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Relevant bibliographies by topics / INDOLENT PRECURSOR

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Journal articles

Academic literature on the topic 'INDOLENT PRECURSOR'

Author: Grafiati

Published: 11 March 2023

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Journal articles on the topic "INDOLENT PRECURSOR"

1

Pinto-Lopes, Pedro, Francisco Adao Fonseca, Roberto Silva, Pedro von Hafe, and Elsa Fonseca. "Indolent systemic mastocytosis limited to the bone: a case report and review of the literature." Sao Paulo Medical Journal 131, no. 3 (2013): 198–204. http://dx.doi.org/10.1590/1516-3180.2013.1313460.

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CONTEXT Systemic mastocytosis is defined as a clonal disorder of mast cells and their precursor cells and is currently classified as a myeloproliferative neoplasm. Its clinical course has a wide spectrum, ranging from indolent disease, with normal life expectancy, to highly aggressive disease, associated with multisystemic involvement and poor overall survival. The aim of this study was to report a case of indolent systemic mastocytosis, focusing on the diagnostic challenges, with a review of the literature. CASE REPORT A 79-year-old Caucasian woman with osteoporosis was evaluated at the Emerg

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2

Canberk, Sule. "Precursor and borderline lesions of the thyroid (indolent lesions of epithelial origin): from theory to practice." Gland Surgery 9, no. 5 (2020): 1724–34. http://dx.doi.org/10.21037/gs-20-429.

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3

Lussier, Tyler, Natalie Schoebe, and Sabine Mai. "Risk Stratification and Treatment in Smoldering Multiple Myeloma." Cells 11, no. 1 (2021): 130. http://dx.doi.org/10.3390/cells11010130.

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Smoldering multiple myeloma is a heterogeneous asymptomatic precursor to multiple myeloma. Since its identification in 1980, risk stratification models have been developed using two main stratification methods: clinical measurement-based and genetics-based. Clinical measurement models can be subdivided in three types: baseline measurements (performed at diagnosis), evolving measurements (performed over time during follow-up appointments), and imaging (for example, magnetic resonance imaging). Genetic approaches include gene expression profiling, DNA/RNA sequencing, and cytogenetics. It is impo

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4

Marchegiani, Giovanni, Stefano Andrianello, Tommaso Pollini, et al. "Natural History of Intraductal Papillary Mucinous Neoplasm of the Pancreas Reappraisal of the Indolent Precursor of Pancreatic Cancer." Journal of the American College of Surgeons 227, no. 4 (2018): e37-e38. http://dx.doi.org/10.1016/j.jamcollsurg.2018.08.099.

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5

Purdy, Adam, Firas Ido, and Deborah Stahlnecker. "Diffuse Idiopathic Pulmonary Neuroendocrine Cell Hyperplasia (DIPNECH): A Case of Indolent Pulmonary Nodules Diagnosed with Robotic-Assisted Navigational Bronchoscopy." Case Reports in Pulmonology 2021 (December 10, 2021): 1–4. http://dx.doi.org/10.1155/2021/6312296.

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Diffuse idiopathic pulmonary neuroendocrine cell hyperplasia (DIPNECH) is an atypical pulmonary disorder with limited understanding. Given the rare nature of this disease, it is essential to obtain adequate tissue pathology to confirm the diagnosis. This disease is mainly diagnosed in middle-aged, nonsmoking females, and it is now accepted as a precursor lesion to pulmonary carcinoid tumors. DIPNECH presents with characteristic radiographic and histologic findings, but its diagnosis, management, and prognosis are often underrecognized and poorly understood. Those with symptoms may present with

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6

Garcia-Montero, Andres C., Maria Jara-Acevedo, Ivan Alvarez-Twose, et al. "KIT D816V Mutation Positive Bone Marrow Mesenchymal Stem Cells in Indolent Systemic Mastocytosis Are Associated with Disease Progression." Blood 126, no. 23 (2015): 4058. http://dx.doi.org/10.1182/blood.v126.23.4058.4058.

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Abstract PURPOSE: Multilineageinvolvement of bone marrow (BM) hematopoiesis by the somatic KIT D816V mutation is present in a subset of adult indolent systemic mastocytosis (ISM) patients in association with a poorer prognosis. Here we investigated the potential involvement of BM mesenchymal stem cells (MSC) from ISM patients by the KIT D816V mutation and its potential impact on disease progression and outcome. METHODS: The KIT D816V mutation was investigated in highly-purified BM MSC and other BM cell populations from 83 ISM patients followed for a median of 116 months. MC clonality was furth

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7

Marchegiani, Giovanni, Stefano Andrianello, Tommaso Pollini, et al. "The natural history of intraductal papillary mucinous neoplasms of the pancreas: Reappraisal of the indolent precursor of pancreatic cancer." Pancreatology 18, no. 4 (2018): S2—S3. http://dx.doi.org/10.1016/j.pan.2018.05.011.

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8

Marchegiani, G., S. Andrianello, T. Pollini, et al. "The natural history of intraductal papillary mucinous neoplasm of the pancreas: Reappraisal of the indolent precursor of pancreatic cancer." HPB 20 (September 2018): S195. http://dx.doi.org/10.1016/j.hpb.2018.06.051.

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9

López, Cristina, Pablo Mozas, Armando López-Guillermo, and Sílvia Beà. "Molecular Pathogenesis of Follicular Lymphoma: From Genetics to Clinical Practice." Hemato 3, no. 4 (2022): 595–614. http://dx.doi.org/10.3390/hemato3040041.

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Follicular lymphoma (FL), a generally indolent disease that derives from germinal center (GC) B cells, represents around 20–25% of all new lymphomas diagnosed in Western countries. The characteristic t(14;18)(q32;q21) translocation that places the BCL2 oncogene under control of the immunoglobulin heavy-chain enhancer occurs in pro- or pre-B cells. However, additional secondary alterations are required for the development of overt FL, which mainly affects genes involved in epigenetic and transcriptional regulation, signaling and B cell differentiation, the BCR/NF-κB pathway, and proliferation/a

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10

Khanlari, Mahsa, and Jennifer R. Chapman. "Follicular lymphoma: updates for pathologists." Journal of Pathology and Translational Medicine 56, no. 1 (2022): 1–15. http://dx.doi.org/10.4132/jptm.2021.09.29.

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Follicular lymphoma (FL) is the most common indolent B-cell lymphoma and originates from germinal center B-cells (centrocytes and centroblasts) of the lymphoid follicle. Tumorigenesis is believed to initiate early in precursor B-cells in the bone marrow (BM) that acquire the t(14;18)(q32;q21). These cells later migrate to lymph nodes to continue their maturation through the germinal center reaction, at which time they acquire additional genetic and epigeneticabnormalities that promote lymphomagenesis. FLs are heterogeneous in terms of their clinicopathologic features. Most FLs are indolent and

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