Academic literature on the topic 'Immunohematologie'

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Journal articles on the topic "Immunohematologie"

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Rosenwasser, L. J. "Immunohematologic diseases." JAMA: The Journal of the American Medical Association 268, no. 20 (November 25, 1992): 2940–45. http://dx.doi.org/10.1001/jama.268.20.2940.

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Patten, Ethel. "Immunohematologic Diseases." JAMA: The Journal of the American Medical Association 258, no. 20 (November 27, 1987): 2945. http://dx.doi.org/10.1001/jama.1987.03400200151018.

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Winkelstein, Alan. "Immunohematologic Disorders." JAMA: The Journal of the American Medical Association 278, no. 22 (December 10, 1997): 1982. http://dx.doi.org/10.1001/jama.1997.03550220188024.

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Winkelstein, A. "Immunohematologic disorders." JAMA: The Journal of the American Medical Association 278, no. 22 (December 10, 1997): 1982–92. http://dx.doi.org/10.1001/jama.278.22.1982.

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Patten, E. "Immunohematologic diseases." JAMA: The Journal of the American Medical Association 258, no. 20 (November 27, 1987): 2945–51. http://dx.doi.org/10.1001/jama.258.20.2945.

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Rosenwasser, Lanny J. "Immunohematologic Diseases." JAMA: The Journal of the American Medical Association 268, no. 20 (November 25, 1992): 2940. http://dx.doi.org/10.1001/jama.1992.03490200192024.

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Lalayanni, Chrisavgi, Niki Stavroyianni, Riad Saloum, Aliki Tsompanakou, and Achilles Anagnostopoulos. "Immunohematology." Hematology 9, no. 4 (August 2004): 287–89. http://dx.doi.org/10.1080/10245330410001714248.

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Trivedi, Deepa H., and James B. Bussel. "21. Immunohematologic disorders." Journal of Allergy and Clinical Immunology 111, no. 2 (February 2003): S669—S676. http://dx.doi.org/10.1067/mai.2003.157.

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Bajpai, Meenu, Ravneet Kaur, and Ekta Gupta. "Automation in Immunohematology." Asian Journal of Transfusion Science 6, no. 2 (2012): 140. http://dx.doi.org/10.4103/0973-6247.98914.

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Wong, Edward C. C. "Blood Banking/Immunohematology." Pediatric Clinics of North America 60, no. 6 (December 2013): 1541–68. http://dx.doi.org/10.1016/j.pcl.2013.08.005.

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Dissertations / Theses on the topic "Immunohematologie"

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Miller, Thomas E. "A Computerized Learning Environment For Exploring Learning Strategies with Immunohematology Students /." The Ohio State University, 1995. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487868114114174.

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Nilsson, Veronica. "Jämförelse av resultat mellan manuell gelkortsanalys och automatisk immunohematologisk gelkortsanalys vid utförande av blodgruppskontroll och antikroppsscreening (BAS-test)." Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-29185.

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Incompatibility between donors and recipients can cause fatal hemolytic transfusion reactions (HTR), kidney damage and death and immunization of the recipient. Antigens on the surface of erythrocytes in the ABO blood group system and regular natural antibodies against these in the patient’s plasma must be considered. An individual always has a corresponding antibody of IgM in their plasma. Irregular antibodies acquired by transfusion or pregnancy and active in body temperature also cause the HTR incompatibility. Before blood transfusion a test is performed to investigate the patient’s ABO antigen and the antigen Rh D, and an antibody screen, a compability test called BAS-test. The test is manual performed using a gel card technique. Immunohematology 1000 (IH-1000) is a fully automatic computerized system that automatically performs the test and passes the results directly to the patient file. To investigate whether these methods provide comparable results blood sample from the vein from a total of 118 blood donors and patients (50 men and 68 women) in the ages of 16-99 years were collected. Both manual gel card technique and analysis with the IH-1000 were performed. The results showed a good agreement (100%) between the two techniques for both affiliation of ABO of erythrocytes and findings of irregular antibodies in plasma. The patients who received a positive antibody screen with the manual technique also received a positive antibody screen with erythrocytes from same test donor in the analysis of IH-1000. Between the two methods no clinically significant difference was observed in the antibody response in the strength of the reactions. BAS-test can be performed with IH-1000 instead of manually performed technique.
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Faustino, Fabiana Garcia. "Teste de antiglobulina direta, pesquisa de anticorpo irregular e prova de compatibilidade em macacos-pregos (Sapajus sp.) e em macacos bugios (Alouatta sp.)." Botucatu, 2019. http://hdl.handle.net/11449/191428.

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Orientador: Lucilene Silva Ruiz e Resende
Resumo: Realizaram-se estudos imuno-hematológicos em 19 macacos sul- americanos, sendo 9 Sapajus sp. (macacos-prego) e 10 Alouatta sp. (macacos bugios). O Teste de Antiglobulina Direta (TAD) não revelou moléculas IgG de imunoglobulina e C3d do complemento, semelhantes às humanas, nas membranas dos eritrócitos dos animais. A Pesquisa de Anticorpos Irregulares (PAI) mostrou-se positiva nos soros de 18 animais adultos sendo negativa no soro do único filhote do estudo, um Sapajus sp. de 2 meses de idade. A Prova de Compatibilidade (PC) foi negativa dentro de cada gênero, revelando-se positiva entre os 2 gêneros de macacos e, destes, com humanos. O filhote de macaco-prego, entretanto, mostrou PC compatível, tanto com o gênero Alouatta sp., quanto com humanos.
Abstract: An immunohematological study was carried out in 19 South-American monkeys corresponding to 9 Sapajus sp. (Capuchin monkeys) and 10 Alouatta sp. (Howler monkeys). The Direct Antiglobulin Test (DAT) didn’t show humanlike molecules of IgG-type immunoglobulin and C3d complement fraction on the animal’s erythrocyte membranes. Serum Irregular Antibodies Screening (IAS) was found in 18 adult animals but not in the only studied cub, which was a 2 month-old Capuchin monkey. The Compatibility Test (CT) was negative among members of a same genre of animals being positive among Sapajus sp. and Alouatta sp., and among the monkey’s genres and humans. The Capuchin monkey cub, however, showed a negative CT with both, Howler monkeys and humans.
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Jacobs, Peter. "Immunohaematopoietic stem and progenitor cell transplantation - a thirty year prospective and systematic research investigation." Thesis, Stellenbosch : University of Stellenbosch, 2010. http://hdl.handle.net/10019.1/5232.

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Credidio, Débora Castilho. "Variantes do antígeno RhD = estudo sorológico e molecular." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310414.

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Orientador: Lilian Maria de Castilho
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
Made available in DSpace on 2018-08-16T17:52:53Z (GMT). No. of bitstreams: 1 Credidio_DeboraCastilho_M.pdf: 2805791 bytes, checksum: d7fa60ce846ffb7baee62f0493fdd327 (MD5) Previous issue date: 2010
Resumo: Discrepâncias na tipagem Rh ocorrem devido à presença de variantes do antígeno RhD, em especial D fraco e D parcial. Diferentes reagentes anti-D monoclonais tem sido desenvolvidos para identificar estas variantes, mas a caracterização molecular pode garantir um resultado mais específico com melhor definição do tipo de variante presente. O fenótipo D fraco ocorre por alterações de nucleotídeos que levam a substituição de aminoácidos na região transmembranar e intracelular da proteína Rh enquanto que o fenótipo D parcial ocorre por alterações de nucleotídeos ou rearranjos gênicos que levam a substituição de aminoácidos na região extracelular da proteína Rh, resultando em fenótipos sorológicos distintos e aloimunização anti-D. Nossos objetivos foram avaliar reagentes anti-D e métodos sorológicos e moleculares na detecção e identificação destas variantes. Foram estudadas 335 amostras de sangue e DNA de doadores e pacientes fenotipados previamente como RhD fraco, ou que apresentaram resultados discrepantes na tipagem RhD. Das 335 amostras estudadas, 215 (64,1%) foram identificadas como D fraco, 89 (26,5%) como D parcial, 3 (1%) como DEL, 11 como RhD-positivo e 17 como RhD-negativo. Entre as amostras D fraco, 76 eram DF1 (35,3%); 75 DF2 (34,9%); 13 DF3 (6,1%); 49 DF4 (22,8%) e 2 DF5 (0,9%). Entre as amostras D parcial, 9 (10,1%) eram DAR, 25 (28,1%) DFR, 6 (6,7%) DBT, 1 (1,1%) DHMi, 1(1,1%) RoHAR, 26 (29,2%) DVI, 14 (15,8%) DVa, 5 DIVb (6,6%) e 2 (2,3%) DVII . Nas amostras DEL, duas foram identificadas molecularmente como RHD(IVS5-38DEL4) e uma como RHD(K409K). Nossos resultados demonstram que a utilização de diferentes reagentes anti-D e métodos na rotina sorológica pode indicar a presença de uma variante do antígeno RhD que deve ser investigada por análise molecular. Esta estratégia pode auxiliar na diferenciação entre D fraco e D parcial e caracterizar as variantes que levam a aloimunização anti-D. Esta distinção é importante na seleção de sangue para pacientes e na prevenção da doença hemolítica do feto e recém-nascido
Abstract: Rh discrepancies are becoming a problem during routine testing due to partial D or weak D phenotypes. Panels of monoclonal antibodies (MoAb) are being developed in order to identify D variants such as partial D and weak D when there are anomalous RhD typing results but molecular characterization offers a more specific grouping of weak and partial D. The weak D phenotype is caused by many different RHD alleles encoding aberrant RhD proteins, resulting in distinct serologic phenotypes and anti-D immunizations. We evaluated currently used serologic methods and reagents to detect and identify D variants and correlated the results with molecular analyses. A total of 335 blood samples from Brazilian blood donors and patients with discrepant results of D typing in routine were analyzed. In total, 215 (64.1%) weak D, 89 (26.5%) partial D, 3 (1%) DEL, 11 RhD-positive and 17 RhD-negative were identified. Among weak D samples, 76 weak D Type 1 (35.3%); 75 weak D Type 2 (34.9%); 13 weak D Type 3 (6.1%); 49 weak D Type 4 (22.8%) and 2 weak D Type 5 (0.9%) alleles were found. Among the partial D samples, 9 (10.1%) DAR, 25 (28.1%) DFR, 6 (6.7%) DBT, 1 (1.1%) DHMi, 1(1.1%) RoHAR, 26 (29.2%) DVI, 14 (15.8%) DVa, 5 DIVb (6.6%) and 2 (2.3%) DVII were observed. Two samples identified as DEL by adsorption/elution were characterized by molecular analyses as RHD(IVS5-38DEL4) and one sample as RHD(K409K). Our results showed that the use of different methods and anti-D reagents in the serologic routine reveal some D variants that can be further investigated. Molecular methods can help to differentiate between partial D and weak D and characterize the weak D types providing additional information of value in the RhD typing. This distinction is important for selection of blood products and to prevent anti-D hemolytic disease of the fetus and newborn
Mestrado
Ciencias Basicas
Mestre em Clinica Medica
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Esteves, Vanessa Sinnott. "Frequência de tipos sangüíneos em uma população de cães de raça de Porto Alegre e região metropolitana." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/13403.

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Nas últimas décadas, a transfusão sangüínea e o estudo da imunohematologia em cães tornou-se extremamente importante no tratamento de diversas doenças. O avanço da técnica de hemoterapia e a maior segurança ao empregá-la estão diretamente relacionadas ao maior conhecimento dos tipos sangüíneos caninos e testes de compatibilidade. No entanto, poucos estudos foram realizados até o momento no sentido de definir as freqüências de tais tipos sangüíneos em diferentes populações, incluindo cães de diferentes raças. Estudos sobre tipagem sangüínea canina já foram realizados em diversos países do mundo, sendo que este é o primeiro estudo realizado no Rio Grande do Sul. Cães apresentam 5 tipos sangüíneos considerados principais: DEA 1 (1.1, 1.2), DEA 3, DEA 4, DEA 5 e DEA 7. O objetivo do trabalho foi determinar a freqüência dos tipos sangüíneos em uma população definida de cães de raça (Golden Retriever, Pastor Alemão, Rottweiler, Dogue Alemão e Dogo Argentino) de Porto Alegre e Região Metropolitana. Foram selecionados 100 cães, sendo 20 de cada uma das raças anteriormente citadas, entre 1 e 8 anos, sem distinção de sexo e clinicamente saudáveis. Amostras de sangue foram coletadas das veias cefálica ou safena lateral e a tipagem realizada através do teste de aglutinação em tubos, padronizado pelo Laboratório de Imunohematologia e Sorologia da Universidade de Michigan (Michigan - Estados Unidos) empregando-se os reagentes específicos (soro policlonal produzido em cães por meio de iso-imunização). Os resultados encontrados estão de acordo com os relatados previamente na literatura. As freqüências gerais observadas para as amostras populacionais utilizadas foram de 62% para o tipo DEA 1.1, 21% para o tipo DEA 1.2, 7% para o tipo DEA 3, 100% para o tipo DEA 4, 9% para o tipo DEA 5 e 16% para o tipo DEA 7. Concluindo, há particularidades entre os tipos sangüíneos de cada raça, que devem ser consideradas em procedimentos de transfusão. Além disso, testes de compatibilidade devem ser empregados em conjunto com a tipagem sangüínea para minimizar riscos de reações transfusionais.
Over the past few decades, blood transfusions and the study of canine immunohematology have become extremely important for the treatment of a various diseases. Technical advances and greater safety in the use of transfusions are directly related to improved knowledge on canine blood types and compatibility tests. However, few studies have been conducted to date to define the frequencies of these different blood types in different populations, including dogs from different breeds. Studies on canine blood typing have already been conducted in several countries around the world, and this is the first study conducted in the state of Rio Grande do Sul and the second in Brazil. Dogs have 5 principal blood types: DEA 1 (1.1, 1.2), DEA 3, DEA 4, DEA 5 and DEA 7. The objective of the study was to determine the frequency of the blood types in a given population of 5 selected breeds (Golden Retriever, German Shepherd, Rottweiler, Great Dane and Dogo Argentino) from Porto Alegre and the surrounding Region. One hundred clinically healthy dogs were chosen, 20 from each of the aforementioned breeds, with ages between 1 and 8, with no distinction between genders. Blood samples were taken from cephalic or lateral saphenous veins and the blood was typed using the agglutination test in tubes, standardized by the Immunohematology and Serology Laboratory of the University of Michigan (Michigan – United States) using specific reagents (polyclonal serum produced in dogs by means of isoimmunization). The results of this study are in agreement with those previously found in the literature. The overall frequencies observed for the population sample used were 62% for type DEA 1.1, 21% for type DEA 1.2, 7% for type DEA 3, 100% for type DEA 4, 9% for type DEA 5 and 16% for type DEA 7. In conclusion, there are specific frequencies among the blood types of each breed, which must be taken into account during blood transfusions. In addition, compatibility tests should be used together with blood typing to minimize risks of transfusion reactions.
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Ferreira, Angela Melgaço. "Avaliação da estabilidade de anticorpos anti-eritrocitários irregulares em amostras biológicas para produção de controles internos em laboratórios de imuno-hematologia." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17155/tde-07062017-142345/.

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O uso de amostras \"Controle Interno\" (CI) para validação de técnicas e reagentes imunohematológicos é exigido por lei e indispensável para identificar erros nos processos laboratoriais. No Brasil, as legislações que regem a hemoterapia aprovam a produção dos reagentes CI pelos serviços de hemoterapia (SH), desde que o processo seja previamente validado e tenha critérios de aceitação bem definidos. O estudo analisou a estabilidade de anticorpos anti-eritrocitários irregulares, da classe IgG, presentes nos plasma fresco congelado (PFC) de doadores e nas amostras pré-transfusionais de pacientes com o objetivo de utilizar esta matéria prima para a produção de CI nos laboratórios de imuno-hematologia. Foram avaliados o título, o escore e a reatividade (testes de potência) de 12 amostras de PFC com anticorpos anti-D, -E e -K e de 25 amostras de pacientes com anticorpos anti-D, -E, -e, -c, -C e -K, ambas estocadas em freezer (-25°C) e em geladeira (2 a 8°C), durante seis e três meses respectivamente. Nas amostras de PFC os valores iniciais do título e reatividade foram mantidos durante o estudo, sendo que as quedas ocorridas não ultrapassaram uma titulação ou graduação em freezer e geladeira. Todavia, o tempo de armazenamento dos PFC antes do início do estudo impactou negativamente o escore das amostras estocadas em freezer (p=0,021) e geladeira (p=0,027). Nas amostras de pacientes o título sofreu um impacto negativo significante, sendo afetado tanto pelo tempo prévio de seu armazenamento antes do início do estudo quanto pelo tipo de estocagem durante a execução dos testes. Amostras com tempo de armazenamento prévio prolongado entre 2 e 8°C tiveram seus títulos significantemente impactados quando estocadas em freezer (p=0,014) e geladeira (p=0,039), indicando que sua permanência demorada em temperaturas mais altas pode acarretar a degradação dos anticorpos de forma mais acentuada. As alíquotas armazenadas em geladeira durante o estudo tiveram uma significante redução do título (p=0,016), quando comparadas com as amostras armazenadas em freezer, confirmando uma menor viabilidade dos anticorpos mantidos em temperaturas menos frias. Ao avaliarmos uma possível associação entre os valores iniciais do título, escore e reatividade e uma propensão à queda nos resultados dos testes de potência, verificamos que os anticorpos com esses valores inicialmente baixos não apresentaram menor estabilidade se comparados aos anticorpos com valores mais altos. Esta característica indica que anticorpos irregulares IgG com diferentes perfis iniciais de potência podem ser utilizados para a produção de CI desde que corretamente caracterizados, armazenados e validados. É pertinente ampliar o conhecimento acerca da estabilidade das diferentes especificidades de anticorpos que podem compor um painel de CI, além das avaliadas neste estudo.
The use of \"Internal Control\" (IC) samples for immunohematological techniques and reagents validation is required by law and necessary to identify laboratory processes errors. In Brazil the hemotherapy legislation approve the IC reagents production by hemotherapic services (HS), if previously validated and with well-defined acceptance criteria. The study examined the stability of irregular red blood cell antibodies (IgG class) present in fresh frozen plasma (FFP) of donor and pre-transfusion patients samples with the aim of using this raw material to produce IC in immunohematology laboratories. The title, score and reactivity (potency tests) of 12 FFP with anti-D, -E and -K and 25 patients samples with anti-D, -E, -e, - c, -C, and -K were evaluated, both stored in freezer (-25°C) and refrigerator (2 to 8°C), for six and three months respectively. In FFP samples the initial values of title and reactivity were maintained during the study and the falls didn\'t exceed a titration or graduation in freezer and refrigerator. However, the storage time of the FFP before the study beginning impacted negatively the score of samples stored in freezer (p=0.021) and refrigerator (p=0.027). In patient samples the title had a negative impact, being affected by the previous storage time before the study beginning and by the storage type during the execution of the tests (freezer and refrigerator). Samples with prolonged previous storage time between 2-8°C had their title significantly impacted when stored in freezer (p=0.014) and refrigerator (p=0.039), indicating that the delayed stay at highter temperatures may lead to degradation of antibodies more sharply. The samples stored in refrigerator during the study had a significant reduction in the title (p=0,016) when compared to the samples stored in freezer, confirming a lower viability of the antibodies kept in less cold temperatures. When was evaluated a possible association between initial title, score and reactivity values and a tendency to fall their potency tests results, it was found that antibodies with initially low values didn\'t show lower stability when compared to antibodies with initial higher values. This feature indicates that IgG irregular antibodies with different inicial potency profiles can be used for IC production provided they are properly characterized, stored and validated. It\'s pertinent to extend the knowledge about the stability of different antibody specificities that can compose a IC panel in addition to those evaluated in this study.
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Carlsson, Malin. "Verifiering av en förstärkningslösning vid manuella serologiska metoder." Thesis, Örebro universitet, Institutionen för hälsovetenskaper, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-92905.

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IntroduktionDe humana blodgrupperna består av A, B, AB samt O. Antigen kan detekteras av alloantikroppar, att en reaktion sinsemellan kan ske är ett krav för att en molekyl ska kunna kallas antigen. Antigen och antikroppar är vitala för analys inom transfusionsmedicin och möjliggör transfusioner av blod samtidigt som man förhindrar allvarliga transfusionsreaktioner. För att effektivisera manuella serologiska analyser används saltlösning med låg jonstyrka. Syftet med arbetet var att verifiera lösningen MLB 2 (Bio-Rad Medical Diagnostics GmbH, Dreieich, Tyskland). Denna lösning ska förstärka reaktioner i antikroppsidentifiering, blodgruppskontroll med antikropps-screening samt förenlighetsprövning, men även för fenotypning av erytrocyter gällande antigenen s, Fya, Fyb, Kpa, Kpb samt Lua. MetodTotalt fenotypades 61 prover varav 31 var positiva och 30 var negativa för sökt fenotyp. Av de 31 positiva var 17 fenotyper av känt heterozygot anlag. Vid varje analystillfälle utfördes kontroller som bestod av heterozygot positiva samt negativa celler från Örebropanelen för vardera fenotyp. För antikroppsidentifiering ämnade två antikroppar, anti-E samt anti-Lea, att analyseras från två prover där de tidigare identifierats. Beträffande blodgruppskontroll med antikroppsscreening samt förenlighetsprövning analyserades tre prover från patienter som varit i behov av transfusioner och som tidigare påvisat antikroppar. Resultat och slutsatsSamtliga provers resultat överensstämde fullständigt vid fenotypning. För antikroppsidentifiering bekräftas tidigare påvisad anti-E för ett av proven, men för det andra provet erhölls inte resultat fullständigt överensstämmande med patientens anti- Lea. För blodgruppskontroller med antikropps-screen samt förenlighetsprövning överensstämde samtliga resultat. Effekten av förstärkningslösningen MLB 2 bedöms som god. Tydligt blir dock att gelteknik, som idag används som golden standard, är överlägsen de äldre rörteknikerna.
BackgroundHuman blood groups are defined as A, B, AB and O. Antigens are detectable by alloantibodies and a reaction between them is necessary in order to denominate as such. Antigens and antibodies respectively are essential within immunohematology to enable transfusion therapy while evading severe transfusion reactions. To improve the efficiency of manual serological test, low ionic strength saline is used. The purpose of this study was to verify MLB 2 (Bio-Rad Medical Diagnostics GmbH, Dreieich, Germany) for use as low ionic saline solution (LISS) in indirect agglutination technique (IAT) tube-tests for antibody identification, blood group verification with antibody-screen and compatibility test, also for phenotyping antigens s, Fya, Fyb, Kpa, Kpb and Lua. MethodsA total of 61 samples, of which 31 were previously known positive and 31 negative were tested. Of the 31 positive, 17 were heterozygous. At every assay, positive heterozygous and negative cells for each of the phenotype was applied as control, from the Örebro panel. For antibody identification, two previously positive samples were used. For blood group verification with antibody-screen and compatibility test, three samples with previous need for transfusion therapy and positive antibody screen were used. Results and conclusionAll samples phenotyped present fully consistent results compared to the previous ones. For antibody identification the results for one of the samples are completely confirmed. The second sample showed inconsistencies to the previous result. For blood group verification with antibody-screen and compatibility test all samples had equivalent results. The effect from MLB 2 in IAT-LISS tube tests are adequate. A distinct observation can be made; gel column technology is superior to the tube tests.
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Daniella, Perri. "The Role of Intravenous Immunoglobulin Anti-A and Anti-B in Complement Activation and Red Blood Cell Phagocytosis." Thesis, 2009. http://hdl.handle.net/1807/18976.

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Intravenous immunoglobulin is a human blood derived product that is used to treat immunodeficiencies and autoimmune disorders. An adverse side effect of IVIg therapy is hemolysis. Patients who experience hemolysis are mainly blood group A or AB. Clinical laboratory studies have demonstrated that IVIg contains ABO blood group antibodies, which can bind complement proteins. This study hypothesizes that anti-A/B in IVIg will bind to A/B antigens and activate complement in a dose dependant manner, which may lead to enhanced RBC phagocytosis. This study observed that the quantity of ABO antigens does not affect the in vitro binding of IVIg to RBCs. IVIg induced C3b deposition at high doses; however, the amount of complement deposition was insufficient to enhance phagocytosis of IVIg-sensitized RBCs by monocytic THP-1 cells in vitro. These studies emphasize that hemolytic reactions involve many factors in conjunction with antibodies and complement proteins.
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Steffensen, Bjørn. "Expression of epithelial blood group substances in gingival and junctional epithelium a dissertation [sic] submitted in partial fulfillment ... periodontics ... /." 1986. http://books.google.com/books?id=55U9AAAAMAAJ.

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Books on the topic "Immunohematologie"

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Clinical immunohematology: Basic concepts and clinical applications. Boston: Blackwell Scientific Publications, 1990.

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Whitlock, Sheryl. Immunohematology. Albany: Delmar Publishers, 1997.

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Ness, Paul M., Steve R. Sloan, and JoAnn M. Moulds, eds. BeadChip Molecular Immunohematology. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-7512-6.

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Judd, W. John. Methods in immunohematology. Miami, FL: Montgomery Scientific Publications, 1988.

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5

Essentials of immunohematology. Philadelphia: Saunders, 1998.

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6

T, Johnson Susan, Storry Jill, Judd W. John, and American Association of Blood Banks., eds. Judd's methods in immunohematology. 3rd ed. Bethesda, MD: AABB Press, 2008.

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7

An introduction to immunohematology. 3rd ed. Philadelphia: W.B. Saunders Co., 1994.

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8

Principles of clinical immunohematology. Chicago: Year Book Medical Publishers, 1986.

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9

Friedman, Mark T., Kamille A. West, and Peyman Bizargity. Immunohematology and Transfusion Medicine. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22342-1.

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Ajmani, Pritam Singh. Immunohematology and Blood banking. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-8435-0.

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Book chapters on the topic "Immunohematologie"

1

Cruse, Julius M., and Robert E. Lewis. "Immunohematology." In Atlas of Immunology, 267–81. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-662-11196-3_15.

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2

Virella, Gabriel, and Armand Glassman. "Immunohematology." In Medical Immunology, 303–19. 7th edition. | Boca Raton : Taylor & Francis, 2020.: CRC Press, 2019. http://dx.doi.org/10.1201/9780429278990-22.

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Salama, A., and O. Meyer. "Immunohematology." In New Diagnostic Methods in Oncology and Hematology, 197–235. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-58803-7_6.

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Jackson, Karen V. "Immunohematology and Hemostasis." In Equine Clinical Pathology, 37–69. Chichester, UK: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118718704.ch3.

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Eisenbrey, A. Bradley. "Essentials of Immunohematology." In Laboratory Hematology Practice, 553–61. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781444398595.ch43.

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Fisher, M. J. "Immunohematologic Mechanisms in Stroke." In Brain Ischemia, 97–103. London: Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2073-5_13.

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Tenorio, Grace C., Snehalata C. Gupte, and Reinhold Munker. "Transfusion Medicine and Immunohematology." In Modern Hematology, 401–32. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-149-9_22.

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Moulds, John J. "An Overview of the Classic Serological Methods: Limitations and Benefits of Serology and DNA Testing." In BeadChip Molecular Immunohematology, 1–7. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7512-6_1.

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Reid, Marion E., and Christine Halter Hipsky. "Looking Beyond HEA: Matching SCD Patients for RH Variants." In BeadChip Molecular Immunohematology, 101–20. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7512-6_10.

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Vege, Sunitha, and Connie M. Westhoff. "Identification of Altered RHD and RHCE Alleles: A Comparison of Manual and Automated Molecular Methods." In BeadChip Molecular Immunohematology, 121–31. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7512-6_11.

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