Journal articles: 'Hyperglobulinemia'

1

LINDEBOOM, G. A. "Hyperglobulinemia and Pregnancy." Acta Medica Scandinavica 131, no. 4 (April 24, 2009): 368–79. http://dx.doi.org/10.1111/j.0954-6820.1948.tb04529.x.

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BING, JENS. "Further Investigations on Hyperglobulinemia." Acta Medica Scandinavica 103, no. 6 (April 24, 2009): 547–64. http://dx.doi.org/10.1111/j.0954-6820.1940.tb09055.x.

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BING, JENS. "Further Investigations on Hyperglobulinemia." Acta Medica Scandinavica 103, no. 6 (April 24, 2009): 565–83. http://dx.doi.org/10.1111/j.0954-6820.1940.tb09056.x.

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Malhotra, Amit, and Prasad R. Koduri. "Polyclonal Hyperglobulinemia and Spurious Hypophosphatemia." Annals of Internal Medicine 131, no. 4 (August 17, 1999): 314. http://dx.doi.org/10.7326/0003-4819-131-4-199908170-00026.

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Mouallem, Meir, Natalia Antipov, Haim Mayan, Ben Ami Sela, and Zvi Farfel. "Hyperglobulinemia in Amiodarone-induced Pneumonitis." Cardiovascular Drugs and Therapy 21, no. 1 (February 2007): 63–67. http://dx.doi.org/10.1007/s10557-007-6006-6.

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Liu, Hui, Guojin Wang, Jia Song, Jing Guan, Zonghong Shao, and Rong Fu. "Pseudo-monoclonal gammopathy due to autoimmune disease: a case report." Journal of International Medical Research 48, no. 2 (September 25, 2019): 030006051986661. http://dx.doi.org/10.1177/0300060519866618.

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Hyperglobulinemia is a common manifestation of plasma cell disease, and it is sometimes caused by autoimmune diseases (AIDs). We report an uncommon presentation of hyperglobulinemia, with a high amount of plasma cells in bone marrow, pancytopenia, hematemesis, and splenomegaly in an 18-year-old woman. Some examinations were performed to determine the diagnosis, including serum protein electrophoresis, immunofixation electrophoresis, the free light chain assay, abdominal enhanced computed tomography (CT) and CT venography, and positron-emission tomography-CT. The patient was finally diagnosed with AID. Considerable improvement in her symptoms was observed after immunosuppressive therapy. Findings in this case highlight that not only differentiation of hyperglobulinemia caused by monoclonal or polyclonal immunoglobulin, but also AIDs, need to be considered to exclude non-Hodgkin’s lymphoma and plasma cell disease.

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Ohteki, Takaaki, Chie Tomida, Michihiro Goto, Jouichi Usui, Kaori Muro, Kunihiro Yamagata, and Akio Koyama. "Recurrent retroperitoneal fibrosis associated with hyperglobulinemia." Nihon Naika Gakkai Zasshi 89, no. 10 (2000): 2183–85. http://dx.doi.org/10.2169/naika.89.2183.

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8

Kirschbaum, Barry. "Hyperglobulinemia With an Increased Anion Gap." American Journal of the Medical Sciences 316, no. 6 (December 1998): 393–97. http://dx.doi.org/10.1016/s0002-9629(15)40450-1.

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KIRSCHBAUM, BARRY. "Hyperglobulinemia With an Increased Anion Gap." American Journal of the Medical Sciences 316, no. 6 (December 1998): 393–97. http://dx.doi.org/10.1097/00000441-199812000-00007.

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Haller, M., P. Deplazes, F. Guscetti, JC Sardinas, I. Reichler, and J. Eckert. "Surgical and chemotherapeutic treatment of alveolar echinococcosis in a dog." Journal of the American Animal Hospital Association 34, no. 4 (July 1, 1998): 309–14. http://dx.doi.org/10.5326/15473317-34-4-309.

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Surgical removal of macroscopically detectable metacestode tissue followed by postoperative chemotherapy according to established human protocols resulted in complete clinical remission and immediate normalization of hyperglobulinemia in a dog with alveolar echinococcosis (AE). The disease is caused by the metacestode stage of the cestode, Echinococcus multilocularis. In endemic areas, AE should be included in the differential diagnosis of polycystic liver masses, especially if concomitant hyperglobulinemia is present. However, the importance of AE is not only the disease of the single dog itself but also the potential risk of infection for humans in an endemic area.

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HAMMARSTEN, GRETA, GUNNAR LINDGREN, BÖRJE OLHAGEN, and RUNE ORDELL. "Hyperglobulinemia with a Unique Thermolabile γ-Component." Acta Medica Scandinavica 123, no. 1 (April 24, 2009): 50–55. http://dx.doi.org/10.1111/j.0954-6820.1945.tb06142.x.

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Ranstrøm, S. "On the terms »Essential Hyperglobulinemia» and »Premyeloma»." Acta Medica Scandinavica 124, no. 2 (April 24, 2009): 134–47. http://dx.doi.org/10.1111/j.0954-6820.1946.tb09197.x.

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Konta, T., and M. Matsui. "Cryoglobulin nephropathy with nodular glomerulosclerosis and IgM hyperglobulinemia." Clinical and Experimental Nephrology 4, no. 2 (June 28, 2000): 144–46. http://dx.doi.org/10.1007/pl00012166.

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Adler, Sharon G., Stewart A. Laidlaw, Michael M. Lubran, and Joel D. Kopple. "Hyperglobulinemia May Spuriously Elevate Measured Serum Inorganic Phosphate Levels." American Journal of Kidney Diseases 11, no. 3 (March 1988): 260–63. http://dx.doi.org/10.1016/s0272-6386(88)80159-8.

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15

Whittemore, J. C., J. R. Hawley, S. V. Radecki, J. D. Steinberg, and M. R. Lappin. "Bartonella Species Antibodies and Hyperglobulinemia in Privately Owned Cats." Journal of Veterinary Internal Medicine 26, no. 3 (April 10, 2012): 639–44. http://dx.doi.org/10.1111/j.1939-1676.2012.00925.x.

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WALDENSTRÖM, JAN. "Incipient myelomatosis or «essential« hyperglobulinemia with fibrinogenopenia - a new syndrome?" Acta Medica Scandinavica 117, no. 3-4 (April 24, 2009): 216–47. http://dx.doi.org/10.1111/j.0954-6820.1944.tb03955.x.

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ANDERSSON, BO, and AYMAR SAMUELSON. "A Case of Hyperglobulinemia with pronounced Eye Changes and Acrocyanosis." Acta Medica Scandinavica 117, no. 3-4 (April 24, 2009): 248–60. http://dx.doi.org/10.1111/j.0954-6820.1944.tb03956.x.

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Seelig, Davis M., James A. Perry, Anne C. Avery, and Paul R. Avery. "Monoclonal gammopathy without hyperglobulinemia in 2 dogs with IgA secretory neoplasms." Veterinary Clinical Pathology 39, no. 4 (October 22, 2010): 447–53. http://dx.doi.org/10.1111/j.1939-165x.2010.00262.x.

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McHutchison, J. G., A. Polito, J. L. Person, S. Govindarajan, B. Valinluck, R. Dinello, S. Quan, and A. G. Redeker. "Assessment of Hepatitis C Antibody Tests in Homosexual Men with Hyperglobulinemia." Journal of Infectious Diseases 164, no. 1 (July 1, 1991): 217–18. http://dx.doi.org/10.1093/infdis/164.1.217.

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BICHEL, JÖRGEN, JENS BING, and NIELS HARBOE. "Another Case of Hyperglobulinemia and Affection of the Central Nervous System1." Acta Medica Scandinavica 138, no. 1 (April 24, 2009): 1–14. http://dx.doi.org/10.1111/j.0954-6820.1950.tb10091.x.

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21

Genesca, Joan, Antonio Gonzalez, Mireia Torregrosa, Abel Mujal, and Rosa Segura. "High Levels of Endotoxin Antibodies Contribute to Hyperglobulinemia of Cirrhotic Patients." American Journal of Gastroenterology 93, no. 4 (April 1998): 664–65. http://dx.doi.org/10.1111/j.1572-0241.1998.664_b.x.

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22

Doi, Hiroyoshi, Eiichi Hayashi, Jun Arai, Masayuki Tojo, Kenichi Morikawa, Junichi Eguchi, Takayoshi Ito, Tatsuya Kanto, David E. Kaplan, and Hitoshi Yoshida. "Enhanced B-cell differentiation driven by advanced cirrhosis resulting in hyperglobulinemia." Journal of Gastroenterology and Hepatology 33, no. 9 (March 13, 2018): 1667–76. http://dx.doi.org/10.1111/jgh.14123.

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HAMADA, ATSUO, NAOHIRO WATANABE, MASASHI KOBAYASHI, EIICHI OKUSAWA, TOMOYOSHI NOZAKI, IVETE BARBOSA, SEIKI TATENO, and AKIO KOBAYASHI. "The etiological factor for eosinophilia and hyperglobulinemia E in Brazilian school children." Japanese Journal of Tropical Medicine and Hygiene 19, no. 2 (1991): 203–8. http://dx.doi.org/10.2149/tmh1973.19.203.

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24

Toy, Pearl T. C. Y., Marion E. Reid, and Maryann Burns. "Positive direct antiglobulin test associated with hyperglobulinemia in acquired immunodeficiency syndrome (AIDS)." American Journal of Hematology 19, no. 2 (June 1985): 145–50. http://dx.doi.org/10.1002/ajh.2830190206.

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25

Pacca, Rafael Lopes, Jiviane Beatriz Cunha Barretto da Silva, Kallen de Carvalho e. Souza, and Rebeca Barbosa Carbinatto. "Autoimmune hemolytic anemia and hyperglobulinemia leading to the diagnosis of multiple myeloma." Revista Brasileira de Hematologia e Hemoterapia 39, no. 4 (October 2017): 357–59. http://dx.doi.org/10.1016/j.bjhh.2017.07.005.

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Heeb, Heather L., Melinda J. Wilkerson, Ruthanne Chun, and Roman Reddy Ganta. "Large Granular Lymphocytosis, Lymphocyte Subset Inversion, Thrombocytopenia, Dysproteinemia, and Positive Ehrlichia Serology in a Dog." Journal of the American Animal Hospital Association 39, no. 4 (July 1, 2003): 379–84. http://dx.doi.org/10.5326/0390379.

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A 7-year-old, mixed-breed dog was presented for evaluation of a possible lymphocytic leukemia. Results of laboratory testing included thrombocytopenia, large granular lymphocytosis, inverted CD4:CD8 ratio, hyperglobulinemia, and hypoalbuminemia. Results of a tick-borne disease panel indicated a positive immunoglobulin G serum titer (1:2,048) to Ehrlichia canis, supporting exposure to this organism. The dog responded to a combination treatment of doxycycline and prednisone. A review of the literature and novel diagnostic methods that aided in the diagnosis of this case are discussed.

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27

Sin, Sang-Hoon, Sun Ah Kang, Yongbaek Kim, Anthony Eason, Kelly Tan, Hyowon An, and Dirk P. Dittmer. "Kaposi's Sarcoma-Associated Herpesvirus Latency Locus Compensates for Interleukin-6 in Initial B Cell Activation." Journal of Virology 90, no. 4 (December 9, 2015): 2150–54. http://dx.doi.org/10.1128/jvi.02456-15.

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Interleukin 6 (IL-6) is considered a proliferation and survival factor for B cells. To assess the role of IL-6 in Kaposi sarcoma-associated herpesvirus (KSHV) latency, KSHV latency locus-transgenic mice (referred to as latency mice) lacking IL-6 were evaluated. IL-6−/−latency mice had the same phenotypes as the latency mice, i.e., increased frequency of marginal zone B cells, hyperplasia, and hyperglobulinemia, indicating that the KSHV latency locus, which includes all viral microRNAs (miRNAs), can compensate for lack of IL-6 in premalignant B cell activation.

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Fradkin, JM, AM Braniecki, TM Craig, F. Ramiro-Ibanez, KS Rogers, and DL Zoran. "Elevated parathyroid hormone-related protein and hypercalcemia in two dogs with schistosomiasis." Journal of the American Animal Hospital Association 37, no. 4 (July 1, 2001): 349–55. http://dx.doi.org/10.5326/15473317-37-4-349.

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Two adult dogs were evaluated for hypercalcemia. Diagnostic evaluation identified elevated parathyroid hormone-related protein (PTHrP) and presumptive humoral hypercalcemia of malignancy. At necropsy, schistosomiasis was diagnosed. North American schistosomiasis is caused by Heterobilharzia americana. Clinical findings may include dermatitis, coughing, diarrhea, and anorexia. Clinicopathological findings may include hypercalcemia, hyperglobulinemia, hypoalbuminemia, anemia, and eosinophilia. Diagnosis by fecal examination is difficult. Praziquantel or fenbendazole treatment may be curative or palliative. These are the first reported cases of hypercalcemia with elevated PTHrP in animals without diagnosed malignancy. Elevation of PTHrP has not been previously reported in hypercalcemic humans or in animals with granulomatous inflammation.

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Ruth, Jeffrey. "Heterobilharzia americana Infection and Glomerulonephritis in a Dog." Journal of the American Animal Hospital Association 46, no. 3 (May 1, 2010): 203–8. http://dx.doi.org/10.5326/0460203.

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Schistosomiasis is an uncommonly reported disease that usually causes weight loss, anemia, and gastrointestinal signs. A 6-year-old, neutered male dog developed membranoproliferative glomerulonephritis concurrent with infection with the trematode parasite Heterobilharzia americana. At presentation, the dog had proteinuria, hypoalbuminemia, hyperglobulinemia, and anemia. Diagnosis was based upon the histopathological appearance of the kidney. Clinical signs, biochemical and hematological abnormalities, and proteinuria resolved following treatment with fenbendazole and praziquantel. Fecal examination by saline sedimentation, miracidia hatching, or Heterobilharzia polymerase chain reaction assay may be indicated when examining a dog that is presented with unexplained glomerulonephritis and is from an endemic area.

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Watanabe, Kazuya, Chikako Takeuchi, Mutsuko Yokota, Tomiko Yamaguchi, Hiroyasu Yasuda, Hitoshi Ohto, Eiko Yoshida, and Yoshimasa J. Aoki. "False negative agglutination phenomenon in hyperglobulinemia serum in the polyethyleneglycol indirect anti-globulin test." Journal of the Japan Society of Blood Transfusion 48, no. 4 (2002): 342–49. http://dx.doi.org/10.3925/jjtc1958.48.342.

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Yasukawa, Kana, Naoka Kato, Asuka Hamasaka, and Hiroo Hata. "Unusual annular purpura and erythema in a patient with malignant lymphoma accompanied with hyperglobulinemia." International Journal of Dermatology 47, no. 3 (March 2008): 302–4. http://dx.doi.org/10.1111/j.1365-4632.2008.03357.x.

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32

Weingart, Christiane, Achim D. Gruber, Mathias Brunnberg, and Barbara Kohn. "Hypernatremia in a Cat with Toxoplasma-Induced Panencephalitis." Journal of the American Animal Hospital Association 52, no. 1 (January 1, 2016): 63–67. http://dx.doi.org/10.5326/jaaha-ms-6257.

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A 12 yr old female neutered Carthusian crossbreed cat was presented due to progressive neurological signs. Clinical signs included dehydration, stupor, and anisocoria. Laboratory examination revealed severe hypernatremia, azotemia, hyperglobulinemia, and an erythrocytosis. Clinical signs and hypernatremia suggested an intracranial process. Imaging studies revealed a loss of structure in the cerebrum, hypothalamus, and pituitary gland. Due to a poor prognosis, the cat was euthanatized. Histopathological examination revealed a subacute granulomatous and necrotizing panencephalitis with Toxoplasma-typical protozoa. The Toxoplasma-induced dysfunction of the hypothalamus and pituitary gland led to diabetes insipidus, which was, in combination with insufficient water intake, the most likely cause for the hypernatremia.

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Peterson, EN, and AC Meininger. "Immunoglobulin A and immunoglobulin G biclonal gammopathy in a dog with multiple myeloma." Journal of the American Animal Hospital Association 33, no. 1 (January 1, 1997): 45–47. http://dx.doi.org/10.5326/15473317-33-1-45.

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A nine-year-old golden retriever was referred for evaluation of chronic anorexia, vomiting, and diarrhea. Low body weight, mucous membrane pallor, and palpably enlarged liver and spleen were detected by physical examination. Anemia, hyperglobulinemia, and concurrent trichuriasis and coccidiosis were identified upon initial diagnostic evaluation. Punctate vertebral lysis was apparent radiographically. Atypical plasma cell proliferation was found in the bone marrow, liver, and spleen. An immunoglobulin A and immunoglobulin G biclonal gammopathy was demonstrated by serum protein electrophoresis and immunoelectrophoresis. The dog was diagnosed with multiple myeloma and euthanized per owner request. Multiple myeloma should be considered in the differential diagnosis for biclonal gammopathy in the dog.

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Jakobovich, E., B. Koplewitz, E. Marva, and E. Granot. "Yersinia enterocoliticaInfection Simulating Lymphoproliferative Disease, after Liver Transplant." Case Reports in Transplantation 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/923058.

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We describe a 14-year-old girl, who was 13 y after liver transplantation for biliary atresia with an unremarkable postoperative course. She presented with fever of up to 40°C, extreme fatigue, malaise, anorexia, and occasional vomiting. On physical examination the only finding was splenomegaly. Lab results showed hyperglobulinemia and an elevated sedimentation rate. Liver function tests were normal except for mild elevation ofγGTP. Abdominal U/S and CT demonstrated an enlarged spleen with retroperitoneal and mesenteric lymph nodes enlargement. An exhaustive evaluation for infectious causes, autoimmune conditions, and malignancy was negative. A full recovery after 5 months prompted testing for self-limited infectious etiologies.Yersinia enterocoliticainfection was diagnosed.

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McConkey, Sandra, Alfonso López, and John Pringle. "Extramedullary Plasmacytoma in a Horse with Ptyalism and Dysphagia." Journal of Veterinary Diagnostic Investigation 12, no. 3 (May 2000): 282–84. http://dx.doi.org/10.1177/104063870001200318.

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A Clydesdale mare was examined for weight loss, inappetence, ptyalism, and dysphagia. The main abnormality revealed by serum biochemistry was a marked hyperglobulinemia, and protein electrophoresis revealed a monoclonal gammopathy in the gamma region. The urine was positive for Bence Jones proteins. These findings suggested a plasma cell tumor. The neoplasm could not be located with extensive antemortem examination. At postmortem, neoplastic cells morphologically compatible with plasma cells and positive for equine IgG with imunoperoxidase staining infiltrated the pericardium, mediastinal stromal tissues, adrenal glands, meninges, atrioventricular valves, aorta, abdominal and thoracic fat, and nerves, including the trigeminal nerve. The neoplastic cells invading the cranial nerves were responsible for many of the presenting signs.

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BING, JENS, and N. O. CHRISTENSEN. "The Connection between Plasma Cells and the Occurrence of Hyperglobulinemia in Horses and Cattle.1." Acta Medica Scandinavica 116, no. 3-4 (April 24, 2009): 382–95. http://dx.doi.org/10.1111/j.0954-6820.1944.tb11940.x.

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WALDENSTRÖM, JAN. "The Occurrence of Benign, Essential Monoclonal (M Type), Non-macromolecular Hyperglobulinemia and its Differential Diagnosis." Acta Medica Scandinavica 176, no. 3 (April 24, 2009): 345–65. http://dx.doi.org/10.1111/j.0954-6820.1964.tb00942.x.

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38

Lloyd, Rebecca, Emmanouil Nikolousis, Bhuvan Kishore, Richard Lovell, Paneesha Shankara, Nervana Abou Zeid, Claire Horgan, et al. "Autoimmune Cytopenias Developing Late Post Alemtuzumab-Based Allogeneic Stem Cell Transplantation: Presentation of Short Case Series from a Transplant Center." Cell Transplantation 29 (January 1, 2020): 096368972095064. http://dx.doi.org/10.1177/0963689720950641.

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Stem cell transplantation remains the curative option for many patients with hematological malignancies. The long-term effects of these treatments on the patients and their immune systems have been extensively investigated, but there remains a paucity of data regarding autoimmune manifestations post-transplant, although these effects are well recognized. Herein we present the clinical picture and therapeutic approach in three patients (cases 1–3), with varied presentations of autoimmune disease post-transplant. Case 1 exhibited autoimmune hemolytic anemia and other autoimmune manifestations (serositis, thyroiditis), that were probably linked to graft versus relapsed leukemia effect. Cases 2 and 3 had pure red white cell aplasia and pure red cell aplasia, respectively, which were associated with hyperglobulinemia and a clonal T cell expansion.

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Broderson, J. R., W. L. Chapman, and W. L. Hanson. "Experimental Visceral Leishmaniasis in the Owl Monkey." Veterinary Pathology 23, no. 3 (May 1986): 293–302. http://dx.doi.org/10.1177/030098588602300310.

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Visceral leishmaniasis developed in eight owl monkeys ( Aotus trivirgatus) after intravenous inoculation with a Khartoum strain (WR378) of Leishmania donovani. Six monkeys died within 93 days, and two monkeys recovered from the disease. Clinically, signs were weight loss, anemia, and hepatosplenomegaly. Hematologic findings included anemia, granulocytopenia, thrombocytopenia, and lymphocytosis. Analysis of serum or plasma revealed hyperbilirubinemia, azotemia, hyperglobulinemia, hypoalbuminemia, and altered hemostasis. All monkeys developed positive antibody titers to promastigotes of L. donovani and had increases in immunoglobulins M and subsequently G. Liver, spleen, bone marrow, and lymph nodes were the principal organs containing numerous parasitized macrophages. The owl monkey was highly susceptible to L. donovani infection and should be a useful animal model for the study of visceral leishmaniasis.

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Witzens, M., G. Egerer, D. Stahl, E. Werle, H. Goldschmidt, and R. Haas. "A case of μ heavy-chain disease associated with hyperglobulinemia, anemia, and a positive Coombs test." Annals of Hematology 77, no. 5 (November 26, 1998): 231–34. http://dx.doi.org/10.1007/s002770050448.

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Polińska, Beata, Joanna Matowicka-Karna, and Halina Kemona. "The role of the erythrocyte sedimentation rate in the diagnostics of different clinical conditions." Diagnostyka Laboratoryjna 51, no. 4 (January 17, 2016): 321–26. http://dx.doi.org/10.5604/01.3001.0008.2548.

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Erythrocyte sedimentation rate (ESR, “Biernacki’s reaction”) is one of the oldest, routine laboratory tests. ESR is sensitive test with low specificity. Erythrocyte sedimentation rate is relatively constant in healthy subjects and unequal between different diseases. ESR technique uses venous blood collected into tubes with anticoagulant (sodium citrate or EDTA), and erythrocyte sedimentation rate is assessed after 60 minutes. The results are expressed in mm/1 h and its reference values depend on age and gender. Increased ESR is present in inflammatory, necrotic changes, autoimmune diseases and cancer, anaemias, hyperglobulinemia and hypoalbuminemia. The characteristic ESR >100 mm/1 h is observed in patients with multiple myeloma, Hodgkin’s disease and liver cirrhosis. Reduced ESR occurs mainly in polycythemia vera and fibrinogen deficiency. Physiologically, the increased ESR is observed during pregnancy, puerperium, menstruation or while taking certain medications.

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Lennox, Angela, Susan Clubb, April Romagnano, Norman H. Altman, and Carolyn Cray. "Monoclonal Hyperglobulinemia in Lymphosarcoma in a Cockatiel (Nymphicus hollandicus) and a Blue and Gold Macaw (Ara ararauna)." Avian Diseases 58, no. 2 (June 2014): 326–29. http://dx.doi.org/10.1637/10683-100413.

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Neuwald, Elisa B., Luciele V. Teixeira, Francisco O. Conrado, Mariana O. D. da Silva, Nicole R. C. Hlavac, and Félix H. D. González. "Epidemiological, clinical and immunohistochemical aspects of canine lymphoma in the region of Porto Alegre, Brazil." Pesquisa Veterinária Brasileira 34, no. 4 (April 2014): 349–54. http://dx.doi.org/10.1590/s0100-736x2014000400009.

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This paper describes the epidemiological, clinical and immunohistochemical characteristics of canine lymphomas diagnosed in the region of Porto Alegre, Brazil. Thirty dogs were enrolled in the study; most of them were male (60%), mixed-breed (23%) and middle-aged or older. The majority (87%) of affected dogs showed the multicentric form. The B-cell phenotype was most frequently detected (62%); 37% of the animals were in clinical stage IV, and 83% were classified as sub-stage "b". Lymphadenopathy was observed in 67% of the cases, and dyspnea, prostration, decreased appetite and vomiting were the most common clinical signs encountered. Anemia was a frequently encountered laboratory alteration (57%), as were leukocytosis (40%), thrombocytopenia (33%), lymphopenia (30%), hyperglobulinemia (20%) and hypercalcemia (13%). The results of this study indicate that the clinical features of dogs with lymphoma in the region of Porto Alegre are similar to those observed worldwide.

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Bischoff, Sebastian, Kakharman Yesmembetov, Christoph Antoni, Janina Sollors, Matthias Evert, Matthias Ebert, and Andreas Teufel. "Autoimmune Hepatitis: a Review of Established and Evolving Treatments." Journal of Gastrointestinal and Liver Diseases 29, no. 3 (September 9, 2020): 429–43. http://dx.doi.org/10.15403/jgld-2667.

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Autoimmune hepatitis (AIH) is a necroinflammatory liver disease commonly presenting with a fluctuating course of activity, presence of circulating autoantibodies, hyperglobulinemia of IgG, and/or response to immunosuppressive drugs. However, the disease displays a considerable heterogeneity. No single clinical or biochemical test may establish diagnosis of AIH. Thus, diagnosis still requires extensive clinical evaluation and experience. Prednisolone and azathioprine are considered standard treatment leading to remission in most patients. However, this standard treatment may not be effective in some patients or not be feasible due to one of these drugs. Over the past two decades additional immunosuppressant drugs for the treatment of AIH have been evaluated and have significantly extended the therapeutic spectrum. Among those novel drugs are mycophenolate mofetil, tacrolimus, everolimus, 6-mercaptopurine, infliximab, rituximab and several others. In this review we summarize the current standard of therapy but also efforts of providing novel therapeutic strategies to AIH patients.

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TOMA, Gina Corina, Marian TAULESCU, Viorica MIRCEAN, Angela Monica IONICĂ, Roxana CORA, Cornel CĂTOI, and Mirabela Oana DUMITRACHE. "Imported Canine leishmaniasis in Romania: a Case Report." Bulletin of University of Agricultural Sciences and Veterinary Medicine Cluj-Napoca. Veterinary Medicine 75, no. 1 (May 19, 2018): 125. http://dx.doi.org/10.15835/buasvmcn-vm:002317.

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Canine leishmaniasis (CanL) is a zoonotic disease considered endemic in the Mediterranean region. Romania is traditionally regarded as a non-endemic country.Considering the zoonotic character of the disease, this study aims to increase the knowledge on diagnostic aspects of CanL.A 2-year-old, mix breed male dog, recently returned from Italy had a history of progressive weight loss and skin lesions. The clinical examination was followed by hematology and serum biochemistry, fine-needle aspiration of lymph nodes, impression smears, VetExpert® rapid test Leishmania Ab, and PCR. The clinical examination revealed muscle atrophy, non-pruritic crusting dermatitis, ulcers, and lymphadenopathy. Hematology showed severe anemia. The serum biochemistry revealed hyperproteinaemia, hypoalbuminemia, hyperglobulinemia. Cytological exams evidenced the intracellular amastigotes in macrophages, confirmed by rapid test and PCR.In Romania, under the light of the new case reports, leishmaniasis should be reconsidered from both veterinary and public health perspective.

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Wang, Taina, Bin Xu, Rong Fan, Zhihong Liu, and Dehua Gong. "Asparaginase-associated concurrence of hyperlipidemia, hyperglobulinemia, and thrombocytosis was successfully treated by centrifuge/membrane hybrid double-filtration plasmapheresis." Journal of Clinical Lipidology 10, no. 3 (May 2016): 646–49. http://dx.doi.org/10.1016/j.jacl.2015.12.010.

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Kameyama, Mamoru, Yoshiharu Ishikawa, Tomoyuki Shibahara, and Koichi Kadota. "Plasma Cell Myeloma Producing IgG, IgM, and IgA Immunoglobulins in a Cow." Journal of Veterinary Diagnostic Investigation 15, no. 2 (March 2003): 166–69. http://dx.doi.org/10.1177/104063870301500212.

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A diagnosis of plasma cell myeloma was made in a 6-year-old Holstein cow that showed continuous nosebleed, progressing emaciation, and hyperglobulinemia. Necropsy revealed enlargement of the caudal mesenteric lymph nodes. Lymph nodes and spleen were infiltrated by neoplastic plasma cells. Renal lymph nodes, bone marrow, and kidneys also were affected. Amyloid-like material that was negative for Congo red was present in most of the affected lymph nodes. Congo red–positive amyloid material was noted in spleen, kidneys, liver, and adrenal glands. Neoplastic plasma cells exhibited immunohistochemical reactions for λ-light chain and 3 classes of heavy chain but not for κ-light chain. Nonamyloid deposits were labeled with antisera against λ-light chain and heavy chains. Plasmacytoid cells, which stained positively for IgG, IgM, or IgA, were intermingled in all lesions. Production of 3 types of immunoglobulins may be caused by heavy-chain class switching, constantly occurring in certain proportions.

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BING, JENS, MOGENS FOG, and AXEL V. NEEL. "Reports of a third case of hyperglobulinemia with affection of the central nervous system on a toxi-infectious basis." Acta Medica Scandinavica 91, no. 4-5 (April 24, 2009): 409–27. http://dx.doi.org/10.1111/j.0954-6820.1937.tb16053.x.

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Zhang, Baohua, Kun Chen, and Erru Ni. "Comparison of three methods for extraction of HCV RNA in sera collected from individuals with hyperlipidemia, hyperbilirubinemia and hyperglobulinemia." Journal of Virological Methods 212 (February 2015): 44–46. http://dx.doi.org/10.1016/j.jviromet.2014.11.004.

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Boyle, Tonya E., Marie K. Holowaychuk, Allison K. Adams, and Steven L. Marks. "Treatment of Three Cats with Hyperviscosity Syndrome and Congestive Heart Failure Using Plasmapheresis." Journal of the American Animal Hospital Association 47, no. 1 (January 1, 2011): 50–55. http://dx.doi.org/10.5326/jaaha-ms-5635.

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Three cats were evaluated at a veterinary teaching hospital for congestive heart failure (CHF) secondary to hyperviscosity syndrome from plasma cell neoplasia. All cats had severe hyperproteinemia due to hyperglobulinemia. Multiple myeloma or plasma cell neoplasia was diagnosed based on cytopathology and post mortem examination. The cats presented with signs of CHF including acute collapse, tachypnea, increased respiratory effort, and pulmonary crackles. All cats had heart murmurs and echocardiographic signs consistent with hypertrophic cardiomyopathy. An enlarged left atrium was found in all cats and two of three cats also had spontaneous echocardiographic contrast. Plasmapheresis (centrifugal plasma exchange) was performed on all three cats by the removal of whole blood and the infusion of a balanced electrolyte solution while the whole blood was centrifuged and separated. The RBCs were then washed before being readministered to the patient. Plasmapheresis alleviated the clinical signs of CHF (tachypnea) in all three cats. Plasmapheresis should be considered in cases of CHF secondary to hyperviscosity syndrome to rapidly alleviate clinical signs associated with heart failure while diagnosis of the underlying cause is made and appropriate therapy implemented.

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Journal articles on the topic 'Hyperglobulinemia'

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