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Dissertations / Theses on the topic 'In vitro ischemia model'

Author: Grafiati

Published: 9 March 2023

Last updated: 10 March 2023

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1

Champattanachai, Voraratt. "Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2008. https://www.mhsl.uab.edu/dt/2008d/champattanachai.pdf.

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2

Frantseva, Marina. "Mechanisms of free radical formation and toxicity in an in vitro model of ischemia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ45776.pdf.

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3

Zwaini, Zinah Dheyaa Razzaq. "In vitro and in vivo models of renal ischemia reperfusion injury." Thesis, University of Leicester, 2017. http://hdl.handle.net/2381/39344.

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Successful kidney transplantation is a life-saving procedure to patients with irreversible chronic renal failure. Despite the presence of various obstacles facing this surgery, preserving donor kidney and consequent ischemia reperfusion injury (IRI) are still major challenges affecting renal function as well as prognosis of transplant surgery. This study pursued two main aims: firstly, characterising changes in damage associated inflammatory gene expressions through developing, and analysis of an in vitro model of proximal tubular epithelial cells (PTEC) of normal human kidney mimicking renal

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4

Zur, Nedden Stephanie. "Targeting the purine salvage pathway in in vitro models of cerebral ischemia." Thesis, University of Warwick, 2011. http://wrap.warwick.ac.uk/45926/.

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An interruption of the blood supply to the brain, as occurs during ischemic stroke, results in a rapid decline of ATP levels and a subsequent loss of neuronal function and viability. Under physiological conditions the brain reuses ATP degradation metabolites, such as hypoxanthine, via the purine salvage pathway, to restore its ATP pool. However, the massive degradation of ATP during ischemia results in the accumulation and loss of diffusible purine metabolites and thereby leads to a reduction in the post-ischemic ATP pool size, leaving the brain more vulnerable to secondary ischemic insults (r

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5

CARROZZINI, TATIANA. "Nutrition interventions in aging: study of coffee-derived compounds antioxidant properties in an in vitro model of ischemia." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2021. http://hdl.handle.net/10281/309808.

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Negli ultimi secoli, l'aspettativa di vita è aumentata grazie a uno stile di vita migliore, ma di conseguenza sono aumentate le patologie legate all'invecchiamento. L'invecchiamento è un processo fisiologico complesso e modificazioni legate all'età sono evidenti anatomicamente e fisiologicamente nella BEE. L'accumulo di danno ossidativo alle macromolecole da parte di RONS e ROS in BEE può essere cruciale nello sviluppo e nella progressione di diverse patologie del SNC. In questa situazione l'ischemia cerebrale potrebbe alterare ulteriormente l'equilibrio ossidante/antiossidante a favore degli

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6

Kim-Lee, Myung Hi. "Perfusion/reperfusion cell damage in vitro : an ischemic model in CNS /." The Ohio State University, 1991. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487758680162691.

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7

Wang, Jie. "The Study of the Effects of (1S,2E,4R,6R,-7E,11E)-2,7,11-cembratriene-4,6-diol on Microglia Polarization Using an Ischemia in Vitro Model." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1491559717910191.

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8

Tsang, Hing-wai, and 曾慶威. "In vitro studies of hypoxic ischemic down-regulated 1 (HID-1) protein encoded by a novel gene down-regulated in neonatal hypoxic-ischemicencephalopathy in different cell death paradigms." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45608192.

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9

Sánchez, Opazo Guillem. "Estudi dels mecanismes de mort cel·lular induïts per un model d’isquèmia cerebral in vitro: implicació dels antagonistes dels receptors de mortJosé Rodríguez Álvarez." Doctoral thesis, Universitat Autònoma de Barcelona, 2014. http://hdl.handle.net/10803/284058.

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L’ictus o accident cerebrovascular és la segona causa de mort en els països industrialitzats i constitueix la primera causa de discapacitat en adults. L’únic tractament aprovat en l’actualitat és el trombolític activador del plasminògen tissular (tPA), el qual només es pot aplicar en un nombre molt reduït de pacients i dintre d’una estreta finestra terapèutica. Els mecanismes de mort cel·lular en la isquèmia cerebral són amplis i venen provocats per l’interrupció del flux sanguini al cervell, el qual provoca una mort ràpida i eminentment necròtica en el nucli de la zona afectada i una mort de

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10

BACIGALUPPI, SUSANNA. "Ruolo e potenziale delle cellule progenitrici endoteliali nel vasospamo cerebrale." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2011. http://hdl.handle.net/10281/27113.

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Title: Role and potential of endothelial progenitor cells in cerebral vasospasm Abstract: Background and aim: Despite many treatment approaches, cerebral vasospasm and delayed ischemic neuronal damage (DIND) still represent a serious threat to patients with subarachnoid haemorrhage (SAH). Endothelial progenitor cells (EPC) have been involved as prognostic indicators in several vascular diseases and mesenchymal stem cells already have shown some benefits in ischemic injury. Aim o

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11

Lübbe, Katharina. "Entwicklung und Einsatz eines In-vitro-Ischämiemodels zur Untersuchung zellulärer Pathomechanismen der Klauenrehe des Rindes." Doctoral thesis, Universitätsbibliothek Leipzig, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-171594.

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Die subklinische Klauenrehe oder claw horn disruption (CHD) ist von großer wirtschaftlicher Bedeutung für die Rinderhaltung, da sie zu Lahmheiten, Beeinträchtigungen des Allgemeinbefindens sowie einer eingeschränkten Leistungsfähigkeit der Tiere führt. Trotz zahlreicher Untersuchungen sind die pathophysiologischen Grundlagen der CHD noch immer nicht vollständig geklärt. Die derzeitigen Hypothesen weisen auf eine Ischämie in den noch lebensfähigen Epidermisschichten infolge einer veränderten dermalen Mikrozirkulation. Diese hat pathophysiologische Veränderungen zur Folge, die eine Störung der e

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12

Lübbe, Katharina [Verfasser], Christoph K. W. [Akademischer Betreuer] Mülling, and Alois [Gutachter] Boos. "Entwicklung und Einsatz eines In-vitro-Ischämiemodels zur Untersuchung zellulärer Pathomechanismen der Klauenrehe des Rindes : Development and experimental application of an in vitro ischemia model for investigating the cellular pathomechanism of laminitis in cattle / Katharina Lübbe ; Gutachter: Alois Boos ; Betreuer: Christoph K.W. Mülling." Leipzig : Universitätsbibliothek Leipzig, 2015. http://d-nb.info/1239566565/34.

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13

Poizat, Adrien. "Contrôle temporel de la cavitation ultrasonore : application à la thrombolyse ultrasonore extracorporelle." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1031/document.

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Les ultrasons focalisés permettent d’effectuer des traitements thérapeutiques ciblés dans le corps humain. Dans le domaine des applications cardiovasculaires, ils permettent de détruire des caillots sanguins susceptibles de se former dans le système vasculaire. Dans ce cas, les mécanismes de thrombolyse sont largement liés à la cavitation ultrasonore, dont la dynamique complexe reste un obstacle à l’élaboration d’un dispositif thérapeutique. Dans le cadre de cette thèse, un système permettant le contrôle temporel de l’activité de cavitation en régime pulsé a été développé puis caractérisé. Ce

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14

Barandier, Christine. "Potentiel thérapeutique du manganèse et de l'un de ses dérivés synthétiques sur le système cardiovasculaire." Université Joseph Fourier (Grenoble), 1998. http://www.theses.fr/1998GRE10238.

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Le present travail qui s'inscrit dans le cadre general des etudes consacrees a la protection pharmacologique des tissus cardiaque et vasculaire au cours de la reperfusion post-ischemique, comprend deux parties principales. La premiere a ete realisee sur un modele d'ischemie/reperfusion myocardique sur un modele experimental de coeur isole de rat. Les resultats sont exprimes en termes de donnees fonctionnelles, metaboliques et ultrastructurales. La seconde partie est une etude pharmacologique menee sur un modele d'anneaux d'aorte isolee de rat et comporte essentiellement des mesures de contract

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15

SAKAMOTO, NOBUO, TATSUAKI MATSUBARA, YOSHIHIRO KAKINUMA, and TATSUO HASHIMOTO. "MYOCARDIAL METABOLIC MARKERS OF TOTAL ISCHEMIA IN VITRO." Nagoya University School of Medicine, 1994. http://hdl.handle.net/2237/15927.

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16

Thomas, Sunu Samuel. "Murine models of cerebral ischemia, development of a mouse model of global cerebral ischemia; response of GluR2 knockout mice in a model of permanent focal cerebral ischemia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0026/MQ50439.pdf.

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17

Pruitt, Christopher Rogers. "Early Mediators of Cutaneous Ischemia Reperfusion Injury: A Mouse Model." VCU Scholars Compass, 2006. http://hdl.handle.net/10156/2024.

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18

Saito, Masao. "Pirfenidone alleviates lung ischemia-reperfusion injury in a rat model." Kyoto University, 2019. http://hdl.handle.net/2433/242360.

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19

Chan, Chu-fung. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B40687284.

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20

Todd, Michael. "The effects of multiple ischemia and survival times on hippocampal CA1 neuronal cell loss in a rat model of global ischemia: A long-term ischemia maturation study." Thesis, University of Ottawa (Canada), 1998. http://hdl.handle.net/10393/4230.

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We investigated the effects of varying ischemia and survival times on CA1 neuronal loss. Histological analysis of the hippocampus was performed at 2, 7, 14, 28 and 90 days following 3, 5, 7, 10 and 13 minutes of global forebrain ischemia. Our results indicate that the ischemic maturation process extends beyond 7 days. Ten and thirteen minutes of ischemia produced a significant degree of cell loss by 7 days (70.53% and 83.25% respectively), while average cell death at 90 days survival was approximately 12% higher. Most strikingly, seven minutes of ischemia produced approximately 30% CA1 cell lo

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21

Todd, Mike. "The effects of multiple ischemia and survival times on hippocampal CA1 neuronal cell loss in a rat model of global ischemia, a long-term ischemia maturation study." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ36746.pdf.

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22

Ariga, Suely Kunimi Kubo. ""Modulação térmica da lesão isquêmica: estudo in vitro"." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-14092006-144005/.

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A isquemia cerebral causada pela parada cardíaca leva ao desapareciemnto neuronal. studamos os mecanismos de morte celular envolvidos na isquemia in vitro em linhagem de neuroblastoma.O insulto isquêmicao foi reproduzido cultivando as células sem fatores de crescimento, sem glicose e em embiente hipóxico produzido por um sistema de anaerobiose. Os resultados sugerem que a privação de oxigênio, glicose e fatores de cresciemtno do meio de cultura reproduzem o fenômeno semelhante a isquemia. INvestigamos ainda a participação de processo apoptótico e sua modulação térmica. Observams que a hipoterm

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23

Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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24

BRESCHI, GIAN LUCA. "STUDIO ANATOMICO E FUNZIONALE DELLA REGIONE DI PENOMBRA IN UN MODELLO DI ISCHEMIA IN VITRO." Doctoral thesis, Università degli Studi di Milano, 2013. http://hdl.handle.net/2434/215236.

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ANATOMICAL AND FUNCTIONAL STUDY OF THE PENUMBRA REGION IN AN IN VITRO MODEL OF FOCAL CEREBRAL ISCHEMIA Experimental data have shown that the ischemic brain region is characterized by a highly damaged core surrounded by a rim of reversibly altered tissue, commonly referred to as the ischemic penumbra. The core region is characterized by a severely compromised CBF, whereas in the penumbra CBF is reduced but cellular metabolism is still preserved (Hossmann, 2008). One possible approach to identify core and penumbra is based on the evaluation of both metabolism and perfusion of the cerebral tis

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25

Shaw, Matthew J. "Apoptosis following ischemia-reperfusion injury in a rabbit lung ex-vivo model." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq64450.pdf.

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26

Shaw, Matthew J. "Apoptosis following ischemia-reperfusion injury in a rabbit lung ex-vivo model." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30745.

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Background. Apoptosis is postulated as a mechanism involved in lung ischemia-reperfusion (IR) injury, however, the relative contributions of ischemia and reperfusion are unclear.<br>Methods. Heart-lung blocks were harvested from New Zealand white rabbits (3.0--4.0 kg) and exposed to 0, 6, or 18 hours of cold ischemia (4°C), followed by 3 hours of reperfusion in an ex vivo model. Terminal dUTP nick-end labeling (TUNEL), the technique used most often for detection of apoptosis, was performed on the tissue sections.<br>Results. TUNEL demonstrated minimal apoptosis in lungs exposed to 0, 6, or 18

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27

O, H.-Ici Darach Michael. "Multi-modal imaging of myocardial ischemia and reperfusion in a rat model." Thesis, King's College London (University of London), 2017. https://kclpure.kcl.ac.uk/portal/en/theses/multimodal-imaging-of-myocardial-ischemia-and-reperfusion-in-a-rat-model(43bf5945-ce02-48cc-a634-2ebf00cffc99).html.

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Myocardial ischaemia causes progressive cellular injury. Initially there is loss of function, followed by formation of oedema. Ischemia, if prolonged, will eventually lead to cell death. Revascularisation of occluded coronary arteries is an efficient tool to reduce infarct size in acute myocardial infarction. However, reperfusion in itself can be associated with a significant amount of myocardial damage. The structural and metabolic changes occurring in myocardial ischemia and reperfusion can now be studied in-vivo using magnetic resonance imaging (MRI) and are targets to optimise the treatmen

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28

Siu, Ada Hoi Ling. "Cardioprotective effects of herba cistanche on ischemia/reperfusion injury ex vivo and oxidative injury in vitro /." View abstract or full-text, 2008. http://library.ust.hk/cgi/db/thesis.pl?BICH%202008%20SIU.

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29

Ohsumi, Akihiro. "Protective effect of pre-recovery surfactant inhalation on lungs donated after cardiac death in a canine lung transplantation model." Kyoto University, 2017. http://hdl.handle.net/2433/218007.

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30

Cardella, Jonathan A. "A novel cell culture model to study ischemia-reperfusion injury in lung transplantation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ46106.pdf.

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31

Wang, Yanxin. "Hypoxic-ischemic injury in the neonatal rat model prediction of irreversible infarction size by Diffusion Weighted MR Imaging /." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B35757577.

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32

Balkaya, Mustafa G. [Verfasser]. "Stroke, stress, and depression - evidence from a brain ischemia mouse model / Mustafa G. Balkaya." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1052221882/34.

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33

Liu, Lingguang, and 刘灵光. "Neuroprotection of melatonin and/or electro-acupuncture in a rat model of focal cerebral ischemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/198928.

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Stroke is a serious cerebral vascular event and a leading cause of death and disability worldwide, and ischemic stroke is the most common type. Evidence from animal research in acute cerebral ischemia shows that a combination of neuroprotectants might be more efficacious than the single agent given individually. Both melatonin and electro-acupuncture (EA) have been suggested to be effective treatments against cerebral ischemia. However, it is unknown whether a combination of these two therapies could be beneficial against focal cerebral ischemia. In the first study, the effect of post-tr

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34

Chan, Pui-shan. "Effects of NPY-Y1 receptor activation or inhibition on free radical generation during in vitro or in vivo cerebral ischemia." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B35760825.

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35

MARIANI, JACOPO. "MULTICENTRE AND MULTISPECIES PRECLINICAL TRIAL OF REMOTE ISCHEMIC CONDITIONING IN ANIMAL MODEL OF ACUTE ISCHEMIC STROKE (TRICS–BASIC)." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2023. https://hdl.handle.net/10281/403043.

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Il condizionamento ischemico remoto (RIC) risulta essere un candidato ideale per essere investigato in uno studio multicentrico volto al trattamento dell'ictus ischemico acuto (AIS). L’ efficacia terapeutica del RIC è stata dimostrata a livello preclinico, come riportato da precedenti studi ottenuti da singoli laboratori; ciò nonostante, gli studi clinici di fase II-III non hanno ancora fornito risultati soddisfacenti. TRICS Basic è definita come la fase preclinica del progetto TRICS, un Trial traslazionale multicentrico di Condizionamento Ischemico Remoto in modelli animali di Ictus Ischemic

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36

Chase, Teena D. "An in vitro model of reactive astrogliosis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ49329.pdf.

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37

Worrall, Lisa Kirsty. "A 3D in vitro breast cancer model." Thesis, University of Nottingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.436812.

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38

Vaskas, Jonas. "Impaired Cardiac cAMP-specific PDE4, β1-AR, and NE in an Ischemia-Reperfusion Rat Model". Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31741.

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Ischemic injury in the heart is followed by an increase in SNS activity and the higher this activity, the poorer patient outcomes. An index of SNS activity in models of ischemia can be achieved by measuring NE, β-AR, and perhaps indirectly PDE4 to give an intracellular aspect on SNS signaling. A 20 minute ischemia-reperfusion was induced in a rat model with physiological measurements at 2-5 weeks post IR. At 3 weeks post IR, rats displayed increased PDE4 expression, decreased β 1-AR expression, increased plasma NE, decreased tissue NE storage, increased Doppler E/A ratio and unchanged LV eject

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39

Corbisiero, Rafael M. "An ischemia reperfusion compartment syndrome model in the canine hindlimb : analysis of present treatment modalities." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61886.

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40

Zanoni, Diogo Sousa [UNESP]. "Experimental glaucoma model (ischemia and reperfusion): histology, morphometry, protein and gene espression of apoptosis pathway." Universidade Estadual Paulista (UNESP), 2015. http://hdl.handle.net/11449/132013.

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Made available in DSpace on 2015-12-10T14:23:03Z (GMT). No. of bitstreams: 0 Previous issue date: 2015-05-28. Added 1 bitstream(s) on 2015-12-10T14:29:18Z : No. of bitstreams: 1 000852681.pdf: 1224340 bytes, checksum: 7e3993edae8f0d4badfed835114ffa9b (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Purpose: The aims of this study were to better understand the mechanism of cell death by apoptosis in a glaucoma model (ischemia / reperfusion) and evaluate the role of apoptosis in this model and if treatment with Sildenafil helps prevent apoptosis. Methods: 36 rats, from

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41

Zanoni, Diogo Sousa. "Experimental glaucoma model (ischemia and reperfusion) : histology, morphometry, protein and gene espression of apoptosis pathway /." Botucatu, 2015. http://hdl.handle.net/11449/132013.

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Orientador: Renée Laufer Amorim<br>Coorientador: José Luiz Laus<br>Banca: Juliany Quitzan Gomes<br>Banca: Alvio Isao Shiguematsu<br>Resumo: Não disponível<br>Abstract: Purpose: The aims of this study were to better understand the mechanism of cell death by apoptosis in a glaucoma model (ischemia / reperfusion) and evaluate the role of apoptosis in this model and if treatment with Sildenafil helps prevent apoptosis. Methods: 36 rats, from 4 to 6 months, males, Lewis and weighing ± 350g were divided in 5 groups: control group (6 animals) and for groups with ischemia / reperfusion (7 and 21 days)

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42

Abood, Sarah K. "Effect of glutamine on mucosal permeability in a canine model of ischemia and reperfusion injury /." The Ohio State University, 1997. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487947908401747.

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43

Kanazawa, Hiroyuki. "Bone marrow-derived mesenchymal stem cells ameliorate hepatic ischemia reperfusion injury in a rat model." Kyoto University, 2012. http://hdl.handle.net/2433/157437.

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44

Albiero, Mattia. "The role of p66Shc knockout in a murine model of diabetic ulcers and peripheral ischemia." Doctoral thesis, Università degli studi di Padova, 2010. http://hdl.handle.net/11577/3427554.

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This thesis studied the role of p66shc in a murine model of ulcers complicated with peripheral ischemia. This study showed that the knockout of p66Shc improved wound healing in the setting of diabetes and ischemia. Moreover, diabetes increases the expression of p66Shc.<br>Questa tesi ha studiato il ruolo di p66Shc in un modello murino di ulcere diabetiche complicate da ischemia periferica. Questo studio ha dimostrato che il knockout di p66Shc migliora la guarigione delle ulcere nel contesto di diabete ed ischemia. Il diabete, inoltre, aumenta l'espressione di p66Shc.

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Giedt, Randy James. "Real-Time Acquisition and Analysis of Endothelial Mitochondrial Superoxide Radical Production and Membrane Potential During In Vitro Ischemia/Reperfusion." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243541457.

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46

Chan, Pui-shan, and 陳佩珊. "Effects of NPY-Y1 receptor activation or inhibition on free radical generation during in vitro or in vivo cerebral ischemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B35760825.

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47

Akinrinmade, Olusiji Alex. "Potential neuroprotective effects of fermented rooibos herbal tea in a rat model of ischemic brain injury." University of the Western Cape, 2015. http://hdl.handle.net/11394/4705.

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Magister Scientiae (Medical Bioscience) - MSc(MBS)<br>Stroke is the third leading cause of death in South Africa, killing about 240 people a day and leaving survivors with residual disabilities. There is no clinically approved neuroprotective agent for stroke at the moment but the consumption of plant polyphenols has been suggested to offer neuroprotection against stroke and other neurodegenerative diseases. In this study, we investigated the effects of long term consumption of fermented rooibos herbal tea (FRHT) on ischemia reperfusion brain injury (I-RBI) in rats. Male adult Wistar rats were

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48

Na, Rongrui [Verfasser], and René [Akademischer Betreuer] Schramm. "Intravital microscopy of lung ischemia reperfusion injury in a rat model / Rongrui Na ; Betreuer: René Schramm." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2021. http://d-nb.info/1227840012/34.

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49

Rich, Barbara Sharon. "Development of an in vitro model for accommodation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq28982.pdf.

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50

Aldsworth, Timothy Grant. "Microbial in vitro model of root surface caries." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360285.

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