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Relevant bibliographies by topics / Immunodeficit

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Academic literature on the topic 'Immunodeficit'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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Journal articles on the topic "Immunodeficit"

1

Bonilla, Francisco A., and Raif S. Geha. "Are you immunodeficient?" Journal of Allergy and Clinical Immunology 116, no. 2 (August 2005): 423–25. http://dx.doi.org/10.1016/j.jaci.2005.05.026.

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Bondarenko, Andrey V., Sergey I. Pokhil, Marianna V. Lytvynenko, Tatyana V. Bocharova, and Vitaliy V. Gargin. "_EXPERIMENTAL IMMUNODEFICIENT STATE MODEL." Wiadomości Lekarskie 72, no. 9 (2019): 1761–64. http://dx.doi.org/10.36740/wlek201909210.

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Introduction: The recently described anaplasmosis infection is widespread but concerns to the insufficiently known group of diseases. The aim of our research is the development of uniform biological model for reproducing of artificial immunodeficient state by experimental anaplasmosis. Materials and methods: Algorithm of experimental anaplasmosis reproducing, consisted of such consecutive stages: 1) artificial forming of the immunodeficient state at nonlinear white mise (Mus musculus L.); 2) preparation of the tested biological material samples; 3) inoculation by prepared samples of the laboratory animals with the artificially formed immunodeficient state; 4) sampling from the dead or slaughtered (by the method of chloroformed anesthesia) experimental animals of sectional material (organs and targets tissues); 5) verification of aetiology by express detection of causative agents by the method of PCR in the selected samples of sectional material. Results: Biological model of experimental anaplasmosis have been created suitable for realization of both diagnostic and epidemiological, epizootic, ecobiological and other researches of different origin biological material samples, including samples of solid and liquid consistency material. Formed model realised in premature death of experimental animals in 17.4 % cases; resulted in an onset of disease clinical signs without death during the term of supervision in 43.8 % cases; coursed in the absence of the expressed symptoms of infection in 31.3 % cases. Conclusions: Developed biological model of experimental anaplasmosis consists in that as laboratory animals with the increased sensitiveness to the infection and accumulation of causative agent are used white nonlinear mice with the artificially formed immunodeficient state.

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Brown, Karen L., Karen Stewart, Moira E. Bruce, and Hugh Fraser. "Scrapie in Immunodeficient mice." Biochemical Society Transactions 25, no. 2 (May 1, 1997): 173S. http://dx.doi.org/10.1042/bst025173s.

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Le Bras, Alexandra. "A new immunodeficient pig model." Lab Animal 48, no. 7 (June 19, 2019): 198. http://dx.doi.org/10.1038/s41684-019-0339-1.

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JAVIER, LAZO, ANTONIO CARLOS OLIVEIRA MENESES, ADEMIR ROCHA, MARCELO SIMÃO FERREIRA, JAIME OLAVO MARQUEZ, EDMUNDO CHAPADEIRO, and EDILSON REIS LOPES. "Chagasic meningoencephalitis in the immunodeficient." Arquivos de Neuro-Psiquiatria 56, no. 1 (March 1998): 93–97. http://dx.doi.org/10.1590/s0004-282x1998000100015.

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Based on their own experience and on the literature, the authors compare the brain pathology due to HIV+ associated Trypanosoma cruzi reactived infection to that described for the natural history of the Chagas' disease (CD). The peculiar focal necrotizing chagasic meningoencephalitis (MECNF) which appears only in immunedeficient chagasics, especially when the deficiency is due HIV is a safe criterion for reactivation of CD. MECNF morphologic findings are unlike to those found either for some cases of acute phase CD or for chronic nervous form of CD.

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Leonard, Arnold S., Michael W. Mulholland, and Alexandra H. Filipovich. "Surgery of the Immunodeficient Child." Surgical Clinics of North America 65, no. 6 (December 1985): 1505–25. http://dx.doi.org/10.1016/s0039-6109(16)43784-9.

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Boshoff, C., and R. Weiss. "Cancer processes in immunodeficient populations." European Journal of Cancer 37, no. 10 (July 2001): 1202–8. http://dx.doi.org/10.1016/s0959-8049(01)00114-9.

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Belizario, Jose E. "Immunodeficient Mouse Models: An Overview." Open Immunology Journal 2, no. 1 (August 19, 2009): 79–85. http://dx.doi.org/10.2174/1874226200902010079.

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Kaplan, Jonathan. "Opportunistic Infections in Immunodeficient Populations." Emerging Infectious Diseases 4, no. 3 (September 1998): 421–22. http://dx.doi.org/10.3201/eid0403.980321.

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Castanet, J., F. Monpoux, R. Mariani, J. P. Ortonne, and J. Ph Lacour. "Demodicidosis in an Immunodeficient Child." Pediatric Dermatology 14, no. 3 (May 1997): 219–20. http://dx.doi.org/10.1111/j.1525-1470.1997.tb00242.x.

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Dissertations / Theses on the topic "Immunodeficit"

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Arveiler, Benoît. "Biologie moleculaire de maladies liees au chromosome x : localisation des genes responsables de trois immunodeficiences et de deux formes de retard mental non specifique, cartographie genetique et physique de la region xq26-q28 contenant le locus de l'x fragile." Université Louis Pasteur (Strasbourg) (1971-2008), 1988. http://www.theses.fr/1988STR13191.

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Messiah, Antoine. "Etude cas-temoin du sida chez les homosexuels masculins en france." Paris 7, 1987. http://www.theses.fr/1987PA077277.

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Gan, Jade Ho Yue School of Biomedical Engineering UNSW. "Characterisation of bone defect models in immunodeficient animals." Awarded by:University of New South Wales. School of Biomedical Engineering, 2005. http://handle.unsw.edu.au/1959.4/22429.

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Bone defects resulting from non-unions, fractures, significant revision joint replacements, tumour resection and osteolysis present a clinical problem. While autografts are considered the gold standard, ubiquitous use of this reparative technique is limited by graft supply and site morbidity. Recent progresses in tissue engineering using stem cells, bone enhancing molecules and gene therapy have provided more hypotheses for bone defect treatment. In vivo assessment to test these hypotheses requires animal models to mimic human conditions. Immunodeficient or nude animals have the advantage of hosting materials from human and other xenographic origins without immuno-intolerance or rejection. A thorough understanding of the biology in nude animals is vital for the further advancement of connective tissue healing and regeneration strategies. Nude mice are excellent xenographic hosts for in- vivo characterisation and provide a reproducible animal source. The immune deficiencies of nude compared to normal animals may however, influence bone healing and need to be addressed. This dissertation (a) investigated potential bone defect models in nude mice and nude rats (b) incorporated the selected bone defect model to evaluate the effect of T cell deficiency and age on bone defect healing in nude animals (c) determined the feasibility of a critical size defect (CSD) in nude mice. A distal-femur-condylar-defect (DFCD) model was successfully performed in nude mice and rats. The model was found to have some advantages as a bone defect model: (1) located at a weight-bearing skeletal site (2) no requirements for an internal or external fixator (3) does not obstruct or limit mobility (4) location is not in close proximity to any major organs such as the brain (5) easy identification of surface anatomy (6) defect size is standardised and reproducible (7) does not require lengthy and complicated surgery and (8) cost effective. This dissertation confirmed that bone healing in nude mice is similar to that of normal immunocompetent mice. Absence of T lymphocytes did not delay or inhibit bone repair. Use of older nude mice did not seem to affect the healing rate, in contrast to older normal mice, which showed delay in bone healing in the initial phase. Establishment of critical sized defects in mice at a weight-bearing location was not feasible due to the robust healing of murine. This dissertation recommends that the DFCD model could be utilized for the assessment of xenogenic materials at early time point.

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Aucouturier, Pierre. "Etude des sous-classes d'immunoglobulines chez l'homme : analyse quantitative par immunoenzymologie à l'aide d'anticorps mononclonaux." Poitiers, 1987. http://www.theses.fr/1987POIT2027.

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Engström, Gunilla Norhagen. "Mucosal conditions in immunodeficient individuals with special emphasis on salivary immunoglobulins." Stockholm : Department of Clinical Immunology, and the Department of Periodontology, Karolinska Institutet, 1993. http://catalog.hathitrust.org/api/volumes/oclc/28436070.html.

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Antognoli, Agnese <1981&gt. "Rag2-/-;gammac-/- immunodeficient mice, a new preclinical model to study antitumor approaches." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2010. http://amsdottorato.unibo.it/2333/.

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Animal models have been relevant to study the molecular mechanisms of cancer and to develop new antitumor agents. Anyway, the huge divergence in mouse and human evolution made difficult the translation of the gained achievements in preclinical mouse based studies. The generation of clinically relevant murine models requires their humanization both concerning the creation of transgenic models and the generation of humanized mice in which to engraft a functional human immune system, and reproduce the physiological effects and molecular mechanisms of growth and metastasization of human tumors. In particular, the availability of genotypically stable immunodepressed mice able to accept tumor injection and allow human tumor growth and metastasization would be important to develop anti-tumor and anti-metastatic strategies. Recently, Rag2-/-;gammac-/- mice, double knockout for genes involved in lymphocyte differentiation, had been developed (CIEA, Central Institute for Experimental Animals, Kawasaki, Japan). Studies of human sarcoma metastasization in Rag2-/-; gammac-/- mice (lacking B, T and NK functionality) revealed their high metastatic efficiency and allowed the expression of human metastatic phenotypes not detectable in the conventionally used nude murine model. In vitro analysis to investigate the molecular mechanisms involved in the specific pattern of human sarcomas metastasization revealed the importance of liver-produced growth and motility factors, in particular the insulin-like growth factors (IGFs). The involvement of this growth factor was then demonstrated in vivo through inhibition of IGF signalling pathway. Due to the high growth and metastatic propensity of tumor cells, Rag2-/-;gammac-/- mice were used as model to investigate the metastatic behavior of rhabdomyosarcoma cells engineered to improve the differentiation. It has been recently shown that this immunodeficient model can be reconstituted with a human immune system through the injection of human cord blood progenitor cells. The work illustrated in this thesis revealed that the injection of different human progenitor cells (CD34+ or CD133+) showed peculiar engraftment and differentiation abilities. Experiments of cell vaccination were performed to investigate the functionality of the engrafted human immune system and the induction of specific human immune responses. Results from such experiments will allow to collect informations about human immune responses activated during cell vaccination and to define the best reconstitution and experimental conditions to create a humanized model in which to study, in a preclinical setting, immunological antitumor strategies.

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Pumeechockchai, Wanna. "Density heterogeneity of hepatitis C virus RNA in immunocompetent and immunodeficient patients." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324936.

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Britt, Nicholas Mason, Madeleine Kate Miller, Donald B. Ph D. Hoover, and John B. M. D. Schweitzer. "IMMUNODEFICIENT R2G2 MOUSE STRAIN YIELDS SPLEENS WITH UNUSUAL CYTOARCHITECTURE AND SYMPATHETIC INNERVATION." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/asrf/2018/schedule/205.

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The nervous system and immune system contact one another through two-way communication in order to establish and preserve homeostasis. The sympathetic neurotransmitter norepinephrine has an impact on how the immune system responds by affecting regional blood flow and activation of adrenergic receptors on leukocytes. Former studies showed that immune cells are capable of releasing nerve growth factor allowing for the establishment and continuation of sympathetic nerves in targeted tissues. From this gathered information, it was hypothesized that sympathetic nerves would prove to be less frequent in spleens from the immunodeficient R2G2 mouse strain (Envigo) when compared to 129P3/J (129) and C57BL/6 (C57) strains. R2G2 mice are an immunodeficient strain that lacks functional T, B, and natural killer cells. Ten to eleven week aged-matched male mice were measured by body weight, spleen weight, and temperature. Spleens were cut and fixed for histological investigation. Sympathetic nerves were labeled by immunostaining tyrosine hydroxylase (TH). Hematoxylin & eosin (H&E) was used to stain spleen sections in order to evaluate cytoarchitecture. Von Willebrand factor (VWF) was used to immunostain for megakaryocytes. R2G2 mice showed slightly higher temperatures and body weights but yielded a significantly smaller spleen weight (R2G2, 38.20 ± 1.48; 129, 65.08 ± 11.71; C57, 81.33 ± 8.38; P< 0.0001, ANOVA). TH stain revealed sympathetic innervation in all strains but location and morphology differed in R2G2 mice compared to controls. Control spleens had nerves which entered white pulp regions of the spleen and were closely related to leukocytes. Fiber profiles in the controls were filamentous with small acute bends. R2G2 differed by having (TH+) nerve fibers more associated with arteries and less localized in the surrounding parenchyma. The fibers were abnormally swollen and held a more granular shape instead of a filamentous shape. The H&E stain showed clear red and white pulp zones in the control spleens with 129 showing more distinct germinal centers than C57. R2G2 H&E sections showed cytoarchitecture with indistinct pulp areas. VWF staining revealed R2G2 mice had an abundant amount of megakaryocytes versus control mice megakaryocyte counts (R2G2, 11.28 ± 3.87 per 20X field; 129, 1.73 ± 0.70; C57, 1.42 ± 0.13; P< 0.0001, ANOVA) and extramedullary hematopoiesis was highly prominent. This evidence supports that leukocytes secrete neurotrophic factors or are vital to establishing normal growth of TH+ nerves toward the white pulp. Leukocytes may not be required for sympathetic innervation of blood vessels in the spleen, however, lack of leukocytes shows TH+ nerve fibers with abnormal morphology in severely immune threatened mice.

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Fell, A. H. "Studies on the growth of Theileria infected bovine cells in immunodeficient mice." Thesis, University of Edinburgh, 1990. http://hdl.handle.net/1842/14831.

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The experiments described in this thesis investigated the growth of bovine cells infected with Theileria sp. macroschizonts in various strains of immunodeficient mice. It was hoped that it would be possible to create a system for the growth of Theileria infected cells in vivo without using bovine hosts. Previous work had shown that T.parva infected cells would establish as subcutaneous tumours in irradiated Swiss and nude mice (Irvin et al, 1977; Veterinary Parasitology 3, p.141-160). Most of the work in this thesis concerned the related parasite, T.annulata. T.anmilata infected cells were found to grow as subcutaneous tumours in irradiated Balb/c, irradiated NIMR and Balb/c nude, and unirradiated C.B-17 scid (severe combined immunodeficiency) mice. Infected cells failed to establish in C57 beige mice, with or without irradiation. The growth and survival of the tumours was dependent on the degree of immunosuppression of the host, and the size of the cell dose administered. In Balb/c mice, T.annulata tumours regressed as mice recovered from irradiation. Analysis of lymphocyte subsets using a fluorescence-activated cell sorter (FACS) showed differential susceptibility of B-cells, T-helper and cytotoxic T-cells to a sublethal dose of ionising radiation (46y). The numbers of these lymphocytes increased more rapidly after irradiation in tumour bearing mice than in those without tumours. In irradiated (46y) Balb/c nude and scid mice, subcutaneous T.annulata tumours failed to regress, despite the development of general haemorrhage and central necrosis. In scid mice, high doses (2xl07) of T.annulata infected cells injected intraperitoneally gave rise to ascites. However, low doses (2xl06 cells) did not. The presence of macroschizont infected cells in the peritoneal cavity was accompanied by a proliferation of macrophages. Several lines of evidence indicated that natural killer (NK) cells were not effective against T.annulata-infected cells in scid mice, but that macrophages were capable of controlling and eliminating the cells, particularly in the peritoneal cavity. In all the strains of mice examined, subcutaneous T.annulata tumours appeared to be damaged by natural immune mechanisms (macrophages) and neutrophils leading to haemorrhage and necrosis. However, the ability of the mice to completely reject the tumours depended on the presence of T and B-cells. Tumour Necrosis (TNF) was not detected in serum, or in tumour extracts. Attempts to induce tumour rejection using a preparation of rabbit TNF were not successful. Attempts to affect the growth of r.annu/ato-infected cells, injected i/p into scid mice, with human alpha and gamma interferons (HulFN-g, were also unsuccessful. The neutralisation of endogenous murine IFN-g with a monoclonal antibody allowed increased growth of T.annulata and 7".parva-infected cells in the intraperi-toneal site in scid mice. Of the mouse strains examined, the scid mouse was the most favourable host for Theileria-infected cells. Attempts to establish uninfected bovine cells in scid mice were not successful, and these results cast doubt on the use of scid mice as hosts for uninfected cells.

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Lieder, Anja. "The lymphocyte reconstituted severe combinmed immunodeficient mouse as a model of human allograft vasculopathy." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275269.

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Books on the topic "Immunodeficit"

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Carlos, Rodríguez-Gallego, and Arnaiz-Villena Antonio, eds. Human T-lymphocyte activation deficiencies. Austin, TX: R.G. Landes, 1994.

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M, McCrae, and Society for General Microbiology, eds. Molecular aspects of host-pathogen interaction: Fifty-fifth Symposium of the Society for General Microbiology : held at Heriot-Watt University, Edinburgh, March 1997. Cambridge: Cambridge University Press, 1997.

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Immunodeficient Rodents. Washington, D.C.: National Academies Press, 1989. http://dx.doi.org/10.17226/1051.

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H, Fiebig H., and Berger D. P, eds. Immunodeficient mice in oncology. Basel: Karger, 1992.

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Fiebig, H. H., and D. P. Berger, eds. Immunodeficient Mice in Oncology. S. Karger AG, 1992. http://dx.doi.org/10.1159/isbn.978-3-318-03425-7.

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1939-, Arnold W., Köpf-Maier P, Micheel B, and Workshop on Immunodeficient Laboratory Animals (1993 : Berlin, Germany), eds. Immunodeficient animals: Models for cancer research. Basel: Karger, 1996.

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Arnold, W., P. Köpf-Maier, and B. Michael, eds. Immunodeficient Animals: Models for Cancer Research. S. Karger AG, 1996. http://dx.doi.org/10.1159/isbn.978-3-318-03434-9.

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Bjorneby, John Maurice. Immunotherapy in immunodeficient hosts persistently infected with Cryptosporidium parvum. 1990.

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Sciences, Commission on Life, National Research Council, Institute for Laboratory Animal Research Staff, and Committee on Immunologically Compromised Rodents. Immunodeficient Rodents: A Guide to Their Immunobiology, Husbandry, and Use. National Academies Press, 1989.

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Institute of Laboratory Animal Resources (U.S.). Committee on Immunologically Compromised Rodents., ed. Immunodeficient rodents: A guide to their immunobiology, husbandry, and use. Washington, D.C: National Academy Press, 1989.

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Book chapters on the topic "Immunodeficit"

1

Barthold, Stephen W. "Coliform Typhlocolitis, Immunodeficient Mice." In Digestive System, 429–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60473-7_68.

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Barthold, Stephen W. "Coliform Typhlocolitis, Immunodeficient Mice." In Digestive System, 429–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-25996-2_68.

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Itin, Peter H., and Manuel Battegay. "Skin Problems in Immunodeficient Patients." In Transplantation Dermatology, 9–17. Basel: KARGER, 2012. http://dx.doi.org/10.1159/000335139.

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Shackley, Fiona. "Management of the Immunodeficient Child." In Scott-Brown’s Otorhinolaryngology Head and Neck Surgery, 47–53. Eighth edition. | Boca Raton : CRC Press, [2018] | Preceded by Scott-Brown’s otorhinolaryngology, head and neck surgery.: CRC Press, 2018. http://dx.doi.org/10.1201/9780203731017-7.

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Rüdlinger, R., P. Buchmann, R. Grob, F. Colla, R. Steiner, and M. Meandzija. "Genitoanal HPV Infections in Immunodeficient Individuals." In Genital Papillomavirus Infections, 249–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75723-5_18.

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Sprecher, Eli, Leonard D. Shultz, and Yechiel Becker. "Epidermal Dendritic Cells in Immunodeficient Mice." In Skin Langerhans (Dendritic) Cells in Virus Infections and AIDS, 59–76. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3942-1_4.

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"Immunodeficient." In Encyclopedia of Pain, 1576. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_201015.

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Hirenallur-Shanthappa, D. K., J. A. Ramírez, and B. M. Iritani. "Immunodeficient Mice." In Patient Derived Tumor Xenograft Models, 57–73. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-804010-2.00005-9.

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"Immunodeficient Animal." In Encyclopedia of Immunotoxicology, 451. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_200762.

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"Immunodeficient Nude Mice." In Encyclopedia of Cancer, 1816. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2986.

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Conference papers on the topic "Immunodeficit"

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Bexell, Daniel, Noémie Braekeveldt, Siv Beckman, Sofie Mohlin, David Gisselsson, and Sven Påhlman. "Abstract B57: Modeling human metastatic neuroblastoma in immunodeficient mice." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-b57.

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Maugham, Michelle L., Patrick B. Thomas, Gabrielle J. Crisp, Lisa K. Philp, Esha T. Shah, Adrian C. Herington, Chen Chen, et al. "Abstract 4819: Insights from engraftable immunodeficient mouse models of hyperinsulinaemia." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-4819.

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Noto, Fallon K., Angela Arey, Christopher McClain, Wei Zhang, and Tseten Yeshi. "Abstract 807: A novel immunodeficient rat for modeling human cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-807.

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Hernandez, Arlene, Emily Aventura, Farrukh Gill, Judd E. Shellito, and Carol M. Mason. "RARE CASE OF A MEDIASTINAL PLEOMORPHIC SARCOMA IN AN IMMUNODEFICIENT PATIENT." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5887.

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Serin, Guillaume, Loïc Morgand, Marie Leblanc, and Francis Bichat. "Abstract 2790: Establishment and characterization of human skingraft model in immunodeficient mice." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2790.

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Balivada, Sivasai, Matthew T. Basel, Marla Pyle, Amanda P. Beck, Ada G. Cino-Ozuna, Maureen A. Kerrigan, Jennifer L. Hill, Carol R. Wyatt, Robert R. R. Rowland, and Deryl L. Troyer. "Abstract LB-116: Immunodeficient pigs as a large animal model for human tumors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-lb-116.

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Lollini, Pier-Luigi, Giordano Nicoletti, Marianna Ianzano, Stefania Croci, Valentina Grosso, Arianna Palladini, Manuela Iezzi, Lorena Landuzzi, Carla De Giovanni, and Patrizia Nanni. "Abstract 1403: Multiorogan metastasis of human HER-2+ breast cancer in immunodeficient mice." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1403.

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Cho, Chi-Heum, So-Jin Shin, Sang-Hoon Kwon, and Soon-Do Cha. "Abstract 1357: Creation and establishment of xenograft model for uterine leiomyoma in immunodeficient mice." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1357.

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Wildt, Sheryl J., Jamie McClellan, and Mandy Horn. "Abstract 2812: Development and characterization of the ultra immunodeficient B6;129-Rag2tm1FwaIL2rgtm1Rsky/DwlHsd (R2G2) mouse model." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2812.

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Gonzalez, Sergio Enderica, S. John Weroha, Marc Becker, Sean Harrington, Xiaonan Hou, and Paul Haluska. "Abstract 3274: Development of a clinically relevantin vivomodel of ovarian cancer tumorgrafts in immunodeficient mice." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3274.

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Reports on the topic "Immunodeficit"

1

Libertin, C. R., S. Gavinski, and G. E. Woloschak. Evidence of T-cell abnormalities in immunodeficient wasted mice. Office of Scientific and Technical Information (OSTI), December 1991. http://dx.doi.org/10.2172/10173435.

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2

Libertin, C. R., L. Ling-Indeck, P. Weaver, Chin-Mei Chang-Liu, V. Strezoska, B. Heckert, and G. E. Woloschak. Dysregulation of temperature and liver cytokine gene expression in immunodeficient wasted mice. Office of Scientific and Technical Information (OSTI), April 1995. http://dx.doi.org/10.2172/208319.

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3

Libertin, C. R., P. Weaver, G. E. Woloschak, and S. Mobarhan. Subnormal albumin gene expression is associated with weight loss in immunodeficient/DNA-repair-deficient wasted mice. Office of Scientific and Technical Information (OSTI), September 1993. http://dx.doi.org/10.2172/10184609.

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