Relevant bibliographies by topics / Incompatibility system

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Academic literature on the topic 'Incompatibility system'

Author: Grafiati
Published: 25 May 2024

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Journal articles on the topic "Incompatibility system"

1

Rashid, Abdul, and Peter A. Peterson. "The RSS system of unidirectional cross-incompatibility in maize. 2. Cytology." Genome 35, no. 4 (1992): 560–64. http://dx.doi.org/10.1139/g92-083.

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In 1975, a number of genetic lines discovered in our maize genetics nursery in Ames, Iowa, showed unidirectional cross-incompatibility. Later, it was found that this unidirectional cross-incompatibility is controlled by three recessive genes. One locus (cif) controls the incompatibility reaction in the female tissue and the other two (cim1 and cim2) control the incompatibility reaction in the pollen grain. The cross is incompatible only when the female parent is homozygous recessive for the cif and the male parent is homozygously recessive for the cim1 as well as the cim2 locus. Cytological st
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2

Paoletti, M., and C. Clavé. "The Fungus-Specific HET Domain Mediates Programmed Cell Death in Podospora anserina." Eukaryotic Cell 6, no. 11 (2007): 2001–8. http://dx.doi.org/10.1128/ec.00129-07.

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ABSTRACT Vegetative incompatibility is a programmed cell death reaction that occurs when fungal cells of unlike genotypes fuse. Genes defining vegetative incompatibility (het genes) are highly polymorphic, and most if not all incompatibility systems include a protein partner bearing the fungus-specific domain termed the HET domain. The nonallelic het-C/het-E incompatibility system is the best-characterized incompatibility system in Podospora anserina. Cell death is triggered by interaction of specific alleles of het-C, encoding a glycolipid transfer protein, and het-E, encoding a HET domain an
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3

Uyenoyama, M. K. "On the evolution of genetic incompatibility systems. VI. A three-locus modifier model for the origin of gametophytic self-incompatibility." Genetics 128, no. 2 (1991): 453–69. http://dx.doi.org/10.1093/genetics/128.2.453.

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Abstract Recent genetic analyses have demonstrated that self-incompatibility in flowering plants derives from the coordinated expression of a system of loci. To address the selective mechanisms through which a genetic system of this kind evolves, I present a three-locus model for the origin of gametophytic self-incompatibility. Conventional models assume that a single locus encodes all physiological effects associated with self-incompatibility and that the viability of offspring depends only on whether they were derived by selfing or outcrossing. My model explicitly represents the genetic dete
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4

Gabrielov, A., V. Keilis-Borok, and D. D. Jackson. "Geometric incompatibility in a fault system." Proceedings of the National Academy of Sciences 93, no. 9 (1996): 3838–42. http://dx.doi.org/10.1073/pnas.93.9.3838.

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5

Saupe, Sven J. "Molecular Genetics of Heterokaryon Incompatibility in Filamentous Ascomycetes." Microbiology and Molecular Biology Reviews 64, no. 3 (2000): 489–502. http://dx.doi.org/10.1128/mmbr.64.3.489-502.2000.

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SUMMARY Filamentous fungi spontaneously undergo vegetative cell fusion events within but also between individuals. These cell fusions (anastomoses) lead to cytoplasmic mixing and to the formation of vegetative heterokaryons (i.e., cells containing different nuclear types). The viability of these heterokaryons is genetically controlled by specific loci termed het loci (for heterokaryon incompatibility). Heterokaryotic cells formed between individuals of unlike het genotypes undergo a characteristic cell death reaction or else are severely inhibited in their growth. The biological significance o
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6

Kemp, R. F. O. "Incompatibility in basidiomycetes: The heterogenic Pentax." Edinburgh Journal of Botany 52, no. 1 (1995): 71–89. http://dx.doi.org/10.1017/s0960428600001931.

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A heterogenic system of incompatibility is described in Coprinus bisporus which involves two alleles at two loci, in addition to the unifactorial homogenic incompatibility locus already described for this two-spored species. The patterns of non-allelic heterogenic incompatibility found in C. bisporus are used to predict those expected in species with bifactorial homogenic incompatibility. This type of heterogenic incompatibility could lead to speciation.
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7

Leppäjärvi, Leevi, and Michal Sedlák. "Incompatibility of quantum instruments." Quantum 8 (February 12, 2024): 1246. http://dx.doi.org/10.22331/q-2024-02-12-1246.

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Quantum instruments describe outcome probability as well as state change induced by measurement of a quantum system. Incompatibility of two instruments, i. e. the impossibility to realize them simultaneously on a given quantum system, generalizes incompatibility of channels and incompatibility of positive operator-valued measures (POVMs). We derive implications of instrument compatibility for the induced POVMs and channels. We also study relation of instrument compatibility to the concept of non-disturbance. Finally, we prove equivalence between instrument compatibility and postprocessing of c
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8

KIM, KYUNGMEE O., and WAY KUO. "TWO-LEVEL BURN-IN FOR RELIABILITY AND ECONOMY IN REPAIRABLE SERIES SYSTEMS HAVING INCOMPATIBILITY." International Journal of Reliability, Quality and Safety Engineering 11, no. 03 (2004): 197–211. http://dx.doi.org/10.1142/s0218539304001464.

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When a system is assembled from components, incompatibility often occurs as a result of the assembly process. The ability to quantify incompatibility is very important for making burn-in decisions because the goal of system burn-in is to minimize the incompatibility factor. In the past, incompatibility has been only partially represented in the system prediction models because it was assumed that assembly had no effect on the components. This paper presents a more accurate model for system prediction by allowing for the possibility that, in some cases, assembly adversely affects the components
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9

Uyenoyama, M. K. "A generalized least-squares estimate for the origin of sporophytic self-incompatibility." Genetics 139, no. 2 (1995): 975–92. http://dx.doi.org/10.1093/genetics/139.2.975.

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Abstract Analysis of nucleotide sequences that regulate the expression of self-incompatibility in flowering plants affords a direct means of examining classical hypotheses for the origin and evolution of this major feature of mating systems. Departing from the classical view of monophyly of all forms of self-incompatibility, the current paradigm for the origin of self-incompatibility postulates multiple episodes of recruitment and modification of preexisting genes. In Brassica, the S locus, which regulates sporophytic self-incompatibility, shows homology to a multigene family present both in s
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10

Franklin-Tong, Vernonica E., and F. C. H. Franklin. "The different mechanisms of gametophytic self–incompatibility." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 358, no. 1434 (2003): 1025–32. http://dx.doi.org/10.1098/rstb.2003.1287.

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Self–incompatibility (SI) involves the recognition and rejection of self or genetically identical pollen. Gametophytic SI is probably the most widespread of the SI systems and, so far, two completely different SI mechanisms, which appear to have evolved separately, have been identified. One mechanism is the RNase system, which is found in the Solanaceae, Rosaceae and Scrophulariaceae. The other is a complex system, so far found only in the Papaveraceae, which involves the triggering of signal transduction cascade(s) that result in rapid pollen tube inhibition and cell death. Here, we present a
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