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1

C, Prendergast George, and Jaffee Elizabeth M, eds. Cancer immunotherapy: Immune suppression and tumor growth. Academic Press, 2007.

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2

I, Gabrilovich Dmitry, and Hurwitz Arthur A, eds. Tumor-induced immune suppression: Mechanisms and therapeutic reversal. Springer, 2008.

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3

Cancer Immunotherapy: Immune Suppression and Tumor Growth. Academic Press, 2007.

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Cancer immunotherapy: Immune suppression and tumor growth. Elsevier Academic Press, 2007.

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5

Prendergast, George C., and Elizabeth M. Jaffee. Cancer Immunotherapy: Immune Suppression and Tumor Growth. Elsevier Science & Technology Books, 2011.

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6

(Editor), George C. Prendergast, and Elizabeth M. Jaffee (Editor), eds. Cancer Immunotherapy: Immune Suppression and Tumor Growth. Academic Press, 2007.

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7

Prendergast, George C., and Elizabeth M. Jaffee. Cancer Immunotherapy: Immune Suppression and Tumor Growth. Elsevier Science & Technology Books, 2013.

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8

Suppression and regulation of immune responses: Methods and protocols. Humana Press, 2011.

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9

Cuturi, Maria Cristina, and Ignacio Anegon. Suppression and Regulation of Immune Responses: Methods and Protocols. Humana Press, 2016.

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10

Suppression and Regulation of Immune Responses: Methods and Protocols. Humana Press, 2011.

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11

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2016.

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12

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2010.

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13

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer London, Limited, 2014.

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14

Gabrilovich, Dmitry I., and Arthur Andrew Hurwitz. Tumor-Induced Immune Suppression: Mechanisms and Therapeutic Reversal. Springer, 2014.

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15

Cuturi, Maria Cristina, and Ignacio Anegon. Suppression and Regulation of Immune Responses: Methods and Protocols, Volume II. Springer New York, 2016.

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16

Cuturi, Maria Cristina, and Ignacio Anegon. Suppression and Regulation of Immune Responses: Methods and Protocols, Volume II. Springer New York, 2015.

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17

Misbah, Siraj. Immunosuppressive therapy and therapeutic monoclonal antibodies. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0302.

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The term immunosuppressive therapy encompasses all forms of treatment that dampens function of the recipient’s immune system, with a view to controlling severe autoimmune, inflammatory, or allergic disease. The predominant targets of these agents are T-lymphocytes with multiple downstream effects, including containment of T-cell activation, inhibition of cytokine production, restriction of clonal expansion, and varying degrees of suppression of B-cell function. This chapter reviews the clinical use of monoclonal antibodies and other immunosuppressive agents, and their mechanisms of action.
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18

Abe, Hiroyuki, Amane Sasada, Shigeki Tabata, and Minako Abe. Heat Shock Protein Vaccine Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0009.

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Despite advances in chemotherapeutic regimens, ovarian cancer has a poor prognosis. Therefore important effective treatments are urgently needed. Many studies have reported that the immune system plays a critical role in disease progression and overall survival. One known effective immunotherapy is the dendritic cell (DC)-based vaccine pulsed with tumor-associated antigens. This chapter reports on a method of production of a novel DC-based vaccine. The key technologies are (a) monocyte collection without leukapheresis, (b) monocyte expansion, (c) production of dendritic cells, (d) multiple ove
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19

Kulkarni, Kunal, James Harrison, Mohamed Baguneid, and Bernard Prendergast, eds. Transplantation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198729426.003.0030.

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Organ transplantation is now a well-established therapy for patients with end-stage organ failure. Over the last 20 years, the results of transplantation have improved incrementally for many reasons, including better recipient selection, improved anaesthetic and surgical techniques, the introduction of more effective antiviral agents, and better post-transplant immunosuppressive management. The problem of early graft loss from acute rejection is now uncommon, and the main challenges today are chronic allograft rejection and the side effects of non-specific suppression of the immune response. R
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20

Muche, Marion, and Seema Baid-Agrawal. Hepatitis B. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0185_update_001.

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Hepatitis B virus (HBV) has been causally linked to a variety of renal diseases, the most common being glomerular diseases and systemic autoimmune disease. Membranous nephropathy (MN) is the commonest HBV-associated glomerulonephritis (HBV-GN), followed by membranoproliferative glomerulonephritis (MPGN), mesangial proliferative glomerulonephritis, immunoglobulin (Ig)-A nephropathy, and focal segmental glomerulosclerosis (FSGS). Polyarteritis nodosa is a rare manifestation. The incidence of HBV-associated renal diseases seems to be decreasing with the introduction of vaccination programmes.HBV-
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