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Relevant bibliographies by topics / Hypophonia

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Academic literature on the topic 'Hypophonia'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Hypophonia"

1

Sekar, Murali Krishnan, Jon Arcelus, and Robert L. Palmer. "Micrographia and hypophonia in anorexia nervosa." International Journal of Eating Disorders 43, no. 8 (November 2, 2009): 762–65. http://dx.doi.org/10.1002/eat.20768.

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Kim, Soo H., James J. Kearney, and Joseph P. Atkins. "Percutaneous Laryngeal Collagen Augmentation for Treatment of Parkinsonian Hypophonia." Otolaryngology–Head and Neck Surgery 126, no. 6 (June 2002): 653–56. http://dx.doi.org/10.1067/mhn.2002.125756.

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OBJECTIVE: Our goal was to determine whether percutaneous laryngeal collagen augmentation improves hypophonia in parkinsonian patients. STUDY DESIGN AND SETTING: A retrospective review of 18 patients was performed. Patients and/or caretakers were called on the telephone and asked about their response to the procedure, if any. Videostroboscopic examinations for all patients were reviewed. RESULTS: Of 18 patients, 11 (61%) reported improvement in their hypophonia for a period of at least 2 months. Five of 7 patients without improvement were relatively aphonic both before and after the procedure. Five of 7 patients without improvement had severe dysphagia, which in 3 necessitated gastrostomy tube placement. Four of 7 patients without improvement were not ambulatory at the time of the procedure. CONCLUSIONS: Percutaneous laryngeal collagen augmentation is an effective treatment for parkinsonian hypophonia in a majority of patients. Patients with advanced neurologic disease with aphonia, difficulty with speech initiation, dysphagia, or ambulatory difficulty are less likely to respond to this procedure and should be so informed.

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Prabhakar, AppaswamyT, AtifI Shaikh, Ashish Vijayaraghavan, and Gideon Rynjah. "Thalamic hypophonia and the neural control of phonation." Neurology India 66, no. 6 (2018): 1815. http://dx.doi.org/10.4103/0028-3886.246247.

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Berke, Gerald S., Bruce Gerratt, Jody Kreiman, and Katherine Jackson. "Treatment of Parkinson Hypophonia With Percutaneous Collagen Augmentation." Laryngoscope 109, no. 8 (August 1999): 1295–99. http://dx.doi.org/10.1097/00005537-199908000-00020.

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Yoon, Ji Hye, Yisuh Ahn, Sook Hui Kim, Juhee Chin, Seongbeom Park, and Duk L. Na. "Neuroanatomical Correlates of Hypophonia in Subcortical Vascular Cognitive Impairment." European Neurology 78, no. 1-2 (2017): 93–104. http://dx.doi.org/10.1159/000479023.

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Rodríguez-López, Claudia, Blanca Ayuso García, and Sara Moreno García. "Hypophonia as a sign of thalamus lesion: a case report." International Journal of Neuroscience 128, no. 1 (August 6, 2017): 100–101. http://dx.doi.org/10.1080/00207454.2017.1358716.

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Murray, B. J., R. Llinas, L. R. Caplan, T. Scammell, and A. Pascual-Leone. "Cerebral deep venous thrombosis presenting as acute micrographia and hypophonia." Neurology 54, no. 3 (February 8, 2000): 751. http://dx.doi.org/10.1212/wnl.54.3.751.

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Zenon, Alexandre, and Etienne Olivier. "Contribution of the basal ganglia to spoken language: Is speech production like the other motor skills?" Behavioral and Brain Sciences 37, no. 6 (December 2014): 576. http://dx.doi.org/10.1017/s0140525x13004238.

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AbstractTwo of the roles assigned to the basal ganglia in spoken language parallel very well their contribution to motor behaviour: (1) their role in sequence processing, resulting in syntax deficits, and (2) their role in movement “vigor,” leading to “hypokinetic dysarthria” or “hypophonia.” This is an additional example of how the motor system has served the emergence of high-level cognitive functions, such as language.

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Knowles, Thea, Scott G. Adams, Allyson Page, Daryn Cushnie-Sparrow, and Mandar Jog. "A Comparison of Speech Amplification and Personal Communication Devices for Hypophonia." Journal of Speech, Language, and Hearing Research 63, no. 8 (August 10, 2020): 2695–712. http://dx.doi.org/10.1044/2020_jslhr-20-00085.

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Purpose This study compared the performance of three amplification devices hypothesized to improve speech communication in individuals with hypophonia (HP), as well as to identify individuals' device preferences. Method Twenty-two individuals with HP and their primary communication partners participated in a cross-over design study comparing three different speech amplification devices: a wired portable amplifier (Device A), a wireless stationary amplifier (Device B), and a one-way personal communication system (Device C). Participants attended one laboratory visit followed by 1-week trial periods with each device. At the first visit, HP participants completed speech tasks with and without the devices, in quiet and in noise. Following the in-laboratory test period, participants trialed each device at home for approximately 1 week per device. Following completion of the study, participants indicated whether or not they would like to continue using a device. Results Overall, in the presence of noise, all three devices demonstrated significant improvements in speech-to-noise levels and speech intelligibility compared to no device. A clear device hierarchy emerged such that the personal communication device (Device C) was associated with significantly better speech outcomes compared to the other two devices. The majority of participants elected to continue using a device at the completion of the study. Device preferences, however, did not clearly reflect the objective device hierarchy that was found for the objective speech measures. Each of the three devices was selected as a preferred device by at least three participants at the completion of the study. Conclusion Results from this study demonstrated clear differences in device performance in three distinct forms of amplification devices for individuals with HP. Findings suggest that amplification device use may be beneficial for this clinical population and underscore the potential to improve device availability and device selection criteria in future research. Supplemental Material https://doi.org/10.23641/asha.12735875

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Baker, K. K., L. O. Ramig, E. S. Luschei, and M. E. Smith. "Thyroarytenoid muscle activity associated with hypophonia in Parkinson disease and aging." Neurology 51, no. 6 (December 1, 1998): 1592–98. http://dx.doi.org/10.1212/wnl.51.6.1592.

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Dissertations / Theses on the topic "Hypophonia"

1

Watkins, Lynn Marie. "How individuals with Parkinson's disease modify their speech in a repetition for clarification." Diss., CLICK HERE for online access, 2005. http://contentdm.lib.byu.edu/ETD/image/etd997.pdf.

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Book chapters on the topic "Hypophonia"

1

Wang, E. Q. "Hypophonia." In Encyclopedia of Movement Disorders, 57–59. Elsevier, 2010. http://dx.doi.org/10.1016/b978-0-12-374105-9.00333-6.

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Lang, Anthony E. "Severe Hypophonia and Parkinsonism With Hepatitis C and Elevated Mn: Lessons From a Drug Abuser." In Movement Disorders. New York, NY: Springer Publishing Company, 2012. http://dx.doi.org/10.1891/9781617050589.0014.

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Mehta, Gautam, and Bilal Iqbal. "Central Nervous System." In Clinical Medicine for the MRCP PACES. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780199542550.003.0011.

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As with all neurological patients, you will be more likely to pick up the diagnosis if you take a step back and look at the whole patient. Take some time to assess their facial expressions, speech, tremor, and posture. A common instruction at this station, with the patient seated on a chair is ‘Look at this patient, and examine as appropriate’. Candidates are often baffled, when given this instruction. Often the patients with Parkinson’s disease are given specific instructions to interlock the fingers of both hands, or place hands flat on their lap to mask the tremor. Picking up an expressionless face and low volume monotonous speech from the outset will provide useful clues to the diagnosis. If you are not sure at this stage, proceed to examining the gait. Once you are certain, that this is Parkinson’s disease, you may proceed to demonstrate the other features. 1. Patients with Parkinson’s disease have characteristic expressionless facies (hypomimia), often described as ‘mask-like’. This is a manifestation of bradykinesia. There is a reduced blink rate. The glabellar tap (Myerson’s sign) is an unreliable sign and is not recommended in the examination. This involves tapping the patient’s forehead repeatedly. Normal subjects will stop blinking, but in Parkinson’s disease, the patient will continue to blink. The patient may be drooling saliva (resulting from dysphagia and sialorrhoea-due to autonomic dysfunction) 2. Patients may have soft speech (hypophonia). This is also a manifestation of bradykinesia, and characteristically, the speech is low-volume, monotonous and tremulous (appears slurred). 3. Blepharoclonus is tremor of the eyelids. This will only be demonstrated if the eyes are gently closed, as opposed to tightly closing the eyes. 4. The classic tremor is present at rest and asymmetrical (more marked on one side). It is classically described as being 4–6Hz and is the initial symptom in 60% of cases, although 20% of patients never have a tremor. The tremor may appear as a ‘pill-rolling’ motion of the hand or a simple oscillation of the hand or arm. It is easier to spot a tremor if you ask the patient to rest their arms in their lap in the semi-prone position.

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