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Journal articles on the topic 'Idiopathic Parkinson's syndrome'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Koschel, Jiri, Andreas Fleiner, and Wolfgang H. Jost. "Smoking behavior and idiopathic Parkinson's syndrome." Basal Ganglia 8 (May 2017): 5. http://dx.doi.org/10.1016/j.baga.2017.02.016.

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2

Raghavan, Ravi, Clare Khin-Nu, Andrew Brown, et al. "Detection of Lewy Bodies in Trisomy 21 (Down's Syndrome)." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 20, no. 1 (1993): 48–51. http://dx.doi.org/10.1017/s0317167100047405.

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ABSTRACT:The presence of cortical senile plaques and neurofibrillary tangles sufficient to warrant a neuropatho-logical diagnosis of Alzheimer's disease is well established in middle-aged individuals with Trisomy 21 (Down's syndrome). In contrast a relationship between Down's syndrome and Lewy bodies, one of the major neuropathological features of Parkinson's disease, has not been previously reported. In a cliniconeuropathological survey of 23 cases of Down's Syndrome, two patients, aged 50 and 56 years respectively, were found to have Lewy body formation in the substantia nigra in addition to

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3

Kim, Jong Hyun, Taek-Hyun Kwon, Seong-Beom Koh, and Jung Youl Park. "Parkinsonism-Hyperpyrexia Syndrome After Deep Brain Stimulation Surgery." Neurosurgery 66, no. 5 (2010): E1029. http://dx.doi.org/10.1227/01.neu.0000367799.38332.43.

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Abstract OBJECTIVE Deep brain stimulation is an alternative treatment for advanced Parkinson's disease. Levodopa medications are usually discontinued the night before surgery to localize the optimal response site to intraoperative macrostimulation. However, abrupt withdrawal of medication may result in side effects. We report a case of parkinsonism-hyperpyrexia syndrome (PHS), a rare complication resulting from discontinuation of antiparkinsonian medication, after a deep brain stimulation (DBS) procedure for bilateral subthalamic-nucleus (STN). CLINICAL PRESENTATION A 66-year-old woman with an

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4

Bravo-San Pedro, José M., Rubén Gómez-Sánchez, Elisa Pizarro-Estrella, Mireia Niso-Santano, Rosa A. González-Polo, and José M. Fuentes Rodríguez. "Parkinson's Disease: Leucine-Rich Repeat Kinase 2 and Autophagy, Intimate Enemies." Parkinson's Disease 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/151039.

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Parkinson's disease is the second common neurodegenerative disorder, after Alzheimer's disease. It is a clinical syndrome characterized by loss of dopamine-generating cells in the substancia nigra, a region of the midbrain. The etiology of Parkinson's disease has long been through to involve both genetic and environmental factors. Mutations in the leucine-rich repeat kinase 2 gene cause late-onset Parkinson's disease with a clinical appearance indistinguishable from Parkinson's disease idiopathic. Autophagy is an intracellular catabolic mechanism whereby a cell recycles or degrades damage prot

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Pinkhardt, Elmar H., Reinhart Jürgens, Wolfgang Becker, Federica Valdarno, Albert C. Ludolph, and Jan Kassubek. "Differential diagnostic value of eye movement recording in PSP-parkinsonism, Richardson's syndrome, and idiopathic Parkinson's disease." Journal of Neurology 255, no. 12 (2008): 1916–25. http://dx.doi.org/10.1007/s00415-009-0027-y.

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6

Crowe, Simon F., Richard F. Peppard, Ruth Borenstein, and John H. Lloyd. "Diffuse Lewy Body Disease and Progressive Dementia in a Young Woman." Australian & New Zealand Journal of Psychiatry 26, no. 3 (1992): 507–11. http://dx.doi.org/10.3109/00048679209072078.

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This report details the emergence of a progressive parkinsonian syndrome, dementia and behavioural disturbance in a 33 year-old woman which can be dated to the delivery of her first child. The findings of this case indicate that cortical Lewy body disease should be considered in any patient with temporoparietal dementia and idiopathic Parkinson's disease irrespective of the age of onset.

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Kyrtsos, Christina Rose, Mark C. Stahl, Paul Eslinger, Thyagarajan Subramanian, and Elisabeth B. Lucassen. "Capgras Syndrome in a Patient with Parkinson's Disease after Bilateral Subthalamic Nucleus Deep Brain Stimulation: A Case Report." Case Reports in Neurology 7, no. 2 (2015): 127–33. http://dx.doi.org/10.1159/000431081.

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Capgras syndrome is a delusional misidentification syndrome (DMS) which can be seen in neurodegenerative diseases such as Lewy body dementia and, to a lesser extent, in Parkinson's disease (PD). Here, we report the case of a 78-year-old man with a history of idiopathic PD who developed Capgras syndrome following bilateral subthalamic nucleus deep brain stimulation (DBS) implantation. As the risk of DMS has been related to deficits in executive, memory, and visuospatial function preoperatively, this case highlights the importance of continuing to improve patient selection for DBS surgery. Capgr

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8

Naidu, Y., V. Metta, S. Tluk, J. Roomi, O. Bernadette, and K. R. Chaudhuri. "1.284 Daytime fatigue in idiopathic restless legs syndrome: Evaluation with Parkinson's fatigue scale." Parkinsonism & Related Disorders 13 (January 2007): S72. http://dx.doi.org/10.1016/s1353-8020(08)70519-4.

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9

Etoom, Mohammad, Mohammad Alwardat, Ala’ S. Aburub, et al. "Therapeutic interventions for Pisa syndrome in idiopathic Parkinson's disease. A Scoping Systematic Review." Clinical Neurology and Neurosurgery 198 (November 2020): 106242. http://dx.doi.org/10.1016/j.clineuro.2020.106242.

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10

Hossein Tabatabaei, Seyed Amir, David Pedrosa, Carsten Eggers, et al. "Machine Learning Techniques for Parkinson’s Disease Detection using Wearables during a Timed-up-and-Go-Test." Current Directions in Biomedical Engineering 6, no. 3 (2020): 376–79. http://dx.doi.org/10.1515/cdbme-2020-3097.

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AbstractIn this paper, the classification models for Idiopathic Parkinson's syndrome (iPS) detection through timed-up-and-go test performed on iPS-patients are given. The models are based on the supervised learning. The data are extracted via Myo gesture armband worn on two hands. The corresponding models are based on extracted features from signal data and raw signal data respectively. The achieved accuracy from both models are 0.91 and 0.93 with reasonable specificity and sensitivity.

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11

Markopoulou, K., K. W. Larsen, E. K. Wszolek, et al. "Olfactory dysfunction in familial parkinsonism." Neurology 49, no. 5 (1997): 1262–67. http://dx.doi.org/10.1212/wnl.49.5.1262.

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Impaired olfactory function is commonly observed in idiopathic Parkinson's disease (IPD). However, it is unknown whether it is also found in familial parkinsonism. To address this issue we administered a smell test to 12 affected, three monosymptomatic, and 12 at-risk individuals from six large parkinsonian kindreds. Three kindreds exhibited an IPD phenotype and three exhibited a parkinsonism-plus syndrome (PPS) phenotype. All but one of the affected individuals had impaired olfactory function. In contrast, only five of the 12 at-risk individuals had impaired olfactory function. The degree of

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12

Zhu, Xiao-Ying, Ye Liu, Xiao-Jin Zhang, et al. "Clinical characteristics of leg restlessness in Parkinson's disease compared with idiopathic Restless Legs Syndrome." Journal of the Neurological Sciences 357, no. 1-2 (2015): 109–14. http://dx.doi.org/10.1016/j.jns.2015.07.008.

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13

Bellon, A. K., W. H. Jost, and K. Schimrigk. "Is cardiovascular dysregulation in idiopathic Parkinson's syndrome correlated with decreased hormonal release?; TO THE EDITOR." European Journal of Neurology 4, no. 2 (1997): 199–200. http://dx.doi.org/10.1111/j.1468-1331.1997.tb00331.x.

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14

Boczarska-Jedynak, M., M. Sałata, B. Jasińska-Myga, and G. Opala. "P3.176 Idiopathic restless legs syndrome in Parkinson's disease – a risk factor or a chance association?" Parkinsonism & Related Disorders 15 (December 2009): S193. http://dx.doi.org/10.1016/s1353-8020(09)70740-0.

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15

Dragan, Elizabeth M., Zhongxue Chen, and William G. Ondo. "Does idiopathic restless legs syndrome delay onset and reduce severity of Parkinson's disease: a pilot study." International Journal of Neuroscience 125, no. 7 (2014): 526–30. http://dx.doi.org/10.3109/00207454.2014.987771.

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16

Ekmekci, H., E. Kaplan, and S. Ozturk. "The comparison of sleep structure, REM period features and tonus in between idiopathic Parkinson's disease and Parkinson's plus syndrome patients with healthy subjects." Sleep Medicine 16 (December 2015): S348—S349. http://dx.doi.org/10.1016/j.sleep.2015.02.455.

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17

Zhao, P., B. Zhang, and S. Gao. "2.141 THE 18[F]-FDG PET STUDY ON THE IDIOPATHIC PARKINSON'S DISEASE AND SEVERAL PARKINSONIAN-PLUS SYNDROME." Parkinsonism & Related Disorders 18 (January 2012): S112. http://dx.doi.org/10.1016/s1353-8020(11)70513-2.

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18

Valko, P. O., S. Hauser, E. Werth, D. Waldvogel, and C. R. Baumann. "1.036 HEART RATE VARIABILITY IN PATIENTS WITH IDIOPATHIC PARKINSON'S DISEASE WITH AND WITHOUT OBSTRUCTIVE SLEEP APNEA SYNDROME." Parkinsonism & Related Disorders 18 (January 2012): S19. http://dx.doi.org/10.1016/s1353-8020(11)70150-x.

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19

Schulz, J�rg B., Martin Skalej, Dirk Wedekind, et al. "Magnetic resonance imaging-based volumetry differentiates idiopathic Parkinson's syndrome from multiple system atrophy and progressive supranuclear palsy." Annals of Neurology 45, no. 1 (1999): 65–74. http://dx.doi.org/10.1002/1531-8249(199901)45:1<65::aid-art12>3.0.co;2-1.

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20

Castiglioni, Emanuela, Dario Finazzi, Stefano Goldwurm, et al. "Analysis of Nucleotide Variations in Genes of Iron Management in Patients of Parkinson's Disease and Other Movement Disorders." Parkinson's Disease 2011 (2011): 1–6. http://dx.doi.org/10.4061/2011/827693.

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The capacity to act as an electron donor and acceptor makes iron an essential cofactor of many vital processes. Its balance in the body has to be tightly regulated since its excess can be harmful by favouring oxidative damage, while its deficiency can impair fundamental activities like erythropoiesis. In the brain, an accumulation of iron or an increase in its availability has been associated with the development and/or progression of different degenerative processes, including Parkinson's disease, while iron paucity seems to be associated with cognitive deficits, motor dysfunction, and restle

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21

Iacono, Robert P., Jonathan D. Carlson, Sandra Kuniyoshi, Aymen Mohamed, Christine Meltzer, and Shokei Yamada. "Contemporaneous bilateral pallidotomy." Neurosurgical Focus 2, no. 3 (1997): E7. http://dx.doi.org/10.3171/foc.1997.2.3.8.

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To investigate the effects of bilateral posteroventral pallidotomy (PVP) for Parkinson's disease (PD), the authors reviewed the technique and neurological outcome in a number of patients who had undergone bilateral pallidotomy. The authors have previously reported the outcome of PVP in 25 bilateral and 25 unilateral surgeries, rating the patient's postural stability, speech, and total Unified Parkinson's Disease Rating Scale scores. A second, separate group of 23 patients who underwent contemporaneous bilateral PVP were evaluated for early onset idiopathic PD, levodopa failure syndrome, and se

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22

Gibb, W. R. G., and A. J. Lees. "The progression of idiopathic Parkinson's disease is not explained by age-related changes. Clinical and pathological comparisons with post-encephalitic parkinsonian syndrome." Acta Neuropathologica 73, no. 2 (1987): 195–201. http://dx.doi.org/10.1007/bf00693789.

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23

Rajput, A. H., B. Rozdilsky, and Alex Rajput. "Accuracy of Clinical Diagnosis in Parkinsonism — A Prospective Study." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 18, no. 3 (1991): 275–78. http://dx.doi.org/10.1017/s0317167100031814.

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ABSTRACT:Clinical diagnosis of Parkinson's syndrome (PS) is reasonably easy in most cases but the distinction between different variants of PS may be difficult in early cases. The correct diagnosis is not only important for counselling and management of patients but also in conducting pharmacological and epidemiological studies. There is very little critical literature on the pathological verification of the clinical diagnosis in PS. We report our 22 year experience to address that issue. Between 1968 and 1990, 65 PS patients came to autopsy. Complete data are available in 59 (M- 50, F-19) cas

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24

van Eimeren, Thilo, Joseph Claßen, Alexander Drzezga, et al. "Empfehlung zum differenzierten Einsatz nuklearmedizinischer Diagnostik bei Parkinson-Syndromen." Fortschritte der Neurologie · Psychiatrie 88, no. 09 (2020): 609–19. http://dx.doi.org/10.1055/a-1207-0515.

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ZusammenfassungDie vorliegende Arbeit gibt einen Überblick über die verschiedenen nuklearmedizinischen Verfahren in der Diagnostik bei neurodegenerativen Parkinson-Syndromen sowie ihre Evidenzlage und soll praxistaugliche Entscheidungshilfen in der Anwendung und Interpretation der Methoden und Befunde ermöglichen. Die Wertigkeit der Verfahren unterscheidet sich erheblich in Bezug auf die beiden relevanten diagnostischen Fragestellungen. Dies ist zum einen die Frage, ob überhaupt ein neurodegeneratives Parkinson-Syndrom vorliegt, zum anderen die Frage, welches. Während zur Beantwortung der erst

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25

Eggers, C., A. Holstein, C. Schneider, et al. "123I-FP-CIT SPECT imaging of the dopaminergic state." Nuklearmedizin 51, no. 06 (2012): 244–51. http://dx.doi.org/10.3413/nukmed-0449-11-12.

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Summary123I-N-ω-fluoropropyl-2β-carbomethoxy-3β- (4-iodophenyl)nortropan (123I-FP-CIT) single photon emission computed tomography (SPECT) can be evaluated by both visual assessment and quantitative analysis to assess the striatal dopamine state in vivo. The aim of our study was to investigate if visual assessment according to a predefined image grading scale reflects the results of quantitative assessment techniques. Patients, methods: 195 patients with a clinical diagnosis of idiopathic Parkinson's disease (n = 134), atypical parkinsonian syndrome (n = 47) or essential tremor (n = 14) were ex

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26

de Keyser, J., L. Herroelen, M. Vervaeck, M. Bruyland, and G. Ebinger. "Idiopathic hemiparetic parkinsonism, a syndrome distinct from idiopathic parkinsonism." Lancet 339, no. 8786 (1992): 149–50. http://dx.doi.org/10.1016/0140-6736(92)90213-m.

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27

Jenkins, Mary, Dan Mendonca, Andrew Parrent, and Mandar S. Jog. "Hemiparkinsonism-Somatic Hemiatrophy Syndrome." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 29, no. 2 (2002): 184–87. http://dx.doi.org/10.1017/s0317167100120992.

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Abstract:Purpose:This paper highlights the hemiparkinsonism-hemiatrophy (HPHA) syndrome as a unique presentation of the parkinsonian state. Clinically relevant diagnostic and treatment aspects are reviewed.Method:We report a case of HPHA, in a 21-year-old, otherwise healthy, woman. Clinical and radiographic features of our case are presented. We also review the current literature on the clinical, radiological and pathophysiological mechanisms of HPHA.Results:In our case, despite the lack of benefit from anticholinergics and dopamine agonists (the patient declined treatment with levodopa), the

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Sebastian, Roshan, William K. Gray, Aishling Foley, et al. "What Predicts Hospital Admissions in Community-Dwelling People With Parkinsonism?" Journal of Geriatric Psychiatry and Neurology 33, no. 2 (2019): 103–8. http://dx.doi.org/10.1177/0891988719868309.

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Objectives: Previous studies have looked at the reasons for hospital admission in people with parkinsonism (PwP), yet few have looked at factors that precipitate admission. Methods: People with parkinsonism with a diagnosis of idiopathic Parkinson disease of Hoehn and Yahr stage III-V and those with Parkinson plus syndromes were assessed for motor and nonmotor symptoms, quality of life, and functional performance. Logistic regression was used to investigate predictors of hospital admission over the subsequent 2 years. Results: Overall, 162 patients consented to be part of the study. Seventy-on

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Anderson, Richard C., Jessica J. Grant, Robert de la Paz, Steven Frucht, and Robert R. Goodman. "Volumetric measurements in the detection of reduced ventricular volume in patients with normal-pressure hydrocephalus whose clinical condition improved after ventriculoperitoneal shunt placement." Journal of Neurosurgery 97, no. 1 (2002): 73–79. http://dx.doi.org/10.3171/jns.2002.97.1.0073.

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Object. The syndrome of normal-pressure hydrocephalus (NPH) refers to the clinical triad of gait disturbance, dementia, and urinary incontinence in association with idiopathic ventriculomegaly and normal intracranial pressure. Ventriculoperitoneal (VP) shunt placement often yields significant clinical improvements, sometimes without apparent reduction of ventricular size. The authors hypothesized that careful volumetric measurements would show a decrease in ventricular volume in these patients. Methods. Twenty consecutive patients with NPH underwent placement of VP shunts equipped with program

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Fereshtehnejad, Seyed-Mohammad, and Johan Lökk. "Orthostatic Hypotension in Patients with Parkinson’s Disease and Atypical Parkinsonism." Parkinson's Disease 2014 (2014): 1–10. http://dx.doi.org/10.1155/2014/475854.

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Orthostatic hypotension (OH) is one of the commonly occurring nonmotor symptoms in patients with idiopathic Parkinson’s disease (IPD) and atypical parkinsonism (AP). We aimed to review current evidences on epidemiology, diagnosis, treatment, and prognosis of OH in patients with IPD and AP. Major electronic medical databases were assessed including PubMed/MEDLINE and Embase up to February 2013. English-written original or review articles with keywords such as “Parkinson’s disease,” “atypical parkinsonism,” and “orthostatic hypotension” were searched for relevant evidences. We addressed differen

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Coppedè, Fabio. "Genetics and Epigenetics of Parkinson's Disease." Scientific World Journal 2012 (2012): 1–12. http://dx.doi.org/10.1100/2012/489830.

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In 1997 a mutation in thea-synuclein(SNCA) gene was associated with familial autosomal dominant Parkinson’s disease (PD). Since then, several loci (PARK1-15) and genes have been linked to familial forms of the disease. There is now sufficient evidence that six of the so far identified genes at PARK loci (a-synuclein, leucine-rich repeat kinase 2, parkin, PTEN-induced putative kinase 1, DJ-1, andATP13A2) cause inherited forms of typical PD or parkinsonian syndromes. Other genes at non-PARK loci (MAPT, SCA1, SCA2, spatacsin, POLG1) cause syndromes with parkinsonism as one of the symptoms. The ma

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32

Berger, L., S. Gauthier, and R. Leblanc. "Akinetic Mutism and Parkinsonism Associated with Obstructive Hydrocephalus." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 12, no. 3 (1985): 255–58. http://dx.doi.org/10.1017/s0317167100047119.

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ABSTRACT:We report the case of a patient with idiopathic aqueductal stenosis and hydrocephalus who had several episodes of akinetic mutism, each preceded by shunt malfunction, that resolved with shunt revision. She also developed a parkinson’s syndrome resistant to shunt revision but responsive to antiparkinsonian medications. The Parkinson’s syndrome and the episodes of akinetic mutism may be related to a reduction of dopaminergic input to the striatum and to the cingulate and frontal cortex brought about or worsened by ventricular dilatation.

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Bor-Seng-Shu, Edson, José Luiz Pedroso, Daniel Ciampi de Andrade, et al. "Transcranial sonography in Parkinson's disease." Einstein (São Paulo) 10, no. 2 (2012): 242–46. http://dx.doi.org/10.1590/s1679-45082012000200022.

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Transcranial sonography has become a useful tool in the differential diagnosis of parkinsonian syndromes. This is a non-invasive, low cost procedure. The main finding on transcranial sonography in patients with idiopathic Parkinson's disease is an increased echogenicity of the mesencephalic substantia nigra region. This hyperechogenicity is present in more than 90% of cases, and reflects a dysfunction in the dopaminergic nigrostriatal pathway. This study discussed how the hyperechogenicity of the substantia nigra may facilitate the differential diagnosis of parkinsonian syndromes.

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Finocchiaro, Gherardo, Harshil Dhutia, Belinda Gray, et al. "Diagnostic yield of hypertrophic cardiomyopathy in first-degree relatives of decedents with idiopathic left ventricular hypertrophy." EP Europace 22, no. 4 (2020): 632–42. http://dx.doi.org/10.1093/europace/euaa012.

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Abstract Aims Idiopathic left ventricular hypertrophy (LVH) is defined as LVH in the absence of myocyte disarray or secondary causes. It is unclear whether idiopathic LVH represents the phenotypic spectrum of hypertrophic cardiomyopathy (HCM) or whether it is a unique disease entity. We aimed to ascertain the prevalence of HCM in first-degree relatives of decedents from sudden death with idiopathic LVH at autopsy. Decedents also underwent molecular autopsy to identify the presence of pathogenic variants in genes implicated in HCM. Methods and results Families of 46 decedents with idiopathic LV

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Poirier, Judes, Sandra Kogan, and Serge Gauthier. "Environment, Genetics and Idiopathic Parkinson's Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 18, no. 1 (1991): 70–76. http://dx.doi.org/10.1017/s0317167100031334.

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ABSTRACT:Since Idiopathic Parkinson's disease (IPD) was first described more than 170 years ago, there have been major advances in the understanding of the etiology of the disease as well as in its treatment. This article will review current knowledge concerning the role of the environment, genetic hypotheses and the aging factor in the etiology of IPD and proposes a complex interaction involving all these factors. Hypotheses regarding mitochondrial inhibition and free radical generation in IPD are discussed in relation to the mechanism of action of neurotoxins known to produce parkinsonian sy

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Bruno, Veronica A., Susan H. Fox, Deborah Mancini, and Janis M. Miyasaki. "Botulinum Toxin Use in Refractory Pain and Other Symptoms in Parkinsonism." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 43, no. 5 (2016): 697–702. http://dx.doi.org/10.1017/cjn.2016.279.

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AbstractBackground: Parkinson’s disease (PD) and other parkinsonian syndromes are chronic, progressive neurodegenerative diseases. With advancing disease, both motor and non-motor symptoms represent a considerable burden and symptom relief and quality of life improvement become the main goal of treatment. Botulinum toxins (BTX) are an effective treatment modality for many neurological conditions. Methods: To understand the potential usefulness of BTX in this population, we performed a retrospective chart review of all patients with a clinical diagnosis of idiopathic PD and atypical parkinsonis

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Yudina, V. V., O. N. Voskresenskaya, and G. K. Yudina. "Comorbidity of normotensive hydrocephalia and Parkinson’s disease." Russian neurological journal 26, no. 2 (2021): 30–36. http://dx.doi.org/10.30629/2658-7947-2021-26-2-30-36.

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Normotensive hydrocephalus (NTH) is a syndrome characterized by enlarged ventricles of the brain, gait disturbance, cognitive impairment, and incontinence. In the elderly with gait disturbances of unspecified etiology, NTH should always be excluded. It is especially difficult to diagnose NTH in patients with neurodegenerative diseases, primarily with idiopathic Parkinson’s disease (PD), and vice versa, to diagnose PD in patients with NTH. We report on an 80-year-old patient with a five-year history of NTH, manifested by the classic clinical triad of symptoms and the subsequent development of P

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Pinkhardt, E. H., R. Jürgens, W. Becker, F. Valdarno, A. C. Ludolph, and J. Kassubek. "195. Differential diagnostic value of eye movement recording in PSP-Parkinsonism, Richardson’s syndrome, and idiopathic Parkinson’s disease." Clinical Neurophysiology 120, no. 1 (2009): e80-e81. http://dx.doi.org/10.1016/j.clinph.2008.07.193.

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Butcher, James. "Eradication of H. pylori Modifies Idiopathic Parkinsonism Syndrome." Clinical Psychiatry News 35, no. 6 (2007): 38. http://dx.doi.org/10.1016/s0270-6644(07)70380-6.

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40

Khakimova, A. R., K. B. Timorshina, E. Z. Yakupov, and N. A. Popova. "Idiopathic generalized dystonia." Kazan medical journal 98, no. 6 (2017): 1044–47. http://dx.doi.org/10.17750/kmj2017-1044.

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Idiopathic generalized dystonia is a group of neurodegenerative diseases primarily charecterized by dystonic hyperkinetic disorder (irregular slow involuntary movements of different body parts accompanied by specific muscle tone changes and pathological poses). A 54-year-old female was admitted with various complaints. The leading syndrome was extrapyramidal due to which the differential diagnosis included Parkinson’s disease, multiple system atrophy, corticobasal degeneration, etc. The diagnosis of idiopathic generalized dystonia, in the form of lower extremity hyperkinesia with dystonic equi

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41

Gątarek, Paulina, Joanna Kałużna-Czaplińska, Małgorzata Pawełczyk, et al. "LC-MS/MS Determination of Modified Nucleosides in The Urine of Parkinson’s Disease and Parkinsonian Syndromes Patients." Molecules 25, no. 21 (2020): 4959. http://dx.doi.org/10.3390/molecules25214959.

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Epigenetic modifications play a key role in gene regulation and expression and are involved in numerous cellular processes. Due to the limited research on nucleosides in Parkinson’s disease (PD), it is very important to consider epigenetic factors and their role in the development of PD. The aim of this study was to investigate and compare the levels of modified nucleosides, such as O-methylguanosine, N6-methyl-2′-deoxyadenosine, 1-methyladenosine, 1-methylguanine, 7-methylguanine, 3-methyladenine and 7-methylguanosine in the urine of Parkinson’s disease (PD) patients and the control group, an

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Gołąb-Janowska, Monika, Dariusz Kotlęga, Krzysztof Safranow, Agnieszka Meller, Anna Budzianowska, and Krystyna Honczarenko. "Risk Factors of Fatigue in Idiopathic Parkinson’s Disease in a Polish Population." Parkinson's Disease 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/2835945.

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Introduction.Fatigue syndrome is one of the nonmotor symptoms in Parkinson’s disease (PD). The aim of the study was assessment of prevalence of fatigue syndrome in PD and answering the question what are the independent risk factors connected with intensity of fatigue in PD.Methods. 114 patients with idiopathic PD (mean age 62.2 + 10.8 years) were enrolled. The fatigue was assessed according to the Fatigue Severity Scale (FSS). We analyzed associations between fatigue and sex, age, education, duration and severity of the disease, everyday activity, intensity of the main symptoms, treatment, pre

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Chang, Ya-Ting, Wen-Neng Chang, Nai-Wen Tsai, et al. "Clinical Features Associated with Frozen Shoulder Syndrome in Parkinson’s Disease." Parkinson's Disease 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/232958.

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Background. Frozen shoulder syndrome is a common musculoskeletal disease of idiopathic Parkinson’s disease (PD) that causes long-term pain and physical disability. A better understanding of the associated factors can help identify PD patients who will require prevention to improve their quality of life.Methodology. This prospective study evaluated 60 shoulders of 30 PD patients. Correlation analysis was used to evaluate the relationships between clinical factors and shoulder sonography findings.Results. Frozen shoulder syndrome was found in 14 of 30 PD patients affecting 19 shoulders, includin

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Park, Sangmin, Jung Geol Lim, Hee Jin Chang, and Eungseok Oh. "What Shall We Do for the Patients with Shaky Leg Syndrome? A Review of 23 Patients." Neurodegenerative Diseases 20, no. 1 (2020): 46–54. http://dx.doi.org/10.1159/000509411.

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Orthostatic tremor (OT) is not an uncommon symptom in various neurodegenerative diseases. However, the nature and pathophysiology of OT involve a complex network of tremors and dopaminergic pathways. We assessed patients who complained of prominent leg tremors described as “shaky leg.” We analyzed their characteristics and evaluated them with neuroimaging and electrophysiological tools. A total of 23 patients who experienced an uncomfortable symptom of leg tremor were retrospectively enrolled from April 2014 to October 2019. Previous medical history, brain MRI, and surface electromyography (EM

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Nakhleh, M. K., S. Badarny, R. Winer, R. Jeries, J. Finberg, and H. Haick. "Distinguishing idiopathic Parkinson's disease from other parkinsonian syndromes by breath test." Parkinsonism & Related Disorders 21, no. 2 (2015): 150–53. http://dx.doi.org/10.1016/j.parkreldis.2014.11.023.

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Kohl, Zacharias, Jürgen Winkler, and Horst J. Koch. "Seasonal pattern of births in patients with idiopathic Parkinson syndrome." Biological Rhythm Research 39, no. 1 (2008): 39–41. http://dx.doi.org/10.1080/09291010701233882.

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Kose, Sedat, Cem Barcin, Atilla Iyisoy, Hurkan Kursaklioglu, Ersoy Isik, and Ertan Demirtas. "Idiopathic Ventricular Fibrillation in a Patient with Wolff-Parkinson-White Syndrome." Japanese Heart Journal 43, no. 3 (2002): 283–87. http://dx.doi.org/10.1536/jhj.43.283.

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Ryu, Jung Ho, Myung Sik Lee, and Jong Sam Baik. "Sonographic abnormalities in idiopathic restless legs syndrome (RLS) and RLS in Parkinson’s disease." Parkinsonism & Related Disorders 17, no. 3 (2011): 201–3. http://dx.doi.org/10.1016/j.parkreldis.2010.11.014.

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Lyons, Mark K., Barry D. Birch, Renee A. Hillman, Orland K. Boucher, and Virgilio Gerald H. Evidente. "Long-term follow-up of deep brain stimulation for Meige syndrome." Neurosurgical Focus 29, no. 2 (2010): E5. http://dx.doi.org/10.3171/2010.4.focus1067.

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Object Meige syndrome is characterized by blepharospasm, cervical dystonia, and facial oromandibular dystonia. The medical treatment of this condition is largely unsuccessful over time and is a major source of decreased quality of life in those patients suffering from this disease. Recent advances in the application of deep brain stimulation (DBS) surgery techniques for many disorders have prompted several recent reports of DBS for medically refractory cases of Meige syndrome. While the etiology for this disorder is unknown, it is considered by many investigators to be a form of idiopathic tor

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Asghar, M., and P. Whitty. "Foreign accent syndrome and psychosis: a case report." Irish Journal of Psychological Medicine 37, no. 2 (2017): 118–21. http://dx.doi.org/10.1017/ipm.2017.12.

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This is a case report on a 57-year-old lady who presented to the Psychiatry Department of Tallaght Hospital after being referred by Neurology Department. She was initially attending Neurology Department for idiopathic Parkinson’s disease which was later refuted as there was no objective evidence of response to treatment. She attended the A&E department with altered consciousness and headache, but her computed tomography of the brain was normal, and was diagnosed with migraine. But it was noted that she was reporting paranoia and auditory hallucinations, and her family had noticed marked ch

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