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Relevant bibliographies by topics / Hybridized induced polymorphism

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Academic literature on the topic 'Hybridized induced polymorphism'

Author: Grafiati

Published: 6 September 2023

Last updated: 31 July 2025

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Journal articles on the topic "Hybridized induced polymorphism"

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Tsuchida, Shirou, Noriaki Himi, Yuuki Miura, et al. "Photoinduced electron transfer detection method for identifying UGT1A1*28 microsatellites." PLOS ONE 18, no. 8 (2023): e0289506. http://dx.doi.org/10.1371/journal.pone.0289506.

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During development of a novel detection method for the UDP-glucuronosyl transferase 1A1 (UGT1A1)*28, the fluorescence intensity of a dye conjugated to cytosine (C) at the end of a DNA strand decreased upon hybridization with guanine (G). This phenomenon is referred to as photoinduced electron transfer (PeT). Using this phenomenon, we devised a method for the naked-eye detection of UGT1A1*28 (thymine-adenine (TA)-repeat polymorphism). Fluorescently labeled single-stranded DNA (ssDNA) oligonucleotides (probes) were designed and hybridized with complementary strand DNAs (target DNAs). Base pair f

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Omori, A., C. Stephens, J. Cooc, et al. "Microarray analysis of formalin-fixed paraffin-embedded specimens shows distinct gene expression patterns in tumors containing the transforming growth factor beta receptor 6A polymorphism (TGFBR1*6A)." Journal of Clinical Oncology 25, no. 18_suppl (2007): 4111. http://dx.doi.org/10.1200/jco.2007.25.18_suppl.4111.

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4111 Introduction: A frequent polymorphism of the type I transforming growth factor beta receptor (TGFBR1) is TGFBR1*6A (6A), which has a deletion of 3 CGC triplets coding for alanine within a 9-alanine (9A) repeat of TGFBR1 exon 1. 6A may act as a tumor susceptibility allele through switching TGF-beta growth inhibitory signals into growth stimulatory signals and also appears to be acquired in some cases by primary colon cancers and their liver metastases. Our aim in this study was to compare the gene expression profiles of colorectal tumors bearing the 6A and the more common 9A genotypes to d

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Steensma, David P., Jessemy D. Neiger, Julie C. Porcher, et al. "Rearrangements of IER3 Represent a Novel and Recurrent Molecular Abnormality in Myelodysplastic Syndromes (MDS)." Blood 112, no. 11 (2008): 2679. http://dx.doi.org/10.1182/blood.v112.11.2679.2679.

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Abstract Background: For most patients with MDS, the disease-associated molecular abnormalities are unknown, which contributes to diagnostic uncertainty and has limited development of effective therapies. Chromosomal deletions such as del(5q) or monosomy 7 are more common than translocations in MDS, but balanced translocations are more likely to be pathobiologically informative. Here we report cloning of an MDS-associated translocation that revealed a novel recurrent molecular abnormality. Methods: We first studied a 71-year old man who presented with MDS and t(6;9)(p21.3;q34) as an isolated c

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Zhang, Xu, Wei Zhang, Binal Shah, et al. "Hydroxyurea Treatment Is Associated with Elevated Serum Erythropoietin Concentration but Suppressed Global Hypoxic Transcriptional Responses in Sickle Cell Disease." Blood 126, no. 23 (2015): 3380. http://dx.doi.org/10.1182/blood.v126.23.3380.3380.

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Abstract Background The level of distorted erythrocytes due to polymerization of hemoglobin S in sickle cell disease (SCD) (Science 1949;110:543) is a major determinant of the severity of hemolysis and microvascular occlusion (Lancet 2010;376:2018). Erythropoietin (EPO) is elevated in SCD due to hemolytic anemia and a related increase in hypoxia-inducible factors (HIFs) (Eur J Haematol 2007;78:183). Hydroxyurea (HU) is widely used in the treatment of SCD. HU inhibits ribonucleotide reductase (Semin Oncol 1992;19(3 Suppl 9):1-10) and promotes γ globin synthesis thereby increasing HbF-containing

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Brown, W. E., D. S. Robertson, and J. L. Bennetzen. "Molecular analysis of multiple mutator-derived alleles of the bronze locus of maize." Genetics 122, no. 2 (1989): 439–45. http://dx.doi.org/10.1093/genetics/122.2.439.

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Abstract Very few mutations derived from Mutator maize lines have been studied at the molecular level. The variety of Mu elements that can induce mutations, the relative frequency of mutant induction by insertion of a given class of Mu elements or by a Mu-induced genomic rearrangement, a possible intragenic insertion site specificity, and the molecular nature of reversion events are all unknown in the Mutator system. To address these questions, we have isolated several partially or fully inactivated bronze alleles from Mutator maize lines and structurally characterized them by gel blot hybridi

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Banks, P. M., P. J. Larkin, H. S. Bariana, et al. "The use of cell culture for subchromosomal introgressions of barley yellow dwarf virus resistance from Thinopyrum intermedium to wheat." Genome 38, no. 2 (1995): 395–405. http://dx.doi.org/10.1139/g95-051.

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Barley yellow dwarf virus (BYDV) resistance has been transferred to wheat from a group 7 chromosome of Thinopyrum (Agropyron) intermedium. The source of the resistance gene was the L1 disomic addition line, which carries the 7Ai-1 chromosome. The resistance locus is on the long arm of this chromosome. BYDV resistant recombinant lines were identified after three or more generations of selection against a group 7 Th. intermedium short arm marker (red coleoptile) and selection for the presence of BYDV resistance. One recombinant line produced by ph. mutant induced homoeologous pairing and 14 reco

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Johnson, David C., Suzanne Grindle, Faith Davies, et al. "Inherited Genetic Variation and the Risk of Developing Venous Thrombosis with Thalidomide Treatment in Myeloma Patients." Blood 106, no. 11 (2005): 1624. http://dx.doi.org/10.1182/blood.v106.11.1624.1624.

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Abstract There is a high background risk of venous thrombotic events (VTE) in patients with Multiple Myeloma (MM) and this is significantly increased following treatment with Thalidomide. The risk may be mediated by the production of a prothrombotic state, via an angiopathic mechanism, or alternatively it may arise from the release of prothrombotic factors from dying myeloma cells targeted by the treatment. No unifying evidence of a prothrombotic state has been demonstrated to date and it remains important to understand the mechanism underlying VTEs in order to prevent them effectively. Inheri

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Friebe, B., N. S. Kim, J. Kuspira, and B. S. Gill. "Genetic and cytogenetic analyses of the A genome of Triticum monococcum. VI. Production and identification of primary trisomics using the C-banding technique." Genome 33, no. 4 (1990): 542–55. http://dx.doi.org/10.1139/g90-081.

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Cytogenetic studies in Triticum monococcum (2n = 2x = 14) are nonexistent. To initiate such investigations in this species, a series of primary trisomics was generated from autotriploids derived from crosses between induced autotetraploids and diploids. All trisomics differed phenotypically from their diploid progenitors. Only two of the seven possible primary trisomic types produced distinct morphological features on the basis of which they could be distinguished. The chromosomes in the karyotype were morphologically very similar and could not be unequivocally identified using standard techni

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Dissertations / Theses on the topic "Hybridized induced polymorphism"

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Sarkar, Sounak. "High-Resolution Charge Density Studies on Electronic Nature of Weak Interactions and Correlation of Molecular Conformation with Packing in Solid State." Thesis, 2018. https://etd.iisc.ac.in/handle/2005/5281.

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The thesis entitled “High-Resolution Charge Density Studies on Electronic Nature of Weak Interactions and Correlation of Molecular Conformation with Packing in Solid State” consist of five chapters. Chapter 1 is a brief introduction to the methodologies and techniques utilized in modelling electron densities and the topics relevant to the work. The subsequent four chapters are divided into two parts-Part A and Part B. Part A has two chapters that discusses the electronic nature of unexplored weak intermolecular interactionspnicogen bonding in nitrogen atom and hydrophobic interactions be

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