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Journal articles on the topic 'Humoral immune responses'

Author: Grafiati

Published: 4 June 2021

Last updated: 19 February 2022

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1

Stegall, Mark D., Natalie Moore, Timucin Taner, Han Li, and Patrick G. Dean. "Down-Regulating Humoral Immune Responses." Transplantation Journal 97, no. 3 (2014): 247–57. http://dx.doi.org/10.1097/tp.0b013e3182a72115.

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2

Jyonouchi, Harumi. "Nucleotide Actions on Humoral Immune Responses." Journal of Nutrition 124, suppl_1 (1994): 138S—143S. http://dx.doi.org/10.1093/jn/124.suppl_1.138s.

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3

YANG, HYUN MO. "A MATHEMATICAL MODEL TO ASSESS THE IMMUNE RESPONSE AGAINSTTRYPANOSOMA CRUZIINFECTION." Journal of Biological Systems 23, no. 01 (2015): 131–63. http://dx.doi.org/10.1142/s0218339015500084.

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A mathematical model is developed to assess humoral and cellular immune responses against Trypanosoma cruzi infection. Analysis of the model shows a unique non-trivial equilibrium, which is locally asymptotically stable, except in the case of a strong cellular response. When the proliferation of the activated CD8 T cells is increased, this equilibrium becomes unstable and a limit cycle appears. However, this behavior can be avoided by increasing the action of the humoral response. Therefore, unbalanced humoral and cellular responses can be responsible for long asymptomatic period, and the cont

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4

Ebersole, Jeffrey L. "Systemic Humoral Immune Responses in Periodontal Disease." Critical Reviews in Oral Biology & Medicine 1, no. 4 (1990): 283–331. http://dx.doi.org/10.1177/10454411900010040601.

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5

Alcántara, Patricia, and Dolores Correa. "Human humoral immune responses against Trichinella spiralis." International Journal for Parasitology 23, no. 5 (1993): 657–60. http://dx.doi.org/10.1016/0020-7519(93)90173-v.

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6

Vyas, Ashish Kumar, Mojahidul lslam, Garima Garg, Anirudh K. Singh, and Nirupma Trehanpati. "Humoral Immune Responses and Hepatitis B Infection." Digestive Diseases 39, no. 5 (2021): 516–25. http://dx.doi.org/10.1159/000514274.

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<b><i>Background:</i></b> Chronicity or seroclearance of hepatitis B virus (HBV) antigens is determined by the host immune responses. Current approaches to treat HBV patients are based on inhibition of replication using different antivirals (nucleoside or nucleotide analogs) as monotherapy, or along with immune modulators as combination therapy is being used worldwide for reducing the viral load. Understanding the role of immune cellular therapies with currently available treatments for persistent viral-mediated responses in HBV patients is unexplored. However, the gene

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7

Alam, KH Md Faisal, Mohd Harun Or Rashid, Md Azizul Haque, et al. "Humoral Immune Responses to Amebic Liver Abscess." TAJ: Journal of Teachers Association 34, no. 1 (2021): 5–8. http://dx.doi.org/10.3329/taj.v34i1.54899.

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Background: Amebic liver abscess (ALA) is an ancient parasitic disease caused by E. histolytica described first by Hippocrates. It is endemic worldwide, mainly in tropic and subtropics countries. About 50 million true E. histolytica infections and approximately 100,000 deaths occur each year globally. In Bangladesh, exact incidences of amebic liver abscess cases are not estimated, but hospital reports indicate that it is endemic. Immunity and immune responses in acute and post infections of ALA are not well understood to date. However, the understanding of immunology is essential to know disea

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8

Renshaw, B. R., W. C. Fanslow, R. J. Armitage, et al. "Humoral immune responses in CD40 ligand-deficient mice." Journal of Experimental Medicine 180, no. 5 (1994): 1889–900. http://dx.doi.org/10.1084/jem.180.5.1889.

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Individuals with X-linked hyper-IgM syndrome fail to express functional CD40 ligand (CD40L) and, as a consequence, are incapable of mounting protective antibody responses to opportunistic bacterial infections. To address the role of CD40L in humoral immunity, we created, through homologous recombination, mice deficient in CD40L expression. These mice exhibited no gross developmental deficiencies or health abnormalities and contained normal percentages of B and T cell subpopulations. CD40L-deficient mice did display selective deficiencies in humoral immunity; basal serum isotype levels were sig

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9

Zhang, Jingyao, Wenjuan Gao, Qirui Guo, et al. "Helminth Protein Vaccine Induced Follicular T Helper Cell for Enhancement of Humoral Immunity againstSchistosoma japonicum." BioMed Research International 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/798164.

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Protein vaccines combined with adjuvants have been widely used to induce immune responses, especially the humoral immune response, against molecular targets including parasites. Follicular T helper (Tfh) cells are the specialized providers of B-cell help, however, the induction of Tfh cells in protein vaccination has been rarely studied. Here, we report that theSchistosoma japonicumrecombinant protein (SjGST-32) combined with tacrolimus (FK506) augmented the induction of Tfh cells, which expressed the canonical markers CXCR5, BCL6, and IL-21, and enhanced the humoral immune responses in BALB/c

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10

Pollard, Andrew J., Rachel Galassini, Eileene M. Rouppe van der Voort, et al. "Humoral Immune Responses to Neisseria meningitidis in Children." Infection and Immunity 67, no. 5 (1999): 2441–51. http://dx.doi.org/10.1128/iai.67.5.2441-2451.1999.

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ABSTRACT An understanding of the nature of immunity to serogroup B meningococci in childhood is necessary in order to establish the reasons for poor responses to candidate vaccines in infancy. We sought to examine the nature of humoral immune responses following infection in relation to age. Serum bactericidal activity was poor in children under 12 months of age despite recent infection with Neisseria meningitidis. The highest levels of bactericidal activity were seen in children over 10 years of age. However, infants produced levels of total immunoglobulin G (IgG) and IgG subclass antibodies

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11

Cerón-Gómez, Miller, and Hyun Mo Yang. "Global dynamics of humoral and cellular immune responses to virus infection." Universitas Scientiarum 24, no. 2 (2019): 407–23. http://dx.doi.org/10.11144/javeriana.sc24-2.gdoh.

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We study the global stability of a model of virus dynamics with consideration of humoral and cellular immune responses. We use a Lyapunov direct method to obtain sufficient conditions for the global stability of virus free and viruspresence equilibriums. First, we analyze the model without an immune response and found that if the reproductive number of the virus is less than or equal to one, the virus-free equilibrium is globally asymptotically stable. However, for the virus-presence equilibrium, global stability is obtained if the virus entrance rate into the target cells is less than one. We

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12

Hirsch, Vanessa M., Sampa Santra, Simoy Goldstein, et al. "Immune Failure in the Absence of Profound CD4+ T-Lymphocyte Depletion in Simian Immunodeficiency Virus-Infected Rapid Progressor Macaques." Journal of Virology 78, no. 1 (2004): 275–84. http://dx.doi.org/10.1128/jvi.78.1.275-284.2004.

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ABSTRACT A fraction of simian immunodeficiency virus (SIV)-infected macaques develop rapidly progressive disease in the apparent absence of detectable SIV-specific antibody responses. To characterize the immunopathogenesis of this syndrome, we studied viral load, CD4+ T-lymphocyte numbers as well as cellular and humoral immune responses to SIV and other exogenous antigens in four SIVsm-infected rhesus macaques that progressed to AIDS 9 to 16 weeks postinoculation. Each of these animals exhibited high levels of viremia but showed relatively preserved CD4 T lymphocytes in blood and lymphoid tiss

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13

Kawachi, Izumi. "Humoral and cellular immune responses in neuromyelitis optica." Rinsho Shinkeigaku 50, no. 11 (2010): 873–74. http://dx.doi.org/10.5692/clinicalneurol.50.873.

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14

Kinane, Denis Francis. "Cellular and humoral immune responses in periodontal disease." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 38, Supplement1 (1996): 43. http://dx.doi.org/10.2329/perio.38.supplement1_43.

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15

Kinane, Denis F. "Cellular and Humoral Immune Responses in Periodontal Disease." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 44, Supplement1 (2002): 31–32. http://dx.doi.org/10.2329/perio.44.supplement1_31.

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16

Dobson, Ruth, Ute C. Meier, and Gavin Giovannoni. "More to come: Humoral immune responses in MS." Journal of Neuroimmunology 240-241 (December 2011): 13–21. http://dx.doi.org/10.1016/j.jneuroim.2011.09.009.

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17

Rezaeipoor, R., L. Ziaei Sanei, and M. Kamalinejad. "Effects of Isatis cappadocica on humoral immune responses." Fitoterapia 71, no. 1 (2000): 14–18. http://dx.doi.org/10.1016/s0367-326x(99)00092-1.

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18

Bharadwaj, Mausumi, Robert Nofchissey, Diane Goade, Frederick Koster, and Brian Hjelle. "Humoral Immune Responses in the Hantavirus Cardiopulmonary Syndrome." Journal of Infectious Diseases 182, no. 1 (2000): 43–48. http://dx.doi.org/10.1086/315657.

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19

Cerar, Tjaša, Katarina Ogrinc, Franc Strle, and Eva Ružić-Sabljić. "Humoral Immune Responses in Patients with Lyme Neuroborreliosis." Clinical and Vaccine Immunology 17, no. 4 (2010): 645–50. http://dx.doi.org/10.1128/cvi.00341-09.

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ABSTRACT The aim of this study was to analyze and compare the humoral immune responses in serum and cerebrospinal fluid (CSF) for 34 adult patients with clinically evident Lyme neuroborreliosis, 27 patients with clinically suspected Lyme neuroborreliosis, and 32 patients with tick-borne encephalitis. Additionally, we wanted to compare the findings of two methods for the detection of intrathecally synthesized borrelial antibodies: the IDEIA Lyme neuroborreliosis test using flagellar antigen and an approach based on the Liaison indirect chemiluminescence immunoassay using the OspC and VlsE antig

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20

Iritani, Nobuhiro, Toshiyuki Seto, Hideji Hattori, et al. "Humoral immune responses against norovirus infections of children." Journal of Medical Virology 79, no. 8 (2007): 1187–93. http://dx.doi.org/10.1002/jmv.20897.

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21

Barletta, Ana Beatriz Ferreira, Thiago Luiz Alves e Silva, Octavio A. C. Talyuli, et al. "Prostaglandins regulate humoral immune responses in Aedes aegypti." PLOS Neglected Tropical Diseases 14, no. 10 (2020): e0008706. http://dx.doi.org/10.1371/journal.pntd.0008706.

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22

Böttger, Erik C., and Dieter Bitter-Suerman. "Complement and the regulation of humoral immune responses." Immunology Today 8, no. 9 (1987): 261–64. http://dx.doi.org/10.1016/0167-5699(87)90184-8.

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23

Konakajima, Y., A. Tani, K. Ohura, et al. "Humoral immune responses in experimental gingivitis in rats." Archives of Oral Biology 35 (1990): S181—S183. http://dx.doi.org/10.1016/0003-9969(90)90153-2.

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24

Mediratta, P. K., Neeta Das, V. S. Gupta, and P. Sen. "Modulation of humoral immune responses by endogenous opioids." Journal of Allergy and Clinical Immunology 81, no. 1 (1988): 27–32. http://dx.doi.org/10.1016/0091-6749(88)90216-3.

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25

Bernard, Claude C., and Nicole Kerlero de Rosbo. "Cellular and humoral immune responses in multiple sclerosis." Journal of Neuroimmunology 35 (January 1991): 15. http://dx.doi.org/10.1016/0165-5728(91)90855-2.

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26

Mahallawi, Waleed H. "Humoral immune responses in hospitalized COVID-19 patients." Saudi Journal of Biological Sciences 28, no. 7 (2021): 4055–61. http://dx.doi.org/10.1016/j.sjbs.2021.04.032.

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27

Mitra, Roshni, Sarvjeet Singh, and Ashok Khar. "Antitumour immune responses." Expert Reviews in Molecular Medicine 5, no. 3 (2003): 1–22. http://dx.doi.org/10.1017/s1462399403005623.

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The role of the immune system in combating tumour progression has been studied extensively. The two branches of the immune response – humoral and cell-mediated – act both independently and in concert to combat tumour progression, the success of which depends on the immunogenicity of the tumour cells. The immune system discriminates between transformed cells and normal cells by virtue of the presence of unique antigens on tumour cells. Despite this, the immune system is not always able to detect and kill cancerous cells because neoplasms have also evolved various strategies to escape immune sur

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28

Hu, Hui, Xinya Lu, Ling Tao, et al. "Induction of Specific Immune Responses by Severe Acute Respiratory Syndrome Coronavirus Spike DNA Vaccine with or without Interleukin-2 Immunization Using Different Vaccination Routes in Mice." Clinical and Vaccine Immunology 14, no. 7 (2007): 894–901. http://dx.doi.org/10.1128/cvi.00019-07.

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ABSTRACT DNA vaccines induce humoral and cellular immune responses in animal models and humans. To analyze the immunogenicity of the severe acute respiratory syndrome (SARS) coronavirus (CoV), SARS-CoV, spike DNA vaccine and the immunoregulatory activity of interleukin-2 (IL-2), DNA vaccine plasmids pcDNA-S and pcDNA-IL-2 were constructed and inoculated into BALB/c mice with or without pcDNA-IL-2 by using three different immunization routes (the intramuscular route, electroporation, or the oral route with live attenuated Salmonella enterica serovar Typhimurium). The cellular and humoral immune

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29

van Spriel, Annemiek B. "Tetraspanins in the humoral immune response." Biochemical Society Transactions 39, no. 2 (2011): 512–17. http://dx.doi.org/10.1042/bst0390512.

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The tetraspanins represent a large superfamily of four-transmembrane proteins that are expressed on all nucleated cells. Tetraspanins play a prominent role in the organization of the plasma membrane by co-ordinating the spatial localization of transmembrane proteins and signalling molecules into ‘tetraspanin microdomains’. In immune cells, tetraspanins interact with key leucocyte receptors [including MHC molecules, integrins, CD4/CD8 and the BCR (B-cell receptor) complex] and as such can modulate leucocyte receptor activation and downstream signalling pathways. There is now ample evidence that

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30

Hocknell, Peter K., Rebecca D. Wiley, Xiuqing Wang, et al. "Expression of Human Immunodeficiency Virus Type 1 gp120 from Herpes Simplex Virus Type 1-Derived Amplicons Results in Potent, Specific, and Durable Cellular and Humoral Immune Responses." Journal of Virology 76, no. 11 (2002): 5565–80. http://dx.doi.org/10.1128/jvi.76.11.5565-5580.2002.

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ABSTRACT Herpes simplex virus type 1 (HSV-1) infects a wide range of cells, including dendritic cells. Consequently, HSV-1 vectors may be capable of eliciting strong immune responses to vectored antigens. To test this hypothesis, an HSV-1 amplicon plasmid encoding human immunodeficiency virus type 1 gp120 was constructed, and murine immune responses to helper virus-free amplicon preparations derived from this construct were evaluated. Initial studies revealed that a single intramuscular (i.m.) injection of 106 infectious units (i.u.) of HSV:gp120 amplicon particles (HSV:gp120) elicited Env-spe

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31

Kong, Wing-pui, Ling Xu, Konrad Stadler, et al. "Modulation of the Immune Response to the Severe Acute Respiratory Syndrome Spike Glycoprotein by Gene-Based and Inactivated Virus Immunization." Journal of Virology 79, no. 22 (2005): 13915–23. http://dx.doi.org/10.1128/jvi.79.22.13915-13923.2005.

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ABSTRACT Although the initial isolates of the severe acute respiratory syndrome (SARS) coronavirus (CoV) are sensitive to neutralization by antibodies through their spike (S) glycoprotein, variants of S have since been identified that are resistant to such inhibition. Optimal vaccine strategies would therefore make use of additional determinants of immune recognition, either through cellular or expanded, cross-reactive humoral immunity. Here, the cellular and humoral immune responses elicited by different combinations of gene-based and inactivated viral particles with various adjuvants have be

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32

Hamsten, Carl, Georgina Tjipura-Zaire, Laura McAuliffe, et al. "Protein-Specific Analysis of Humoral Immune Responses in a Clinical Trial for Vaccines against Contagious Bovine Pleuropneumonia." Clinical and Vaccine Immunology 17, no. 5 (2010): 853–61. http://dx.doi.org/10.1128/cvi.00019-10.

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ABSTRACT Specific humoral immune responses in a clinical trial on cattle for vaccines against contagious bovine pleuropneumonia (CBPP) were investigated. The trial included a subunit vaccine consisting of five recombinant putative variable surface proteins of the infectious agent Mycoplasma mycoides subsp. mycoides small colony type (M. mycoides SC) compared to the currently approved attenuated vaccine strain T1/44 and untreated controls. Humoral immune responses to 65 individual recombinant surface proteins of M. mycoides SC were monitored by a recently developed bead-based array assay. Respo

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33

Lendvai, Nikoletta, Sacha Gnjatic, Erika Ritter, et al. "Host Immune Responses Against CT Antigens in Multiple Myeloma Patients." Blood 108, no. 11 (2006): 3492. http://dx.doi.org/10.1182/blood.v108.11.3492.3492.

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Abstract The type I Melanoma Antigen GEne (MAGE) proteins belong to the Cancer-Testis (CT) family of tumor-associated antigens and are widely expressed in solid and hematologic malignancies. They are immunogenic and frequently elicit spontaneous immune responses in patients with CT antigen-expressing tumors, particularly in malignant melanoma. In melanoma patients, there is high concordance between humoral and cellular immunity. Based on these findings, CT antigens are widely investigated as potential antigenic targets for tumor-specific therapeutic vaccines. We previously showed that the type

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34

Kersh, Gilbert J., Kelly A. Fitzpatrick, Joshua S. Self, Brad J. Biggerstaff, and Robert F. Massung. "Long-Term Immune Responses to Coxiella burnetii after Vaccination." Clinical and Vaccine Immunology 20, no. 2 (2012): 129–33. http://dx.doi.org/10.1128/cvi.00613-12.

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ABSTRACTQ fever is a zoonotic disease caused by infection with the bacteriumCoxiella burnetii. Infection withC. burnetiiresults in humoral and cellular immune responses, both of which are thought to contribute to protection against subsequent infection. Whole-cell formalin-inactivated vaccines have also been shown to induce both humoral and cellular immunity and provide protection. Whether measurement of cellular or humoral immunity is a better indicator of immune protection is not known, and the duration of immunity induced by natural infection or vaccination is also poorly understood. To bet

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35

Ahn, Young Keun, and Joung Hoon Kim. "Effects of squalene on the immune responses in mice(I): Humoral immune responses of squalene." Archives of Pharmacal Research 14, no. 4 (1991): 370–78. http://dx.doi.org/10.1007/bf02876887.

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36

Tedder, T. F., K. M. Haas, and J. C. Poe. "CD19-CD21 complex regulates an intrinsic Src family kinase amplification loop that links innate immunity with B-lymphocyte intracellular calcium responses." Biochemical Society Transactions 30, no. 4 (2002): 807–11. http://dx.doi.org/10.1042/bst0300807.

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CD19 is a B-lymphocyte cell surface molecule that functions as a general response regulator or rheostat, which defines intrinsic and B-cell antigen receptor-induced signalling thresholds that are critical for humoral immunity and expansion of the peripheral B-cell pool. In addition, B-cell responses are influenced by signals transduced through a CD19-CD21 cell surface receptor complex, where the binding of complement C3d to CD21 links humoral immune responses with the innate immune system. This review outlines recent biochemical and genetic studies that characterize the signal transduction pat

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37

Alfonso, Christopher, Jung-Ok Han, G. Stuart Williams, and Lars Karlsson. "The Impact of H2-DM on Humoral Immune Responses." Journal of Immunology 167, no. 11 (2001): 6348–55. http://dx.doi.org/10.4049/jimmunol.167.11.6348.

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38

Shet, Uttom, Hee-Kyun Oh, Hyun-Ju Chung, et al. "Humoral immune responses to periodontal pathogens in the elderly." Journal of Periodontal & Implant Science 45, no. 5 (2015): 178. http://dx.doi.org/10.5051/jpis.2015.45.5.178.

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39

Naveed, Khalid, Aqeel Javeed, Muhammad Ashraf, Amjad Riaz, Aamir Ghafoor, and Adeel Sattar. "Effect of nabumetone on humoral immune responses in mice." Arquivo Brasileiro de Medicina Veterinária e Zootecnia 72, no. 3 (2020): 915–20. http://dx.doi.org/10.1590/1678-4162-11460.

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ABSTRACT Nabumetone is used to reduce the pain and inflammation in rheumatoid arthritis. In the current study, immunomodulatory effect of Nabumetone is investigated in mice. The control group was administered normal saline orally as placebo. Nabumetone was administered orally via gavage in two treatment groups at 14mg/kg.b.w. doses and 28mg/kgb.w., respectively. Haemagglutination (HA) assay, Jerne hemolytic plaque and mice lethality assays were applied. In HA assay, the titer was significantly decreased in Nabumetone treatment groups (P< 0.001). In Jerne hemolytic plaque formation assay, th

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40

Kennedy, Richard B., Inna G. Ovsyannikova, V. Shane Pankratz, et al. "Gender effects on humoral immune responses to smallpox vaccine." Vaccine 27, no. 25-26 (2009): 3319–23. http://dx.doi.org/10.1016/j.vaccine.2009.01.086.

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41

Landers, D. M., K. A. Kubitz, T. Robinson, and J. S. Skinner. "PSYCHOLOGICAL, HUMORAL IMMUNE, AND HEALTH RESPONSES TO CHRONIC EXERCISE." Medicine and Science in Sports and Exercise 21, Supplement (1989): S50. http://dx.doi.org/10.1249/00005768-198904001-00297.

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42

Dummer, Reinhard, Jurg Willers, and Daria Kanduc. "INV 30 Melanocytic differentiation antigens and humoral immune responses." Melanoma Research 17, no. 1 (2007): A13—A14. http://dx.doi.org/10.1097/00008390-200702000-00040.

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43

Baldwin, C. I., F. Medeiros, M. Malecela, and D. A. Denham. "Humoral immune responses in cats repeatedly infected withBrugia pahangi." Parasite 1, no. 1S (1994): S40. http://dx.doi.org/10.1051/parasite/199401s1040.

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GOTTLIEB, D. J., H. G. PRENTICE, H. E. HESLOP, C. BELLO, and M. K. BRENNER. "IL-2 infusion abrogates humoral immune responses in humans." Clinical & Experimental Immunology 87, no. 3 (2008): 493–98. http://dx.doi.org/10.1111/j.1365-2249.1992.tb03025.x.

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45

Waag, David M., Marilyn J. England, and David DeShazer. "Humoral Immune Responses in a Human Case of Glanders." Clinical and Vaccine Immunology 19, no. 5 (2012): 814–16. http://dx.doi.org/10.1128/cvi.05567-11.

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ABSTRACTWithin 2 months of acquiring glanders, a patient developed 8-, 16-, and 4-fold increases, respectively, in specific IgA, IgG, and IgM serological titers againstBurkholderia mallei. Within 14 months of infection, the titers decreased to the baseline. Serum from this patient was also highly reactive againstBurkholderia pseudomalleiwhole cells.Burkholderia malleiwhole cells did not react with sera from patients with other diseases. Therefore, an assay using aB. malleicellular diagnostic antigen may be useful for the serodiagnosis of glanders.

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Kajiwara, Koji, Andrew P. Byrnes, Yoshinori Ohmoto, Harry M. Charlton, Matthew J. A. Wood, and Kathryn J. Wood. "Humoral immune responses to adenovirus vectors in the brain." Journal of Neuroimmunology 103, no. 1 (2000): 8–15. http://dx.doi.org/10.1016/s0165-5728(99)00220-9.

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47

Mestecky, J., Z. Moldoveanu, R. C. Alexander, and M. Raska. "Induction of humoral immune responses in the genital tract." Journal of Reproductive Immunology 81, no. 2 (2009): 127. http://dx.doi.org/10.1016/j.jri.2009.06.157.

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48

Cramer, Donald V., and Haval Shirwan. "The importance of humoral immune responses in chronic rejection." Transplantation Reviews 12, no. 4 (1998): 166–76. http://dx.doi.org/10.1016/s0955-470x(98)80007-4.

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49

Lang, Susanne I., Nathalia A. Giese, Jean Rommelaere, Christiane Dinsart, and Jan J. Cornelis. "Humoral immune responses against minute virus of mice vectors." Journal of Gene Medicine 8, no. 9 (2006): 1141–50. http://dx.doi.org/10.1002/jgm.940.

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50

Struijk, Geertrude H., Robert C. Minnee, Sven D. Koch, et al. "Maintenance immunosuppressive therapy with everolimus preserves humoral immune responses." Kidney International 78, no. 9 (2010): 934–40. http://dx.doi.org/10.1038/ki.2010.269.

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