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1

Hammerschmidt, R. "Inducing genes, inducing resistance." Physiological and Molecular Plant Pathology 81 (January 2013): v—vi. http://dx.doi.org/10.1016/j.pmpp.2013.01.005.

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2

Hammerschmidt, R. "Inducing resistance in different ways." Physiological and Molecular Plant Pathology 82 (April 2013): iii—iv. http://dx.doi.org/10.1016/s0885-5765(13)00026-x.

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3

Degani, Marcos H., and Luis V. A. Scalvi. "Subband mixing inducing negative resistance." Solid State Communications 86, no. 5 (1993): 301–4. http://dx.doi.org/10.1016/0038-1098(93)90377-y.

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4

O'Donnell, Will M. "Inducing ampicillin resistance in Escherichia coli." Transactions of the Kansas Academy of Science 106, no. 1 & 2 (2003): 99–104. http://dx.doi.org/10.1660/0022-8443(2003)106[0099:iariec]2.0.co;2.

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5

Hoffmann, M. R. "On inducing equations for vegetation resistance." Journal of Hydraulic Research 47, no. 2 (2009): 281–82. http://dx.doi.org/10.1080/00221686.2009.9521996.

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6

Warren, Amye, Katherine Hulse-Trotter, and Ernest C. Tubbs. "Inducing resistance to suggestibility in children." Law and Human Behavior 15, no. 3 (1991): 273–85. http://dx.doi.org/10.1007/bf01061713.

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7

Baptist, M. J., V. Babovic, J. Rodríguez Uthurburu, et al. "On inducing equations for vegetation resistance." Journal of Hydraulic Research 45, no. 4 (2007): 435–50. http://dx.doi.org/10.1080/00221686.2007.9521778.

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8

WANI, Shabir Hussain. "Inducing Fungus-Resistance into Plants through Biotechnology." Notulae Scientia Biologicae 2, no. 2 (2010): 14–21. http://dx.doi.org/10.15835/nsb224594.

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Plant diseases are caused by a variety of plant pathogens including fungi, and their management requires the use of techniques like transgenic technology, molecular biology, and genetics. There have been attempts to use gene technology as an alternative method to protect plants from microbial diseases, in addition to the development of novel agrochemicals and the conventional breeding of resistant cultivars. Various genes have been introduced into plants, and the enhanced resistance against fungi has been demonstrated. These include: genes that express proteins, peptides, or antimicrobial compounds that are directly toxic to pathogens or that reduce their growth in situ; gene products that directly inhibit pathogen virulence products or enhance plant structural defense genes, that directly or indirectly activate general plant defense responses; and resistance genes involved in the hypersensitive response and in the interactions with virulence factors. The introduction of the tabtoxin acetyltransferase gene, the stilbene synthase gene, the ribosome-inactivation protein gene and the glucose oxidase gene brought enhanced resistance in different plants. Genes encoding hydrolytic enzymes such as chitinase and glucanase, which can deteriorate fungal cell-wall components, are attractive candidates for this approach and are preferentially used for the production of fungal disease-resistant plants. In addition to this, RNA-mediated gene silencing is being tried as a reverse tool for gene targeting in plant diseases caused by fungal pathogens. In this review, different mechanisms of fungal disease resistance through biotechnological approaches are discussed and the recent advances in fungal disease management through transgenic approach are reviewed.
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9

Raza, Syed Abbas, Richard T. DeWitt, Herbert Chen, Thomas F. Warner, and Robert D. Blank. "CATECHOLAMINE EXCESS IN PHEOCHROMOCYTOMA INDUCING INSULIN RESISTANCE." Endocrine Practice 10, no. 2 (2004): 149–52. http://dx.doi.org/10.4158/ep.10.2.149.

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10

Manong’a, Theresa, and Adesh Kumar. "Effect of Growth Promoting and Resistance Inducing Chemicals on Yield Attributing Characteristics of Tomato." Journal of Pure and Applied Microbiology 11, no. 3 (2017): 1479–85. http://dx.doi.org/10.22207/jpam.11.3.32.

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11

Omer, Amal. "INDUCING PLANT RESISTANCE AGAINST SALINITY USING SOME RHIZOBACTERIA." Egyptian Journal of Desert Research 67, no. 1 (2017): 185–206. http://dx.doi.org/10.21608/ejdr.2017.6498.

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12

Holscher, Christian, Lidy van Aalten, and Calum Sutherland. "Anaesthesia generates neuronal insulin resistance by inducing hypothermia." BMC Neuroscience 9, no. 1 (2008): 100. http://dx.doi.org/10.1186/1471-2202-9-100.

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13

Ienaga, K., M. Kurohashi, K. Nakamura, T. Nakanishi, T. Ichii, and J. Konishi. "Naturally occurring heterocycles inducing drought resistance in plants." Experientia 44, no. 4 (1988): 356–57. http://dx.doi.org/10.1007/bf01961283.

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14

Fisette, Alexandre, Marc Lapointe, and Katherine Cianflone. "Obesity-inducing diet promotes acylation stimulating protein resistance." Biochemical and Biophysical Research Communications 437, no. 3 (2013): 403–7. http://dx.doi.org/10.1016/j.bbrc.2013.06.090.

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15

BRUNK, DOUG. "Tips to Avoid Inducing Resistance in CA-MRSA." Family Practice News 39, no. 7 (2009): 20. http://dx.doi.org/10.1016/s0300-7073(09)70262-2.

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16

Ienaga, Kazuharu, Ko Nakamura, Masaharu Kurohashi, Tetsu Nakanishi, and Takao Ichii. "Hydroxyproline-containing diketopiperazines inducing drought resistance in rice." Phytochemistry 29, no. 1 (1990): 35–39. http://dx.doi.org/10.1016/0031-9422(90)89005-t.

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17

Věchet, L., J. Martinková, M. Šindelářová, and L. Burketová. "Compounds of natural origin inducing winter wheat resistance to powdery mildew (Blumeria graminis f.sp. tritici)." Plant, Soil and Environment 51, No. 10 (2011): 469–75. http://dx.doi.org/10.17221/3619-pse.

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In laboratory and small-field experiments inducers of synthetic origin: benzothiadiazole (BTH), salicylic acid, and inducers of biological origin: glycine betaine, extracts prepared from oak bark (Quercus robur L.), Reynoutria sacchaliensis L., curcuma (Curcuma longa L.), ginger (Zingiber officinale Roscoe) were effective against powdery mildew on the winter wheat (cv. Kanzler) susceptible to this disease. All studied inducers slightly effected the synthesis of new proteins (PR-proteins) that were localized in extracellular space. The efficacy of inducers was long-term. The most effective inducer was BTH; its application produced a number of chlorotic blotches on leaves
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18

Kolupaev, Yu E., A. I. Kokorev, T. O. Yastreb, and E. I. Horielova. "Hydrogen peroxide as a signal mediator at inducing heat resistance in wheat seedlings by putrescine." Ukrainian Biochemical Journal 91, no. 6 (2019): 103–11. http://dx.doi.org/10.15407/ubj91.06.103.

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19

Qi, Lin, Yuan Ge, Tian Xia, et al. "Rare earth oxide nanoparticles promote soil microbial antibiotic resistance by selectively enriching antibiotic resistance genes." Environmental Science: Nano 6, no. 2 (2019): 456–66. http://dx.doi.org/10.1039/c8en01129j.

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This study demonstrates that rare earth oxide nanoparticles can enhance soil microbial antibiotic resistance by inducing the enrichment and spread of antibiotic resistance genes in soil microbial communities.
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20

Turk, Thomas A. "Takeover Resistance, Information Leakage, and Target Firm Value." Journal of Management 18, no. 3 (1992): 503–22. http://dx.doi.org/10.1177/014920639201800305.

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This study examines the effect of management responses to takeovers on the value of the targetfirm. Three categories of takeover responses are identified: auction-inducing resistance, competitionreducing resistance, and support. Auction-inducing resistance is shown to increase competition for control of the target firm. This increased competition leads to increased gains to the target firm during the takeover battle relative to gains obtained when the initial takeover offer is supported. Competition-reducing resistance is shown to have the opposite effect.
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21

Tarakanov, Rashit, Balzhima Shagdarova, Valery Varlamov, and Fevzi Dzhalilov. "Biocidal and resistance-inducing effects of chitosan on phytopathogens." E3S Web of Conferences 254 (2021): 05007. http://dx.doi.org/10.1051/e3sconf/202125405007.

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In connection with the of the law adoption on organic products in the Russian Federation, as well as the appearance of fungicide-resistant forms of phytopathogens, the search for new preparations of biological origin for plant protection from disease remains relevant. Chitosan is a promising substance obtained from shells of crustaceans, zygomycete fungi: on the one hand, it prevents the pathogen's penetration into the plant inducing a horizontal resistance, and on the other, it has a bactericidal effect against some bacteria and fungi. This article shows the biocidal effect of chitosan hydrolysate with a molecular weight of the main fraction of 33.7 kDa in relation to 5 phytopathogenic bacteria. The minimum bactericidal concentration ranged from 0.25 to 0.5%. It was noted that the substance acts more actively against Gram-negative bacteria than in relation to Gram-positive. Biological substance efficacy on artificial infectious background of cucumber downy mildew during preventive treatment amounted to the average of 55.4% compared to control with no treatment.
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22

Cheng, Xingkai, Xuejing Man, Zitong Wang, et al. "Fungicide SYP-14288 Inducing Multidrug Resistance in Rhizoctonia solani." Plant Disease 104, no. 10 (2020): 2563–70. http://dx.doi.org/10.1094/pdis-01-20-0048-re.

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Rhizoctonia solani is a widely distributed soilborne plant pathogen, and can cause significant economic losses to crop production. In chemical controls, SYP-14288 is highly effective against plant pathogens, including R. solani. To examine the sensitivity to SYP-14288, 112 R. solani isolates were collected from infected rice plants. An established baseline sensitivity showed that values of effective concentration for 50% growth inhibition (EC50) ranged from 0.0003 to 0.0138 μg/ml, with an average of 0.0055 ± 0.0030 μg/ml. The frequency distribution of the EC50 was unimodal and the range of variation factor (the ratio of maximal over minimal EC50) was 46.03, indicating that all wild-type strains were sensitive to SYP-14288. To examine the risk of fungicide resistance, 20 SYP-14288-resistant mutants were generated on agar plates amended with SYP-14288. Eighteen mutants remained resistant after 10 transfers, and their fitness was significantly different from the parental strain. All of the mutants grew more slowly but showed high virulence to rice plants, though lower than the parental strain. A cross-resistance assay demonstrated that there was a positive correlation between SYP-14288 and fungicides having or not having the same mode of action with SYP-14288, including fluazinam, fentin chloride, fludioxonil, difenoconazole, cyazofamid, chlorothalonil, and 2,4-dinitrophen. This result showed a multidrug resistance induced by SYP-14288, which could be a concern in increasing the spectrum of resistance in R. solani to commonly used fungicides.
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23

Peterhänsel, Christoph, and Thomas Lahaye. "Be fruitful and multiply: gene amplification inducing pathogen resistance." Trends in Plant Science 10, no. 6 (2005): 257–60. http://dx.doi.org/10.1016/j.tplants.2005.04.005.

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24

van Loon, L. C. "Manipulating the Plant’s innate immune system by inducing resistance." Phytoparasitica 36, no. 2 (2008): 103–6. http://dx.doi.org/10.1007/bf02981323.

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25

Liu, Ping. "Drug resistance and apoptosis-inducing strategies in cancer therapy." Biomedicine & Pharmacotherapy 62, no. 7 (2008): 426–27. http://dx.doi.org/10.1016/j.biopha.2008.06.020.

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26

Sandri, Marco. "FOXOphagy path to inducing stress resistance and cell survival." Nature Cell Biology 14, no. 8 (2012): 786–88. http://dx.doi.org/10.1038/ncb2550.

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27

Oliveira, Vinicius de Souza, Johnatan Jair de Paula Marchiori, Lusiane de Sousa Ferreira, et al. "Role of Biostimulants in Inducing Resistance for Phytosanitary Management." International Journal of Plant & Soil Science 36, no. 1 (2024): 112–19. http://dx.doi.org/10.9734/ijpss/2024/v36i14336.

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Biostimulants perform functions that improve plant metabolism and can positively interfere with processes such as respiration, photosynthesis, synthesis of nucleic acids and absorption of ions. Furthermore, they improve the roots’ ability to absorb nutrients. Thus, bistimulants can help to induce plant resistance by improving the nutritional capacity of plants, making them more tolerant to pathogen attack. Therefore, the use of biostimulants can have beneficial effects, providing an increase in plant productivity, whether through stimulating their physiology or controlling diseases.
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28

Lee Díaz, Ana Shein, Desiré Macheda, Haymanti Saha, Ursula Ploll, Dimitri Orine, and Arjen Biere. "Tackling the Context-Dependency of Microbial-Induced Resistance." Agronomy 11, no. 7 (2021): 1293. http://dx.doi.org/10.3390/agronomy11071293.

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Plant protection with beneficial microbes is considered to be a promising alternative to chemical control of pests and pathogens. Beneficial microbes can boost plant defences via induced systemic resistance (ISR), enhancing plant resistance against future biotic stresses. Although the use of ISR-inducing microbes in agriculture seems promising, the activation of ISR is context-dependent: it often occurs only under particular biotic and abiotic conditions, thus making its use unpredictable and hindering its application. Although major breakthroughs in research on mechanistic aspects of ISR have been reported, ISR research is mainly conducted under highly controlled conditions, differing from those in agricultural systems. This forms one of the bottlenecks for the development of applications based on ISR-inducing microbes in commercial agriculture. We propose an approach that explicitly incorporates context-dependent factors in ISR research to improve the predictability of ISR induction under environmentally variable conditions. Here, we highlight how abiotic and biotic factors influence plant–microbe interactions in the context of ISR. We also discuss the need to raise awareness in harnessing interdisciplinary efforts between researchers and stakeholders partaking in the development of applications involving ISR-inducing microbes for sustainable agriculture.
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29

Duguid, S. D., and A. L. Brûlé-Babel. "Inheritance of resistance to a necrosis- and chlorosis-inducing isolate from Race 1 of Pyrenophora tritici-repentis in spring wheat." Canadian Journal of Plant Science 81, no. 3 (2001): 519–25. http://dx.doi.org/10.4141/p00-037.

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Tan spot is a stubble-borne foliar disease of wheat (Triticum aestivum L.) caused by Pyrenophora tritici-repentis (Died.) Drechs. The potential for yield losses due to tan spot has increased with the adoption of conservation tillage practices. The main objective of this study was to determine the inheritance of resistance among seven wheat genotypes to the tan necrosis- and chlorosis-in ducing, race 1, isolate ASC1 (nec+ chl+), and the necrosis-inducing toxin, Ptr ToxA. Crosses were made between four resistant (Erik, ST6, 6B367, 6B1043) and three susceptible genotypes (Katepwa, BH1146, ST15). Parental, F1 and F2 populations were inoculated with ASC1 and infiltrated with Ptr ToxA under controlled environments. F2-derived F3 families were grown in the field and inoculated with ASC1. No reciprocal differences were observed. Resistance to the tan necrosis-inducing component of ASC1 and insensitivity to Ptr ToxA was controlled by a single recessive gene, whereas resistance to the chlorosis-inducing component of ASC1 was controlled by a single dominant gene. Genetic control of responses to each component (tan necrosis- or chlorosis-inducing) of ASC1 was independent. Lack of segregation among F2 progeny from crosses between resistant genotypes indicated that resistant genotypes carry at least one gene in common for resistance to ASC1. Key words: Triticum aestivum, Pyrenophora tritici-repentis, disease resistance, inheritance, Ptr ToxA, necrosis, chlorosis, toxin, tan spot, leaf spot
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30

Zhang, Chen, Xiyi Lu, Xinyin Liu, et al. "Carbonic Anhydrase IX Controls Vulnerability to Ferroptosis in Gefitinib-Resistant Lung Cancer." Oxidative Medicine and Cellular Longevity 2023 (January 31, 2023): 1–21. http://dx.doi.org/10.1155/2023/1367938.

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Acquired resistance to epidermal growth factor receptor tyrosine kinase inhibitors (EGFR-TKI, such as gefitinib) in lung cancer continues to be a major problem. Recent studies have shown the promise of ferroptosis-inducing therapy in EGFR-TKI resistant cancer, but have not been translated into clinical benefits. Here, we identified carbonic anhydrase IX (CA9) was upregulated in gefitinib-resistant lung cancer. Then we measured the cell viability, intracellular reactive oxygen species (ROS) levels, and labile iron levels after the treatment of ferroptosis inducer erastin. We found that CA9 confers resistance to ferroptosis-inducing drugs. Mechanistically, CA9 is involved in the inhibition of transferrin endocytosis and the stabilization of ferritin, leading to resistance to ferroptosis. Targeting CA9 promotes iron uptake and release, thus triggering gefitinib-resistant cell ferroptosis. Notably, CA9 inhibitor enhances the ferroptosis-inducing effect of cisplatin on gefitinib-resistant cells, thus eliminating resistant cells in heterogeneous tumor tissues. Taken together, CA9-targeting therapy is a promising approach to improve the therapeutic effect of gefitinib-resistant lung cancer by inducing ferroptosis.
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31

CĂTANĂ, Laura, Raul CĂTANĂ, Roxana CORA, Ştefan RĂILEANU, and Mihai CERNEA. "In Vitro Study of Benzimidazole Derivatives, Tetrahydropyrimidines and Macrocyclic Lactones Therapeutic Efficacy in Dog Hookworms." Bulletin of University of Agricultural Sciences and Veterinary Medicine Cluj-Napoca. Veterinary Medicine 75, no. 2 (2018): 199. http://dx.doi.org/10.15835/buasvmcn-vm:2018.0019.

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The study was conducted using faecal samples from 62 dogs. We tested the ovicidal and larvicidal effects of albendazole (ABZ), mebendazole (MBZ), fenbendazole (FBZ) and flubendazole (FLU) by Egg hatch assay (EHA) and larval development assay (LDA). For pyrantel (PYR) and selamectin (SEL) we tested the larvicidal effects by LDA. In all in vitro tests, benzimidazoles efficacy was low, with a high risk of inducing resistance phenomena. In EHA more than 50% of the hookworm eggs hatched, revealing a low efficacy of all tested benzimidazoles. The regression line was positive for all benzimidazoles, FBZ having the smallest value of the Y parameter (62.62), and lower risk of resistance. When testing the larvicidal effects, a superior efficacy of benzimidazoles was observed. The lowest MIC was for MBZ (0.8672μg/ml). ABZ had a very poor effect (8.46750 μg/ml). The Y parameter showed a lower risk of inducing resistance for MBZ (Y= -64.14) and FBZ (Y= -27.89). Pyrantel and Selamectin were very effective, presenting also a very low risk of inducing resistance phenomena. For PYR and SEL, MIC was 0.2131 μg/ml and 2.7921 μg/ml, respectively. The Y parameter was -448.37 for PYR and -62.74 for SEL, with minimal risk of inducing the adaptive phenomena.
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32

Muthulakshmi, P., P. Narayanasamy, and H. Koganezawa. "Serological evidence for inducing resistance to rice tungro viruses using antiviral principles." International Rice Research Notes 21, no. 2-3 (1996): 77. https://doi.org/10.5281/zenodo.6880635.

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This article 'Serological evidence for inducing resistance to rice tungro viruses using antiviral principles' appeared in the International Rice Research Notes series, created by the International Rice Research Institute (IRRI) to expedite communication among scientists concerned with the development of improved technology for rice and rice-based systems. The series is a mechanism to help scientists keep each other informed of current rice research findings. The concise scientific notes are meant to encourage rice scientists to communicate with one another to obtain details on the research reported.
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33

Qin, Xuan, Xiaodong Shi, Licheng Tu, et al. "Autophagy inducing cyclic peptides constructed by methionine alkylation." Chemical Communications 55, no. 29 (2019): 4198–201. http://dx.doi.org/10.1039/c9cc01027k.

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34

Acharya, R., P. Patra, N. Chakraborty, N. S. Gupta, and K. Acharya. "Footprint of Nitric oxide in induced systemic resistance." NBU Journal of Plant Sciences 7, no. 1 (2013): 55–61. http://dx.doi.org/10.55734/nbujps.2013.v07i01.008.

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Nitric oxide (NO) is a potent signaling molecule with diverse physiological functions in plants. Several rhizobacterial strains may have capacity to induce systemic resistance in (ISR) plants but how far the biochemical mechanisms in which No participates in this signaling pathway is still an open question. The present study have shown in Pseudomonas aeruginosa WS-1 mediated ISR inducing system in Catharanthus roseus induces defense enzyme and phenolics and also showed a two fold increase in NO production when challenge with Alternaria alternata. Furthermore, NO donor treatment in the host produced same defense molecules in a comparable manner. From those observations it is suggested that NO might have possible signaling role in ISR during crosstalk between the ISR inducing agent and pathogen within the host system.
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35

Li, Jingwen, Anbang Li, Yupeng Li, et al. "Preparation of Chitooligosaccharides with Specific Sequence Arrangement and Their Effect on Inducing Salt Resistance in Wheat Seedlings." Polymers 17, no. 9 (2025): 1194. https://doi.org/10.3390/polym17091194.

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Chitooligosaccharides (COS) exhibits good activity of inducing plant resistance, but the structure–activity relationship is still unclear. In this study, chitin oligosaccharides (CHOS) with a degree of polymerization (DP) of 2~6 were used as raw materials. Three deacetylases (NodB, VcCOD, and ArCE4A) were employed to prepare three different sequence-arranged COSs, namely N-COS, C-COS, and A-COS, and their structures were characterized by infrared spectroscopy, high-performance liquid chromatography, and mass spectrometry. Further studies were conducted on inducing the plant salt resistance of the three different sequence-arranged COSs on wheat seedlings. The results showed a sequence-dependent effect of COS inducing plant salt resistance. Among them, A-COS exhibited the best activity. When sprayed at a concentration of 10 mg/L on wheat seedlings under salt stress for 3 days, the leaf length of the wheat seedlings sprayed with A-COS was recovered, and the wet mass and dry mass were recovered by 20.40% and 6.64%, respectively. Following the enhancement of proline accumulation, the malondialdehyde content decreased by 34.75%, and the Na+/K+ ratio also exhibited a significant reduction, thereby alleviating salt stress-induced damage. This study was the first to demonstrate the effect of COS with specific sequences on inducing plant salt resistance, providing a theoretical basis for the development of a new generation of efficient COS plant biostimulator.
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36

Shafie, Radwa, Aly Hamed, and Hany El-Sharkawy. "Inducing Systemic Resistance against Cucumber Mosaic Cucumovirus using Streptomyces spp." Egyptian Journal of Phytopathology 44, no. 1 (2016): 127–42. http://dx.doi.org/10.21608/ejp.2016.91931.

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37

Seo, Pil Joon, and Chung-Mo Park. "Cuticular wax biosynthesis as a way of inducing drought resistance." Plant Signaling & Behavior 6, no. 7 (2011): 1043–45. http://dx.doi.org/10.4161/psb.6.7.15606.

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38

Zhang, Song-Fa, Xin-Yu Wang, Zhi-Qin Fu, et al. "TXNDC17 promotes paclitaxel resistance via inducing autophagy in ovarian cancer." Autophagy 11, no. 2 (2015): 225–38. http://dx.doi.org/10.1080/15548627.2014.998931.

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39

MEZIANE, HAMID, IENTSE VAN DER SLUIS, LEENDERT C. VAN LOON, MONICA HÖFTE, and PETER A. H. M. BAKKER. "Determinants ofPseudomonas putidaWCS358 involved in inducing systemic resistance in plants." Molecular Plant Pathology 6, no. 2 (2005): 177–85. http://dx.doi.org/10.1111/j.1364-3703.2005.00276.x.

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40

Hiramoto, T., R. Tobimatsu, N. Abe, et al. "Signal Molecules Inducing Systemic Resistance and Susceptibility in Barley Seedlings." Journal of Phytopathology 143, no. 1 (1995): 47–51. http://dx.doi.org/10.1111/j.1439-0434.1995.tb00199.x.

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41

Van der Ent, Sjoerd, Saskia C. M. Van Wees, and Corné M. J. Pieterse. "Jasmonate signaling in plant interactions with resistance-inducing beneficial microbes." Phytochemistry 70, no. 13-14 (2009): 1581–88. http://dx.doi.org/10.1016/j.phytochem.2009.06.009.

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42

Dankel, Scott J., Kevin T. Mattocks, Matthew B. Jessee, et al. "Frequency: The Overlooked Resistance Training Variable for Inducing Muscle Hypertrophy?" Sports Medicine 47, no. 5 (2016): 799–805. http://dx.doi.org/10.1007/s40279-016-0640-8.

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43

Lehrer, Axel T., Dereje Dugassa-Gobena, Stefan Vidal, and Karlheinz Seifert. "Transport of Resistance - Inducing Sterols in Phloem Sap of Barley§." Zeitschrift für Naturforschung C 55, no. 11-12 (2000): 948–52. http://dx.doi.org/10.1515/znc-2000-11-1216.

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After root application of [7α-3H]-7β-hydroxysitosterol and [3α,6β-3H2]-6α-hydroxylathosterol these sterols could be detected in the leaves and phloem sap feeding aphids. These results imply that the phloem sap is a sterol transport system in barley plants.
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44

Mousa, Alaa Raad, and Aalaa Khudair Hassan. "INDUCING SYSTEMIC ACQUIRED RESISTANCE IN PEPPER PLANTS AGAINST RHIZOCTONIA SOLANI." Iraqi Journal of Market Research and Consumer Protection 15, no. 1 (2023): 92–105. http://dx.doi.org/10.28936/jmracpc15.1.2023.(9).

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This study was initiated to assess the efficacy of some biological materials separately or mixed to control Rhizoctonia root rot disease caused by the fungus Rhizoctonia solani. In vitro efficacy assessment showed; glutathione could inhabit fungal growth up to 100% at concentration 3000 mg/L. Whereas, the bacterium Azospirillum brasilense scored 78.63% inhibitory at 10-5 concentration. The fungal bio-agent Trichoderma viride scored 1.33 highest antagonistic activity 5 days of inoculation on PDA medium. Under greenhouse conditions, (Tr + Az + G+ R. solani) and (Tr + G+ R. solani) combination treatments could decrease R.solani infectivity and disease severity up to 0.00% compared to 73.33 and 68.33% for control treatment, respectively. Similarly, these two treatments could induce systemic acquired resistance (SAR) when scored the highest polyphenol oxidase (PPO) activity 6 and 12d of pathogenic fungus inoculation compared to healthy control. They scored 82.14 and 67.07, 78.12 and 65.33 absorbance increase rate (AIR)/min/g fresh leaf weight, respectively, compared to 41.67, 40.08 for AIR/min/g fresh leaf weight, respectively, for healthy control. Amongst other treatments, (Az + R.solani) scored 11.553% highest protein content compared to 9.433% for healthy control.
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45

Amin, Hadeer Hammad, Abdelanser Badaey Elsayed, Hanafey Farouk Maswada, and Nabil Ibrahim Elsheery. "Enhancing Sugar Beet Plant Health with Zinc Nanoparticles: A Sustainable Solution for Disease Management." Journal of Soil, Plant and Environment 2, no. 1 (2023): 1–20. http://dx.doi.org/10.56946/jspae.v2i1.129.

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Sugar beet (Beta vulgaris L.) is susceptible to various diseases, especially powdery mildew, caused by Erysiphe betae. Using nanotechnology in agriculture could revolutionize the sector by providing new tools for fast disease diagnosis and disease resistance. This study investigated the potential of Zn nanoparticles in inducing resistance to powdery mildew in sugar beet plants through two experiments. The first experiment assessed the susceptibility of sugar beet cultivars to powdery mildew, with Puma being the most resistant and Top being the most susceptible. The second experiment examined the impact of Zn NPs in inducing resistance to powdery mildew. Zinc-oxide nanoparticles (ZN) and zinc sulfate (ZS) at concentrations of 100, 50 and 10 ppm were used as foliar applications. The results showed that most treatments significantly increased levels of chlorophyll a, b, and total chlorophyll, total soluble sugars, endogenous H2O2, and activity of peroxidase (POD) and polyphenol oxidase (PPO), while reducing the severity of powdery mildew disease, lipid peroxidation (MDA), phenolics concentrations and catalase activity, especially Zn at concentrations of 100 and 50 ppm compared to infected control. The physiological role of Zn NPs in inducing resistance against powdery mildew disease is attributed to the production and accumulation of reactive oxygen species (ROS) and oxidative reactions of phenolic compounds catalyzed by PPO and/or POD. Our results suggested that ZnO nanoparticles at 100 and 50 ppm can be used as a foliar spray to reduce the harmful impacts of biotic stress caused by E. betae in sugar beet plants by inducing resistance to the pathogen.
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46

Lenk, Miriam, Marion Wenig, Kornelia Bauer, et al. "Pipecolic Acid Is Induced in Barley upon Infection and Triggers Immune Responses Associated with Elevated Nitric Oxide Accumulation." Molecular Plant-Microbe Interactions® 32, no. 10 (2019): 1303–13. http://dx.doi.org/10.1094/mpmi-01-19-0013-r.

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Pipecolic acid (Pip) is an essential component of systemic acquired resistance, priming resistance in Arabidopsis thaliana against (hemi)biotrophic pathogens. Here, we studied the potential role of Pip in bacteria-induced systemic immunity in barley. Exudates of barley leaves infected with the systemic immunity–inducing pathogen Pseudomonas syringae pv. japonica induced immune responses in A. thaliana. The same leaf exudates contained elevated Pip levels compared with those of mock-treated barley leaves. Exogenous application of Pip induced resistance in barley against the hemibiotrophic bacterial pathogen Xanthomonas translucens pv. cerealis. Furthermore, both a systemic immunity–inducing infection and exogenous application of Pip enhanced the resistance of barley against the biotrophic powdery mildew pathogen Blumeria graminis f. sp. hordei. In contrast to a systemic immunity-inducing infection, Pip application did not influence lesion formation by a systemically applied inoculum of the necrotrophic fungus Pyrenophora teres. Nitric oxide (NO) levels in barley leaves increased after Pip application. Furthermore, X. translucens pv. cerealis induced the accumulation of superoxide anion radicals and this response was stronger in Pip-pretreated compared with mock-pretreated plants. Thus, the data suggest that Pip induces barley innate immune responses by triggering NO and priming reactive oxygen species accumulation.
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Wang, Y., K. G. Jia, H. J. Xing, et al. "Interaction of SENP6 with PINK1 promotes temozolomide resistance in neuroglioma cells via inducing the mitophagy." Молекулярная биология 58, no. 1 (2024): 126–29. http://dx.doi.org/10.31857/s0026898424010112.

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Temozolomide resistance is a major cause of recurrence and poor prognosis in neuroglioma. Recently, growing evidence has suggested that mitophagy is involved in drug resistance in various tumor types. However, the role and molecular mechanisms of mitophagy in temozolomide resistance in glioma remain unclear. In this study, mitophagy levels in temozolomide-resistant and -sensitive cell lines were evaluated. The mechanisms underlying the regulation of mitophagy were explored through RNA sequencing, and the roles of differentially expressed genes in mitophagy and temozolomide resistance were investigated. We found that mitophagy promotes temozolomide resistance in glioma. Specifically, small ubiquitin-like modifier specific protease 6 (SENP6) promoted temozolomide resistance in glioma by inducing mitophagy. Protein-protein interactions between SENP6 and the mitophagy executive protein PTEN-induced kinase 1 (PINK1) resulted in a reduction in small ubiquitin-like modifier 2 (SUMO2)ylation of PINK1, thereby enhancing mitophagy. Our study demonstrates that by inducing mitophagy, the interaction of SENP6 with PINK1 promotes temozolomide resistance in glioblastoma. Therefore, targeting SENP6 or directly regulating mitophagy could be a potential and novel therapeutic targets for reversing temozolomide resistance in glioma.
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48

Schabdach, H., S. Johne, U. Steiner, and K. Seifert. "Plant Disease Resistance Inducing Activity of 7-Oxo- and 7-Hydroxysterols." Zeitschrift für Naturforschung C 50, no. 3-4 (1995): 257–62. http://dx.doi.org/10.1515/znc-1995-3-415.

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The 7-oxosterols 1-2 and the 7-hydroxysterols 3-6 induce resistance toward the fungal pathogens Puccinia striiformis West, and Puccinia hordei Otth in barley and wheat. Primary leaves of the plants were sprayed with solutions of the compounds ( 10-4 mol/l in 1% aqu. ethanol) followed, 2 days later, by challenge inoculation with the fungal pathogens. The results indicate that 7α- and 7β-hydroxylated epimers of β-sitosterol and cholesterol show the highest value of induced resistance (39-49% reduction of infection sites). No enhanced resistance toward the fungi Erysiphe graminis DC f.sp. tricitic and hordei and Cochliobolus sativus Ito & Kuribayashi was observed.
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He, Hongyang, Tiejun Li, Yuxiang Lin, Shuya Yang, Maojing Li, and Jinyan Pan. "Memory Performance Enhancement by Inducing Conductive Channel via Doping." Journal of Physics: Conference Series 2566, no. 1 (2023): 012131. http://dx.doi.org/10.1088/1742-6596/2566/1/012131.

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Abstract Due to the excellent nonvolatile resistance characteristics demonstrated by hafnium oxide, it is the potential to facilitate the use of resistive random access memory. In this paper, an ingenious method using doping to locate conductive channels is presented to improve the stable rheostatic performance of HfO2-based rheostatic memory. Metal particles are located to enhance the electric field locally to spur on conductive filaments in situ so that the resistive parameters Vset and Vreset are reduced, the relative fluctuation value (standard deviation/mean) of the Vset is reduced from 22.57% to 18.16%, and that of Vreset is reduced from 19.59% to 16.77%. Consequently, the device gains more stable resistance switching with a larger resistive window.
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50

Susiana, Purwantisari, Priyatmojo Achmadi, P. Sancayaningsih Retno, S. Kasiamdari Rina, and Budihardjo Kadarwati. "The Resistance of Potatoes by Application of Trichoderma viride Antagonists Fungus." E3S Web of Conferences 73 (2018): 06014. http://dx.doi.org/10.1051/e3sconf/20187306014.

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Leaf blight disease caused by pathogenic fungus Phytophthora infestans is the major disease in potato that can reduce its production up to 100%. The use of biological agent Trichoderma viride as an inducing potato resistance against leaf blight disease has been considered potential method. The purpose of this study was to evaluate the use of biological agent Trichoderma viride in inducing potato plant resistance. The parameters observed were the growth of the potato plant and leaf blight intensity. Experimental research with complete randomized design with 6 treatments was applied. The results showed that the application of Trichoderma viride could reduce the intensity of leaf blight disease and increase the growth of the potato plant. Trichoderma viride application could improve the systemic resistance of potato plants.
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