2023. http://dx.doi.org/10.1136/thorax-2023-btsabstracts.119.

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Tomilova, Yulia, Yulia Khvorostova, Mikhail Ivanov, et al. "Complex PCR-diagnostics of respiratory infections in A children's hospital." In Proceedings of the International Congress Public Health - Achievements and Challenges. Institute of Public Health of Serbia "Dr Milan Jovanović Batut", 2024. http://dx.doi.org/10.5937/batutphco24027t.

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Background: Accurate differential etiological diagnosis allows to select the right therapy or adjust the course of treatment. The aim of the study was to analyze the etiological structure of childhood respiratory infections of viral and bacterial origin using the PCR method. Methods and Objectives: Nasoropharyngeal swabs of children admitted to hospital with symptoms of respiratory disease were examined (n=1443). The analysis was carried out using kits for DNA/RNA extraction and PCR kits "RealBest" (AO "Vector-Best"), designed to detect DNA/RNA of influenza A and B viruses, parainfluenza types
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Smith, Claire, Priya Radhakrishnan, Dani DoHyang Lee, Edith M. Hessel, Rick Williamson, and Chris O'Callaghan. "Rhinovirus infection of human ciliated respiratory epithelial cultures." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2608.

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Pelikan, Jonathan B., Shahina Wiehler, Raza S. Zaheer, and David Proud. "Mechanisms Of Human Rhinovirus-Induced Viperin Expression In Human Airway Epithelial Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5384.

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Yamaya, M., M. Asada, M. Yoshida, Y. Hatachi, H. Kubo, and H. Nishimura. "Ambroxol Inhibits Rhinovirus Infection in Human Airway Epithelial Cells." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5945.

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Yamaya, Mutsuo, Yukimasa Hatachi, Hiroshi Kubo, and Hidekazu Nishimura. "Tulobuterol Inhibits Rhinovirus Infection In Human Airway Epithelial Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4453.

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ILARRAZA, RAMSES, YINGQI WU, and DARRYL J. ADAMKO. "Rhinovirus Has Unique Properties To Activate Human T-Cells." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5337.

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