Relevant bibliographies by topics / Hypercholesterolemie

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hypercholesterolemie'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 June 2024

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Journal articles on the topic "Hypercholesterolemie"

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Nussbaumerová, Barbora, and Hana Rosolová. "Epidemiology of hypercholesterolemia." Vnitřní lékařství 64, no. 1 (January 1, 2018): 30–37. http://dx.doi.org/10.36290/vnl.2018.005.

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DE BACKER G. "Geïsoleerde hypercholesterolemie." Tijdschrift voor Geneeskunde 55, no. 22 (January 1, 1999): 1625. http://dx.doi.org/10.2143/tvg.55.22.5000602.

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DE BACKER G. "Geïsoleerde hypercholesterolemie." Tijdschrift voor Geneeskunde, no. 22 (January 1, 1999): 1625. http://dx.doi.org/10.47671/tvg.55.22.5000602.

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Češka, Richard, Michaela Šnejdrlová, Michal Vrablík, Tereza Altschmiedová, and Tomáš Štulc. "Treating hypercholesterolaemia with evolocumab." Intervenční a akutní kardiologie 17, no. 4 (December 1, 2018): 212–17. http://dx.doi.org/10.36290/kar.2018.057.

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VAN CLEEMPUT J. "Zware hypercholesterolemie: behandeling?" Tijdschrift voor Geneeskunde, no. 4 (2009): 159–60. http://dx.doi.org/10.47671/tvg.65.04.2000488.

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Karásek, David. "Ezetimibe in the treatment of hypercholesterolaemia." Interní medicína pro praxi 21, no. 2 (May 10, 2019): 112–16. http://dx.doi.org/10.36290/int.2019.017.

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 . "Therapietrouw bij familiaire hypercholesterolemie." Medisch-Farmaceutische Mededelingen 41, no. 6 (June 2003): 180. http://dx.doi.org/10.1007/bf03058199.

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Broekhuizen, Lidewij. "Spoor familiaire hypercholesterolemie op." Huisarts en wetenschap 60, no. 1 (January 2017): 3. http://dx.doi.org/10.1007/s12445-017-0003-1.

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Lafeber, M., and GA Rongen. "PCSK9-remmers bij familiaire hypercholesterolemie." Ge-Bu 108-115, no. 10 (2021): 1. http://dx.doi.org/10.35351/gebu.nl.2021.10.20.

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Wiegman, A. "Vetstofwisselingsafwijkingen bij kinderenfamiliaire hypercholesterolemie cholesterol." Bijblijven 19, no. 2 (February 2003): 79–82. http://dx.doi.org/10.1007/bf03059691.

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Dissertations / Theses on the topic "Hypercholesterolemie"

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Boileau, Catherine, and J. P. GELOSO. "Syndrome de marfan et hypercholesterolemie : modeles d'heterogeneite genetique." Paris 7, 1993. http://www.theses.fr/1993PA077318.

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Le syndrome de marfan (sm) et l'hypercholesterolemie familiale (hf) correspondent a deux groupes de pathologies a transmission autosomique dominante pour lesquelles une heterogeneite genetique a longtemps ete soupconnee. Pour identifier les facteurs genetiques associes a ces maladies, nous avons utilise une approche combinant des methodes genetiques et moleculaires, appliquees tant a des analyses de familles que de populations. Par l'approche gene candidat utilisee dans l'etude d'une grande famille presentant une forme variante du sm, nous avons montre que, a la difference des formes classiques, la maladie n'etait pas liee au gene de la fibrilline (fib15). Ce resultat a conduit a la definition d'une nouvelle entite clinique et moleculaire. Apres l'exclusion de genes codant pour d'autres constituants de la matrice extracellulaire, nous avons entrepris une carte d'exclusion genomique qui a deja permis d'eliminer plus de 65% du genome. Dans le cas de l'hf, nous avons identifie 7 nouvelles mutations du gene du recepteur ldl (ldlr) demontrant ainsi une importante heterogeneite allelique. Nous avons aussi retrouve la mutation 3500 du gene de l'apo b (apob) chez un sujet. Cette mutation etait associee a un haplotype identique a celui rapporte chez d'autres proposants, confirmant la nature unique et europeenne de cette mutation. Par ailleurs, la liaison aux genes ldlr et apob a pu etre exclue dans une fraction importante de familles de hf, demontrant ainsi l'implication d'au moins un troisieme facteur genetique a effet dominant. Enfin, nous n'avons pas observe de desequilibre de liaison entre l'hypercholesterolemie et les marqueurs des genes apoa1-c3-a4. L'ensemble de ces resultats va maintenant etre suivie de l'identification des nouveaux facteurs genetiques impliques dans le sm et l'hf permettant ainsi de tendre vers des tests de diagnostic specifiques
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BARRET, FRANCOISE. "Hypercholesterolemie familiale de l'enfant : a propos de deux observations." Limoges, 1988. http://www.theses.fr/1988LIMO0127.

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COLOMES, MARYVONNE. "Hypercholesterolemies : etude de l'efficacite et de la tolerance de la fluvastatine." Toulouse 3, 1991. http://www.theses.fr/1991TOU31523.

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DIEBOLT, PASCAL. "Les hypercholesterolemies : nouvelle approche therapeutique." Strasbourg 1, 1990. http://www.theses.fr/1990STR15042.

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Bent, Patrick. "Lipides et qualites du sperme." Clermont-Ferrand 1, 1992. http://www.theses.fr/1992CLF13832.

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DE, LA FARGE FLORENCE. "Interets du bilan lipidique chez le sujet age." Toulouse 3, 1992. http://www.theses.fr/1992TOU31126.

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VANEL, NATHALIE. "L'hypercholesterolemie dans le sang du cordon humain : recherche d'une signification pronostique." Lyon 1, 1989. http://www.theses.fr/1989LYO1M480.

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SETBON, JEAN-MARC. "L'hypercholesterolemie familiale homozygote : possibilites therapeutiques actuelles ; a propos de deux cas d'une meme fratrie." Aix-Marseille 2, 1989. http://www.theses.fr/1989AIX20046.

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Chanial, Christine. "Incidence de l'hypothyroi͏̈die dans l'hypercholestérolémie : étude prospective sur 135 cas." Montpellier 1, 1991. http://www.theses.fr/1991MON11095.

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FAUROUX, MARIE-FRANCOISE. "Etude comparative pravastatine : cholestyramine chez 67 patients atteints d'hypercholesterolemie familiale." Toulouse 3, 1989. http://www.theses.fr/1989TOU31033.

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Books on the topic "Hypercholesterolemie"

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Action plan for high cholesterol. Champaign, IL: Human Kinetics, 2006.

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Geddis, Alison Elaine. Haemostasis in hypercholesterolemia. [S.l: The Author], 1992.

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Gayle, Baird, and University College Dublin. Centre for Health Economics., eds. The economics of treating hypercholesterolemia. Dublin: University College Dublin, Centre for Health Economics, 1996.

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1924-, Stokes Joseph, and Mancini M, eds. Hypercholesterolemia: Clinical and therapeutic implications. New York: Raven Press, 1988.

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Malmendier, Claude L., P. Alaupovic, and H. Bryan Brewer, eds. Hypercholesterolemia, Hypocholesterolemia, Hypertriglyceridemia, in Vivo Kinetics. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5904-3.

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International, Colloquium on Atherosclerosis (5th 1990 Brussels Belgium). Hypercholesterolemia, hypocholesterolemia, hypertriglyceridemia, in vivo kinetics. New York: Plenum Press, 1991.

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1922-, Steinberg Daniel, and Olefsky Jerrold M, eds. Hypercholesterolemia and atherosclerosis: Pathogenesis and prevention. New York: Churchill Livingstone, 1987.

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Gross, Peter Lawrence. Modification of rabbit platelet function in hypercholesterolemia. Ottawa: National Library of Canada, 1991.

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C, Defesche Joep, and Lansberg Peter J, eds. The molecular basis and treatment of familial hypercholesterolemia. Amsterdam: Thesis Publishers, 1993.

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Association, American Heart. Dietary treatment of hypercholesterolemia: A manual for patients. Dallas, Tex: The Association, 1988.

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Book chapters on the topic "Hypercholesterolemie"

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Erkelens, D. W. "Wat is het controleschema bij familiaire hypercholesterolemie?" In Vademecum permanente nascholing huisartsen, 2334. Houten: Bohn Stafleu van Loghum, 2006. http://dx.doi.org/10.1007/978-90-313-8808-0_1229.

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Walma, E. P., and Tj Wiersma. "NHG-Standpunt Diagnostiek en behandeling van familiaire hypercholesterolemie." In NHG-Standaarden voor de huisarts 2009, 153–59. Houten: Bohn Stafleu van Loghum, 2009. http://dx.doi.org/10.1007/978-90-313-6614-9_7.

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Walma, EP, and Tj Wiersma. "NHG-Standpunt Diagnostiek en behandeling van familiaire hypercholesterolemie." In NHG- Standaarden voor de huisarts 2011, 409–15. Houten: Bohn Stafleu van Loghum, 2011. http://dx.doi.org/10.1007/978-90-313-8279-8_21.

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Erkelens, D. W. "Wat is het effect van de verschillende behandelingen bij hypercholesterolemie?" In Vademecum permanente nascholing huisartsen, 2184–85. Houten: Bohn Stafleu van Loghum, 2006. http://dx.doi.org/10.1007/978-90-313-8808-0_1153.

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Erkelens, D. W. "Wat is de behandeling van familiaire hypercholesterolemie op de kinderleeftijd (6-16 jaar)?" In Vademecum permanente nascholing huisartsen, 2435. Houten: Bohn Stafleu van Loghum, 2006. http://dx.doi.org/10.1007/978-90-313-8808-0_1285.

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Kastelein, J. J. P., and H. J. Avis. "Is het éénmalig bepalen van het cholesterolgehalte voldoende om familiaire hypercholesterolemie uit te sluiten?" In Vademecum permanente nascholing huisartsen, 1984–85. Houten: Bohn Stafleu van Loghum, 2006. http://dx.doi.org/10.1007/978-90-313-8808-0_1055.

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder, et al. "Hypercholesterolemia." In Encyclopedia of Behavioral Medicine, 1008. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100841.

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Sainburg, Robert L., Andrew L. Clark, George E. Billman, Zachary J. Schlader, Toby Mündel, Kevin Milne, Earl G. Noble, et al. "Hypercholesterolemia." In Encyclopedia of Exercise Medicine in Health and Disease, 415. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2504.

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Agha, Ali, and Christie M. Ballantyne. "Hypercholesterolemia." In Contemporary Cardiology, 61–71. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-98824-1_4.

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Gumina, Stefano, Vittorio Candela, and Daniele Passaretti. "Hypercholesterolemia." In Rotator Cuff Tear, 83–85. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33355-7_10.

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Conference papers on the topic "Hypercholesterolemie"

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Purwanti, Nunik, Rahmadiar Aditya Putri, and Siti Nurjanah. "Effectiveness of Sembung Tree Honey on Decreasing Cholesterol Level in Hypercolesterolemia." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.05.22.

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ABSTRACT Background: Lifestyle changes, causing an increase in cholesterol. Honey has done a lot of research. The content of flavonoids can lower cholesterol. Many people consume Sembung tree honey, but not much research has been done, including its effectiveness in reducing cholesterol. This study aimed to analyze the effectiveness of sembung tree honey to reduce cholesterol levels in hypercholesterolemia. Subjects and Method: This was a quasi-experiment with pretest and posttest control group. A sample of 20 hypercholesterolemia sufferers in Kedensari Village was selected by purposive sampling. The dependent variable was hypercholesterolemia. The independent variable was sembung tree honey. The data was analyzed by Wilcoxon. Results: Cholesterol level after intervention (<240 mg / dL), lower before intervention (> 240 mg / dl), significant value p = 0.000. The mean coverage of cholesterol levels after the intervention (Mean = 185.50; SD = 6.05) was lower than that before the intervention (Mean = 247.20; SD = 6.96), it was statistically significant (p <0.001). Conclusion: Sembung tree honey is effective in reducing cholesterol levels. Keywords: Sembung tree honey, Hypercholesterolemia Correspondence: Nunik Purwanti. Faculty of Nursing and Midwifery, Universitas Nahdatul Ulama, Surabaya. Jl SMEA No 57 Surabaya. Email: noniek@unusa.ac.id. Mobile: 082141511655 DOI: https://doi.org/10.26911/the7thicph.05.22
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Medini, L., P. Maderna, E. Tremoli, and C. Galli. "PLATELETS FROM TYPE IIA HYPERCHOLESTEROLEMIC PATIENTS GENERATE MORE INOSITOLPHOSPHATES AFTER THROMBIN STIMULATION IN COMPARISON WITH THOSE OF NORMAL SUBJECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643415.

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Functional and biochemical responses of platelets to stimulating agents have been reported to be amplified in several pathological conditions, including hyperlipidemia. Enhanced aggregation and thromboxane formation are, e.g. frequently observed in the presence of high plasma cholesterol levels (type Ila hypercholesterolemia). Stimulation of phosphoinositides breakdown through specific phosphohydrolases (phospholipase C) resulting in the formation of inositolphosphates (IPs), is one of the early events in platelet activation. A study was thus designed in order to investigate IPs generation in thrombin stimulated platelets from type Ila hypercholesterolemic patients in comparison with a group of normocholesterolemic subjects. Preparation of washed platelets, prelabelling with 2[3H] myo inositol and separation of lipid and water soluble inositides was carried out according to Watson et al (1). Product generation (inositoltrisphosphate, IP3; inositolbisphosphate, IP2; inositol mono phosphate, IP) in relation to the 2[3H) inositol incorporated in phosphoinositides was evaluated at 10 and 90 s after platelet stimulation with 1 U/ml NIH in the presence of 10 mM lithium chloride. The major differences found in platelets from type Ila patients in comparison with those of controls were the following:1) in non stimulated platelets a greater incorporation of myoinositol in phosphoinositides and lower levels of labelled IP2; 2) after stimulation, the levels of all labelled IPs were significantly greater, and those of IP2 were double than in controls. In particular the percent increment of IP2 over basal values in platelets of type Ila patients was more than two fold greater. It is concluded that the enhanced generation of IPs in platelets from type IIa patients, following thrombin stimulation, may contribute to the greater sensitivity to agonists of the aggregatory process in this pathological condition.1) Watson P.S., McConnell R.T. and Lapetina E.G., J. Biol. Chem.21:13199, 1984
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Baba, Waqas, and Sajid Maqsood. "Novel antihypertensive and anticholesterolemic peptides from peptic hydrolysates of camel whey proteins." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/qecs2081.

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Hypercholesterolemia and hypertension are major growing concerns that are managed by drugs that inhibit various metabolic enzymes. Milk hydrolysates have been reported to contain various bioactive peptides (BAP) that can inhibit various metabolic enzymes for enhancing human health. As such camel whey proteins were subjected to peptic hydrolysis using a full factorial model (33) with hydrolysis time, temperature, and enzyme concentration as factors. The resulting hydrolysates were analyzed for anti-hypercholesterolemic and hypertensive properties by studying the in vitro inhibition of various enzymatic markers. The hydrolysates with lowest IC50 values were further subjected to LC-MS-QTOF that revealed presence of 185 peptides. Selected peptides that had Peptide Ranker Score greater than 0.8 were further studied for prediction of possible interactions with enzyme markers: pancreatic lipase (PL) cholesterol esterase (CE) and angiotensin converting enzyme (ACE) using in silico analysis. The data generated suggested that most of the peptides could bind active site of PL while as only three peptides could bind active site of CE. Based on higher number of reactive residues in the bioactive peptides (BAP) and greater number of substrate binding sites, FCCLGPVPP was identified as potential CE inhibitory peptide while PAGNFLPPVAAAPVM, MLPLMLPFTMGY, and LRFPL were identified as PL inhibitors. While peptides PAGNFLP, FCCLGPVPP, PAGNFLMNGLMHR, PAVACCLPPLPCHM were identified as potential ACE inhibitors. Molecular docking of selected peptides showed hydrophilic and hydrophobic interactions between peptides and target enzymes. Moreover, due to the importance of renin in managing hypertension, peptides from hydrolysates with high ACE inhibiting potential were predicted for potential to interact with renin using in silico analysis. Molecular docking was subsequently employed to identify how the identified peptides, PVAAAPVM and LRPFL, could interact with renin and potentially inhibit it. Thus, non-bovine (camel) whey hydrolysates might be used as functional ingredients for production of functional foods with antihypertensive and anticholesterolemic properties.
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Lan, Xiufeng, Jiangang Liu, Ying Liu, Xiaosen Luo, Jian Lu, and Xiaowu Ni. "Fluorimetric determination of cholesterol in hypercholesterolemia serum." In Photonics Asia 2004, edited by Britton Chance, Mingzhe Chen, Arthur E. T. Chiou, and Qingming Luo. SPIE, 2005. http://dx.doi.org/10.1117/12.573054.

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Bakkoury, Zohra, Assia Rharbi, and Abdelkader Betari. "Integration of biological data : The familial hypercholesterolemia." In 2009 IEEE/ACS International Conference on Computer Systems and Applications. IEEE, 2009. http://dx.doi.org/10.1109/aiccsa.2009.5069295.

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Ferrer Machín, A., S. Martin Rodriguez, J. Vilar Rodriguez, MDLA Padron Garcia, M. Vera Cabrera, J. Arias Blaco, and MDC Villastrigo Garcia. "4CPS-178 Effect of pcsk9 inhibitors on hypercholesterolemia." In 28th EAHP Congress, Bordeaux, France, 20-21-22 March 2024. British Medical Journal Publishing Group, 2024. http://dx.doi.org/10.1136/ejhpharm-2024-eahp.282.

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Milanic, Matija, Asgeir Bjorgan, Marcus Larsson, Tomas Strömberg, and Lise Lyngsnes Randeberg. "Detection of hypercholesterolemia using hyperspectral imaging of human skin." In European Conference on Biomedical Optics. Washington, D.C.: OSA, 2015. http://dx.doi.org/10.1364/ecbo.2015.95370c.

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Waivers, L. E., and J. T. Busher. "A program for the screening and treatment of hypercholesterolemia." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.95170.

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Dubonosova, Ekaterina, Anastasia Lamasova, Elizaveta Leonova, Alina Pankova, and Kamilla Efendieva. "26 Familial hypercholesterolemia: a rare case of early diagnosis." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.26.

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Milanic, Matija, Asgeir Bjorgan, Marcus Larsson, Tomas Strömberg, and Lise L. Randeberg. "Detection of hypercholesterolemia using hyperspectral imaging of human skin." In European Conferences on Biomedical Optics, edited by J. Quincy Brown and Volker Deckert. SPIE, 2015. http://dx.doi.org/10.1117/12.2183880.

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Reports on the topic "Hypercholesterolemie"

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Zhang, Lingnan, Fang Zhang, Xinwei Jia, Junmin Xie, Yeran Zhu, Xiaozhe Zhou, and Chang Meng. Targeting PCSK9 in Heterozygous familial hypercholesterolemia: a meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2024. http://dx.doi.org/10.37766/inplasy2024.3.0095.

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Cheng, Yonglang, Peng Zeng, Zhiwei Huang, Hao Shi, Tianying Cai, Tongxi Li, Yifan Chen, Wenguang Fu, and Qiu Li. Lactobacillus reuteri alleviates lipid levels in patients with hypercholesterolemia: a meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0160.

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Chen, Jen-Jen H. Adherence to Hypercholesterolemia Management Guidelines By Health Care Providers in a United States Air Force Medical Treatment Facility. Fort Belvoir, VA: Defense Technical Information Center, April 2001. http://dx.doi.org/10.21236/ad1012387.

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Chen, Jen-Jen H. Adherence to Hypercholesterolemia Management Guidelines by Health Care Providers in a United States Air Force Medical Treatment Facility. Fort Belvoir, VA: Defense Technical Information Center, May 2001. http://dx.doi.org/10.21236/ada421097.

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Yu, Yani, Lei Chen, Honghong Zhang, Zihao Fu, Qi Liu, Haijing Zhao, Yuqi Liu, and Yundai Chen. Association between Familial Hypercholesterolemia and Risk of Cardiovascular Events in different subgroups:A Meta-Analysis of 1.1 Million Subjects. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0010.

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Kanner, Joseph, Edwin Frankel, Stella Harel, and Bruce German. Grapes, Wines and By-products as Potential Sources of Antioxidants. United States Department of Agriculture, January 1995. http://dx.doi.org/10.32747/1995.7568767.bard.

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Several grape varieties and red wines were found to contain large concentration of phenolic compounds which work as antioxidant in-vitro and in-vivo. Wastes from wine production contain antioxidants in large amounts, between 2-6% on dry material basis. Red wines but also white wines were found to prevent lipid peroxidation of turkey muscle tissues stored at 5oC. The antioxidant reaction of flavonoids found in red wines against lipid peroxidation were found to depend on the structure of the molecule. Red wine flavonoids containing an orthodihydroxy structure around the B ring were found highly active against LDL and membrane lipid peroxidation. The antioxidant activity of red wine polyphenols were also found to be dependent on the catalyzer used. In the presence of H2O2-activated myoglobin, the inhibition efficiency was malvidin 3-glucoside>catechin>malvidin>resveratol. However, in the presence of an iron redox cycle catalyzer, the order of effectiveness was resveratol>malvidin 3-glucoside = malvidin>catechin. Differences in protein binding were found to affect antioxidant activity in inhibiting LDL oxidation. A model protein such as BSA, was investigated on the antioxidant activity of phenolic compounds, grape extracts, and red wines in a lecithin-liposome model system. Ferulic acid followed by malvidin and rutin were the most efficient in inhibiting both lipid and protein oxidation. Catechin, a flavonal found in red-wines in relatively high concentration was found to inhibit myoglobin catalyzed linoleate membrane lipid peroxidation at a relatively very low concentration. This effect was studied by the determination of the by-products generated from linoleate during oxidation. The study showed that hydroperoxides are catalytically broken down, not to an alcohol but most probably to a non-radical adduct. The ability of wine-phenolics to reduce iron and from complexes with metals were also demonstrated. Low concentration of wine phenolics were found to inhibit lipoxygenase type II activity. An attempt to understand the bioavailability in humans of antocyanins from red wine showed that two antocyanins from red wine were found unchanged in human urine. Other antocyanins seems to undergo molecular modification. In hypercholesterolemic hamsters, aortic lipid deposition was significantly less in animals fed diets supplemented with either catechin or vitamin E. The rate of LDL accumulation in the carotid arteries was also significantly lower in the catechin and vitamin E animal groups. These results suggested a novel mechanism by which wine phenolics are associated with decreased risk of coronary heart diseases. This study proves in part our hypothesis that the "French Paradox" could be explained by the action of the antioxidant effects of phenolic compounds found at high concentration in red wines. The results of this study argue that it is in the interest of public health to increase the consumption of dietary plant falvonoids. Our results and these from others, show that the consumption of red wine or plant derived polyphenolics can change the antioxidant tone of animal and human plasma and its isolated components towards oxidative reactions. However, we need more research to better understand bioavailability and the mechanism of how polyphenolics affect health and disease.
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