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Journal articles on the topic 'Hypotonic shock'

Author: Grafiati

Published: 9 March 2023

Last updated: 31 July 2025

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1

Dinh, Xuan Tu, Huynh Thi Diem Suong Le, and Minh Ly Nguyen. "The chromosome numbers of Panax vietnamensis Ha et Grushv." Can Tho University Journal of Science 14, CBA (2022): 86–90. http://dx.doi.org/10.22144/ctu.jen.2022.033.

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The somatic chromosome number of Panax vietnamensis Ha et Grushv. was determined to be 2n = 24, based on the hypotonic shock method by potassium chloride solution. In this study, we investigated the effect of potassium chloride and colchicine solutions on chromosome dispersion of Panax vietnamensis at different concentrations. The treatment using 0.2% KCl solution in 45 minutes combined with 0.05% colchicine solution in 2 hours subsequently resulted in proper hypotonia. The result showed that chromosomes were evenly dispersed. The hypotonic shock method seemed to be effective in equally distri

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2

Beck, J. S., S. Breton, G. Giebisch, and R. Laprade. "Potassium conductance regulation by pH during volume regulation in rabbit proximal convoluted tubules." American Journal of Physiology-Renal Physiology 263, no. 3 (1992): F453—F458. http://dx.doi.org/10.1152/ajprenal.1992.263.3.f453.

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When rabbit proximal convoluted tubules were microperfused in the presence of bicarbonate, a 90 mosmol hypotonic shock hyperpolarized the basolateral membrane by 5.5 +/- 1.4 mV, increased basolateral potassium selectivity (tK) from 0.30 +/- 0.02 to 0.45 +/- 0.02, and reduced the basolateral membrane resistance from 4,887 +/- 821 to 2,836 +/- 602 omega.cm. These data show that the hypotonic shock increased absolute basolateral potassium conductance. The same hypotonic shock elevated intracellular pH from 7.18 +/- 0.04 to 7.31 +/- 0.04. When bath pH was increased by 0.2 pH units (by reduction of

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3

Marshall, W. S., S. E. Bryson, and T. Luby. "Control of epithelial Cl(−) secretion by basolateral osmolality in the euryhaline teleost Fundulus heteroclitus." Journal of Experimental Biology 203, no. 12 (2000): 1897–905. http://dx.doi.org/10.1242/jeb.203.12.1897.

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Euryhaline teleost fish adapt rapidly to salinity change and reduce their rate of ion secretion on entry to fresh water. Killifish (Fundulus heteroclitus) transferred from full-strength sea water to fresh water showed large reductions in plasma [Na(+)] and osmolality at 6 h which were corrected by 24 h. To mimic this in vitro, a hypotonic shock of 20–70 mosmol kg(−)(1) was applied on the basolateral side of opercular epithelia. This hypotonic shock reversibly reduced the short-circuit current (I(sc), equivalent to the rate of secretion of Cl(−)) in a dose-dependent fashion, with a 40 mosmol kg

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4

Bear, C. E. "A nonselective cation channel in rat liver cells is activated by membrane stretch." American Journal of Physiology-Cell Physiology 258, no. 3 (1990): C421—C428. http://dx.doi.org/10.1152/ajpcell.1990.258.3.c421.

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A 16-pS channel was studied using patch-clamp electrophysiology in freshly dissociated rat liver cells and rat hepatoma cells. The channel was found to be cation selective and permeable to Na+, K+, and Ca2+. Its gating was unaffected by addition of the calcium ionophore A23187 (5 microM) in the presence of extracellular Ca2+ (2 mM). Ca2+ channel blockers, nifedipine, verapamil, and lanthanum, failed to inhibit the channel. The channel was activated by stretch, applied as suction to the interior of the patch pipette, and by cell swelling, induced by hypotonic shock or organic solute uptake (10

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5

Fujii, Shuhei, and Johan A. Hellebust. "Release of intracellular glycerol and pore formation in Dunaliella tertiolecta exposed to hypotonic stress." Canadian Journal of Botany 70, no. 7 (1992): 1313–18. http://dx.doi.org/10.1139/b92-164.

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When Dunaliella tertiolecta cells, previously cultured in a 0.5 M NaCl medium, were resuspended in a 0.25 NaCl medium, about 50% of the intracellular glycerol was lost within 2 min. A corresponding amount of glycerol appeared in the medium, while other organic solutes, such as amino acids and sugars, were not detected. These results indicate that intracellular glycerol is rapidly released without significant concomitant cell damage. Rubidum, in the case of rubidium-loaded cells, was also rapidly released to the medium in response to hypotonic shock. Gramicidin, dimers of which form stable memb

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6

Sawaied, Alaa, Bat-El Levy, Eden Arazi, Eitan Lunenfeld, Qinghua Shi, and Mahmoud Huleihel. "Follicle-Stimulating Hormone and Testosterone Play a Role in the Regulation of Sertoli Cell Functions Following Germ Cell Depletion In Vitro." International Journal of Molecular Sciences 26, no. 6 (2025): 2702. https://doi.org/10.3390/ijms26062702.

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Spermatogenesis is a process of self-renewal of spermatogonial stem cells and their proliferation and differentiation to generate mature sperm. This process involves interactions between testicular somatic (mainly Sertoli cells) and spermatogonial cells at their different stages of development. The functionality of Sertoli cells is regulated by hormones and testicular autocrine/paracrine factors. In this study, we investigated the effects of follicle-stimulating hormone (FSH) and testosterone addition on Sertoli cell cultures that undergo hypotonic shock, with a primary focus on Sertoli cell a

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7

Macri, P., S. Breton, J. S. Beck, J. Cardinal, and R. Laprade. "Basolateral K+, Cl-, and HCO3- conductances and cell volume regulation in rabbit PCT." American Journal of Physiology-Renal Physiology 264, no. 2 (1993): F365—F376. http://dx.doi.org/10.1152/ajprenal.1993.264.2.f365.

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The relationship between changes in cellular volume, intracellular pH (pHi), basolateral membrane potential (VBL), and membrane partial basolateral conductances to K+ (tK) and Cl- (tCl) and mediated by the Na-HCO3 cotransporter (tNaHCO3) was determined in the collapsed proximal convoluted tubule (PCT) submitted to a 125-mosmol/kg hypotonic shock. The shock that produces a rapid swelling followed by partial volume regulation was accompanied by a rapid and transient VBL hyperpolarization of 10.0 +/- 1.5 mV and a second gradual hyperpolarization of 5.0 +/- 0.7 mV with respect to a control value o

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8

Galietta, L. J., S. Falzoni, F. Di Virgilio, G. Romeo, and O. Zegarra-Moran. "Characterization of volume-sensitive taurine- and Cl(-)-permeable channels." American Journal of Physiology-Cell Physiology 273, no. 1 (1997): C57—C66. http://dx.doi.org/10.1152/ajpcell.1997.273.1.c57.

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Volume-sensitive Cl- channels [ICl(vol)] were studied using taurine efflux and patch-clamp experiments in 9HTEo- human tracheal cells. Cells were stimulated with the Ca(2+)- elevating agents ATP and ionomycin in isotonic medium or in hypotonic solutions. ATP (100 microM) or ionomycin (1 microM) and hypotonic shock produced a synergic effect. Indeed, the resulting taurine efflux was much higher than the sum of the single effects elicited by ATP, ionomycin, or hypotonic medium. The taurine release elicited by hypotonic shock and the potentiation by ATP and ionomycin were markedly inhibited by us

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9

Smets, Ilse, Marcel Ameloot, Paul Steels, and Willy Van Driessche. "Loss of cell volume regulation during metabolic inhibition in renal epithelial cells (A6): role of intracellular pH." American Journal of Physiology-Cell Physiology 283, no. 2 (2002): C535—C544. http://dx.doi.org/10.1152/ajpcell.00371.2001.

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In renal ischemia, tubular obstruction induced by swelling of epithelial cells might be an important mechanism for reduction of the glomerular filtration rate. We investigated ischemic cell swelling by examining volume regulation of A6 cells during metabolic inhibition (MI) induced by cyanide and 2-deoxyglucose. Changes in cell volume were monitored by recording cell thickness ( T c). Intracellular pH (pHc) measurements were performed with the pH-sensitive probe 5-chloromethyl-fluoresceine diacetate. T c measurements showed that MI increases cell volume. Cell swelling during MI is proportional

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10

Shpakova, Natalia, and Natalia Orlova. "About the Mechanism of Mammalian Erythrocytes Osmotic Stability." Problems of Cryobiology and Cryomedicine 30, no. 4 (2020): 331–42. http://dx.doi.org/10.15407/cryo30.04.331.

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The peculiarities of the effect of hypertonic shock and hypotonic stress on erythrocytes of different species of mammals (human, bull, horse, rabbit, dog, rat) have been investigated. Based on the results of correlation analysis (using the Spearman’s rank correlation coefficient), the relationship between osmotic sensitivity of mammalian erythrocytes and the well-known structural and functional characteristics of these cells was assessed. The paper presents and analyzes the significant relationships. Under hypotonic stress of mammalian erythrocytes, the values of the threshold concentration of

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11

Batiza, Ann F., Tara Schulz, and Patrick H. Masson. "Yeast Respond to Hypotonic Shock with a Calcium Pulse." Journal of Biological Chemistry 271, no. 38 (1996): 23357–62. http://dx.doi.org/10.1074/jbc.271.38.23357.

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12

Huang, L. E., L. Caruccio, A. Y. Liu, and K. Y. Chen. "Rapid activation of the heat shock transcription factor, HSF1, by hypo-osmotic stress in mammalian cells." Biochemical Journal 307, no. 2 (1995): 347–52. http://dx.doi.org/10.1042/bj3070347.

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Osmoregulation is important to living organisms for survival in responding to environmental changes of water and ionic strength. We demonstrated here for the first time that exposure of HeLa cells to a hypotonic medium (30% growth medium and 70% water) prominently induced the binding activity of the heat shock transcription factor (HSF). Pretreatment of cells with cycloheximide did not inhibit the induction of HSF-binding activity, indicating that the mechanisms of induction are independent of new protein synthesis. The magnitude of hypo-osmotic stress-induced HSF-binding activity was comparab

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13

Dube, L., L. Parent, and R. Sauve. "Hypotonic shock activates a maxi K+ channel in primary cultured proximal tubule cells." American Journal of Physiology-Renal Physiology 259, no. 2 (1990): F348—F356. http://dx.doi.org/10.1152/ajprenal.1990.259.2.f348.

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The nature and function of the ionic channels at the apical membrane of primary cultured proximal tubule cells (PT) was investigated by use of the extracellular patch-clamp method. Several types of ionic channels were observed, including a calcium-dependent K+ channel of 206 pS in symmetrical 162 mM KCl activated at depolarizing potentials [maxi K+(Ca2+)]. Whole cell experiments were also carried out that clearly indicated that the PT cells respond to a hypotonic shock by activating electroconductive pathways. This response consisted of an initial hyperpolarization (from -47 to -58 mV, SD = 3,

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14

Breton, S., M. Marsolais, J. Y. Lapointe, and R. Laprade. "Cell volume increases of physiologic amplitude activate basolateral K and CI conductances in the rabbit proximal convoluted tubule." Journal of the American Society of Nephrology 7, no. 10 (1996): 2072–87. http://dx.doi.org/10.1681/asn.v7102072.

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The effects of increases in cell volume (CV) of physiologic amplitude, induced either hypotonically or isotonically, were studied on the three major basolateral conductances of rabbit isolated proximal convoluted tubules. CV increases were produced by a 40 mosmol/kg H2O hypotonic shock or by the isotonic replacement of mannitol by 40 mM glucose or alanine. The hypotonic shock led to an increase in CV of 17 +/- 3% (N = 8), whereas additions of glucose and alanine led to increases in CV of 22.6 +/- 2.5% (N = 7) and 28.3 +/- 3.5 (N = 5), respectively. Under all of these conditions, the absolute c

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15

Duranton, C., E. Mikulovic, M. Tauc, M. Avella, and P. Poujeol. "Potassium channels in primary cultures of seawater fish gill cells. II. Channel activation by hypotonic shock." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 279, no. 5 (2000): R1659—R1670. http://dx.doi.org/10.1152/ajpregu.2000.279.5.r1659.

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Previous studies performed on apical membranes of seawater fish gills in primary culture have demonstrated the existence of stretch-activated K+channels with a conductance of 122 pS. The present report examines the involvement of K+ channels in ion transport mechanisms and cell swelling. In the whole cell patch-clamp configuration, K+ currents were produced by exposing cells to a hypotonic solution or to 1 μM ionomycin. These K+ currents were inhibited by the addition of quinidine and charybdotoxin to the bath solution. Isotopic efflux measurements were performed on cells grown on permeable su

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16

Cutler, Adrian J., and Mohammed Saleem. "Permeabilizing Soybean Protoplasts to Macromolecules Using Electroporation and Hypotonic Shock." Plant Physiology 83, no. 1 (1987): 24–28. http://dx.doi.org/10.1104/pp.83.1.24.

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17

Rugolo, Michela, Teresa Mastocola, Alberto Flamigni, and Giorgio Lenaz. "Chloride transport in human fibroblasts is activated by hypotonic shock." Biochemical and Biophysical Research Communications 160, no. 3 (1989): 1330–38. http://dx.doi.org/10.1016/s0006-291x(89)80149-4.

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18

Mastrocola, Teresa, Alberto Flamigni, and Michela Rugolo. "Hypotonic shock activated Cl− and K+ pathways in human fibroblasts." Biochimica et Biophysica Acta (BBA) - Biomembranes 1069, no. 2 (1991): 201–8. http://dx.doi.org/10.1016/0005-2736(91)90125-r.

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19

Bianchini, L., B. Fossat, J. Porthe-Nibelle, J. C. Ellory, and B. Lahlou. "Effects of hyposmotic shock on ion fluxes in isolated trout hepatocytes." Journal of Experimental Biology 137, no. 1 (1988): 303–18. http://dx.doi.org/10.1242/jeb.137.1.303.

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Isolated trout hepatocytes exposed to hypotonic Hank's medium (isotonicity × 0.70) swelled to 1.17 times the control volume after 3 min; by 15 min the cell volume had returned to normal. The ouabain-insensitive K+ uptake increased, indicating an immediate rise in K+ membrane permeability. As indicated by analysis of cellular contents, the regulatory volume decrease (RVD) was ensured by a release of intracellular K+. Na+ was not implicated in this mechanism. This potassium permeability induced by hypotonic shock was transient (maximum at 6 min), insensitive to blocking agents of voltage- and Ca

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20

Zhang, Zheng, and David M. Cohen. "Hypotonicity increases transcription, expression, and action ofEgr-1in murine renal medullary mIMCD3 cells." American Journal of Physiology-Renal Physiology 273, no. 5 (1997): F837—F842. http://dx.doi.org/10.1152/ajprenal.1997.273.5.f837.

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In cells of the murine renal inner medullary collecting duct (mIMCD3) cell line, acute hypotonic shock (50% dilution of medium with sterile water but not with sterile 150 mM NaCl) increased Egr-1 mRNA abundance 2.5-fold at 6 h, as determined by Northern analysis. This increase was accompanied by increased Egr-1 transcription, as quantitated by luciferase reporter gene assay. Increased transcription was dose dependent, additive with other Egr-1 transcriptional activators, and occurred in the absence of overt cytotoxicy, as quantitated via a fluorometric viability assay. In addition, hypotonic s

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21

Leeson, Cale E., Brianna-Lee Beaudry, and Geoffrey R. Wignall. "Septic Shock Immediately following Percutaneous Suprapubic Catheterization." Case Reports in Urology 2021 (August 31, 2021): 1–3. http://dx.doi.org/10.1155/2021/2184866.

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Suprapubic catheterization (SPC) is considered a safe and effective procedure for long-term bladder decompression. With proper technique and appropriate patient selection, significant complications of SPC are rare. Immediate postoperative septic shock (i.e., within the first 24 hours of surgery) is rarely reported. We report a case of an 83-year-old patient who developed septic shock within one hour of suprapubic catheterization for a chronic hypotonic bladder, highlighting the importance of early recognition of complications from SPC and prompt management to ensure positive outcomes.

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Straub, Susanne G., Samira Daniel та Geoffrey W. G. Sharp. "Hyposmotic shock stimulates insulin secretion by two distinct mechanisms. Studies with the βHC9 cell". American Journal of Physiology-Endocrinology and Metabolism 282, № 5 (2002): E1070—E1076. http://dx.doi.org/10.1152/ajpendo.00176.2001.

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Exposure of βHC9 cells to a Krebs-Ringer bicarbonate-HEPES buffer (KRBH) made hypotonic by a reduction of 25 mM NaCl resulted in a prompt stimulation of insulin release. The stimulation was transient, and release rates returned to basal levels after 10 min. The response resembles that of the first phase of glucose-stimulated insulin release. The response did not occur if the reduction in NaCl was compensated for by the addition of an equivalent osmolar amount of sorbitol, so the stimulation of release was due to the osmolarity change and not the reduction in NaCl. The hyposmotic shock released

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Daborn, K., R. R. F. Cozzi, and W. S. Marshall. "Dynamics of Pavement Cell–Chloride Cell Interactions During Abrupt Salinity Change in FUNDULUS HETEROCLITUS." Journal of Experimental Biology 204, no. 11 (2001): 1889–99. http://dx.doi.org/10.1242/jeb.204.11.1889.

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SUMMARY Freshwater-adapted killifish (Fundulus heteroclitus) opercular epithelia were dissected and subjected to blood-side hypertonic bathing solution in Ussing-style chambers to simulate the increase in blood osmolality during migration to sea water. Conversely, seawater-acclimated killifish opercular epithelia were subjected to hypotonic bathing solutions to simulate the initial stages of migration to fresh water. Freshwater-acclimation (hypertonic stress) induced a rapid (approximately 30min) increase in membrane conductance (Gt) from 3.10±0.56 to 7.52±1.15mScm−2 (P&lt;0.01, N=27), whe

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Rubera, Isabelle, Hervé Barrière, Michel Tauc, et al. "Extracellular adenosine modulates a volume-sensitive-like chloride conductance in immortalized rabbit DC1 cells." American Journal of Physiology-Renal Physiology 280, no. 1 (2001): F126—F145. http://dx.doi.org/10.1152/ajprenal.2001.280.1.f126.

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Cl−currents induced by cell swelling were characterized in an immortalized cell line (DC1) derived from rabbit distal bright convoluted tubule by the whole cell patch-clamp techniques and by125I− efflux experiments. Exposure of cells to a hypotonic shock induced outwardly rectifying Cl−currents that could be blocked by 0.1 mM 5-nitro-2-(3-phenylpropyl-amino)benzoic acid, 1 mM DIDS, and by 1 mM diphenylamine-2-carboxylate. 125I− efflux experiments showed that exposure of the monolayer to a hypotonic medium increased 125I− loss. Preincubation of cells with LaCl3 or GdCl3 prevented the developmen

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WILKINS, R. J., T. P. AFAIRFAX, M. E. DAVIES, M. C. MUZYAMBA, and J. S. GIBSON. "Homeostasis of intracellular Ca2+ in equine chondrocytes: response to hypotonic shock." Equine Veterinary Journal 35, no. 5 (2010): 439–43. http://dx.doi.org/10.2746/042516403775600541.

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Sánchez, J. C., and R. J. Wilkins. "Effects of hypotonic shock on intracellular pH in bovine articular chondrocytes." Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 135, no. 4 (2003): 575–83. http://dx.doi.org/10.1016/s1095-6433(03)00138-7.

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Olmos, Gemma, L. Alfredo Lotero, M. Cristina Tejedor, and José C. Diez. "Delivery to Macrophages of Interleukin 3 Loaded in Mouse Erythrocytes." Bioscience Reports 20, no. 5 (2000): 399–410. http://dx.doi.org/10.1023/a:1010334118492.

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Mouse carrier erythrocytes containing 125I-interleukin 3 have been prepared and treated with band 3 crosslinking reagents. The incorporation of interleukin 3 by hypotonic treatment into mouse erythrocytes reached levels of about 15% of the interleukin 3 added to the medium being predominantly present in the cytosolic fraction (73%). Uptake fell to about 7.4% when using the same conditions but omitting hypotonic shock. The interaction of band 3 crosslinked interleukin 3 loaded erythrocytes with macrophages was also studied. A high level of incorporation of interleukin 3 into macrophages was obs

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Galizia, L., M. P. Flamenco, V. Rivarola, C. Capurro, and P. Ford. "Role of AQP2 in activation of calcium entry by hypotonicity: implications in cell volume regulation." American Journal of Physiology-Renal Physiology 294, no. 3 (2008): F582—F590. http://dx.doi.org/10.1152/ajprenal.00427.2007.

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We previously reported in a rat cortical collecting duct cell line (RCCD1) that the presence of aquaporin 2 (AQP2) in the cell membrane is critical for the rapid activation of regulatory volume decrease mechanisms (RVD) (Ford et al. Biol Cell 97: 687–697, 2005). The aim of our present work was to investigate the signaling pathway that links AQP2 to this rapid RVD activation. Since it has been previously described that hypotonic conditions induce intracellular calcium ([Ca2+]i) increases in different cell types, we tested the hypothesis that AQP2 could have a role in activation of calcium entry

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Mitchell, C. H., J. J. Zhang, L. Wang, and T. J. Jacob. "Volume-sensitive chloride current in pigmented ciliary epithelial cells: role of phospholipases." American Journal of Physiology-Cell Physiology 272, no. 1 (1997): C212—C222. http://dx.doi.org/10.1152/ajpcell.1997.272.1.c212.

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The whole cell recording technique was used to examine an outwardly rectifying chloride current activated by hypotonic shock in bovine pigmented ciliary epithelial (PCE) cells. Removal of internal and external Ca2+ did not affect the activation of these currents, but they were abolished by the phospholipase C inhibitor neomycin. The current was blocked by 5-nitro-2-(3-phenylpropylamino)benzoic acid, 4-acetamido-4'-isothiocyanostilbene-2,2'-disulfonic acid, and 4,4'-disothiocyanostilbene-2,2'-disulfonic acid (DIDS) in a voltage-dependent manner, but tamoxifen, dideoxyforskolin, and quinidine di

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Breton, S., J. S. Beck, J. Cardinal, G. Giebisch, and R. Laprade. "Involvement and source of calcium in volume regulatory decrease of collapsed proximal convoluted tubule." American Journal of Physiology-Renal Physiology 263, no. 4 (1992): F656—F664. http://dx.doi.org/10.1152/ajprenal.1992.263.4.f656.

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We examined the role of Ca2+ in the volume regulatory decrease (VRD) of rabbit collapsed proximal tubules. Reduction of bath osmolality by 125 mosmol/kgH2O led to an initial cell swelling of 62.3 +/- 7.5% followed by a partial regulatory phase bringing cell volume to a value of 13.3 +/- 2.9% above control (n = 5). This swelling was accompanied by a transient intracellular Ca2+ ([Ca2+]i) increase from 174 +/- 33 to 306 +/- 67 nM (P < 0.05, n = 8). In the same condition, but in absence of extracellular Ca2+ ([Ca2+]e) [1 mM ethylene glycol-bis(beta-aminoethyl ether)-N,N,N',N'-tetraacetic acid

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Li, Jinqing, Patrick De Smet, Danny Jans, Jeannine Simaels, and Willy Van Driessche. "Swelling-activated cation-selective channels in A6 epithelia are permeable to large cations." American Journal of Physiology-Cell Physiology 275, no. 2 (1998): C358—C366. http://dx.doi.org/10.1152/ajpcell.1998.275.2.c358.

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Effects of basolateral monovalent cation replacements (Na+ by Li+, K+, Cs+, methylammonium, and guanidinium) on permeability to86Rb of volume-sensitive cation channels (VSCC) in the basolateral membrane and on regulatory volume decrease (RVD), elicited by a hyposmotic shock, were studied in A6 epithelia in the absence of apical Na+ uptake. A complete and quick RVD occurred only when the cells were perfused with Na+ or Li+ saline. With both cations, hypotonicity increased basolateral86Rb release ([Formula: see text]), which reached a maximum after 15 min and declined back to control level. When

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32

Lemoine, J. L., R. Farley, and L. Huang. "Mechanism of efficient transfection of the nasal airway epithelium by hypotonic shock." Gene Therapy 12, no. 16 (2005): 1275–82. http://dx.doi.org/10.1038/sj.gt.3302548.

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Schlotmann, R., F. Ch Mooren, R. Stoll, and W. Domschke. "Hypotonic shock causes an intracellular calcium increase in rat gastric parietal cells." Gastroenterology 108, no. 4 (1995): A214. http://dx.doi.org/10.1016/0016-5085(95)23517-5.

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McGrath, John J., and Peter C. Thomas. "The influence of osmotic species on human erythrocyte hypotonic cold shock hemolysis." Cryobiology 23, no. 6 (1986): 546. http://dx.doi.org/10.1016/0011-2240(86)90072-6.

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Faggio, Caterina, Agata Torre, Elisa Pelle, Federica Raffa, Valentina Villari, and Francesca Trischitta. "Cell volume regulation following hypotonic shock in hepatocytes isolated from Sparus aurata." Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 158, no. 1 (2011): 143–49. http://dx.doi.org/10.1016/j.cbpa.2010.10.002.

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36

Mignen, Olivier, Christelle Le Gall, Brian J. Harvey, and Serge Thomas. "Volume regulation following hypotonic shock in isolated crypts of mouse distal colon." Journal of Physiology 515, no. 2 (1999): 501–10. http://dx.doi.org/10.1111/j.1469-7793.1999.501ac.x.

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37

Pogorelov, A. G., and V. N. Pogorelova. "Dynamics of cell volume in early mouse embryos subjected to hypotonic shock." Biophysics 54, no. 3 (2009): 336–39. http://dx.doi.org/10.1134/s0006350909030130.

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LÖVKVIST WALLSTRÖM, Eva, Koichi TAKAO, Anna WENDT, Cristina VARGIU, Hong YIN, and Lo PERSSON. "Importance of the 3′ untranslated region of ornithine decarboxylase mRNA in the translational regulation of the enzyme." Biochemical Journal 356, no. 2 (2001): 627–34. http://dx.doi.org/10.1042/bj3560627.

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Translational regulation of ornithine decarboxylase (ODC), which catalyses the first step in the biosynthesis of polyamines, appears to be an important mechanism in the strong feedback control as well as in the hypotonic induction of the enzyme. However, the exact mechanisms are not yet understood. The ODC mRNA has long 5′ and 3′ untranslated regions (UTRs) which may be involved in the translational control of the enzyme. In the present study we have used a series of stable transfectants of Chinese Hamster ovary cells expressing ODC mRNAs with various truncations in the 5′ and 3′ UTRs to inves

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Adorante, Joseph S., and Jeffrey L. Edelman. "Letters to the Editor." American Journal of Physiology-Cell Physiology 273, no. 4 (1997): C1435—C1436. http://dx.doi.org/10.1152/ajpcell.1997.273.4.c1435.

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The following is the abstract of the article discussed in the subsequent letter: Mitchell, Claire H., Jin Jun Zhang, Liwei Wang, and Tim J. C. Jacob. Volume-sensitive chloride current in pigmented ciliary epithelial cells: role of phospholipases. Am. J. Physiol. 272 ( Cell Physiol. 41): C212–C222, 1997.—The whole cell recording technique was used to examine an outwardly rectifying chloride current activated by hypotonic shock in bovine pigmented ciliary epithelial (PCE) cells. Removal of internal and external Ca2+ did not affect the activation of these currents, but they were abolished by the

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Bianchini, L., B. Fossat, J. Porthe-Nibelle, and B. Lahlou. "Activation by N-ethylmaleimide of a Cl--dependent K+ flux in isolated trout hepatocytes." Journal of Experimental Biology 157, no. 1 (1991): 335–48. http://dx.doi.org/10.1242/jeb.157.1.335.

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Isolated trout hepatocytes when swollen in hypotonic medium undergo a regulatory volume decrease (RVD), which occurs via KCl loss. The system shows characteristics similar to those of the transporter described in red cells. This led us to investigate, in trout hepatocytes, the effect of another signal known to activate this flux in red cells, i.e. treatment with the sulphhydryl-group reagent N-ethylmaleimide (NEM). NEM treatment resulted in a striking increase in ouabain-resistant K+ uptake measured by an isotope pulse uptake technique. The time course of the response to NEM was similar to tha

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Hoffmann, Tamara, Clara Boiangiu, Susanne Moses, and Erhard Bremer. "Responses of Bacillus subtilis to Hypotonic Challenges: Physiological Contributions of Mechanosensitive Channels to Cellular Survival." Applied and Environmental Microbiology 74, no. 8 (2008): 2454–60. http://dx.doi.org/10.1128/aem.01573-07.

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ABSTRACT Mechanosensitive channels are thought to function as safety valves for the release of cytoplasmic solutes from cells that have to manage a rapid transition from high- to low-osmolarity environments. Subsequent to an osmotic down-shock of cells grown at high osmolarity, Bacillus subtilis rapidly releases the previously accumulated compatible solute glycine betaine in accordance with the degree of the osmotic downshift. Database searches suggest that B. subtilis possesses one copy of a gene for a mechanosensitive channel of large conductance (mscL) and three copies of genes encoding pro

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Nanduri, Jayasri, and Alan M. Tartakoff. "Perturbation of the Nucleus: A Novel Hog1p-independent, Pkc1p-dependent Consequence of Hypertonic Shock in Yeast." Molecular Biology of the Cell 12, no. 6 (2001): 1835–41. http://dx.doi.org/10.1091/mbc.12.6.1835.

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Hypertonic shock of Saccharomyces cerevisiaeactivates the Hog1p MAP kinase cascade. In contrast, protein kinase C (Pkc1p) and the “cell integrity” MAP kinase cascade are critical for the response to hypotonic shock. We observed that hypertonic shock transiently relocated many, but not all, nuclear and nucleolar proteins to the cytoplasm. We hypothesized that the relocation of nuclear proteins was due to activation of the Hog1p kinase cascade, yet, surprisingly, Hog1p was not required for these effects. In contrast, Pkc1p kinase activity was required, although the Pkc1p MAP kinase cascade and s

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Solenov, Evgeny I. "Cell Volume and Sodium Content in Rat Kidney Collecting Duct Principal Cells During Hypotonic Shock." Journal of Biophysics 2008 (July 27, 2008): 1–5. http://dx.doi.org/10.1155/2008/420963.

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The purpose of this study was to investigate the time course of the volume-regulatory response and intracellular sodium concentration ([Na+]i) in the principal cells of rat kidney outer medulla collecting duct (OMCD) epithelia during acute swelling in hypotonic medium. Hypotonic shock was created by PBS diluted with 50% of water. Changes in cell volume were measured with calcein quenching method. Intracellular sodium concentration was studied with fluorescence dye Sodium Green. Principal cells of microdissected OMCD fragments swelled very fast. The characteristic time of swelling (τ1) was 0.65

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Waseem, Tatyana V., Sergei V. Konev, and Sergei V. Fedorovich. "Influence of Hypotonic Shock on Glutamate and GABA Uptake in Rat Brain Synaptosomes." Neurochemical Research 29, no. 9 (2004): 1653–58. http://dx.doi.org/10.1023/b:nere.0000035799.79422.d1.

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Fret, T., L. Heylen, M. Nuydens, F. De Jongh, and T. Meert. "Hypotonic shock in mice as in vivo model for cytotoxic brain oedema formation." European Journal of Anaesthesiology 25, Supplement 43 (2008): 1. http://dx.doi.org/10.1097/00003643-200801001-00001.

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Ubl, J., H. Murer, and H. A. Kolb. "Hypotonic shock evokes opening of Ca2+-activated K channels in opossum kidney cells." Pflügers Archiv - European Journal of Physiology 412, no. 5 (1988): 551–53. http://dx.doi.org/10.1007/bf00582547.

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Avella, Martine, Olivier Ducoudret, Didier F. Pisani, and Philippe Poujeol. "Swelling-activated transport of taurine in cultured gill cells of sea bass: physiological adaptation and pavement cell plasticity." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 4 (2009): R1149—R1160. http://dx.doi.org/10.1152/ajpregu.90615.2008.

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We have investigated volume-activated taurine transport and ultrastructural swelling response of sea bass gill cells in culture, assuming that euryhaline fish may have developed particularly efficient mechanisms of salinity adaptation. In vivo, when sea basses were progressively transferred from seawater to freshwater, we noticed a decrease in blood osmotic pressure. When gill cells in culture were subjected to 30% hypotonic shock, we observed a five-fold stimulation of [3H]taurine efflux. This transport was reduced by various anion channel inhibitors with the following efficiency: 5-nitro-2-(

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L'Hoste, S., H. Barriere, R. Belfodil, et al. "Extracellular pH alkalinization by Cl−/HCO3− exchanger is crucial for TASK2 activation by hypotonic shock in proximal cell lines from mouse kidney." American Journal of Physiology-Renal Physiology 292, no. 2 (2007): F628—F638. http://dx.doi.org/10.1152/ajprenal.00132.2006.

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We have previously shown that K+-selective TASK2 channels and swelling-activated Cl− currents are involved in a regulatory volume decrease (RVD; Barriere H, Belfodil R, Rubera I, Tauc M, Lesage F, Poujeol C, Guy N, Barhanin J, Poujeol P. J Gen Physiol 122: 177–190, 2003; Belfodil R, Barriere H, Rubera I, Tauc M, Poujeol C, Bidet M, Poujeol P. Am J Physiol Renal Physiol 284: F812–F828, 2003). The aim of this study was to determine the mechanism responsible for the activation of TASK2 channels during RVD in proximal cell lines from mouse kidney. For this purpose, the patch-clamp whole-cell techn

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Sblano, Cesare, Silvia Micelli та Daniela Meleleo. "Effects of n-Octyl-β-D-Glucopyranoside on Human and Rat Erythrocyte Membrane Stability Against Hemolysis". Open Biology Journal 5, № 1 (2012): 1–5. http://dx.doi.org/10.2174/1874196701205010001.

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The practical importance for the pharmaceutical and cosmetics industries of the interactions between biological membranes and surfactant molecules has led to intensive research within this area. The interactions of non-ionic surfactant n-octyl-β-D-glucopyranoside (OG) with the human and rat erythrocyte membranes were studied. The in vitro hemolytic and antihemolytic activities were determined by employing a method in which both erythrocytes were added to the hypotonic medium containing OG at different concentrations, and the amount of haemoglobin released was determined. noctyl- β-D-glucopyran

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Meng, Qinglei, Zhenmin Bao, Zhaoping Wang, et al. "Growth and Reproductive Performance of Triploid Yesso Scallops (Patinopecten yessoensis) Induced by Hypotonic Shock." Journal of Shellfish Research 31, no. 4 (2012): 1113–22. http://dx.doi.org/10.2983/035.031.0422.

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