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Journal articles on the topic 'Hypoproteinemia'

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1

BING, JENS. "Hypoproteinemia." Acta Medica Scandinavica 126, no. 4-5 (2009): 273–85. http://dx.doi.org/10.1111/j.0954-6820.1946.tb19019.x.

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2

Cummings, J. J., D. P. Carlton, F. R. Poulain, J. U. Raj, and R. D. Bland. "Hypoproteinemia slows lung liquid clearance in young lambs." Journal of Applied Physiology 74, no. 1 (1993): 153–60. http://dx.doi.org/10.1152/jappl.1993.74.1.153.

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To determine whether hypoproteinemia slows the rate at which liquid is cleared from the lung lumen, we studied 36 lambs, 18 of which underwent repeated plasmapheresis, reducing plasma protein concentration by 37% and plasma protein osmotic pressure by 39%. We killed 29 lambs (14 hypoproteinemic and 15 normoproteinemic) and removed their lungs 1, 2, or 6 h after intratracheal instillation of isotonic saline (6 ml/kg body wt). We measured extravascular lung water and determined the percentage of tracheally instilled liquid that was cleared from the lungs by comparison with control lambs that did
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3

Valenzuela, G. J., C. W. Hewitt, G. C. Kramer, Y. Do, and W. A. Hseuh. "Effects of sustained lymph drainage on cardiovascular function and thoracic duct lymph in sheep." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 256, no. 4 (1989): R867—R874. http://dx.doi.org/10.1152/ajpregu.1989.256.4.r867.

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We studied the effect of lowering the plasma protein concentration on the cardiovascular function and thoracic duct lymph in awake adult sheep. Hypoproteinemia was induced in seven nonpregnant, splenectomized sheep by drainage of the thoracic duct lymph over a 5-day period. The plasma protein went from a mean of 6.4 +/- 0.2 (SE) to 4.9 +/- 0.2 g/dl on day 5, and the lymph-to-plasma protein concentration ratio decreased from 0.74 +/- 0.01 on day 1 to 0.48 +/- 0.04 on day 5. The percentage composition of the protein fractions in plasma and lymph remained unchanged. Lymph flow was 1.79 +/- 0.37 a
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4

Parfenov, A. I., and L. M. Krums. "Protein-losing enteropathy." Terapevticheskii arkhiv 89, no. 2 (2017): 4–9. http://dx.doi.org/10.17116/terarkh20178924-9.

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Protein-losing enteropathy (PLE) is a rare complication of intestinal diseases. Its main manifestation is hypoproteinemic edema. The diagnosis of PLE is based on the verification of protein loss into the intestinal lumen, by determining fecal α1-antitrypsin concentration and clearance. The localization of the affected colonic segment is clarified using radiologic and endoscopic techniques. The mainstay of treatment for PLE is a fat-free diet enriched with medium-chain triglycerides. Surgical resection of the affected segment of the colon may be the treatment of choice for severe hypoproteinemi
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5

Parker, R. E., N. E. Wickersham, R. J. Roselli, T. R. Harris, and K. L. Brigham. "Effects of hypoproteinemia on lung microvascular protein sieving and lung lymph flow." Journal of Applied Physiology 60, no. 4 (1986): 1293–99. http://dx.doi.org/10.1152/jappl.1986.60.4.1293.

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Experiments were conducted on five chronically instrumented unanesthetized sheep to determine the effects of sustained hypoproteinemia on lung fluid balance. Plasma total protein concentration was decreased from a control value of 6.17 +/- 0.019 to 3.97 +/- 0.17 g/dl (mean +/- SE) by acute plasmapheresis and maintained at this level by chronic thoracic lymph duct drainage. We measured pulmonary arterial pressure, left atrial pressure, aortic pressure, central venous pressure, cardiac output, oncotic pressures of both plasma and lung lymph, lung lymph flow rate, and lung lymph-to-plasma ratio o
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6

BRØCHNER-MORTENSEN, KNUD. "Hypoproteinemia in Chronic Pemphigus." Acta Medica Scandinavica 97, no. 3-4 (2009): 329–34. http://dx.doi.org/10.1111/j.0954-6820.1938.tb09978.x.

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7

Tuglak, Nadeem, Sunil Goenka, Kamlesh Sharma, and Nitin Pandey. "Incidence of Anemia and Serum Protein Deficiencies in Patients with Acute Traumatic Spinal Cord Injury." International Journal of Pharmaceutical and Clinical Research 15, no. 5 (2023): 1071–75. https://doi.org/10.5281/zenodo.12587689.

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<strong>Introduction:&nbsp;</strong>In the acute phase of traumatic Spinal Cord Injury, many patients suffer from anemia, hypoproteinemia and hypoalbuminemia which can contribute to easy fatiguability and an overall hindrance to the rehabilitation effort. This study was conducted to further investigate the incidence of anemia, hypoproteinemia and hypoalbuminemia in patients with acute traumatic spinal cord injury (SCI).&nbsp;<strong>Methods:</strong>&nbsp;This prospective study was carried out at department of physical medicine and rehabilitation, SMS Medical College, Jaipur. Adults with acute
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8

Brinson, Robert, and D. Neil Granger. "Hypoproteinemia-induced mucosal albumin leakage." Digestive Diseases and Sciences 34, no. 1 (1989): 97–102. http://dx.doi.org/10.1007/bf01536161.

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9

Xu, Yu, and Yao-Feng Zhu. "Nursing case of necrotizing fasciitis in a patient with hypoproteinemia after spinal tumor resection." Frontiers of Nursing 12, no. 2 (2025): 279–86. https://doi.org/10.2478/fon-2025-0030.

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Abstract Objective To summarize the nursing experience of a patient with necrotizing fasciitis (NF) secondary to hypoproteinemia after spinal tumor resection. Methods We analyzed the clinical data of a patient with hypoproteinemia and described in detail his symptoms, examination results, and diagnosis. We explored the evaluation method of hypoproteinemia in orthopedic patients and its relationship with adverse postoperative outcomes, including the risk of reoperation and rehospitalization, the impact on postoperative wound healing, and the increased probability of wound infection. We also int
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10

Wilson, Robert C., and Nancy K. Green. "Pharmacokinetics of minocycline hydrochloride in clinically normal and hypoproteinemic sheep." American Journal of Veterinary Research 47, no. 3 (1986): 650–52. https://doi.org/10.2460/ajvr.1986.47.03.650.

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SUMMARY Eight adult sheep were given 2.2 mg of minocycline hydrochloride/kg of body weight iv before and after blood was collected to induce hypoproteinemia. The blood collection produced a significant (P &lt; 0.01) reduction in pcv, hemoglobin, and total serum protein values. Pharmacokinetic evaluation of the serum drug concentration vs time data was performed, using a noncompartmental model based on statistical moment theory. Pharmacokinetic values obtained from the sheep with normal serum protein were 5.94 ± 1.78 ml/kg/min for clearance, 1.32 ± 0.16 L/kg for the steady-state volume of distr
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11

Wen, Jianxia, Xing Chen, Shizhang Wei, Xiao Ma, and Yanling Zhao. "Research Progress and Treatment Status of Liver Cirrhosis with Hypoproteinemia." Evidence-Based Complementary and Alternative Medicine 2022 (February 24, 2022): 1–8. http://dx.doi.org/10.1155/2022/2245491.

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Liver cirrhosis is the 14th leading cause of death in adults worldwide. The liver is an important organ for the metabolism of sugar, protein, and fat. Liver cirrhosis with hypoproteinemia (LCH) can lead to metabolic disorders of the nutrients such as sugar, protein, and fat, as well as insufficient protein intake, digestion and absorption disorders, and continuous leakage of plasma protein into the abdominal cavity. Severe hypoproteinemia leads to a poor prognosis in patients. For every 10 g/L decrease in peripheral blood albumin, the risk of secondary liver disease complications will increase
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12

Faris, Shahi, and Muhammad Hussain Safi. "Study of Risk Factors in Patients with Post Lapartomy Wound Dehisence." Journal of Health and Rehabilitation Research 4, no. 2 (2024): 1161–65. http://dx.doi.org/10.61919/jhrr.v4i2.1029.

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Background: Post-laparotomy wound dehiscence is a significant complication in abdominal surgeries, characterized by the partial or complete separation of the surgical wound layers. This complication can lead to severe morbidity, extended hospital stays, and increased healthcare costs. Objective: To assess the risk factors associated with wound dehiscence in patients post-laparotomy. Methods: A cross-sectional study was conducted at the Department of Surgery from January 25, 2024, to April 25, 2024. A total of eighty patients who developed wound dehiscence following emergency or elective laparo
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13

Mu??iz, Antonio E., Sam Bartle, and Robin Foster. "Edema, Anemia, Hypoproteinemia, and Acrodermatitis Enteropathica." Pediatric Emergency Care 20, no. 2 (2004): 112–14. http://dx.doi.org/10.1097/01.pec.0000113881.10140.50.

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14

Nakasato, Shigeki, Satoru Okamura, Keigo Kudo, and Yasunori Takeda. "Gigantic ameloblastoma associated with secondary hypoproteinemia." Journal of Oral and Maxillofacial Surgery 49, no. 7 (1991): 764–67. http://dx.doi.org/10.1016/s0278-2391(10)80246-0.

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15

Novembre, E., G. Leo, A. Cianferoni, R. Bernardini, N. Pucci, and A. Vierucci. "Severe hypoproteinemia in infant with AD." Allergy 58, no. 1 (2003): 88–89. http://dx.doi.org/10.1034/j.1398-9995.2003.23710_6.x.

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16

Tan, Yan, Wei Xiang, Yi Chen, Jing Huang, and Dong Sun. "Effect of hypoproteinemia on mortality of elderly male patients with chronic heart failure." Medicine 103, no. 5 (2024): e37078. http://dx.doi.org/10.1097/md.0000000000037078.

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To explore the effect of hypoproteinemia on cardiac function and prognosis in elderly male patients with chronic heart failure. Among the patients with chronic heart failure hospitalized in the General Hospital of Southern Theater Command from December 2014 to December 2015, 100 elderly male patients with chronic heart failure were selected. The patients were divided into 2 groups based on their serum albumin (ALB) levels: 53 cases in the normal group (ALB ≥ 35 g/L) and 47 cases in the hypoproteinemia group (ALB &lt; 35 g/L). Using the method of prospective study, under the condition of routin
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17

Tan, Xiuwei, Yanlan Wu, Fengxin Li, et al. "Development and validation of a prediction model for hypoproteinemia after traumatic spinal cord injury: A multicenter retrospective clinical study." Medicine 103, no. 25 (2024): e38081. http://dx.doi.org/10.1097/md.0000000000038081.

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A multicenter retrospective analysis of conventionally collected data. To identify the potential causes of hypoproteinemia after traumatic spinal cord injury (TSCI) and provide a diagnostic model for predicting an individual likelihood of developing hypoproteinemia. Hypoproteinemia is a complication of spinal cord injury (SCI), an independent risk factor for respiratory failure in elderly patients with SCI, and a predictor of outcomes in patients with cervical SCI. Few nomogram-based studies have used clinical indicators to predict the likelihood of hypoproteinemia following TSCI. This multice
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18

Katayama, Kan, Ryosuke Saiki, and Kaoru Dohi. "Special Issue “Nephrotic Syndrome: Pathomechanism, Diagnostics, and Novel Treatment Options”." Biomedicines 12, no. 12 (2024): 2862. https://doi.org/10.3390/biomedicines12122862.

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19

Chen, S., G. Lalazar, O. Barak, T. Adar, V. Doviner, and M. Mizrahi. "Protein-Loosing Entropathy Induced by Unique Combination of CMV and HP in an Immunocompetent Patient." Case Reports in Medicine 2012 (2012): 1–4. http://dx.doi.org/10.1155/2012/361892.

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Protein-losing gastroenteropathies are characterized by an excessive loss of serum proteins into the gastrointestinal tract, resulting in hypoproteinemia (detected as hypoalbuminemia), edema, and, in some cases, pleural and pericardial effusions. Protein-losing gastroenteropathies can be caused by a diverse group of disorders and should be suspected in a patient with hypoproteinemia in whom other causes, such as malnutrition, proteinuria, and impaired liver protein synthesis, have been excluded. In this paper, we present a case of protein-losing enteropathy in a 22-year-old immunocompetent mal
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20

Каленчиц, Т. И., С. Л. Кабак, М. В. Кузьмич, and Е. К. Абражевич. "Polyserositis Caused by Hypoproteinemia: a Case Report." Рецепт 27, no. 3 (2024): 475–80. http://dx.doi.org/10.34883/pi.2024.27.3.013.

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В статье описан случай двустороннего экссудативного плеврита, асцита и гидроперикарда, обусловленных энтеропатией с потерей белка в сочетании с первичным гипотиреозом. Полисерозит не был связан с гипотиреозом, а обусловлен потерей белка, который определялся на уровне 47,2 г/л. Снижение содержания альбумина до уровня 20,9 г/л привело к появлению анасарки в результате выхода жидкости из сосудистого русла. При этом не было выявлено нарушений функции печени и почек, которые могли бы стать причиной снижения содержания белка. Сохранение в пределах нормы фракции выброса левого желудочка и уровень нат
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21

Morton, A. Ross, and Gavril Hercz. "Serum Calcium and Hypoproteinemia in Dialysis Patients." Seminars in Dialysis 5, no. 3 (2007): 242–43. http://dx.doi.org/10.1111/j.1525-139x.1992.tb00486.x.

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22

Osaki, Tokio, Kazuo Ryoke, Terumasa Nagami, Takatsugu Ogawa, and Takeshi Hamada. "Ameloblastoma with hypoproteinemia due to protein leakage." International Journal of Oral Surgery 14, no. 3 (1985): 302–6. http://dx.doi.org/10.1016/s0300-9785(85)80045-4.

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23

Elemen, Levent, Dicle Inanc, Feriha Oz, and Ergun Erdogan. "Segmental dilatation of the ileum accompanying hypoproteinemia." Journal of Pediatric Surgery 43, no. 7 (2008): e15-e18. http://dx.doi.org/10.1016/j.jpedsurg.2008.02.084.

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24

Jo, So Yoon, Chan-Ho Lee, Woo-Jin Jung, Sung-Won Kim, and Yoon-Ha Hwang. "Common features of atopic dermatitis with hypoproteinemia." Korean Journal of Pediatrics 61, no. 11 (2018): 348–54. http://dx.doi.org/10.3345/kjp.2018.06324.

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25

Joles, J. A., H. A. Koomans, W. Kortlandt, P. Boer, and E. J. Dorhout Mees. "Hypoproteinemia and recovery from edema in dogs." American Journal of Physiology-Renal Physiology 254, no. 6 (1988): F887—F894. http://dx.doi.org/10.1152/ajprenal.1988.254.6.f887.

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We studied the effects of hypoproteinemia following 12 days of repeated plasmapheresis and low-protein diet on sodium balance, fluid volumes, and renal hemodynamics in six conscious dogs on 50 mmol sodium intake. Measurements during hypoproteinemia were obtained during a 5-day recovery period starting 20 h after the final plasmapheresis session, with continued low-protein diet. During the plasmapheresis period sodium was retained. Sodium balance became negative on the first recovery day when plasma protein was 29 +/- 1 g/l (control 60 +/- 2 g/l, P less than 0.01), and plasma colloid osmotic pr
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26

Hata, H., M. Ebihara, T. Onitsuka, M. Nakagawa, Y. Kitagawa, and Y. Ota. "Large ameloblastoma of the mandible with hypoproteinemia." International Journal of Oral and Maxillofacial Surgery 37, no. 9 (2008): 866–69. http://dx.doi.org/10.1016/j.ijom.2008.04.001.

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27

Maindad, Gayatri Gawade, and Dadasaheb Maindad. "Hypoproteinemia: An eye of suspicion towards the diagnosis of rapunzel syndrome." International Journal of Clinical Biochemistry and Research 9, no. 2 (2022): 182–84. http://dx.doi.org/10.18231/j.ijcbr.2022.035.

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The Rapunzel syndrome is the usually name given to the trichobezoar related condition in which there is extension of tail of trichobezoar from its origin to the distant location. Trichobezoar is the rare clinical entity found usually in the pediatric population and young females with underlying psychiatric condition. There is huge collection of undigested materials like hairs, threads, etc. leading to various clinical complications if not diagnosed and removed on time. Hypoproteinemia is a rare presentation of complication of this Rapunzel syndrome due to protein losing enteropathy. We encount
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28

Jameshorani, Maryam, Akram Pourshams, Anahita Sadeghi, Hiva Saffar, and Reza Malekzadeh. "Celiac Crisis in a Young Woman: Raising Awareness of a Life-Threatening Condition." Middle East Journal of Digestive Diseases 11, no. 4 (2019): 230–33. http://dx.doi.org/10.15171/mejdd.2019.154.

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Celiac crisis is a rare, acute, and life-threatening presentation of celiac disease. Its clinical presentations consist of severe watery non-bloody diarrhea, electrolyte disturbances (i.e. hypokalemia, hyponatremia, hypomagnesemia, hypocalcemia, and metabolic acidosis), hypoproteinemia, and dehydration. Here we present a 33-year-old woman who referred with profuse diarrhea, weight loss, hemodynamic instability, hypokalemia, hypoproteinemia, ascites, pancytopenia, and iron deficiency anemia. She used herbal medicines for constipation and had severe weakness after her childbirth. The patient was
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29

GOLDEN, MICHAEL H. N., and BARBARA E. GOLDEN. "Zinc Deficiency and Edema." Pediatrics 77, no. 1 (1986): 132–33. http://dx.doi.org/10.1542/peds.77.1.132a.

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To the Editor.— Kumar and Anday1 describe three premature infants presenting with edema and hypoproteinemia—the classical signs of kwashiorkor—between 5 and 9 weeks of age. Such cases are not uncommon in developing countries. Kumar and Anday's patients had low plasma zinc concentrations (43, 37, and 42 µg/dL). On this basis the authors claim that edema and hypoproteinemia is a clinical presentation of zinc deficiency not previously reported. We reported2 a clear association between "nutritional" edema and a low plasma zinc concentration in 1979; our subsequent experience has confirmed that ede
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30

Yoshioka, Katsunobu, Masanori Kishibuchi, and Ko Takada. "Protein losing gastroenteropathy due to Helicobacter pylori infection without giant rugal folds, erosion or polyposis." BMJ Case Reports 12, no. 11 (2019): e230870. http://dx.doi.org/10.1136/bcr-2019-230870.

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An 85-year-old woman was admitted to our hospital because of progressive hypoproteinemia and generalised oedema. Technetium-99m human albumin scintigraphy revealed protein leakage in the gastrointestinal tract. Upper gastrointestinal endoscopy revealed small whitish nodules from the gastric body up to the duodenal bulb. The urease test for Helicobacter pylori infection was positive. We diagnosed her as having protein-losing gastroenteropathy (PLGE) caused by H. pylori infection. The patient’s hypoproteinemia and clinical symptoms promptly resolved after H. pylori eradication. Our results sugge
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31

KUMAR, SAVITRI P., and ENDLA K. ANDAY. "Zinc Deficiency: Cause of Hypoproteinemia or Its Result?" Pediatrics 75, no. 1 (1985): 128–29. http://dx.doi.org/10.1542/peds.75.1.128b.

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In Reply.— We agree with Weizman that quantification of enteric protein loss is best carried out by the use of51 Cr-albumin. Although this test has been used in adults and older children, there is no published study on the use of this method in neonates or low-birth-weight infants. The use of radioactive isotopes and the necessity of obtaining fecal samples without urinary contamination makes this an undesirable test in infants. Although the fecal clearance of α1-antitrypsin overcomes all the drawbacks of the 51Cr-albumin method, there are again no studies to date, on its use in neonates.
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32

WEIZMAN, ZVI. "Zinc Deficiency: Cause of Hypoproteinemia or Its Result?" Pediatrics 75, no. 1 (1985): 128. http://dx.doi.org/10.1542/peds.75.1.128a.

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To the Editor.— I read with great interest the paper by Kumar and Anday.1 They suggest that hypoproteinemia in their patients is the direct result of zinc deficiency, which interferes with protein synthesis. However, in their discussion there is no mention of a different interpretation, namely that zinc deficiency might be the result of protein loss rather than a cause of poor synthesis. Although liver disease and/or urinary protein loss have been ruled out, there are no data regarding a possible protein-losing enteropathy in these infants. Several disorders, such as cow milk intolerance (case
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33

Ito, Sakuya, Chikei Natori, Akiko Nagata, Atsuko Ohara, and Junichi Tsuchiya. "A Case of Filarial Chyluria with Severe Hypoproteinemia." Nihon Naika Gakkai Zasshi 102, no. 12 (2013): 3227–29. http://dx.doi.org/10.2169/naika.102.3227.

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34

BROCH, OLE JACOB. "The Effects of Plasma Transfusions in Nephrogenic Hypoproteinemia." Acta Medica Scandinavica 134, no. 1 (2009): 6–22. http://dx.doi.org/10.1111/j.0954-6820.1949.tb06926.x.

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35

Fustik, S., T. Jakovska, and L. Spirevska. "Hypoproteinemia and anemia in infants with cystic fibrosis." Journal of Cystic Fibrosis 7 (June 2008): S97. http://dx.doi.org/10.1016/s1569-1993(08)60369-0.

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36

Fujii, Tatsuya, Tohru Yorifuji, Takehiko Okuno, Shinya Toyokuni, Shigeru Okada, and Haruki Mikawa. "Congenital myotonic dystrophy with progressive edema and hypoproteinemia." Brain and Development 13, no. 1 (1991): 58–60. http://dx.doi.org/10.1016/s0387-7604(12)80300-7.

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37

Park, Jong Ha, Yonghye Shin, Geun Hwa Park, and Sung Won Kim. "The investigation of hypoproteinemia in pediatric atopic dermatitis." Allergy, Asthma & Respiratory Disease 1, no. 4 (2013): 344. http://dx.doi.org/10.4168/aard.2013.1.4.344.

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38

Serebruany, Victor L., Marie J. Christenson, John Pescetti, and Robert H. McLean. "Hypoproteinemia in the hemolytic-uremic syndrome of childhood." Pediatric Nephrology 7, no. 1 (1993): 72–73. http://dx.doi.org/10.1007/bf00861576.

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39

Manzali, E., F. Vincenzi, A. Fugazza, et al. "Hypoproteinemia in childhood: What should you look for?" Digestive and Liver Disease 45 (September 2013): e290. http://dx.doi.org/10.1016/j.dld.2013.08.197.

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40

Joseph, Nishi Mary, and Sam Christy Mammen. "Stress hyperglycemia after pancreatobiliary surgeries." International Surgery Journal 7, no. 6 (2020): 1768. http://dx.doi.org/10.18203/2349-2902.isj20202380.

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Background: Diabetes mellitus is a common problem and stress hyperglycemia occurring in patients undergoing surgery without history of diabetes mellitus has been shown to have a poorer clinical outcome. Effective glycemic control in the perioperative period results in marked improvement in clinical outcome.Methods: This prospective study was conducted among 100 cases of the pancreatobiliary postoperative patients admitted in General Surgery Department, Government Medical College Kottayam during November 2015 to November 2016. The post-operative blood sugar values, age, sex, BMI, family history
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Fu, Wenjun, Min Li, Hongrong Lin, et al. "Cyclosporine A relieved proteinuria and hypoproteinemia in DGKE nephropathy." Clinica Chimica Acta 518 (July 2021): 78–82. http://dx.doi.org/10.1016/j.cca.2021.02.021.

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42

Deng, Juan, Xiaoxia Chen, Hui Sun, et al. "Hypoproteinemia being a manifestation of immunotherapy-related liver dysfunction." Annals of Translational Medicine 8, no. 14 (2020): 889. http://dx.doi.org/10.21037/atm-20-4980.

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43

Fukue, Mayumi, Toshio Nakahara, and Keisuke Sarai. "Hypoproteinemia Related with Chronic Lithium Therapy in Two Patients." Psychiatry and Clinical Neurosciences 44, no. 1 (1990): 55–58. http://dx.doi.org/10.1111/j.1440-1819.1990.tb00440.x.

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44

YOSHIKAWA, Fumihiro, Kinya HIGUCHI, Motohiro TSUJINO, et al. "Mandibular ameloblastoma complicating severe hypoproteinemia: Report of a case." Japanese Journal of Oral & Maxillofacial Surgery 36, no. 3 (1990): 589–93. http://dx.doi.org/10.5794/jjoms.36.589.

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45

Nomura, Ichiro, Toshio Katsunuma, Morimitsu Tomikawa, et al. "Hypoproteinemia in severe childhood atopic dermatitis: A serious complication." Pediatric Allergy and Immunology 13, no. 4 (2002): 287–94. http://dx.doi.org/10.1034/j.1399-3038.2002.01041.x.

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46

Belamarich, Peter F., Anahi Ortiz, and Richard L. Mones. "Severe Hypoproteinemia and Edema in Association with Varicella Infection." Clinical Pediatrics 30, no. 1 (1991): 56–58. http://dx.doi.org/10.1177/000992289103000110.

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47

Ueyama, Yoshiya, Gohichi Tsukamoto, and Tomohiro Matsumura. "Gigantic ameloblastoma of the mandible complicating hypoproteinemia: case report." Journal of Cranio-Maxillofacial Surgery 23, no. 1 (1995): 47–49. http://dx.doi.org/10.1016/s1010-5182(05)80255-0.

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48

Raff, Hershel, Scott Brock, and James W. Findling. "Cosyntropin-stimulated salivary cortisol in hospitalized patients with hypoproteinemia." Endocrine 34, no. 1-3 (2008): 68–74. http://dx.doi.org/10.1007/s12020-008-9101-y.

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KAWAI, MASAHIKO, TOHRU YORIFUJI, CHUTARO YAMANAKA, et al. "Liver transplantation in a case of hypoproteinemia and coagulopathy." Pediatrics International 40, no. 1 (2007): 96–98. http://dx.doi.org/10.1111/j.1442-200x.1998.tb01414.x.

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AMENOMORI, Masaki, Tomio UMEMOTO, Ryoji KUSHIMA, and Takanori HATTORI. "Spontaneous Remission of Hypertrophic Lymphocytic Gastritis Associated with Hypoproteinemia." Internal Medicine 37, no. 12 (1998): 1019–22. http://dx.doi.org/10.2169/internalmedicine.37.1019.

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