Academic literature on the topic 'Hyperlipidemia – Epidemiology'

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Journal articles on the topic "Hyperlipidemia – Epidemiology"

1

Cote, David J., Bernard A. Rosner, Stephanie A. Smith-Warner, Kathleen M. Egan, and Meir J. Stampfer. "Statin use, hyperlipidemia, and risk of glioma." European Journal of Epidemiology 34, no. 11 (2019): 997–1011. http://dx.doi.org/10.1007/s10654-019-00565-8.

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2

Gotto, A. M., and C. M. Ballantyne. "Dietary Treatment of Hyperlipidemia." European Journal of Cardiovascular Prevention & Rehabilitation 1, no. 4 (1994): 283–85. http://dx.doi.org/10.1177/174182679400100401.

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3

Wang, Dongming, Jianghao Chen, Yun Zhou, et al. "Association between sleep duration, sleep quality and hyperlipidemia in middle-aged and older Chinese: The Dongfeng–Tongji Cohort Study." European Journal of Preventive Cardiology 26, no. 12 (2019): 1288–97. http://dx.doi.org/10.1177/2047487319843068.

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Aims This study aimed to evaluate the relationship between sleep duration, sleep quality and hyperlipidemia in middle-aged and older Chinese. Methods We included 20,712 individuals at baseline from September 2008 to June 2010, and they were followed-up until October 2013. Hyperlipidemia was defined according to the Chinese guidelines on the prevention and treatment of dyslipidemia in adults. Sleep duration was self-reported and sleep quality was evaluated with a questionnaire that was designed according to the Pittsburgh Sleep Quality Index. Logistic regression and Cox proportional hazard models were conducted to explore the associations. Results In the cross-sectional analyses, longer sleep duration (≥10 h) was significantly associated with higher prevalence of hyperlipidemia (odds ratio (OR) = 1.17, 95% confidence interval (CI) = 1.02–1.35) after adjusting for potential confounders. The ORs of hyperlipidemia were significantly elevated among participants with impaired sleep quality (OR = 1.14, 95% CI = 1.08–1.22) and poor sleep quality (OR = 1.20, 95% CI = 1.08–1.34) when compared to those with good sleep quality. In the longitudinal analyses, compared to participants with a sleep duration of 7–<8 h, those with a sleep duration of 9–<10 h (hazard ratio (HR) = 1.19, 95% CI = 1.04–1.35) and ≥10 h (HR = 1.27, 95% CI = 1.02–1.58) showed significantly higher risk of hyperlipidemia after adjusting for potential confounders. However, no statistically significant association was found between impaired or poor sleep quality and hyperlipidemia. Conclusions Longer sleep duration was significantly associated with higher risk of hyperlipidemia. Impaired or poor sleep quality were associated with elevated prevalence of hyperlipidemia, but not with the incidence of hyperlipidemia.
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Spracklen, Cassandra N., Caitlin J. Smith, Audrey F. Saftlas, Jennifer G. Robinson, and Kelli K. Ryckman. "Maternal Hyperlipidemia and the Risk of Preeclampsia: a Meta-Analysis." American Journal of Epidemiology 180, no. 4 (2014): 346–58. http://dx.doi.org/10.1093/aje/kwu145.

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5

Rabeya, Rokshana, Mohammad Hayatun Nabi, Ariful Bari Chowdhury, Sanjana Zaman, Mohammad Niaz Morshed Khan, and Mohammad Delwer Hossain Hawlader. "Epidemiology of Dyslipidemia Among Adult Population of Bangladesh." Romanian Journal of Diabetes Nutrition and Metabolic Diseases 26, no. 2 (2019): 99–106. http://dx.doi.org/10.2478/rjdnmd-2019-0011.

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Abstract Background and aims: evatedEl level serum of lipids stimulate atherosclerosis, which is the risk factor for stroke, peripheral vascular taeohrrratrrocvtra disease. The aim of this study was to explore the pattern and associated factors of dyslipidemia among Bangladeshi adult population. Material and methods: A descriptive cross-sectional study was conducted at the outpatient department (OPD) of four Medical College Hospitals, Bangladesh. 200 adults aged 20 to 65 years diagnosed case of dyslipidemia were randomly selected. Fasting CHO, HDL, LDL and TG were measured. According to the criteria of the National Cholesterol Education Program-Adult Treatment Panel III (NCEP-ATP III), dyslipidemia was classified into (a) Hyper-lipidemia: TC>200 mg/dl, TG>150 mg/dl, (b) Hyper cholesterolemia: TC>200 mg/dl, (c) Hyper-triglyceridemia: TG>150 mg/dl, and (d) Atherogenic-dyslipidemia: TG>150 mg/dl, LDLC>165 mg/dl. Results: Study found 46% hyperlipidemia, 37% atherogenic dyslipidemia, 13.5% hypercholesterolemia and only 3.5% hypertriglyceridemia. BMI, FBS and HDL-C were significantly higher among female compare to male (p=<0.01, <0.01 and 0.04 respectively). TC and TG were significantly higher among higher calorie intake group in compare to normal intake group (p=0.04). Conclusions: Results of this study concluded that hyperlipidemia and atherogenic dyslipidemia are common and female dyslipidemic patients are susceptible to develop higher BMI, FBS, and HDL-C.
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6

Albright, Karen C., Amelia K. Boehme, Rikki M. Tanner, et al. "Addressing Stroke Risk Factors in Black and White Americans: Findings from the National Health and Nutrition Examination Survey, 2009-2010." Ethnicity & Disease 26, no. 1 (2016): 9. http://dx.doi.org/10.18865/ed.26.1.9.

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<p><strong>Objectives: </strong> Recurrent stroke affects 5%-15% of stroke survivors, is higher among Blacks, and preventable with secondary stroke prevention medications. Our study aimed to examine racial differences in risk factors being addressed (defined as either on active treatment or within guideline levels) among stroke survivors and those at risk for stroke.</p><p><strong>Methods: </strong> A cross-sectional study using NHANES 2009-2010 standardized interviews of Whites and Blacks aged ≥18 years. Risk factors were defined as being addressed if: 1) for hypertension, SBP <140, DBP <90 (SBP<130, DBP<80 for diabetics) or using BP-lowering medications; 2) for current smoking, using cessation medications; and 3) for hyperlipidemia, LDL<100 (LDL<70 for stroke survivors) or using lipid-lowering medications. Participants were stratified by stroke history. Prevalence of addressed risk factors was compared by race.</p><p><strong>Results: </strong>Among 4005 participants (mean age 48, 52% women, 15% Black), 4% reported a history of stroke. Among stroke survivors, there were no statistically significant differences in Blacks and Whites having their hypertension or hyperlipidemia addressed. Among stroke naïve participants, the prevalence of addressed hypertension (P<.01) and hyperlipidemia (P<.01) was lower in Blacks compared with Whites. </p><strong>Conclusions: </strong> We found that addressed hypertension and hyperlipidemia in stroke naïve participants were significantly lower in Blacks than Whites. Our observations call attention to areas that require further investigation, such as why black Americans may not be receiving evidence-based pharmacologic therapy for hypertension and hyperlipidemia or why Black Americans are not at goal blood pressure or goal LDL. A better understanding of this information is critical to preventing stroke and other vascular diseases. <em>Ethn Dis</em>. 2016;26(1):9-16; doi:10.18865/ed.26.1.9
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7

Wallace, Robert B., and Patricia L. Colsher. "Blood lipid distributions in older persons prevalence and correlates of hyperlipidemia." Annals of Epidemiology 2, no. 1-2 (1992): 15–21. http://dx.doi.org/10.1016/1047-2797(92)90032-l.

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8

Crouse, John R., Jacqueline E. Ryu, and Frederic R. Kahl. "Changes in Pharmacologic Treatment of Hyperlipidemia." American Journal of Preventive Medicine 5, no. 2 (1989): 90–94. http://dx.doi.org/10.1016/s0749-3797(18)31110-3.

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9

Jalali, Farzad Seyyed, K. Hajian, and M. R. Niaki. "P-276 Can Linseed Correct Hyperlipidemia?" CVD Prevention and Control 4 (May 2009): S130. http://dx.doi.org/10.1016/s1875-4570(09)60468-x.

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10

Papadimitriou, L., J. Skoumas, C. Pitsavos, et al. "Abstract: P361 EPIDEMIOLOGY OF FAMILIAL COMBINED HYPERLIPIDEMIA IN A GREEK POPULATION: 10 YEAR FOLLOW UP." Atherosclerosis Supplements 10, no. 2 (2009): e671. http://dx.doi.org/10.1016/s1567-5688(09)70656-7.

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