Relevant bibliographies by topics / Impulsive aggression

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Impulsive aggression'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "Impulsive aggression"

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Coccaro, Emil F., Jennifer R. Fanning, K. Luan Phan, and Royce Lee. "Serotonin and impulsive aggression." CNS Spectrums 20, no. 3 (May 22, 2015): 295–302. http://dx.doi.org/10.1017/s1092852915000310.

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Aggression is a behavior with evolutionary origins, but is often both destructive and maladaptive in today’s society. Research over the past several decades has confirmed the involvement of neurotransmitter function in aggressive behavior. This research has centered around the “serotonin hypothesis.” As this literature continues to grow, guided by pre-clinical research and aided by the application of increasingly sophisticated neuroimaging methodology, a more complex picture has emerged. As current pharmacological and therapeutic interventions are effective but imperfect, it is hoped that new insights into the neurobiology of aggression will reveal novel avenues for treatment of this destructive and costly behavior.
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Coccaro, Emil F. "Central Serotonin and Impulsive Aggression." British Journal of Psychiatry 155, S8 (December 1989): 52–62. http://dx.doi.org/10.1192/s0007125000291769.

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The role of central serotonergic (5-HT) system dysfunction in the regulation of aggression in both animals and man has been investigated for more than the past two decades. Evidence for reduced central 5-HT in the mediation of aggression comes from both behavioural and correlative studies. Functional reduction and augmentation of 5-HT activity is respectively associated with increased and decreased aggression in various animal models of aggression. While similar studies in man have not been performed, strong and consistent associations between indices reflecting reduced pre-synaptic 5-HT activity and aggression have been reported. Evidence of post-synaptic receptor upregulation in the brains of suicide victims has also been reported leaving the functional status 5-HT activity in such patients an open question. However, reduced neuroendocrine (i.e. prolactin) responses to fenfluramine, a 5-HT uptake inhibitor/releaser, which activates both pre- and post-synaptic sides of the 5-HT synapse, strongly suggest that overall central 5-HT activity is reduced in mood and/or personality disorder patients with history of suicidal and/or impulsive aggressive behaviour. Preliminary data with the 5-HT receptor agonist m-chlorophenylpiperazine further suggest that reduced activity of post-synaptic 5-HT receptors may be an important correlate of impulsive aggressive behaviour. Pharmacological agents with potent 5-HT pre- and/or post-synaptic augmenting effects should be tested clinically to determine their efficacy in the treatment of impulsive aggressive behaviour in psychiatric patients.
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Hadi, Fatima, Kathleen Franco, Teresa Hermida, Karl Douton, and Rebecca Zarko. "CITALOPRAM FOR IMPULSIVE AGGRESSION." Journal of the American Academy of Child & Adolescent Psychiatry 42, no. 7 (July 2003): 749. http://dx.doi.org/10.1097/01.chi.0000046883.27264.e6.

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Felthous, Alan R. "Introduction to impulsive aggression." Behavioral Sciences & the Law 16, no. 3 (1998): 281–83. http://dx.doi.org/10.1002/(sici)1099-0798(199822)16:3<281::aid-bsl310>3.0.co;2-2.

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Telesheva, Klavdiya, Farit Safuanov, Anna Kirenskaya, Zinaida Storozheva, and Vadim Myamlin. "Pathocharacterologic features as predictors of impulsive aggression." Applied psychology and pedagogy 5, no. 2 (April 29, 2020): 51–66. http://dx.doi.org/10.12737/2500-0543-2020-51-66.

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In the original study, in order to identify predisposition factors and mechanisms of criminal impulsive aggression, psychometric testing was performed with further evaluation of the dependence of the probability of impulsive aggression on the results of psychometric testing by constructing a binary logistic regression model. On groups of mentally healthy individuals and individuals with organic mental disorder, the dependence of the presence or absence of the fact of impulsive aggression and the diagnosis (mentally healthy/organic mental disorder) on the variable test results was evaluated. The results prove the extra-nosological nature of the formation of propensity to impulsive aggression, the contribution of pathocharacterological predictors of aggressive behavior, but not clinical ones. The specificity of mechanisms of formation of propensity to impulsive aggression in mental health and organic mental disorder is determined. A method for determining probability has been developed.
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Ostapenko, A. V., and S. Kolov. "Interrelations between anger and aggression among male veterans with combat-related posttraumatic stress disorder." European Psychiatry 26, S2 (March 2011): 1077. http://dx.doi.org/10.1016/s0924-9338(11)72782-1.

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ObjectivesCombatants with PTSD show a higher level of aggression and dysfunctional anger influencing clinical picture and determining tolerance to treatment. Interrelations between emotional dysfunctions (anger), symptoms of combat-related PTSD and aggression are not clear.MethodWe studied relations between aggression, anger and PTSD symptoms in the group of 557 males-combatants (109 patients had marked symptoms of PTSD, 448 men showed subclinical symptoms), and in the control group of 234 healthy males. We used the Mississippi Scale for quantitative evaluation of PTSD and authorial methods for assessing anger and aggression.ResultsImpulsive aggression is the main form in veterans with PTSD, exceeding sevenfold indices in the control group (21,17 ± 1,92; 3,18 ± 0,38, P < 0,00001). Indices of this aggression are equal to level of instrumental aggression (3,03 ± 0,27; 3,88 ± 0,19, P < 0,001), in the control group various forms of aggression differed greatly, level of these types of aggressive behaviour was lower (0,45±0,06; 1,88±0,09, P < 0,0000001).Correlation analysis of indices of different aggressive behaviour and anger showed that dysfunctional anger has strong positive interrelations only with indices of impulsive (affective-hostile) aggression (P < 0,01), similar correlation with instrumental aggression was not discovered. In the control group significant correlations between anger and aggressive behaviour were not found.ConclusionsImpulsive aggression is mostly expressed in combatants with PTSD. Anger is the main determining factor for level, strength, intensity and frequency of impulsive aggression. Association between anger and PTSD is unique for combatants with impulsive aggression. Dysfunctional anger serves a general pathogenetic factor for combat-related PTSD and impulsive aggression.
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Lopez-Castroman, J., I. Jaussent, S. Beziat, S. Guillaume, E. Baca-Garcia, C. Genty, E. Olié, and P. Courtet. "Increased severity of suicidal behavior in impulsive aggressive patients exposed to familial adversities." Psychological Medicine 44, no. 14 (March 26, 2014): 3059–68. http://dx.doi.org/10.1017/s0033291714000646.

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BackgroundThe mechanisms by which childhood abuse and family history of suicidal behavior (FHS) lead to an increased risk of suicidal behavior are still unknown. Impulsive aggression may play an intermediate role. We investigated whether greater scores for aggression and impulsivity might be associated with the effects of FHS and/or childhood abuse on the severity of suicidal behavior.MethodWe examined the scores of three scales measuring impulsive aggression in a sample of 696 suicide attempters. We compared the highest and lowest scores with regard to reports of childhood abuse and FHS using adjusted multinomial regression models. Genetic polymorphisms of the serotonergic system known to be associated with impulsive aggression were also analyzed.ResultsPatients with high impulsive aggressive scores showed significant differences in sociodemographic, clinical and suicidal features compared with patients with low impulsive aggressive scores. Adjusted results showed that combinations of some types of childhood abuse and FHS, particularly emotional abuse and emotional neglect, are associated with high impulsivity and hostility scores. The SS genotype of the serotonin transporter gene (5-HTTLPR) was associated with high levels of impulsivity when the subjects reported emotional abuse [odds ratio (OR) 5.55, 95% confidence interval (CI) 1.75–17.5] or physical abuse (OR 5.03, 95% CI 1.50–16.9) in their childhood.ConclusionsOur results support the role of impulsive aggression as one of the links that may connect childhood abuse and FHS with severity of suicidal behavior.
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Cruz, Ana Rita, Rita Pasion, Andreia Castro Rodrigues, Carmen Zabala, Jorge Ricarte, and Fernando Barbosa. "Psychometric properties of the Impulsive/Premeditated Aggression Scale in Portuguese community and forensic samples." Trends in Psychiatry and Psychotherapy 41, no. 2 (June 2019): 144–48. http://dx.doi.org/10.1590/2237-6089-2018-0055.

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Abstract Introduction Aggression can be defined according to impulsive or premeditated features. Impulsivity is defined as an uncontrolled and unplanned form of aggression. On the contrary, premeditation requires planning and is goal-oriented. Objective The purpose of this study was to validate the basic psychometric properties of the Impulsive/Premeditated Aggression Scale (IPAS) into European Portuguese. The scale evaluates aggression according to impulsive and premeditated features, which are considered the predominant forms of aggressive behavior, and can be used in community, forensic and clinical settings. Methods Participants from a community sample (n = 957; 424 male) and incarcerated individuals (n = 115, all male) completed the IPAS. Results Internal consistency and reliability indicated that the scale has good psychometric properties in both samples. Data from a principal component analysis (PCA) demonstrated similarities to previous structures reported in the literature. Conclusions The scale demonstrated to be sensitive to the bimodal classification of aggression in community and forensic samples, indicating its utility in the characterization of aggressive patterns.
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Fulwiler, Carl, Joy Eckstine, and Sapna Kalsy. "Impulsive-Aggressive Traits, Serotonin Function, and Alcohol-Enhanced Aggression." Journal of Clinical Pharmacology 45, no. 1 (January 2005): 94–100. http://dx.doi.org/10.1177/0091270004270457.

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Kavoussi, Richard, Phyllis Armstead, and Emil Coccaro. "THE NEUROBIOLOGY OF IMPULSIVE AGGRESSION." Psychiatric Clinics of North America 20, no. 2 (June 1997): 395–403. http://dx.doi.org/10.1016/s0193-953x(05)70319-1.

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Dissertations / Theses on the topic "Impulsive aggression"

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Henton, Lynn Bishop. "Subtypes of impulsive aggression in children and adolescents with impulsive aggression /." Digital version accessible at:, 1999. http://wwwlib.umi.com/cr/utexas/main.

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Rice, Judy A. "Behind the Rage: the Neurobiology of Impulsive Aggression." Digital Commons @ East Tennessee State University, 2003. https://dc.etsu.edu/etsu-works/7615.

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McGirr, Alexander. "The role of impulsive and impulsive aggressive behaviours in the risk for suicide and the familial transmission of suicidal behaviours /." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116037.

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One of the most difficult and serious challenges facing the mental health professional is the prevention of suicide. Efficient prevention, however, depends on early detection of patients at risk for suicidal behaviour, which in turn depends on a better understanding of the predisposing factors. Over the last years, based on a large volume of data, it has become increasingly clear that subjects who commit suicide present high levels of impulsive and impulsive-aggressive behaviours.
In the first approach, we investigate whether levels of the impulsive aggressive diathesis were more important in suicide occurring at different life stages. In the second, we investigate whether levels of the diathesis predicted when during the course of major depressive disorder suicide occurs.
Family studies have consistently indicated that suicidal behaviour tends to cluster in families. However, relatives also present increased risk for psychiatric morbidity, and therefore, the critical question is whether or not the liability to suicidal behavior is given by the same predisposition to the coexistent psychiatric disorders.
Therefore, in the third study, we examined the independence of familial liability using a three group design. The relatives of deceased suicide probands who died in the context of a major depressive episode are compared to the relatives of living depressed subjects without a history of suicide and to the relatives of healthy controls screened for the absence of major depression and suicide.
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Checknita, David. "Monoamine oxidase A gene promoter methylation and impulsive aggression in an offender population with antisocial personality disorder." Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=121478.

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Antisocial personality disorder (ASPD) is a condition characterized by elevated impulsive aggression and increased risk for criminal behaviour and incarceration. Deficient activity of the monoamine oxidase A (MAOA) gene is suggested to contribute to serotonergic system dysregulation strongly associated with impulsive aggression and ASPD. The potential contribution of epigenetic processes, which modulate gene expression without altering the underlying genomic code, towards the dysregulation of MAOA in ASPD is not yet understood.The current study aimed to elucidate the role of epigenetic processes in altered MAOA expression and serotonin regulation in a population of offenders with ASPD compared to healthy controls. Results suggest MAOA promoter hypermethylation contributes to downregulated gene expression and elevated whole-blood serotonin in offenders with ASPD. These results are consistent with prior literature suggesting MAOA and serotonergic dysregulation in antisocial populations. Further, our results offer the first evidence suggesting epigenetic mechanisms may contribute to MAOA dysregulation in antisocial offenders.
Le trouble de personnalité antisociale est une condition qui se caractérise par un niveau élevé d'agression impulsive ainsi qu'un risque accru de comportements criminels et d'incarcération. Il a été suggéré qu'une activation déficitaire du gène monoamine oxydase A (MAOA) contribuerait à une dysrégulation du système sérotogénique, qui est fortement associé à l'agression impulsive et au trouble de personnalité antisociale. La potentielle contribution des processus épigénétiques modulant l'expression génétique sans altérer le code génomique sous jacent dans la dysrégulation du MAOA chez les individus atteint du trouble de personnalité antisociale n'est pas encore comprise. L'étude suivante avait comme objectif d'élucider le rôle des processus épigénétiques dans l'altération de l'expression du MAOA et de la régulation de la sérotonine dans une population incarcérée avec un trouble de personnalité antisociale, lorsque comparés à des contrôles sains. Les résultats suggérent que le promoteur d'hyperméthylation MAOA contribue à une réduction de l'expression génétique et à un niveau élevé de sérotonine sanguin chez les incarcérés avec un trouble de personnalité antisociale. Ces résultats sont cohérents avec la littérature suggérant que la dysrégulation du MAOA et de la sérotonine est présente dans les populations antisociales. De plus, nos résultats représentent la première évidence suggérant que les méchanismes épigénétiques pourraient contribuer à la dysrégulation du MAOA chez les incarcérés avec un trouble de personnalité antisociale.Le trouble de personnalité antisociale est une condition qui se caractérise par un niveau élevé d'agression impulsive ainsi qu'un risque accru de comportements criminels et d'incarcération. Il a été suggéré qu'une activation déficitaire du gène monoamine oxydase A (MAOA) contribuerait à une dysrégulation du système sérotogénique, qui est fortement associé à l'agression impulsive et au trouble de personnalité antisociale. La potentielle contribution des processus épigénétiques modulant l'expression génétique sans altérer le code génomique sous jacent dans la dysrégulation du MAOA chez les individus atteint du trouble de personnalité antisociale n'est pas encore comprise. L'étude suivante avait comme objectif d'élucider le rôle des processus épigénétiques dans l'altération de l'expression du MAOA et de la régulation de la sérotonine dans une population incarcérée avec un trouble de personnalité antisociale, lorsque comparés à des contrôles sains. Les résultats suggérent que le promoteur d'hyperméthylation MAOA contribue à une réduction de l'expression génétique et à un niveau élevé de sérotonine sanguin chez les incarcérés avec un trouble de personnalité antisociale. Ces résultats sont cohérents avec la littérature suggérant que la dysrégulation du MAOA et de la sérotonine est présente dans les populations antisociales. De plus, nos résultats représentent la première évidence suggérant que les méchanismes épigénétiques pourraient contribuer à la dysrégulation du MAOA chez les incarcérés avec un trouble de personnalité antisociale.
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Rylands, Angela J. "An investigation of cognitive function and the brain serotonin (5HT) system in impulsive aggression (IA), using positron emission tomography." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.511920.

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Colby, M. Amanda Earl Stanford Matthew S. "Verbal and working memory deficits in an impulsive aggressive college sample." Waco, Tex. : Baylor University, 2008. http://hdl.handle.net/2104/5168.

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Zouk, Hana. "Investigation of impulsive-aggressive behaviors in suicide : a clinical and genetic approach." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111928.

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Impulsivity and aggression have been shown to be important behavioral correlates of suicide. Neurobiological and genetic studies, mostly focusing on the serotonergic system, have demonstrated that these behaviors not only have clinical implications in suicide but also play an important biological role in increasing suicide risk. It remains unclear, however, how impulsivity and aggression might mediate suicide risk. The first study presented here was carried out to investigate clinical, behavioral and psychosocial correlates of impulsivity in suicide completers. Impulsive suicides were characterized by a greater psychiatric comorbidity as well as increased levels of aggression, and were more likely to be affected by negative life events. In a separate study, the effect of genetic variants of the 5-HT1B gene on impulsive aggressive behaviors (IABs) in suicide, as well as their contribution to overall suicide risk, was investigated. One 5-HT1B promoter variant significantly influenced levels of aggressive behaviors in suicide completers, suggesting that aggression plays a role as an intermediate phenotype that increases propensity to suicide. Both studies highlight the importance of the role of IABs in mediating suicide at both clinical and biological levels.
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LeMarquand, David Gordon 1966. "Serotonin and disorders of human disinhibition : alcohol abuse and dependence, aggression and impulsivity." Thesis, McGill University, 1997. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=34998.

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A wealth of data supports the hypothesis that the neurotransmitter serotonin regulates the intake of ethanol, and is involved in the development of alcoholism in humans. Reduced functioning of the serotonergic system hypothetically increases alcohol intake in both animals and humans. In this thesis, it was proposed that the effect of lowered serotonergic function on alcohol intake is mediated by an increase in disinhibition. The hypothesis that lowered serotonin increases disinhibition was tested in separate groups of individuals at high risk for the development of psychopathology: nonalcoholic young men with a strong family history of paternal alcoholism, and adolescent men with previous histories of physically aggressive behavior. Lowered serotonergic synthesis (and thus presumably function) was experimentally induced through a transient dietary reduction in the availability of the amino add precursor of serotonin, tryptophan. Disinhibition was quantified using a go/no-go task previously shown to characterize psychopaths and children with attention deficit hyperactivity disorder as disinhibited. In the first study, acute tryptophan depletion had no effect on aggressive responding on a modified competitive reaction time aggression task, but increased disinhibition in young men at risk for alcoholism. This effect was independent of the tryptophan depletion-induced mood alterations. The effect tryptophan depletion on disinhibition was not replicated in the second study with previously aggressive adolescent men. A number of explanations for this were posited, including the presence of a ceiling effect. An association between disinhibition and executive functioning (cognitive abilities associated with proper functioning of the prefrontal cortex, such as working memory, planning abilities) was demonstrated in the second study. In a third preliminary study, no association between disinhibition on the go/no-go task and allelic polymorphisms of the dopamine D4 receptor
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Fisher, William I. "The relationship between memory and event-related potentials in pathologically impulsive aggressive juveniles : a retrospective chart study /." View online, 2009. http://ecommons.txstate.edu/psyctad/2/.

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Tanskanen, Sanna-Leena. "Associations between TBI, facial emotion recognition, impulse control and aggression in delinquent and vulnerable young people." Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/18524.

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Objectives: There is evidence that childhood traumatic brain injury (TBI) is associated with increased risk of offending and violent crime. This study aimed to explore associations between TBI in a group of delinquent and vulnerable young people (VYP) at risk of offending, and facial emotion recognition (FER) abilities, inhibition control (Stop-IT) and self-reported reactive-proactive aggression (RPQ). Methods: There were two studies. The first study used a cross sectional between group design to compare 45 VYP (with and without TBI) and a control group of 59 students on FER task measuring emotion recognition accuracy of six basic emotions. The second study examined differences between TBI and non-TBI groups in the VYP sample (N=21) on a Stop-IT task, FER accuracy and self-reported reactive-proactive aggression. Results: A history of TBI was reported by 60% of the VYP group (48.9% with loss of consciousness [LoC]), whereas 30% of the control group reported a history of TBI (25.4% with LoC). The VYP group (with and without TBI) demonstrated a similar pattern of reduced overall FER accuracy that was significantly different to the control group. Compared to the control group, The VYP groups (with and without TBI) were less accurate on recognising anger, disgust, sadness and surprise, but not happy and fear. There were no significant differences between the TBI- and non-TBI groups. The second study did not find any significant differences between the TBI and non-TBI groups on overall FER accuracy, Stop-IT performance, and RPQ scores. There were also no significant associations between these measures. Conclusions: Future research requires larger samples that enable investigating the association between different severity of TBI, FER and inhibition control ability. Better and more youth-friendly measures are also needed.
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Books on the topic "Impulsive aggression"

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S, Malta Loretta, and Blanchard Edward B, eds. Road rage: Assessment and treatment of the angry, aggressive driver. Washington, DC: American Psychological Association, 2006.

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Fanning, Jennifer R., and Emil F. Coccaro. Neurobiology of Impulsive Aggression. Edited by Phillip M. Kleespies. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199352722.013.24.

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Aggression is a behavior with evolutionary origins, but in today’s society it’s often both destructive and maladaptive. The fact that aggression has a strong basis in biological factors has long been apparent from case histories of traumatic brain damage. Research over the past several decades has confirmed the involvement of neurotransmitter function and abnormalities in brain structure and function in aggressive behavior. This research has centered around the “serotonin hypothesis” and on dysfunction in prefrontal brain regions. As this literature continues to grow, guided by preclinical research and aided by the application of increasingly sophisticated neuroimaging methodology, a more complex picture has emerged, implicating diverse neurotransmitter and neuropeptide systems (e.g., glutamate, vasopressin, and oxytocin) and neural circuits. As the current pharmacological and therapeutic interventions are effective but imperfect, it is hoped that new insights into the neurobiology of aggression will reveal novel avenues for treatment of this destructive and costly behavior.
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Michael, Maes, and Coccaro Emil F, eds. Neurobiology and clinical views on aggression and impulsivity. Chichester: Wiley, 1998.

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Lee, Royce, Jennifer R. Fanning, and Emil F. Coccaro. The Clinical Neuroscience of Impulsive Aggression. Edited by Christian Schmahl, K. Luan Phan, Robert O. Friedel, and Larry J. Siever. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199362318.003.0008.

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Aggression can be categorized into three subtypes: premeditated aggression, frustration-related aggression, and impulsive aggression (IA), which is the focus of this chapter. It first delineates the social information processing model of IA and its neurobiological underpinnings, with a special focus on ventral prefrontal-amygdala, frontostriatal, and frontoparietal circuits. In these circuits, structural as well as functional alterations have been associated with IA. A large body of basic and clinical research has examined the role of neurotransmitters (glutamate, GABA) and neuromodulators (monoamines and neuropeptides) in mediating IA. The important role of the monoamines dopamine, serotonin, norepinephrine, and acetylcholine in the mediation of different aspects of IA and the pharmacological potential resulting from these alterations are depicted in the second half of the chapter. The chapter concludes with an overview of the most important etiological factors.
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Siever, Larry J., and Joshua E. Kuluva. Aggression, Impulsivity, and Personality Disorders. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0030.

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Aggressivity and impulsivity are traits that are core features of the Cluster B personality disorders. Within these disorders, impulsive aggression leads to a significant amount of morbidity and mortality. This type of behavior is intrinsically linked to violence, suicide, and substance abuse. In this chapter, we will discuss the phenomenology of these traits, the neurobiology of impulsive aggression, and some potential treatment options. We will conclude with some thoughts on the future direction of research in this filed.
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Trestman, Robert L. Aggression. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199360574.003.0048.

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Managing aggression is a challenge for psychiatry in all settings. Recognizing opportunities for appropriate assessment and intervention in correctional settings is an important component of correctional psychiatry. Studies reflect significant risks of violence for both correctional officers and inmates. Although prison homicides occur at rates below estimated community homicide rates, the rate of non-lethal violence is substantial. The data for assault are less clear, as definitions of what constitutes assault vary. Inmate-on-inmate assault has been estimated to range from 2 per 1000 inmates to as high as 200 per 1000 inmates. However assault is defined, correctional officers who have been the target of offender violence have elevated risk of emotional exhaustion and burnout. Effectively addressing aggression requires a thoughtful and comprehensive approach that may incorporate elements of environmental management, evaluation of potential motivating factors, differential diagnosis, and a coordinated intervention. This always involves includes effective communication among stakeholders including the patient. Recommended milieu changes and psychotherapeutic and / or pharmacologic interventions need to be explicitly defined; available data are described in this chapter. Consistent oversight and follow up to measure the effects of each component of the intervention(s) is critical, as aggressive behavior may be both habitual and episodic. This chapter reviews the factors that contribute to the broad range of assaultive behavior observed in correctional settings, and some of the pragmatic issues and opportunities for assessment, diagnosis, and treatment of aggressive behaviors, both impulsive and predatory.
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Trestman, Robert L. Aggression. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199360574.003.0048_update_001.

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Managing aggression is a challenge for psychiatry in all settings. Recognizing opportunities for appropriate assessment and intervention in correctional settings is an important component of correctional psychiatry. Studies reflect significant risks of violence for both correctional officers and inmates. Although prison homicides occur at rates below estimated community homicide rates, the rate of non-lethal violence is substantial. The data for assault are less clear, as definitions of what constitutes assault vary. Inmate-on-inmate assault has been estimated to range from 2 per 1000 inmates to as high as 200 per 1000 inmates. However assault is defined, correctional officers who have been the target of offender violence have elevated risk of emotional exhaustion and burnout. Effectively addressing aggression requires a thoughtful and comprehensive approach that may incorporate elements of environmental management, evaluation of potential motivating factors, differential diagnosis, and a coordinated intervention. This always involves includes effective communication among stakeholders including the patient. Recommended milieu changes and psychotherapeutic and / or pharmacologic interventions need to be explicitly defined; available data are described in this chapter. Consistent oversight and follow up to measure the effects of each component of the intervention(s) is critical, as aggressive behavior may be both habitual and episodic. This chapter reviews the factors that contribute to the broad range of assaultive behavior observed in correctional settings, and some of the pragmatic issues and opportunities for assessment, diagnosis, and treatment of aggressive behaviors, both impulsive and predatory.
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Buckholtz, Joshua W., and Andreas Meyer-Lindenberg. Genetic Perspectives on the Neurochemistry of Human Aggression and Violence. Edited by Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.009.

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Violence is a devastating social phenomenon that is costly both to affected individuals and to society at large. Pathological aggression, especially reactive/impulsive aggression, is a cardinal symptom common to several psychiatric disorders—including antisocial personality disorder, borderline personality disorder, and psychopathy—that are associated with risk for violence. Thus, understanding the factors that predispose people to impulsive violence represents a crucial goal for psychology, neuroscience, and psychiatry. Although we are far from a full understanding of the etiopathophysiology of violence, impulsive aggression is heritable, suggesting that genetic mechanisms may be important for determining individual variation in susceptibility. This chapter synthesizes available preclinical and human data to propose a compelling neurogenetic mechanism for violence, specifically arguing that a genetically determined excess in serotonin signaling during a critical developmental period leads to dysregulation within a key corticolimbic circuit for emotional arousal and regulation, inhibitory control, and social cognition.
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Feinstein, Robert E., and Brian Rothberg. Violence. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0013.

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Potentially violent patients need immediate attention and evaluation to determine their risk of imminent violence. A past history of violence is the best predictor of future violent behavior, and individuals who have committed violent acts in the past and have been arrested for assaultive behavior represent the highest risk; people who carry weapons or have access to weapons are of relatively high risk. Individuals with violent impulses who are either intoxicated or are in withdrawal have the most extreme risk for imminent violence. The treatment of acute aggression or agitation involves the judicious use of sedative-anxiolytics or low doses of second-generation antipsychotics. SSRIs have been used to treat aggressive, impulsive, and violent symptoms, particularly in individuals with head injuries, and lithium carbonate can reduce impulsive aggression to extremely low levels in some aggressive patients. Two Tarasoff decisions have become national standards for clinical practice regarding “duty to warn” and “duty to protect” all potential victims of life-threatening danger from a homicidal patient.
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Patton, Jim H., and Matthew S. Stanford. Psychology of Impulsivity. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0086.

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Impulsive behavior is generally viewed as counterproductive by society, and individual differences in impulsivity have been found to be related to a number of socially relevant behaviors. Yet, there are times when acting quickly and without thinking may seem desirable, even adaptive. With the possible exception of intelligence, no other personality dimension or trait so broadly influences various areas of human endeavor: interpersonal relationships, education, fiscal responsibility, personal moral behavior, business ethics and entrepreneurship, aggression, and criminality. This chapter gives an overview of impulsivity from a personality theory perspective. Topics discussed include the historical development of the construct, the place of impulsivity in a broader personality theory, self-report and behavioral assessment, and the role of impulsiveness in impulse control disorders.
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Book chapters on the topic "Impulsive aggression"

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Carroll, Marilyn E., Peter A. Santi, Joseph Zohar, Thomas R. E. Barnes, Peter Verheart, Per Svenningsson, Per E. Andrén, et al. "Impulsive Aggression." In Encyclopedia of Psychopharmacology, 624. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1022.

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Dorfman, Hayley M., Andreas Meyer-Lindenberg, and Joshua W. Buckholtz. "Neurobiological Mechanisms for Impulsive-Aggression: The Role of MAOA." In Neuroscience of Aggression, 297–313. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/7854_2013_272.

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Bernagie, Katrien, Ingeborg Goethals, Kurt Audenaert, Filip Jacobs, Kathelijne Peremans, Rudi Dierckx, and C. van Heeringen. "Functional Brain Imaging of Impulsive Aggression." In Nuclear Medicine in Psychiatry, 487–505. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18773-5_29.

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Berkowitz, Leonard. "On the Determinants and Regulation of Impulsive Aggression." In The Plenum Series in Social/Clinical Psychology, 187–211. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5883-5_12.

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Janke, Wilhelm. "Erfassung von aggressivem und impulsivem Verhalten: Ansätze der Psychologie." In Aggression und Autoaggression, 35–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77431-7_3.

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Boutros, Nash N. "Violence Aggression and Impulse Dyscontrol." In Standard EEG: A Research Roadmap for Neuropsychiatry, 83–94. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-13867-1_9.

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Peremans, Kathelijne, Simon Vermeire, Tim Waelbers, Eva Vandermeulen, André Dobbeleir, Bart De Spiegeleer, and Kurt Audenaert. "Brain SPECT in the Impulsive-Aggressive Dog." In PET and SPECT in Psychiatry, 621–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-40384-2_26.

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Stowe, Robert M. "Impulse Control Disorder." In Handbook of Aggressive and Destructive Behavior in Psychiatric Patients, 287–304. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2403-8_17.

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O'Leary, K. Daniel, and Ingrid Solano. "Relationship Discord, Intimate Partner Physical Aggression, and Externalizing Problems of Children." In The Wiley Handbook of Disruptive and Impulse-Control Disorders, 291–305. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119092254.ch18.

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Mehler-Wex, Claudia, Marcel Romanos, and Andreas Warnke. "Aggressive and Autoaggressive Behavior, Impulse Control Disorder, and Conduct Disorder." In Psychiatric Drugs in Children and Adolescents, 337–50. Vienna: Springer Vienna, 2014. http://dx.doi.org/10.1007/978-3-7091-1501-5_9.

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Conference papers on the topic "Impulsive aggression"

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Wilde, Douglass J., and Sohyeong Kim. "Creative and Supporting Cognitive Modes." In ASME 2007 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/detc2007-35764.

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The psychiatrist Carl G. Jung has asserted that the “creative impulse” — the drive to solve problems — is almost an instinct on a par with hunger, sex drive, aggression and flight from danger. Unlike the instincts, however, creativity can be extinguished or can atrophy from disuse. Jung’s personality theory identifies eight “cognitive modes” which channel the creative impulse in different directions for various people. A given person’s “creative mode” — the most conscious — can be identified by the four-letter code of the Myers-Briggs Type Indicator (MBTI®). Of the remaining seven modes, as many as three “supporting” modes can be found by simple calculation using the four “clarity index” numbers associated with the MBTI® letters. Although these supporting modes are not the “creative” one, they back it up during problem-solving and at Stanford have been found useful for forming, organizing and analyzing student design teams.
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Dolatabadi, Nader, Stephanos Theodossiades, and Steve J. Rothberg. "Application of Nonlinear Vibration Absorbers to the Control of Piston Secondary Motion in Internal Combustion Engines." In ASME 2015 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/detc2015-47410.

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The impulsive behavior of piston plays a key role in the Noise, Vibration and Harshness (NVH) of internal combustion engines. There have been several studies on the identification and quantification of piston impacting action under various operation conditions. In the current study, the dynamics of piston secondary motion are briefly explored, since this is fundamental to understanding the aggressive oscillations, energy loss and noise generation. Concepts of controlling piston secondary motion (and thus, impacts) are investigated and a new passive control approach is presented based on the nonlinear energy absorption of the highly transient oscillations. The effectiveness of this new method on the improvement of piston impact behavior is discussed, using a preliminary optimization exercise (with respect to engine excitation/speed, damping and stiffness of the nonlinear oscillator) that leads to the conceptual design of a nonlinear energy absorber.
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Meena, J., A. Parthasarathy, R. Vatsa, N. Singh, S. Kumar, K. K. Roy, and S. Singhal. "Radical excision of a massive vulvo-vaginal mass." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685391.

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Background: Vulvo-vaginal masses has a varied presentation and causes. The most common differential diagnosis are condylomata acuminata, vulvular abscess, vulvular/vaginal cysts, vulval carcinoma, vulval lipoma, angiomyofibroblastoma and aggressive angiomyxoma. Surgical excision of the mass is the main method of treatment and the outcome differs with the histological diagnosis. We present a video of excision of a massive Vulvo vaginal mass in toto. Case: A 45 year old P3 L3 female, presented with complaint of mass in perineal area and discharge per vaginum for 2 years. The mass was growing progressively and reached the present size. On examination there was a 9 × 8 cm irregular firm to cystic mass, arising from posterior wall of vagina and protruding out of introitus with bossellated surface. The mass also extended into right ischiorectal fossa, 10 × 10 cm mass with cystic, smooth surface that was irreducible with no cough impulse. CECT abdomen and pelvis revealed a well-defined 12 × 10 × 8 cm mass in right perineum arising from right lower lateral vaginal wall with ischiorectal fossa extension. There was no extension into cervix, bladder or rectum. Biopsy taken from the mass was inconclusive. A wide local excision was done under general anesthesia wherein an ischiorectal and vaginal mass of size 30 × 10 cm with irregular margin was excised in toto. Histopathology was suggestive of aggressive angiomyxoma. The patient is under follow up. Discussion: Aggressive Angiomyxoma is a rare slow growing locally invasive mesenchymal tumor and has a substantial potential for recurrence. It is often misdiagnosed. Pre-operative diagnosis is difficult due to rarity of this entity and absence of diagnostic features, but it should be considered in case of masses in genital, perianal and pelvic region in a woman of reproductive age. Radical surgical excision is the first line of management. A long term follow up of the case is necessary and MRI is preferred method for detecting recurrences.
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Meena, J., A. Parthasarathy, R. Vatsa, N. Singh, S. Kumar, K. K. Roy, and S. Singhal. "Radical excision of a massive vulvo-vaginal mass." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685394.

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Background: Vulvo-vaginal masses has a varied presentation and causes. The most common differential diagnosis are Condylomata acuminata, Vulvular abscess, Vulvular/vaginal cysts, Vulval carcinoma, Vulval lipoma, Angiomyofibroblastoma and Aggressive Angiomyxoma. Surgical excision of the mass is the main method of treatment and the outcome differs with the histological diagnosis. We present a video of excision of a massiveVulvo vaginal mass in toto. Case: A 45 year old P3 L3female, presented with complaint of mass in perineal area & discharge per vaginum for 2 years. The mass was growing progressively and reached the present size. On examination there was a 9 X 8 cm irregular firm to cystic mass, arising from posterior wall of vagina and protruding out of introitus with bossellated surface. The mass also extended into right ischiorectal fossa, 10 X 10 cm mass with cystic, smooth surface that was irreducible with no cough impulse. CECT abdomen and pelvis revealed a well-defined 12 X 10 X 8 cm mass in right perineum arising from right lower lateral vaginal wall with ischiorectal fossa extension. There was no extension into cervix, bladder or rectum. Biopsy taken from the mass was inconclusive. A wide local excision was done under general anesthesia wherein an ischiorectal and vaginal mass of size 30 X 10 cm with irregular margin was excised in toto. Histopathology was suggestive of Aggressive Angiomyxoma. The patient is under follow up. Discussion: Aggressive Angiomyxoma is a rare slow growing locally invasive mesenchymal tumor and has a substantial potential for recurrence. It is often misdiagnosed. Pre-operative diagnosis is difficult due to rarity of this entity and absence of diagnostic features, but it should be considered in case of masses in genital, perianal and pelvic region in a woman of reproductive age. Radical surgical excision is the first line of management. A long term follow up of the case is necessary and MRI is preferred method for detecting recurrences.
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Meena, J., A. Parthasarathy, R. Vatsa, N. Singh, S. Kumar, K. K. Roy, and S. Singhal. "Radical excision of a massive vulvo: Vaginal mass." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685374.

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Background: Vulvo-vaginal masses has a varied presentation and causes. The most common differential diagnosis are Condylomata acuminata, Vulvular abscess, Vulvular/vaginal cysts, Vulval carcinoma, Vulval lipoma, Angiomyofibroblastoma and Aggressive Angiomyxoma. Surgical excision of the mass is the main method of treatment and the outcome differs with the histological diagnosis. We present a video of excision of a massive Vulvo vaginal mass in toto. Case: A 45 year old P3 L3 female, presented with complaint of mass in perineal area & discharge per vaginum for 2 years. The mass was growing progressively and reached the present size. On examination there was a 9 X 8 cm irregular firm to cystic mass, arising from posterior wall of vagina and protruding out of introitus with bossellated surface. The mass also extended into right ischiorectal fossa, 10 X 10 cm mass with cystic, smooth surface that was irreducible with no cough impulse. CECT abdomen and pelvis revealed a well-defined 12 X 10 X 8 cm mass in right perineum arising from right lower lateral vaginal wall with ischiorectal fossa extension. There was no extension into cervix, bladder or rectum. Biopsy taken from the mass was inconclusive. A wide local excision was done under general anesthesia wherein an ischiorectal and vaginal mass of size 30 X 10 cm with irregular margin was excised in toto. Histopathology was suggestive of Aggressive Angiomyxoma. The patient is under follow up. Discussion: Aggressive Angiomyxoma is a rare slow growing locally invasive mesenchymal tumor and has a substantial potential for recurrence. It is often misdiagnosed. Pre-operative diagnosis is difficult due to rarity of this entity and absence of diagnostic features, but it should be considered in case of masses in genital, perianal and pelvic region in a woman of reproductive age. Radical surgical excision is the first line of management. A long term follow up of the case is necessary and MRI is preferred method for detecting recurrences.
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Anakapalli, Manoj, P. Raju Mantena, Ahmed Al-Ostaz, and S. Jimmy Hwang. "Impulse-Frequency Response and EIS Based NDE for Characterizing Bond Integrity of Adhesive Joints Subjected to Environmental Degradation." In ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-79421.

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Single-Lap Joint (SLJ) and three-point end-notched flexure (ENF) joint configurations were used to bond 1” × 1/8″ (25.4mm × 3.175mm) aluminum 2024 T-4 adherends using a range of 3M™ high performance pressure sensitive adhesives (Adhesives 69, 73 and 85) and VHB™ acrylic foam tapes (Foam 41, 50, 52). Batches of bonded specimens were subjected to two types of aggressive environments simulating extreme service conditions: freeze-thaw cycling from 10°F to 50°F at 6 cycles per day (ASTM C666 Procedure A) for 21 days with samples immersed in water; heat-cool cycling (with 90% of maximum recommended temperature by the manufacturer of both acrylic foam and adhesive transfer tapes attained at 70% relative humidity) and 3 cycles per day for 21 days. Electrochemical Impedance Spectroscopy (EIS) and Fast Fourier Transform (FFT) based impulse frequency response vibration Non-Destructive Evaluation (NDE) techniques were used to monitor overall bond integrity.
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Anakapalli, Manoj, P. Raju Mantena, Ahmed Al-Ostaz, and S. Jimmy Hwang. "Quasi-Static Lap Shear and Dynamic Impact of High Performance VHB™ Acrylic Foam and Adhesive Transfer Tape Bonded Aluminum Joints Subjected to Environmental Degradation." In ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-79425.

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A range of 3M™ VHB™ acrylic foam tapes and high performance adhesive transfer tapes were used to bond 1” × 1/8″ (25.4 mm × 3.175 mm) aluminum 2024 T-4 adherends in single-lap joint (SLJ) and three-point end-notched flexure (ENF) configurations. Three types of 0.045” thick double-coated acrylic foam tapes: Foam 41, 50 and 52 (firm, soft and softer), and three types of adhesive transfer tapes: Adhesives 69, 73 and 85 (0.005”, 0.01” and 0.005” thick, respectively) were used for this study. The samples were subjected to two types of aggressive environments simulating extreme service conditions: freeze-thaw cycling from 10°F to 50°F at 6 cycles per day (ASTM C666 Procedure A) for 21 days with samples immersed in water; heat-cool cycling (with 90% of maximum recommended temperature by the manufacturer of both acrylic foam and adhesive transfer tapes attained at 70% relative humidity) and 3 cycles per day for 21 days. Initially the impulse-frequency response vibration and electrochemical impedance spectroscopy (EIS) techniques were used for monitoring bond quality nondestructively and selecting the best out of 250 fabricated samples. After obtaining baseline data, the specimens were subjected to quasi-static lap-shear and dynamic impact loading to compare their lap-shear failure loads and shear energy along with the impact load and energy absorbed.
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