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Journal articles on the topic 'Hydropsy fetalis'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Pejic, Katarina, Borisav Jankovic, Zeljko Mikovic, Zorica Rakonjac, Jelena Martic, and Natasa Stajic. "Non-immune hydrops fetalis: Clinical experience in newborn infants." Medical review 64, no. 9-10 (2011): 507–10. http://dx.doi.org/10.2298/mpns1110507p.

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Introduction. Non-immune hydrops fetalis is a condition of excessive accumulation of extravascular fluid without identifiable circulating antibody to erythrocytes membrane antigens. In newborn infants it is characterized by skin oedema and pleural, pericardial or peritoneal effusion. In the era of routine Rh immunization for the prevention of foetal erythroblastosis, non-immune pathophysiologic mechanisms are presented in 76-87% of all hydropic newborns. Non-immune hydrops fetalis can be associated with numerous and various disorders. The mortality rate may exceed 50%. This study was aimed at

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2

Srinivas, G., D.V. Ramanjaneyulu, E. Muralinath, et al. "An Essential Parameters of Parvo Virus Include Patho Physiology, Histo Pathology, Diagnosis, Differential Diagnosis, Treatment and Prognosis." Journal of Research and Reviews in Nursing Science and Education 2, no. 2 (2025): 11–17. https://doi.org/10.5281/zenodo.15541791.

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<em>One virus that only affects people is parvovirus B19. The fifth illness, also known as erythema infectious or slapped cheek syndrome, is known to be caused by it. It primarily affects young children, though it can sometimes affect adults. In addition, it may result in Poly arthropathy, popular-purpuric gloves and socks syndrome (PPGSS) in young people, certain anemias, hydrops fetalis, particularly in pregnant women, and an aplastic crisis. Viral transmission occurs through both respiratory secretions and blood products. The virus can infect a pregnant woman and then infect her unborn chil

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3

Das, Niladri, Sushma Malik, Prachi Gandhi, Vinaya A. Singh, and Poonam Wade. "Non-immune hydrops fetalis: a case series." International Journal of Contemporary Pediatrics 7, no. 5 (2020): 1195. http://dx.doi.org/10.18203/2349-3291.ijcp20201661.

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Hydrops fetalis is a clinical condition characterized by pathological fluid accumulation in soft tissues and serous cavities of the fetus like peritoneal cavity, pleural cavity, pericardial space, and body wall edema. Hydrops fetalis is broadly classified into Immune Hydrops Fetalis (IHF) and Non-Immune Hydrops Fetalis (NIHF). Incidence of immune hydrops fetalis due to Erythroblastosis fetalis secondary to Rh Iso-immunisation has drastically reduced due to widespread use of anti-D immunoglobulin. In the last few decades, the majority of cases are identified as non-immune hydrops. It is importa

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4

Krasniqi, Faton, Edmond Pistulli, Astrit M. Gashi, and Isabere Krasniqi. "Non-immunologic hydrops fetalis and coronavirus disease (COVID-19) – A case report." Romanian Journal of Pediatrics 70, no. 1 (2021): 75–79. http://dx.doi.org/10.37897/rjp.2021.1.14.

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Non-immune hydrops fetalis is a severe fetal condition defined as the excessive accumulation of fetal fluid within the fetal extravascular compartments and body cavities. The prevalence of non-immune hydrops fetalis is unknown. Currently, about 90% of cases of hydrops fetalis are non-immune hydrops fetalis. Non-immune hydrops fetalis causes are multi-factorial. The pathophysiological mechanism of non-immunologic hydrops fetalis is related to abnormal fluid transportation between plasma and tissues. This is due to the increase in hydrostatic capillary pressure and capillary permeability and a r

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5

Yuan, Shi-Min. "Cardiac Etiologies of Hydrops Fetalis." Zeitschrift für Geburtshilfe und Neonatologie 221, no. 02 (2017): 67–72. http://dx.doi.org/10.1055/s-0042-123825.

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AbstractCardiac etiologies of hydrops fetalis have been a topic of concern due to challenging perinatal management. The common cardiac etiologies leading to hydrops fetalis include structural cardiac anomalies, cardiac dysrhythmias, cardiac tumors, cardiomyopathy and myocarditis. The mechanisms of cardiogenic hydrops fetalis may be: 1) elevation of atrial pressure and volume overload, 2) decrease of cardiac output, and 3) development of congestive heart failure. The diagnosis of hydrops fetalis was usually made at 19–36 gestational weeks, when ultrasound is a highly effective diagnostic method

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6

Lonsdale, Angela. "Hydrops Fetalis." Canadian Journal of Medical Sonography 1, no. 2 (2010): 9–12. http://dx.doi.org/10.3138/cjms.v1i2.9.

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Hydrops fetalis is a complex and fascinating disorder. This article explains the characteristics of each type of hydrops fetalis, immune and non-immune. The etiology and the associated underlying conditions leading to the development of these disorders are also discussed. The ultrasonographic appearance of a classic case of hydrops fetalis is described including variations in its appearance and its numerous differential diagnoses. Several causes of non-immune hydrops fetalis are outlined and briefly discussed. The article concludes with delivery recommendations and discusses a future plan of a

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7

Oliver, Kate E., Kimberly W. Hickey, and Scott M. Petersen. "Spontaneous Resolution of Mirror Syndrome following Demise of Hydropic Twin." Case Reports in Obstetrics and Gynecology 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/783408.

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Maternal mirror syndrome is a rare consequence of fetal hydrops. By convention, delivery is recommended in pregnancies complicated by mirror syndrome due to grave fetal prognosis. We describe a case of a dichorionic, diamniotic twin gestation complicated by hydrops fetalis of twin B. The patient declined selective feticide. Two weeks later, intrauterine fetal demise of fetus B was diagnosed and complete resolution of mirror syndrome followed. Unaddressed, mirror syndrome can lead to significant maternal and fetal complications. This case illustrates resolution of mirror syndrome following spon

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8

González González, Luis Alberto, Zaida Selene Zamora Chávez, Katya Miroslava Mora Rivera, Maria Elizabeth Jimenez Ahumada, and Maria Tsuyuko Shiguematsu. "Caso Clínico: Hydrops Fetal No Inmune." Estudios y Perspectivas Revista Científica y Académica 4, no. 2 (2024): 155–67. http://dx.doi.org/10.61384/r.c.a..v4i2.205.

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Introducción: Se define hydrops fetalis como la presencia de 2 o más colecciones anormales de líquido en el feto, con evidencia mediante ecografía, 9, 11. Estas colecciones de líquido se presentan como ascitis, derrame pleural, derrame pericárdico y edema cutáneo generalizado (con espesor de piel >5 mm), 10, 12. Hay dos categorías de hydrops fetalis: inmune y no inmune. El hydrops fetalis no inmune es específicamente de casos sin aloinmunización con glóbulos rojos, 9. Actualmente se ha repostado que la incidencia de hydrops fetalis inmune a disminuido en los últimos años de manera significa

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9

Agarwal, Arjit, Shubhra Agarwal, Astha Lalwani, Rehana Najam, and Ashutosh Kumar. "Fetal bradyarrhythmia causing hydrops fetalis: A journey from fetal echo to autopsy." Ultrasound 28, no. 4 (2020): 266–70. http://dx.doi.org/10.1177/1742271x20933996.

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Introduction Non-immune hydrops fetalis is a condition with poor fetal prognosis. The incidence of this clinical condition is increasing as compared to its iso-immune variant. The diagnosis of hydrops fetalis is straightforward; however, delineating the primary cause of non-immune hydrops fetalis requires a holistic approach and background knowledge of the entity. Case report We present a case of non-immune hydrops fetalis due to a rare functional cardiac disorder demonstrated by features of cardiac failure in the form of clinically significant tricuspid regurgitation detected on echocardiogra

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10

Randenberg, Andrea. "Nonimmune Hydrops Fetalis Part II: Does Etiology Influence Mortality?" Neonatal Network 29, no. 6 (2010): 367–80. http://dx.doi.org/10.1891/0730-0832.29.6.367.

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HYDROPS FETALIS IS A CONDITION in which there is an excess of total body fluid, primarily within the fetal interstitial spaces.1,2 Etymologically, hydrops fetalis is a Latin term meaning “edema of the fetus.”2,3 In addition to generalized edema, the fetus has at least one of the following: ascites, pericardial effusion, pleural effusion(s), and an abnormally thick (>6 cm) placenta.4–6 Hydrops is classified as nonimmune hydrops fetalis (NIHF) when it occurs without evidence of isoimmunization.7,8

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11

Burdis, Leighann, and Donald D. Koch. "The Use of Cardiopulmonary Bypass and Extracorporeal Membrane Oxygenation for Support During Removal of Two Teratomas and Hydrops Fetalis." Journal of ExtraCorporeal Technology 36, no. 2 (2004): 182–84. http://dx.doi.org/10.1051/ject/2004362182.

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The removal of massive tissue tumors often leads to rapid blood loss and decreased lung compliance because of large volume shifts. Cardiopulmonary bypass (CPB) and extracorporeal membrane oxygenation (ECMO) have both been used as a means of support during resection of saccrococcygeal teratomas (1). Hydrops fetalis is the accumulation of fluid in extravascular spaces and body cavities. This leads to edema and sometimes hypoxia. ECMO has been used for support during treatment of hydrops fetalis (2). This patient was diagnosed, via sonogram, at 30 weeks gestation to have two teratomas and hydrops

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12

Mayer, Beate, and Alexander Weichert. "Frühe und schwer verlaufende hämolytische Erkrankung des Fetus und Neugeborenen." Transfusionsmedizin - Immunhämatologie, Hämotherapie, Immungenetik, Zelltherapie 10, no. 02 (2020): 89–93. http://dx.doi.org/10.1055/a-0972-6307.

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ZusammenfassungEine schwere Alloimmunisierung der Schwangeren gegen erythrozytäre Blutgruppenmerkmale kann insbesondere bei Antikörpern der Spezifität Anti-K bereits vor der 20. Schwangerschaftswoche zu einer fetalen Anämie führen. Wir berichten über eine 37-jährige Schwangere (Gravida 6, Para 5) mit bekanntem Anti-K. In der Vorgeschichte kam es aufgrund einer K-Inkompatibilität in der 4. Schwangerschaft in der 34. Schwangerschaftswoche (SSW) zu einem Hydrops fetalis und in der 5. Schwangerschaft in der 22. SSW zu einem intrauterinen Fruchttod. Mittels eines nicht invasiven Pränataltests (NIPT

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13

Azmi, Jofril, and Roza Sriyanti. "Hydrops Fetalis." JOURNAL OBGIN EMAS 3, no. 2 (2019): 151–55. http://dx.doi.org/10.25077/aoj.3.2.151-155.2019.

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Objective: To report cases of hydrops fetalisMethod: Case reportResults: The case was a female patient aged 36 years, with a diagnosis of G3P1A1H1 gravid 23-24 weeks + Hydrops Fetalis + 1x SC former. On ultrasound examination at 23-24 weeks of age, fetal biometry was found; BPD: 58.9 mm, HC: 211.0 mm, AC: 202.5 mm, FL: 44.4 mm, HL: 40.7 mm, EFW: 417 gr, SDP: 12.79 cm, FHR: 162x / minute, shows anasarcoma edema (+), hydrothoric (+), ascites (+), impression: gravid 23-24 weeks according to biometry, live fetus, Hydrops fetalis, polyhydramnios. Then amniocentesis was carried out followed by a chr

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14

A, Pulatova G., and Yusupbaev R. B. "PRENATAL INVESTIGATION AND MANAGEMENT OF NON-IMMUNE HYDROPS FETALIS." European Journal of Medical Genetics and Clinical Biology 1, no. 8 (2024): 389–93. https://doi.org/10.61796/jmgcb.v1i8.903.

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Background: Advances in medicine have significantly improved our ability to address various diseases affecting the unborn child, with fetal medicine emerging as a rapidly evolving field. Specific Background: Nonimmune hydrops fetalis, a severe antenatal pathology requiring advanced minimally invasive treatment techniques, exemplifies the critical challenges and progress in fetal medicine. Knowledge Gap: Despite advancements, there remains a need for a comprehensive review of nonimmune hydrops fetalis, encompassing its etiology, clinical presentation, and management, particularly with regard to

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15

Vökt, Cora Alexandra, Eva Visca, and Sevgi Tercanli. "Sonographische Befunde bei materno-fetalen Infektionen." Therapeutische Umschau 65, no. 11 (2008): 667–74. http://dx.doi.org/10.1024/0040-5930.65.11.667.

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Mindestens 5% aller Frauen erkranken an einer symptomatischen, viralen Infektion während der Schwangerschaft. Weit mehr Schwangere durchleben eine akute Infektion, welche subklinisch oder gänzlich asymptomatisch verläuft und somit in der Regel unentdeckt bleibt [1]. Eine transplazentare Transmission mit konsekutiver fetaler Infektion ist kein seltenes Ereignis und kann schwerwiegende Folgen für die Schwangerschaft und die Gesundheit des Kindes haben. Die bedeutendsten diesbezüglichen materno-fetalen Infektionen werden unter dem Akronym TORCH (Toxoplasmose, Others, Röteln, Cytomegalie, Herpes)

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16

KHTIRA, Ayoub. "A case of non-immune hydrops fetalis with cystic hygroma complicated by intrauterine fetal death." Journal of Medical Science And clinical Research 11, no. 08 (2023): 90–94. http://dx.doi.org/10.18535/jmscr/v11i8.10.

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Hydrops fetalis refers to the excessive accumulation of fluid in the extracellular compartment of the fetus. Cystic hygroma is a congenital malformation of the cervical lymphatic system responsible for an accumulation of lymphatic fluid commonly in the retrocervical region. Cystic hygromas are most often associated with chromosomal abnormalities. When a cystic hygroma is diagnosed in utero, the fetal survival rate is only 2-6%. When hydrops fetalis is present with cystic hygroma, the mortality rate is close to 100%. We present here the case of a 23-year-old primigravida in whom an ultrasound p

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17

Kosinski, Przemyslaw, Pawel Krajewski, Miroslaw Wielgos, and Aleksandra Jezela-Stanek. "Nonimmune Hydrops Fetalis—Prenatal Diagnosis, Genetic Investigation, Outcomes and Literature Review." Journal of Clinical Medicine 9, no. 6 (2020): 1789. http://dx.doi.org/10.3390/jcm9061789.

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The aim of this paper is to review the outcomes and discuss the genetic and non-genetic aetiology of nonimmune hydrops fetalis in order to support differential ultrasound and genetic evaluations and family counselling. This single-centre study includes all cases of nonimmune hydrops fetalis diagnosed prenatally from 2009 to 2019. Two sources of data were used for this study (prenatal and neonatal) to compare and summarise the findings. Data from genetic testing and ultrasound scans were collected. In total, 33 pregnant women with prenatally diagnosed nonimmune hydrops fetalis were studied. The

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Tholu, Chandana Prasad, Iffat Wakeel Ahmed, Shalini Dange, Amruta Abhijit Choudhary, Anusha Kamath, and Anita Yadav. "Unveiling the elusive: a case of missed hydrops fetalis." International Journal of Research in Medical Sciences 12, no. 7 (2024): 2664–67. http://dx.doi.org/10.18203/2320-6012.ijrms20241932.

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Hydrops fetalis, characterized by abnormal fluid accumulation in fetal tissues, poses diagnostic challenges often leading to adverse outcomes. We present a case of a 26-year-old multigravida diagnosed with hydrops fetalis at 26 weeks, despite unremarkable antenatal visits. Detailed ultrasound revealed characteristic signs, prompting urgent intervention. Despite aggressive measures, the neonate succumbed shortly after birth. This case highlights the complexities of diagnosing and managing hydrops fetalis, emphasizing the need for heightened clinical awareness. Diagnostic steps, including fetal

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19

Ergunay, Koray, Gulcin Altinok, Bora Gurel, et al. "Identifying the etiologic role of Parvovirus B19 in non-immune hydrops fetalis by histopathology, immunohistochemistry and nucleic acid testing: a retrospective study." Open Medicine 2, no. 3 (2007): 271–79. http://dx.doi.org/10.2478/s11536-007-0029-z.

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AbstractIntrauterine Parvovirus B19 infections may cause fetal anemia, non-immune hydrops fetalis or abortion. This study focuses on the pathogenic role of Parvovirus B19 in non-immune hydrops fetalis at Hacettepe University, a major reference hospital in Turkey. Twenty-two cases of non-immune hydrops fetalis were retrospectively selected out of a total of 431 hydrops fetalis specimens from the Department of Pathology archieves. Paraffine embedded tissue sections from placental and liver tissues from each case were evaluated by histopathology, immunohistochemistry, nested PCR and commercial qu

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20

Narayanan, Monisha, Sypara Dhuka, Srilatha Alapati, and Robert P. Kauffman. "Complete resolution of arrhythmia-induced hydrops fetalis in utero." BMJ Case Reports 13, no. 10 (2020): e235827. http://dx.doi.org/10.1136/bcr-2020-235827.

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A 21-year-old G3P2011 Caucasian woman at 27 weeks’ gestation presented with fetal tachyarrhythmia between 240 and 270 beats per minute. Fetal supraventricular tachycardia, abdominal ascites, pleural effusion and pericardial effusion indicated hydrops fetalis. Management with digoxin and flecainide converted the fetus to sinus rhythm and resolved the ascites and pleural effusion within 4 days of treatment. Flecainide was discontinued at 31 weeks’ gestation due to elevated liver enzymes. Intrahepatic cholestasis was treated with ursodiol. Caesarean section was performed at 37 weeks’ gestation. N

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Takeuchi, Makoto, Masahiro Nakayama, Arihiro Tamura, and Hiroyuki Kitajima. "Hydrops Fetalis Due to Agenesis of the Ductus Venosus: New Hepatic Histological Features." Pediatric and Developmental Pathology 12, no. 3 (2009): 239–43. http://dx.doi.org/10.2350/07-10-0363.1.

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We describe the clinical course and autopsy findings of a male infant with hydrops fetalis due to agenesis of the ductus venosus. Fetal echocardiography at 27 weeks in gestation demonstrated hydrops fetalis due to unknown causes. The baby was born at 28 weeks in gestation by emergency caesarean section because of preeclampsia and progressive hydrop fetalis but died immediately at birth. The umbilical vein catheter ran an unusual course: left renal vein and inferior vena cava were opacified after postmortem injection of radiopaque dye into the umbilical vein. The autopsy demonstrated agenesis o

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Kang, Sok-Leng, David Howe, Matthew Coleman, Kevin Roman, and James Gnanapragasam. "Foetal supraventricular tachycardia with hydrops fetalis: a role for direct intraperitoneal amiodarone." Cardiology in the Young 25, no. 3 (2014): 447–53. http://dx.doi.org/10.1017/s104795111400002x.

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AbstractIntroduction: Persistent foetal tachyarrythmias complicated by hydrops fetalis carry a poor prognosis, with foetal death reported in excess of a quarter despite treatment. We present our experience with direct intraperitoneal amiodarone administration in eight hydropic foetuses with resistant supraventricular tachycardia. Methods: Amiodarone was injected slowly into foetal peritoneal cavity under ultrasound guidance. All mothers were loaded with oral amiodarone before the procedure and maintained on it. The procedure was repeated guided by foetal rhythm. Result: All eight cases had sev

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Gao, Guofeng, Jason Tovar, and Katherine Raven. "An Autopsy Case of Severe Nonimmune Hydrops Fetalis Secondary to Premature Closure of the Foramen Ovale." American Journal of Clinical Pathology 152, Supplement_1 (2019): S77. http://dx.doi.org/10.1093/ajcp/aqz114.003.

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Abstract Hydrops fetalis is a condition in the fetus characterized by excessive fluid accumulation within the fetal extravascular compartments and body cavities occurring prenatally. Nonimmune hydrops fetalis is the predominant (>85%) cause of all affected individuals (cardiovascular is the leading etiological category). Herein we present an autopsy case of severe nonimmune hydrops fetalis. The decedent was delivered from a blood type A Rh-positive mother. The overall autopsy findings are diffuse subcutaneous edema, significant ascites, pleural effusion, and pericardial effusion. The major

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Wyatt-Ashmead, Josephine. "Antenatal Closure of the Ductus Arteriosus and Hydrops Fetalis." Pediatric and Developmental Pathology 14, no. 6 (2011): 469–74. http://dx.doi.org/10.2350/07-11-0368.1.

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Antenatal closure of the ductus arteriosus has been speculated, but rarely reported, as a cause of hydrops fetalis. The purpose of this prospective autopsy study was to find the incidence of antenatal closure of the ductus arteriosus and hydrops fetalis in a high-risk obstetric population and to find associated factors that might give clues to the cause of antenatal closure of the ductus arteriosus. Antenatal closure of the ductus arteriosus had to be stringently sought by in situ examination. Fifteen stillborns with antenatal closure of the ductus arteriosus were found in 684 perinatal autops

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Cooper, Nina, Jens-Christian Wolff, and Birgit Ertl. "Erythrozytäre Alloantikörper in der Muttermilch – Ursache einer prolongierten Anämie bei hämolytischer Krankheit des Neugeborenen?" Transfusionsmedizin - Immunhämatologie, Hämotherapie, Immungenetik, Zelltherapie 10, no. 02 (2020): 94–96. http://dx.doi.org/10.1055/a-1085-1093.

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ZusammenfassungMaternale IgG-Alloantikörper gegen fetale erythrozytäre Blutgruppenantigene haben eine klinische Bedeutung in der Auslösung einer hämolytischen Erkrankung des Fetus und Neugeborenen (HDFN). Schwere Fälle gehen mit einer fetalen Anämie, Hydrops fetalis und postnatalem Kernikterus des Neugeborenen einher. Muttermilch enthält hauptsächlich sekretorische IgA-Antikörper, obwohl IgM- und IgG-Isotypen in niedrigeren Konzentrationen detektiert werden konnten. In den letzten Jahren gab es wenige Berichte einer Assoziation erythrozytärer Alloantikörper vom Typ IgG in der Muttermilch mit e

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Remacha, Angel F., Isabel Badell, Núria Pujol-Moix, et al. "Hydrops fetalis–associated congenital dyserythropoietic anemia treated with intrauterine transfusions and bone marrow transplantation." Blood 100, no. 1 (2002): 356–58. http://dx.doi.org/10.1182/blood-2001-12-0351.

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Abstract Hydrops fetalis is rarely caused by congenital dyserythropoietic anemia (CDA). We report a patient with hydrops fetalis as a result of severe anemia. This patient needed intrauterine transfusions from 21 weeks of gestation until birth. The hematologic study showed an atypical CDA (hydrops fetalis–associated CDA) characterized by features resembling CDA type II, but negative acidified serum lysis test (HEMPAS negative). The patient was regularly transfused for a year, after which an allogeneic bone marrow transplantation (BMT) from an HLA-identical sibling was successfully carried out.

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Ho, Sherry Sze Yee, Samuel S. Chong, Evelyn SC Koay, et al. "Microsatellite Markers within —SEA Breakpoints for Prenatal Diagnosis of HbBarts Hydrops Fetalis." Clinical Chemistry 53, no. 2 (2007): 173–79. http://dx.doi.org/10.1373/clinchem.2006.075085.

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Abstract Background: We sought to develop a rapid prenatal diagnostic test for simultaneous detection of HbBarts hydrops fetalis and exclusion of maternal contamination. Methods: We developed a multiplex quantitative fluorescent PCR (QF-PCR) test that detects the presence/ absence of 2 microsatellite markers (16PTEL05/16PTEL06) located within breakpoints of the Southeast Asia (—SEA) deletion. HbBarts hydrops fetalis (—SEA/—SEA) is diagnosed by absence of both markers, and maternal contamination of fetal DNA is excluded by absence of noninherited maternal alleles. Fetal and parental DNA samples

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Mascaretti, Renata Suman, Mário Cícero Falcão, Andrea M. Silva, Flávio Adolfo Costa Vaz, and Cléa Rodrigues Leone. "Characterization of newborns with nonimmune hydrops fetalis admitted to a neonatal intensive care unit." Revista do Hospital das Clínicas 58, no. 3 (2003): 125–32. http://dx.doi.org/10.1590/s0041-87812003000300001.

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PURPOSE: To determine the incidence and characteristics of nonimmune hydrops fetalis in the newborn population. METHOD: A retrospective study of the period between 1996 and 2000, including all newborns with a prenatal or early neonatal diagnosis of nonimmune hydrops fetalis, based on clinical history, physical examination, and laboratory evaluation. The following were analyzed: prenatal follow-up, delivery type, gender, birth weight, gestational age, presence of perinatal asphyxia, nutritional classification, etiopathic diagnosis, length of hospital stay, mortality, and age at death. RESULTS:

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Simoncic, Milanka, Silvo Kopriva, Ziva Zupancic, et al. "Mediastinal teratoma with hydrops fetalis in a newborn and development of chronic respiratory insufficiency." Radiology and Oncology 48, no. 4 (2014): 397–402. http://dx.doi.org/10.2478/raon-2013-0080.

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Abstract Background. Mediastinal fetal teratoma can be detected as a mass in the chest during a routine prenatal ultrasound screening. Because of the pressure on mediastinal structures it can be the cause of non-immune hydrops fetalis and polyhydramnion. The development of hydrops fetalis leads to fetal death or premature delivery in most reported cases. Early surgical removal is important, but, the result of treatment depends on the stage of development of mediastinal organs and complications in the postoperative period. Case report. A 31-year-old gravida carrying twins, with spontaneous memb

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Yanase, Yuri, Sirinart Sirilert, Phudit Jatavan, Mallika Pomrop, Krittaya Phirom, and Theera Tongsong. "Hydrops Fetalis Caused by Congenital Syphilis: Case Series and a Comprehensive Review." Journal of Clinical Medicine 14, no. 11 (2025): 3671. https://doi.org/10.3390/jcm14113671.

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A total of 30 hydropic fetuses, including 25 cases from published reports and 5 from our own series, were reviewed, validated, and analyzed. This review yielded the following key findings: (1) Unlike most cases of nonimmune hydrops fetalis (NIHF), hydrops caused by syphilis is not only preventable but also treatable, with complete resolution possible when appropriately managed. (2) Syphilis-associated hydrops carries a poor prognosis if timely and appropriate treatment is not administered. (3) Based on limited data, intravenous penicillin G is probably more effective than intramuscular benzath

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Dumova, S. V., H. A. Sarkisyan, O. L. Chugunova, et al. "Non-immune hydrops fetalis: etiology, pathogenesis, features of diagnosis and treatment in the fetus and newborn." CHILDREN INFECTIONS 23, no. 1 (2024): 35–43. http://dx.doi.org/10.22627/2072-8107-2024-23-1-35-43.

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Non-immune hydrops fetalis is a polyetiological disease characterized by high perinatal mortality. The development of non-immune fetal hydrops is associated with diseases of the cardiovascular and lymphatic systems, genetic and chromosomal diseases, infectious agents and many other reasons. In this regard, there is no single approach to the treatment of this extremely dangerous pathological condition, which requires the joint work of specialists from different specialties: obstetricians-gynecologists, neonatologists, cardiologists, surgeons, hematologists and geneticists. This article provides

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Al-Riyami, Arwa Z., Mouza Al-Salmani, Sabria N. Al-Hashami, et al. "Intrauterine Fetal Blood Transfusion: Descriptive study of the first four years’ experience in Oman." Sultan Qaboos University Medical Journal [SQUMJ] 18, no. 1 (2018): 34. http://dx.doi.org/10.18295/squmj.2018.18.01.006.

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Objectives: Haemolytic disease of the fetus and newborn (HDFN) causes hydrops fetalis. The successful treatment of HDFN has been reported with intrauterine blood transfusion (IUT). This study aimed to describe the initial experience with IUT procedures in Oman. Methods: This retrospective observational study took place at the Royal Hospital and Sultan Qaboos University Hospital Blood Bank, Muscat, Oman, and included all women who underwent IUT procedures in Oman between March 2012 and March 2016. Gestational and neonatal outcomes were assessed, including complications, morbidity, neurodevelopm

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Buran, Cebrail, and Betül Tosun. "Nursing Care According to Gordon's Functional Health Patterns Model: A Case Presentation of a Baby with Hydrops Fetalis and Meningomyelocele." Ordu Üniversitesi Hemşirelik Çalışmaları Dergisi 8, no. 1 (2025): 238–51. https://doi.org/10.38108/ouhcd.1394437.

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Objective: This study presents a rare and complex case of concurrent hydrops fetalis and meningomyelocele in an infant. Methods: The patient was assessed using Gordon's functional health pattern model during the study conducted between April 30, 2022, and June 30, 2022. This model was employed to plan nursing care and provide guidance. It encompasses 11 functional patterns used to evaluate vital processes and daily life, specifically for babies with hydrops fetalis and meningomyelocele. Results: The study highlights the challenges posed by the co-occurrence of two conditions, hydrops fetalis a

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O'Malley, Aiveen, Carole Barry-Kinsella, Caroline Hughes, et al. "Parvovirus Infects Cardiac Myocytes in Hydrops Fetalis." Pediatric and Developmental Pathology 6, no. 5 (2003): 414–20. http://dx.doi.org/10.1007/s10024-001-0269-x.

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Parvovirus infection during pregnancy is an important cause of hydrops fetalis. It is attributed to anemia caused by viral-induced destruction of red blood cells. Infection of other organs has been reported including the heart, liver, and lungs. Few of these reports, however, convincingly demonstrate virions within the functional parenchyma of the tissue. This is of particular concern regarding myocardium in the context of hydrops fetalis which is, in part, due to cardiac failure. The problem in routine pathology practice is that most fetuses with the infection are macerated. This, in part, pr

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Bajpai, Shivani, Kausik Mandal, Kirti Naranje, and Anita Singh. "Galactosialidosis presenting as non-immune hydrops." BMJ Case Reports 17, no. 10 (2024): e260906. http://dx.doi.org/10.1136/bcr-2024-260906.

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Hydrops fetalis is an abnormal accumulation of fluid in two or more foetal compartments which is easily detected using prenatal ultrasonography. It can be categorised into immune and non-immune. The non-immune hydrops can result from various aetiologies, including cardiovascular, respiratory, genitourinary infections, chromosomal anomalies and metabolic causes. The metabolic causes, including lysosomal storage disorders (LSD), are increasingly being recognised as the causes of non-immune hydrops. The hydrops fetalis associated with metabolic disorders is usually severe with huge ascites, hepat

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Popescu, Daniela Eugenia, Andreea Cioca, Cezara Muresan, et al. "A Case of COVID-19 Pregnancy Complicated with Hydrops Fetalis and Intrauterine Death." Medicina 57, no. 7 (2021): 667. http://dx.doi.org/10.3390/medicina57070667.

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Coronavirus disease 2019 (COVID-19) has rapidly evolved into a worldwide pandemic causing a serious global public health problem. The risk of vertical transmission of SARS-CoV-2 is still debated, and the consequences of this virus on pregnant women and their fetuses remain unknown. We report a case of pregnancy complicated with hydrops fetalis that developed 7 weeks after recovery from a mild SARS-CoV-2 infection, leading to intrauterine death of the foetus. Evidence of SARS-CoV-2 placentitis was demonstrated by the presence of viral particles in the placenta identified by immunohistochemistry

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Patel, Bhavya, Abhijit Mehta, Binoy Shah, Ashish Mehta, and Manish Shah. "A preterm neonate with fetal anemia and immune hydrops fetalis requiring intrauterine transfusion and postnatal exchange transfusion: a case report." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 13, no. 7 (2024): 1873–77. http://dx.doi.org/10.18203/2320-1770.ijrcog20241794.

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Hydrops fetalis is a presenting illness with various immune and non-immune etiologies. It involves fluid accumulation in body cavities, and symptoms specific to its underlying cause. In this case, we report on a preterm neonate with a history of bad obstetrics who presented with hydrops fetalis due to fetal anemia related to RH incompatibility. The patient received an intrauterine transfusion for severe fetal anemia and subsequently required NICU admission. Routine preterm care was provided, along with specific management for jaundice resulting from isoimmune hemolytic anemia.

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Hasnain, Afia, Sherri Burnett, Ronald Agatep, Elizabeth Spriggs, Bernard Chodirker, and Aizeddin (Aziz) A. Mhanni. "Prenatal hydrops fetalis associated with infantile free sialic acid storage disease due to a novel homozygous deletion in the SLC17A5 gene." Molecular Case Studies 7, no. 5 (2021): a006106. http://dx.doi.org/10.1101/mcs.a006106.

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Nonimmune hydrops fetalis, the excessive accumulation of serous fluid in the subcutaneous tissues and serous cavities of the fetus, has many possible etiologies, providing a diagnostic challenge for the physician. Lysosomal storage diseases have been reported in up to 5%–16% of nonimmune hydrops fetalis pregnancies. Infantile free sialic acid storage disease (ISSD) (OMIM #269920) is a severe form of autosomal recessive sialic acid storage disease. ISSD is caused by mutations in SLC17A5 (OMIM #604322), which encodes sialin, a lysosomal-membrane sialic acid transporter. We describe a case of fet

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Bührer, C. "Konnatale CMV-Infektionen." Kinder- und Jugendmedizin 14, no. 02 (2014): 75–78. http://dx.doi.org/10.1055/s-0038-1629377.

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ZusammenfassungWährend effektive Präventionsstrategien zu einem deutlichen Rückgang der meisten vertikalen Infektionen geführt haben (Röteln, HIV, Hepatitis B, Lues, B-Streptokokken), dürften jedes Jahr in Deutschland mehrere Tau-send Kinder mit einer konnatalen Zytomegalie-Virus (CMV)-Infektion auf die Welt kommen, von denen schätzungsweise 90 % jedoch nicht diagnostiziert werden. Während in Mitteleuropa jeweils rund die Hälfte der konnatalen CMV-Infektionen Kinder betrifft, deren Mütter ihre CMV-Erstinfektion entweder in der Schwangerschaft oder bereits vor der Schwangerschaft durchgemacht h

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Huang, Yu-Yun, Yu-Jun Chang, Lih-Ju Chen, et al. "Survival of Hydrops Fetalis with and without Fetal Intervention." Children 9, no. 4 (2022): 530. http://dx.doi.org/10.3390/children9040530.

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Objectives: To investigate the survival rate of hydrops fetalis after fetal interventions and neonatal intensive care. Methods: We reviewed the medical records of patients diagnosed with hydrops fetalis from January 2009 to December 2019 at Changhua Christian Children’s Hospital. All cases had abnormal fluid accumulation in at least two body compartments during pre- and postnatal examination. The primary outcome measure was the mortality rate. We also collected information regarding disease etiology, duration of hospital stay, Apgar score, gestational age at birth, initial hydrops fetalis diag

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Janssen, Olivia, and Jing Lin. "Postnatal IVIG treatment for persistent anaemia in neonate due to congenital parvovirus infection." BMJ Case Reports 14, no. 1 (2021): e237393. http://dx.doi.org/10.1136/bcr-2020-237393.

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Congenital parvovirus B19 infection is a rare but serious condition that can result in hydrops fetalis and fetal death. Due to the virus’ cytotoxic effect on fetal red blood cell precursors, postnatal infection can cause a neonatal viremia and secondary pure red cell aplasia. Here, we describe a case of congenital parvovirus infection in a preterm infant complicated by hydrops fetalis and chronic anaemia that responded to postnatal treatment with intravenous immunoglobulin administered on day of life 44. After treatment, the anaemia resolved as the neonate exhibited interval increases in haemo

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Janssen, Olivia. "Neonatal Intravenous Immunoglobulin (IVIG) Treatment for Chronic Anemia Due to Congenital Parvovirus Infection: A Case Report." Corpus Journal of Case Reports (CJCR) 01, no. 01 (2020): 1–2. http://dx.doi.org/10.54026/cjcr/1002.

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Congenital Parvovirus B19 infection is a rare but serious condition that can result in hydrops fetalis and fetal death. Due to the virus’ cytotoxic effect on fetal red blood cell precursors, postnatal infection can cause a neonatal viremia and secondary pure red cell aplasia. Here, we describe a case of congenital parvovirus infection in a preterm infant complicated by hydrops fetalis and chronic anemia that responded to postnatal treatment with intravenous immunoglobulin (IVIG) administered on day of life 44. After treatment, the anemia resolved as the neonate exhibited interval increases in

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Yusrawati, Yusrawati, and Dona Mirsa Putri. "A Case of Prenatal Diagnosis of Turner Syndrome with Ultrasonography." Journal Obgin Emas 5, no. 1 (2021): 119–29. http://dx.doi.org/10.25077/aoj.5.1.119-129.2021.

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Objective: To report a case of Turner syndrome diagnosed in prenatal care.Method: A case report.Case: Case of Mrs. Y 26-year-old woman G2P1A0L1 diagnosed on 19-20 weeks of gestation with Turner syndrome. Ultrasound examination findings were hydrops fetalis on the neck region, multiple septated cystic masses, subcutaneous edema, ascites, and hydrothorax. Subsequently, amniocentesis was performed and the chromosome analysis result showed Turner syndrome (45, X0). The patient was induced vaginal delivery on 22-23 weeks of pregnancy due to intrauterine fetal death indication. The baby was born wit

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Bets, Olga G., Irina G. Khmelevskaya, Alexandra V. Seriozhkina, Anna A. Bulka, Lyubov G. Khodulapova, and Olga S. Yavlyanskaya. "Clinical case of non-immune hydrops in a preterm infant with IgG to SARS-CoV-2 at birth." Человек и его здоровье 25, no. 2 (2022): 4–12. http://dx.doi.org/10.21626/vestnik/2022-2/01.

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Non-immune hydrops of the fetus is a heterogeneous pathology, which is accompanied by excessive accumulation of fluid in two or more serous cavities (pericardial cavity, abdominal cavity, pleural cavity) and fetal tissues in the absence of signs of immune sensitization. This pathology in most cases leads to high mortality in both the antenatal and postnatal periods. The incidence of this disease ranges from 0.05 to 5%. Perinatal mortality in nonimmune hydrops fetalis accounts for about 80%, but with the manifestation of the disease in the early stages of pregnancy, the frequency of deaths is a

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Gaither, Kecia, Andrea Ardite, and Sarita Dhuper. "In Utero Diagnosis of Agenesis of the Ductus Arteriosus in a Twin Pregnancy: An Unusual Case Presentation." ISRN Obstetrics and Gynecology 2011 (December 1, 2011): 1–3. http://dx.doi.org/10.5402/2011/258431.

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Arora, Sunita, Gursharn Singh Narang, Anmol Kaur, and Taranjeet Kaur. "Congenital Chylothorax as a Cause of Non Immune Hydrops: A Case Report." Journal of Nepal Paediatric Society 42, no. 2 (2022): 89–91. http://dx.doi.org/10.3126/jnps.v42i2.39150.

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Chylothorax is a rare cause of non immune hydrops fetalis & presents with respiratory distress at birth. We present a late preterm diagnosed antenatally as hydrops with chylothorax with bilateral pleural effusion and respiratory distress at birth requiring mechanical ventilation. Baby was managed successfully with intercostal drainage, octreotide infusion & MCT milk formula.

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Singh, Paul, and Matthew Connell. "Primary Congenital Lymphedema Complicated by Hydrops Fetalis: A Case Report and Review of the Literature." Case Reports in Obstetrics and Gynecology 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/186173.

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Introduction. Primary congenital lymphedema is a rare disorder associated with insufficient development of lymphatic vessels. Usually most patients present with lower extremity edema seen sonographically. Rarely primary congenital lymphedema may be associated with severe lymphatic dysfunction resulting in hydrops fetalis.Case. A 27-year-old primigravida with a family history of leg swelling throughout multiple generations was diagnosed early in the third trimester with hydrops fetalis. Delivery was undertaken at 32 weeks for nonreassuring fetal status and the infant expired at approximately 45

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Paul, Jerin Monisha, Prabhukumar Sakthigirisamy, Ilangumaran Lakshmanan, Sucindar Mullainathan, and Saravanan Sambandam. "Giant fetal lymphangioma with non-immune hydrops fetalis." International Journal of Contemporary Pediatrics 7, no. 10 (2020): 2080. http://dx.doi.org/10.18203/2349-3291.ijcp20204056.

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Fetal lymphangioma is one of the rarest congenital malformation occuring in the newborn. We are reporting a giant fetal lymphangioma which may be the largest one so far reported in medical literature and its association with non immune hydrops fetalis. A late preterm 35+3 weeks gestation female neonate with birth weight of 3.8 kg was delivered via emergency caesarean section, for prevention of birth injuries in view of large for gestation. Baby had weak cry at birth. On examination a giant cystic mass extending from right hemithorax to right anterolateral abdominal wall measuring 25×12.5×9 cm

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Jojić, Dragica, Jelica Predojević-Samardžić, Gordana Guzijan, and Snežana Petrović-Tepić. "Immune hydrops fetalis." Scripta Medica 46, no. 1 (2015): 80–83. http://dx.doi.org/10.5937/scrimed1501080j.

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Chan, Elaine S., Julie Lauzon, Lothar Resch, Bob Argiropoulos, Laura Schmitt, and Rati Chadha. "Atypical Prenatal Ultrasound Presentation and Neuropathological Findings in a Neonate With Alpha Thalassemia Major: A Case Report." Pediatric and Developmental Pathology 22, no. 2 (2018): 166–70. http://dx.doi.org/10.1177/1093526618817655.

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Alpha thalassemia major is a hemoglobinopathy caused by the inactivation or deletion of all 4 α-globin alleles. We describe a case of α-thalassemia major with atypical ultrasound and neuropathological findings. The mother had her first prenatal visit at 27 4/7 gestational weeks. Ultrasound revealed a hydropic fetus with multiple anomalies. However, the middle cerebral artery peak systolic velocity (MCA-PSV) suggested that the likelihood of fetal anemia was low. Given the poor prognosis of hydrops fetalis, the parents opted for termination of pregnancy. The neonate died shortly after birth. Aut

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