Relevant bibliographies by topics / IL-17 members

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'IL-17 members'

Author: Grafiati
Published: 11 March 2023

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Journal articles on the topic "IL-17 members"

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Chung, Soo-Hyun, Xiao-Qi Ye, and Yoichiro Iwakura. "Interleukin-17 family members in health and disease." International Immunology 33, no. 12 (2021): 723–29. http://dx.doi.org/10.1093/intimm/dxab075.

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Abstract The interleukin-17 (IL-17) family consists of six family members (IL-17A–IL-17F) and all the corresponding receptors have been identified recently. This family is mainly involved in the host defense mechanisms against bacteria, fungi and helminth infection by inducing cytokines and chemokines, recruiting neutrophils, inducing anti-microbial proteins and modifying T-helper cell differentiation. IL-17A and some other family cytokines are also involved in the development of psoriasis, psoriatic arthritis and ankylosing spondylitis by inducing inflammatory cytokines and chemokines, and an
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Kleinschek, Melanie A., Alexander M. Owyang, Barbara Joyce-Shaikh, et al. "IL-25 regulates Th17 function in autoimmune inflammation." Journal of Experimental Medicine 204, no. 1 (2007): 161–70. http://dx.doi.org/10.1084/jem.20061738.

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Interleukin (IL)-25 is a member of the IL-17 family of cytokines. However, unlike the other members of this family, IL-25 promotes T helper (Th) 2 responses. We now show that IL-25 also regulates the development of autoimmune inflammation mediated by IL-17–producing T cells. We have generated IL-25–deficient (il25−/−) mice and found that they are highly susceptible to experimental autoimmune encephalomyelitis (EAE). The accelerated disease in the il25−/− mice is associated with an increase of IL-23 in the periphery and a subsequent increase in the number of inflammatory IL-17–, IFNγ-, and TNF-
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Han, Q., S. Das, M. Hirano, et al. "Characterization of Lamprey IL-17 Family Members and Their Receptors." Journal of Immunology 195, no. 11 (2015): 5440–51. http://dx.doi.org/10.4049/jimmunol.1500892.

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Towne, Jennifer E., Yu Zhang, Lori A. Siegel, Antony Symons, and Alison L. Budelsky. "PS2-045. Role of IL-17 family members in psoriasis." Cytokine 56, no. 1 (2011): 75. http://dx.doi.org/10.1016/j.cyto.2011.07.205.

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Majumder, Saikat, and Mandy J. McGeachy. "IL-17 in the Pathogenesis of Disease: Good Intentions Gone Awry." Annual Review of Immunology 39, no. 1 (2021): 537–56. http://dx.doi.org/10.1146/annurev-immunol-101819-092536.

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The IL-17 family is an evolutionarily old cytokine family consisting of six members (IL-17A through IL-17F). IL-17 family cytokines signal through heterodimeric receptors that include the shared IL-17RA subunit, which is widely expressed throughout the body on both hematopoietic and nonhematopoietic cells. The founding family member, IL-17A, is usually referred to as IL-17 and has received the most attention for proinflammatory roles in autoimmune diseases like psoriasis. However, IL-17 is associated with a wide array of diseases with perhaps surprisingly variable pathologies. This review focu
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Li, H., J. Chen, A. Huang, et al. "Cloning and characterization of IL-17B and IL-17C, two new members of the IL-17 cytokine family." Proceedings of the National Academy of Sciences 97, no. 2 (2000): 773–78. http://dx.doi.org/10.1073/pnas.97.2.773.

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Paroli, Marino, Luca Spadea, Rosalba Caccavale, Leopoldo Spadea, Maria Pia Paroli, and Nicola Nante. "The Role of Interleukin-17 in Juvenile Idiopathic Arthritis: From Pathogenesis to Treatment." Medicina 58, no. 11 (2022): 1552. http://dx.doi.org/10.3390/medicina58111552.

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Background and Objectives: Interleukin-17 (IL-17) is a cytokine family consisting of six members and five specific receptors. IL-17A was the first member to be identified in 1993. Since then, several studies have elucidated that IL-17 has predominantly pro-inflammatory activity and that its production is involved in both the defense against pathogens and the genesis of autoimmune processes. Materials and Methods: In this review, we provide an overview of the role of interleukin-17 in the pathogenesis of juvenile idiopathic arthritis (JIA) and its relationship with IL-23, the so-called IL-23–IL
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Cho, Mila, Seon-Yeong Lee, Jin-Sil Park, et al. "Up-Regulation of Stromal Cell-Derived Factor by IL-17 and IL-18 via Phosphatidylinositol 3-Kinase Dependent Pathway (171.8)." Journal of Immunology 188, no. 1_Supplement (2012): 171.8. http://dx.doi.org/10.4049/jimmunol.188.supp.171.8.

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Abstract IL-17 producing Th17 has a key role in the pathogenesis of autoimmune inflammation. Among various cytokines which are interwoven with the IL-17 pathway, members of IL-1beta family including IL-18 have recently gained significance. In the present study, we stimulated synovial fibroblasts with the combination of IL-17 and IL-18, and quantified the cellular production of stromal cell-derived factor-1 (SDF-1) with ELISA and its mRNA with RT-PCR. Both IL-17 and IL-18 significantly increased the level of SDF-1, not only individually but also synergistically (p<0.05). The synergism wa
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Guo, Yifei, Wei Cao, and Ying Zhu. "Immunoregulatory Functions of the IL-12 Family of Cytokines in Antiviral Systems." Viruses 11, no. 9 (2019): 772. http://dx.doi.org/10.3390/v11090772.

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Members of the interleukin 12 (IL-12) family have been known to be inflammatory factors since their discovery. The IL-12 family consists of IL-12, IL-23, IL-27, IL-35, and a new member, IL-39, which has recently been identified and has not yet been studied extensively. Current literature has described the mechanisms of immunity of these cytokines and potential uses for therapy and medical cures. IL-12 was found first and is effective in combatting a wide range of naturally occurring viral infections through the upregulation of various cytokines to clear the infected cells. IL-23 has an essenti
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Broxmeyer, Hal E., Trevor Starnes, Heather Ramsey, et al. "The IL-17 cytokine family members are inhibitors of human hematopoietic progenitor proliferation." Blood 108, no. 2 (2006): 770. http://dx.doi.org/10.1182/blood-2006-01-0292.

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Dissertations / Theses on the topic "IL-17 members"

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Michaudel, Chloé. "Pollution à l'ozone : maintien de la barrière pulmonaire via l'IL-33, implication des autres membres de la famille IL-1 et régulation cytokinique via AhR." Thesis, Orléans, 2017. http://www.theses.fr/2017ORLE2040.

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L’ozone est un des polluants présents dans l’air que nous respirons. Les pics de ce polluant entrainent une augmentation des hospitalisations et des cas d’exacerbations d’asthme allergique. L’objectif de ce travail est d’étudier plus en détails les mécanismes inflammatoires mis en place après exposition à l’ozone. Cette étude s’est déroulée en trois axes, les deux premiers traitant du rôle de deux alarmines, l’IL-33 et l’IL-1α et le troisième se focalisant sur AhR, un récepteur impliqué dans la réponse à de nombreux polluants. Pour ce faire, des souris ont été exposées à l’ozone selon deux mod
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Souza, Natália Satchiko Hojo de. "Estudo da ocorrência de polimorfismos de nucleotídeo único em genes codificadores de membros da família das citocinas IL-1 e IL-17 e suas associações com a cardiomiopatia chagásica humana." Universidade Federal de Minas Gerais, 2012. http://hdl.handle.net/1843/BUOS-8UBGX2.

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Chagas disease, caused by the protozoan Trypanosoma cruzi, is characterized by an acute phase followed by a chronic phase. Most patients in the chronic phase do not present any clinical signs being clinically classified as indeterminate. On the other hand, about 30% of the patients develop the symptomatic clinical forms (cardiac and/or digestive), and the chronic chagasic cardiomyopathy (CCC) is the most severe form of Chagas disease. The wide variation in clinical presentation of chagasic patients allow us to hypothesize that genetic factors related to the host, particularly genetic polymorph
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Silva, Fabio Arruda e. "Molecular Mechanisms Regulating Cytokine Production by Human Neutrophils." Doctoral thesis, 2018. http://hdl.handle.net/11562/984950.

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Neutrophils are known to perform a series of effector functions that are crucial for the innate and adaptive responses towards infections. Furthermore, neutrophils respond to various stimuli, including microbial components, by synthetizing and secreting a variety of cytokines. In this context, the main objective of this study was to re-evaluate the capacity of human neutrophils to express and produce cytokines of the IL-17 family, including IL-17A, IL-17B, IL-17F and IL-17AF since the current literature on this topic is discordant. By performing RT-qPCR, immunohistochemistry (IHC), immunoblott
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Book chapters on the topic "IL-17 members"

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Sato, Emi, and Shinichi Imafuku. "Effects of IL-17 on Epidermal Development." In Structural, Immunological and Molecular Biology of Keratinocytes [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.101602.

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Immunotherapies targeting interleukin 17 (IL-17) have a strong effect on plaque psoriasis. However, many previous studies on IL-17 focused only on the T-helper 17 (Th17) immune response, and a few studies have reported that IL-17A may affect psoriatic epidermal structure. IL-17 includes six family members, namely IL-17A–F, which are involved in a wide variety of biological responses. IL-17A is produced mainly by Th17 cells or group 3 innate lymphoid cells (ILC3), while IL-17C is locally produced by epithelial cells, such as keratinocytes. In contrast to IL-17C, which is locally produced in various cells such as keratinocytes, it is predicted that IL-17A, which is produced by limited cells and has systemic effects, has different roles in epidermal development. For example, several research studies have shown that IL-17A affects terminal differentiation of epidermis by suppressing the expression of filaggrin or loricrin in keratinocytes. On the other hand, IL-17C, which is produced by keratinocytes themselves, does not have as strong as an effect on epidermal development as IL-17A. In this chapter, we summarized the effects of IL-17A and other IL-17 members on epidermal development and their comprehensive roles based on previously reported papers.
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Evans Adunyah, Samuel, Richard Akomeah, Fareed K.N. Arthur, Roland S. Cooper, and Joshua C.M. Williams. "IL-17 Biological Effects and Signaling Mechanisms in Human Leukemia U937 Cells." In Interleukins - The Immune and Non-Immune Systems’ Related Cytokines. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96422.

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Human Interlekin-17 is produced by memory activated CD4+ T cells and other cells. It was initially considered unique in that its specific receptor is distinct from other cytokine receptors. IL-17 receptor is ubiquitously expressed by different cells including T cells. IL-17 plays a role in regulating growth, immune response and pro-inflammatory responses. It regulates differentiation of a subset of Th0 cells into Th-17 cells, which produce IL-17-induced cytokines. The IL-17R belongs to type 1 cytokine receptors. IL-17 belongs to a superfamily of its own, which includes IL-17A, IL-17B, IL-17C, IL-17E and IL-17F. These members of IL-17 superfamily have some sequence homology but bind to different receptors. Prior to this investigation, limited information existed on the effects of IL-17A in human leukemia cell lines. Our results show that IL-17A promotes growth, anti-apoptotic effects, chemotaxis, cytokine expression and transcriptional factor activation in leukemia cells. IL-17A activates multiple signaling pathways including PI-3 K, Jak–STAT, Raf-ERK1/2 and SRC kinase pathways, which mediate different biological effects of IL-17A in leukemia cells. Our findings implicate IL-17A in leukemia cell growth and survival, supporting potential leukemia therapy via development of anti-IL-17A drugs. This chapter focuses on IL-17A, herein referred to as IL-17.
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