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Journal articles on the topic 'Hyperkineses'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2025

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1

MUNASIPOVA, S. E., Z. A. ZALYALOVA, and D. M. KHASANOVA. "Comorbidity of tic hyperkineses with impulsive disorders." Practical medicine 18, no. 5 (2020): 88–92. http://dx.doi.org/10.32000/2072-1757-2020-5-88-92.

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Tourette’s syndrome, the most common cause of tics, manifests itself in a wide range of motor and behavioral disorders and is found in men about three times more often than in women. The purpose — to establish the comorbidity of neuropsychiatric disorders in patients with tic hyperkinesis. Material and methods. The study included 111 patients with tic hyperkinesis of various ages and sex, who were outpatiently observed at the Clinical and Diagnostic Center for Movement Disorders and Botulinum Therapy of the Republic of Tatarstan from 2011 to 2014. Results and conclusions. In the group of patie

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2

Judelson, Y. B., and N. Р. Gribova. "Exteroceptive suppression in facial hyperkineses and dystonias." Neurology Bulletin XXIX, no. 1-2 (1997): 88–90. http://dx.doi.org/10.17816/nb79930.

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On the basis of study of results of exteroceptive suppression parameters in 140 patients with different variants of facial hyperkineses the state of reflex inhibitory neuronal chains was defined more precisely. Received data may be used in process selection and monitoring of therapy and promote to understanding of facial musculature dyskinesia forming mechanisms.

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3

Ivanichev, G. A., and K. Levit. "Pathogenesis of the secondary contracture of musces of expression." Kazan medical journal 76, no. 3 (1995): 224–29. http://dx.doi.org/10.17816/kazmj100848.

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The role of local muscular hypertensions of muscles of expression in contracture pathogenesis is stated. Hypertensions are developed pathologically in only average extent of the nerve damage. Synkineses and hyperkineses of muscles of expression in contracture result from the formation of transversal synapse between muscles, divided by weak insulator in conditions of dystrophic changes of the muscular cell membrane.

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4

Khanenko, N., N. Svyrydova, G. Chuprina, et al. "Hyperkinesis: pathogenesis, clinical features, diagnosis, treatment (clinical lecture)." East European Journal of Neurology, no. 3(21) (September 20, 2018): 13–18. http://dx.doi.org/10.33444/2411-5797.2018.3(21).13-18.

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Traditionally, the basic etiological concepts are considered in the views on the morphophysiological basis of hyperkinesis. Hyperkinesia is associated with hypotonia, a decrease in muscle tone, and hyperkinetic disorders are psychogenic and manifest in childhood. Hyperkinesia can be caused by a large number of various diseases, including metabolic disorders, endocrine disruption, hereditary disorders, vascular disorders or traumatic disorders. Other causes include intoxication of the nervous system, autoimmune diseases and infections. The classification of hyperkinesis is that hyperkinetic mot

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5

Yakimovskii, A. F. "INFLUENCE OF ZINC ALIMENTARY TREATMENT ON NORMAL AND PATHOLOGICAL MOTOR BEHAVIOR OF RATS." Trace Elements in Medicine (Moscow) 21, no. 2 (2020): 34–40. http://dx.doi.org/10.19112/2413-6174-2020-21-2-34-40.

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Chronic experiments were performed on rats to study the influence of prolonged acetic zinc alimentary treatment on normal (spontaneous movement in “open field and condition active avoidance reflex in “shuttle box”) and abnormal (choreomyoclonic hyperkinesia, produced by intrastriatal microinjections GABA-A receptors antagonist picrotoxin  2 mcg) motor behavior. 4 mg acetic zinc is used by rats with food ball once a day. 12 mg zinc сonsumption by rats per week did not affected on normal behavior. While 24 mg is produced smaller negative effects on rats reflex performance to 6570% correct resp

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6

Starikov, A. S. "The evaluation of hyperkineses in hepatocerebral dystrophy (Wilson—Konovalov disease) from the position of the theory of muscle spindles." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 115, no. 4 (2015): 54. http://dx.doi.org/10.17116/jnevro20151154154-60.

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7

Bogdanov, O. V., D. Yu Pinchuk, E. V. Pisar'kova, A. M. Shelyakin, and K. T. Sirbiladze. "The use of the method of transcranial micropolarization to decrease the severity hyperkineses in patients with infantile cerebral palsy." Neuroscience and Behavioral Physiology 24, no. 5 (1994): 442–45. http://dx.doi.org/10.1007/bf02359800.

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8

Tappakhov, A. A., and O. S. Nizhegorodova. "Prevalence of stereotypies among adults without neurological disorders and intellectual decline." YAKUT MEDICAL JOURNAL 86, no. 1 (2024): 13–17. https://doi.org/10.25789/ymj.2024.86.03.

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Stereotypies, along with tics, are the most common hyperkineses, especially among children. It has been esTableished that stereotypies can occur among children with normal development. At the same time, we found only one article on the prevalence of stereotypies among relatively healthy adults. The aim of the study is to assess the prevalence and nature of stereotypies among adults without neurological and intellectual disorders and to determine its relationship with tics and associated psychiatric symptoms. Material and methods. We have created an online questionnaire that includes 32 questio

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9

Churton, Michael W. "Hyperkinesis: A Review of Literature." Adapted Physical Activity Quarterly 6, no. 4 (1989): 313–27. http://dx.doi.org/10.1123/apaq.6.4.313.

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The review of literature focuses upon a disorder that affects between 2 and 25% of school-age children. Commonly referred to as hyperkinesis, the disorder lacks definitive consensus on nomenclature, etiology, treatment, and symptomatology. The divergence in identifying hyperkinesis as a homogeneous disorder has prevented the development of data based educational strategies. The disorder is often associated with learning disabilities, and research in hyperkinesis or attentional deficit disorder relative to psychomotor skills and learning has been limited. Subsequently, motor activity programs h

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10

Millichap, J. Gordon. "Food-Induced Hyperkinesis." Pediatric Neurology Briefs 6, no. 8 (1992): 63. http://dx.doi.org/10.15844/pedneurbriefs-6-8-10.

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11

Levitina, E. V., M. U. Kolchanova, O. A. Rakhmanina, and E. B. Hramova. "Structure of comorbide disorders in ticose hyperkinesis in children." Medical Science And Education Of Ural 21, no. 4 (2020): 72–74. http://dx.doi.org/10.36361/1814-8999-2020-21-4-72-74.

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Purpose. To study the structure of comorbid disorders in children with tycotic hyperkinesis of the city of Tyumen. Materials and methods. We examined 103 children aged 5 to 14 years. Patients were divided into 2 groups: group 1 – local tics (n = 43), group 2 – common tics (n = 60). All patients received anticitotic therapy. Using various scales and questionnaires, an assessment of tic hyperkinesis, the level of anxiety, a study of the rational, emotional and behavioral components of the relationship between parents and a child, an investigation of the emotional sphere, and neuropsychological t

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12

Birkeland, S., J. Westby, K. Grong, and J. Lekven. "Effect of afterload and beta-adrenergic blockade on nonischemic myocardial contraction pattern." American Journal of Physiology-Heart and Circulatory Physiology 263, no. 6 (1992): H1716—H1723. http://dx.doi.org/10.1152/ajpheart.1992.263.6.h1716.

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We studied how changes in afterload affect regional contraction in the anterior wall of the left ventricular after circumflex coronary arterial (CFX) occlusion and subsequent beta-adrenergic blockade in pentobarbital sodium-anesthetized cats. Regional function was determined by orthogonal sonomicrometry. CFX occlusion produced nonuniform hyperkinesis in the nonischemic anterior wall; shortening of circumferential segments increased from 10.1 to 14.1% (P < 0.001), whereas shortening of longitudinal segments increased from 3.0 to 9.6% (P < 0.001). Hyperkinesis of longitudinal segments was

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13

Nurmatova, Dilorom, Zainutdinhuzha Sayfitdinhuzhaev, and Zhakhongir Okhunboev. "TIC EPILEPSY IN THE UZBEK POPULATION: CLINICAL AND ENCEPHALOGRAPHIC ASPECTS (PILOT STUDY)." Medical science of Uzbekistan, no. 3 (June 28, 2025): 40–42. https://doi.org/10.56121/2181-3612-2025-3-40-42.

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Relevance. Tic disorders in children are one of the most common forms of hyperkinesis in pediatric neurological practice, demonstrating a high incidence of comorbidity with various neuropsychiatric and neurological disorders. One of these conditions, which has a significant impact on the course and prognosis of the disease, is epilepsy. The aim of the study was to carry out a clinical and physiological study to analyze the pathogenesis and clinical characteristics of epilepsy in children with tic hyperkinesis. Materials and methods of research. The present study analyzes the pathophysiological

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14

Rosenbloom, Lewis. "LEARNING DISABILITIES AND HYPERKINESIS." Developmental Medicine & Child Neurology 14, no. 3 (2008): 394–95. http://dx.doi.org/10.1111/j.1469-8749.1972.tb02606.x.

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15

May, Mark. "Management of Facial Hyperkinesis." Otology & Neurotology 23, Sup 1 (2002): S70. http://dx.doi.org/10.1097/00129492-200200001-00179.

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16

Ruziev, A. Sh. "CLINICAL AND NEUROLOGICAL CHARACTERISTICS OF TIC HYPERKINESIS IN CHILDREN." Oriental Journal of Medicine and Pharmacology 04, no. 02 (2024): 1–6. http://dx.doi.org/10.37547/supsci-ojmp-04-02-01.

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The article presents examination data of 70 children with tic hyperkinesis. Tic hyperkinesis affects the cognitive and emotional spheres, and these changes are most pronounced with common tics. Based on the Toulouse-Pieron test, which characterizes the ability to voluntarily concentrate attention, it has been proven that children with local tics have a significantly higher concentration of attention compared to common ones.

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17

Tsygan, N. V., Yu I. Vainshenker, I. V. Litvinenko, and M. M. Odinak. "Muscular-tonic disorders in a vegetative and a minimal consciousness states." Russian neurological journal 28, no. 1 (2023): 25–32. http://dx.doi.org/10.30629/2658-7947-2023-28-1-25-32.

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Muscular-tonic disorders (MTD) in prolonged disorders of consciousness (PDoC), including a vegetative state (VS) and a minimal consciousness state (MCS), are poorly understood.Aim. To systematize MTD in PDoC, to highlight the features of their dynamics depending on the change in consciousness.Material and methods. 87 patients in PDoC (VS — 52, MCS — 35) resulting from brain damage of diff erent etiology, lasting from 2 months up to 10 years. MTD, provoking hyperkinesis factors and consciousness were analyzed retrospectively in the dynamics and complex.Results. MTD had 98% of patients in PDoC.

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18

Dy, Marisela E., Nathaniel A. Chuang, and Jennifer Friedman. "Holoprosencephaly-Associated Hyperkinesia." Movement Disorders Clinical Practice 2, no. 3 (2015): 310–12. http://dx.doi.org/10.1002/mdc3.12176.

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19

Yakimovskii, Аndrey F. "The influence of acizol into effects of picrotoxin, injected in rat’s neostriatum." Medical academic journal 19, no. 2 (2019): 57–62. http://dx.doi.org/10.17816/maj19257-62.

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The aim of the article. The article is devoted to investigation of of zinc donator acizol influence to rat’s behavior, broken by intrastriatal injection of GABA-A receptor antagonist picrotoxin. Materials and methods. Adult male Wistar rats with avoidance conditioning reflexes in “shuttle box” and free locomotor activity in “open field” were used. Daily microinjection of picrotoxin (2 mcg/1 mcl) bilateral into rostral neostriatum in term of 15 days were made. Zinc donator acizol was injected intraperitoneal (24 mg/kg). Results. Steady losses of avoidance conditioning and choreo-mio

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20

Davletshinр, U. В., and R. V. Magzhanov. "Some questions of pathogenesis and cure of ticous hyperkinesia in children." Neurology Bulletin XXVII, no. 1-2 (1995): 19–21. http://dx.doi.org/10.17816/nb77837.

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Data on clinical and hormonal examination of child ren with ticous hyperkinesia are presented. The obtained results allow to judge activation of dofaminergic neurotransmission and thyroid function in children with tics and to recommend dofamin antagonistseglonil (sulpirid), cerucal (metoclonramid) for curing ticous hyperkinesia.

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21

Bogacheva, V. A., D. V. Zakharov, and V. A. Mikhailov. "Efficacy of antiparkinsonian therapy in tremor correction according to tremorography." Neurology, Neuropsychiatry, Psychosomatics 16, no. 2 (2024): 34–40. http://dx.doi.org/10.14412/2074-2711-2024-2-34-40.

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Objective: to evaluate the efficacy of antiparkinsonian drugs of different groups considering the frequency-amplitude characteristics in the correction of tremulous hyperkinesis in Parkinson's disease (PD) using tremorography. Material and methods. 172 patients with a confirmed diagnosis of PD were observed. Taking into account age and contraindications, patients were divided into four groups receiving one of four drugs in monotherapy: dopamine receptor agonists (ADR; pramipexole), levodopa (Tidomet forte), anticholinergics (biperiden), amantadines (amantadine sulfate). Before starting treatme

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22

Maxamatjanova, N., and G. Ishankhodjaeva. "Psychocorrection of children with ticose hyperkinesis." Parkinsonism & Related Disorders 79 (October 2020): e108. http://dx.doi.org/10.1016/j.parkreldis.2020.06.393.

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23

Ciniglio, Ricardo, Mark Kime, Trudy L. Burns, and Byron F. Vandenberg. "Rapid Resolution of Hyperkinesis After Exercise." Chest 104, no. 3 (1993): 712–17. http://dx.doi.org/10.1378/chest.104.3.712.

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24

Gazzetta, Joshua, Betty Fan, Paul Bonner, and John Galante. "Cholecystectomy for Biliary Hyperkinesia." American Surgeon 85, no. 2 (2019): 219–22. http://dx.doi.org/10.1177/000313481908500232.

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Patients with classic biliary colic symptoms and documented gallbladder ejection fractions on the higher end of the spectrum on hepatobiliary iminoacetic acid scans with cholecystokinin stimulation are presently understudied and the benefits of cholecystectomy are unclear. To determine whether patients with biliary-type pain and biliary hyperkinesia (defined as a gallbladder ejection fractions of 80% or greater) benefit from laparoscopic cholecystectomy, a retrospective chart review encompassing five community hospitals was performed. Patients 16 years and older with diagnosed biliary hyperkin

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25

Tolmacheva, V. A., P. G. Yuzbashyan, R. M. Shishorin, B. A. Volel, and D. V. Romanov. "Functional writer's cramp as psychogenic focal dystonia." Neurology, Neuropsychiatry, Psychosomatics 14, no. 1 (2022): 115–21. http://dx.doi.org/10.14412/2074-2711-2022-1-115-121.

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We present a description of a functional writer's cramp case. The disease manifested as pain and tension in the right hand when writing; thus, we suspected kinesigenic dystonia in the form of writer's cramp. However, the motor pattern and the presence of additional manifestations made it possible to assume the neurotic nature of hyperkinesis. A psychiatrist diagnosed a combined conversion motor and undifferentiated somatoform disorder as a part of personality dynamics of the dramatic cluster at the involutionary age. Treatment included cognitive-behavioral therapy and periciazine administratio

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26

Newstead, Shaundra M., and Josef Finsterer. "Hyperkinesias in Leigh-like Syndrome with Complex-I Deficiency Due to m.10191T>C in MT-ND3." Annals of African Medicine 23, no. 3 (2024): 512–13. http://dx.doi.org/10.4103/aam.aam_32_23.

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Hyperkinesias in a patient with complex-I deficiency due to the variant m.10191T>C in MT-ND3 have not been previously reported. The patient is a 32 years-old female with multisystem mitochondrial disease due to variant m.10191T>C in MT-ND3, who has been experiencing episodic, spontaneous or induced abnormal movements since age 23. The abnormal movements started as right hemi-athetosis, bilateral dystonia of the legs, or unilateral dystonia of the right arm and leg. They often progressed to severe ballism, involving the trunk, and limbs. The arms were more dystonic than the legs. In concl

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27

Naimov, Olim, Rustambek Matmurodov, and Khanifa Khalimova. "Striatal hyperkinesis and neuron specific protein S100B." Journal of the Neurological Sciences 429 (October 2021): 119525. http://dx.doi.org/10.1016/j.jns.2021.119525.

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28

Hersher, Leonard. "The effectiveness of behavior modification on hyperkinesis." Child Psychiatry & Human Development 16, no. 2 (1985): 87–96. http://dx.doi.org/10.1007/bf00705943.

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29

Maxamatjanova, N. M., and G. T. Ishanxodjayeva. "Neuropsychological studies in children with tic hyperkinesis." Parkinsonism & Related Disorders 134 (May 2025): 107437. https://doi.org/10.1016/j.parkreldis.2025.107437.

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30

Bartenstein, P., O. Schober, C. Oberwittler, H. Lerch, H. Masur, and H. B. Saur. "D2-Rezeptorszintigraphie (123J-IBZM) im Vergleich zur zerebralen Perfusion (99mTc-HMPAO) bei extrapyramidalen Erkrankungen." Nuklearmedizin 33, no. 05 (1994): 184–88. http://dx.doi.org/10.1055/s-0038-1629752.

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ZusammenfassungZiel der Studie war es, zu überprüfen ob eine Korrelation zwischen zerebraler Perfusion (99mTc-HMPAO) und D2-Rezeptorbindung (123J-IBZM) bei extrapyramidalen Erkrankungen besteht. 13 Patienten mit Parkinson-Syndrom und 13 Patienten mit extrapyramidalen Hyperkinesien wurden untersucht. Bei allen Patienten wurden SPECT-Studien mit 99mTc-HMPAO und 123J-IBZM im Abstand von 2 bis 7 Tagen durchgeführt. Die Auswertung erfolgte semiquantitativ zu Frontalhirn und Zerebellum als Referenzregion für die Basalganglien. Die Streuung der Quotienten Basalganglien/Referenz war in beiden Gruppen

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31

Giovanni, Defazio. "Tic, tremors and other hyperkinesias." Toxicon 51 (June 2008): 26. http://dx.doi.org/10.1016/j.toxicon.2008.04.080.

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32

McKay, Jake H., and Jay A. van Gerpen. "Different orthostatic hyperkinesias go “Thump”." Clinical Neurophysiology Practice 2 (2017): 144–46. http://dx.doi.org/10.1016/j.cnp.2017.06.005.

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33

Ozel‐Kizil, Erguvan Tugba, M. Cenk Akbostanci, Halise D. Ozguven, and Esref Cem Atbasoglu. "Secondary social anxiety in hyperkinesias." Movement Disorders 23, no. 5 (2008): 641–45. http://dx.doi.org/10.1002/mds.21764.

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34

Popelyansky, Ya Y., A. A. Bykov, and V. N. Larina. "Role of myofascial indurations in pathogenesis of brain organic lesions." Neurology Bulletin XXXII, no. 1-2 (2000): 40–42. http://dx.doi.org/10.17816/nb77769.

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Types of motor disorders (including hyperkinesis) were studied in 42 patients with cerebral diseases. Therapeutic dynamics can be observed when stimulating peripheric foci and performing manual treatment. А major role of myofascial indurations in formation of centrale motor disorders has been revealed.

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35

Yakimovskii, Аndrey F. "Zinc, brain, behavior." Reviews on Clinical Pharmacology and Drug Therapy 19, no. 1 (2021): 23–35. http://dx.doi.org/10.17816/rcf19123-35.

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The purpose of the review was to analyze current notions about role of essential trace element zinc in brain activity and therefore in behavior. At the beginning of the review the basic data about zinc metabolism was described. The facts of zinc involvement into neurologic disorders and human cognition were represented. The results of the own investigation, devoted zinc peroral treatment and intrabrain microinjections influence on rats normal and pathological motor behavior were described. In particular, it is shown that zinc, depending on the dose and its mode of entry into the organism, can

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36

Omonov, B., R. Matmurodov, and I. Qlichev. "Etiological structure of stroke and secondary extrapyramidal hyperkinesis." Parkinsonism & Related Disorders 79 (October 2020): e104. http://dx.doi.org/10.1016/j.parkreldis.2020.06.377.

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37

SALAMONE, J. D., and M. S. COUSINES. "COMPARATIVE RESEARCH ON CHILDHOOD HYPERKINESIS/-ATTENTION DEFICIT DISORDER." Behavioural Pharmacology 7, Supplement 1 (1996): 98. http://dx.doi.org/10.1097/00008877-199605001-00224.

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38

Foreman, D. M., D. Foreman, M. Prendergast, and B. Minty. "Is clinic prevalence of ICD-10 hyperkinesis underestimated?" European Child & Adolescent Psychiatry 10, no. 2 (2001): 130–34. http://dx.doi.org/10.1007/s007870170036.

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39

Matmurodov, R. J. "INFLUENCING FACTORS ON LIFE QUALITY IN PATIENTS WITH EXTRAPYRAMIDAL DISTURBANCES." National Journal of Neurology 1, no. 09 (2016): 47–52. http://dx.doi.org/10.61788/njn.v1i16.07.

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Quality of life of patients with extrapyramidal hyperkinesia (EPH) depends on variety of different factors with leads to development of social-economic problems. 76 patients with different forms of hyperkinesia were observed. Clinical neurologic and neuropsychological examination and estimation of life quality questioner scale were conducted. Results show that quality of life of patients with EPH depends on clinical and non-clinical factors. Analysis reveal, that main factor is inheritance, so patients and all close relatives must go through genealogic and clinical examination. Timely done dif

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40

Abdukadirova, D. T. "HYPERKINESE IN TUMORS OF THE POSTERIOR CRANIAL FOSSA." National Journal of Neurology 2017, Special (2017): 110–12. http://dx.doi.org/10.61788/njn.spec.17.18.

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In connection with the rarity and unusualness of hyperkinesia and static tremor in tumors of the cerebellum and other neoplasms of the posterior cranial fossa, we give our observations of tumors of suptenorial localization, in the clinical picture of which there was a tremor of limb rest.

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41

Kurbanov, Oybek, Nodir Nurmetov, Zarifboy Ibodullaev, and Dilshoda Abdullaeva. "Pathogenetic mechanisms in epileptic myoclonic hyperkinesia." Parkinsonism & Related Disorders 22 (January 2016): e132. http://dx.doi.org/10.1016/j.parkreldis.2015.10.316.

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42

Tan, Eng-King, and Ling-Ling Chan. "Spectrum of anxiety symptoms in hyperkinesias." Movement Disorders 23, no. 12 (2008): 1795. http://dx.doi.org/10.1002/mds.22105.

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43

Bruno, Elisa, Paul Holmes, and Michalis Koutroumanidis. "A Complex Hyperkinesia Induced by Valproate." Movement Disorders Clinical Practice 3, no. 5 (2016): 513–14. http://dx.doi.org/10.1002/mdc3.12324.

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44

Rao, S. S., and R. A. Hatfield. "Paroxysmal anal hyperkinesis: a characteristic feature of proctalgia fugax." Gut 39, no. 4 (1996): 609–12. http://dx.doi.org/10.1136/gut.39.4.609.

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45

Pilina, G., and A. Ageev. "Hydroxyzine in the treatment of children with tic hyperkinesis." European Journal of Paediatric Neurology 12 (May 2008): S50. http://dx.doi.org/10.1016/s1090-3798(08)70167-3.

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46

Scheel, A., and C. Reimers. "Sonographische Darstellung von Faszikulationen und anderen Formen muskulärer Hyperkinesen." Ultraschall in der Medizin - European Journal of Ultrasound 25, no. 05 (2004): 337–41. http://dx.doi.org/10.1055/s-2004-813488.

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47

Zykov, V. P., E. A. Kashirina та Yu V. Naugolnykh. "POSSIBILITIES OF TIC HYPERKINESIA THERAPY USING ANTIСONVULSANTS". Epilepsia and paroxyzmal conditions 8, № 3 (2016): 45–51. http://dx.doi.org/10.17749/2077-8333.2016.8.3.045-051.

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48

Hallett, Joseph J., and Louise S. Kiessling. "NEUROIMMUNOLOGY OF TICS AND OTHER CHILDHOOD HYPERKINESIAS." Neurologic Clinics 15, no. 2 (1997): 333–44. http://dx.doi.org/10.1016/s0733-8619(05)70316-9.

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49

Liu, Jia, and Lu-Ning Wang. "Hyperkinesia in ancient China: Perspectives and prescriptions." Parkinsonism & Related Disorders 22 (January 2016): e133. http://dx.doi.org/10.1016/j.parkreldis.2015.10.319.

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50

Liu, Jia, and Lu-ning Wang. "Hyperkinesia in ancient China: Perspectives and prescriptions." Parkinsonism & Related Disorders 22 (January 2016): e76-e77. http://dx.doi.org/10.1016/j.parkreldis.2015.10.568.

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