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Journal articles on the topic 'IL-5'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Kotsimbos, ATC, and Q. Hamid. "IL-5 and IL-5 receptor in asthma." Memórias do Instituto Oswaldo Cruz 92, suppl 2 (1997): 75–91. http://dx.doi.org/10.1590/s0074-02761997000800012.

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2

TAKATSU, Kiyoshi. "Interleukin-5, IL-5." Journal of Japan Atherosclerosis Society 23, no. 10 (1996): 599–603. http://dx.doi.org/10.5551/jat1973.23.10_599.

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3

ANIL, MISHRA. "Long term Th2 cytokines immunotherapy is beneficial for EoE pediatric patients?" Int J Public Health Awar 5, no. 1-2 (2022): 13–15. https://doi.org/10.725722/IJPHA.ISP.

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Eosinohilic esophagitis (EoE) and gastroesophageal reflux diseases (GERD) are closely related esophageal disorders, and both accumulate eosinophils in the esophagus. However, origin of both these diseases are very different. The evidence indicates that acid reflux is the main cause of GERD; whereas EoE is induced in response to food or environment allergens exposed accumulation of inflammatory cells like eosinophils, mast cells and T cell subsets. EoE is a global problem, and effect even 1 year old children to adult population. Currently, mepolizumab (anti-IL-5) neutralization therapy sdata sh

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4

Oberhofer, Elke. "IL-5- und IL-5-Rezeptor-Blocker als Option?" hautnah dermatologie 40, no. 1 (2024): 12. http://dx.doi.org/10.1007/s15012-023-8326-z.

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5

Malhi, Gin S. "DSM-5: Il buono, il cattivo, il brutto." Australian & New Zealand Journal of Psychiatry 47, no. 7 (2013): 595–98. http://dx.doi.org/10.1177/0004867413496363.

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6

Moermans, C., C. Brion, G. Bock, et al. "Sputum IL-5 predicts the response to anti-IL-5/IL-5R therapy." Revue des Maladies Respiratoires 40, no. 2 (2023): 112. http://dx.doi.org/10.1016/j.rmr.2022.11.008.

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7

Oberhofer, Elke. "IL-5- und IL-5-Rezeptor-Blocker bei refraktärem DRESS." Allergo Journal 32, no. 4 (2023): 16–17. http://dx.doi.org/10.1007/s15007-023-5752-5.

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8

Stein, Miguel L., Joyce M. Villanueva, Bridget K. Buckmeier, et al. "Anti–IL-5 (mepolizumab) therapy reduces eosinophil activation ex vivo and increases IL-5 and IL-5 receptor levels." Journal of Allergy and Clinical Immunology 121, no. 6 (2008): 1473–83. http://dx.doi.org/10.1016/j.jaci.2008.02.033.

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9

NAKANISHI, KENJI. "Interleukin cascade of IL-4,IL-5 and IL-2." Japanese Journal of Clinical Immunology 13, no. 5 (1990): 438–40. http://dx.doi.org/10.2177/jsci.13.438.

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10

Warren, H. S., B. F. Kinnear, J. H. Phillips, and L. L. Lanier. "Production of IL-5 by human NK cells and regulation of IL-5 secretion by IL-4, IL-10, and IL-12." Journal of Immunology 154, no. 10 (1995): 5144–52. http://dx.doi.org/10.4049/jimmunol.154.10.5144.

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Abstract Human NK cells produce IFN-gamma, TNF-alpha, and granulocyte macrophage-CSF when stimulated with susceptible target cells or through the CD16 and CD94 cell surface molecules. This study reports that NK cells also produce IL-5, a cytokine typically produced by Th2 cells, which mediates mobilization and differentiation of eosinophils. Polyclonal NK cell populations and NK cell clones produce IL-5 when stimulated to proliferate with gamma-irradiated MM-170 melanoma cells or JY B-lymphoblastoid cells and rIL-2. IL-5 is produced in cultures generated from freshly isolated NK cells (primary

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11

Esnault, Stephane, Mats W. Johansson, and Sameer K. Mathur. "Eosinophils, beyond IL-5." Cells 10, no. 10 (2021): 2615. http://dx.doi.org/10.3390/cells10102615.

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12

Takatsu, Kiyoshi, and Hiroshi Nakajima. "IL-5 and eosinophilia." Current Opinion in Immunology 20, no. 3 (2008): 288–94. http://dx.doi.org/10.1016/j.coi.2008.04.001.

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13

Chung, K. F. "IL-5 in asthma." Thorax 57, no. 8 (2002): 751. http://dx.doi.org/10.1136/thorax.57.8.751.

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14

Matthaei, Klaus I., Paul S. Foster, and Ian G. Young. "The role of interleukin-5 (IL-5 ) in vivo: studies with IL-5 deficient mice." Memórias do Instituto Oswaldo Cruz 92, suppl 2 (1997): 63–68. http://dx.doi.org/10.1590/s0074-02761997000800010.

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15

Frohberger, Stefan J., Jesuthas Ajendra, Jayagopi Surendar, et al. "Susceptibility to L. sigmodontis infection is highest in animals lacking IL-4R/IL-5 compared to single knockouts of IL-4R, IL-5 or eosinophils." Parasites & Vectors 12, no. 1 (2019): 248. https://doi.org/10.1186/s13071-019-3502-z.

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<strong>Background: </strong>Mice are susceptible to infections with the rodent filarial nematode <i>Litomosoides sigmodontis</i> and develop immune responses that resemble those of human filarial infections. Thus, the <i>L. sigmodontis</i> model is used to study filarial immunomodulation, protective immune responses against filariae and to screen drug candidates for human filarial diseases. While previous studies showed that type 2 immune responses are protective against <i>L. sigmodontis</i>, the present study directly compared the impact of eosinophils, IL-5, and the IL-4R on the outcome of

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16

KOPF, MANFRED, GRAHAM LE GROS, ANTHONY J. COYLE, MARIE KOSCO-VTLBOIS, FRANK BROMBACHER, and Georges Kohler. "Immune Responses of IL-4, IL-5, IL-6 Deficient Mice." Immunological Reviews 148, no. 1 (1995): 45–69. http://dx.doi.org/10.1111/j.1600-065x.1995.tb00093.x.

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17

Takatsu, Kiyoshi. "Interleukin 5 (IL-5) and Its Receptor." Microbiology and Immunology 35, no. 8 (1991): 593–606. http://dx.doi.org/10.1111/j.1348-0421.1991.tb01591.x.

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18

Dickason, R. R., M. M. Huston, and D. P. Huston. "Delineation of IL-5 domains predicted to engage the IL-5 receptor complex." Journal of Immunology 156, no. 3 (1996): 1030–37. http://dx.doi.org/10.4049/jimmunol.156.3.1030.

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Abstract IL-5 is an interdigitating homodimeric glycoprotein and a member of the helical bundle family of cytokines. IL-5 is a potent activator of eosinophils and a specific promoter of their differentiation. This activity has implicated IL-5 in the pathogenesis of asthma and allergic disease. A detailed understanding of IL-5 structure and function is required to develop immunomodulators of IL-5-mediated inflammatory responses. We generated a panel of neutralizing anti-IL-5 mAbs which were used to map functional domains on IL-5. In addition, the nucleotide sequences for human IL-5, murine IL-5

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19

Noor, Jamal Mohammed, and A. Al-fahham Ali. "Pathophysiology and Potential Clinical Significance of Il-5: A Review Article." INTERNATIONAL JOURNAL OF HEALTH & MEDICAL RESEARCH 04, no. 01 (2025): 18–21. https://doi.org/10.5281/zenodo.14627363.

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Interleukins belong to the group of cytokines that play a key role in immune system cell signaling. Among them, Interleukin-5 (IL-5) gains paramount significance in the regulation of eosinophils, which are thinly scattered in most allergic as well as inflammatory conditions, especially asthma. A biochemical characterization of IL-5 is a prerequisite for understanding the basic aspects of its function and finding a precise treatment. Interleukin-5 (IL-5) is a critical cytokine that primarily drives the development and differentiation of eosinophils and B cells, playing a key role in the allergi

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20

STEIN, M., A. MUNITZ, and M. ROTHENBERG. "Increased High Molecular Weight IL-5 Complex After Anti-IL-5 (Mepolizumab) Therapy." Journal of Allergy and Clinical Immunology 121, no. 2 (2008): S118. http://dx.doi.org/10.1016/j.jaci.2007.12.468.

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21

Li, Shijie, Shijie Wang, Eric Fordjour та ін. "Development and characterization of anti-IL-5 monoclonal antibody Fab fragment for blocking IL-5/IL-5Rα binding". International Immunopharmacology 124 (листопад 2023): 111032. http://dx.doi.org/10.1016/j.intimp.2023.111032.

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22

Ohbo, Kazuyuki, Hironobu Asoa, Kouro Taku та ін. "Demonstration of a cross-talk between IL-2 and IL-5 in Phosphorylation of IL-2 and IL-5 receptor β chains". International Immunology 8, № 6 (1996): 951–60. http://dx.doi.org/10.1093/intimm/8.6.951.

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23

Yamaguchi, Y., T. Suda, H. Shiozaki, et al. "Role of IL-5 in IL-2-induced eosinophilia. In vivo and in vitro expression of IL-5 mRNA by IL-2." Journal of Immunology 145, no. 3 (1990): 873–77. http://dx.doi.org/10.4049/jimmunol.145.3.873.

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Abstract We recently demonstrated in vivo that IL-5 was an important mediator of eosinophilia in mice with parasite infections. In this study, we examined whether or not IL-5 was actually responsible for the eosinophilia induced by injection of human rIL-2. Mice administered hIL-2 developed eosinophilia during the course of the series of injections. This eosinophilia could be suppressed by a single injection of mAb against murine IL-5. The number of eosinophilic precursors increased more in the spleen cells of the IL-2-treated mice in comparison to the control mice, although in bone marrow pre

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24

Martínez-Saldaña, Ma. Consolación. "CYPERMETHRIN CAUSE ALLERGIC INFLAMMATORY RESPONSE IN RAT LUNG." World Journal of Pharmaceutical Science and Research 2, no. 6 (2023): 263–76. https://doi.org/10.5281/zenodo.10421769.

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Cypermethrin is a type 2 photosensitive liposoluble pyrethroid for agricultural and domestic use, has a half-life in the ambient air for days, its metabolism in human liver generate a cyan group (CN), an alcohol fraction (dimethylcyclopropanecarboxylic acid) (DCCA), and 3-acid phenoxybenzoic acid (3-PBA) with reactive oxygen species (ROS) properties, there are scarce studies when is inhaled. The objective of this work was to evaluate the structural changes and activation of allergic inflammatory response in lower airway components and alveoli induced by inhaled exposure to low doses of cyperme

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25

TAKATSU, SEISHI. "IL-5 and its receptor." Japanese Journal of Clinical Immunology 13, no. 5 (1990): 441–43. http://dx.doi.org/10.2177/jsci.13.441.

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26

Narendra, Dharani K., and Nicola A. Hanania. "Targeting IL-5 in COPD." International Journal of Chronic Obstructive Pulmonary Disease Volume 14 (May 2019): 1045–51. http://dx.doi.org/10.2147/copd.s155306.

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27

BAGLEY, C., A. LOPEZ, and M. VADAS. "New frontiers for IL-5." Journal of Allergy and Clinical Immunology 99, no. 6 (1997): 725–28. http://dx.doi.org/10.1016/s0091-6749(97)80002-4.

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28

Leung, Donald Y. M., Harold S. Nelson, Stanley J. Szefler, and William W. Busse. "Anti–IL-5 for hypereosinophilia." Journal of Allergy and Clinical Immunology 113, no. 1 (2004): 2. http://dx.doi.org/10.1016/j.jaci.2003.11.004.

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29

Ameri, Abdol A. "Weiterer IL-5-Antikörper verfügbar." MMW - Fortschritte der Medizin 159, no. 1 (2017): 67. http://dx.doi.org/10.1007/s15006-017-9167-7.

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30

Klein, Friederike. "IL-5-Antikörper bei Vaskulitis." MMW - Fortschritte der Medizin 165, no. 19 (2023): 66. http://dx.doi.org/10.1007/s15006-023-3119-1.

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31

Gregory, Bernard, Antje Kirchem, Simon Phipps та ін. "Differential Regulation of Human Eosinophil IL-3, IL-5, and GM-CSF Receptor α-Chain Expression by Cytokines: IL-3, IL-5, and GM-CSF Down-Regulate IL-5 Receptor α Expression with Loss of IL-5 Responsiveness, but Up-Regulate IL-3 Receptor α Expression". Journal of Immunology 170, № 11 (2003): 5359–66. http://dx.doi.org/10.4049/jimmunol.170.11.5359.

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32

Webb, Dianne C., Andrew N. J. McKenzie, Aulikki M. L. Koskinen, Ming Yang, Joërg Mattes, and Paul S. Foster. "Integrated Signals Between IL-13, IL-4, and IL-5 Regulate Airways Hyperreactivity." Journal of Immunology 165, no. 1 (2000): 108–13. http://dx.doi.org/10.4049/jimmunol.165.1.108.

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33

Fort, Madeline M., Jeanne Cheung, David Yen, et al. "IL-25 Induces IL-4, IL-5, and IL-13 and Th2-Associated Pathologies In Vivo." Immunity 15, no. 6 (2001): 985–95. http://dx.doi.org/10.1016/s1074-7613(01)00243-6.

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34

Tominaga, A., T. Takahashi, Y. Kikuchi, et al. "Role of carbohydrate moiety of IL-5. Effect of tunicamycin on the glycosylation of IL-5 and the biologic activity of deglycosylated IL-5." Journal of Immunology 144, no. 4 (1990): 1345–52. http://dx.doi.org/10.4049/jimmunol.144.4.1345.

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Abstract IL-5 is a T cell-derived lymphokine that induces B cell growth and differentiation in murine systems. In this study, we examined the role of carbohydrate moiety of IL-5 in the expression of biological function. IL-5 polypeptides translated in Xenopus oocytes were heterogeneous in terms of isoelectric point (pI 4.7 to 8.0) and m.w. (45,000 to 60,000 under nonreducing conditions) and yielded m.w. of 25,000 to 30,000 under reducing conditions. Treatment of rIL-5 with N-glycanase under reducing conditions yielded an IL-5 monomer of m.w. 12,000 to 14,000. Furthermore, deglycosylated rIL-5

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35

Randall, T. D., F. E. Lund, J. W. Brewer, C. Aldridge, R. Wall, and R. B. Corley. "Interleukin-5 (IL-5) and IL-6 define two molecularly distinct pathways of B-cell differentiation." Molecular and Cellular Biology 13, no. 7 (1993): 3929–36. http://dx.doi.org/10.1128/mcb.13.7.3929-3936.1993.

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Interleukin-5 (IL-5) and IL-6 have both been reported to act as B-cell differentiation factors by stimulating activated B cells to secrete antibody. However, it has not been possible to directly compare the effects of these two lymphokines because of the lack of a suitable B-cell line capable of responding to both. We have identified a clonal, inducible B-cell lymphoma, CH12, that has this property. Both IL-5 and IL-6 can independently stimulate increases in steady-state levels of immunoglobulin and J-chain mRNA and proteins, and they both induce the differentiation of CH12 into high-rate anti

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36

Randall, T. D., F. E. Lund, J. W. Brewer, C. Aldridge, R. Wall, and R. B. Corley. "Interleukin-5 (IL-5) and IL-6 define two molecularly distinct pathways of B-cell differentiation." Molecular and Cellular Biology 13, no. 7 (1993): 3929–36. http://dx.doi.org/10.1128/mcb.13.7.3929.

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Interleukin-5 (IL-5) and IL-6 have both been reported to act as B-cell differentiation factors by stimulating activated B cells to secrete antibody. However, it has not been possible to directly compare the effects of these two lymphokines because of the lack of a suitable B-cell line capable of responding to both. We have identified a clonal, inducible B-cell lymphoma, CH12, that has this property. Both IL-5 and IL-6 can independently stimulate increases in steady-state levels of immunoglobulin and J-chain mRNA and proteins, and they both induce the differentiation of CH12 into high-rate anti

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37

H, Ha, Spicer T, He Xy, et al. "IL-4 AND IL-5 THERAPY INHIBITS HEYMANN NEPHRITIS (HN)." Nephrology 7, no. 1 (2002): A113. http://dx.doi.org/10.1046/j.1440-1797.2002.00007-1-113.x.

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38

Hashiguchi, Masaaki, Yuji Kashiwakura, Yumiko Kanno, Hidefumi Kojima, and Tetsuji Kobata. "IL-21 and IL-5 coordinately induce surface IgA+ cells." Immunology Letters 224 (August 2020): 21–27. http://dx.doi.org/10.1016/j.imlet.2020.05.004.

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39

Bourdenet, V., G. Devouassoux, B. Antoine, et al. "Eczéma paradoxal sous biothérapie ciblant l’axe IL-5/IL-5R." Annales de Dermatologie et de Vénéréologie - FMC 2, no. 8 (2022): A64. http://dx.doi.org/10.1016/j.fander.2022.09.062.

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40

Solinger, Alan M. "IL-1-5 Blocking IL-1β in type 2 diabetes". Cytokine 52, № 1-2 (2010): 10. http://dx.doi.org/10.1016/j.cyto.2010.07.043.

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41

Bourdenet, V., G. Devouassoux, A. Beurnier, et al. "Eczémas paradoxaux sous biothérapies ciblant l’axe IL-5/IL-5R." Revue Française d'Allergologie 63, no. 3 (2023): 103611. http://dx.doi.org/10.1016/j.reval.2023.103611.

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42

Reiman, Rachael M., Robert W. Thompson, Carl G. Feng, et al. "Interleukin-5 (IL-5) Augments the Progression of Liver Fibrosis by Regulating IL-13 Activity." Infection and Immunity 74, no. 3 (2006): 1471–79. http://dx.doi.org/10.1128/iai.74.3.1471-1479.2006.

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ABSTRACT Eosinophils are frequently found in increased numbers in a variety of chronic fibrotic diseases; however, their role in the development of hepatic fibrosis has not been dissected in vivo. Here, we used interleukin-5 (IL-5) knockout (KO) mice to determine whether eosinophils contribute to the progressive liver fibrosis that develops in response to chronic Schistosoma mansoni infection. Although infection intensities were similar in C57BL/6 and IL-5 KO mice, the average size of granulomas was significantly smaller in both acutely and chronically infected IL-5 KO mice. Their granulomas w

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43

Drick, Nora, Katrin Milger, Benjamin Seeliger та ін. "Switch from IL-5 to IL-5-Receptor α Antibody Treatment in Severe Eosinophilic Asthma". Journal of Asthma and Allergy Volume 13 (листопад 2020): 605–14. http://dx.doi.org/10.2147/jaa.s270298.

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44

Hall, B., K. Plain, M. Nomura, et al. "ALLOGRAFT TOLERANCE MEDIATING CD4+T CELLS DEPEND UPON INTERLEUKIN-5 (IL-5), NOT IL-4." Transplantation 86, Supplement (2008): 136. http://dx.doi.org/10.1097/01.tp.0000332495.28926.ce.

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45

Namkung, J. H., J. E. Lee, E. Kim, et al. "IL-5 and IL-5 receptor alpha polymorphisms are associated with atopic dermatitis in Koreans." Allergy 62, no. 8 (2007): 934–42. http://dx.doi.org/10.1111/j.1398-9995.2007.01445.x.

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46

Zhu, W., N. Liu, Y. Zhao, H. Jia, B. Cui, and G. Ning. "Association analysis of polymorphisms in IL-3, IL-4, IL-5, IL-9, and IL-13 with Graves’ disease." Journal of Endocrinological Investigation 33, no. 10 (2010): 751–55. http://dx.doi.org/10.1007/bf03346682.

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47

TAKATSU, Kiyoshi. "Structure and Function of IL-5 Receptor." YAKUGAKU ZASSHI 115, no. 8 (1995): 570–83. http://dx.doi.org/10.1248/yakushi1947.115.8_570.

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48

Hatzistilianou, M., C. Aggouridaki, B. Tsotsou, A. Thymi, G. Koulouses та A. Stogias. "Serum TNF-α, IL-1β, IL-4 and IL-5 levels in bronchial asthma". Immunology Letters 56 (травень 1997): 155. http://dx.doi.org/10.1016/s0165-2478(97)85619-2.

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49

Hatzistilianou, M. "Serum TNF-α, IL-1β, IL-4 and IL-5 levels in bronchial asthma". Immunology Letters 56, № 1-3 (1997): 155. http://dx.doi.org/10.1016/s0165-2478(97)87457-3.

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50

Brown, Pamela M., Philip Tagari, Kevin R. Rowan, et al. "Epitope-labeled Soluble Human Interleukin-5 (IL-5) Receptors." Journal of Biological Chemistry 270, no. 49 (1995): 29236–43. http://dx.doi.org/10.1074/jbc.270.49.29236.

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