Relevant bibliographies by topics / Hyperkinetický syndrom

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Hyperkinetický syndrom'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Hyperkinetický syndrom"

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Lejčarová, Alena, and Martina Skálová. "The use of canistherapy in a child with hyperkinetic syndrome." Kontakt 11, no. 2 (December 18, 2009): 413–23. http://dx.doi.org/10.32725/kont.2009.064.

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EGGER, JOSEPH. "Hyperkinetic Syndrome." Journal of Nutritional & Environmental Medicine 7, no. 4 (January 1997): 353–57. http://dx.doi.org/10.1080/13590849762484.

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Glushchenko, Vita Valentinovna. "Rational pharmacotherapy of hyperkinetic disorders." Reviews on Clinical Pharmacology and Drug Therapy 12, no. 4 (December 15, 2014): 58–63. http://dx.doi.org/10.17816/rcf12458-63.

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The data concerning hyperkinetic disorder syndrome are shortly reviewed in the paper. Principal attention is focused on the analysis of effects of metabolic activators (atomoxetine), neuroleptics (thioridazine, chlorprotixene) and different antidepressants (fluoxetine, fluvoxamine, sertraline, tianeptine, pipophesine).
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Kay, A. B., A. J. Frew, J. O. Warner, M. H. Lessof, Joseph Egger, Adelheid Stolla, and LeonardM Mcewen. "Hyposensitisation for food-induced hyperkinetic syndrome." Lancet 341, no. 8837 (January 1993): 114–15. http://dx.doi.org/10.1016/0140-6736(93)92592-h.

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Franklin, A. J. "Hyposensitisation for food-induced hyperkinetic syndrome." Lancet 341, no. 8842 (February 1993): 437. http://dx.doi.org/10.1016/0140-6736(93)93031-u.

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Haddad, P. M., and M. E. Garralda. "Hyperkinetic Syndrome and Disruptive Early Experiences." British Journal of Psychiatry 161, no. 5 (November 1992): 700–703. http://dx.doi.org/10.1192/bjp.161.5.700.

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Five children aged 8–10 years presenting to child psychiatric clinics with hyperkinetic syndrome and severe disruptive early experiences are described. At referral all had been living in stable families for a minimum of four years. Biological risk factors were absent. It is proposed that the disruption of early attachments resulted in ill-regulated search and exploratory behaviour and that this had a continuing effect on the children's ability to focus and control attention skills and motor behaviour.
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Rapoport, Judith L., and H. Bruce Ferguson. "Biological Validation of the Hyperkinetic Syndrome." Developmental Medicine & Child Neurology 23, no. 6 (November 12, 2008): 667–82. http://dx.doi.org/10.1111/j.1469-8749.1981.tb02053.x.

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Steinhausen, Hans-Christoph, and Dietmar G�bel. "The validity of the hyperkinetic syndrome." European Archives of Psychiatry and Neurological Sciences 235, no. 2 (1985): 122–28. http://dx.doi.org/10.1007/bf00633484.

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Cameron, Mary, and Peter Hill. "Hyperkinetic disorder: assessment and treatment." Advances in Psychiatric Treatment 2, no. 3 (May 1996): 94–102. http://dx.doi.org/10.1192/apt.2.3.94.

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Hyperkinetic disorder is the generic ICD-10 (WHO, 1992) term used to describe one of the most common childhood psychiatric disorders. It is a severe form of a syndrome which is referred to in DSM–IV (APA, 1994) and the American literature as attention deficit hyperactivity disorder (ADHD). Hyperactivity or hyperkinesis can be defined as “an enduring disposition to behave in a restless, inattentive, distractible and disorganised fashion” (Taylor, 1994). It is thus more than motor overactivity. Diagnostically there are three main groups of symptomatology: overactivity, inattentiveness and impulsiveness.
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TP, Borysova, Badogina LP, Allahverdieva ZS, and Samsonenko SV. "Hyperkinetic Syndrome in Adolescent with HIV-Infection." Acta Scientific Paediatrics 4, no. 5 (April 23, 2020): 09–12. http://dx.doi.org/10.31080/aspe.2020.03.0243.

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Dissertations / Theses on the topic "Hyperkinetický syndrom"

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Eekhof, Job Lambert Adam. "Electrophysiological investigations in cranial hyperkinetic syndromes." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2000. http://dare.uva.nl/document/81828.

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Volodkovič, Stanislava. "Vaikų turinčių hiperkinezinį sindromą socializacijos ypatumai pradinėse klasėse." Master's thesis, Lithuanian Academic Libraries Network (LABT), 2009. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2009~D_20090630_094115-00810.

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Daugeliui žmonių vaikai yra ne tik didžiausia gyvenimo palaima, bet ir rimčiausias iššūkis. Šioje grupėje ypač išsiskiria negebantys sutelkti dėmesio, judrūs, impulsyvūs vaikai. Kuo labiau padidėjęs vaiko aktyvumas ir impulsyvumas, tuo dažniau jam kyla psichosocialinės adaptacijos sunkumų, ypač vaikų kolektyve. Sutrikusio elgesio, nedėmesingų, impulsyvų vaikų socializacija mokykloje yra svarbi ir sudėtinga pedagoginė problema, kartu tai vienas iš dažniausiai pasitaikančių elgesio ir socialinės raidos sutrikimų tarp vaikų. Išanalizuotoje literatūroje radau, kad visuomenėje yra nuo 3 iki 10 procentų vaikų turinčių hiperkinezinį sindromą. Tirtose mokyklose buvo nustatyta 8% vaikų, turinčių hiperkinezinį sindromą. Nemažas procentas yra impulsyvių, nedėmesingų, neklusnų vaikų. Mokinių, turinčių hiperkinezinį sindromą, tėvai ne visada supranta šio sutrikimo esmę ir dažnai nereaguoja į vaiko hiperaktyvumą, dėmesio stoką ir impulsyvumą. Dauguma vaikų tirtose mokyklose negali pakankamai ilgai susikaupti dėmesį ruošiant pamokas. Tokiems vaikams ne tik sunku paruošti pamokas, bet ir mokytis mokykloje, atlikti tam tikrą veiklą.. Pradinių klasių mokytojai negeba naudotis teisingumu dirbdami su tokiais mokiniais, turinčiais hiperkinezinį sindromą: pernelyg retai vaikas iš anksto supažindinamas su tinkamo elgesio taisyklėmis, retai jam deferencijojamos ir dozuojamos užduotis, vaikas pernelyg retai gauna teigiamą įvertinimą. Mokyklose nėra specialiųjų pedagogu, niekas neužsiima vaiko... [toliau žr. visą tekstą]
Problem. Socialisation of primary school pupils with hyperkinetic syndrome. The object of research. Primary school pupils with hyperkinetic syndrome. The aim of research. Analyse the peculiarities and problems of socialisation of Polish primary school pupils with hyperkinetic syndrome. Objectives: 1. To give general characteristics of children with hyperkinetic syndrome. 2. To reveal the problems of Polish – speaking children with hyperkinetic syndrome and suggest some ways to their solution. 3. To define the role of an educator in helping the children with hyperkinetic syndrome. Most people`s children are not only the greatest blessing but also the most serious challenge. This group includes excessively active and impulsive children with attention – deficit. The more active and impulsive are children, the more often they have psychosocial adaptation difficulties, especially in a children`s group. Socialisation of inattentive, impulsive school children with disruptive behaviour is an important and complicated pedagogical problem. Moreover, it is one of the most often disorders of behaviour and social development in children. Having analysed some literary resources, I have found out that in some western countries, as well as in the USA, there are 3 to 10 per cent of children with hyperkinetic syndrome. Such statistical researches have not been conducted in Lithuania. The research which I have conducted can`t be considered to... [to full text]
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Cavallin, Mara. "Physiopathologie moléculaire et cellulaire des anomalies du développement du cortex cérébral : le syndrome d'Aicardi WDR81 mutations cause extreme microcephaly and impair mitotic progression in human fibroblasts and Drosophila neural stem cells TLE1, a key player in neurogenesis, a new candidate gene for autosomal recessive postnatal microcephaly Mutations in TBR1 gene leads to cortical malformations and intellectual disability Aicardi syndrome: Exome, genome and RNA-sequencing of a large cohort of 19 patients failed to detect the genetic cause Recurrent RTTN mutation leading to severe microcephaly, polymicrogyria and growth restriction Recurrent KIF2A mutations are responsible for classic lissencephaly Recurrent KIF5C mutation leading to frontal pachygyria without microcephaly Rare ACTG1 variants in fetal microlissencephaly De novo TUBB2B mutation causes fetal akinesia deformation sequence with microlissencephaly: An unusual presentation of tubulinopathy A novel recurrent LIS1 splice site mutation in classic lissencephaly Further refinement of COL4A1 and COL4A2 related cortical malformations Prenatal and postnatal presentations of corpus callosum agenesis with polymicrogyria caused By EGP5 mutation Delineating FOXG1 syndrome from congenital microcephaly to hyperkinetic encephalopathy Delineating FOXG1 syndrome: From congenital microcephaly to hyperkinetic encephalopathy." Thesis, Sorbonne Paris Cité, 2019. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2213&f=18201.

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Les malformations du cortex cérébral (MDC) représentent une cause importante de handicap et d'épilepsie pharmaco-résistante. Le séquençage à haut débit a permis une amélioration considérable de l'identification des bases moléculaires des MDC non syndromiques. Toutefois, certaines formes, notamment les MDC complexes, demeurent inexpliquées. Mon projet de thèse a pour objectif de progresser dans la compréhension des MDC complexes en utilisant deux modèles : les microlissencéphalies (MLIS) et le syndrome d'Aicardi (AIC), une forme syndromique particulière associant des malformations de l'oeil et du cerveau uniquement rapporté chez les filles. L'étude par séquençage d'exome en trios de 16 familles MLIS m'a permis d'identifier et de caractériser un nouveau gène, WDR81, impliqué dans le cycle cellulaire. Par la même stratégie, j'ai pu identifier un variant homozygote pathogène dans TLE1, un partenaire majeur de FOXG1 dans la balance prolifération/différenciation de progéniteurs neuronaux, dans une famille consanguine de microcéphalie postnatale dont le phénotype est proche du syndrome FOXG1. En parallèle, mes travaux ont permis de préciser les spectres phénotypiques associés à RTTN, EPG5, COL4A1, COL4A2, TBR1, KIF5C, KIF2A et FOXG1. La deuxième partie de mon projet avait pour objet l'identification des bases moléculaires du syndrome d'Aicardi à partir d'une cohorte internationale de 19 patientes. Après avoir exclu un biais d'inactivation du chromosome X et la présence de microremaniements chromosomiques, j'ai réalisé un séquençage d'exome en trio. Aucun variant récurrent n'a été retrouvé dans les séquences codantes. Dans un second temps, j'ai testé une approche combinant les données du séquençage de génome et l'analyse du transcriptome (RNA-Seq) sur fibroblastes, me permettant d'identifier des transcrits dérégulés qui étaient impliqués dans le développement du cerveau et de l'oeil. J'ai comparé les résultats de cette analyse avec ceux de l'analyse du génome dans le but d'identifier des variants dans ces gènes candidats. En conclusion, mon travail de thèse a permis d'améliorer la connaissance des bases moléculaires des MDC complexes et d'ouvrir des perspectives de nouveaux mécanismes tels que ceux engageant les gènes WDR81 et EPG5, et le rôle des endosomes et de l'autophagie dans les MDC, et aussi TLE1 comme nouvelle cause de microcéphalies postnatales. Mes travaux ont également permis de générer une collection de données de séquençage haut débit (WES, WGS et RNA-Seq) qui seront mises en commun dans le cadre d'un consortium international afin de développer des nouvelles stratégies d'analyse en particulier pour les séquences non codantes. Cette approche permettra également d'ouvrir la voie vers la compréhension des mécanismes cellulaires impliqués dans la formation du cerveau et de l' œil
Malformations of cortical development (MCD) are a major cause of intellectual disability and drug-resistant epilepsy. Next Generation Sequencing (NGS) has considerably improved the identification of the molecular basis of non-syndromic MCD. However, certain forms, including complex MCD, remain unexplained. My PhD project aimed to improve the understanding of complex MCD using two disorders: Microlissencephaly (MLIS) and Aicardi Syndrome (AIC), the latter associating brain and eye malformations and only reported in girls. Trio Whole Exome Sequencing (WES) performed in 16 MLIS families allowed me to identify and functionally characterize a new MLIS gene, WDR81, in which mutations lead to cell cycle alteration. Moreover, using the same strategy, I was able to identify a pathogenic homozygous variant in TLE1 in a patient from consanguineous family with a postnatal microcephaly, suggestive of a FOXG1-like presentation. Interestingly, TLE1 is a major partner of FOXG1, a gene involved in maintaining the balance between progenitor proliferation and differentiation. In parallel, my work allowed me to redefine the phenotypic spectrum associated with RTTN, EPG5, COL4A1 and COL4A2, TBR1, KIF5C, KIF2A and FOXG1. The second part of my PhD program was aimed at identifying the genetic basis of AIC in an international cohort of 19 patients. After excluding a skewed X chromosome inactivation and the presence of chromosomal rearrangements, I performed WES in trios. The analysis of the data from WES did not allow me to identify any recurrent variants. I therefore tested a new approach combining Whole Genome Sequencing (WGS) and RNA-Sequencing (RNA-Seq) on fibroblast cells. I identified a number of deregulated transcripts implicated in brain and eye development. I compared the results of this analysis with the WGS analysis in order to find variants in these candidate genes. In conclusion, these studies have improved the knowledge of the molecular basis of complex MCD, such as TLE1 in postnatal microcephaly, and revealed the pathogenic mechanisms such as WDR81 in cell cycle progression and EPG5 in endosomes and autophagy. My work has also generated a collection of NGS data (WES, WGS and RNA-Seq) that will be shared in an international consortium to develop new analytical strategies, in particular for the non-coding DNA regions. This novel strategy provides opportunities to improve understanding of the cellular mechanisms involved in brain and eye development
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Lukáš, Martin. "Využití hiporehabilitace u dětí s poruchami chování a ADHD." Master's thesis, 2014. http://www.nusl.cz/ntk/nusl-335076.

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Therapeutic horseback riding is one of the most popular and the most used form of animal based therapy. The term "therapeutic horseback riding" itself is very broad and it consists of four basic branches. The most used, yet least described, is the branch that has been called "pedagogical-psychological horseback riding therapy" since 2011. This branch is further divided into two sub-branches, Equine assisted psychotherapy and Horse-based activities. These two sub-branches overlap each other and are usually conducted together. With school-age children they are mostly used in cases of behavioral disorders and ADHD. The goal of the thesis is to evaluate the effectiveness of the methods with children diagnosed as such. The research was realized in "Stredisko hyporehabilitace Pirueta" which operates within the Mental Hospital in Havlickuv Brod. The method of the research was observation of five subjects within the one complete therapy unit. At the beginning and at the end of the unit the tutors of the subjects were given observational scale, which served as supplementary technique for diagnostics. Data obtained from own observation and the data gained from observational scaled matched each other approximately in 90%. During the therapy unit all the subjects improved in areas of self-confidence, self-...
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VETCHÁ, Veronika. "ADHD v MŠ - jak se rodí diagnóza." Master's thesis, 2010. http://www.nusl.cz/ntk/nusl-53426.

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The thesis is focuced on the origin of the syndrom ADHD and its influence and impact on ensuant life of the pre-schoolers in kindergarten. The main terms connected with ADHD are explained at the beginning of the work, containig terminology, partial symptoms such as hyperaktivity, impulsivity and attention deficit, atc. Furthermore, there are mentioned its possible etiologies, various ways of rectification and treatment and convenient and professional approaches to hyperactive children. The describtion of the the procedures and ways of the revealing and later professional assignment of the diagnosis of this disorder is also mentioned in these thesis. The practical part of the thesis is focused just on the proces and proceeding to the professional assignment of ADHD and its causes and circumstances for this decision , who did that and how the diagnosis influenced life of that child and his/her surroundings.
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EISERTOVÁ, Jaroslava. "Canisterapie jako podpůrná terapeutická metoda u dětí s hyperkinetickým syndromem a dětskou mozkovou obrnou." Doctoral thesis, 2009. http://www.nusl.cz/ntk/nusl-52476.

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Books on the topic "Hyperkinetický syndrom"

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Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Hyperkinetic movement disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0010.

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This chapter discusses the clinical features and evidence-based pharmacological management of tremor, dystonia (focal, generalized, and dopa-responsive dystonia), tics and Tourette’s syndrome, chorea (Huntington’s disease (HD) and Sydenham’s chorea), ballism and athetosis, myoclonus, and restless legs syndrome (RLS).
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Oades, Robert D. Attention Deficit/Hyperactivity Disorder (Ad/hd) And the Hyperkinetic Syndrome (Hks): Current Ideas And Ways Forward. Nova Science Publishers, 2006.

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D, Oades Robert, ed. Attention-deficit/hyperactivity disorder (AD/HD) and the hyperkinetic syndrome (HKS): Current ideas and ways forward. New York: Nova Science Publishers, 2006.

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Fox, Susan H. Delayed and Often Persistent. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0021.

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Tardive syndromes are drug-induced hyperkinetic movement disorders that occur as a consequence of dopamine D2 receptor antagonism/blockade. There are several types, including classical tardive dyskinesia, tardive dystonia, tardive tics, tardive myoclonus, and tardive tremor, and it is important to the management of these disorders that the type of movement disorder induced is identified. Tardive syndromes can occur with all antipsychotic drugs, including so-called atypical drugs. Patients taking these drugs should be evaluated frequently for side effects. Evaluating the nature of the movement (i.e., chorea or dystonia) is important because treatment options can differ according to the type of dyskinesia present.
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Walsh, Richard A. “It Has to Be Functional!”. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0026.

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The paroxysmal dyskinesias are a heterogeneous group of rare movement disorders, characterized by the abrupt onset of involuntary hyperkinetic movements with or without trigger factors and of variable duration. Interictal periods are marked by relative normality, although there is evidence for an association between some genotypes and migraine, episodic ataxia, and seizure disorders. Three genes have been identified that are associated with the three most common syndromes; however, these do not account for some cases with an otherwise typical history. The clinical phenotype continues to evolve with increasing characterization of genetically proven cases. Paroxysmal kinesigenic dyskinesia responds well to carbamazepine therapy.
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Book chapters on the topic "Hyperkinetický syndrom"

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Hyperkinetic Syndrome." In Encyclopedia of Molecular Mechanisms of Disease, 903. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7558.

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Bloch, Michael H., Michael H. Bloch, Mark A. Geyer, David C. S. Roberts, Eileen M. Joyce, Jonathan P. Roiser, John H. Halpern, et al. "Hyperkinetic Child Syndrome." In Encyclopedia of Psychopharmacology, 606. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_3308.

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Hamilton-Stubbs, Pamela, and Arthur S. Walters. "Restless Legs Syndrome." In Hyperkinetic Movement Disorders, 311–30. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444346183.ch20.

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Jankovic, Joseph. "Tics and Tourette Syndrome." In Hyperkinetic Movement Disorders, 188–99. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444346183.ch12.

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Roze, Emmanuel, and Emmanuelle Apartis. "Inherited Myoclonus Syndromes." In Hyperkinetic Movement Disorders, 207–20. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444346183.ch14.

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Zauber, S. Elizabeth, and Christopher G. Goetz. "Dyskinesias in Parkinsonian Syndromes." In Hyperkinetic Movement Disorders, 297–310. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444346183.ch19.

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Bernard, Geneviève, Paul Lespérance, Francois Richer, and Sylvain Chouinard. "Tourette Syndrome and the Spectrum of Neurodevelopmental Tic Disorders." In Hyperkinetic Movement Disorders, 85–111. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60327-120-2_4.

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, Ben J. Harrison, Christos Pantelis, Christof Baltes, Thomas Mueggler, et al. "Model Organisms of Hyperkinetic Syndrome." In Encyclopedia of Psychopharmacology, 790. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_3403.

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Prastaro, Marco, and Floriano Girotti. "Extrapyramidal Diseases: Hyperkinetic Movement Disorders – Tics and Tourette Syndrome." In Prognosis of Neurological Diseases, 381–83. Milano: Springer Milan, 2015. http://dx.doi.org/10.1007/978-88-470-5755-5_31.

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"Hyperkinetic Syndrome." In Encyclopedia of Child Behavior and Development, 770. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_4225.

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