Relevant bibliographies by topics / Immune response genes

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Immune response genes'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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Journal articles on the topic "Immune response genes"

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Du, Liping, Aize Kijlstra, and Peizeng Yang. "Immune Response Genes in Uveitis." Ocular Immunology and Inflammation 17, no. 4 (2009): 249–56. http://dx.doi.org/10.1080/09273940902999356.

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Cox, Diane W. "Genes of the immune response: disease associations." Genome 31, no. 2 (1989): 1085–86. http://dx.doi.org/10.1139/g89-186.

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Béhar, Ghislaine, Yves Bourlet, Nancy Fréchin, François Guillemot, Rima Zoorob, and Charles Auffray. "Molecular analysis of chicken immune response genes." Biochimie 70, no. 7 (1988): 909–17. http://dx.doi.org/10.1016/0300-9084(88)90232-5.

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Hartman, Zachary, Xiao Yi Yang, Gangjun Lei, et al. "Modulation of adenoviral vector immune responses through the over-expression of immune adaptor and viral immuno-modulatory genes (48.17)." Journal of Immunology 178, no. 1_Supplement (2007): S77—S78. http://dx.doi.org/10.4049/jimmunol.178.supp.48.17.

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Abstract For the past two decades, adenoviral vector platforms have been used as both gene therapy vehicles and vaccine platforms in various human clinical trials. However, the use of these, as well as other viral vector platforms, has been tempered by the innate immune and adaptive immune responses elicited. Despite the development of advanced generation adenoviral vectors, the innate responses precipitated by these vectors have remained largely unchanged due to the particular nature of the viral capsid and infectious process. To more effectively use these vectors for different purposes, we c
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Knapek, Katie J., Hanah M. Georges, Hana Van Campen, et al. "Fetal Lymphoid Organ Immune Responses to Transient and Persistent Infection with Bovine Viral Diarrhea Virus." Viruses 12, no. 8 (2020): 816. http://dx.doi.org/10.3390/v12080816.

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Bovine Viral Diarrhea Virus (BVDV) fetal infections occur in two forms; persistent infection (PI) or transient infection (TI), depending on what stage of gestation the fetus is infected. Examination of lymphoid organs from both PI and TI fetuses reveals drastically different fetal responses, dependent upon the developmental stage of the fetal immune system. Total RNA was extracted from the thymuses and spleens of uninfected control, PI, and TI fetuses collected on day 190 of gestation to test the hypothesis that BVDV infection impairs the innate and adaptive immune response in the fetal thymus
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Marchalonis, John J., Peter J. Morris, and Alan W. Harris. "SPECULATIONS ON THE FUNCTION OF IMMUNE RESPONSE GENES." International Journal of Immunogenetics 1, no. 1 (2007): 63–67. http://dx.doi.org/10.1111/j.1744-313x.1974.tb00292.x.

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Blumenthal, M., B. Johnson, D. Marcus, et al. "558 Immune response genes of ragweed sensitive individuals." Journal of Allergy and Clinical Immunology 81, no. 1 (1988): 307. http://dx.doi.org/10.1016/0091-6749(88)90792-0.

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Damulewicz, Milena, Michał Świątek, Agnieszka Łoboda, et al. "Daily Regulation of Phototransduction, Circadian Clock, DNA Repair, and Immune Gene Expression by Heme Oxygenase in the Retina of Drosophila." Genes 10, no. 1 (2018): 6. http://dx.doi.org/10.3390/genes10010006.

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The daily expression of genes and the changes in gene expression after silencing the heme oxygenase (ho) gene were examined in the retina of Drosophila using microarray and SybrGreen qPCR (quantitative polymerase chain reaction) methods. The HO decrease in the morning upregulated 83 genes and downregulated 57 genes. At night, 80 genes were upregulated and 22 were downregulated. The top 20 genes downregulated after ho silencing in the morning modulate phototransduction, immune responses, autophagy, phagocytosis, apoptosis, the carbon monoxide (CO) response, the oxidative stress/UV response, and
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Hsu, S. D., C. K. Anders, C. R. Acharya, et al. "Immune signatures hold prognostic import across solids tumors." Journal of Clinical Oncology 25, no. 18_suppl (2007): 21041. http://dx.doi.org/10.1200/jco.2007.25.18_suppl.21041.

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21041 Background: The host immune response can impact cancer growth, prognosis, and response to therapy. In colorectal cancer, the presence of cells involved with T-cell mediated adaptive immunity better predicts of survival than the current staging method. Immune signatures based on host response to cancer have the potential to predict prognosis and facilitate target specific therapy. Methods: We used the gene expression data associated with immune host response to colorectal cancer (Galon et al., Science 2006) to perform hierarchical clustering of solid tumors for which the clinical annotati
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Wu, Louisa P., Kwang-Min Choe, Yiran Lu, and Kathryn V. Anderson. "Drosophila Immunity: Genes on the Third Chromosome Required for the Response to Bacterial Infection." Genetics 159, no. 1 (2001): 189–99. http://dx.doi.org/10.1093/genetics/159.1.189.

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Abstract We have screened the third chromosome of Drosophila melanogaster for mutations that prevent the normal immune response. We identified mutant lines on the basis of their failure to induce transcription of an antibacterial peptide gene in response to infection or their failure to form melanized clots at the site of wounding. These mutations define 14 genes [immune response deficient (ird) genes] that have distinct roles in the immune response. We have identified the molecular basis of several ird phenotypes. Two genes, scribble and kurtz/modulo, affect the cellular organization of the f
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Dissertations / Theses on the topic "Immune response genes"

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Aldred, Patricia M. R. "Variation in human immune response genes." Thesis, University of Nottingham, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415720.

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Lorenzi, Roberto. "Studies on gene conversion as a mutational mechanism in the evolution of major histocompatibility complex genes." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336766.

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Milner, Caroline M. "Characterisation of novel genes in the human major histocompatibility complex : the HSP70 & G9a genes." Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302867.

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Ghaffari, Emma Louise Marie. "Early growth response genes -2 and -3 are essential for optimal immune responses." Thesis, Brunel University, 2013. http://bura.brunel.ac.uk/handle/2438/8134.

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Early Growth Response Genes (EGR) is a family of four transcription factors containing a unique zinc finger domain. EGR-2 and EGR-3 are important for hindbrain development and myelination. These transcription factors are also necessary for lymphocyte function however, the mechanisms are still unclear. Previous findings have shown that EGR-2cKO mice develop lupus-like autoimmune disease with high levels of pro-inflammatory cytokines despite showing normal T and B cell proliferation after mitogenic stimulation. Therefore we established the CD2-EGR-2-/-EGR-3-/- mouse model to explore the phenotyp
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Kendall, Elaine. "Molecular characterisation of the human major histocompatibility complex." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333402.

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O'Neill, Ann Marie Ewald Sandra J. "Polymorphism in chicken immune response genes and resistance to disease." Auburn, Ala., 2007. http://repo.lib.auburn.edu/2007%20Fall%20Dissertations/O'Neill_Ann_48.pdf.

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Daniels, Garry D. "Cloning and characterisation of cytokine and cytokine receptor genes in rainbow trout, Oncorhynchus mykiss." Thesis, University of Aberdeen, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265893.

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Convincing molecular evidence for the existence of both cytokines and their receptors in teleost fish is presented. TGF-β is present in <I>O. mykiss</I> encoding a 112 amino acid mature peptide. An integrin binding site (RGD) and a characteristic tetrabasic cleavage site (RKKR) are present, as is the TGF-β superfamily motif. The mature peptide has 9 conserved cysteine residues (8 of which occur in pairs) as well as two additional conserved TGF-β superfamily residues (Pro<sub>36</sub> and Gly<sub>46</sub>). TGF-β exhibits a wide range tissue distribution including head-kidney macrophages, PBL,
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Emonts, M. "Polymorphisms in immune response genes in infectious diseases and autoimmune diseases." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/14316.

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Modesto, P. "EXPRESSION PROFILE OF IMMUNE RESPONSE GENES IN GOATS WITH EXPERIMENTALLY INDUCED STAPHYLOCOCCUS AUREUS MASTITIS." Doctoral thesis, Università degli Studi di Milano, 2012. http://hdl.handle.net/2434/169565.

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Counteracting infectious diseases of farm animals are an everlasting challenge in food production from livestock and preserving the health of farm animals is highly relevant to maintaining high standards of food quality. Clinical mastitis (CM) is the primary health reason for involuntary culling in dairy small ruminants and causes additional economic losses from costs of veterinary treatments. Complementary strategies are needed, since the classical prophylactic measures sometimes appear too demanding to breeders in terms of time and care, and efficient vaccination against the main pathogens i
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MAGRO, GIADA. "BOVINE STAPHYLOCOCCUS AUREUS MASTITIS: FROM THE MAMMARY IMMUNE RESPONSE TO THE BACTERIA VIRULENCE GENES." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/548380.

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Staphylococcus aureus (S. aureus) is one of the most important bacteria in veterinary medicine. In dairy herds, it is a contagious bacterium responsible mainly for subclinical mastitis in cattle, which frequently gives rise to persistent and chronic infection. Mastitis cause considerable economic losses due to i) decreased milk production, (ii) reduced milk quality, and (iii) treatment costs. Mastitis is also a public health problem. Indeed, the strains isolated from infected glands could produce enterotoxins. Three factors interact in mastitis: the host, the pathogen and the environment. This
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Books on the topic "Immune response genes"

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Mak, Tak W. Handbook of immune response genes. Plenum Press, 1998.

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Marc, Feldmann, and McMichael Andrew J, eds. Regulation of immune gene expression. Humana Press, 1986.

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J, Kay, ed. Genes and proteins in immunity. Biochemical Society, 1986.

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B, Schook Lawrence, Tew John G, and International RES Symposium (1987 : Richmond, Va.), eds. Antigen presenting cells: Diversity, differentiation, and regulation : proceedings of a symposium held in Richmond, Virginia, March 26-29, 1987. Liss, 1988.

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Ottenhoff, Thomas Henricus Maria. HLA class II immune response genes in leprosy: Studies on the recognition of Mycobacterium leprae antigens and class II molecules by cloned human T cells. [s.n.], 1986.

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Derek, Wakelin, and Blackwell J. M, eds. Genetics of resistance to bacterial and parasitic infection. Taylor & Francis, 1988.

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1943-, Watson James D., and Marbrook John, eds. Recognition and regulation in cell-mediated immunity. M. Dekker, 1985.

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Mak, Tak W., and John J. L. Simard. Handbook of Immune Response Genes. Springer, 2013.

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Feldmann, Marc, and Andrew McMichael. Regulation of Immune Gene Expression. Humana Press, 2012.

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Feldmann, Marc, and Andrew McMichael. Regulation of Immune Gene Expression. Humana Press, 2012.

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Book chapters on the topic "Immune response genes"

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Winchester, Robert. "Human Immune Response Genes." In Immunopharmacology in Autoimmune Diseases and Transplantation. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4899-1167-4_1.

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Nepom, Gerald T., and John A. Hansen. "Human Immune Response Genes." In Immunology of Rheumatic Diseases. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2493-5_1.

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Bontrop, R. E. "MHC Genes, Immune Response, and Vaccines." In Molecular Biology and Evolution of Blood Group and MHC Antigens in Primates. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59086-3_20.

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GÜnther, E. "Heat Shock Protein Genes and the Major Histocompatibility Complex." In Heat Shock Proteins and Immune Response. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75875-1_3.

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Bell, J. I., and H. O. McDevitt. "Molecular Polymorphism of Human Immune-Response-Genes." In HLA Class II Antigens. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70367-6_26.

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Greco, Raffaella, and Dominique Farge. "CART Cells and Other Cell Therapies (ie MSC, Tregs) in Autoimmune Diseases." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_93.

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AbstractAuto-immune diseases (AD) are heterogeneous conditions, characterized by polyclonal activation of the immune system with a defect of B or T lymphocyte selection and altered lymphocytic reactions to auto-antigens components (Burnet 1959a, b), although it is rare to identify a single antigenic epitope. The native immune system and its tissue environment play an important role to determine if exposure to a given antigen will induce an immune response or tolerance or anergy. The role of the genes coding for the major histocompatibility system molecules, but also of many other genes, is important in the regulation of the immune response, although this does not explain all the observed phenomena during loss of tolerance (Matzinger 1994; Rioux and Abbas 2005).
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Meignier, Bernard, and Bernard Roizman. "Genetic Engineering and Properties of Novel Herpes Simplex Viruses for Use as Potential Vaccines and as Vectors of Foreign Genes." In The Immune Response to Viral Infections. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5712-4_17.

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Allan, Alison L., and Ann F. Chambers. "Genes and metastasis: experimental advances and clinical implications." In Selected Aspects of Cancer Progression: Metastasis, Apoptosis and Immune Response. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6729-7_4.

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Running, Katherine L. D., and Justin D. Faris. "Rapid Cloning of Disease Resistance Genes in Wheat." In Compendium of Plant Genomes. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-38294-9_10.

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AbstractWheat is challenged by rapidly evolving pathogen populations, resulting in yield losses. Plants use innate immune systems involving the recognition of pathogen effectors and subsequent activation of defense responses to respond to pathogen infections. Understanding the genes, genetic networks, and mechanisms governing plant-pathogen interactions is key to the development of varieties with robust resistance whether through conventional breeding techniques coupled with marker selection, gene editing, or other novel strategies. With regards to plant-pathogen interactions, the most useful targets for crop improvement are the plant genes responsible for pathogen effector recognition, referred to as resistance (R) or susceptibility (S) genes, because they govern the plant’s defense response. Historically, the molecular identification of R/S genes in wheat has been extremely difficult due to the large and repetitive nature of the wheat genome. However, recent advances in gene cloning methods that exploit reduced representation sequencing methods to reduce genome complexity have greatly expedited R/S gene cloning in wheat. Such rapid cloning methods referred to as MutRenSeq, AgRenSeq, k-mer GWAS, and MutChromSeq allow the identification of candidate genes without the development and screening of high-resolution mapping populations, which is a highly laborious step often required in traditional positional cloning methods. These new cloning methods can now be coupled with a wide range of wheat genome assemblies, additional genomic resources such as TILLING populations, and advances in bioinformatics and data analysis, to revolutionize the gene cloning landscape for wheat. Today, 58 R/S genes have been identified with 42 of them having been identified in the past six years alone. Thus, wheat researchers now have the means to enhance global food security through the discovery of R/S genes, paving the way for rapid R gene deployment or S gene elimination, manipulation through gene editing, and understanding wheat-pathogen interactions at the molecular level to guard against crop losses due to pathogens.
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Nissar, Saniya, Aga Syed Sameer, and Mujeeb Zafar Banday. "Genetic Polymorphisms of Essential Immune Pathogenic Response Genes and Risk of Cervical Cancer." In Genetic Polymorphism and cancer susceptibility. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6699-2_7.

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Conference papers on the topic "Immune response genes"

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Zinovieva, S. V., Z. V. Udalova, and F. K. Khasanov. "EXPRESSION OF IMMUNE SYSTEM GENES IN TOMATO PLANTS INFECTED BY MELOIDOGYNE INCOGNITA." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2024. http://dx.doi.org/10.31016/978-5-6050437-8-2.2024.25.135-139.

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Data were presented from a study on expression of resistance gene Mi-1.2 of protective genes of the PR gene family (PR-2, PR-3) and genes of serine and cysteine proteinase (PIser PIcys) inhibitors in tissues of tomato plants of resistant and susceptible hybrids infected by gall nematodes and an assessment of their role in parasite resistance was given. Differences were detected in the expression of the studied genes at all stages of nematode development in the roots of resistant and susceptible plants. The studies showed that the infection of resistant plants caused an increase in the study ge
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Liu, Xiao-Ling. "DECIPHERING THE GENETIC LINKS BETWEEN PSYCHOLOGICAL STRESS, AUTOPHAGY, AND DERMATOLOGICAL HEALTH: INSIGHTS FROM BIOINFORMATICS, SINGLE-CELL ANALYSIS, AND MACHINE LEARNING IN PSORIASIS AND ANXIETY DISORDERS." In BioClina 2024 – International Conference on Biological & Clinical Studies, 21-22 June, Singapore. Global Research & Development Services, 2024. http://dx.doi.org/10.20319/icrlsh.2024.8687.

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The relationship between psychological stress, altered skin immunity, and autophagy-related genes (ATGs) is currently unclear. Psoriasis is a chronic skin inflammation of unclear etiology that is characterized by persistence and recurrence. Immune dysregulation and emotional disturbances are recognized as significant risk factors. Emerging clinical evidence suggests a possible connection between anxiety disorders, heightened immune system activation, and altered skin immunity, offering a fresh perspective on the initiation of psoriasis. The aim of this study was to explore the potential shared
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Uvarova, A. N., E. A. Tkachenko, K. V. Korneev, and D. V. Kuprash. "FUNCTIONAL ANALYSIS OF SNPS ASSOCIATED WITH SEVERE VIRAL RESPIRATORY DISEASES AND LOCATED IN THE REGULATORY REGIONS OF ANTIVIRAL IMMUNE RESPONSE GENES." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-378.

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We investigated the regulatory regions of the CD55, LGALS1, and IFNAR2 genes in cell models of human macrophages and B-cells and analyzed several SNPs located in the regulatory regions of these genes and associated with severe COVID-19 or flu. In the course of this work, we characterized the IFNAR2 gene enhancer and performed functional annotation of several SNPs in the CD55 and LGALS1 promoters.
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Muntyan, Victoria S., Alla S. Saksaganskaia, Alexey N. Muntyan, Mariia E. Vladimirova, and Marina L. Roumiantseva. "STRESS AND IMMUNITY OF NODULE BACTERIA SINORHIZOBIUM MELILOTI: LOCALIZATION, POLYMORPHISM AND PHYLOGENY OF GENETIC DETERMINANTS." In 22nd SGEM International Multidisciplinary Scientific GeoConference 2022. STEF92 Technology, 2022. http://dx.doi.org/10.5593/sgem2022/6.1/s25.15.

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Sinorhizobium meliloti are agriculturally valuable species of soil bacteria that form nitrogen-fixing symbiosis with alfalfa plants. Global climate changes lead to an increase of agricultural areas subjected to salinity. Current knowledge about about high-salt stress impact on soil saprophitic root nodulated microsymbionts of legumes is weakly studied and rhizobia gene pool responsible for salt tolerance are fragment and far from clear. An increase of bacteria nonspecific resistance (immune status) to unfavorable stress factors can occur through the induction of defense mechanisms like restric
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Buslaev, V. Yu, A. V. Torgunakova, Irina Milentyeva, Lyubov Dyshlyuk, and V. I. Minina. "POPYMORPHISM OF IMMUNE RESPONSE GENES AND LUNG CANCER RISK IN NON-SMOKING RESIDENTS OF KUZBASS." In I International Congress “The Latest Achievements of Medicine, Healthcare, and Health-Saving Technologies”. Kemerovo State University, 2023. http://dx.doi.org/10.21603/-i-ic-17.

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Lung cancer (LC) is leading oncological pathology, posing a serious threat for patient’s lives.&#x0D; Accordingly to World Health Organization (WHO) 2,1 million of new cases and 1,8 of deaths are&#x0D; annually registered. It was accumulated a lot of information about significant influence of smoking on&#x0D; increased risk of LC development. 80-90% of patients with LC are namely smokers. However at present&#x0D; time it was registered increased level of mortality from this pathology among non-smoking patients [1].&#x0D; LC formation in non-smoking individuals can occur due to environmental po
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Božić, Dragica, Katarina Živančević, Katarina ,. Baralić, et al. "APPLYING „IN SILICO“ TOXICOGENOMIC DATA MINING TO PREDICT MOLECULAR MECHANISMS AND PATHWAYS AGAINST CARCINOMA: IMMUNOMODULATOR SULFORAPHANE AS A CASE STUDY." In 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac,, 2021. http://dx.doi.org/10.46793/iccbi21.470b.

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The aim of this study was to predict the molecular mechanisms and pathways of immunomodulator sulforaphane (SFN) against carcinoma using in silico toxicogenomic data mining. Three key tools applied in our analysis were Comparative Toxicogenomics Database (CTD; http://CTD.mdibl. org), ToppGene Suite portal (https://toppgene.cchmc.org) and Reactome Knowledgebase (https://reactome.org). Sulforaphane interacted with a total of 1896, among which NFE2L2, NQO1, HMOX1, GCLC, TXNRD1, IL1B, IFNG, AGT, KEAP1, and CASP3 had the highest number of interactions. In the CTD, there were direct evidences that S
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Chumacheva, Yu V., and D. S. Stashkevich. "COMBINATIONS OF SNPS TNFRSF11B AND TNFA GENOTYPES IN PATIENTS WITH RHEUMATOID ARTHRITIS OF THE BASHKIR POPULATION IN CHELYABINSK REGION." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-392.

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Cytokine genes influence the nature of the immune response in rheumatoid arthritis (RA) through the level of production of encoded proteins, which makes the functional polymorphism of cytokine genes particularly interesting for research in RA. In our study, we evaluated combinations of frequencies of combinations genotype of polymorphisms of TNFRSF11B genes at point G1181C and TNFA at points G-308A, G-238A in population of Bashkir RA patients and a control group to identify possible associations.
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Han, Sohee, Qing Lan, Ae Kyung Park, et al. "Abstract 925: Common variants in genes related to immune response and childhood leukemia risk." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-925.

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Mbofung, Rina M., Jodi A. McKenzie, Shruti Malu, et al. "Abstract 4360: Inhibition of HSP90 enhances T cell-mediated antitumor immune responses through expression of interferon-alpha response Genes." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4360.

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Kamareddine, Layla, Hoda Najjar, Abeer Mohbeddin, Nawar Haj Ahmed, and Paula Watnick. "Between Immunity, Metabolism, and Development: A story of a Fly Gut!" In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0141.

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In addition to its role in initiating immune response in the body, the innate immune system seems to also play a critical role in maintaining homeostatic balance in the gut epithelium. Our recent studies in the Drosophila melanogaster fruit fly model suggest that different innate immune pathways contribute to this homeostatic balance through activating the transcription of genes encoding antimicrobial peptides. We provide evidence that several metabolic parameters are altered in immune deficient flies. We also highlight a role of the gut flora, particularly through its short chain fatty acid,
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Reports on the topic "Immune response genes"

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Cahaner, Avigdor, Susan J. Lamont, E. Dan Heller, and Jossi Hillel. Molecular Genetic Dissection of Complex Immunocompetence Traits in Broilers. United States Department of Agriculture, 2003. http://dx.doi.org/10.32747/2003.7586461.bard.

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Objectives: (1) Evaluate Immunocompetence-OTL-containing Chromosomal Regions (ICRs), marked by microsatellites or candidate genes, for magnitude of direct effect and for contribution to relationships among multiple immunocompetence, disease-resistance, and growth traits, in order to estimate epistatic and pleiotropic effects and to predict the potential breeding applications of such markers. (2) Evaluate the interaction of the ICRs with genetic backgrounds from multiple sources and of multiple levels of genetic variation, in order to predict the general applicability of molecular genetic marke
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Evans, Donald L., Avigdor Eldar, Liliana Jaso-Friedmann, and Herve Bercovier. Streptococcus Iniae Infection in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Towards the Pathogen and Vaccine Formulation. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7586538.bard.

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The objectives of the BARD proposal were to determine the mechanisms of nonspecific cytotoxic cells (NCC) that are necessary to provide heightened innate resistance to infection and to identify the antigenic determinants in Streptococcus iniae that are best suited for vaccine development. Our central hypothesis was that anti-bacterial immunity in trout and tilapia can only be acquired by combining "innate" NCC responses with antibody responses to polysaccharide antigens. These Objectives were accomplished by experiments delineated by the following Specific Aims: Specific aim (SA) #1 (USA) "Clo
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Chejanovsky, Nor, and Bruce A. Webb. Potentiation of pest control by insect immunosuppression. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7587236.bard.

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Our original aims were to elucidate the mechanisms through which the immunosuppressive insect virus, the Campoletis sonorensis polydnavirus (CsV) promotes replication of a well-characterized pathogenic virus, the Autographa californica multiple nucleopolyhedrovirus (AcMNPV) in hosts that are mildly or non-permissive to virus replication. According to the BARD panels criticism we modified our short-term goals (see below). Thus, in this feasibility study (one-year funding) we aimed to show that: 1. S. littoralis larvae mount an immune response against a baculovirus infection. 2. Immunosuppressio
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Chejanovsky, Nor, and Bruce A. Webb. Potentiation of Pest Control by Insect Immunosuppression. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592113.bard.

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The restricted host range of many baculoviruses, highly pathogenic to Lepidoptera and non-pathogenic to mammals, limits their use to single or few closely related Lepidopteran species and is an obstacle to extending their implementation for pest control. The insect immune response is a major determinant of the ability of an insect pathogen to efficiently multiply and propagate. We have developed an original model system to study the Lepidopteran antiviral immune response based on Spodoptera littoralis resistance to AcMNPV (Autographa californica multiple nucleopolyhedrovirus) infection and the
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David, Lior, Yaniv Palti, Moshe Kotler, Gideon Hulata, and Eric M. Hallerman. Genetic Basis of Cyprinid Herpes Virus-3 Resistance in Common Carp. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7592645.bard.

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The goal of this project was to provide scientific and technical basis for initiating the development of breeding protocols using marker assisted selection for viral disease resistance in common carp. The specific objectives were: 1) Establishing families and characterizing the phenotypic and genetic variation of viral resistance; 2) Measuring the dynamics of immune response and developing a method to measure the long term immune memory; 3) Developing markers and generating a new genetic linkage map, which will enable initial QTL mapping; and, 4) Identifying genetic linkage of markers and cand
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McElwain, Terry F., Eugene Pipano, Guy H. Palmer, Varda Shkap, Stephn A. Hines, and Wendy C. Brown. Protection of Cattle against Babesiosis: Immunization against Babesia bovis with an Optimized RAP-1/Apical Complex Construct. United States Department of Agriculture, 1999. http://dx.doi.org/10.32747/1999.7573063.bard.

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Previous research and current efforts at control of babesiosis fall short of meeting the needs of countries where the disease is endemic, such as Israel, as well as the needs of exporting countries and countries bordering on endemic areas, such as the U.S. Our long-term goal is to develop improved methods of immunization against bovine babesiosis based on an understanding of the molecular mechanisms of immune protection and parasite targets of a protective immune response. In our previous BARD project, we established the basis for focusing on rhoptry antigens as components of a subunit vaccine
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Shpigel, Nahum Y., Ynte Schukken, and Ilan Rosenshine. Identification of genes involved in virulence of Escherichia coli mastitis by signature tagged mutagenesis. United States Department of Agriculture, 2014. http://dx.doi.org/10.32747/2014.7699853.bard.

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Mastitis, an inflammatory response of the mammary tissue to invading pathogenic bacteria, is the largest health problem in the dairy industry and is responsible for multibillion dollar economic losses. E. coli are a leading cause of acute mastitis in dairy animals worldwide and certainly in Israel and North America. The species E. coli comprises a highly heterogeneous group of pathogens, some of which are commensal residents of the gut, infecting the mammary gland after contamination of the teat skin from the environment. As compared to other gut microflora, mammary pathogenic E. coli (MPEC) m
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Eldar, Avigdor, and Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575286.bard.

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In Israel and in the U.S., Streptococcus iniae is responsible for considerable losses in various fish species. Poor understanding of its virulence factors and limited know-how-to of vaccine formulation and administration are the main reasons for the limited efficacy of vaccines. Our strategy was that in order to Improve control measures, both aspects should be equally addressed. Our proposal included the following objectives: (i) construction of host-pathogen interaction models; (ii) characterization of virulence factors and immunodominant antigens, with assessment of their relative importance
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Sessa, Guido, and Gregory B. Martin. molecular link from PAMP perception to a MAPK cascade associated with tomato disease resistance. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7597918.bard.

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The research problem: The detection of pathogen-associated molecular patterns (PAMPs) by plant pattern recognition receptors (PRRs) is a key mechanism by which plants activate an effective immune response against pathogen attack. MAPK cascades are important signaling components downstream of PRRs that transduce the PAMP signal to activate various defense responses. Preliminary experiments suggested that the receptor-like cytoplasmickinase (RLCK) Mai5 plays a positive role in pattern-triggered immunity (PTI) and interacts with the MAPKKK M3Kε. We thus hypothesized that Mai5, as other RLCKs, fun
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Chejanovsky, Nor, Diana Cox-Foster, Victoria Soroker, and Ron Ophir. Honeybee modulation of infection with the Israeli acute paralysis virus, in asymptomatic, acutely infected and CCD colonies. United States Department of Agriculture, 2013. http://dx.doi.org/10.32747/2013.7594392.bard.

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Honey bee (Apis mellifera) colony losses pose a severe risk to the food chain. The IAPV (Israeli acute paralysis virus) was correlated with CCD, a particular case of colony collapse. Honey bees severely infected with IAPV show shivering wings that progress to paralysis and subsequent death. Bee viruses, including IAPV, are widely present in honey bee colonies but often there are no pathological symptoms. Infestation of the beehive with Varroa mites or exposure to stress factors leads to significant increase in viral titers and fatal infections. We hypothesized that the honey bee is regulating/
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