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Relevant bibliographies by topics / Human skeletal muscle

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Academic literature on the topic 'Human skeletal muscle'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 March 2023

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Journal articles on the topic "Human skeletal muscle"

1

Sandage, Mary J., and Audrey G. Smith. "Muscle Bioenergetic Considerations for Intrinsic Laryngeal Skeletal Muscle Physiology." Journal of Speech, Language, and Hearing Research 60, no. 5 (May 24, 2017): 1254–63. http://dx.doi.org/10.1044/2016_jslhr-s-16-0192.

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PurposeIntrinsic laryngeal skeletal muscle bioenergetics, the means by which muscles produce fuel for muscle metabolism, is an understudied aspect of laryngeal physiology with direct implications for voice habilitation and rehabilitation. The purpose of this review is to describe bioenergetic pathways identified in limb skeletal muscle and introduce bioenergetic physiology as a necessary parameter for theoretical models of laryngeal skeletal muscle function.MethodA comprehensive review of the human intrinsic laryngeal skeletal muscle physiology literature was conducted. Findings regarding intrinsic laryngeal muscle fiber complement and muscle metabolism in human models are summarized and exercise physiology methodology is applied to identify probable bioenergetic pathways used for voice function.ResultsIntrinsic laryngeal skeletal muscle fibers described in human models support the fast, high-intensity physiological requirements of these muscles for biological functions of airway protection. Inclusion of muscle bioenergetic constructs in theoretical modeling of voice training, detraining, fatigue, and voice loading have been limited.ConclusionsMuscle bioenergetics, a key component for muscle training, detraining, and fatigue models in exercise science, is a little-considered aspect of intrinsic laryngeal skeletal muscle physiology. Partnered with knowledge of occupation-specific voice requirements, application of bioenergetics may inform novel considerations for voice habilitation and rehabilitation.

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Ahmed, A., D. L. Maxwell, P. M. Taylor, and M. J. Rennie. "Glutamine transport in human skeletal muscle." American Journal of Physiology-Endocrinology and Metabolism 264, no. 6 (June 1, 1993): E993—E1000. http://dx.doi.org/10.1152/ajpendo.1993.264.6.e993.

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Sarcolemmal vesicles isolated from human skeletal muscle obtained at surgery showed approximately 14-fold enrichment of sarcolemmal marker enzymes 5'-nucleotidase and K-stimulated phosphatase. [3H]glutamine transport in these vesicles was stereospecific, largely Na dependent, and tolerated Li-for-Na substitution. Glutamine transport was stimulated by an inside negative membrane potential, and 25 mM glutamine stimulated 22Na (0.1 mM) uptake into vesicles by 50%, indicating rheogenic cotransport of Na and glutamine. Alanine transport was Na dependent but did not tolerate Li-for-Na substitution. Transport of L-[3H]glutamine was inhibited by 35-65% with a 20-fold excess of glutamine, asparagine, and alanine; cysteine, alpha-(methylamino)isobutyrate, and 2-amino-2-norborane carboxylic acid had smaller inhibitory effects, although cysteine had an unusually large inhibitory effect on glutamine transport at 1,000-fold excess compared with most other amino acids. Glutamine transport showed sensitivity to pH values < 7.0. Glutamine transport consisted of a Na-dependent and a Na-independent component, both of which appeared saturable. The kinetic characteristics of the Na-dependent component were different in different types of muscles, with half-maximal concentrations (mM) varying from 1.6 +/- 0.4 (tibialis anterior) to 0.56 +/- 0.0.2 (gluteus maximus) and maximal velocity (pmol.mg protein-1.s-1) of 1.3 +/- 0.27 to 5 +/- 1.25 in the same muscles. The results demonstrate both marked similarities and important differences between the principal glutamine transporter in human skeletal muscle and the known system Nm transporter in rat skeletal muscle.

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Norheim, Frode, Truls Raastad, Bernd Thiede, Arild C. Rustan, Christian A. Drevon, and Fred Haugen. "Proteomic identification of secreted proteins from human skeletal muscle cells and expression in response to strength training." American Journal of Physiology-Endocrinology and Metabolism 301, no. 5 (November 2011): E1013—E1021. http://dx.doi.org/10.1152/ajpendo.00326.2011.

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Regular physical activity protects against several types of diseases. This may involve altered secretion of signaling proteins from skeletal muscle. Our aim was to identify the most abundantly secreted proteins in cultures of human skeletal muscle cells and to monitor their expression in muscles of strength-training individuals. A total of 236 proteins were detected by proteome analysis in medium conditioned by cultured human myotubes, which was narrowed down to identification of 18 classically secreted proteins expressed in skeletal muscle, using the SignalP 3.0 and Human Genome Expression Profile databases together with a published mRNA-based reconstruction of the human skeletal muscle secretome. For 17 of the secreted proteins, expression was confirmed at the mRNA level in cultured human myotubes as well as in biopsies of human skeletal muscles. RT-PCR analyses showed that 15 of the secreted muscle proteins had significantly enhanced mRNA expression in m. vastus lateralis and/or m. trapezius after 11 wk of strength training among healthy volunteers. For example, secreted protein acidic and rich in cysteine, a secretory protein in the membrane fraction of skeletal muscle fibers, was increased 3- and 10-fold in m. vastus lateralis and m. trapezius, respectively. Identification of proteins secreted by skeletal muscle cells in vitro facilitated the discovery of novel responses in skeletal muscles of strength-training individuals.

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Maganaris, Constantinos N., Vasilios Baltzopoulos, D. Ball, and Anthony J. Sargeant. "In vivo specific tension of human skeletal muscle." Journal of Applied Physiology 90, no. 3 (March 1, 2001): 865–72. http://dx.doi.org/10.1152/jappl.2001.90.3.865.

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In this study, we estimated the specific tensions of soleus (Sol) and tibialis anterior (TA) muscles in six men. Joint moments were measured during maximum voluntary contraction (MVC) and during electrical stimulation. Moment arm lengths and muscle volumes were measured using magnetic resonance imaging, and pennation angles and fascicular lengths were measured using ultrasonography. Tendon and muscle forces were modeled. Two approaches were followed to estimate specific tension. First, muscle moments during electrical stimulation and moment arm lengths, fascicular lengths, and pennation angles during MVC were used ( data set A). Then, MVC moments, moment arm lengths at rest, and cadaveric fascicular lengths and pennation angles were used ( data set B). The use of data set B yielded the unrealistic specific tension estimates of 104 kN/m2 in Sol and 658 kN/m2 in TA. The use of data set A, however, yielded values of 150 and 155 kN/m2 in Sol and TA, respectively, which agree with in vitro results from fiber type I-predominant muscles. In fact, both Sol and TA are such muscles. Our study demonstrates the feasibility of accurate in vivo estimates of human muscle intrinsic strength.

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Hansen, J., G. D. Thomas, T. N. Jacobsen, and R. G. Victor. "Muscle metaboreflex triggers parallel sympathetic activation in exercising and resting human skeletal muscle." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 6 (June 1, 1994): H2508—H2514. http://dx.doi.org/10.1152/ajpheart.1994.266.6.h2508.

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Activation of a metabolically generated reflex in exercising skeletal muscle (muscle metaboreflex) in humans is known to trigger increases in sympathetic nerve activity (SNA) to resting skeletal muscles. In seven healthy human subjects, to determine whether this reflex mechanism also increases SNA to the exercising muscles, we recorded muscle SNA with microelectrodes in the right peroneal nerve and in fascicles of the left peroneal nerve selectively innervating the exercising muscles of the left foot. Subjects performed static toe extension at 20% maximal voluntary contraction alone or in combination with foot ischemia. Only static toe extension at 20% MVC during ischemia activated the muscle metaboreflex. This paradigm caused increases in SNA to exercising muscle that paralleled those to the resting muscles: during the first minute of exercise SNA was unchanged, but during the second minute SNA increased from 29 +/- 2 to 38 +/- 2 bursts/min (P < 0.05) to the exercising muscles and from 30 +/- 3 to 40 +/- 2 bursts/min (P < 0.05) to the resting muscles. These bilateral increases in SNA were maintained when metaboreflex activation was sustained by postexercise foot ischemia. In conclusion, these data provide neurophysiological evidence that the muscle metaboreflex evokes parallel sympathetic activation in exercising and resting human skeletal muscle.

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Tirrell, T. F., M. S. Cook, J. A. Carr, E. Lin, S. R. Ward, and R. L. Lieber. "Human skeletal muscle biochemical diversity." Journal of Experimental Biology 215, no. 15 (July 11, 2012): 2551–59. http://dx.doi.org/10.1242/jeb.069385.

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Tirrell, T. F., M. S. Cook, J. A. Carr, E. Lin, S. R. Ward, and R. L. Lieber. "Human skeletal muscle biochemical diversity." Journal of Experimental Biology 215, no. 16 (July 25, 2012): 2931. http://dx.doi.org/10.1242/jeb.077347.

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Vicart, S., D. Sternberg, B. Fontaine, and G. Meola. "Human skeletal muscle sodium channelopathies." Neurological Sciences 26, no. 4 (October 2005): 194–202. http://dx.doi.org/10.1007/s10072-005-0461-x.

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Rasmussen, U. F., and H. N. Rasmussen. "Human skeletal muscle mitochondrial capacity." Acta Physiologica Scandinavica 168, no. 4 (April 2000): 473–80. http://dx.doi.org/10.1046/j.1365-201x.2000.00699.x.

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Zhang, Tan, Xin Feng, Bo Feng, Juan Dong, Karen Haas, Barbara M. Nicklas, Osvaldo Delbono, and Stephen Kritchevsky. "CARDIAC TROPONIN T MEDIATED AUTOIMMUNE RESPONSE AND ITS ROLE IN SKELETAL MUSCLE AGING." Innovation in Aging 3, Supplement_1 (November 2019): S882. http://dx.doi.org/10.1093/geroni/igz038.3231.

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Abstract Cardiac troponin T (cTnT), a key component of contractile machinery essential for muscle contraction, is also expressed in skeletal muscle under certain conditions (e.g. neuromuscular diseases and aging). We have reported that skeletal muscle cTnT regulates neuromuscular junction denervation preferentially in fast skeletal muscle of old mice. Here, we further report that cTnT is also enriched within some myofibers, and/or along microvascular walls in old mice fast skeletal muscle. Strikingly, immunoglobulin G (IgG), together with markers of complement system activation, cell death (necroptosis or apoptosis), and macrophage infiltration, were all found to be co-localized with cTnT and IgG in those areas. In addition, elevated cTnT and IgG are associated with lower dystrophin expression on muscle fiber membrane, lower muscle capillary density, and reduced muscle performance (wire hanging test). Using purified recombinant TnT proteins, we confirmed that only cTnT, but not slow or fast skeletal muscle TnT1 or TnT3, was detected by immunoblot using sera from old (but not young) mice with pre-determined elevated cTnT and IgG in their skeletal muscle, indicating the existence of anti-cTnT autoantibodies in sera (previously found in human blood) and skeletal muscle of old mice. Immunoblotting further revealed that the age related changes in skeletaI muscle cTnT and IgG are more prominent in fast skeletal muscle than in slow. Importantly, elevated cTnT and IgG were also detected in skeletal muscles from 4 older adults (65-70 yrs, IMFIT). Our finding suggests a novel autoimmune mechanism mediated by cTnT that underlies age related skeletal muscle abnormalities and dysfunction.

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Dissertations / Theses on the topic "Human skeletal muscle"

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Tallon, Mark J. "Carnosine metabolism in human skeletal muscle." Thesis, University of Chichester, 2005. http://eprints.chi.ac.uk/843/.

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Adamo, Kristi Bree. "Proglycogen and macroglycogen in human skeletal muscle." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ31807.pdf.

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Saxton, John Michael. "Exercise-induced damage to human skeletal muscle." Thesis, University of Wolverhampton, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.385185.

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Hurel, Steven J. "Insulin action in cultured human skeletal muscle." Thesis, University of Newcastle Upon Tyne, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363891.

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Stephens, Francis B. "Carnitine transport and metabolism in human skeletal muscle." Thesis, University of Nottingham, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.430645.

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Pickersgill, Laura. "Lipid-induced insulin resistance in human skeletal muscle." Thesis, University of Newcastle Upon Tyne, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413955.

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Kennedy, Paul. "Magnetic resonance elastography studies of human skeletal muscle." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25776.

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A robust, reliable method to non-invasively measure in-vivo mechanical properties of large tissue areas was unavailable until the advent of a new Magnetic Resonance Imaging (MRI) technique known as Magnetic Resonance Elastography (MRE). MRE is a phase-contrast imaging technique that enables quantification of tissue mechanical properties by capturing the motion of induced shear waves via a synchronised Motion Encoding Gradient (MEG). The complex shear modulus is determined via mathematical inversion and reported as the magnitude of the complex shear modulus, |G*|, and phase angle, φ. The work reported in this thesis focuses on the development of MRE data acquisition and analysis protocols optimised to study thigh muscle mechanical properties. The protocols are subsequently applied in healthy volunteers to study natural phenomena such as contraction and ageing and interventions such as an experimental protocol to produce Exercise Induced Muscle Damage (EIMD). Methodological advances made throughout this work include moving from 2D to 3D MRE data acquisition protocols and the application of advanced inversion software to extract muscle viscoelastic properties from the acquired MRE data. Results obtained include observation of reduced muscle stiffness in 6 elderly subjects (>80 years old) compared to 4 young subjects in the Vastus Lateralis (32%), quadriceps muscle group (22%) and entire thigh cross-section (19%), higher resting stiffness of agonist quadriceps compared to antagonist hamstrings (18%) and an increase in quadriceps stiffness (40%) during a leg raise task in 11 healthy subjects. Variability in muscle group recruitment patterns during the contraction were also observed, with the phase angle of the Vastus Intermedius (VI) increasing significantly during contraction. The final experiment involved the recruitment of 20 healthy male subjects to perform an eccentric exercise protocol designed to induce EIMD. Subjects who displayed a Maximum Voluntary Contraction (MVC) force deficit of >10% were considered to have experienced EIMD. A further severe EIMD group were defined based on the presence of hyper-intense signal on T2 weighted imaging following the protocol. The T2 hyper-intensity was found to occur exclusively in the Rectus Femoris (RF) and VI muscle groups. Increased muscle stiffness was observed in the RF muscle in subjects who experienced moderate EIMD (6%). A greater increase in RF stiffness (48%) was observed in the severe EIMD group. The severe EIMD group also displayed significantly increased VI stiffness (14%). The experiments carried out provide several novel findings which can support the development of beneficial strategies to promote both healthy ageing and rehabilitation in athletes, and potentially contribute to improving muscle performance evaluation tests which will expand the opportunities for clinical applications of muscle MRE.

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Crowther, Gregory John. "An analysis of metabolic fluxes in contracting human skeletal muscle /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/10538.

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Kosek, David J. "Aging differences in mechanisms of human skeletal muscle hypertrophy." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2009r/kosek.pdf.

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O'Leary, Mary Frances. "The role of adipose and skeletal muscle derived cytokines in primary human myogenesis : implications for ageing skeletal muscle." Thesis, University of Birmingham, 2018. http://etheses.bham.ac.uk//id/eprint/8089/.

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Sarcopenia is the age-related loss of skeletal muscle mass and function; inflammation is thought to be one aetiological factor in its development. Adipose tissue accumulates with advancing age and adipose-derived cytokines (adipokines) contribute to inflammaging. Skeletal muscle myogenesis is one adaptaive mechanism by which skeletal muscle mass is sustained throughout the human lifespan. The effect of the adipose inflammatory milieu on such myogenesis is unknown, as is the relative importance of its constituent adipokines to myogenesis. This work demonstrates that conditioned medium generated from obese subcutaneuous adipose tissue has a detrimental effect on in vitro primary human myogenesis. Resistin is shown to be – in part – responsible for this phenomenon and is demonstrated to inhibit myogenesis by activating the classical NFκB pathway. Resistin is further shown to be a metabolic stressor of primary human myotubes, promoting increased oxygen consumption, fatty acid oxidation and lipid accumulation. It is important to identify more avenues for the development of pharmacological interventions in sarcopenia. To that end, this thesis also demonstrates for the first time that the myokine IL-15: 1) is pro-myogenic in primary human cultures; 2) can mitigate the detrimental effects of an inflammatory environment on myogenesis; and 3) supports myogenesis at autocrine concentrations.

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Books on the topic "Human skeletal muscle"

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A, Stone Judith, ed. Atlas of the skeletal muscles. Dubuque, Iowa: Wm. C. Brown, 1989.

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Kinesiology: The skeletal system and muscle function. 2nd ed. St. Louis, Mo: Mosby/Elsevier, 2011.

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A, Stone Judith, ed. Atlas of skeletal muscles. 3rd ed. Boston: McGraw-Hill, 2000.

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Stone, Robert J. Atlas of skeletal muscles. 6th ed. Boston: McGraw-Hill Higher Education, 2009.

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A, Stone Judith, ed. Atlas of skeletal muscles. 6th ed. Boston: McGraw-Hill Higher Education, 2009.

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Stone, Robert J. Atlas of skeletal muscles. 2nd ed. Dubuque, IA: Wm. C. Brown Publishers, 1997.

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Stone, Robert J. Atlas of skeletal muscles. 6th ed. Boston: McGraw-Hill Higher Education, 2009.

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M, Bowden Joan, ed. An illustrated atlas of the skeletal muscles. 3rd ed. Englewood, CO: Morton, 2010.

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Child, R. B. Exercise and free radical induced damage to human skeletal muscle. Wolverhampton: University of Wolverhampton, 1997.

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Joan, Bowden, ed. An illustrated atlas of the skeletal muscles. 2nd ed. Englewood, CO: Morton, 2005.

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Book chapters on the topic "Human skeletal muscle"

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van der Ven, Peter F. M. "Skeletal Muscle." In Human Cell Culture, 65–101. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/0-306-46870-0_5.

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Ernst, Linda M., and Patrick Shannon. "Skeletal Muscle." In Color Atlas of Human Fetal and Neonatal Histology, 367–74. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11425-1_33.

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Windhorst, U., and W. F. H. M. Mommaerts. "Physiology of Skeletal Muscle." In Comprehensive Human Physiology, 911–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60946-6_46.

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Yamada, Hiroshi, and Eiichi Tanaka. "Active Stress Models of Cardiac Muscle, Smooth Muscle and Skeletal Muscle." In Human Biomechanics and Injury Prevention, 161–66. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-66967-8_21.

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Görlach, G., H. H. Scheld, J. Mulch, J. Schaper, and F. W. Hehrlein. "Ultrastructure of the Human Myocardium after Intermittent Ischemia Compared to Cardioplegia." In Myocardial and Skeletal Muscle Bioenergetics, 439–49. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5107-8_33.

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Shortland, Adam. "Skeletal Muscle Structure in Spastic Cerebral Palsy." In Handbook of Human Motion, 1075–89. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-14418-4_51.

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Shortland, Adam. "Skeletal Muscle Structure in Spastic Cerebral Palsy." In Handbook of Human Motion, 1–15. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-30808-1_51-1.

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Abou-Khalil, Rana, Fabien Le Grand, and Bénédicte Chazaud. "Human and Murine Skeletal Muscle Reserve Cells." In Stem Cell Niche, 165–77. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-508-8_14.

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Clarke, Mark S. F. "Skeletal Muscle Culture Under Spaceflight Conditions." In Effect of Spaceflight and Spaceflight Analogue Culture on Human and Microbial Cells, 151–74. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3277-1_8.

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Shansky, Janet, Paulette Ferland, Sharon McGuire, Courtney Powell, Michael DelTatto, Martin Nackman, James Hennessey, and Herman H. Vandenburgh. "Tissue Engineering Human Skeletal Muscle for Clinical Applications." In Culture of Cells for Tissue Engineering, 239–57. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2006. http://dx.doi.org/10.1002/0471741817.ch10.

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Conference papers on the topic "Human skeletal muscle"

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Odegard, G. M., T. L. Haut Donahue, D. A. Morrow, and K. R. Kaufman. "Constitutive Modeling of Skeletal Muscle Tissue." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175848.

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The main functions of the human musculoskeletal system are to sustain loads and provide mobility. Bones and joints themselves cannot produce movement; skeletal muscles provide the ability to move. Knowledge of muscle forces during given activities can provide insight into muscle mechanics, muscle physiology, musculoskeletal mechanics, neurophysiology, and motor control. However, clinical examinations or instrumented strength testing only provides information regarding muscle groups. Musculoskeletal models are typically needed to calculate individual muscle forces.

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Burmeister and Lehman. "Force Relaxation In Human Skeletal Muscle." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.589829.

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Burmeister, E. E., and S. L. Lehman. "Force relaxation in human skeletal muscle." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761946.

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Koeppen, Ryan, Meghan E. Huber, Dagmar Sternad, and Neville Hogan. "Controlling Physical Interactions: Humans Do Not Minimize Muscle Effort." In ASME 2017 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dscc2017-5202.

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Physical interaction with tools is ubiquitous in functional activities of daily living. While tool use is considered a hallmark of human behavior, how humans control such physical interactions is still poorly understood. When humans perform a motor task, it is commonly suggested that the central nervous system coordinates the musculo-skeletal system to minimize muscle effort. In this paper, we tested if this notion holds true for motor tasks that involve physical interaction. Specifically, we investigated whether humans minimize muscle forces to control physical interaction with a circular kinematic constraint. Using a simplified arm model, we derived three predictions for how humans should behave if they were minimizing muscular effort to perform the task. First, we predicted that subjects would exert workless, radial forces on the constraint. Second, we predicted that the muscles would be deactivated when they could not contribute to work. Third, we predicted that when moving very slowly along the constraint, the pattern of muscle activity would not differ between clockwise (CW) and counterclockwise (CCW) motions. To test these predictions, we instructed human subjects to move a robot handle around a virtual, circular constraint at a constant tangential velocity. To reduce the effect of forces that might arise from incomplete compensation of neuro-musculo-skeletal dynamics, the target tangential speed was set to an extremely slow pace (∼1 revolution every 13.3 seconds). Ultimately, the results of human experiment did not support the predictions derived from our model of minimizing muscular effort. While subjects did exert workless forces, they did not deactivate muscles as predicted. Furthermore, muscle activation patterns differed between CW and CCW motions about the constraint. These findings demonstrate that minimizing muscle effort is not a significant factor in human performance of this constrained-motion task. Instead, the central nervous system likely prioritizes reducing other costs, such as computational effort, over muscle effort to control physical interactions.

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Al-Ibadi, Alaa, Samia Nefti-Meziani, and Steve Davis. "A circular pneumatic muscle actuator (CPMA) inspired by human skeletal muscles." In 2018 IEEE International Conference on Soft Robotics (RoboSoft). IEEE, 2018. http://dx.doi.org/10.1109/robosoft.2018.8404889.

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"A NEW METABOLISM MODEL FOR HUMAN SKELETAL MUSCLE." In International Conference on Biomedical Electronics and Devices. SciTePress - Science and and Technology Publications, 2008. http://dx.doi.org/10.5220/0001051202380243.

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Selk Ghafari, Ali, Ali Meghdari, and Gholam Reza Vossoughi. "Modeling of Human Lower Extremity Musculo-Skeletal Structure Using Bond Graph Approach." In ASME 2007 International Mechanical Engineering Congress and Exposition. ASMEDC, 2007. http://dx.doi.org/10.1115/imece2007-41558.

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A vector bond graph approach for dynamic modeling of human musculo-skeletal system is addressed in this article. In the proposed model, human body is modeled as a ten-segment, nine degree of freedom, mechanical linkage, actuated by ten muscles in sagittal plane. The head, arm and torso (HAT) are modeled as a single rigid body. Interaction of the feet with the ground is modeled using a spring-damper unit placed under the sole of each foot. The path of each muscle is represented by a straight line. Each actuator is modeled as a three-element, Hill-type muscle in series with tendon. The governing equations of motion generated by the proposed method are equivalent to those developed with more traditional techniques. However the models can be more easily used in conjunction with control models of neuro-muscular function for the simulation of overall dynamic motor performance. In the proposed structure, segments can be easily added or removed. Such a model may have applications in clinical diagnosis and modeling of paraplegic patients during robotic-assisted walking.

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Pagliara, Valentina, Rosarita Nasso, Antonio Ascione, Mariorosario Masullo, and Rosaria Arcone. "Myostatin and plasticity of skeletal muscle tissue." In Journal of Human Sport and Exercise - 2019 - Summer Conferences of Sports Science. Universidad de Alicante, 2019. http://dx.doi.org/10.14198/jhse.2019.14.proc5.12.

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Ramirez, Angelica Maria, Begoña Calvo Calzada, and Jorge Grasa. "The Effect of the Fascia on the Stress Distribution in Skeletal Muscle." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19696.

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The human and vertebrate interaction with the environment is done primarily through the movement. This is possible due the skeletal muscle: anatomical structure able to contract voluntarily. The skeletal muscles are made up of contractile proteins which slide one over another allowing the muscle shortening and the body force generation. This protein structure of actin and myosin maintains its organization through the connective tissue that surrounds it (endomysium, perimysium and epimysium), creating arrays of myofibrils, fibre bundles, fascicles until conform the whole muscle. All this connective tissue extends to the ends of the muscle to form the tendon.

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Ito, Akira, Hirokazu Akiyama, Yasunori Yamamoto, Yoshinori Kawabe, and Masamichi Kamihira. "Skeletal muscle tissue engineering using functional magnetite nanoparticles." In 2009 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2009. http://dx.doi.org/10.1109/mhs.2009.5351986.

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