Relevant bibliographies by topics / Hypoxic-ischemic injury

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Hypoxic-ischemic injury'

Author: Grafiati
Published: 28 May 2022
Last updated: 30 July 2025

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Journal articles on the topic "Hypoxic-ischemic injury"

1

Biagas, Katherine. "Hypoxic-ischemic brain injury." Current Opinion in Pediatrics 11, no. 3 (1999): 223–28. http://dx.doi.org/10.1097/00008480-199906000-00009.

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Xu, Xiaowen, Xinxin Wang, Li Zhang, et al. "Nicotinamide adenine dinucleotide treatment confers resistance to neonatal ischemia and hypoxia: effects on neurobehavioral phenotypes." Neural Regeneration Research 19, no. 12 (2024): 2760–72. http://dx.doi.org/10.4103/nrr.nrr-d-23-01490.

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JOURNAL/nrgr/04.03/01300535-202412000-00031/figure1/v/2024-04-08T165401Z/r/image-tiff Neonatal hypoxic-ischemic brain injury is the main cause of hypoxic-ischemic encephalopathy and cerebral palsy. Currently, there are few effective clinical treatments for neonatal hypoxic-ischemic brain injury. Here, we investigated the neuroprotective and molecular mechanisms of exogenous nicotinamide adenine dinucleotide, which can protect against hypoxic injury in adulthood, in a mouse model of neonatal hypoxic-ischemic brain injury. In this study, nicotinamide adenine dinucleotide (5 mg/kg) was intraperit
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Jha, Ruchira M., Aaron L. Berkowitz, and Joshua P. Klein. "Evolution of Hypoxic–Ischemic Injury." Neurohospitalist 3, no. 1 (2012): 46. http://dx.doi.org/10.1177/1941874412467807.

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Roland, Elke H., Kenneth Poskitt, Estela Rodriguez, Brian A. Lupton, and Alan Hill. "Perinatal Hypoxic-Ischemic Thalamic Injury." Obstetrical & Gynecological Survey 54, no. 3 (1999): 166–68. http://dx.doi.org/10.1097/00006254-199903000-00011.

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Boggio, Paulo Sérgio, Elizeu Coutinho de Macedo, Alvaro Pascual-Leone, Jose Maria Tormos Muñoz, José Salomáo SchWartzman, and Felipe Fregni. "Neuromodulation in hypoxic-ischemic injury." Brain Stimulation 2, no. 3 (2009): 179–81. http://dx.doi.org/10.1016/j.brs.2008.12.002.

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Povroznik, Jessica M., Elizabeth B. Engler-Chiurazzi, Tania Nanavati, and Paola Pergami. "Absolute lymphocyte and neutrophil counts in neonatal ischemic brain injury." SAGE Open Medicine 6 (January 1, 2018): 205031211775261. http://dx.doi.org/10.1177/2050312117752613.

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Objectives: This study aimed to identify differences in absolute neutrophils, lymphocytes, and neutrophil-to-lymphocyte ratio between neonates with two forms of ischemic brain injury, hypoxic-ischemic encephalopathy, and acute ischemic stroke, compared to controls. We also aimed to determine whether this neutrophil/lymphocyte response pattern is associated with disease severity or is a consequence of the effects of total-body cooling, an approved treatment for moderate-to-severe hypoxic-ischemic encephalopathy. Methods: A retrospective chart review of 101 neonates with hypoxic-ischemic encepha
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Davydenko, A. V. "GENE POLYMORPHISM AS A PREDICTOR DEVELOPMENT OF THE CHILDREN DISEASE." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 22, no. 3-4 (2022): 225–30. http://dx.doi.org/10.31718/2077-1096.22.3.4.225.

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Neonates suffering from severe birth asphyxia may develop hypoxic ischemic encephalopathy and in some cases to permanent neurological damage. Around 20 – 50% of neonates with birth asphyxia who have hypoxic ischemic encephalopathy symptoms die in the neonatal period. Our study aims to clarify the role and impact of gene polymorphisms on the occurrence of hypoxic-ischemic encephalopathy. Hypoxic-ischaemic encephalopathy is a common cause of death and disability in newborns. It causes long-term or permanent damage, such as cerebral palsy, epilepsy, and certain forms of mental retardation. Autore
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Lai, Ming-Chi, and San-Nan Yang. "Perinatal Hypoxic-Ischemic Encephalopathy." Journal of Biomedicine and Biotechnology 2011 (2011): 1–6. http://dx.doi.org/10.1155/2011/609813.

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Perinatal hypoxic-ischemic encephalopathy (HIE) is an important cause of brain injury in the newborn and can result in long-term devastating consequences. Perinatal hypoxia is a vital cause of long-term neurologic complications varying from mild behavioural deficits to severe seizure, mental retardation, and/or cerebral palsy in the newborn. In the mammalian developing brain, ongoing research into pathophysiological mechanism of neuronal injury and therapeutic strategy after perinatal hypoxia is still limited. With the advent of promising therapy of hypothermia in HIE, this paper reviews the p
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Zhang, Ruilan, Zhenggang Zhang, and Michael Chopp. "Function of neural stem cells in ischemic brain repair processes." Journal of Cerebral Blood Flow & Metabolism 36, no. 12 (2016): 2034–43. http://dx.doi.org/10.1177/0271678x16674487.

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Hypoxic/ischemic injury is the single most important cause of disabilities in infants, while stroke remains a leading cause of morbidity in children and adults around the world. The injured brain has limited repair capacity, and thereby only modest improvement of neurological function is evident post injury. In rodents, embryonic neural stem cells in the ventricular zone generate cortical neurons, and adult neural stem cells in the ventricular–subventricular zone of the lateral ventricle produce new neurons through animal life. In addition to generation of new neurons, neural stem cells contri
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Carroll, J. "Stem cells for neonatal hypoxic-ischemic injury." Cell and Organ Transplantology 1, no. 1 (2013): 10–13. http://dx.doi.org/10.22494/cot.v1i1.44.

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Many types of adult stem cells have been used in pre-clinical situations to treat experimental hypoxic-ischemic (HI) injury in neonatal animals. Numerous laboratory reports have appeared in the literature indicating that this treatment is beneficial, and the route of cell administration does not appear to be critical. The success of treatment occurs with administration soon after the injury, and this early administration of the cells proximate to the time of injury appears to be decisive. The mechanism of benefit relates to preservation of intrinsic neurons at the site of injury rather than ce
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Dissertations / Theses on the topic "Hypoxic-ischemic injury"

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Crooks, Suzanne. "Long-term neuropsychological and psychosocial outcomes of hypoxic-ischemic brain injury." Thesis, Queen's University Belfast, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.676279.

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Purpose: This study aimed to identify the long-term cognitive impairments arising from Hypoxic Ischaemic Brian Injury (HIBI) in a group of patients at least one year post insult. The long term psycho-social impact and carer burden associated with HIBI were also investigated. Method: A case series design was employed to facilitate detailed analysis of individual profiles. A battery of neuropsychological measures was administered to all participants. Cognitive domains assessed included premorbid and current IQ, visual and auditory recognition, immediate and delayed recall, auditory and visual at
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Dobrucki, Wawrzyniec L. "Nitrogen and Oxygen Radicals in Ischemic and Hypoxic Injury of the Brain." Ohio University / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1057171850.

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Widerøe, Marius. "Magnetic Resonance Imaging of Hypoxic-Ischemic Brain Injury Development in the Newborn Rat." Doctoral thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for laboratoriemedisin, barne- og kvinnesykdommer, 2012. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-17210.

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Leonardo, Christopher C. "The Role of Extracellular Matrix and Matrix-Degrading Proteases in Neonatal Hypoxic-Ischemic Injury." [Tampa, Fla] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002587.

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Petrashenko, Viktoriia Oleksandrivna, D. Alper, Вікторія Олександрівна Петрашенко, and Виктория Александровна Петрашенко. "Diagnostical markers of perinatal hypoxic and ischemic injury of central nervous system at premature newborns." Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27542.

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Петрашенко, Вікторія Олександрівна, Виктория Александровна Петрашенко та Viktoriia Oleksandrivna Petrashenko. "Нейроспецифічна енолаза - ранній маркер тяжкості гіпоксично-ішемічного ураження ЦНС у недоношених новонароджених". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27453.

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Редько, Олена Костянтинівна, Елена Константиновна Редько, Olena Kostiantynivna Redko, А. Сичона та А. Салех. "Комплексная терапия церебральной ишемии у доношенных новорожденных и кортексин". Thesis, Издательство СумГУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27461.

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Включение кортексина в комплексную терапию гипоксически- ишемического поражения ЦНС у доношенных новорожденных позволяет быстрее восстановить соотношение процессов возбуждения и торможения, нормализовать неврологический статус и снизить сроки госпитализации. При цитировании документа, используйте ссылку http://essuir.sumdu.edu.ua/handle/123456789/27461
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Ranasinghe, Himani Sumudumalee. "Mechanisms underlying hypoxic ischemic injury to the developing brain: The significance of matrix metalloproteinase 2 and 9." Thesis, University of Auckland, 2009. http://hdl.handle.net/2292/4962.

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Perinatal hypoxic ischemic (HI) injury is a leading cause of long-term neurological complications in newborn babies. Matrix metalloproteinases (MMPs) are a family of endopeptidases that are capable of degrading the extracellular matrix (ECM) components. They are considered to be integral in many physiological processes. However, recently it has been demonstrated that the inappropriate activity of these proteases, particularly MMP-2 and 9, contribute to the pathogenesis of cerebral ischemia in the adult brain. Given that ECM disruption is frequently observed following injury to the developing
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Wang, Yanxin, and 王燕欣. "Hypoxic-ischemic injury in the neonatal rat model: prediction of irreversible infarction size by DiffusionWeighted MR Imaging." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B35757577.

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Riparini, Giulia. "Perinatal Brain Injury: Mechanism and potential pharmacological therapies - The role of SIRT1 in the neuroprotective effect of Melatonin and of Endothelin-1 in Oligodendrocyte Progenitor Cell Development." Doctoral thesis, Urbino, 2018. http://hdl.handle.net/11576/2663578.

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Books on the topic "Hypoxic-ischemic injury"

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Kuan, Chia-Yi. Neural Stem Cells in Hypoxic-Ischemic And Hemorrhagic Brain Injury in Newborns. Morgan & Claypool Life Science Publishers, 2012.

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Chang, Eugene. Neuroprotection for Premature Birth and Neonatal Brain Injury. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0014.

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Preterm birth is associated with increased risk of perinatal brain injury. Although there has been little headway made in reducing preterm birth rates, survival of infants born prematurely has improved greatly. Because of this, the neurodevelopmental consequences related to prematurity have become significant issues, especially in those infants born at less than 32 weeks gestation. Hypoxic-ischemic encephalopathy commonly leads to neonatal brain injury both before and after delivery. While perinatal birth asphyxia accounts for a proportion of neonatal brain injury in neonates younger than 37 w
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Chong, Ji Y., and Michael P. Lerario. Cardiac Arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0028.

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Hypoxic–ischemic brain injury is common following cardiopulmonary arrest and is associated with high rates of mortality and morbidity. Therapeutic hypothermia has been helpful in increasing survival and functional outcomes in these patients. The neurological examination, neuroimaging studies, and ancillary serological and neurophysiological testing can be helpful in prognostication post-arrest.
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Stocchetti, Nino, and Marco Carbonara. Pharmacologic Neuroprotection. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0002.

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Acute cerebral injury sets into motion a cascade of deleterious biochemical events that cause further neuronal damage and amplify deleterious effects. This cascade develops over time and potentially may be attenuated or limited by pharmacologic manipulation. The neuroprotective properties of several molecules have been clearly demonstrated in experimental models of various pathologies. Based on these findings, many promising compounds have been tested in clinical trials. Large randomized controlled trials, however, have repeatedly failed to provide evidence of clinical efficacy. The authors pr
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Shein, Steven L., and Robert S. B. Clark. Neurocritical Care. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199918027.003.0009.

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Brain injury is the most common proximate cause of death in pediatric intensive care units. For children who survive critical illness, long-standing brain damage and residual brain dysfunction can affect quality of life significantly. Therefore, minimizing neurological injury to improve patient outcomes is a priority of neurocritical care. This may be accomplished by implementing specific targeted therapies, avoiding pathophysiological conditions that exacerbate neurological injury, and using a multidisciplinary team that focuses on contemporary care of children with neurological injury and di
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Tymianski, Michael. A study of neuronal Cap2s+ homeostasis: application to the pathophysiology and treatment of early excitotoxic and hypoxic/ischemic neuronal injury in vitro and in vivo. 1994.

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Book chapters on the topic "Hypoxic-ischemic injury"

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Furer, Tzvi, Aaron J. Hauptman, and Lindsey Gurin. "“Depression” After Hypoxic-Ischemic Injury." In Pediatric Neuropsychiatry. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94998-7_4.

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Martinez-Biarge, Miriam, and Frances M. Cowan. "Hypoxic-Ischemic Encephalopathy in Preterm Infants." In Neonatal Brain Injury. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-55972-3_7.

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AbstractHypoxic-ischemic encephalopathy (HIE) in preterm infants is uncommon, though the true incidence is not known for certain. This condition may not be easy to identify, especially in the most immature infants, as clinical signs overlap those of prematurity. Hypothermia is currently not approved for infants born at <35 weeks gestation (GA), and a large clinical trial of therapeutic hypothermia in preterm infants recently completed has not shown benefit. As in term-born infants, MRI in the neonatal period shows the site and severity of hypoxic-ischemic injury well and is one of the best
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Hellickson, Jodi D., and Eelco F. M. Wijdicks. "Hypoxic-Ischemic Injury After Cardiac Arrest." In Neurocritical Care for the Advanced Practice Clinician. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48669-7_17.

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Dugan, Laura L., M. Margarita Behrens, and Sameh S. Ali. "Oxidative Stress in Hypoxic-Ischemic Brain Injury." In Brain Hypoxia and Ischemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-579-8_12.

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Doherty, Dermot R., and James S. Hutchison. "Hypoxic Ischemic Encephalopathy After Cardiorespiratory Arrest." In The Central Nervous System in Pediatric Critical Illness and Injury. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84800-993-6_8.

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Mohammad, Khorshid, Linda S. de Vries, Gerda Meijler, and Frances M. Cowan. "Hypoxic-Ischemic Encephalopathy (HIE) in Term and Near-Term Infants." In Neonatal Brain Injury. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-55972-3_8.

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AbstractHypoxic-ischemic encephalopathy (HIE) remains a major burden for infants, their families, and society all over the world, while the incidence of HIE has fallen slightly in high-income countries (HICs) that is not the situation in many poorer economic environments. Still, in many cases, a clear cause for and the timing of the insult remains unclear. The introduction of therapeutic cooling in HICs for moderate and severe HIE has resulted in better early recognition of encephalopathy, better monitoring with EEG, and an emphasis on optimal early management with early seizure treatments and
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Decker, Robert S., and Marlene L. Decker. "Lysosomal integrity in ischemic, hypoxic and recovering heart." In Pathophysiology of Severe Ischemic Myocardial Injury. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0475-0_7.

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Hayakawa, Masahiro. "Pathophysiology and Pathology of Neonatal Hypoxic-Ischemic Encephalopathy." In Cell Therapy for Perinatal Brain Injury. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-1412-3_3.

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Carroll, James. "Stem Cell Therapy for Neonatal Hypoxic–Ischemic Brain Injury." In Cell Therapy for Brain Injury. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15063-5_16.

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Martini, Silvia, Roberta Parladori, and Luigi Corvaglia. "Biomarkers of Oxidative Stress in Neonatal Hypoxic-Ischemic Encephalopathy." In Biomarkers in Trauma, Injury and Critical Care. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-07395-3_12.

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Conference papers on the topic "Hypoxic-ischemic injury"

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Wu, Dan, Anastassios Bezerianos, David Sherman, Xiaofeng Jia, and Nitish V. Thakor. "Causal interactions between thalamic and cortical LFPs following hypoxic-ischemic brain injury." In 5th International IEEE/EMBS Conference on Neural Engineering (NER 2011). IEEE, 2011. http://dx.doi.org/10.1109/ner.2011.5910518.

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Bakalov, V., J. Hertel, A. Ku, and B. E. DiSilvio. "Significant Recovery in Functional Capacity After Hypoxic-Ischemic Brain Injury in Young Female Patient." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6633.

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Hegab, Ahmed E., Vi Luan Ha, Jennifer L. Gilbert, Derek W. Nickerson, Aik Ooi, and Brigitte N. Gomperts. "Repair And Regeneration Of The Tracheal Epithelium And Submucosal Glands After Hypoxic-Ischemic Injury." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5294.

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Chapman, Silena C., Blair Britt, and Maria Dizon. "Hypoxic-ischemic Injury Alters the Expression of Micrornas Known to Regulate Oligodendrocyte Progenitor Cell Differentiation." In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.747.

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Chen, Cheng, Anil Maybhate, and Nitish V. Thakor. "Granger causality analysis reveals the changes of thalamocortical functionality after cardiac arrest induced hypoxic-ischemic injury." In 2012 38th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2012. http://dx.doi.org/10.1109/nebc.2012.6206963.

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Dandan Zhang, M. Hathi, Zeng-Jin Yang, Haiyan Ding, R. Koehler, and N. Thakor. "Hypoxic-ischemic brain injury in neonatal piglets with different histological outcomes: An amplitude-integrated EEG study." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5333439.

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Mauro, I., A. Franz, E. Baraldi, et al. "The Albino Trail Effect of Allopurinol in Addition to Hypothermia for Hypoxic-Ischemic Brain Injury on Neurocognitive Outcome." In 7th International Conference on Clinical Neonatology—Selected Abstracts. Thieme Medical Publishers, 2018. http://dx.doi.org/10.1055/s-0038-1647082.

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Yang, Jian, and Ed X. Wu. "Manganese-enhanced MRI detected the gray matter lesions in the late phase of mild hypoxic-ischemic injury in neonatal rat." In 2007 29th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2007. http://dx.doi.org/10.1109/iembs.2007.4352220.

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Milanish, Yulia. "OPPORTUNITIES OF RATIONAL EMOTIVE BEHAVIOR THERAPY IN WORKING WITH MOTHERS OF NEWBORNS WITH INTRAVENTRICULAR HEMORRHAGE AND HYPOXIC-ISCHEMIC CENTRAL NERVOUS SYSTEM INJURY." In 4th SGEM International Multidisciplinary Scientific Conferences on SOCIAL SCIENCES and ARTS Proceedings. STEF92 Technology, 2017. http://dx.doi.org/10.5593/sgemsocial2017/32/s11.030.

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