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Journal articles on the topic 'Induction 1'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 May 2024

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1

Hutter, H., and R. Schnabel. "glp-1 and inductions establishing embryonic axes in C. elegans." Development 120, no. 7 (1994): 2051–64. http://dx.doi.org/10.1242/dev.120.7.2051.

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Two successive inductions specify blastomere identities, that is complex cell lineages and not specific tissues, in a major part of the early C. elegans embryo. The first induction acts along the anterior-posterior axis of the embryo and the second along the left-right axis. During the first induction a specific lineage program is induced in the posterior of the two AB blastomeres present in the four cell embryo. During the second induction, almost all of the left-right differences of the embryo are specified by interactions between a single signalling blastomere, MS, and the AB blastomeres th

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2

Goldfield, Joseph A. "Rapnotic Induction No. 1." Journal of Systemic Therapies 12, no. 2 (1993): 89–91. http://dx.doi.org/10.1521/jsyt.1993.12.2.89.

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3

Oh, Myoung-don, Debra P. Merrill, and Martin S. Hirsch. "Induction and Maintenance Treatment Regimens for HIV-1 Infection in Vitro." Antiviral Therapy 4, no. 1 (1999): 29–34. http://dx.doi.org/10.1177/135965359900400104.

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Can aggressive anti-human immunodeficiency virus (HIV) induction regimens be simplified after sufficient virus suppression is achieved? In vitro studies were conducted to evaluate the hypothesis that aggressive induction regimens could be followed by less aggressive maintenance regimens. A clinical HIV-1 isolate and lymphoblastoid cell line (H9) were employed. Virus multiplicities were varied, as were drug inhibitory concentrations (IC90, IC99) and induction periods (1, 2 and 3 weeks) of a three-drug regimen (zidovudine plus lamivudine and indinavir), following which maintenance regimens (no d

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4

Hawa, M. I., and R. D. G. Leslie. "Early induction of type 1 diabetes." Clinical & Experimental Immunology 126, no. 2 (2001): 181–83. http://dx.doi.org/10.1046/j.1365-2249.2001.01724.x.

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5

Li Volti, Giovanni, and Paolo Murabito. "Pharmacologic Induction of Heme Oxygenase-1." Critical Care Medicine 42, no. 8 (2014): 1967–68. http://dx.doi.org/10.1097/ccm.0000000000000458.

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6

Li, Cheng, Parastu Hossieny, Ben J. Wu, Abdelqader Qawasmeh, Konstanze Beck, and Roland Stocker. "Pharmacologic Induction of Heme Oxygenase-1." Antioxidants & Redox Signaling 9, no. 12 (2007): 2227–40. http://dx.doi.org/10.1089/ars.2007.1783.

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7

Balen, Adam. "1 Endocrine methods of ovulation induction." Baillière's Clinical Obstetrics and Gynaecology 12, no. 4 (1998): 521–39. http://dx.doi.org/10.1016/s0950-3552(98)80049-7.

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8

Calame, Kathryn. "Activation-dependent induction of Blimp-1." Current Opinion in Immunology 20, no. 3 (2008): 259–64. http://dx.doi.org/10.1016/j.coi.2008.04.010.

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9

Hatzikiriakou, Kostas. "Algebraic disguises ofΣ 1 0 induction". Archive for Mathematical Logic 29, № 1 (1989): 47–51. http://dx.doi.org/10.1007/bf01630810.

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10

Spain-Santana, Tammy A., Stephanie Marglin, Francis A. Ennis, and Alan L. Rothman. "MIP-1? and MIP-1? induction by dengue virus." Journal of Medical Virology 65, no. 2 (2001): 324–30. http://dx.doi.org/10.1002/jmv.2037.

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11

Farshchi Zarabi, Sara, Steven M. Chan, Vikas Gupta, et al. "Remissions after Third Induction Chemotherapy for Primary Non-Responders with Acute Myeloid Leukemia (AML) Are Uncommon and Short-Lived." Blood 128, no. 22 (2016): 2800. http://dx.doi.org/10.1182/blood.v128.22.2800.2800.

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Abstract The outcome of adult patients with AML who are primary non-responders to two courses of induction chemotherapy is poor. However, the utility of a 3rd induction for a select subgroup of these patients is uncertain. Here, we evaluated the rates of response and survival after a 3rd course of induction chemotherapy for primary non-responders with AML. We identified 98 patients from the Princess Margaret Cancer Centre between May 1999 and March 2015 who were non-responders to induction and reinduction chemotherapy. No-response to re-induction chemotherapy was defined according to the Revis

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12

Lutz, Justin D., Brian J. Kirby, Lu Wang, et al. "Cytochrome P450 3A Induction Predicts P-glycoprotein Induction; Part 1: Establishing Induction Relationships Using Ascending Dose Rifampin." Clinical Pharmacology & Therapeutics 104, no. 6 (2018): 1182–90. http://dx.doi.org/10.1002/cpt.1073.

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13

Shao, Ningsheng, YuLi Chai, Jian-qi Cui та ін. "Induction of apoptosis by Elk-1 and ΔElk-1 proteins". Oncogene 17, № 4 (1998): 527–32. http://dx.doi.org/10.1038/sj.onc.1201931.

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14

Osman, Hanan, Claire Murigande, Anne Nadakal, and Alessandro M. Capponi. "Repression of DAX-1 and Induction of SF-1 Expression." Journal of Biological Chemistry 277, no. 43 (2002): 41259–67. http://dx.doi.org/10.1074/jbc.m206595200.

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15

Agnez-Lima, Lucymara F., Paolo Di Mascio, Bruce Demple, and Carlos F. M. Menck. "Singlet Molecular Oxygen Triggers the soxRS Regulon of Escherichia coli." Biological Chemistry 382, no. 7 (2001): 1071–75. http://dx.doi.org/10.1515/bc.2001.134.

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AbstractThe electronically excited molecular oxygen (singlet oxygen, [1]O[2]) can be detrimental to cells in several ways, although recent reports indicate that it may play a role as an intercellular signal in eukaryotes. Here we present evidence that [1]O[2], generated by thermodissociation of disodium 3,3(1,4-naphthylidene) diproprionate endoperoxide, activates transcription of genes of the soxRS regulon, and that this induction is paralleled by induction of a soxS::lacZ operon fusion. The inductions were dependent on a functional soxR gene. These data imply that protective responses, such a

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16

Agrawal, Sudhir, and R. Russell Martin. "Was Induction of HIV-1 Through TLR9?" Journal of Immunology 171, no. 4 (2003): 1621–22. http://dx.doi.org/10.4049/jimmunol.171.4.1621.

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17

Devadas, Krishnakumar, Neil J. Hardegen, Larry M. Wahl, et al. "Mechanisms for Macrophage-Mediated HIV-1 Induction." Journal of Immunology 173, no. 11 (2004): 6735–44. http://dx.doi.org/10.4049/jimmunol.173.11.6735.

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18

Lee, Joshua A. "Induction of Adventitious Shoots in Cotton 1." Crop Science 27, no. 2 (1987): 349–50. http://dx.doi.org/10.2135/cropsci1987.0011183x002700020046x.

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19

Simpson, Stephen G. "Baire Categoricity and $\Sigma^{0}_{1}$ -Induction." Notre Dame Journal of Formal Logic 55, no. 1 (2014): 75–78. http://dx.doi.org/10.1215/00294527-2377887.

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20

Okamoto, Isao. "Peculiar Black-Hole Unipolar Induction: Fig. 1." Publications of the Astronomical Society of Japan 64, no. 3 (2012): 50. http://dx.doi.org/10.1093/pasj/64.3.50.

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21

Imai, Kenichi, Kenji Nakata, Kazuaki Kawai, et al. "Induction ofOGG1Gene Expression by HIV-1 Tat." Journal of Biological Chemistry 280, no. 29 (2005): 26701–13. http://dx.doi.org/10.1074/jbc.m503313200.

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22

Tan, Shulian, Yang Li, Jinxing Xia, et al. "Type 1 diabetes induction in humanized mice." Proceedings of the National Academy of Sciences 114, no. 41 (2017): 10954–59. http://dx.doi.org/10.1073/pnas.1710415114.

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23

Löfstedt, Tobias, Annika Jögi, Mikael Sigvardsson, et al. "Induction ofID2Expression by Hypoxia-inducible Factor-1." Journal of Biological Chemistry 279, no. 38 (2004): 39223–31. http://dx.doi.org/10.1074/jbc.m402904200.

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24

Kawai, Keiichi, Hiroyuki Horitsu, Kazuhiro Hamada, and Masahiro Watanabe. "Induction of Cadmium-resistance ofPseudomonas putidaGAM-1." Agricultural and Biological Chemistry 54, no. 6 (1990): 1553–55. http://dx.doi.org/10.1080/00021369.1990.10870178.

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25

Curlin, Marcel E., Timothy Wilkin, and John Mittler. "Induction–maintenance therapy for HIV-1 infection." Future HIV Therapy 2, no. 2 (2008): 175–85. http://dx.doi.org/10.2217/17469600.2.2.175.

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26

Motterlini, Roberto, Roberta Foresti, Rekha Bassi, Vittorio Calabrese, James E. Clark, and Colin J. Green. "Endothelial Heme Oxygenase-1 Induction by Hypoxia." Journal of Biological Chemistry 275, no. 18 (2000): 13613–20. http://dx.doi.org/10.1074/jbc.275.18.13613.

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27

Dhawle, Ajay S. "Induction of Labor." Journal of South Asian Federation of Obstetrics and Gynaecology 4, no. 1 (2012): 0. http://dx.doi.org/10.5005/jsafog-4-1-vi.

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28

Nikiforov, V. N. "Magnetic induction hyperthermia." Russian Physics Journal 50, no. 9 (2007): 913–24. http://dx.doi.org/10.1007/s11182-007-0133-1.

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29

BI, Yongyi, Richard D. PALMITER, Kristi M. WOOD, and Qiang MA. "Induction of metallothionein I by phenolic antioxidants requires metal-activated transcription factor 1 (MTF-1) and zinc." Biochemical Journal 380, no. 3 (2004): 695–703. http://dx.doi.org/10.1042/bj20031677.

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Phenolic antioxidants, such as tBHQ [2,5-di-(t-butyl)-1,4-hydroquinone], induce Mt1 (metallothionein 1) gene expression and accumulation of MT protein. Induction of Mt1 mRNA does not depend on protein synthesis, and correlates with oxidation–reduction functions of the antioxidants. In the present study, we analysed the biochemical pathway of the induction. Induction depends on the presence of MTF-1 (metal-activated transcription factor 1), a transcription factor that is required for metal-induced transcription of Mt1, but does not require nuclear factor erythroid 2-related factor 2, a tBHQ-act

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30

Tannahill, C. L., S. A. Stevenot, E. Y. Eaker, J. E. Sallustio, H. S. Nick, and J. F. Valentine. "Regulation of superoxide dismutase in primary cultures of rat colonic smooth muscle cells." American Journal of Physiology-Gastrointestinal and Liver Physiology 272, no. 5 (1997): G1230—G1235. http://dx.doi.org/10.1152/ajpgi.1997.272.5.g1230.

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We have observed a rapid induction of manganese superoxide dismutase (MnSOD) in epithelial, neuronal, and smooth muscle cells (SMC) after acetic acid-induced colitis. To examine the regulation of MnSOD in the SMC more specifically, primary cultures of colonic SMC were developed by enzymatic digestion of the circular muscle layer from an adult rat. SMC were treated for 2-72 h with 0.5 microgram/ml Escherichia coli endotoxin [lipopolysaccharide (LPS)], 10 ng/ml tumor necrosis factor (TNF)-alpha, or 2 ng/ml interleukin-1 beta (IL-1 beta). Cotreatments were performed with IL-1 beta and 4 microM ac

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31

Lerman, Oren Z., Matthew R. Greives, Sunil P. Singh, et al. "Low-dose radiation augments vasculogenesis signaling through HIF-1–dependent and –independent SDF-1 induction." Blood 116, no. 18 (2010): 3669–76. http://dx.doi.org/10.1182/blood-2009-03-213629.

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Abstract The inflammatory response to ionizing radiation (IR) includes a proangiogenic effect that could be counterproductive in cancer but can be exploited for treating impaired wound healing. We demonstrate for the first time that IR stimulates hypoxia-inducible factor-1α (HIF-1α) up-regulation in endothelial cells (ECs), a HIF-1α–independent up-regulation of stromal cell–derived factor-1 (SDF-1), as well as endothelial migration, all of which are essential for angiogenesis. 5 Gray IR-induced EC HIF-1α and SDF-1 expression was greater when combined with hypoxia suggesting an additive effect.

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32

Losev, G. L., and V. A. Eltishchev. "Induction method of the solidification front localization." ВЕСТНИК ПЕРМСКОГО УНИВЕРСИТЕТА. ФИЗИКА, no. 1 (2023): 57–61. http://dx.doi.org/10.17072/1994-3598-2023-1-57-61.

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The paper describes the development and testing of an inductive sensor designed for determining the position of the solidification front of electricallyconductive melts. The operation of the sensor is based on the eddy current transducer principle. We compared induction and ultrasonic methods of localizing the solidification front of a gallium alloy on a model problem of directional solidification of liquid metal in a flat vertical layer. The principles of the induction method are described, its ap-plicability is shown, the measurement error has been found.

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33

Yan, Rui, Matijs van Meurs, Eliane R. Popa та ін. "Endothelial Interferon Regulatory Factor 1 Regulates Lipopolysaccharide-Induced VCAM-1 Expression Independent of NFκB". Journal of Innate Immunity 9, № 6 (2017): 546–60. http://dx.doi.org/10.1159/000477211.

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Sepsis is a severe systemic inflammatory response to infection. Endothelial activation and dysfunction play a critical role in the pathophysiology of sepsis and represent an important therapeutic target to reduce sepsis mortality. Interferon regulatory factor 1 (IRF-1) was recently identified as a downstream target of TNF-α-mediated signal transduction in endothelial cells. The aim of this study was to explore the importance of IRF-1 as a regulator of lipopolysaccharide (LPS)-induced endothelial proinflammatory activation. We found that renal IRF-1 was upregulated by LPS in vivo as well as in

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34

Devadas, Krishnakumar, and Subhash Dhawan. "Hemin Activation Ameliorates HIV-1 Infection via Heme Oxygenase-1 Induction." Journal of Immunology 176, no. 7 (2006): 4252–57. http://dx.doi.org/10.4049/jimmunol.176.7.4252.

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35

BULGER, EILEEN M. "INDUCTION OF HEME-OXYGENASE 1 (HO-1) AND THE PROINFLAMMATORY RESPONSE." Shock 21, Supplement (2004): 44. http://dx.doi.org/10.1097/00024382-200403001-00174.

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36

Turner, John L., Richard J. Trauger, Anne E. Daigle, and Dennis J. Carlo. "HIV-1 immunogen induction of HIV-1-specific delayed-type hypersensitivity." AIDS 8, no. 10 (1994): 1429–36. http://dx.doi.org/10.1097/00002030-199410000-00009.

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37

CORNELISSEN, A., and J. OVERDULVE. ": Induction of sexuality." Cell Biology International Reports 10, no. 9 (1986): 735–44. http://dx.doi.org/10.1016/0309-1651(86)90131-1.

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38

Parent, Ted. "NOTE ON INDUCTION." Think 12, no. 33 (2013): 37–39. http://dx.doi.org/10.1017/s1477175612000322.

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Some logic textbooks say, as if it were the received wisdom, that inductive arguments are partly defined by the thinker's intentions. The claim is that an inductive argument is one where the premises are intended to make the conclusion likely. This contrasts with a deductive argument, where the premises are intended to entail the conclusion. However, since entailing is one way of making more likely, a further way to distinguish induction is needed. The addition offered is that the premises are not intended to entail the conclusion. Taken together, the result is: (1) An argument is inductive if

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39

George, Eric M., Marietta Arany, Kathy Cockrell, Megan V. Storm, David E. Stec, and Joey P. Granger. "Induction of heme oxygenase-1 attenuates sFlt-1-induced hypertension in pregnant rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 301, no. 5 (2011): R1495—R1500. http://dx.doi.org/10.1152/ajpregu.00325.2011.

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Preeclampsia (PE) is one of the leading causes of fetal and maternal morbidity, affecting 5–10% of all pregnancies, and lacks an effective treatment. The exact etiology of the disorder is unclear, but placental ischemia has been shown to be a central causative agent. In response to placental ischemia, the antiangiogenic protein fms-like tyrosine kinase-1 (sFlt-1), a VEGF antagonist, and reactive oxygen species are secreted, leading to the maternal syndrome. One promising therapeutic approach to treat PE is through manipulation of the heme oxygenase-1 (HO-1) protein. It has been previously repo

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40

Chen, C. Y., L. W. Forman, and D. V. Faller. "Calcium-dependent immediate-early gene induction in lymphocytes is negatively regulated by p21Ha-ras." Molecular and Cellular Biology 16, no. 11 (1996): 6582–92. http://dx.doi.org/10.1128/mcb.16.11.6582.

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The induction of immediate-early (IE) response genes, such as egr-1, c-fos, and c-jun, occurs rapidly after the activation of T lymphocytes. The process of activation involves calcium mobilization, activation of protein kinase C (PKC), and phosphorylation of tyrosine kinases. p21(ras), a guanine nucleotide binding factor, mediates T-cell signal transduction through PKC-dependent and PKC-independent pathways. The involvement of p21(ras) in the regulation of calcium-dependent signals has been suggested through analysis of its role in the activation of NF-AT. We have investigated the inductions o

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41

Tripathi, G. "Thyroid Hormone-Induced Changes in Cytoplasmic and Mitochondrial Proteins of a Teleost." Zeitschrift für Naturforschung C 53, no. 1-2 (1998): 120–24. http://dx.doi.org/10.1515/znc-1998-1-221.

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AbstractThe effect of triiodothyronine (T3) on the cytoplasmic and mitochondrial protein contents were studied in the liver and skeletal muscle of a freshwater teleost. The fish exposed to thiouracil for 28 days showed 1.5-2 times reduction in the total protein contents of cytoplasmic and mitochondrial fractions. A single injection of T3 to thiouracil exposed fish caused the earliest induction in the liver and skeletal muscle mitochondrial protein and the skeletal muscle cytoplasmic protein at 12 hr of lapses. However, the initial induction in the cytoplasmic protein of the liver was observed

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42

De Simoni, M. G., M. Sironi, A. De Luigi, A. Manfridi, A. Mantovani, and P. Ghezzi. "Intracerebroventricular injection of interleukin 1 induces high circulating levels of interleukin 6." Journal of Experimental Medicine 171, no. 5 (1990): 1773–78. http://dx.doi.org/10.1084/jem.171.5.1773.

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IL-1 is known to have a central role in the induction of acute-phase response, and some of its activities (including induction of some acute-phase proteins) were reported to be mediated by an induction of IL-6. Administration to rats of 200 ng of human rIL-1 by intracerebroventricular injection resulted in a more marked induction of circulating IL-6 than the same dose of IL-1 administered systemically (intravenously or intraperitoneally). Induction of serum IL-6 by centrally administered IL-1 was also observed in hypophysectomized or adrenalectomized rats, suggesting that activation of the hyp

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43

Ratajczyk, Z. "Arithmetical transfinite induction and hierarchies of functions." Fundamenta Mathematicae 141, no. 1 (1992): 1–20. http://dx.doi.org/10.4064/fm-141-1-1-20.

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44

Hariz Santoso, Asfari, Rhezal Agung Ananto, Ahmad Hermawan, and Sri Wahyuni Dali. "Desain Motor Kapasitor Dua Fasa Simetris 1 kW." Elposys: Jurnal Sistem Kelistrikan 11, no. 1 (2024): 40–44. http://dx.doi.org/10.33795/elposys.v11i1.4507.

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Single phase induction motors are one of the most frequently used types of motors because of their economical, sturdy, simple and easy maintenance advantages. One application is for electric vehicles. When single phase induction motors are applied to electric vehicles there are problems related to torque regulation based on vehicle operating conditions where when operating on light road conditions the torque produced by the motor remains high. In this research, a single-phase induction motor was made to have the same proportion of torque produced by the main coil and auxiliary coil or a 1kW 10

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45

Martinelli, V., and G. Comi. "Induction versus escalation therapy." Neurological Sciences 26, S4 (2005): s193—s199. http://dx.doi.org/10.1007/s10072-005-0519-1.

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46

Turmambekov, T., and N. Mastenova. "Scientific discovery – electrodynamic induction." Q A Iasaýı atyndaǵy Halyqaralyq qazaq-túrіk ýnıversıtetіnіń habarlary (fızıka matematıka ınformatıka serııasy) 24, no. 1 (2023): 70–79. http://dx.doi.org/10.47526/2023-1/2524-0080.07.

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This article presents experimental evidence and a description of the scientific discovery «The phenomenon of electric current excitation in a conductor moving in an electrostatic field». The first class of effects I will show you are those caused by the electrostatic force. It is the force that controls the movement of atoms, collides, and generates heat and light energy. This force also causes atoms to combine in an infinite number of ways according to nature's fantastic designs, and to form all the amazing structures we see around us. According to Maxwell's theory, an eddy magnetic field sho

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47

Jaster, R., Y. Zhu, M. Pless, S. Bhattacharya, B. Mathey-Prevot, and A. D. D'Andrea. "JAK2 is required for induction of the murine DUB-1 gene." Molecular and Cellular Biology 17, no. 6 (1997): 3364–72. http://dx.doi.org/10.1128/mcb.17.6.3364.

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Cytokine receptors activate multiple signal transduction pathways, resulting in the induction of specific target genes. We have recently identified a hematopoietic cell-specific immediate-early gene, DUB-1, that encodes a growth-regulatory deubiquitinating enzyme. The DUB-1 gene contains a 112-bp enhancer element that is specifically induced by the beta c subunit of the interleukin-3 (IL-3) receptor. To investigate the mechanism of DUB-1 induction, we examined the effects of dominant-negative forms of JAK kinases, STAT transcription factors, and Raf-1 in transient transfection assays. In Ba/F3

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48

Wahl, S. M., G. L. Costa, M. Corcoran, L. M. Wahl, and A. E. Berger. "Transforming growth factor-beta mediates IL-1-dependent induction of IL-1 receptor antagonist." Journal of Immunology 150, no. 8 (1993): 3553–60. http://dx.doi.org/10.4049/jimmunol.150.8.3553.

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Abstract Transforming growth factor-beta (TGF-beta) is a potent immunomodulatory molecule that promotes inflammation through recruitment of monocytes and induction of IL-1 and other cytokines. These proinflammatory processes may be modulated by the ability of TGF-beta to induce de novo synthesis and secretion of an IL-1 receptor antagonist (IL-1ra) that binds to and blocks IL-1 receptors. In this study, we show that the addition of TGF-beta to human peripheral blood monocytes induced the sequential transcription of the 1.8-kb mRNA for IL-1 beta and for IL-1ra. The expression of detectable mRNA

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49

Takacs, L., J. A. Berzofsky, J. York-Jolley, T. Akahoshi, E. Blasi, and S. K. Durum. "IL 1 induction by murine T cell clones: detection of an IL 1-inducing lymphokine." Journal of Immunology 138, no. 7 (1987): 2124–31. http://dx.doi.org/10.4049/jimmunol.138.7.2124.

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Abstract T cell activation is widely believed to depend on interleukin 1 (IL 1) provided by antigen (Ag)-presenting cells (APC). Because IL 1 is not a constitutive product of APC, we examined the features of its production during the interaction of murine T cell clones and APC. We observed that IL 1 was detectable in supernatants of most myoglobin-specific T cell clones grown with APC and Ag. Two of these T cell clones induced exceptionally high levels of IL 1 in their supernatants, and these same clones demonstrated the unusual restriction to I-Ek, which is a low responder type for sperm whal

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50

&NA;. "Induction maintenance approach viable in HIV-1 infection." Inpharma Weekly &NA;, no. 1502 (2005): 13. http://dx.doi.org/10.2165/00128413-200515020-00035.

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