Academic literature on the topic 'Idiopathic Parkinson's syndrome'

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Journal articles on the topic "Idiopathic Parkinson's syndrome"

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Koschel, Jiri, Andreas Fleiner, and Wolfgang H. Jost. "Smoking behavior and idiopathic Parkinson's syndrome." Basal Ganglia 8 (May 2017): 5. http://dx.doi.org/10.1016/j.baga.2017.02.016.

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Raghavan, Ravi, Clare Khin-Nu, Andrew Brown, et al. "Detection of Lewy Bodies in Trisomy 21 (Down's Syndrome)." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 20, no. 1 (1993): 48–51. http://dx.doi.org/10.1017/s0317167100047405.

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ABSTRACT:The presence of cortical senile plaques and neurofibrillary tangles sufficient to warrant a neuropatho-logical diagnosis of Alzheimer's disease is well established in middle-aged individuals with Trisomy 21 (Down's syndrome). In contrast a relationship between Down's syndrome and Lewy bodies, one of the major neuropathological features of Parkinson's disease, has not been previously reported. In a cliniconeuropathological survey of 23 cases of Down's Syndrome, two patients, aged 50 and 56 years respectively, were found to have Lewy body formation in the substantia nigra in addition to
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Kim, Jong Hyun, Taek-Hyun Kwon, Seong-Beom Koh, and Jung Youl Park. "Parkinsonism-Hyperpyrexia Syndrome After Deep Brain Stimulation Surgery." Neurosurgery 66, no. 5 (2010): E1029. http://dx.doi.org/10.1227/01.neu.0000367799.38332.43.

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Abstract OBJECTIVE Deep brain stimulation is an alternative treatment for advanced Parkinson's disease. Levodopa medications are usually discontinued the night before surgery to localize the optimal response site to intraoperative macrostimulation. However, abrupt withdrawal of medication may result in side effects. We report a case of parkinsonism-hyperpyrexia syndrome (PHS), a rare complication resulting from discontinuation of antiparkinsonian medication, after a deep brain stimulation (DBS) procedure for bilateral subthalamic-nucleus (STN). CLINICAL PRESENTATION A 66-year-old woman with an
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Bravo-San Pedro, José M., Rubén Gómez-Sánchez, Elisa Pizarro-Estrella, Mireia Niso-Santano, Rosa A. González-Polo, and José M. Fuentes Rodríguez. "Parkinson's Disease: Leucine-Rich Repeat Kinase 2 and Autophagy, Intimate Enemies." Parkinson's Disease 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/151039.

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Parkinson's disease is the second common neurodegenerative disorder, after Alzheimer's disease. It is a clinical syndrome characterized by loss of dopamine-generating cells in the substancia nigra, a region of the midbrain. The etiology of Parkinson's disease has long been through to involve both genetic and environmental factors. Mutations in the leucine-rich repeat kinase 2 gene cause late-onset Parkinson's disease with a clinical appearance indistinguishable from Parkinson's disease idiopathic. Autophagy is an intracellular catabolic mechanism whereby a cell recycles or degrades damage prot
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Pinkhardt, Elmar H., Reinhart Jürgens, Wolfgang Becker, Federica Valdarno, Albert C. Ludolph, and Jan Kassubek. "Differential diagnostic value of eye movement recording in PSP-parkinsonism, Richardson's syndrome, and idiopathic Parkinson's disease." Journal of Neurology 255, no. 12 (2008): 1916–25. http://dx.doi.org/10.1007/s00415-009-0027-y.

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Crowe, Simon F., Richard F. Peppard, Ruth Borenstein, and John H. Lloyd. "Diffuse Lewy Body Disease and Progressive Dementia in a Young Woman." Australian & New Zealand Journal of Psychiatry 26, no. 3 (1992): 507–11. http://dx.doi.org/10.3109/00048679209072078.

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This report details the emergence of a progressive parkinsonian syndrome, dementia and behavioural disturbance in a 33 year-old woman which can be dated to the delivery of her first child. The findings of this case indicate that cortical Lewy body disease should be considered in any patient with temporoparietal dementia and idiopathic Parkinson's disease irrespective of the age of onset.
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Kyrtsos, Christina Rose, Mark C. Stahl, Paul Eslinger, Thyagarajan Subramanian, and Elisabeth B. Lucassen. "Capgras Syndrome in a Patient with Parkinson's Disease after Bilateral Subthalamic Nucleus Deep Brain Stimulation: A Case Report." Case Reports in Neurology 7, no. 2 (2015): 127–33. http://dx.doi.org/10.1159/000431081.

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Capgras syndrome is a delusional misidentification syndrome (DMS) which can be seen in neurodegenerative diseases such as Lewy body dementia and, to a lesser extent, in Parkinson's disease (PD). Here, we report the case of a 78-year-old man with a history of idiopathic PD who developed Capgras syndrome following bilateral subthalamic nucleus deep brain stimulation (DBS) implantation. As the risk of DMS has been related to deficits in executive, memory, and visuospatial function preoperatively, this case highlights the importance of continuing to improve patient selection for DBS surgery. Capgr
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Naidu, Y., V. Metta, S. Tluk, J. Roomi, O. Bernadette, and K. R. Chaudhuri. "1.284 Daytime fatigue in idiopathic restless legs syndrome: Evaluation with Parkinson's fatigue scale." Parkinsonism & Related Disorders 13 (January 2007): S72. http://dx.doi.org/10.1016/s1353-8020(08)70519-4.

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Etoom, Mohammad, Mohammad Alwardat, Ala’ S. Aburub, et al. "Therapeutic interventions for Pisa syndrome in idiopathic Parkinson's disease. A Scoping Systematic Review." Clinical Neurology and Neurosurgery 198 (November 2020): 106242. http://dx.doi.org/10.1016/j.clineuro.2020.106242.

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Hossein Tabatabaei, Seyed Amir, David Pedrosa, Carsten Eggers, et al. "Machine Learning Techniques for Parkinson’s Disease Detection using Wearables during a Timed-up-and-Go-Test." Current Directions in Biomedical Engineering 6, no. 3 (2020): 376–79. http://dx.doi.org/10.1515/cdbme-2020-3097.

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AbstractIn this paper, the classification models for Idiopathic Parkinson's syndrome (iPS) detection through timed-up-and-go test performed on iPS-patients are given. The models are based on the supervised learning. The data are extracted via Myo gesture armband worn on two hands. The corresponding models are based on extracted features from signal data and raw signal data respectively. The achieved accuracy from both models are 0.91 and 0.93 with reasonable specificity and sensitivity.
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Dissertations / Theses on the topic "Idiopathic Parkinson's syndrome"

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Pantel, Pia Marie. "Innovative MRT-Kontraste zur in-vivo-Differenzierung von Patienten mit typischem idiopathischen Parkinson und atypischen Parkinsonsyndromen." Doctoral thesis, 2013. http://hdl.handle.net/11858/00-1735-0000-0022-60A1-A.

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HINTERGRUND/ ZIELSETZUNG: Vom idiopathischen Parkinsonsyndrom (IPS) können so genannte „atypische“ Parkinsonsyndrome (APS) mit einem Anteil von ca. 20% bezogen auf die Gesamtinzidenz unterschieden werden. Neben zusätzlichen Krankheitssymptomen und einem progredienteren Verlauf zeichnen sie sich durch eine schlechtere Prognose aus, die häufig auf einem Nichtansprechen auf eine dopaminerge Therapie beruht. Eine frühzeitige, korrekte Diagnose ist daher sehr entscheidend, aber im Einzelfall auch für Spezialisten äußerst schwierig. Trotz anerkannter klinischer Diagnosekriterien gibt es besonders i
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Books on the topic "Idiopathic Parkinson's syndrome"

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Silver, Michael, and Stewart A. Factor. History of Parkinsonism. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0001.

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This chapter explores the history of Parkinson’s disease and similar conditions. Dr James Parkinson’s initial description of this syndrome is covered along with all of the major developments in the understanding of the disease since then up to the modern era. Therapies are detailed, from early herbal remedies up to current treatment, including dopamine and levodopa, non-levodopa therapy, and surgery, and glimpses are given of potential future therapies. The conditions that are similar, the so-called “parkinsonian conditions,” are discussed as well. Synonyms: idiopathic Parkinson disease; Parki
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Fletcher, Nicholas. Movement disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0926.

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Almost any neurological disorder can produce a disorder of movement but the ‘movement disorders’ include the akinetic rigid syndromes, hyperkinesias, and some tremors. It can sometimes seem, especially with the use of videotape recordings, that diagnosis of movement disorders is mainly a matter of correct visual recognition. Such an approach is not recommended and can lead to mistakes unless, as in other areas of medicine, the history is considered first and the physical signs second. Obvious examples include the family history in Huntington’s disease, developmental history in dystonic cerebra
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Book chapters on the topic "Idiopathic Parkinson's syndrome"

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Gartler, Stanley M., R. Scott Hansen, Vinzenz Oji, et al. "Idiopathic Parkinson Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_6530.

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Reichmann, Heinz, and Ulrike Sommer. "Dementia in Idiopathic Parkinson’s Syndrome." In Mapping the Progress of Alzheimer’s and Parkinson’s Disease. Springer US, 2002. http://dx.doi.org/10.1007/978-0-306-47593-1_59.

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Earl, C., J. Sautter, W. Oertel, and A. Kupsch. "Neurosurgical and Restorative Possibilities in the Treatment of Idiopathic Parkinson Syndromes: Neurotransplantation and Neurotrophic Factors." In Minimally Invasive Techniques for Neurosurgery. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-58731-3_44.

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van Rheenen, Ronald W. J., Jacoba P. van Amerongen, Sylvia Eshuis, Klaus L. Leenders, Andre Dobbeleir, and Rudi A. J. O. Dierckx. "SPECT Imaging for Idiopatic M. Parkinson and Parkinsonian Syndromes: Guidelines and Comparison with PET and Recent Developments." In PET and SPECT in Neurology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54307-4_27.

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Wilkinson, Ian B., Tim Raine, Kate Wiles, Anna Goodhart, Catriona Hall, and Harriet O’Neill. "Neurology." In Oxford Handbook of Clinical Medicine. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199689903.003.0010.

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This chapter explores neurology, including lesion location, drugs and the nervous system, cerebral blood supply, testing peripheral nerves, dermatomes and peripheral nerves, headache, migraine, blackouts, vertigo and dizziness, hearing loss and tinnitus, acute bilateral leg weakness, abnormal involuntary movements (dyskinesia), stroke, transient ischaemic attack (TIA), subarachnoid haemorrhage (SAH), intracranial venous thrombosis (IVT), subdural haematoma, extradural (epidural) haematoma, delirium (acute confusional state), dementia, Alzheimer’s disease (AD), epilepsy, Parkinsonism, multiple sclerosis (MS), space-occupying lesions (SOL), idiopathic intracranial hypertension, Bell’s palsy, mononeuropathies, polyneuropathies, autonomic neuropathy, motor neuron disease (MND), bulbar palsy, cervical spondylosis, myopathy, myasthenia gravis (MG), Lambert–Eaton myasthenic syndrome (LEMS), neurofibromatosis, syingomyelia, and retroviruses and neurology.
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Bouwmans, Angela E. P., Annemarie M. M. Vlaar, Karin Srulijes, Werner H. Mess, and W. E. J. Weber. "Transcranial Sonography for the Discrimination of Idiopathic Parkinson’s Disease from the Atypical Parkinsonian Syndromes." In International Review of Neurobiology. Elsevier, 2010. http://dx.doi.org/10.1016/s0074-7742(10)90009-3.

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Vedam-Mai, Vinata, Anthony T. Yachnis, Michael Ullman, Saman P. Javedan, and Michael S. Okun. "Fibrous Scarring and Deep Brain Stimulation Lead Implantation." In Deep Brain Stimulation, edited by Laura S. Surillo Dahdah, Padraig O’Suilleabhain, Hrishikesh Dadhich, Mazen Elkurd, Shilpa Chitnis, and Richard B. Dewey. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190647209.003.0028.

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Deep brain stimulation (DBS) has emerged in recent years as a powerful surgical modality for the treatment of several movement disorders as well as neuropsychiatric syndromes. Despite its clinical efficacy, the mode of action of DBS and the cellular responses to this therapy remain unclear. This chapter presents an unusual case from the DBS Brain Tissue Network at the University of Florida of prominent tissue response to DBS. The patient was a 74-year-old man with idiopathic Parkinson disease, status post bilateral STN DBS leads. Neuropathologic examination of the tissue surrounding the distal lead tip of the right DBS using immunohistochemistry revealed a dense, collagen-filled fibrous sheath. There was no intraoperative bleeding during the procedure, and this finding was confirmed by postoperative imaging. Further, there was no perioperative decline in this patient’s condition. It is likely that the fibrous capsule resulted in less than optimal clinical benefit, which was previously reported in this patient. This severe type of exaggerated tissue response to DBS leads has not been observed in other cases in our Brain Bank network and has not been reported previously in the literature, to our knowledge.
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