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Journal articles on the topic 'Hyperkinesis'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 June 2025

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1

Khanenko, N., N. Svyrydova, G. Chuprina, et al. "Hyperkinesis: pathogenesis, clinical features, diagnosis, treatment (clinical lecture)." East European Journal of Neurology, no. 3(21) (September 20, 2018): 13–18. http://dx.doi.org/10.33444/2411-5797.2018.3(21).13-18.

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Traditionally, the basic etiological concepts are considered in the views on the morphophysiological basis of hyperkinesis. Hyperkinesia is associated with hypotonia, a decrease in muscle tone, and hyperkinetic disorders are psychogenic and manifest in childhood. Hyperkinesia can be caused by a large number of various diseases, including metabolic disorders, endocrine disruption, hereditary disorders, vascular disorders or traumatic disorders. Other causes include intoxication of the nervous system, autoimmune diseases and infections. The classification of hyperkinesis is that hyperkinetic motions can be defined as any undesirable, excessive movements that can be distinguished from each other, based on the degree to which they are rhythmic, discrete, repetitive and random. When assessing a patient with suspected hyperkinesia, the doctor thoroughly records in the history of the disease a clear description of the movements, the medications prescribed in the past and present, the family history of the similar diseases, the history of the disease, including past infections, and any other influences. Treatment is aimed at reducing symptoms, restoring normal posture and improving the general condition of the patient.

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2

Yakimovskii, Аndrey F. "The influence of acizol into effects of picrotoxin, injected in rat’s neostriatum." Medical academic journal 19, no. 2 (2019): 57–62. http://dx.doi.org/10.17816/maj19257-62.

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The aim of the article. The article is devoted to investigation of of zinc donator acizol influence to rat’s behavior, broken by intrastriatal injection of GABA-A receptor antagonist picrotoxin. Materials and methods. Adult male Wistar rats with avoidance conditioning reflexes in “shuttle box” and free locomotor activity in “open field” were used. Daily microinjection of picrotoxin (2 mcg/1 mcl) bilateral into rostral neostriatum in term of 15 days were made. Zinc donator acizol was injected intraperitoneal (24 mg/kg). Results. Steady losses of avoidance conditioning and choreo-mioclonic hyperkinesis of limbs and body, similar with human Huntington’s chorea by picrotoxin were produced. Acizol is contribute to restore avoidance conditioning and to prevent the development of hyperkinesis or essentially extend latency and lover duration of it. Conclusion. With the early data obtained, there is reason to propose, that acizol, to increasing the zinc content in the body, especially in the brain, is recover damaged cognitive function and to prevent the picrotoxin-induced hyperkinesis. Acizol should be proposed as perspective drug in extrapyramidal hyperkinetic deviation treatment in human.

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3

Yakimovskii, A. F. "INFLUENCE OF ZINC ALIMENTARY TREATMENT ON NORMAL AND PATHOLOGICAL MOTOR BEHAVIOR OF RATS." Trace Elements in Medicine (Moscow) 21, no. 2 (2020): 34–40. http://dx.doi.org/10.19112/2413-6174-2020-21-2-34-40.

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Chronic experiments were performed on rats to study the influence of prolonged acetic zinc alimentary treatment on normal (spontaneous movement in “open field and condition active avoidance reflex in “shuttle box”) and abnormal (choreomyoclonic hyperkinesia, produced by intrastriatal microinjections GABA-A receptors antagonist picrotoxin  2 mcg) motor behavior. 4 mg acetic zinc is used by rats with food ball once a day. 12 mg zinc сonsumption by rats per week did not affected on normal behavior. While 24 mg is produced smaller negative effects on rats reflex performance to 6570% correct responses (of total present during the experiment) but to improved condition avoidance behavior, violated by picrotoxinin rats and reduce the reproducibility of picrotoxin-induced choreo-mioclonic hyperkinesis (human Huntington disease hyperkinesis analog). The influence of zinc on motor behavior depending on its dose and mode of administration is discussed.

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4

Millichap, J. Gordon. "Food-Induced Hyperkinesis." Pediatric Neurology Briefs 6, no. 8 (1992): 63. http://dx.doi.org/10.15844/pedneurbriefs-6-8-10.

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5

Churton, Michael W. "Hyperkinesis: A Review of Literature." Adapted Physical Activity Quarterly 6, no. 4 (1989): 313–27. http://dx.doi.org/10.1123/apaq.6.4.313.

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The review of literature focuses upon a disorder that affects between 2 and 25% of school-age children. Commonly referred to as hyperkinesis, the disorder lacks definitive consensus on nomenclature, etiology, treatment, and symptomatology. The divergence in identifying hyperkinesis as a homogeneous disorder has prevented the development of data based educational strategies. The disorder is often associated with learning disabilities, and research in hyperkinesis or attentional deficit disorder relative to psychomotor skills and learning has been limited. Subsequently, motor activity programs have not had the resources to address the motor needs of these children. This paper reviews the divergency in the literature on hyperkinesis and offers research considerations in the area of motor learning and development for these children.

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6

Cameron, Mary, and Peter Hill. "Hyperkinetic disorder: assessment and treatment." Advances in Psychiatric Treatment 2, no. 3 (1996): 94–102. http://dx.doi.org/10.1192/apt.2.3.94.

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Hyperkinetic disorder is the generic ICD-10 (WHO, 1992) term used to describe one of the most common childhood psychiatric disorders. It is a severe form of a syndrome which is referred to in DSM–IV (APA, 1994) and the American literature as attention deficit hyperactivity disorder (ADHD). Hyperactivity or hyperkinesis can be defined as “an enduring disposition to behave in a restless, inattentive, distractible and disorganised fashion” (Taylor, 1994). It is thus more than motor overactivity. Diagnostically there are three main groups of symptomatology: overactivity, inattentiveness and impulsiveness.

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7

Levitina, E. V., M. U. Kolchanova, O. A. Rakhmanina, and E. B. Hramova. "Structure of comorbide disorders in ticose hyperkinesis in children." Medical Science And Education Of Ural 21, no. 4 (2020): 72–74. http://dx.doi.org/10.36361/1814-8999-2020-21-4-72-74.

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Purpose. To study the structure of comorbid disorders in children with tycotic hyperkinesis of the city of Tyumen. Materials and methods. We examined 103 children aged 5 to 14 years. Patients were divided into 2 groups: group 1 – local tics (n = 43), group 2 – common tics (n = 60). All patients received anticitotic therapy. Using various scales and questionnaires, an assessment of tic hyperkinesis, the level of anxiety, a study of the rational, emotional and behavioral components of the relationship between parents and a child, an investigation of the emotional sphere, and neuropsychological testing were carried out. Results. Manifestations of ticks contribute to various stressful situations, school adaptation stress. The tics are intensified by emotional overstrain, mental overwork, eye strain. A risk factor for the formation of tic hyperkinesis is family education by the type of symbiosis and hypersocialization. Comorbid disorders in patients with ticose hyperkinesis in most cases are represented by ADHD, anxiety, the prevalence of a combination of stress with pathological compensation, and the presence of a headache. The neuropsychological plan revealed impaired attention, auditory-speech and visual memory, and writing. Conclusion. Identified comorbid disorders in children with tic hyperkinesis significantly reduce the quality of life of patients, disrupts their social adaptation and require timely correction.

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8

Birkeland, S., J. Westby, K. Grong, and J. Lekven. "Effect of afterload and beta-adrenergic blockade on nonischemic myocardial contraction pattern." American Journal of Physiology-Heart and Circulatory Physiology 263, no. 6 (1992): H1716—H1723. http://dx.doi.org/10.1152/ajpheart.1992.263.6.h1716.

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We studied how changes in afterload affect regional contraction in the anterior wall of the left ventricular after circumflex coronary arterial (CFX) occlusion and subsequent beta-adrenergic blockade in pentobarbital sodium-anesthetized cats. Regional function was determined by orthogonal sonomicrometry. CFX occlusion produced nonuniform hyperkinesis in the nonischemic anterior wall; shortening of circumferential segments increased from 10.1 to 14.1% (P < 0.001), whereas shortening of longitudinal segments increased from 3.0 to 9.6% (P < 0.001). Hyperkinesis of longitudinal segments was influenced neither by changes in afterload over a pressure range of +/- 30 mmHg nor by beta-adrenergic blockade, indicating that hyperkinesis of longitudinal segments does not rely on increased inotropic state or resistance to ventricular emptying. Hyperkinesis of longitudinal segments occurred at end-diastolic lengths equal to preocclusion conditions, whereas hyperkinesis of circumferential segments was dependent on activation of the Frank-Starling mechanism. Furthermore, shortening of circumferential segments decreased with increments in afterload, particularly after CFX occlusion and subsequent beta-adrenergic blockade. In conclusion, CFX occlusion alters the contraction pattern of the nonischemic anterior wall. The postocclusion contraction is sensitive to increased afterload in the cardiac minor axis direction. These initial alterations may well direct the following remodeling process in infarcted hearts.

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9

Ruziev, A. Sh. "CLINICAL AND NEUROLOGICAL CHARACTERISTICS OF TIC HYPERKINESIS IN CHILDREN." Oriental Journal of Medicine and Pharmacology 04, no. 02 (2024): 1–6. http://dx.doi.org/10.37547/supsci-ojmp-04-02-01.

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The article presents examination data of 70 children with tic hyperkinesis. Tic hyperkinesis affects the cognitive and emotional spheres, and these changes are most pronounced with common tics. Based on the Toulouse-Pieron test, which characterizes the ability to voluntarily concentrate attention, it has been proven that children with local tics have a significantly higher concentration of attention compared to common ones.

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10

Rosenbloom, Lewis. "LEARNING DISABILITIES AND HYPERKINESIS." Developmental Medicine & Child Neurology 14, no. 3 (2008): 394–95. http://dx.doi.org/10.1111/j.1469-8749.1972.tb02606.x.

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11

May, Mark. "Management of Facial Hyperkinesis." Otology & Neurotology 23, Sup 1 (2002): S70. http://dx.doi.org/10.1097/00129492-200200001-00179.

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12

MUNASIPOVA, S. E., Z. A. ZALYALOVA, and D. M. KHASANOVA. "Comorbidity of tic hyperkineses with impulsive disorders." Practical medicine 18, no. 5 (2020): 88–92. http://dx.doi.org/10.32000/2072-1757-2020-5-88-92.

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Tourette’s syndrome, the most common cause of tics, manifests itself in a wide range of motor and behavioral disorders and is found in men about three times more often than in women. The purpose — to establish the comorbidity of neuropsychiatric disorders in patients with tic hyperkinesis. Material and methods. The study included 111 patients with tic hyperkinesis of various ages and sex, who were outpatiently observed at the Clinical and Diagnostic Center for Movement Disorders and Botulinum Therapy of the Republic of Tatarstan from 2011 to 2014. Results and conclusions. In the group of patients we studied, tics were more common in males (in 71.6% of cases), and patients with Tourette’s syndrome were numerically predominant, taking into account the visits to the Clinical and Diagnostic Center for Movement Disorders and Botulinum Therapy of the Republic of Tatarstan. Clinical manifestations of obsessive-compulsive disorders were found not only in patients with Tourette’s syndrome, but they are characteristic of 53.2% of patients with tic hyperkinesis of various severity and phenomenology. Also, regardless of the structural and phenomenological characteristics of tic hyperkinesis, they were almost always associated with attention deficit hyperactivity disorder with a predominance of hyperactivity.

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13

Tsygan, N. V., Yu I. Vainshenker, I. V. Litvinenko, and M. M. Odinak. "Muscular-tonic disorders in a vegetative and a minimal consciousness states." Russian neurological journal 28, no. 1 (2023): 25–32. http://dx.doi.org/10.30629/2658-7947-2023-28-1-25-32.

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Muscular-tonic disorders (MTD) in prolonged disorders of consciousness (PDoC), including a vegetative state (VS) and a minimal consciousness state (MCS), are poorly understood.Aim. To systematize MTD in PDoC, to highlight the features of their dynamics depending on the change in consciousness.Material and methods. 87 patients in PDoC (VS — 52, MCS — 35) resulting from brain damage of diff erent etiology, lasting from 2 months up to 10 years. MTD, provoking hyperkinesis factors and consciousness were analyzed retrospectively in the dynamics and complex.Results. MTD had 98% of patients in PDoC. The ratio of occurrence of spasticity: hyperkinesis: postural spasms corresponded to 11:11:10, and hyperkinesis — dystonia: myoclonus/myokymia: athetosis: stereotypes: ballism: choreiform hyperkinesis: tremor — 17:10:6:3:2:1:1. Their clinical variants and features are noted. The total dynamics of MTD on improved consciousness was as follows. In general, regardless of the initial VS or MCS, their “change” (especially decrease) prevailed over “no changes” (p < 0.001). Comparatively more often the decrease occurred in MCS “plus” (p < 0.05); appearance/increase/modifi cation — in VS (p < 0.05); “no changes” — in MCS “minus” (p < 0.01). When considered separately, spasticity, dystonia, spasms, hemiballismus and stereotypy in MCS “plus” correlated (p < 0.01) with the change of consciousness. The key provoking hyperkinesis factors in VS were pain and other sensory infl uences (p < 0.01), but their role from MCS “minus” to MCS “plus” decreased, while the role both conscious emotions and movements increased (p < 0.01).Conclusion. Along with academic interest, the data are promising in developing the prognosis, pathogenesis and treatment of PDoC.

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14

Foreman, David M., Alexis Beedie, and Ranjit Kapuge. "Clinic assessment for methylphenidate maintenance therapy in secondary care: are parental questionnaires useful?" Psychiatric Bulletin 30, no. 11 (2006): 406–9. http://dx.doi.org/10.1192/pb.30.11.406.

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Aims and MethodThere is poorer control of hyperactivity symptoms in community clinics than research settings, and difficulty in detecting such symptoms without standardised measures. Hyperkinetic children (n=29) were evaluated at follow-up using the parental version of the Strengths and Difficulties Questionnaire (SDQ) and, independently, routine clinic reports to test the value of a parental questionnaire.ResultsThe parental SDQ identified symptoms in more patients (25, 86% v. 13, 45%), but high levels of symptoms did not necessarily imply impairment. Even clinically identified hyperactivity provoked no change in treatment.Clinical ImplicationsParental questionnaires alone are unlikely to improve clinic practice. Research is needed into what factors influence clinical decisions regarding treatment maintenance for hyperkinesis, and the adaptation of structured protocols from major research trials should be considered.

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15

Tarianyk, K. A. "DIAGNOSTIC DIFFICULTIES IN MOTOR DISORDERS." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 20, no. 2 (2020): 275–79. http://dx.doi.org/10.31718/2077-1096.20.2.275.

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The article is devoted to the diagnosis of motor disorders in extrapyramidal pathology, namely, the course of multisystem atrophy, which is a steadily progressing neurodegenerative disease, accompanied by damage to the pyramidal system, cerebellum and autonomic nervous system and is associated with degeneration of nerve cells in certain parts of the brain. Emphasis is placed on the diagnosis of conditions in the clinical picture of which tremor dominates, which occurs as a result of damage to the structures of the nervous system related to the regulation of muscle tone and unreasonable control of movements (cerebellum, basal ganglia). Tremor is hyperkinesis, which is manifested by involuntary oscillatory rhythmic movements of a part (limbs, head) or the whole body as a result of a constant repetition of muscle contraction and relaxation. Making diagnosis of the diseases underlying this condition is often a challenging task that requires a proper diagnostic approach. There has been presented clinical and paraclinical methods for the diagnosis of trembling hyperkinesis, described advantages of electromyography (tremorography, ultrasonography) in the diagnosis of extrapyramidal diseases. The article includes a case report of a patient with hyperkinetic syndromes at multisystem atrophy, who was examined and treated at the Centre for Patients with Parkinson's disease and other neurodegenerative diseases at the Department of Nervous Diseases with Neurosurgery and Medical Genetics, Ukrainian Medical Dental Academy, Neurological Department of the Poltava Regional Clinical Hospital, details the stages in making final diagnosis. The article sums up on the efficiency of neurophysiological methods in early diagnosis and in disease progression.

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Maxamatjanova, N., and G. Ishankhodjaeva. "Psychocorrection of children with ticose hyperkinesis." Parkinsonism & Related Disorders 79 (October 2020): e108. http://dx.doi.org/10.1016/j.parkreldis.2020.06.393.

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17

Ciniglio, Ricardo, Mark Kime, Trudy L. Burns, and Byron F. Vandenberg. "Rapid Resolution of Hyperkinesis After Exercise." Chest 104, no. 3 (1993): 712–17. http://dx.doi.org/10.1378/chest.104.3.712.

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18

Bogacheva, V. A., D. V. Zakharov, and V. A. Mikhailov. "Efficacy of antiparkinsonian therapy in tremor correction according to tremorography." Neurology, Neuropsychiatry, Psychosomatics 16, no. 2 (2024): 34–40. http://dx.doi.org/10.14412/2074-2711-2024-2-34-40.

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Objective: to evaluate the efficacy of antiparkinsonian drugs of different groups considering the frequency-amplitude characteristics in the correction of tremulous hyperkinesis in Parkinson's disease (PD) using tremorography. Material and methods. 172 patients with a confirmed diagnosis of PD were observed. Taking into account age and contraindications, patients were divided into four groups receiving one of four drugs in monotherapy: dopamine receptor agonists (ADR; pramipexole), levodopa (Tidomet forte), anticholinergics (biperiden), amantadines (amantadine sulfate). Before starting treatment and after completion of the selected therapy, patients underwent tremorography to analyse the frequency, amplitude and duration of tremor over time in four standard tests. Results. All antiparkinsonian drugs reduced rest tremor, and the efficacy of pramipexole in treating tremor was comparable to that of levodopa in the early stages of the disease. As the severity of the disease increased, the efficacy of ADRs decreased, whereas levodopa became more important. Amantadine sulfate showed the lowest efficacy in rest tremor. The efficacy of anticholinergics against tremor is high but inconsistent. Conclusion. We demonstrated feasibility of using tremorography in the selection of an individualised treatment programme for patients with shaking hyperkinesis in PD , and the high efficacy of levodopa in shaking hyperkinesis was confirmed.

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19

Tolmacheva, V. A., P. G. Yuzbashyan, R. M. Shishorin, B. A. Volel, and D. V. Romanov. "Functional writer's cramp as psychogenic focal dystonia." Neurology, Neuropsychiatry, Psychosomatics 14, no. 1 (2022): 115–21. http://dx.doi.org/10.14412/2074-2711-2022-1-115-121.

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We present a description of a functional writer's cramp case. The disease manifested as pain and tension in the right hand when writing; thus, we suspected kinesigenic dystonia in the form of writer's cramp. However, the motor pattern and the presence of additional manifestations made it possible to assume the neurotic nature of hyperkinesis. A psychiatrist diagnosed a combined conversion motor and undifferentiated somatoform disorder as a part of personality dynamics of the dramatic cluster at the involutionary age. Treatment included cognitive-behavioral therapy and periciazine administration with a positive effect. We discuss the aspects of hyperkinesis and mental status, which help differentiate the kinesigenic form of dystonia (writer's cramp) and functional movement disorder.

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20

Kolesnik, Olena. "Clinical and pathogenetic features and correction of hyperkinetic disorders in vegetative dysfunctions." Ukrains'kyi Visnyk Psykhonevrolohii, Volume 28, issue 1 (102) (March 25, 2020): 6–9. http://dx.doi.org/10.36927/20790325-v28-is1-2020-1.

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One of the early syndromes in vascular pathology is motor disorders in the form of tremor. We examined 101 patients with cerebral dystonia on the background of chronic cerebral ischemia with compensated and subcompensated stages (n = 55) and autonomic dystonia syndrome (n = 46). Pathological mechanisms of formation of trembling hyperkinesis in cerebral angiodystonia of different etiology have been studied. Participation in these processes of the autonomic nervous system (ANS) is established. It is proved that the severity of this type of motor disorders depends on the condition of the ANS at different levels of its organization, especially on the background of sympathicotonic effects, as well as the vegetative characteristics of the patient. At the same time, signifi cant changes in electrophysiological parameters, as well as data of vegetative, emotional, psychometric testing, were obtained. For an objective evaluation of the severity of the tremor, a tremograph with the calculation of an integrative tremographic index was used. Developed a complex of cerebral angiodystonia with hyperkinetic syndrome and the use of antioxidants, nootropic drugs, which allowed to reduce or eliminate subjective experiences, to increase the adaptation capacity of the organism with the normalization of sympatho-parasympathetic relationships, to the normalization of the organism. also inhibition of the implementation of trembling hyperkinesis in both research groups. The average values of the tremographic index decreased towards an adequate distribution with sympathicolytic influence on the indices of vegetative testing and caused skin sympathetic potentials. At the same time, a greater effect was achieved when using the proposed method of treatment, with the maximum — in the syndrome of autonomic dystonia, as well as in the compensated stage of chronic brain ischemia. Keywords: cerebral angioedema, cerebral ischemia, autonomic disorders, tremor, treatment

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21

Naimov, Olim, Rustambek Matmurodov, and Khanifa Khalimova. "Striatal hyperkinesis and neuron specific protein S100B." Journal of the Neurological Sciences 429 (October 2021): 119525. http://dx.doi.org/10.1016/j.jns.2021.119525.

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22

Hersher, Leonard. "The effectiveness of behavior modification on hyperkinesis." Child Psychiatry & Human Development 16, no. 2 (1985): 87–96. http://dx.doi.org/10.1007/bf00705943.

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23

Maxamatjanova, N. M., and G. T. Ishanxodjayeva. "Neuropsychological studies in children with tic hyperkinesis." Parkinsonism & Related Disorders 134 (May 2025): 107437. https://doi.org/10.1016/j.parkreldis.2025.107437.

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Omonov, B., R. Matmurodov, and I. Qlichev. "Etiological structure of stroke and secondary extrapyramidal hyperkinesis." Parkinsonism & Related Disorders 79 (October 2020): e104. http://dx.doi.org/10.1016/j.parkreldis.2020.06.377.

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SALAMONE, J. D., and M. S. COUSINES. "COMPARATIVE RESEARCH ON CHILDHOOD HYPERKINESIS/-ATTENTION DEFICIT DISORDER." Behavioural Pharmacology 7, Supplement 1 (1996): 98. http://dx.doi.org/10.1097/00008877-199605001-00224.

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Foreman, D. M., D. Foreman, M. Prendergast, and B. Minty. "Is clinic prevalence of ICD-10 hyperkinesis underestimated?" European Child & Adolescent Psychiatry 10, no. 2 (2001): 130–34. http://dx.doi.org/10.1007/s007870170036.

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Popelyansky, Ya Y., A. A. Bykov, and V. N. Larina. "Role of myofascial indurations in pathogenesis of brain organic lesions." Neurology Bulletin XXXII, no. 1-2 (2000): 40–42. http://dx.doi.org/10.17816/nb77769.

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Types of motor disorders (including hyperkinesis) were studied in 42 patients with cerebral diseases. Therapeutic dynamics can be observed when stimulating peripheric foci and performing manual treatment. А major role of myofascial indurations in formation of centrale motor disorders has been revealed.

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Yakimovskii, Аndrey F. "Zinc, brain, behavior." Reviews on Clinical Pharmacology and Drug Therapy 19, no. 1 (2021): 23–35. http://dx.doi.org/10.17816/rcf19123-35.

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The purpose of the review was to analyze current notions about role of essential trace element zinc in brain activity and therefore in behavior. At the beginning of the review the basic data about zinc metabolism was described. The facts of zinc involvement into neurologic disorders and human cognition were represented. The results of the own investigation, devoted zinc peroral treatment and intrabrain microinjections influence on rats normal and pathological motor behavior were described. In particular, it is shown that zinc, depending on the dose and its mode of entry into the organism, can weaken and prevent the development of picrotoxin-induced neostriatal hyperkinesis (human Huntington horea analog), but it may aggravate hyperkinesis symptoms and even independently cause the motor stereotypy. On the basis of their own data and literary, it was suggested that neurons membranes structures are different sensitive to a certain zinc concentration and what does the specific way of behavior realization is ultimately depend.

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Rao, S. S., and R. A. Hatfield. "Paroxysmal anal hyperkinesis: a characteristic feature of proctalgia fugax." Gut 39, no. 4 (1996): 609–12. http://dx.doi.org/10.1136/gut.39.4.609.

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Pilina, G., and A. Ageev. "Hydroxyzine in the treatment of children with tic hyperkinesis." European Journal of Paediatric Neurology 12 (May 2008): S50. http://dx.doi.org/10.1016/s1090-3798(08)70167-3.

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Birkeland, S., J. Westby, S. Faerestrand, and K. Grong. "Changes in myocardial contraction patterns in response to regional ischemia and sympathetic nerve stimulation." American Journal of Physiology-Heart and Circulatory Physiology 264, no. 2 (1993): H533—H540. http://dx.doi.org/10.1152/ajpheart.1993.264.2.h533.

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We studied the effect of inferior cardiac nerve stimulation on global and regional left ventricular function before and after circumflex coronary arterial (CFX) occlusion in 12 pentobarbital-anesthetized cats. Regional function was evaluated by orthogonal sonomicrometry in the anterior midwall of the left ventricle and by two-dimensional short-axis echocardiography. CFX occlusion provoked hypokinesis in the nonischemic free wall adjacent to the dyskinetic ischemic region (17.6 +/- 2.3 vs. 34.6 +/- 2.2% preocclusion wall thickening; P < 0.01) and gradually improved systolic wall thickening toward the contralateral remote region, where the maximal hyperkinetic response was measured (52.9 +/- 4.4 vs. 32.5 +/- 3.3% preocclusion wall thickening; P < 0.001). In the anterior wall, remote region, hyperkinesis was predominant in longitudinal segments (8.1 +/- 0.8 vs. 2.3 +/- 0.6% preocclusion ejection shortening; P < 0.001) and slight in circumferential segments (11.0 +/- 0.7 vs. 9.1 +/- 0.6% preocclusion ejection shortening; P < 0.01). The postocclusion systolic wall thickening and segment-shortening pattern were not modified by sympathetic nerve stimulation, which increased cardiac output less after coronary occlusion, although contractility (dP/dt) increased markedly. In conclusion, the myocardial contraction pattern in ischemic and nonischemic regions after CFX occlusion is not much modified by sympathetic cardiac nerve stimulation.

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Смагулова, А. Р., Г. Б. Кадржанова, А. Ш. Избасарова, and Н. Д. Тулекова. "FEATURES OF THE MANIFESTATION AND ETIOLOGICAL FACTORS OF DYSKINETIC FORMS OF CEREBRAL PALSY IN PREMATURE INFANTS." Vestnik, no. 3 (December 15, 2021): 273–75. http://dx.doi.org/10.53065/kaznmu.2021.87.80.052.

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В данной статье представлен анализ особенностей проявлений дискинетической формы церебрального паралича у недоношенных детей. В статье описаны ранние проявления данной патологии, сроки формирования гиперкинезов, этиологические факторы. This article presents an analysis of the features of the manifestations of the dyskinetic form of cerebral palsy in premature infants. The article describes the early manifestations of this pathology, the timing of the formation of hyperkinesis, and etiological factors.

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33

Zykov, V. P., E. S. Kashirina, and Yu V. Naugolnykh. "Electromyography in childhood patients with tic hyperkinesis and Tourette’s syndrome." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 121, no. 11 (2021): 19. http://dx.doi.org/10.17116/jnevro202112111219.

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34

Porter, Luz S. "The what, why and how of hyperkinesis: implications for nursing." Journal of Advanced Nursing 13, no. 2 (1988): 229–36. http://dx.doi.org/10.1111/j.1365-2648.1988.tb01412.x.

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35

Sattarova, D., Y. Madjidova, N. Azimova, and K. Achilova. "Psychotherapeutic treatment of cognitive impairtment in children with tic hyperkinesis." Parkinsonism & Related Disorders 46 (January 2018): e49. http://dx.doi.org/10.1016/j.parkreldis.2017.11.168.

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36

Pivik, R. T., and F. Bylsma. "Spinal motoneuronal excitability in hyperkinesis: effects of the Jendrassik manoeuvre." International Journal of Psychophysiology 9, no. 1 (1990): 85–95. http://dx.doi.org/10.1016/0167-8760(90)90010-b.

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37

Hess, Ellen J., Katherine A. Collins, and Michael C. Wilson. "Mouse Model of Hyperkinesis Implicates SNAP-25 in Behavioral Regulation." Journal of Neuroscience 16, no. 9 (1996): 3104–11. http://dx.doi.org/10.1523/jneurosci.16-09-03104.1996.

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38

Filipo, Roberto, Irma Spahiu, Edoardo Covelli, Maria Nicastri, and Gian Antonio Bertoli. "Botulinum toxin in the treatment of facial synkinesis and hyperkinesis." Laryngoscope 122, no. 2 (2012): 266–70. http://dx.doi.org/10.1002/lary.22404.

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39

Anast, Constantine S. "FDA Proposals The National Advisory Committee on Hyperkinesis and Food Additives." Nutrition Reviews 33, no. 7 (2009): 220–23. http://dx.doi.org/10.1111/j.1753-4887.1975.tb05222.x.

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40

Garami, A., E. Pakai, D. L. Oliveira, et al. "Thermoregulatory Phenotype of the Trpv1 Knockout Mouse: Thermoeffector Dysbalance with Hyperkinesis." Journal of Neuroscience 31, no. 5 (2011): 1721–33. http://dx.doi.org/10.1523/jneurosci.4671-10.2011.

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41

GILLBERG, I. CARINA, and CHRISTOPHER GILLBERG. "Generalized Hyperkinesis: Follow-up Study From Age 7 to 13 Years." Journal of the American Academy of Child & Adolescent Psychiatry 27, no. 1 (1988): 55–59. http://dx.doi.org/10.1097/00004583-198801000-00009.

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42

Sackler, A. M., and A. S. Weltman. "Effects of methylphenidate on whirler mice: An animal model for hyperkinesis." Life Sciences 37, no. 5 (1985): 425–31. http://dx.doi.org/10.1016/0024-3205(85)90404-7.

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43

Foreman, D. M. "The association between hyperkinesis and breakdown of parenting in clinic populations." Archives of Disease in Childhood 90, no. 3 (2005): 245–48. http://dx.doi.org/10.1136/adc.2003.039826.

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44

de Maio, Maur??cio, and Ricardo Ferreira Bento. "Botulinum Toxin in Facial Palsy: An Effective Treatment for Contralateral Hyperkinesis." Plastic and Reconstructive Surgery 120, no. 4 (2007): 917–27. http://dx.doi.org/10.1097/01.prs.0000244311.72941.9a.

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45

Burlton-Bennet, Jocelyn A., and Viviane M. J. Robinson. "A Single Subject Evaluation of the K-P Diet for Hyperkinesis." Journal of Learning Disabilities 20, no. 6 (1987): 331–35. http://dx.doi.org/10.1177/002221948702000605.

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46

Calhoun, George, and James A. Bolton. "Hypnotherapy: A Possible Alternative for Treating Pupils Affected with Attention Deficit Disorder." Perceptual and Motor Skills 63, no. 3 (1986): 1191–95. http://dx.doi.org/10.2466/pms.1986.63.3.1191.

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Abstract:

The purpose of this study was to ascertain the effectiveness of hypnotherapy in treating attention-deficit-hyperactivity by certified psychologist and physicians. The 7 boys and 4 girls were enrolled in a Special Day classroom for hyperkinesis and behavior problems and were being given methylphenidate (Ritalin). Three attempts were made to hypnotize these subjects; however, the group had to be reduced from 11 to 3 and finally, from 3 to a single child to be successful. A significant improvement from pre- and post-hypnotic sessions was noted.

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Macić-Džanković, Amra, and Meliha Pozderac-Memija. "Specificity of Electrocardiography and Echocardiography Changes at the Patients with the Pulmonary Embolism." Bosnian Journal of Basic Medical Sciences 6, no. 4 (2006): 72–75. http://dx.doi.org/10.17305/bjbms.2006.3126.

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We evaluated electrocardiographic and echocardiographic changes of 40 patients with pulmonary thrombo-embolism proved by perfusion scintigrams. ECG-changes included sinus tachycardia or absolute tachyarrhythmia de novo, changes type Q1S3T3 and changes in right precordial leads. Analyses of echocardiography included hyperkinesis and then dilatation and apical hypokinesis of right ventricule and tricuspid regurgitation with maximal transvalvular gradients. We emphasize such a sensibility of echocardiographic changes in early estimation of pulmonary thrombo-embolism severity and necessity for echocardiography as early as possible in suspected patients.

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Goodman, Robert. "Neuronal Misconnections and Psychiatric Disorder." British Journal of Psychiatry 154, no. 3 (1989): 292–99. http://dx.doi.org/10.1192/bjp.154.3.292.

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Abstract:

Brain damage can induce anomalous neuronal connections in experimental animals, which can sometimes result in maladaptive behaviour, particularly when damage occurs early in development. Anomalous patterns of neuronal connection can also arise from genetic disorders. In humans, neuronal misconnections could be involved in a variety of psychiatric disorders. For example, they may account for the link between hyperkinesis and childhood hemiplegia, and for the link between schizophrenia and ‘alien tissue’ lesions of the temporal lobes. Predictions from misconnection hypotheses can potentially be tested in neuropathological, neurophysiological, and clinical studies.

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Goto, Yoichi, Yuichiro Igarashi, Osamu Yamada, et al. "-150-MECHANISMS OF HYPERKINESIS IN NONISCHEMIC REGION OF ACUTE ISCHEMIC CANINE HEART." Japanese Circulation Journal 50, no. 6 (1986): 503. http://dx.doi.org/10.1253/jcj.50.503_1.

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Dzhanumova, Galina Mikhailovna, L. B. Ivanov, and A. V. Budkevich. "EXPERIENCE IN USING TENOTEN IN THE TREATMENT OF TICK HYPERKINESIS IN CHILDREN." Neurology, neuropsychiatry, Psychosomatics, no. 1 (March 14, 2010): 53. http://dx.doi.org/10.14412/2074-2711-2010-71.

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