Relevant bibliographies by topics / Hurthle Cell Tumors Thyroid

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Academic literature on the topic 'Hurthle Cell Tumors Thyroid'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Hurthle Cell Tumors Thyroid"

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Cornianu, Marioara. "Proliferative Activity of Thyroid Hurthle Cell Tumors." Acta Endocrinologica (Bucharest) 2, no. 3 (2006): 269–81. http://dx.doi.org/10.4183/aeb.2006.269.

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Suh, Young-Jin, Chung-Soo Chun, Young-Hyug Kim, Hyun-Min Cho, Yong-Sung Won, Hyung-Min Chin, Jun-Gi Kim, and Woo-Bae Park. "Hurthle Cell Tumors of the Thyroid Gland." Korean Journal of Endocrine Surgery 1, no. 1 (2001): 89. http://dx.doi.org/10.16956/kjes.2001.1.1.89.

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Johnson, Terri L., Ricardo V. Lloyd, Richard E. Burney, and Norman W. Thompson. "Hurthle cell thyroid tumors. An immunohistochemical study." Cancer 59, no. 1 (January 1, 1987): 107–12. http://dx.doi.org/10.1002/1097-0142(19870101)59:1<107::aid-cncr2820590123>3.0.co;2-u.

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Ganly, Ian, Julio Ricarte Filho, Stephanie Eng, Ronald Ghossein, Luc G. T. Morris, Yupu Liang, Nicholas Socci, et al. "Genomic Dissection of Hurthle Cell Carcinoma Reveals a Unique Class of Thyroid Malignancy." Journal of Clinical Endocrinology & Metabolism 98, no. 5 (May 1, 2013): E962—E972. http://dx.doi.org/10.1210/jc.2012-3539.

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Context: Hurthle cell cancer (HCC) is an understudied cancer with poor prognosis. Objective: Our objective was to elucidate the genomic foundations of HCC. Design and Setting: We conducted a large-scale integrated analysis of mutations, gene expression profiles, and copy number alterations in HCC at a single tertiary-care cancer institution. Methods: Mass spectrometry-based genotyping was used to interrogate hot spot point mutations in the most common thyroid oncogenes: BRAF, RET, NRAS, HRAS, KRAS, PIK3CA, MAP2K1, and AKT1. In addition, common oncogenic fusions of RET and NTRK1 as well as PAX8/PPARγ and AKAP9-BRAF were also assessed by RT-PCR. Global copy number changes and gene expression profiles were determined in the same tumor set as the mutational analyses. Results: We report that the mutational, transcriptional, and copy number profiles of HCC were distinct from those of papillary thyroid cancer and follicular thyroid cancer, indicating HCC to be a unique type of thyroid malignancy. Unsupervised hierarchical clustering of gene expression showed the 3 groups of Hurthle tumors (Hurthle cell adenoma [HA], minimally invasive Hurthle cell carcinoma [HMIN], and widely invasive Hurthle cell carcinoma [HWIDE] clustered separately with a marked difference between HWIDE and HA. Global copy number analysis also indicated distinct subgroups of tumors that may arise as HWIDE and HMIN. Molecular pathways that differentiate HA from HWIDE included the PIK3CA-Akt-mTOR and Wnt/β-catenin pathways, potentially providing a rationale for new targets for this type of malignancy. Conclusions: Our data provide evidence that HCC may be a unique thyroid cancer distinct from papillary and follicular thyroid cancer.
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Carcangiu, M. L., S. Bianchi, D. Savino, I. M. Voynick, and J. Rosai. "Follicular Hurthle cell tumors of the thyroid gland." Cancer 68, no. 9 (November 1, 1991): 1944–53. http://dx.doi.org/10.1002/1097-0142(19911101)68:9<1944::aid-cncr2820680917>3.0.co;2-i.

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Palo, Seetu, Archana H. Deshpande, and Citrawati B. Gargade. "Hurthle cell adenoma and papillary microcarcinoma: a rare case of thyroid collision tumor." International Journal of Otorhinolaryngology and Head and Neck Surgery 7, no. 4 (March 24, 2021): 695. http://dx.doi.org/10.18203/issn.2454-5929.ijohns20211198.

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<p class="abstract">Collision tumors of the thyroid gland are histologically distinct tumors coexisting within the gland. Here, we depict a case of a 36-year-old woman who presented with anterior neck swelling of one-year duration. Local examination revealed a 3×2 cm, firm, right solitary thyroid nodule. Fine needle aspiration cytology was suggestive of Hurthle cell neoplasm. Patient underwent a right hemi-thyroidectomy. Histopathological examination showed co-existence of Hurthle cell adenoma and papillary thyroid microcarcinoma. The patient was asymptomatic during six months follow up. It is important that the surgeons and pathologists are aware of these collision tumors so that optimal therapeutic interventions can be carried out. </p>
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Chao, Tzu-Chieh, Jen-Der Lin, and Miin-Fu Chen. "Surgical Treatment of Hurthle Cell Tumors of the Thyroid." World Journal of Surgery 29, no. 2 (January 20, 2005): 164–68. http://dx.doi.org/10.1007/s00268-004-7669-9.

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Nagoti, Sarada, Anunayi J., and Manthan Patel. "The role of cell cycle regulatory protein p53 in Follicular neoplasms of thyroid with hurthle cells." International Journal of Scientific Reports 2, no. 5 (May 19, 2016): 99. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20161468.

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<p class="abstract"><strong><span lang="EN-US">Background:</span></strong><span lang="EN-US"> The disease biology of Follicular neoplasms of thyroid with hurthle cells is poorly understood. Very few studies in literature have addressed the role of p53 in these neoplasms. The aim of the present study is to analyze the histomorphological features of Follicular neoplasms with hurthle cell change and to evaluate the role of p53 in their tumor biology.</span></p><p class="abstract"><strong><span lang="EN-US">Methods:</span></strong>32 cases of Follicular neoplasms of thyroid with focal and pure hurthle cell change over a period of 2.5 years were studied histologically and immunohistochemically using p53 antibody (Biogenex). They included 20 follicular adenomas with focal hurthle cells, 10 pure hurthle cell adenomas and 2 hurthle cell carcinomas.</p><p class="abstract"><strong><span lang="EN-US">Results:</span></strong><span lang="EN-US"> All cases showed nuclear p53 positivity in hurthle cells. Muller-Hocker et al criteria was used for frequency scoring. Out of the 32 cases, 12 cases of pure hurthle cells showed score 3, Remaining 20 cases of follicular adenomas with focal hurthle cell change showed score 3 in cases with &gt;50% hurthle cells, score 2 in cases with 20-40% hurthle cells and score 0 in cases with &lt;10% hurthle cells.</span></p><p class="abstract"><strong><span lang="EN-US">Conclusions:</span></strong>The study showed a good correlation of p53 protein expression with tumor progression and aggressiveness. Hence this indicates that molecular alterations in p53 pathway play a role in tumor biology in Follicular neoplasms of thyroid with hurthle cell change.</p>
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Rana, Chanchal. "Hurthle Cell Adenoma and Papillary Microcarcinoma in Thyroid: Collision Tumors." World Journal of Endocrine Surgery 10, no. 2 (2018): 134–36. http://dx.doi.org/10.5005/jp-journals-10002-1232.

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Ding, Li, Yunhui Jiang, and Wan Yang. "Approach the Invasive Potential with Hurthle Cell Tumors of Thyroid." Pathology & Oncology Research 25, no. 2 (December 11, 2018): 697–701. http://dx.doi.org/10.1007/s12253-018-0546-x.

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Dissertations / Theses on the topic "Hurthle Cell Tumors Thyroid"

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Sherret, John, and Joshua Coleman. "Small Cell Medullary Thyroid Cancer: A Therapeutic Dilemma." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/asrf/2020/presentations/57.

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Small cell variant of medullary thyroid carcinoma is an extremely rare histologic entity with a paucity of data. As such, there is a lack of clinical experience regarding this disease. In this case, a 52-year-old patient with small cell variant medullary thyroid carcinoma was experiencing intractable nausea, vomiting, and diarrhea. The initial workup was extensive yet unrevealing. He was refractory to all treatments. On further laboratory analysis, the calcitonin was substantially high and the thyroid stimulating hormone level was mildly elevated. This case is presented to highlight a possible treatment for this rare cancer through thyroxine suppression therapy. This case is presented due to the lack of literature available on small cell medullary thyroid carcinoma and also to discuss a possible direct relationship between thyroid stimulating hormone and calcitonin levels in this disease population.
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Fagan, Erin A. "Identification of the presence and activity of the JAK-STAT pathway in canine solid tumors." Thesis, Virginia Tech, 2017. http://hdl.handle.net/10919/100859.

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Background: The JAK-STAT pathway is a cellular signaling pathway, which acts normally in humans and animals in the control of multiple important functions. Dysregulation of this pathway has been identified in human cancers, as well as a limited number of veterinary cancers. Objectives: The aims of this study were to identify the presence and tentative activity of components of the JAK-STAT pathway in selected canine tumors. Methods: Formalin-fixed, paraffin-embedded samples from mast cell tumors (MCT), hemangiosarcomas (HSA), thyroid carcinomas, and apocrine gland anal sac adenocarcinomas (AGASACA) were obtained from the Diagnostic Histopathology Laboratory at the Virginia Maryland College of Veterinary Medicine. Immunohistochemistry was performed to evaluate protein levels of JAK1, phospho-JAK1, JAK2, phospho-JAK2, STAT3, and phospho-STAT3. Signalment, treatment information, and survival information was obtained from the medical record for each case. Results: Tumor samples were scored for percent positive neoplastic cells. Positive staining was seen for all antibodies in all tumor types, with expression of JAK1, STAT3, and pSTAT3 being highest overall for all tumor types. Significant associations were seen between JAK1 and survival time in MCT (p = 0.03), pJAK1 and survival time in HSA (p = 0.009) and MCT (p = 0.04), and pSTAT3 and metastasis in MCT (p = 0.0008). Conclusions: The finding of positive staining for the components of the JAK-STAT pathway in the tumor samples evaluated indicates presence and tentative activity of this pathway in the studied cancers. Further study of JAK1, pJAK1, and pSTAT3 should be pursued to evaluate their potential as therapeutic targets.
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Malith, V. J. W. "Prevalence, demographic and histological subtypes of hurthle cell tumors of the thyroid: a histopathological audit." Thesis, 2017. http://hdl.handle.net/10539/23399.

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A Dissertation submitted to the Faculty of Health Sciences of University of the Witwatersrand, Johannesburg, in fulfillment of the requirements for the degree of Master of Medicine in Surgery. Johannesburg, 2017
Background: Hurthle cell neoplasms (HCN) are considered a variant of follicular thyroid neoplasms, and accounts for 3-10% of neoplasms of the thyroid gland. They include Hurthle cell adenomas (HCA) and carcinomas (HCC). Differentiating HCA from HCC preoperatively is currently not possible. We retrospectively searched for demographic and histopathological factors which can be used to predict the risk of malignancy in HCN. Aim: To determine the prevalence of HCC and its demographic factors and histopathological features that can be used to predict the risk of malignancy in HCN. Methods: Records of all patients who underwent thyroidectomy at Academic Hospitals associated with University of the Witwatersrand from January 2001 to October 2015 were reviewed. Patients’ demographic data and the final histology of HCN were further analyzed including pre-operative fine needle aspiration cytology (FNAC) results. Data collected included patients’ demographic, final histology, tumor size and preoperative FNAC result. Data was entered into Excel Spreadsheet and analyzed using STATICA 13.1 program. Results: At total of 2641 records of thyroidectomies were found of which 25.6% (676/2641) were for thyroid neoplasms. Only 15.8% (107/676) of the neoplasms were HCNs and 25.2% (27/107) of HCNs were HCCs. Hurthle cell carcinoma made up 5.6% (27/481) of thyroid carcinomas. 70.4% (19/27) of HCCs were incidentally found following thyroidectomy for multinodular goiter (MNG). The mean tumor size was significantly greater for carcinomas than for adenomas (4.9 cm vs. 3.5 cm; p = 0.016). The risk of malignancy increased from 11.1% when the size was less or equal to 1cm, through 33.3% for size of 1-4cm to 51.8% when the size was greater than 4cm in diameter. A total of 58 FNACs results of 107 HCNs were available for further analysis. Thirty one (53.4%: 31/58) of FNAC results were suspicious for HCN (Bethesda IV), seven (12.1%: 7/58) suspicious of papillary carcinoma (Bethesda V) and eight (13.8%: 8/58) were reported as benign (Bethesda II). Around 10.3% (6/58) were non-diagnostic (Bethesda I) whereas 8.6% (5/58) were reported as atypia of unknown significance (Bethesda III). Both HCA and HCC were more prevalent in females, 88.7% (71/80) and 77.8% (21/27); respectively. The mean age of the patients who had HCA and HCC in years was 52.3+/- 15.6 SD and 55.0 +/- 15.0 SD, respectively. Conclusion: Majority of HCCs are diagnosed following thyroidectomy for benign disease. Close to a quarter of HCNs are malignant and the risk of malignancy increases with size. Age and gender are not useful to predict malignancy in HCNs. We recommend total thyroidectomy for thyroid nodule greater than 4cm in diameter if FNAC result is suggestive of HCN as the risk of malignancy is above 50%.
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Books on the topic "Hurthle Cell Tumors Thyroid"

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United States. Environmental Protection Agency. Risk Assessment Forum., ed. Assessment of thyroid follicular cell tumors. Washington, DC: U.S. Environmental Protection Agency, Office of Research and Development, 1998.

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United States. Environmental Protection Agency. Risk Assessment Forum., ed. Assessment of thyroid follicular cell tumors. Washington, DC: U.S. Environmental Protection Agency, Office of Research and Development, 1998.

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United States. Environmental Protection Agency. Risk Assessment Forum, ed. Assessment of thyroid follicular cell tumors. Washington, DC: U.S. Environmental Protection Agency, Office of Research and Development, 1998.

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Keum, NaNa, Mingyang Song, Edward L. Giovannucci, and A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

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In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast (postmenopausal), colorectum, endometrium, esophagus (adenocarcinoma), gallbladder, kidney (renal cell), pancreas, gastric cardia, liver, ovary, prostate (advanced tumors), multiple myeloma, thyroid, and meningioma. Mechanisms by which adipose tissue are thought to promote tumor growth include the endocrine and metabolic effects of fat on sex hormones, growth factors, and inflammation, as well as local chemical or mechanical injury of gastrointestinal organs.
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Book chapters on the topic "Hurthle Cell Tumors Thyroid"

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Kobayashi, Kaoru, Akira Yoshida, and Takehito Igarashi. "CQ25. Does Prognosis of Oxyphilic (Hurthle Cell) Follicular Carcinoma Differ from That of Conventional Follicular Carcinoma?" In Treatment of Thyroid Tumor, 153–55. Tokyo: Springer Japan, 2012. http://dx.doi.org/10.1007/978-4-431-54049-6_36.

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Özkan, Elgin, and Çiğdem Soydal. "Hurthle Cell Carcinoma." In Thyroid and Parathyroid Diseases, 399–402. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78476-2_63.

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Erickson, Lori A. "Hurthle Cell Thyroid Neoplasms." In Atlas of Endocrine Pathology, 63–66. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0443-3_8.

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Heilo, Arne, Eva Sigstad, and Krystyna Grøholt. "Oncocytic Nodule/Hürtle Cell Tumors." In Atlas of Thyroid Lesions, 121. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6010-8_5.

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Eloy, Catarina, José M. Cameselle-Teijeiro, Isabel Amendoeira, Paula Soares, Javier Caneiro-Gómez, Miguel Melo, and Manuel Sobrinho-Simões. "Small Cell Tumours." In Rare Tumors of the Thyroid Gland, 45–56. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61182-2_4.

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Khan, Ashraf, and Manju Prasad. "Tumors of Thyroid Gland: Non-C cell Tumors." In Surgical Pathology of Endocrine and Neuroendocrine Tumors, 41–81. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-396-1_5.

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Lawler, Elizabeth, and Gilbert Fareau. "Advanced Hurthle Cell Carcinoma in a Belizean Woman: Treatment Options in Central America." In CLINICAL - Thyroid Cancer, P3–677—P3–677. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part4.p11.p3-677.

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Gopal, Raj K., Peter M. Sadow, and Ian Ganly. "Hürthle Cell Tumors of the Thyroid." In Surgery of the Thyroid and Parathyroid Glands, 225–28. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-323-66127-0.00025-9.

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Lee, Jung Uee, Min Kyeong Kim, Ji Hye Yim, Kyung-Hye Jung, Min Hee Lee, Seong Eun Lee, Koon Soon Kim, et al. "Distinctive Features of Autophagy in Thyroid Hürthle Cell Tumors." In BASIC/TRANSLATIONAL - Thyroid Cancer, P2–673—P2–673. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part3.p12.p2-673.

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Hoang, Thanh Duc, Vinh Q. Mai, Patrick W. Clyde, and KM Mohamed Shakir. "Tall-Cell Variant Papillary Thyroid Carcinoma Associated with MEN1." In CLINICAL/TRANSLATIONAL - Neuroendocrine Tumors, P3–260—P3–260. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part3.p29.p3-260.

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