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Books on the topic 'Immune organs'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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1

United States. National Aeronautics and Space Administration., ed. New technologies for bioartificial organs. Center for Microgravity Research and Applications, Vanderbilt University, 1997.

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2

Berne, Katrina H. Running on empty: Chronic Fatigue Immune Dysfunction Syndrome (CFIDS). Hunter House, 1995.

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3

Berne, Katrina H. Running on empty: Chronic fatigue immune dysfunction syndrome (CFIDS). Hunter House, 1992.

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4

Inc, World Book, ed. The digestive and urinary systems. World Book, a Scott Fetzer Company, 2014.

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5

Hippeläinen, Mikko. Maturation and site of the peripheral immune function associated with the bursa of Fabricius in the chicken: Surgical techniques for separating the bursa of Fabricius from other lymphoid organs. Dept. of Surgery, University of Kuopio, 1988.

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6

F, Tenholder Michael, ed. Approach to pulmonary infections in the immunocompromised host. Futura Pub. Co., 1991.

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7

Atlan, Henri. Theories of Immune Networks. Springer Berlin Heidelberg, 1989.

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8

Green, Peter H. R. Celiac disease: A hidden epidemic. Collins, 2006.

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9

Leslie, Brent, and Sells Robert A, eds. Organ transplantation: Current clinical and immunological concepts. Baillière Tindall, 1989.

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10

Arthur, Baue, Faist E, Schildberg F. W, and International Congress on the Immune Consequences of Trauma, Shock, and Sepsis--Mechanisms and Therapeutic Approaches (3rd : 1994 : Munich, Germany), eds. The immune consequences of trauma, shock, and sepsis: Mechanisms and therapeutic approaches. Pabst Science Publishers, 1996.

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11

Huang, Lei, Andrew L. Mellor, Anatolij Horuzsko, and Sha Wu, eds. Immune Regulations in Reproductive Organs and Organ Transplant. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-945-4.

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12

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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13

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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14

Soulsby, E. J. L. Immune Response to Parasitic Infections. CRC, 1987.

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15

Soulsby, E. J. L. Immune Responses in Parasitic Infections: Immunology Immunopathology, and Immunoprophylaxis. Crc Pr I Llc, 1987.

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16

Klenerman, Paul. The Immune System: A Very Short Introduction. Oxford University Press, 2017. http://dx.doi.org/10.1093/actrade/9780198753902.001.0001.

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The Immune System: A Very Short Introduction describes the immune system and how it works in health and disease. It focuses on the human immune system, considering how it evolved, and the basic rules that govern its behaviour. The immune system comprises a series of organs, cells, and chemical messengers that work together as a team to provide defence against infection. These components are discussed along with the critical signals that trigger them and how they exert their protective effects, including innate and adaptive responses. The consequences of too little immunity (immunodeficiency),

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17

Buckingham, Robert. Trafficking: How Chronic Inflammation Sabotages the Immune Arsenal and Poisons the Interstitial Space of End Organs. Parchment Global Publishing, 2024.

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18

H. M. Yang, Z. Yang, W. Wang, et al. Effects of different housing systems on visceral organs, serum biochemical proportions, immune performance and egg quality of laying hens. Verlag Eugen Ulmer, 2014. http://dx.doi.org/10.1399/eps.2014.48.

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19

Scott, Gabriel. Healthy Body Organs Cookbook: Immune Boosting Diets Nutrition, Food Science, and Recipes and How to Prevent and Reverse Organ Failure and Everything You Need to Know. Independently Published, 2021.

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20

Slavin, Shimon. Tolerance in Bone Marrow and Organ Transplantation: Achievements and Goals. Elsevier Publishing Company, 1985.

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21

The Digestive and Urinary Systems. World Book, Inc., 2016.

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22

Cleverley, Joanne. The imaging of fungal disease. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0041.

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The imaging of fungal infection is diverse and often non-specific with multiple abnormalities commonly identified, frequently with more than one organ involved. By correlating the clinical information, which should include patient immune status, pre-existing chronic disease, and potential exposure to endemic fungi, and using this information with an awareness of the radiographic findings of fungal infection, a potential diagnosis can be ascertained. In this chapter, the imaging of fungal infection is discussed, concentrating on the various imaging modalities available, their role, and the majo

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23

Patterson, Marc C. Congenital Disorders of Glycosylation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0066.

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Congenital disorders of glycosylation (CDG) comprise a family of multisystem diseases in which N- and O-linked glycosylation and glypiation of a variety of proteins and lipids is deficient. The hypoglycosylation of multiple glycoconjugates impairs normal development of the brain (and other organs), and is associated with both episodic and chronic organ dysfunction. Developmental disorders; seizures; strokelike episodes (and stroke); hearing and visual loss; peripheral neuropathy; coagulopathy; and immune, liver, endocrine, cardiac, and cutaneous manifestations may occur in varying combinations

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24

Haldar, Pranabashis. Tuberculosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0130.

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Tuberculosis (TB) is an infectious disease caused by the bacterial organism Mycobacterium tuberculosis. In this context, reference to the word ‘disease’ is important, as TB implies Mycobacterium tuberculosis infection associated with symptoms. Approximately 10% of Mycobacterium tuberculosis infection is manifest as disease. In the large majority of cases, Mycobacterium tuberculosis infection is latent and defined by evidence of a measurable and significant cell-mediated immune response to mycobacterial antigens, in the absence of clinical or radiological evidence of disease. TB may be clinical

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25

Ussing, Anne Phaff. Development of the immune system during amphibian ontogenesis: Blood cells and haemopoietic organs during thyroid-hormone-induced metamorphosis in the Mexican Axolotl (Amphibia, urodela, Amystoma Mexicanum). 1991.

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26

Meisami, Esmail. CRC Handbook of Human Growth and Developmental Biology, Volume III, Part A. CRC Press, 1990.

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27

Ramsay, Michael A. E. Anaesthesia for transplant surgery. Edited by Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0067.

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The provision of anaesthesia for organ transplantation requires a team of specialist anaesthetists who are available 24 hours a day. The cold and warm ischaemia times may have very deleterious effects on the graft. The team must have a basic understanding of the immune system and the strategies of immunosuppression therapy. The preoperative assessment of the patient requires an understanding of the cause and effects of the compromised organ that is to be replaced. The procedure in many instances will result in a reperfusion syndrome when the graft is revascularized and also an ischaemia–reperf

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28

Bretscher, Peter. Rediscovering the Immune System as an Integrated Organ. FriesenPress, 2016.

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29

Meisami, Esmail. CRC Handbook of Human Growth and Developmental Biology, Volume III, Part B. CRC Press, 1990.

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30

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient

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31

Jones, Rory, and Peter H. R. Green. Celiac Disease: A Hidden Epidemic. Collins, 2006.

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32

Jones, Rory, and Peter H. R. Green. Celiac Disease: A Hidden Epidemic. HarperCollins Publishers, 2020.

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33

Jones, Rory, and Peter H. R. Green. Celiac Disease: A Hidden Epidemic. Collins, 2006.

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34

Jones, Rory, and Peter H. R. Green. Celiac Disease: A Hidden Epidemic. HarperCollins Publishers, 2020.

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35

Immunotoxicology and Immunopharmacology, Third Edition (Target Organ Toxicology Series). 3rd ed. CRC, 2006.

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36

JONES, Racheal. Healthy Body Organ Cookbook: Immune Boosting Diets and Organ Repairs with Meal Plans to Help Boost Your Immune System, Loss Weight and Heal Your Body Totally. Independently Published, 2021.

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37

Organ Transplantation: Current Clinical and Immunological Concepts. W.B. Saunders Company, 1989.

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38

Limaye, Ajit P., and Lynne Strasfeld. Introduction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199938568.003.0200.

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Chapter 2 focuses on the solid organ transplantation (SOT). The Solid organ transplantation (SOT) is undertaken to restore organ function for patients with failing or end-stage disease of the liver, heart, lung, kidneys, and/or pancreas or to re-establish function in patients with short gut or other disorders of the intestinal tract. Organ transplantation requires lifelong maintenance immune suppression to prevent organ rejection. Infection can be related to donor transmission, reactivation from latency in the recipient, or acquisition de novo post-transplant. The evaluation of suspected infec

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39

Fischer, Kevin M., and Shannon S. Carson. Chronic Multiple Organ Dysfunction. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0013.

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This chapter reviews the clinical syndrome of chronic multiple organ dysfunction (MOD) following acute critical illness. Chronic MOD, also referred to as chronic critical illness, occurs in patients who have survived the acute phase of their illness or injury but remain dependent on life support for weeks or months. This condition presents unique physiologic and metabolic abnormalities distinct from those encountered in the acute illness. These include neuroendocrine and immune dysregulation, ICU-acquired weakness, persistent respiratory failure, and brain dysfunction. The symptom burden for t

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40

Maddux, Aline B., and Gordon R. Bernard. Immunomodulation strategies in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0312.

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Severe sepsis is a hyperimmune response to an infectious stimulus resulting in a surge of cytokines and mediators of inflammation. High circulating levels of pro-inflammatory cytokines lead to shock, multiple organ failure, and death in septic patients. It has been recognized that patients with sepsis progress into a state of immune paralysis characterized by immune cell apoptosis and high levels of anti-inflammatory cytokines… Anti-inflammatory cytokines suppress production of pro-inflammatory cytokines and inhibit monocytes from presenting antigens to other immune cells. Immune paralysis pro

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41

Rizzuto, Gabrielle A., and Anna I. Bakardjiev. Listeria monocytogenes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0020.

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Listeria monocytogenes is a intracellular bacterial pathogen that causes serious foodborne illness in humans. Among all infectious diseases caused by gastrointestinal pathogens, listeriosis has the highest mortality rate, likely because of its ability to cross the gastrointestinal barrier and cause sepsis and infection of other organs such as the brain and placenta. Infection of the placenta leads to fetal infection, and otherwise healthy pregnant women have a significantly increased incidence of listeriosis than the general population, likely due to changes in the maternal cell-mediated immun

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42

Condon, Marie, Philippa Dodd, and Liz Lightstone. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0162.

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AbstractSystemic lupus erythematosus (SLE) is a chronic, relapsing, inflammatory, often febrile multisystemic disorder, characterized by involvement of the skin, joints, visceral organs, and serosal membranes. Symptoms and manifestations vary widely over an unpredictable relapsing and remitting course.The presentation of SLE can range from mild forms to severe disease requiring hospitalization. Most commonly it manifests as a combination of constitutional symptoms, particularly fatigue and fever, with cutaneous, musculoskeletal, mild haematological, and serological involvement; however, when r

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43

Ackland, Gareth L. Neural and endocrine function in the immune response to critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0310.

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The neurohormonal physiological response to various stressors is pivotal for maintaining homeostasis. However, the advent of modern critical care has distorted evolutionary biology by generating the entirely new (patho)physiological entity of critical illness. By extending the biological features of the ‘fight or flight’ response beyond the acute phase, distinct neurohormonal, and immune profiles have become increasingly apparent. Both direct and off-target effects of neurohormonal control on immune function are implicated in the disruption of bidirectional links between neurohormones and immu

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44

Bhole, Malini. Hypersensitivity diseases. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0300.

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Hypersensitivity reactions are aberrant immune responses that are provoked by innocuous extrinsic or self-antigens, are mediated by B-cells or T-cells, and may result in tissue or organ damage. Coombs and Gell classified hypersensitivity reactions into four types, based on the different immune responses: type I, or immediate hypersensitivity; type II, or antibody-mediated (humoral) cytotoxicity; type III, or immune-complex disease; and type IV, or delayed hypersensitivity. This chapter reviews the clinical features, diagnosis, and management of hypersensitivity reactions.

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45

Nydegger, Rudy. Dealing with Anxiety. ABC-CLIO, LLC, 2016. http://dx.doi.org/10.5040/9798400638138.

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A concise, accessible introduction to anxiety covering everything from its causes to its symptoms, available treatment options, and prevention. While anxiety is commonly associated with modern stresses and problems, few realize that disorders of this kind have existed since the beginning of time. What defines "anxiety" as a mental health condition? Who gets it and why? This book looks at this highly treatable condition that is responsible for many lost days of school and work and contributes to rising health care costs. Mental health practitioner Rudy Nydegger addresses the full spectrum of an

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46

Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0181_update_001.

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AbstractSchistosomes are blood flukes that parasitize humans, apes, cattle, and other animals. In these definitive hosts they are bisexual, and lay eggs which are shed to fresh water where they complete an asexual cycle in different snails, ending in the release of cercariae which infect the definitive hosts to complete the life cycle.Seven of over 100 species of schistosomes are human pathogens, causing disease in different organs depending on the parasite species. Racial and genetic factors are involved in susceptibility, severity, and sequelae of infection.Morbidity is induced by the host’s

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47

Vinod, Nikhra. Living with COVID-19: The Nemesis, the Hubris, and the Elpis. Heighten Science Publications Inc., 2021. http://dx.doi.org/10.29328/ebook1004.

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The immuno-thrombo-inflammatory disease: COVID-19 is an acute immuno-thrombogenic inflammatory viral disease manifested by dysfunctions related to multiple organs involved in its pathogenesis. Its incidence and prevalence of is related to environmental viability of the virus, various transmission factors associated with the agent and the host, possible modes of transmission, period of infectiousness, and composition and susceptibility of the population. Whereas respiratory route is dominant mode of transmission, transmission through direct contact or fomite transmission do occur.

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48

Heeringa, Peter, and Coen A. Stegeman. The patient with vasculitis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0158.

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Vasculitis is an inflammatory destructive process affecting blood vessels resulting in fibrinoid necrosis of the vessel wall that eventually can lead to occlusion of the vascular lumen and organ failure. Vasculitis may be the primary manifestation of a disease, or be a secondary manifestation of another underlying disease.The pathogenesis of vasculitis is complex, involving innate and adaptive immune effector mechanisms that range from cell-mediated inflammation, immune-complex-mediated inflammation, and inflammation triggered by autoantibodies. Here it is discussed with respect to general pat

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49

Bhagani, Dr Sanjay, Dr Nicholas Easom, Dr Sanjay Bhagani, Dr Nicholas Easom, and Dr Nicholas Easom. Immunocompromised patients, including HIV-positive patients. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00013.

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Chapter 13 discusses immunocompromised patients, including HIV-positive patients, HIV and other causes of immunodeficiency, infections in the HIV-infected patient, post-exposure prophylaxis (PEP) for prevention of HIV infection, non-HIV causes of immunodeficiency, secondary immunodeficiency in malignancy or post-chemotherapy, immune dysfunction and systemic illness, solid organ transplant, and immunosuppressive therapy.

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50

Daly, Ivonne M., and Ali Al-Khafaji. Intensive care management in hepatic and other abdominal organ transplantation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0371.

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Care of the transplant patient post-operatively requires a multidisciplinary approach. The goal of the intensivist is to create an ideal environment for the allograft to recover from its ischaemic insult and return to normal function. An understanding of the recipient’s pretransplant physiology is essential, as the pathological states associated with organ failure may persist for weeks to months after transplant. In particular, cardiac and renal disease may impact care in the immediate post-transplant period. An understanding of immune suppressive strategies will enable the intensivist to miti

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