Relevant bibliographies by topics / Hypothalamic amenorrhea

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Academic literature on the topic 'Hypothalamic amenorrhea'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Hypothalamic amenorrhea"

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Warren, Michelle P., and Joanna L. Fried. "HYPOTHALAMIC AMENORRHEA." Endocrinology and Metabolism Clinics of North America 30, no. 3 (September 2001): 611–29. http://dx.doi.org/10.1016/s0889-8529(05)70204-8.

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Keller, Jennifer L., and Kenneth Faber. "Hypothalamic Amenorrhea." Postgraduate Obstetrics & Gynecology 28, no. 21 (November 2008): 1–5. http://dx.doi.org/10.1097/01.pgo.0000337875.34050.5a.

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&NA;. "Hypothalamic Amenorrhea." Postgraduate Obstetrics & Gynecology 28, no. 21 (November 2008): 6. http://dx.doi.org/10.1097/01.pgo.0000337876.41673.71.

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Meczekalski, B., A. Tonetti, P. Monteleone, F. Bernardi, S. Luisi, M. Stomati, M. Luisi, F. Petraglia, and AR Genazzani. "Hypothalamic amenorrhea with normal body weight: ACTH, allopregnanolone and cortisol responses to corticotropin-releasing hormone test." European Journal of Endocrinology 142, no. 3 (March 1, 2000): 280–85. http://dx.doi.org/10.1530/eje.0.1420280.

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OBJECTIVE: Hypothalamic amenorrhea (HA) is a functional disorder caused by disturbances in gonadotropin-releasing hormone (GnRH) pulsatility. The mechanism by which stress alters GnRH release is not well known. Recently, the role of corticotropin-releasing hormone (CRH) and neurosteroids in the pathophysiology of HA has been considered. The aim of the present study was to explore further the role of the hypothalamic-pituitary-adrenal axis in HA. DESIGN: We included 8 patients (aged 23.16+/-1.72 years) suffering from hypothalamic stress-related amenorrhea with normal body weight and 8 age-matched healthy controls in the follicular phase of the menstrual cycle. METHODS: We measured basal serum levels of FSH, LH, and estradiol and evaluated ACTH, allopregnanolone and cortisol responses to CRH test in both HA patients and healthy women. RESULTS: Serum basal levels of FSH, LH, and estradiol as well as basal levels of allopregnanolone were significantly lower in HA patients than in controls (P<0.001) while basal ACTH and cortisol levels were significantly higher in amenorrheic patients with respect to controls (P<0.001). The response (area under the curve) of ACTH, allopregnanolone and cortisol to CRH was significantly lower in amenorrheic women compared with controls (P<0.001, P<0.05, P<0.05 respectively). CONCLUSIONS: In conclusion, women with HA, despite the high ACTH and cortisol levels and, therefore, hypothalamus-pituitary-adrenal axis hyperactivity, are characterized by low allopregnanolone basal levels, deriving from an impairment of both adrenal and ovarian synthesis. The blunted ACTH, allopregnanolone and cortisol responses to CRH indicate that, in hypothalamic amenorrhea, there is a reduced sensitivity and expression of CRH receptor. These results open new perspectives on the role of neurosteroids in the pathogenesis of hypothalamic amenorrhea.
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Podfigurna, Agnieszka, and Blazej Meczekalski. "Functional Hypothalamic Amenorrhea: A Stress-Based Disease." Endocrines 2, no. 3 (July 24, 2021): 203–11. http://dx.doi.org/10.3390/endocrines2030020.

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The aim of the study is to present the problem of functional hypothalamic amenorrhea, taking into account any disease and treatment, diagnosis, and consequences of this disease. We searched PubMed (MEDLINE) and included 38 original and review articles concerning functional hypothalamic amenorrhea. Functional hypothalamic amenorrhea is the most common cause of secondary amenorrhea in women of childbearing age. It is a reversible disorder caused by stress related to weight loss, excessive exercise and/or traumatic mental experiences. The basis of functional hypothalamic amenorrhea is hormonal, based on impaired pulsatile GnRH secretion in the hypothalamus, then decreased secretion of gonadotropins, and, consequently, impaired hormonal function of the ovaries. This disorder leads to hypoestrogenism, manifested by a disturbance of the menstrual cycle in the form of amenorrhea, leading to anovulation. Prolonged state of hypoestrogenism can be very detrimental to general health, leading to many harmful short- and long-term consequences. Treatment of functional hypothalamic amenorrhea should be started as soon as possible, and it should primarily involve lifestyle modification. Only then should pharmacological treatment be applied. Importantly, treatment is most often long-term, but it results in recovery for the majority of patients. Effective therapy, based on multidirectional action, can protect patients from numerous negative impacts on fertility, cardiovascular system and bone health, as well as reducing mental morbidity.
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Gordon, Catherine M. "Functional Hypothalamic Amenorrhea." New England Journal of Medicine 363, no. 4 (July 22, 2010): 365–71. http://dx.doi.org/10.1056/nejmcp0912024.

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Berga, Sarah L. "Functional hypothalamic amenorrhea." Current Opinion in Endocrinology & Diabetes 8, no. 6 (December 2001): 307–13. http://dx.doi.org/10.1097/00060793-200112000-00008.

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Warren, M. P., F. Voussoughian, E. B. Geer, E. P. Hyle, C. L. Adberg, and R. H. Ramos. "Functional Hypothalamic Amenorrhea." Obstetrical & Gynecological Survey 54, no. 8 (August 1999): 510–11. http://dx.doi.org/10.1097/00006254-199908000-00017.

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Ferin, M. "Stress and hypothalamic amenorrhea." Gynecological Endocrinology 10, sup4 (January 1996): 42–43. http://dx.doi.org/10.3109/09513599609116179.

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Guay, Andre T., Sudhir Bansal, and Mary Beth Hodge. "Possible hypothalamic impotencemale counterpart to hypothalamic amenorrhea?" Urology 38, no. 4 (October 1991): 317–22. http://dx.doi.org/10.1016/0090-4295(91)80143-u.

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Dissertations / Theses on the topic "Hypothalamic amenorrhea"

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Lines, Linda. "Functional Hypothalamic Amenorrhea : Affected Women’s Perspective on Diagnosis and Treatment." Thesis, Uppsala universitet, Institutionen för folkhälso- och vårdvetenskap, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-448003.

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ABSTRACT Functional hypothalamic amenorrhea, FHA, is the clinical diagnosis for a woman in whommenstrual periods have been absent for three months or longer and where no other organic ormedical explanation is found. The aim was to explore characteristics and life experiencesamong women showing signs of FHA and to investigate what diagnoses and treatment/advicethey received when seeking medical help. Self-reported data were collected via an onlinesurvey. Descriptive data were presented regarding global representation, education levels,characteristics, life experiences, diagnoses, treatment/advice, treatment satisfaction, recoverysuccess and congruence between undertaken lifestyle changes and treatment/advice.Respondents consisted of 1,035 women from 71 different countries. The results showedcommon characteristics among respondents: the majority reported body appearancedissatisfaction, drive for thinness, high expectations of oneself, anxiety, obsessive behavioursand perfectionism. Life experiences were childhood bullying, loneliness and having a closerelative with mental health issues. Less than half of the participants received the diagnosisFHA and roughly a third did not receive any diagnosis/were told nothing was wrong. One infive were not given any treatment/advice that would help them regain their menstrual period.Ninety-three percent of those who recovered stated that they had gained weight, engaged lessin physical activity and/or undertaken cognitive behavioural therapy. Seventy-two percentwere dissatisfied with the treatment/advice they were given. In conclusion, the results fromthis study point to FHA as an underdiagnosed/misdiagnosed, neglected and mistreatedmedical condition and public health issue affecting women globally.
SAMMANFATTNING Funktionell hypotalamisk amenorré, FHA, är den kliniska diagnosen för en kvinna som varitutan menstruation i tre månader eller längre, där ingen annan organisk eller medicinskförklaring hittats. Syftet var att utforska förekomsten av personlighetsdrag ochlivserfarenheter bland kvinnor med kliniska tecken på FHA och att undersöka vilka diagnoseroch behandling/råd de fått när de sökt vård. Självrapporterad data inhämtades viainternetbaserade frågeformulär. Deskriptiv data presenterades avseende global representation,utbildningsnivå, personlighetsdrag, livserfarenheter, erhållna diagnoser, erhållnaråd/behandling, tillfredsställelse med behandling, behandlingsframgång och samstämmighetmellan livsstilsförändringar och erhållna råd/behandling. Svarspersonerna bestod av 1 035kvinnor från 71 olika länder. Resultaten visade gemensamma personlighetsdrag blandsvarspersonerna: majoriteten rapporterade kroppsmissnöje (utseendemässigt), strävan eftersmal kropp, höga förväntningar på sig själv, ångest/oro, tvångsmässiga beteenden ochperfektionism. Livserfarenheter var mobbning under barndomen, ensamhet och att ha ensläkting med psykisk ohälsa. Mindre än hälften fick diagnosen FHA och ungefär en tredjedelfick ingen diagnos/fick höra att ingenting var fel. En av fem fick ingen behandling/råd somhade kunnat hjälpa dem att återfå sin menstruation. Sjuttiotre procent var missnöjda medbehandlingen/råden de fick av sin läkare. Nittiotre procent av de som återfick sinmenstruation uppgav att de hade gått upp i vikt, ägnat sig mindre åt fysisk aktivitet och/ellergenomgått kognitiv beteendeterapi. Sjuttiotvå procent var missnöjda med den behandling/rådde gavs. Sammanfattningsvis pekar resultaten från denna studie mot att FHA är ettunderdiagnostiserat/feldiagnostiserat, negligerat och felbehandlat medicinskt tillstånd ochfolkhälsoproblem som drabbar kvinnor över hela världen.
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Timby, Erika. "Allopregnanolone effects in women : clinical studies in relation to the menstrual cycle, premenstrual dysphoric disorder and oral contraceptive use." Doctoral thesis, Umeå universitet, Obstetrik och gynekologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-50058.

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Background: Premenstrual dysphoric disorder (PMDD) affects 3–8% of women in fertile ages. Combined oral contraceptives (OCs) are widely used and some users experience adverse mood effects. The cyclicity of PMDD symptoms coincides with increased endogenous levels of allopregnanolone after ovulation. Allopregnanolone enhances the effect of γ-aminobutyric acid (GABA) on the GABAA receptor, the principal inhibitory transmitter system in the brain. The sensitivity to other GABAA receptor agonists than allopregnanolone (i.e. benzodiazepines, alcohol and the 5 β epimer to allopregnanolone, pregnanolone) has been reported to depend on menstrual cycle phase and/or PMDD diagnosis. Isoallopregnanolone, the 3 β epimer to allopregnanolone, has previously been used to verify specific allopregnanolone GABAA receptor effects. Saccadic eye velocity (SEV) is a sensitive and objective measurement of GABAA receptor function. Aims: To study the pharmacological effects, and any effect on gonadotropin release, of intravenous allopregnanolone in healthy women. A second aim was to explore whether allopregnanolone sensitivity differs over the menstrual cycle or during OC use in healthy women, and thirdly in PMDD patients. Methods: Ten women were challenged with a cumulative dose of intravenous allopregnanolone in the follicular phase of the menstrual cycle. The effect on FSH and LH was compared to women exposed to isoallopregnanolone. A single dose of allopregnanolone was administered once in the follicular phase and once in the luteal phase in another ten healthy women and in ten PMDD patients, and additionally in ten women using OCs. Repeated measurements of SEV, subjectively rated sedation and serum concentrations after allopregnanolone injections were performed in all studies. Results: Allopregnanolone dose-dependently reduced SEV and increased subjectively rated sedation. Healthy women had a decreased SEV response in the luteal phase compared to the follicular phase. By contrast, PMDD patients had a decreased SEV response and subjectively rated sedation response to allopregnanolone in the follicular phase compared to the luteal phase. There was no difference in the SEV response to allopregnanolone between women using oral contraceptives and healthy naturally cycling women. Allopregnanolone decreased serum levels of FSH and LH whereas isoallopregnanolone did not affect FSH and LH levels. Conclusion: Intravenous allopregnanolone was safely given and produced a sedative response in terms of SEV and subjectively rated sedation in women. The sensitivity to allopregnanolone was associated with menstrual cycle phase, but in the opposite direction in healthy women compared to PMDD patients. The results suggest mechanisms of physiological tolerance to allopregnanolone across the menstrual cycle in healthy women and support that PMDD patients have a disturbed GABAA receptor function. In addition, one of our studies suggests that allopregnanolone might be involved in the mechanism behind hypothalamic amenorrhea.
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Franklin, Kim. "Hur påverkar funktionell hypotalamisk amenorré (FHA) fertilitet och eventuell graviditet hos kvinnor med anorexia nervosa?" Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-105708.

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Bakgrund: Ett av sex par har någon gång upplevt problem relaterat till fertilitet under sina reproduktiva år och efter 30 års ålder är infertilitet vanligare hos kvinnor än hos män. Flera delar av menstruationscykeln består av energikrävande processer som exempelvis ägglossning och produktion av könshormoner. Näringsbrist och låg energitillgänglighet leder till brist på substrat till dessa energikrävande processer och i västvärlden orsakas låg energitillgänglighet vanligen av en ätstörning som anorexia nervosa, vilket kan leda till funktionell hypotalamisk amenorré (FHA) hos kvinnor. FHA resulterar i en minskad frisättning av könshormonerna östrogen och progesteron vilket kan leda till infertilitet. En av 20 kvinnor har erfarenhet av ätstörning under graviditeten men få studier har undersökt hur en historik med ätstörning påverkar fertilitet och graviditet. Syfte: Syftet med studien var att undersöka om FHA hos kvinnor med anoreci leder till nedsatt fertilitet och komplikationer vid en eventuell graviditet. Metod: En litteratursökning genomfördes på PubMed och Web of Science med sökorden amenorrhea, fertility, eating disorders, anorexia nervosa, reproduction (1999-2021). Resultat: Åtta studier inkluderades och resultatet visade att kvinnor med anorexi födde färre barn och hade större sannolikhet för att ha genomgått fertilitetsbehandling än friska kvinnor i kontrollgruppen. Vidare visade resultatet att kvinnor med anorexi oftare rapporterade komplicerade graviditeter med till exempel lägre fostertillväxt, prematur födsel och kejsarsnitt. Slutsats: Utifrån resultatet i den aktuella litteraturstudien kan konkluderas att kvinnor med FHA på grund av en ätstörning har lägre fertilitet än friska kvinnor. Kvinnor med ätstörning upplever i högre utsträckning mer komplicerade graviditeter och även fosterutvecklingen verkar påverkas negativt och därför kan tätare kontroller under och efter graviditet vara nödvändigt för dessa kvinnor. Resultatet kan vidare tolkas som att den negativa påverkan på reproduktionsförmågan kan vara reversibel när ätstörninssymptomen behandlats.
Background: One in six couples has sometime during their reproductive years experienced problems related to fertility and after the age of 30, infertility is more common in somen than in men. Several parts of the menstrual cycle require a lot of energy, such as ovulation and the production of sex hormones. Malnutrition and low energy availability is usually caused by an eating disorder such as anorexia nervosa, which can lead to functional hypothalamic amenorrhea (FHA) in women. FHA leads to a reduced release of the sex hormones estrogene and progesterone, which leads to infertility. One in 20 women have experience of an eating disorder during pregnancy, but few studies have examined how a history og eating disorder affects fertility and pregnancy. Aim: The aim of this study was to investigate whether FHA in women with anorexia nervosa leads to reduced fertility and complications in a potential pregnancy.  Method: A literature search was made on PubMed and Web of Science with the keyword´s amenorrhea, fertility, eating disorders, anorexia nervosa reproduction (1999-2021). Results: Eight studies were included, and the results showed that women with anorexia gave birth to fewer children and were more likely to have experienced fertility treatment than healthy women in the control group. Furthermore, the results showed that women with anorexia more often reported more complicated pregnancies with, e.g., lower fetal growth, premature birth, and cesarean section. Conclusion: Based on the results of the current literature study, it can be concluded that women with FHA due to an eating disorder have lower fertility than healthy women. Women with an eating disorder experience more complicated pregnancies and fetal development also seems to be negatively affected and therefore more frequent checks during and after pregnancy may be necessary fore these women. The results can further be interpreted as that the negative impact on reproductive health is reversible when symptoms of eating disorder are treated.
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BLANCHARD, PATRICIA. "Traitement des amenorrhees hypothalamiques par la pompe pulsatile a gnrh : a propos de notre experience nantaise." Nantes, 1988. http://www.theses.fr/1988NANT150M.

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CHAMPAIN, JEAN-FRANCOIS. "Exploration de la fonction gonadotrope dans l'anorexie mentale feminine : relations avec le poids corporel." Aix-Marseille 2, 1988. http://www.theses.fr/1988AIX20387.

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O'Donnell, Emma. "Cardiovascular Consequences of Estrogen Deficiency: Studies in Premenopausal Women." Thesis, 2013. http://hdl.handle.net/1807/43692.

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The influence of estrogen deficiency in physically active women with functional hypothalamic amenorrhea (ExFHA) on cardiovascular regulation is unknown. Three mechanistic studies compared cardiovascular responses to exercise and orthostatic stress in ExFHA women with responses in physically active (ExOv) and sedentary (SedOv) eumenorrheic ovulatory women. Measures included calf blood flow (BF), brachial artery (BA) endothelial dependent and independent function, shear rate (SR), vascular resistance (VR), blood pressure (BP), heart rate (HR), HR variability (HRV), muscle sympathetic nervous activity (MSNA), and serum renin-angiotensin-aldosterone system (RAAS) components. Study one examined the effects of a single bout of dynamic exercise on vascular function in ExFHA (n=12), ExOv (n=14), and SedOv (n=15) women. Pre-exercise, calf BF and BA endothelium-dependent flow-mediated vasodilation (FMD%) were lower (p<0.05) in ExFHA versus ovulatory women in association with higher (p<0.05) calf VR and lower (p<0.05) SR, respectively. Endothelium-independent vasodilation, assessed at baseline only, was also lower (p<0.05) in ExFHA. Post-exercise, calf BF was increased and VR decreased (p<0.05) in ExFHA women, similar (p>0.05) to that observed in ovulatory women. FMD% and SR were augmented (p<0.05) post-exercise, but both remained lower (p<0.05) in ExFHA versus ovulatory women (p<0.05). Study two investigated neurohumoral (MSNA and RAAS) BP regulation during orthostatic stress in ExFHA (n=12) and ExOv (n=17) women. Baseline systolic BP was lower (p<0.05) in ExFHA versus ExOv. Neurohumoral measures did not differ (p>0.05) between the groups at baseline. However, during hypotensive stimuli, MSNA increased to a greater extent (p<0.05), yet angiotensin II and renin were not activated in ExFHA women. Study three examined autonomic control of HR during orthostatic stress in ExFHA (n=11), ExOv (n=17), and SedOv (n=17) women. Lower HR (p<0.05) at rest and during orthostatic stress in ExFHA was associated with markedly elevated (p<0.05) HRV due to higher (p<0.05) parasympathetic modulation. Sympathetic modulation did not differ (p>0.05) between the groups. These studies indicate altered cardiovascular regulation in otherwise healthy ExFHA women. The influence of estrogen deficiency per se in these alterations are not clear, but in light of the etiology of amenorrhea, it is likely that complex interactions between estrogen and energy deficiency and exercise training are involved.
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Book chapters on the topic "Hypothalamic amenorrhea"

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Dicken, Cary, Marie Menke, and Genevieve Neal-Perry. "The Hypothalamic-Pituitary-Ovarian Axis." In Amenorrhea, 1–19. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-864-5_1.

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Berga, Sarah L. "Hypothalamic Amenorrhea." In Menstrual Cycle Related Disorders, 15–26. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14358-9_2.

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Kanj, Rula V., and Catherine M. Gordon. "Hypothalamic Amenorrhea." In Pituitary Disorders of Childhood, 269–77. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11339-1_14.

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Loucks, Tammy L., and Sarah L. Berga. "Clinical Implications of Prolonged Hypothalamic Amenorrhea." In Amenorrhea, 171–86. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-864-5_10.

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Tiedemann, Anne, Catherine Sherrington, Daina L. Sturnieks, Stephen R. Lord, Mark W. Rogers, Marie-Laure Mille, Paavo V. Komi, et al. "Functional Hypothalamic Amenorrhea." In Encyclopedia of Exercise Medicine in Health and Disease, 350. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2420.

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Warren, Michelle P., and Jennifer E. Dominguez. "Leptin and Hypothalamic Amenorrhea." In Leptin and Reproduction, 311–32. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0157-2_19.

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Proske, Uwe, David L. Morgan, Tamara Hew-Butler, Kevin G. Keenan, Roger M. Enoka, Sebastian Sixt, Josef Niebauer, et al. "Exercise-Associated Functional Hypothalamic Amenorrhea." In Encyclopedia of Exercise Medicine in Health and Disease, 324. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4207.

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Podfigurna, Agnieszka, Adam Czyzyk, Anna Szeliga, and Błażej Meczekalski. "Kisspeptin Role in Functional Hypothalamic Amenorrhea." In Menstrual Cycle Related Disorders, 27–42. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14358-9_3.

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Chou, Sharon H., and Christos Mantzoros. "Leptin Therapy in Women with Hypothalamic Amenorrhea." In Leptin, 237–54. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09915-6_19.

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Miller, Karen K. "M55 – Bone Loss in Women with Hypothalamic Amenorrhea." In 2012 Meet-The-Professor: Endocrine Case Management, 168–72. 8401 Connecticut Avenue, Suite 900, Chevy Chase, MD 20815 www.endo-society.org: The Endocrine Society, 2012. http://dx.doi.org/10.1210/mtp1.9781936704729.ch26.

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